Relevant bibliographies by topics / Condition-dependent signalling

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Academic literature on the topic 'Condition-dependent signalling'

Author: Grafiati
Published: 6 September 2023

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Journal articles on the topic "Condition-dependent signalling"

1

LINDSTROM, L., J. J. AHTIAINEN, J. MAPPES, J. S. KOTIAHO, A. LYYTINEN, and R. V. ALATALO. "Negatively condition dependent predation cost of a positively condition dependent sexual signalling." Journal of Evolutionary Biology 19, no. 2 (March 2006): 649–56. http://dx.doi.org/10.1111/j.1420-9101.2005.01043.x.

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Kurtz, Joachim, Klaus Reinhold, and Leif Engqvist. "Immunosuppression under stress: necessary for condition-dependent signalling?" Trends in Ecology & Evolution 15, no. 10 (October 2000): 418–19. http://dx.doi.org/10.1016/s0169-5347(00)01969-8.

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David, Patrice, Tracey Bjorksten, Kevin Fowler, and Andrew Pomiankowski. "Condition-dependent signalling of genetic variation in stalk-eyed flies." Nature 406, no. 6792 (July 2000): 186–88. http://dx.doi.org/10.1038/35018079.

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4

Palchevska, O. L., V. V. Balatskyi, L. L. Macewicz, and O. O. Piven. "Cardiospecific deletion of β-catenin gene associated with an activity violation of signaling cascades involved in the development of myocardial hypertrophy." Visnik ukrains'kogo tovaristva genetikiv i selekcioneriv 15, no. 2 (February 28, 2018): 181–86. http://dx.doi.org/10.7124/visnyk.utgis.15.2.877.

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The aim of our study was to investigate the molecular mechanisms of hypertrophy response under cardiospecific β-catenin haploinsufficiency condition. Materials and methods. Studies were done with β-catenin condtional knockout mice (β-catflox/flox) and α-MHC-Cre-transgenic mice. To induce hypertrophy we used swimming test during 6 weeks. Using western-blot, we have analyzed the level of studied proteins. Results. It has been shown that the β-catenin haploinsufficiency is associated with increased signaling activity of MAPK, PI3-kinase-mTOR-dependent signaling cascades in both: with prolonged physical activity and without it. However, even with an increased activity of this signalling, β-catenin haploinsufficient mice expressed weaker hypertrophic response. Conclusions. The transcriptional activity of β-catenin is necessary for the proper interaction of signaling cascades during heart maturation and adaptation to stress. Keywords: β-catenin, hypertrophy, Wnt-signalling, MAPK signalling, PI3-kinase-mTOR-dependent cascade, PKA-signalling, myocardium.
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5

Garratt, Michael, and Robert C. Brooks. "Oxidative stress and condition-dependent sexual signals: more than just seeing red." Proceedings of the Royal Society B: Biological Sciences 279, no. 1741 (May 30, 2012): 3121–30. http://dx.doi.org/10.1098/rspb.2012.0568.

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The links between fitness, health, sexual signals and mate choice are complex and subject to ongoing study. In 1999, von Schantz et al . made the valuable suggestion that oxidative stress may be an important missing piece of this complex puzzle. Their suggestion has been enthusiastically tested, with over 300 studies citing their paper, but most effort has concerned carotenoid-based (and to a lesser extent melanin-based) visual signals, predominantly in birds and fishes. Today, we know a great deal more about oxidative stress and related physiology, in both a pathological and regulatory sense, than we did in 1999. We revisit von Schantz et al .'s predictions and, more importantly, highlight novel mechanisms that could link oxidative stress with a range of energetically demanding signals, greatly increasing the scope from visual signalling systems that are usually discussed and nearly always tested. In particular, we argue that differences between individuals in their ability to regulate physiology related to oxidative stress may be an important factor influencing the production of sexual signals and the costs that are incurred from investment.
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Runemark, Anna, Bengt Hansson, Marcus Ljungqvist, Mikkel Brydegaard, and Erik I. Svensson. "Has the inbreeding load for a condition-dependent sexual signalling trait been purged in insular lizard populations?" Molecular Ecology 22, no. 5 (January 7, 2013): 1310–21. http://dx.doi.org/10.1111/mec.12178.

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7

HEALEY, MO, and MATS OLSSON. "Too big for his boots: Are social costs keeping condition-dependent status signalling honest in an Australian lizard?" Austral Ecology 34, no. 6 (September 2009): 636–40. http://dx.doi.org/10.1111/j.1442-9993.2009.01968.x.

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8

Steiger, Sandra, Wolf Haberer, and Josef K. Müller. "Social environment determines degree of chemical signalling." Biology Letters 7, no. 6 (June 8, 2011): 822–24. http://dx.doi.org/10.1098/rsbl.2011.0457.

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Few studies have attempted to distinguish between cues and signals in the context of chemical communication. A number of chemical substances have been shown to vary with physiological state, such as stage of oestrus cycle, fertility, dominance status or nutritional condition, but little is known about whether this variation is incidental or adaptive. Here, we provide evidence of a substance whose emission varies with breeding state, but is not merely an incidental by-product of physiological state, but rather, an evolved signal. Breeding females of the facultative biparental burying beetle, Nicrophorus vespilloides , release methyl geranate, a substance that helps males to identify breeding status and to distinguish between their female partners and non-breeding intruders. We demonstrate that females respond flexibly to their social environment and emit high amounts of methyl geranate only in the presence of a male partner, i.e. a receiver. In contrast, cuticular hydrocarbons, which also have been shown to change with breeding status, are not modulated and do not differ between single and paired breeding females. Receiver-dependent chemical signalling is expected to evolve when costs are involved in the production or transmission of the signal; such signal modulation might be more common than previously thought.
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9

Chan, Anita YM, Vernon W. Dolinsky, Carrie-Lynn M. Soltys, and Jason RB Dyck. "DISSECTING THE SIGNALLING PATHWAYS INVOLVED IN THEANTI-HYPERTROPHIC EFFECTS OF RESVERATROL." Clinical & Investigative Medicine 31, no. 4 (August 1, 2008): 7. http://dx.doi.org/10.25011/cim.v31i4.4796.

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Background: Pathological left ventricular hypertrophy is associated with all-cause mortality; however, effective treatment for this condition is currently lacking. We have shown that activation of AMP-activated protein kinase (AMPK) by resveratrol can inhibit myocardial hypertrophy by decreasing protein synthesis and suppressing nuclear factor ofactivated T-cells (NFAT) activation. However, the mechanism by which resveratrol affects AMPK isunknown. Since LKB1 is the upstream kinase of AMPK, we hypothesize that resveratrol signals via LKB1 toactivate AMPK and it is this signalling pathway that contributes to the anti-hypertrophic effects of resveratrol. Methods: Wildtype (WT), LKB1 null, and AMPK null mouseembryonic fibroblasts (MEFs) were treated with vehicle or 100?M resveratrol for 1 h. Cell lysates were subjected to immunoblot analysis to examine the phosphorylation status of the proteins of interest. NFAT-dependent transcription was also measured in these MEFs using a NFAT-luciferase reporter transgene. Results: While resveratrol treatment increased AMPK phosphorylation in WT MEFs, resveratrol was unable to activate AMPK in LKB1 null MEFs. In addition, resveratrol suppressed NFAT-dependent transcription in WT MEFs, yet failed to inhibit NFAT activity in AMPK null MEFs. Conclusion: These data combined with our previous data suggest that resveratrol signals through LKB1 to activate AMPK and that this activation results in suppressed protein synthesis and reduced NFAT activation. As the development of pathologicalcardiac hypertrophy is dependent on protein synthesis and NFAT activation, inhibition of these two pathways by resveratrol may be an exciting new approach for the treatment of pathological cardiac hypertrophy.
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10

Galeotti, P., R. Sacchi, M. Fasola, D. Pellitteri Rosa, M. Marchesi, and D. Ballasina. "Courtship displays and mounting calls are honest, condition-dependent signals that influence mounting success in Hermann's tortoises." Canadian Journal of Zoology 83, no. 10 (October 1, 2005): 1306–13. http://dx.doi.org/10.1139/z05-130.

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Like other terrestrial tortoises, the courtship behaviour of Hermann's tortoises (Testudo hermanni Gmelin, 1789) is based on a multiple signalling system that involves visual, olfactory, tactile, and acoustic signals. In this study, we analysed relationships between male morphology, hematological profile, courtship intensity, vocalizations, and mounting success in Hermann's tortoises breeding in semi-natural enclosures to investigate the effects of male condition on signals exhibited during courtship and on their mounting success. Results showed that mounting success of Hermann's tortoise males was positively affected by the number of sexual interactions/h, number of bites given to the female during interactions, and by call rate and frequency-modulation range. Call rate, frequency-modulation range, and number of sexual interaction/h increased with hematocrit value, while number of bites given to females decreased with leukocyte concentration. In conclusion, courtship signals exhibited by Hermann's tortoise males, including vocalizations, reliably reveal different components of male condition, and females may use these multiple traits to choose high-quality partners. This is the first study documenting the condition-dependent nature of tortoise courting signals and their effect on male mounting success.
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