Relevant bibliographies by topics / Complex physiology

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Complex physiology'

Author: Grafiati
Published: 4 June 2021
Last updated: 9 February 2022

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Journal articles on the topic "Complex physiology"

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Ray, L. B. "From Protein Interactions to Complex Physiology." Science's STKE 2007, no. 395 (July 11, 2007): tw248. http://dx.doi.org/10.1126/stke.3952007tw248.

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SAH, P., E. S. L. FABER, M. LOPEZ DE ARMENTIA, and J. POWER. "The Amygdaloid Complex: Anatomy and Physiology." Physiological Reviews 83, no. 3 (July 2003): 803–34. http://dx.doi.org/10.1152/physrev.00002.2003.

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Sah, P., E. S. L. Faber, M. Lopez de Armentia, and J. Power. The Amygdaloid Complex: Anatomy and Physiology. Physiol Rev 83: 803–834, 2003; 10.1152/physrev.00002.2003.—A converging body of literature over the last 50 years has implicated the amygdala in assigning emotional significance or value to sensory information. In particular, the amygdala has been shown to be an essential component of the circuitry underlying fear-related responses. Disorders in the processing of fear-related information are likely to be the underlying cause of some anxiety disorders in humans such as posttraumatic stress. The amygdaloid complex is a group of more than 10 nuclei that are located in the midtemporal lobe. These nuclei can be distinguished both on cytoarchitectonic and connectional grounds. Anatomical tract tracing studies have shown that these nuclei have extensive intranuclear and internuclear connections. The afferent and efferent connections of the amygdala have also been mapped in detail, showing that the amygdaloid complex has extensive connections with cortical and subcortical regions. Analysis of fear conditioning in rats has suggested that long-term synaptic plasticity of inputs to the amygdala underlies the acquisition and perhaps storage of the fear memory. In agreement with this proposal, synaptic plasticity has been demonstrated at synapses in the amygdala in both in vitro and in vivo studies. In this review, we examine the anatomical and physiological substrates proposed to underlie amygdala function.
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Pollock, David M. "Dissecting the Complex Physiology of Endothelin." Hypertension 56, no. 1 (July 2010): 31–33. http://dx.doi.org/10.1161/hypertensionaha.109.139758.

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Forkink, M., R. Brock, J.Smeitink, P. Willems, and W. Koopman. "Inhibiting mitochondrial Complex I or Complex III differentially affects mitochondrial physiology." Biochimica et Biophysica Acta (BBA) - Bioenergetics 1817 (October 2012): S55. http://dx.doi.org/10.1016/j.bbabio.2012.06.156.

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Griffith, Linda G., and Melody A. Swartz. "Capturing complex 3D tissue physiology in vitro." Nature Reviews Molecular Cell Biology 7, no. 3 (March 2006): 211–24. http://dx.doi.org/10.1038/nrm1858.

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Loscalzo, J., and J. A. Vita. "Ischemia, hyperemia, exercise, and nitric oxide. Complex physiology and complex molecular adaptations." Circulation 90, no. 5 (November 1994): 2556–59. http://dx.doi.org/10.1161/01.cir.90.5.2556.

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Chouaib, Salem, Didier Branellec, and Wim A. Buurman. "More insights into the complex physiology of TNF." Immunology Today 12, no. 5 (January 1991): 141–42. http://dx.doi.org/10.1016/s0167-5699(05)80041-6.

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London, G. M., and B. Pannier. "Arterial functions: how to interpret the complex physiology." Nephrology Dialysis Transplantation 25, no. 12 (October 14, 2010): 3815–23. http://dx.doi.org/10.1093/ndt/gfq614.

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WEST, BRUCE J. "FRACTAL FORMS IN PHYSIOLOGY." International Journal of Modern Physics B 04, no. 10 (August 1990): 1629–69. http://dx.doi.org/10.1142/s0217979290000826.

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The natural variability in physiological structure is herein related to the geometric concept of a fractal. The average dimensions of the branches in the tracheobronchial tree, long thought to be exponential, are shown to be an inverse power law of the generation number modulated by a harmonic variation. A similar functional form is found for the power spectrum of the QRS-complex of the healthy human heart. These results follow from the assumption that the bronchial tree and the cardiac conduction system are fractal forms. The fractal concept provides a mechanism for the morphogenesis of complex structures which are more stable than those generated by classical scaling (i.e., they are more error tolerant).
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Xu, Bai-Nan, Zheng-Hui Sun, Chen Wu, Jin-Li Jiang, Ding-Biao Zhou, Xin-Guang Yu, Garnette R. Sutherland, and Bao-Min Li. "Revascularization for Complex Cerebral Aneurysms." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 38, no. 5 (September 2011): 712–18. http://dx.doi.org/10.1017/s031716710005407x.

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ABSTRACT:Background and Purpose:Complex cerebral aneurysms may require indirect treatment with revascularization. This manuscript describes various surgical revascularization techniques together with clinical outcomes.Methods:Thirty-two consecutive patients with complex cerebral aneurysm were managed from November 2005 to October 2008. Techniques used for revascularization were high-flow bypass, low-flow bypass, branch artery reimplantion, and primary reanastomosis. Physiologic and anatomic monitoring technologies, including electroencephalography, somatosensory evoked potential monitoring, microvascular doppler ultrasonography, and/or indocyanine green videoangiography were used intraoperatively to assess both brain physiology and vascular anatomy. Patient outcome was determined using the Glasgow Outcome Scale at discharge and at a mean of 12 months post operation (range 6-25 months).Results:Two cervical carotid aneurysms (6%) were resected followed by primary reanastomosis, 21 aneurysms (66%) were trapped following saphenous vein high-flow bypasses, five (16%) were clipped after superficial temporal or occipital artery low-flow bypasses, and four (12%) middle cerebral branch arteries were reimplanted. Of the 32 patients at discharge, 29 (91%) had a Glasgow Outcome Scale of four or five, two (6%) had severe disability, and one (3%) died.Conclusion:Cerebral revascularization remains an effective and reliable procedure for treatment of complex cerebral aneurysms. Low morbidity and mortality rates reflect the maturity of patient selection and surgical technique in the management of these lesions.
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Dissertations / Theses on the topic "Complex physiology"

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Dave, Emma. "The physiology of the Escherichia coli pyruvate dehydrogenase complex." Thesis, University of Sheffield, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.364242.

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Sim, Jae Hoon. "Imaging, physiology, and biomechanics of the malleus-incus complex /." May be available electronically:, 2007. http://proquest.umi.com/login?COPT=REJTPTU1MTUmSU5UPTAmVkVSPTI=&clientId=12498.

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Ollivier, Julien. "Scalable methods for modelling complex biochemical networks." Thesis, McGill University, 2011. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=104586.

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In cells, complex networks of interacting biomolecules process both environmental and endogenous signals to control gene expression and other cellular processes. This poses a challenge to researchers who attempt to develop mathematical and computational models of biochemical networks that reflect this complexity. In this thesis, I propose methods that help manage complexity by exploiting the finding that, as for other biological systems, cellular networks are characterized by a modularity that appears at all levels of organization.The first part of this work focuses on the modular properties of proteins and how their function can be characterized through their structure and allosteric properties. I develop a modular rule-based framework and formal modelling language that describes the computations performed by allosteric proteins and that is rooted in biophysical principles. Rule-based modelling conventionally addresses the problem of combinatorial complexity, whereby protein interactions can generate a combinatorial explosion of protein complex states. However, I explore how these same interactions can potentially require a combinatorial number of parameters to describe them. I demonstrate that my rule-based framework effectively addresses this problem of regulatory complexity, and describes allosteric proteins and networks in a unified, consistent, and modular fashion. I use the framework in three applications. First, I show that allostery can make macromolecular assembly more efficacious when a protein that joins two separable parts of a complex is present in excessively high concentrations. Second, I demonstrate that I can straightforwardly analyze the complex cooperative interactions that arise when competitive ligands bind to a multimeric protein. Third, I analyze a new model of G protein-coupled receptor signalling and demonstrate that it explains the functional selectivity of these receptors while being parsimonious in the number of parameters used. Overall, I find that my rule-based modelling framework, implemented as the Allosteric Network Compiler software tool, can ease of modelling and analysis of complex allosteric interactions.If cellular networks are modular, this implies that small sub-systems can be studied in isolation, provided that external inputs and perturbations to the system can be modelled appropriately. However, cellular networks are subject to both intrinsic noise, which is endogenous to the system, but also extrinsic noise, arising from noisy inputs. Furthermore, many inputs may be dynamic, whether due to experimental protocols or perhaps reflecting the cyclic process of cell division. This motivates my development, in the second part of this work, of efficient stochastic simulation algorithms for biochemical networks that can accommodate time-varying biochemical parameters. Starting from Gillespie's well-known First Reaction Method and Gibson and Bruck's Next Reaction Method, I develop two new algorithms that allow time-varying inputs of arbitrary functional form while scaling well to systems comprising many biochemical reactions. I analyze their scaling properties and find that a modified First Reaction Method may scale better than a modified Next Reaction Method in some applications.The third and last part of this thesis introduces a new software tool, Facile, that eases the creation, update and simulation of biochemical network models. Models created through a simple and intuitive textual language are automatically converted into a form usable by downstream tools, for example ordinary differential equations for simulation by Matlab. Also, Facile conveniently accommodates mathematical and time-varying expressions in rate laws.
Au niveau cellulaire, des réseaux complexes d'interaction biomoléculaire traitent les signaux tant environnementaux qu'endogènes dans le but de contrôler l'expression génétique ainsi que d'autres processus cellulaires. Ceci est un défi pour les chercheurs qui veulent concevoir des modèles mathématiques et calculatoires des réseaux biochimiques. Dans cette thèse, je propose des méthodes qui facilitent la gestion de cette complexité en exploitant la constatation que, tout comme d'autres systèmes biologiques, les réseaux cellulaires se caractérisent par une modularité qui transparaît à tous les niveaux d'organisation.Dans la première partie, je mets l'accent sur les propriétés modulaires des protéines et sur la façon de caractériser leur fonction, compte tenu de leur structure et de leurs propriétés allostériques. J'ai mis au point un cadre modulaire à base de règles ainsi qu'un langage formel de modélisation qui permet de décrire les calculs effectués par les protéines allostériques et qui découle de principes biophysiques. La modélisation à base de règles s'adresse conventionnellement au problème de la complexité combinatoire, où les interactions entre les protéines peuvent générer une explosion combinatoire d'états des complexes protéiques. J'examine, cependant, comment il peut s'avérer nécessaire d'utiliser un nombre combinatoire de paramètres pour décrire ces mêmes interactions. Je démontre que notre cadre à base de règles peut régler efficacement ce problème de la complexité régulatoire, et permet de décrire les protéines et les réseaux allostériques de façon unifiée, cohérente et modulaire. J'utilise le cadre développé dans trois applications. Tout d'abord, je montre que l'allostérie peut rendre l'assemblage macromoléculaire plus efficace lorsqu'une protéine qui unit deux parties distinctes d'un complexe protéique est présente en concentration excessive. Deuxièmement, je démontre qu'il est relativement simple d'analyser les interactions coopératives complexes qui surviennent lorsque des ligands compétitifs se lient à une protéine multimérique. En troisième lieu, j'analyse un nouveau modèle de la signalisation des récepteurs couplés aux protéines G qui explique leur sélectivité fonctionnelle tout en limitant le nombre des paramètres utilisés. Globalement, je montre que ce cadre basé sur des règles, qui est implémenté dans le logiciel ‘Allosteric Network Compiler', peut faciliter la modélisation et l'analyse d'interactions allostériques complexes.Si les réseaux cellulaires sont modulaires, il en résulte que des sous-systèmes peuvent être étudiés séparément, à la condition que les entrées et les perturbations externes du système puissent être modélisées adéquatement. Cependant, ces réseaux sont soumis à l'influence du bruit intrinsèque, qui est endogène au système, mais également au bruit extrinsèque, venant des entrées bruyantes. De plus, de nombreuses entrées peuvent être dynamiques. Cela motive, dans la deuxième partie de ce travail, le développement d'algorithmes efficients de simulation stochastique pour les réseaux biochimiques qui peuvent tenir compte de paramètres biochimiques dynamiques. En me fondant sur la méthode maintenant célèbre de Gillespie, d'appellation ‘First Reaction Method', et sur celle de Gibson et Bruck, la ‘Next Reaction Method', j'ai développé deux nouveaux algorithmes qui permettent des entrées dynamiques de forme fonctionnelle arbitraire tout en s'échelonnant bien sur les systèmes qui comportent de nombreuses réactions biochimiques. J'analyse leurs propriétés d'échelonnement et je constate que, pour certaines applications, la ‘First Reaction Method' modifiée s'échelonne mieux que la ‘Next Reaction Method' modifiée.La troisième et dernière partie cette thèse est la présentation d'un nouvel outil informatique, Facile, qui simplifie la création, la mise à jour et la simulation de modèles de réseaux biochimiques.
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Caudron, Quentin. "Neuronal computation on complex dendritic morphologies." Thesis, University of Warwick, 2012. http://wrap.warwick.ac.uk/57056/.

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When we think about neural cells, we immediately recall the wealth of electrical behaviour which, eventually, brings about consciousness. Hidden deep in the frequencies and timings of action potentials, in subthreshold oscillations, and in the cooperation of tens of billions of neurons, are synchronicities and emergent behaviours that result in high-level, system-wide properties such as thought and cognition. However, neurons are even more remarkable for their elaborate morphologies, unique among biological cells. The principal, and most striking, component of neuronal morphologies is the dendritic tree. Despite comprising the vast majority of the surface area and volume of a neuron, dendrites are often neglected in many neuron models, due to their sheer complexity. The vast array of dendritic geometries, combined with heterogeneous properties of the cell membrane, continue to challenge scientists in predicting neuronal input-output relationships, even in the case of subthreshold dendritic currents. In this thesis, we will explore the properties of neuronal dendritic trees, and how they alter and integrate the electrical signals that diffuse along them. After an introduction to neural cell biology and membrane biophysics, we will review Abbott's dendritic path integral in detail, and derive the theoretical convergence of its infinite sum solution. On certain symmetric structures, closed-form solutions will be found; for arbitrary geometries, we will propose algorithms using various heuristics for constructing the solution, and assess their computational convergences on real neuronal morphologies. We will demonstrate how generating terms for the path integral solution in an order that optimises convergence is non-trivial, and how a computationally-significant number of terms is required for reasonable accuracy. We will, however, derive a highly-efficient and accurate algorithm for application to discretised dendritic trees. Finally, a modular method for constructing a solution in the Laplace domain will be developed.
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Wells, Cathy Clarke. "The complex spatial topography of visual attention : behavior and physiology /." View online version; access limited to Brown University users, 2005. http://wwwlib.umi.com/dissertations/fullcit/3174692.

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Florman, Jeremy T. "Neuroendocrine Modulation of Complex Behavior and Physiology in C. elegans." eScholarship@UMMS, 2020. https://escholarship.umassmed.edu/gsbs_diss/1109.

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To survive, animals must adapt to a complex and challenging world in a way that is flexible and responsive, while maintaining internal homeostasis. Neuromodulators provide a means to systemically alter behavioral or physiological state based on intrinsic or extrinsic cues, however dysregulated neuroendocrine signaling has negative consequences for fitness and survival. Here I examine neuroendocrine function and dysfunction using the escape response in Caenorhabditis elegans. The RFamide neuropeptide FLP-18 is a co-transmitter with the monoamine tyramine and functions both synergistically and antagonistically to tyramine in coordinating escape behavior. Using behavioral analysis and calcium imaging, I show that FLP-18 functions primarily through the G-protein coupled receptor (GPCR) NPR-5 to increase calcium levels in muscle, enhancing locomotion rate, bending and reversal behavior during the escape response. Furthermore, I examine the relationship between persistent acute stress and resilience using repeated activation of the escape response as a model of neuroendocrine dysregulation. Repeated activation of the escape response shortens lifespan and renders animals more susceptible to thermal, oxidative, and nutritional stress. Tyramine release is necessary and sufficient for this effect and activity of the tyraminergic RIM neurons is differentially regulated by acute versus long-term stressors. Impaired stress resistance requires both the GPCR TYRA-3 in the intestine and intestinal neuropeptide release. Activation of the insulin receptor DAF-2 is downstream of TYRA-3 and inhibits the transcription factors DAF-16/FOXO, SKN-1/Nrf2 and HSF-1, linking monoamine signaling in acute stress to the insulin signaling pathway and impaired resilience to long-term stressors.
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Burgess, Alexandra Jacquelyn. "The variable light environment within complex 3D canopies." Thesis, University of Nottingham, 2017. http://eprints.nottingham.ac.uk/38967/.

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With an expanding population and uncertain consequences of climate change, the need to both stabilise and increase crop yields is important. The relationship between biomass production and radiation interception suggests one target for improvement. Under optimal growing conditions, biomass production is determined by the amount of light intercepted and the efficiency with which this is converted into dry matter. The amount of light at a given photosynthetic surface is dependent upon solar movement, weather patterns and the structure of the plant, amongst others. Optimising canopy structure provides a method by which we can improve and optimise both radiation interception and also the distribution of light among canopy layers that contribute to net photosynthesis. This requires knowledge of how canopy structure determines light distribution and therefore photosynthetic capacity of a given crop species. The aim of this thesis was to assess the relationships between canopy architecture, the light environment and photosynthesis. This focused on two core areas: the effect of varietal selection and management practices on canopy structure and the light environment and; the effect of variable light on select photosynthetic processes (photoinhibition and acclimation). An image-based reconstruction method based on stereocameras was employed with a forward ray tracing algorithm in order to model canopy structure and light distributions in high-resolution. Empirical models were then applied using parameterisation from manually measured data to predict the effects of variable light on photosynthesis. The plasticity of plants means that the physical structure of the canopy is dependent upon many different factors. Detailed descriptions of canopy architecture are integral to predicting whole canopy photosynthesis due to the spatial and temporal differences in light profiles between canopies. This inherent complexity of the canopy means that previous methods for calculating light interception are often not suitable. 3-dimensional modelling can provide a quick and easy method to retain this complexity by preserving small variations. This provides a means to more accurately quantify light interception and enable the scaling of cellular level processes up to the whole canopy. Results indicate that a canopy with more upright leaves enables greater light penetration to lower canopy layers, and thus higher photosynthetic productivity. This structural characteristic can also limit radiation-induced damage by preventing exposure to high light, particularly around midday. Whilst these features may lead to higher photosynthetic rates per unit leaf area, per unit ground area, photosynthesis is usually determined by total leaf area of the canopies, and within this study, the erect canopies tended to have lower total leaf areas than the more horizontal canopies. The structural arrangement of plant material often led to low levels of light within the lower canopy layers which were punctuated by infrequent, high light events. However, the slow response of photosynthesis to a change in light levels meant that these sun flecks cannot be used by the plant and thus the optimal strategy should be geared towards light harvesting and efficient photosynthesis under low light conditions. The results of this study contribute to our understanding of photosynthetic processes within the whole canopy and provide a foundation for future work in this area.
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Jones, Benjamin R. "Engineering a fluorescent calmodulin for use in complex models of cardiac physiology." Connect to resource, 2009. http://hdl.handle.net/1811/37210.

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Hughes, Ruth. "Serpentine tolerance in the Mimulus guttatus complex." Thesis, University of Exeter, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.286489.

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Stojanovic, Marina. "Role BAP31 complex at the endoplasmic reticulum in normal cell physiology and apoptosis." Thesis, McGill University, 2006. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=102730.

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The endoplasmic reticulum (ER) plays a vital role in synthesis, folding and sorting of newly synthesized secretory cargo proteins. Quality control mechanism in the ER is a key regulator of this process, which separates correctly folded proteins from immature or misfolded secretory proteins, and ultimately retains and disposes the latter before they can exit the ER. Numerous diseases have been associated with defects in the regulation of the egress of the nascent membrane proteins out of ER to the cell surface. BAP31, a polytopic integral membrane protein at the ER, has been implicated as a putative quality control factor and/or cargo protein in regulating the export of specific sets of nascent membrane proteins. Here, I demonstrate BAP31 to be an important player in regulating and maintaining functional integrity of the cell surface. Both truncated (p20BAP31) and deletion (BAP31-null) mutants of BAP31 significantly abrogated expression of tetraspanins at the cell surface, and thus integrin-mediated cell adhesion and survival due to BAP31 effects on tetraspanin transport from the ER. Consistent with its role as a chaperone or cargo receptor, BAP31 was found to interact with Sec61beta and TRAM, components of the nascent protein translocation machinery and to form an important part of the ribosome-translocon complexes at the ER. In addition to its predicted role in quality control mechanism in the ER, BAP31, a BCL-2 associated protein, is recognized as an important regulator of apoptosis, an essential physiological mechanism of cell suicide in development and homeostasis in all multicellular organisms. Here, I characterized SPIKE, a new proposed pro-apoptotic BCL-2 protein and interacting partner of BAP31. SPIKE is a novel and untypical pro-apoptotic BH3 only protein capable of inducing apoptosis without binding to anti-apoptotic BCL-2 partners and endogenous BAP31, whose role in specific apoptotic pathway remains to be elucidated. Thus, BAP31 plays important, but distinct roles in both normal cell physiology and apoptosis.
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Books on the topic "Complex physiology"

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Rammensee, Hans-Georg. MHC ligands and peptide motifs. Austin, Tex: Landes Bioscience, 1997.

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Aizawa, Miki. Major histocompatibility complex of the rat, rattus norvegicus. Sapporo: Hokkaido University School of Medicine, 1988.

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Bianca, C. Towards a mathematical theory of complex biological systems. Singapore: World Scientific, 2011.

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Aizawa, Miki. Major histocompatibility complex of the rat, Rattus norvegicus: Its structure and function. Sapporo: Hokkaido University School of Medicine, 1988.

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The new executive brain: Frontal lobes in a complex world. New York: Oxford University Press, 2009.

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Dehmer, Matthias. Towards an Information Theory of Complex Networks: Statistical Methods and Applications. Boston: Springer Science+Business Media, LLC, 2011.

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New aspects of complement structure and function. Austin, TX: R.G. Landes, 1994.

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Dahlem Workshop on Complex Organismal Functions: Integration and Evolution in Vertebrates (1988 Berlin, Germany). Complex organismal functions: Integration and evolution in vertebrates : report of the Dahlem Workshop on Complex Organismal Functions--Integration and Evolution in Vertebrates, Berlin 1988, August 28-September 2. Edited by Wake David B and Roth, Gerhard, 1942 Aug. 15-. Chichester [England]: Wiley, 1989.

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K, Dana Syamal, Roy Prodyot K, and Kurths J. (Jürgen) 1953-, eds. Complex dynamics in physiological systems: From heart to brain. [Dordrecht]: Springer, 2009.

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Harding, Clifford V. MHC molecules and antigen processing. Austin, Tex: R.G. Landes Co., 1997.

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Book chapters on the topic "Complex physiology"

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Hallett, Mark. "Physiology of Volition." In Understanding Complex Systems, 127–43. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-03205-9_7.

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Ivanov, Plamen Ch, and Ronny P. Bartsch. "Network Physiology: Mapping Interactions Between Networks of Physiologic Networks." In Understanding Complex Systems, 203–22. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-03518-5_10.

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Devesa, J. Manuel. "Complex Procedures for Fecal Incontinence." In Anorectal Physiology, 223–39. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-43811-1_17.

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Chen, Shangbin, and Alexey Zaikin. "Modelling Complex Phenomena in Physiology." In Quantitative Physiology, 189–237. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-33-4033-6_13.

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Küster, Dennis, and Arvid Kappas. "Measuring Emotions Online: Expression and Physiology." In Understanding Complex Systems, 71–93. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-43639-5_5.

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Lee, Hyun-Jong, and Joon-Hyung Doh. "FFR in Complex Lesions." In Coronary Imaging and Physiology, 269–77. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-2787-1_27.

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Hu, James W., and Alain Woda. "Trigeminal Brainstem Nuclear Complex, Physiology." In Encyclopedia of Pain, 4060–65. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-28753-4_4604.

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Orini, Michele, Riccardo Barbieri, Mimma Nardelli, Enzo Pasquale Scilingo, and Gaetano Valenza. "Introduction to Complex Cardiovascular Physiology." In Complexity and Nonlinearity in Cardiovascular Signals, 3–42. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-58709-7_1.

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Chen, Shangbin, and Alexey Zaikin. "Complex and Surprising Dynamics in Gene Regulatory Networks." In Quantitative Physiology, 147–87. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-33-4033-6_12.

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Kolláth-Leiß, Krisztina, and Frank Kempken. "The Fungal MCC/Eisosome Complex: An Unfolding Story." In Physiology and Genetics, 119–30. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-71740-1_4.

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Conference papers on the topic "Complex physiology"

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Abbott, Jonathan A. "Cardiovascular physiology: mechanisms of control." In Complex Adaptive Structures, edited by William B. Spillman, Jr. SPIE, 2001. http://dx.doi.org/10.1117/12.446759.

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Szasz, Andras. "Oncothermia: Complex therapy by EM and fractal physiology." In 2014 XXXIth URSI General Assembly and Scientific Symposium (URSI GASS). IEEE, 2014. http://dx.doi.org/10.1109/ursigass.2014.6930100.

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Vavrinsky, E., D. Moskalova, J. Mihalov, M. Donoval, M. Daricek, and V. Stopjakova. "Complex measurement of human physiology using designed miniature wireless sensors." In 2014 10th International Conference on Advanced Semiconductor Devices & Microsystems (ASDAM). IEEE, 2014. http://dx.doi.org/10.1109/asdam.2014.6998692.

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Tsvankin, Vadim, Dmitry Belchenko, Devon Scott, and Wei Tan. "Anisotropic Strain Effects on Vascular Smooth Muscle Cell Physiology." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176284.

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Biological development is a complex and highly-regulated process, a significant part of which is controlled by mechanostimulus, or the strain imparted on a cell by its environment. Mechanostimulus is important for stem cell differentiation, from cytoskeletal assembly to cell-cell and cell-matrix adhesion [1]. The mechanics of cells and tissues play a critical role in organisms, under both physiological and pathological conditions; abnormal mechanotransduction — the mechanism by which cells sense and respond to strain — has been implicated in a wide range of clinical pathologies [2,3].
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Shrestha, Rajiv P., Yossi Chait, Christopher V. Hollot, Stuart Chipkin, and Claus P. Schmitt. "A Mathematical Model of Parathyroid Hormone Response to Acute Changes in Plasma Ionized Calcium Concentration in Humans." In ASME 2008 International Mechanical Engineering Congress and Exposition. ASMEDC, 2008. http://dx.doi.org/10.1115/imece2008-67551.

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A complex bio-mechanism, referred to as calcium homeostasis, regulates plasma ionized calcium (Ca++) concentration in the human body to within a narrow physiologic range which is crucial for maintaining normal physiology and metabolism. In this paper we present a qualitative model of the calcium homeostatic system and then focus on a particular sub-system, termed Ca-PTH axis. We consider the dynamics of the axis involving the response of the parathyroid glands to acute changes in plasma Ca++ concentration. We use a two-pool, linear time-varying model to describe the Ca-PTH axis. We show that this model, parameterized using a guided iterative parametrization scheme and induced hypocalcemic clamp test data, successfully predicts dynamics observed in clinical tests of induced hypercalcemia in normal humans.
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Kulhanek, Tomas, Marek Matejak, Jan Silar, and Jiri Kofranek. "Parameter estimation of complex mathematical models of human physiology using remote simulation distributed in scientific cloud." In 2014 IEEE-EMBS International Conference on Biomedical and Health Informatics (BHI). IEEE, 2014. http://dx.doi.org/10.1109/bhi.2014.6864463.

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Kryuchkova, E. V. "Plant-bacterial associations in the remediation of complex pollutants." In IX Congress of society physiologists of plants of Russia "Plant physiology is the basis for creating plants of the future". Kazan University Press, 2019. http://dx.doi.org/10.26907/978-5-00130-204-9-2019-241.

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Gorshkova, T. A. "Creation and use of complex carbohydrates: a workshop on plants." In IX Congress of society physiologists of plants of Russia "Plant physiology is the basis for creating plants of the future". Kazan University Press, 2019. http://dx.doi.org/10.26907/978-5-00130-204-9-2019-14.

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Biglino, Giovanni, Alessandro Giardini, Catriona Baker, Tain-Yen Hsia, Richard S. Figliola, Andrew M. Taylor, and Silvia Schievano. "Implementing the Sano Modification in an Experimental Model of the Norwood Circulation." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80273.

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Surgical palliation of hypoplastic left heart syndrome (HLHS) is performed in three stages, the first of which is known as the Norwood procedure [1]. Traditionally, this operation involves securing an unobstructed outlet for the systemic circulation in infants for whom the single right ventricle is the only pump in the system, with pulmonary flow sourced via a modified Blalock-Taussig (BT) shunt. In 2003, Sano et al. have proposed a radical variation of this operation, known as the Sano modification [2]. In this case, the pulmonary circulation is supplied directly from the systemic right ventricle via an unvalved ventriculo-pulmonary Goretex conduit, or Sano shunt. Characteristically, flow in the Sano shunt continues throughout diastole (diastolic runoff). Differences between surgical approaches for stage 1 palliation have been addressed in the literature [3]. A computational model of Norwood procedure and Sano modification has also been proposed [4]. However, an experimental model of this complex physiology is currently lacking. Having recently presented an in vitro model of stage 1 physiology with a BT shunt arrangement [5], we propose here a compact mock circulatory system suitable for simulating the Sano physiology in patient-specific anatomical models.
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Gulin, S. V., and A. G. Pirkin. "FEATURES OF BUSINESS-PROCESSES IN THE CREATION OF ELECTROTECHNOLOGICAL SYSTEMS FOR THE AGRICULTURAL INDUSTRIAL COMPLEX." In INNOVATIVE TECHNOLOGIES IN SCIENCE AND EDUCATION. DSTU-Print, 2020. http://dx.doi.org/10.23947/itno.2020.357-362.

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This article offers a universal methodology for the design, creation and operation of complex electrotechnological systems. This methodology is based on a system-process approach to business modeling. The article provides a detailed description of all private business processes that provide a full cycle of business engineering, and offers a General mathematical expression for a comprehensive assessment of the effectiveness of the business engineering process. The proposed methodology has been tested on the example of designing, creating and operating vegetation climate systems (VCS). This example shows that it is possible to conduct quite serious scientific research at the intersection of plant physiology and electric power engineering, which allows us to create modern self-adjusting systems for automatic microclimate control when growing plants. Application of engineering methods allows to increase the efficiency of development of information systems for automatic control of parameters of the most important physiological processes (photosynthesis, transpiration, etc.) in plants under the influence of environmental factors. The article outlines the prospects for the development of the subject area of engineering in the direction of solving specific problems to integrated energy engineering, and the energy business - from trading individual services to trading models and technologies.
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