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Books on the topic 'Coagulator'

Author: Grafiati
Published: 30 May 2022
Last updated: 31 May 2022

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1

Coagulation-inflammation interface: Coagulation assembly on leukocytes. New York: Chapman & Hall, 1997.

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2

Altieri, Dario C. The coagulation-inflammation interface: Coagulation assembly on leukocytes. New York: Springer, 1997.

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3

Matthias, Fritz Reinhard. Blood Coagulation Disorders. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-83098-3.

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4

Lichtin, Alan, and John Bartholomew, eds. The Coagulation Consult. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-9560-4.

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5

Kwaan, Hau C., and David Green, eds. Coagulation in Cancer. Boston, MA: Springer US, 2009. http://dx.doi.org/10.1007/978-0-387-79962-9.

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6

Coagulation problems during pregnancy. Edinburgh: Churchill Livingstone, 1985.

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7

Aledort, Louis M., Leon W. Hoyer, Jeanne M. Lusher, Howard M. Reisner, and Gilbert C. White, eds. Inhibitors to Coagulation Factors. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4613-0331-2.

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8

Sibinga, C. Th Smit, P. C. Das, and P. M. Mannucci, eds. Coagulation and Blood Transfusion. Boston, MA: Springer US, 1991. http://dx.doi.org/10.1007/978-1-4615-3900-1.

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9

Marques, Marisa B. Quick guide to coagulation testing. 2nd ed. Washington, DC: American Association for Clinical Chemistry, 2009.

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10

Klaus, Strenge, and Vincent B, eds. Coagulation kinetics and structure formation. New York: Plenum Press, 1987.

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11

International, Scientific Symposium on Fibrinogen Thrombosis Coagulation and Fibrinolysis (1989 Taipei Taiwan). Fibrinogen, thrombosis, coagulation, and fibrinolysis. New York: Plenum Press, 1990.

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12

Essential guide to blood coagulation. 2nd ed. Chichester, West Sussex: Wiley-Blackwell, 2013.

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13

Antovic, Jovan P., and Margareta Blombäck, eds. Essential Guide to Blood Coagulation. Oxford, UK: Wiley-Blackwell, 2009. http://dx.doi.org/10.1002/9781444314465.

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14

Antovic, Jovan P., and Margareta Blombäck, eds. Essential Guide to Blood Coagulation. Oxford, UK: John Wiley & Sons Ltd, 2013. http://dx.doi.org/10.1002/9781118327517.

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15

Liu, Chung Yuan, and Shu Chien, eds. Fibrinogen, Thrombosis, Coagulation, and Fibrinolysis. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4615-3806-6.

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16

Sonntag, Hans, and Klaus Strenge. Coagulation Kinetics and Structure Formation. Edited by B. Vincent. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4757-0617-8.

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17

Liu, Chung Yuan. Fibrinogen, Thrombosis, Coagulation, and Fibrinolysis. Boston, MA: Springer US, 1990.

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18

The evolution of vertebrate blood coagulation. Mill Valley, Calif: University Science Books, 2012.

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19

Taloyan, Anett M. Coagulation: Kinetics, structure formation, and disorders. Hauppauge, N.Y: Nova Science Publisher's, 2011.

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20

Coagulation disorders: Quality in laboratory diagnosis. New York: Demos Medical Pub., 2011.

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21

Ranucci, Marco, ed. The Coagulation Labyrinth of Covid-19. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-82938-4.

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22

1935-, Takada Akikazu, Collen D, Gaffney Patrick J, and Nihon Seiri Gakkai Taikai, eds. Recent progress in blood coagulation and fibrinolysis: Proceedings of the International Symposium 'Clot Formation and Lysis' held on March 21-22, 1997, Hamamatsu, Japan, as a satellite symposium of the 74th Annual Meeting of the Japanese Society of Physiology. Amsterdam: Elsevier, 1997.

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23

Theodore S. Zimmerman Memorial Conference: Progress in Vascular Biology, Hemostasis, and Thrombosis (1990 La Jolla, San Diego, Calif.). Progress in vascular biology, hemostasis, and thrombosis. New York, N.Y: New York Academy of Sciences, 1991.

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24

Theodore S. Zimmerman Memorial Conference, Progress in Vascular Biology, Hemostasis, and Thrombosis (1990 La Jolla, San Diego, Calif.). Progress in vascular biology, hemostasis, and thrombosis: Theodore S. Zimmerman Memorial Conference. New York, N.Y: New York Academy of Sciences, 1991.

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25

Operational control of coagulation and filtration processes. 3rd ed. Denver: American Water Works Association, 2010.

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26

A, Triplett Douglas, and College of American Pathologists, eds. Advances in coagulation testing: Interpretation and application. Skokie, Ill: College of American Pathologists, 1986.

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27

Molecular genetics and immunoanalysis in blood coagulation. Weinheim, F.R.G: VCH, 1988.

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28

Tancray, J. Coagulation time measurements applied to polymer surfaces. Manchester: UMIST, 1997.

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29

(Editor), T. P. Driscoll, and J. Kapturauskas (Editor), eds. Application and Optimization of the Galvano-coagulator Process to Heavy Metal Waste Streams: Werf Report Project 00-cts-15et (Werf Report). Intl Water Assn, 2002.

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30

A, Zwaal R. F., and Hemker H. C, eds. Blood coagulation. Amsterdam: Elsevier, 1986.

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31

A, Penner John, and Hassouna Houria I, eds. Coagulation disorders. Philadelphia: Saunders, 1993.

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32

Kuiper, Gerhardus J. A. J. M., and Hugo ten Cate. Coagulation monitoring. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0266.

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Haemostasis is a dynamic process to stop bleeding after vessel wall damage. Platelets form a platelet plug via activation, adherence, and aggregation processes. The coagulation proteins are activated one-by-one, cascading towards fibrin polymerization, a process controlled by thrombin generation. Fibrinolysis is the process responsible for fibrin mesh degradation, which is also controlled by thrombin. Besides procoagulant proteins, anticoagulant proteins maintain a balance in the haemostatic system. Measuring platelet count and function can be done as part of the monitoring of haemostasis, while coagulation times are measured to assess the coagulation proteins. Degradation products of fibrin and lysis times give information about fibrinolysis. Point-of-care monitoring provides simple, rapid bedside testing for platelets and for whole blood using viscoelasticity properties. In trauma-induced coagulopathy (TIC) platelet counts and coagulation times are still common practice to evaluate haemostasis, but point-of-care measurements are being used more and more. Medication interfering with haemostasis is frequently used in intensive care unit patients. Each (group of) drug(s) has its own monitoring tests either based on classical or novel techniques.
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33

Kilkelly, Shannon. Coagulation System. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0090.

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Despite the development of entirely new classes of anticoagulant medication, vitamin K antagonists like warfarin continue to be commonly prescribed for a wide range of cardiovascular diagnoses. Conversely, the advent of low molecular weight heparin has greatly simplified the use of the drug to the point that patients can dose themselves at home with no need for any type of monitoring. Given the widespread use of these medications, it is not surprising that an increasing number of patients requiring urgent or emergent surgery will present with a medically induced coagulopathy. Managing this coagulopathy requires assessment of the urgency of the operation, the patient’s volume status, and the need for reanticoagulation following surgical intervention.
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34

Burger, Christina F., Melissa L. Bellomy, and Joseph J. Schlesinger. Coagulation System. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0091.

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Anticoagulation is increasingly prevalent in the general population and poses a significant risk of increased bleeding in patients needing urgent or emergent surgical procedures. There are two main classes of direct or anticoagulants: direct thrombin inhibitors and factor Xa inhibitors. Management of these patients requires assessment of bleeding risk, possible reversal of anticoagulation, and subsequent management after surgery to prevent postoperative complications associated with either bleeding or clot formation (due to cessation of anticoagulants). This chapter covers the proper assessment and management of patients on oral direct thrombin inhibitor or oral Factor Xa inhibitor therapies.
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35

Blood Coagulation. Elsevier, 1986. http://dx.doi.org/10.1016/s0167-7306(08)x6004-1.

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36

A, Penner John, and Hassouna Houria I, eds. Coagulation disorders. Philadelphia: Saunders, 1992.

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37

Hemostasis And Coagulation. W.B. Saunders Company, 2009.

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38

Coagulation In Cancer. Springer, 2009.

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39

A, Zwaal R. F., ed. Coagulation and lipids. Boca Raton, Fla: CRC Press, 1989.

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40

Kwaan, Hau C., and Green David. Coagulation in Cancer. Springer, 2014.

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41

Stechemesser, Hansjoachim, and Bohuslav Dobias. Coagulation and Flocculation. Taylor & Francis Group, 2005.

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42

Hansjoachim, Stechemesser, and Dobiáš B, eds. Coagulation and flocculation. 2nd ed. Boca Raton: Taylor & Francis, 2005.

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43

University of Sheffield. Biomedical Information Service., ed. Blood coagulation factors. Sheffield: Universi y of Sheffield Biomedical Information Service, 1985.

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44

Huber, Kurt, and Joao Morais. Coagulation and thrombosis. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656653.003.0017.

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Antithrombotic therapy consisting of antiplatelet agents and/or anticoagulants is an important way to avoid atherothrombotic complications, especially in secondary prevention. Primary prevention by antithrombotic measures usually refers to the prevention of stroke in patients with atrial fibrillation and an increased risk for stroke or peripheral thromboembolic events by the use of anticoagulants. In certain situations a combination of anticoagulants and antiplatelet agents is mandatory. This chapter provides the pathophysiological background of coagulation and thrombosis, reports on the epidemiology of antithrombotic treatment, and describes the efficacy and safety of preventive antithrombotic measures in different cardiovascular indications. A short paragraph summarizes the current discussion of skipping aspirin in order to reduce the rate and severity of bleeding events.
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45

Huber, Kurt, and Joao Morais. Coagulation and thrombosis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199656653.003.0017_update_001.

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Antithrombotic therapy consisting of antiplatelet agents and/or anticoagulants is an important way to avoid atherothrombotic complications, especially in secondary prevention. Primary prevention by antithrombotic measures usually refers to the prevention of stroke in patients with atrial fibrillation and an increased risk for stroke or peripheral thromboembolic events by the use of anticoagulants. In certain situations a combination of anticoagulants and antiplatelet agents is mandatory. This chapter provides the pathophysiological background of coagulation and thrombosis, reports on the epidemiology of antithrombotic treatment, and describes the efficacy and safety of preventive antithrombotic measures in different cardiovascular indications. A short paragraph summarizes the current discussion of skipping aspirin in order to reduce the rate and severity of bleeding events.
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46

Hematology and Coagulation. Elsevier, 2015. http://dx.doi.org/10.1016/c2013-0-16034-5.

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47

Hematology and Coagulation. Elsevier, 2020. http://dx.doi.org/10.1016/c2017-0-02293-0.

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48

Donati, Maria Benedetta. Coagulation and Cancer. S Karger AG, 1988.

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49

M, Aledort Louis, and International Symposium on Inhibitors to Coagulation Factors (2nd : 1993 : Chapel Hill, N.C.), eds. Inhibitors to coagulation factors. New York: Plenum Press, 1995.

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50

Immunoassays in Coagulation Testing. Springer, 2012.

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