Relevant bibliographies by topics / Clostridium perfringens

Academic literature on the topic 'Clostridium perfringens'

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Published: 4 June 2021
Last updated: 22 June 2024

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Journal articles on the topic "Clostridium perfringens"

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Oskutis, Morgan, and Matthew Reaven. "Septic Shock and Spontaneous Gangrenous Gas Necrosis of the Spleen Secondary to Clostridium perfringens: The Importance of Source Control." Case Reports in Critical Care 2021 (May 6, 2021): 1–4. http://dx.doi.org/10.1155/2021/5563071.

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Clostridium perfringens is a rare cause of septic shock, occurring most frequently in immunocompromised patients. An uncommon cause of Clostridium perfringen septicemia is spontaneous gangrenous gas necrosis of the spleen, where the primary treatment is splenectomy. We present a case of septic shock caused by spontaneous gangrenous gas necrosis of the spleen secondary to Clostridium perfringens in a patient whose profound pancytopenia made obtaining definitive source control extremely difficult.
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SHEFF, BARBARA. "Clostridium perfringens." Nursing 34, no. 8 (August 2004): 31. http://dx.doi.org/10.1097/00152193-200408000-00031.

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Sheff, Barbara. "Clostridium perfringens." Nursing (Ed. española) 23, no. 7 (August 2005): 50. http://dx.doi.org/10.1016/s0212-5382(05)71474-6.

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Kristopaitis, Theresa, Randy Jensen, and Meena Gujrati. "Clostridium perfringens:." Surgical Neurology 51, no. 4 (April 1999): 448–51. http://dx.doi.org/10.1016/s0090-3019(97)00454-0.

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Labbé, Ronald G. "Clostridium perfringens." Journal of AOAC INTERNATIONAL 74, no. 4 (July 1, 1991): 711–14. http://dx.doi.org/10.1093/jaoac/74.4.711.

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Abstract In the United States and Canada, Clostridium perfringens remains a leading cause of bacterial food poisoning in humans. It has been primarily associated with meat and poultry products prepared in food service establishments. Fecal spore levels of 104 or more per g are considered indicative of a food poisoning outbreak. However, elevated spore levels of this organism are frequently seen in healthy elderly individuals, an observation that complicates investigations of suspected outbreaks. Recent studies with this population indicate that fecal enterotoxin levels are a valuable and effective assay for confirming outbreaks due to this organism. With regard to the toxin itself, a membrane protein of 50 000-70 000 molecular weight has been isolated as a possible enterotoxin-receptor site. It is the subsequent action of the toxin on membrane structure that results in the loss of ions and fluid associated with illness. In addition, the enterotoxin gene has been cloned in E. coil and sequenced. Using toxin-specific DNA probes, only 6% of non-symptomatic farm animals were found to possess the enterotoxin gene, disproving the hypothesis that all strains of this organism can produce the toxin.
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Jerram, Lucy. "Clostridial disease in cattle." Livestock 24, no. 6 (November 2, 2019): 274–79. http://dx.doi.org/10.12968/live.2019.24.6.274.

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Clostridial disease affects cattle across the UK throughout the year but particularly in the grazing season. All clostridial disease occurs when conditions in the muscles or organs become anaerobic allowing for bacterial proliferation and toxin production. The most common pathogenic clostridium is Clostridium chauvoei which causes blackleg and has both skeletal and cardiac forms. Clostridium perfringens is the second most commonly identified clostridia with type A causing enterotoxaemia and type D causing pulpy kidney. Clostridium novyi is the third most common clostridia — type B causes black disease and type D causes bacillary haemoglobinuria. Cattle are most at risk during the grazing season meaning that control and prevention of disease should be particularly targeted at this time of year. The cost of vaccination is so low compared with the cost of losing even one animal that an appropriate protocol should be implemented on all farms.
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Bezborodova, Natalia A., Evgenia N. Shilova, Veronika V. Kozhukhovskaya, Vladlena D. Zubareva, Olga V. Sokolova, and Nikolai A. Martynov. "Improvement of Laboratory Diagnosis for Detection and Identification of Bovine Clostridiosis." WSEAS TRANSACTIONS ON BIOLOGY AND BIOMEDICINE 20 (November 1, 2023): 305–12. http://dx.doi.org/10.37394/23208.2023.20.31.

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Objective: Clostridiosis is a toxic infectious disease; the pathogenicity factor of causative agents is the secreted toxins. A characteristic feature of clostridiosis pathogens is their polytropism. They affect both humans and agricultural, domestic, and wild animals. Our research aimed to monitor Clostridium perfringens and Clostridium difficile spread among agricultural organizations of the Ural region. Materials and Methods: 137 biological samples were obtained from cattle with symptoms of clostridial infection. For PCR species and toxinotype identification commercial kits and previously described protocols were used. Results verification was conducted using MALDI-TOF MS. Results: Out of 137 samples of selected material Clostridium was detected in 40.6% of samples: Cl. difficile in 35.8%, Cl. perfringens in 25.3%, Cl. difficile+Cl. perfringens in 16.4%. Cl. difficile and Cl. perfringens were found in 30.5% of fecal samples, in pathological material from dead calves and cows – 8.7%, in milk samples – 1.4%. Conclusion: Laboratory methods made it possible to verify the diagnosis: infectious anaerobic enterotoxemia of calves in one case, necrotic enteritis in 3 animals, and intestinal toxic infection caused by Cl. perfringens type A in 2 cows and 5 calves. The diagnostics of toxinotypes of Cl. perfringens have made it possible to conduct toxin-specific vaccination against clostridial infection in farms.
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Liu, Ying, Jianfei Zhu, Shaoqi Qu, Jianzhong Shen, and Kui Zhu. "Plant-Derived Xanthones against Clostridial Enteric Infections." Antibiotics 12, no. 2 (January 21, 2023): 232. http://dx.doi.org/10.3390/antibiotics12020232.

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Intestinal bacterial infections are a major threat to human and animal health. In this study, we found plant-derived antibacterial xanthones, particularly α-mangostin (AMG) from the mangosteen peel, exhibiting extraordinary activities against Clostridium perfringens. Structure–activity relationship analysis showed that prenylation modulated the activity of xanthones. The efficacy of AMG (4, 8, 20 mg/kg body weight) was also demonstrated in the broiler chicken necrotic enteritis model infected with Clostridium perfringens. In the models (n = 6 per group), feed supplementation of AMG maintained the homeostasis of the gut microbiome by reducing the colonization of clostridia and promoting the integrity of intestinal barriers via the upregulation of mucin expression. These results suggest that plant-derived xanthones may be a potential alternative to antibiotics for treating clostridial enteric infections in the clinic.
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Bueschel, Dawn, Richard Walker, Leslie Woods, John Kokai-Kun, Bruce McClane, and J. Glenn Songer. "Enterotoxigenic Clostridium perfringens type A necrotic enteritis in a foal." Journal of the American Veterinary Medical Association 213, no. 9 (November 1, 1998): 1305–7. http://dx.doi.org/10.2460/javma.1998.213.09.1305.

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Clostridium perfringens is the most common cause of clostridial enteric disease in domestic animals. Many individuals have suggested that isolation of C perfringens type A from a horse with enteric disease is of little importance, even when other likely causes are ruled out; however, it appears that enterotoxigenic C perfringens type A may cause enteric disease in horses.
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Maikanov, Balgabay, Raikhan Mustafina, Laura Auteleyeva, Jan Wiśniewski, Krzysztof Anusz, Tomasz Grenda, Krzysztof Kwiatek, Magdalena Goldsztejn, and Magdalena Grabczak. "Clostridium botulinum and Clostridium perfringens Occurrence in Kazakh Honey Samples." Toxins 11, no. 8 (August 13, 2019): 472. http://dx.doi.org/10.3390/toxins11080472.

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The aim of this study was to assess occurrence of Clostridium botulinum and Clostridium perfringens in honey samples from Kazakhstan. Analyses were carried out using a set of PCR methods for identification of anaerobic bacteria, and detection of toxin genes of C. botulinum and C. perfringens. Among 197 samples, C. botulinum was noticed in only one (0.5%). The isolated strain of this pathogen showed the presence of the bont/A and ntnh genes. C. perfringens strains were isolated from 18 (9%) samples, and mPCR (multiplex PCR) analysis led to them all being classified as toxin type A with the ability to produce α toxin. Sequence analysis of 16S rDNA genes showed occurrence in 4 samples of other anaerobes related to C. botulinum, which were C. sporogenes and C. beijerinckii strains. C. botulinum prevalence in honey samples from Kazakhstan in comparison to the prevalence in samples collected from the other regions seems to be less. The highest prevalence of Clostridium sp. was noticed in the East Kazakhstan province. Our study is the first survey on BoNT-producing clostridia and C. perfringens prevalence in Kazakh honey.
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Dissertations / Theses on the topic "Clostridium perfringens"

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Canard, Bruno. "Organisation genomique de clostridium perfringens." Paris 7, 1991. http://www.theses.fr/1991PA077145.

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Clostridium perfringens est un bacille a gram positif, pathogene pour l'homme et les animaux. Nous avons construit une carte physique et genetique du chromosome de la souche de reference cpn50, de type a, ainsi que de sept autres isolats appartenant aux serotypes a, b, d et e. Nous avons ainsi pu etudier comparativement l'organisation genomique des genes et loci impliques dans la maintenance des fonctions vitales de la bacterie, dans la virulence, et dans la plasticite genomique. Nous avons caracterise respectivement au niveau moleculaire les operons ribosomiques rrn, le gene nagh codant pour une n-acetyl-beta-d-glucosaminidase, et neuf regions genomiques sujettes a des polymorphismes de restriction. Quatre de ces dernieres sont associees a des facteurs de virulence, et une de ces quatre tolere des variations en taille de l'ordre de 0,5 megabase. L'organisation des genes de virulence et la plasticite genomique sont etroitement imbriquees
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Stiles, Bradley G. "Purification and characterization of Clostridium perfringens iota toxin." Diss., Virginia Polytechnic Institute and State University, 1987. http://hdl.handle.net/10919/76516.

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Clostridium perfringens type E iota toxin is implicated in some cases of fatal diarrhea in calves, lambs, and guinea pigs. A crossreacting "iota-like" toxin, produced by Clostridium spiroforme, is responsible for antibiotic-associated and weaning related enterotoxemias of rabbits. Antisera developed against culture supernatant of either organism neutralized the biological activity of iota or iota-like toxin. By using C. spiroforme antiserum and crossed immunoelectrophoresis (crossed IEP), we found two cross-reacting antigens in C. perfringens type E supernatants. C. perfringens types A, B, C, and D, which do not produce iota toxin, did not cross-react with C. spiroforme antiserum. To determine if either antigen had iota toxin activity, we separated the cross-reacting antigens of C. perfringens by preparative isoelectric focusing (IEF) and tested all IEF fractions for biological activity in guinea pigs and mice. The fraction containing the faster-migrating antigen seen in crossed IEP, designated iota b (ib), had some guinea pig dermonecrotic and mouse lethal activity. Other fractions, including the one containing the slower migrating iota a (ia) antigen, had little to no biological activity. When fractions containing ia and ib were mixed, there was an 8 and 25 fold increase in mouse lethal and dermonecrotic titers, respectively. Activity was neutralized by C. perfringens type E or C. spiroforme antisera and other fractions, when mixed with ia or ib, did not have a synergistic effect. Both components of C. perfringens iota toxin were purified using ammonium sulfate precipitation, DEAE anion exchange chromatography, preparative IEF, Sephadex G-100 gel filtration, and flatbed electrophoresis to yield a 12 and 5% final recovery of ia and ib, respectively. Each protein was homogeneous by SDS PAGE, gradient PAGE, and crossed IEP using homologous antiserum. There was at least an 8 fold increase in mouse lethal titer and 64 fold increase in dermonecrotic titer when equimolar amounts of ia and ib were mixed. Monospecific antisera against purified ia and ib neutralizd the iota or iota-like activity of crude supernatants. A sensitive and specific ELISA was developed using monospecific and C. spiroforme antisera. The ia and ib proteins have a pI of 5.2 and 4.2 and molecular weights of 48,000 and 71,000 (SDS PAGE), respectively. The ia protein is heat stable (85° C/15 min) while ib lost its activity at 55°C. Amino terminus sequencing revealed that both proteins were blocked by an unknown functional group(s). Purified ia, but not ib, has ADP-ribosylating activity specific poly-L-arginine in vitro. Recent evidence suggests that nonmuscle actin, involved in the cytoskeletal structure of eucaryotic cells, may act as the in situ acceptor.
Ph. D.
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Pérez, Janampa David Remy. "Caracterización toxigénica de la fosfolipasa C del Clostridium perfringens (Cp-PLC) y su relación con aislados de C. perfrigens de casos de enterotoxemia en alpacas." Master's thesis, Universidad Nacional Mayor de San Marcos, 2010. https://hdl.handle.net/20.500.12672/3262.

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La enterotoxemia, causada por el Clostridium perfringens, es la enfermedad infecciosa más importante que afecta a las alpacas, debido a que ocasiona elevadas tasas de mortalidad neonatal de hasta 70%. Recientes estudios han sugerido la participación de la Cp-PLC (C. perfringens fosfolipasa C) como factor de virulencia responsable del cuadro enterotoxemico en alpacas y otras especies domesticas. El presente estudio evaluó las características toxigénicas de la Cp-PLC y de sobrenadantes de diferentes aislados de C. perfringens obtenidos de casos de enterotoxemia en alpacas relacionándolos con sus niveles de producción de Cp-PLC. El protocolo de purificación de Cp-PLC mostró ser exitoso, mostrando su comportamiento como una enterotoxina incapaz de generar lesiones entéricas. Asimismo, los aislados de C. perfringens analizados evidenciaron distintas características toxigénicas independientes de la presencia de Cp-PLC. Al parecer, la Cp-PLC no seria un factor esencial del C. perfringens en la producción de lesiones entéricas en casos de enterotoxemias en alpacas. Palabras Claves: Cp-PLC, Clostridium perfringens, enterotoxigenico
--- Enterotoxemia caused by Clostridium perfringens, causes a mortality neonatal rate up to 70%, this is why it is considered as the most important infections disease. Recent studies has suggested that Cp-PLC (Clostridium perfringens phospholipase C) is a main virulence factor responsible of the enterotoxemic lesions found in alpacas and other domestic animals. This study evaluated the toxigenic characteristics of Cp-PLC and of supercultures of C. perfringens isolates from enterotoxemia in alpacas associated with their levels of Cp-PLC production. The Cp-PLC purification protocol used was successful, showing that Cp-PLC as an enterotoxin enteric unable to cause injury. Similarly, C. perfringens isolates analyzed showed different toxigenic characteristics independently of the Cp-PLC production. Apparently, Cp-PLC does not be a essential factor from C. perfringens in the production of enteric lesions in cases of enterotoxemia in alpacas. Key Word: Cp-PLC, Clostridium perfringens, enterotoxigenic.
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Perelle, Sylvie. "Toxine IOTA de "Clostridium perfringens" et toxines apparentées." Paris 11, 1996. http://www.theses.fr/1996PA114811.

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Xie, Xinye. "Purification and characterization of a blood group A₂degrading [alpha]-N-acetylgalactosaminidase from clostridium perfringens." free to MU campus, to others for purchase, 2001. http://wwwlib.umi.com/cr/mo/fullcit?p3012978.

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Eaton, Julian Timothy. "Structural studies of Clostridium perfringens alpha toxin." Thesis, Birkbeck (University of London), 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.417896.

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Griffiths, Nicola Jane. "Studies on Clostridium perfringens in the horse." Thesis, University of Liverpool, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.367091.

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Hunter, Sophie Emma Clare. "Molecular genetics of Clostridium perfringens epsilon-toxin." Thesis, University of Southampton, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.316420.

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Justin, Neil. "Structural studies of clostridium perfringens alpha toxin." Thesis, Birkbeck (University of London), 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.392355.

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Russell, Katherine Margaret. "Intestinal responses to Clostridium perfringens in broilers." Thesis, University of Edinburgh, 2016. http://hdl.handle.net/1842/25514.

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Clostridium perfringens is the aetiological agent of Necrotic enteritis (NE); a disease that impacts on the health and welfare of broilers. This disease is a large cost to the industry and presents as lesions in the small intestine hindering productivity. Antibiotics are commonly used to treat NE but as pressure increases to limit their use further information about disease onset and broiler responses to the bacteria and it’s virulence factors during infection is required to implement new preventative measures and treatments. NetB is a secreted toxin from C. perfringens which has an important role in NE onset. Using an in situ intestinal loop model we have been able to characterise: I) temporal broiler responses to NetB positive bacterial culture supernatant (Chapter 2), ii) early host responses to different isolates possessing NetB (virulent) or not (avirulent) in the presence or absence of bacterial cells (Chapter 3) and iii) the responses of two commercial broiler breeds (Chapter 4) four hours post exposure. Samples collected from these experiments have been used for histology, mRNA expression and immunohistology. We have shown differences in mRNA expression in the duodenum of broilers after exposure to C. perfringens cells as well as the culture supernatant from the isolates used after four hours. The presence of bacteria cells resulted in up-regulation of pro-inflammatory cytokine, IFN-γ, mRNA, whereas it resulted in down-regulation of B-LA, mRNA a gene involved in presentation of pathogens to immune cells. IL-6 mRNA expression was also reduced in the presence of virulent isolates. This could indicate a possible evasion strategy for C. perfringens in broilers. Immunohistochemical analysis indicated that slower growing broilers have increased numbers of immune cells (macrophages and γδ T cells) in their duodenum compared with faster growing broilers, although this did not appear to have an effect on mRNA expression levels of pro-inflammatory cytokines, 4h post antigen infusion. Overall we detect greater changes when bacteria are included with culture supernatant and have highlighted possible mechanisms for C. perfringens to avoid the broiler immune system. Induction of NE in the literature requires pre-disposing factors, including co-infection with other intestinal pathogens and dietary manipulation of the host. The final experiment trialled protocols administering a virulent isolate of C. perfringens in-feed and via gavage along with an increased protein source to induce NE (Chapter 5). These models were not considered to be consistent for further investigation of NE in the future.
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Books on the topic "Clostridium perfringens"

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Belyy, Alexander. Roles of Asp179 and Glu270 in ADP-ribosylation of Actin by Clostridium perfringens Iota Toxin. Freiburg: Universität, 2015.

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Standardization, International Organization for. Microbiology: General guidance for enumeration of Clostridium perfringens : colony count technique = Microbiologie : directives ge ne rales pour le de nombrement de Clostridium perfringens : me thode par comptage des colonies. [Geneva]: International Organization for Standardization, 1985.

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Warriner, Keith. Clostridium Perfringens: Features,Detection and Prevention of Foodborne Disease. Elsevier Science & Technology Books, 2016.

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Publications, ICON Health. Clostridium Perfringens - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References. ICON Health Publications, 2004.

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N. Liu, J.Q. Wang, K.T. Gu, Q.Q. Deng, and J.P. Wang. Effects of aflatoxin, Clostridium perfringens and yeast cell wall on the growth performance and gut health of broilers. Verlag Eugen Ulmer, 2017. http://dx.doi.org/10.1399/eps.2017.207.

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D. Józefiak, S. Świątkiewicz, B. Kierończyk, M. Rawski, J. Długosz, Ricarda Margarete Engberg, and O. Højberg. Clostridium perfringens challenge and dietary fat type modifies performance, microbiota composition and histomorphology of the broiler chicken gastrointestinal tract. Verlag Eugen Ulmer, 2016. http://dx.doi.org/10.1399/eps.2016.130.

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Mehta, Tanmay. Clostridium Perfringenes: A Medical Comic Book. Independently Published, 2020.

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Book chapters on the topic "Clostridium perfringens"

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Coia, John, and Heather Cubie. "Clostridium perfringens." In The Immunoassay Kit Directory, 686–87. Dordrecht: Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-009-0359-3_8.

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Heredia, Norma L., and Ronald G. Labbé. "Clostridium perfringens." In Guide to Foodborne Pathogens, 82–90. Oxford: John Wiley & Sons, 2013. http://dx.doi.org/10.1002/9781118684856.ch5.

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Juneja, Vijay K., John S. Novak, and Ronald J. Labbe. "Clostridium perfringens." In Pathogens and Toxins in Foods, 53–70. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555815936.ch4.

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da Silva, Neusely, Marta Hiromi Taniwaki, Valéria Christina Amstalden Junqueira, Neliane Ferraz de Arruda Silveira, Margarete Midori Okazaki, and Renato Abeilar Romeiro Gomes. "Clostridium perfringens." In Microbiological Examination Methods of Food and Water, 161–76. Second edition. | Leiden, The Netherlands ; Boca Raton : CRC Press/Balkema, [2018]: CRC Press, 2018. http://dx.doi.org/10.1201/9781315165011-12.

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McClane, Bruce A., Susan L. Robertson, and Jihong Li. "Clostridium perfringens." In Food Microbiology, 465–89. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555818463.ch18.

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García, Santos, Jorge E. Vidal, Norma Heredia, and Vijay K. Juneja. "Clostridium perfringens." In Food Microbiology, 513–40. Washington, DC, USA: ASM Press, 2019. http://dx.doi.org/10.1128/9781555819972.ch19.

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da Silva, Neusely, Marta Hiromi Taniwaki, Valéria Christina Amstalden Junqueira, Neliane Ferraz de Arruda Silveira, Margarete Midori Okazaki, and Renato Abeilar Romeiro Gomes. "Clostridium perfringens." In Microbiological Examination Methods of Food and Water, 161–76. Second edition. | Leiden, The Netherlands ; Boca Raton : CRC Press/Balkema, [2018]: CRC Press, 2017. http://dx.doi.org/10.1201/b13740-12.

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Hassan, Karl A., and Ian T. Paulsen. "Clostridium perfringens." In Genomes of Foodborne and Waterborne Pathogens, 213–21. Washington, DC: ASM Press, 2014. http://dx.doi.org/10.1128/9781555816902.ch14.

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Narayan, Krishna Gopal, Dharmendra Kumar Sinha, and Dhirendra Kumar Singh. "Clostridium perfringens." In Veterinary Public Health & Epidemiology, 317–22. Singapore: Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-19-7800-5_34.

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Bhunia, Arun K. "Clostridium botulinum, Clostridium perfringens, Clostridium difficile." In Foodborne Microbial Pathogens, 209–28. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7349-1_12.

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Conference papers on the topic "Clostridium perfringens"

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Thuler, Jaqueline Rodrigues. "AVALIAÇÃO DAS TOXINFECÇÕES ALIMENTARES POR Clostridium perfringens." In II Congresso Nacional de Microbiologia On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/ii-conamic/9734.

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Farooq, M. F., and P. Grover. "Clostridium Perfringens Liver Abscess with Massive Intravascular Hemolysis." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a3549.

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Chen, Lin, Ting Bai, Wei Wang, Tian-Fei Liu, Li-Li Ji, and Jia-Min Zhang. "Analysis of Codon Bias on Clostridium Perfringens Del1 Genome." In 2017 2nd International Conference on Biological Sciences and Technology (BST 2017). Paris, France: Atlantis Press, 2018. http://dx.doi.org/10.2991/bst-17.2018.20.

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Moreno, Andrea M., Thais S. P. Ferreira, Renata R. Almeida, Luciane T. S. Zucon, Renata Paixão, Débora D. S. Gobbi, Cleise R. Gomes, and Antônio J. P. Fereirra. "Isolation of Clostridium perfringens from swine carcasses and feces." In Sixth International Symposium on the Epidemiology and Control of Foodborne Pathogens in Pork. Iowa State University, Digital Press, 2005. http://dx.doi.org/10.31274/safepork-180809-771.

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Shen, Juezhou. "Clostridium Perfringens Alpha Toxin Inhibits Granulocyte Colony-Stimulating Factor." In ISAIMS 2022: 2022 3rd International Symposium on Artificial Intelligence for Medicine Sciences. New York, NY, USA: ACM, 2022. http://dx.doi.org/10.1145/3570773.3570789.

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Kammeyer, P., D. Hartmann, S. Braune, H. Nagengast, and S. Kleinschmidt. "Hämorrhagische Enteritis: Clostridium perfringens Typ A bei einem Hund." In 66. Jahrestagung der Fachgruppe Pathologie der Deutschen Veterinärmedizinischen Gesellschaft. Georg Thieme Verlag, 2023. http://dx.doi.org/10.1055/s-0043-1770877.

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Islam, M. I., and S. Schecter. "Clostridium Perfringens - A Rare Cause of Spontaneous Bacterial Peritonitis." In American Thoracic Society 2023 International Conference, May 19-24, 2023 - Washington, DC. American Thoracic Society, 2023. http://dx.doi.org/10.1164/ajrccm-conference.2023.207.1_meetingabstracts.a5371.

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Moreno, Andrea M., Flávio Hirose, Luciane T. S. Zucon, Daniela S. Doto, Renata Paixão, Thais S. P. Ferreira, Renata R. Almeida, and Roberto A. Bordin. "Characterization of Clostridium perfringens isolated from meat and bone meal." In Sixth International Symposium on the Epidemiology and Control of Foodborne Pathogens in Pork. Iowa State University, Digital Press, 2005. http://dx.doi.org/10.31274/safepork-180809-772.

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Shadid, A. M., R. Nabaty, D. Kapadia, and D. R. Ouellette. "Clostridium Perfringens Pleural Effusion as First Presentation for Malignant Mesothelioma." In American Thoracic Society 2023 International Conference, May 19-24, 2023 - Washington, DC. American Thoracic Society, 2023. http://dx.doi.org/10.1164/ajrccm-conference.2023.207.1_meetingabstracts.a2106.

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Dudiki, N., S. Summers, M. Maxfield, and R. Leslie. "A Rare Case of Hydropneumothorax and Necrotizing Pneumonia by Clostridium Perfringens." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6844.

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Reports on the topic "Clostridium perfringens"

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Jackson, Armitra, Gary Sullivan, Joseph G. Sebranek, and James S. Dickson. Growth of Clostridium perfringens on Natural and Organic Frankfurters. Ames (Iowa): Iowa State University, January 2009. http://dx.doi.org/10.31274/ans_air-180814-114.

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Jackson, Armitra, Gary Sullivan, Joseph G. Sebranek, and James S. Dickson. Use of Natural Ingredients to Control Growth of Clostridium perfringens on Frankfurters and Ham. Ames (Iowa): Iowa State University, January 2010. http://dx.doi.org/10.31274/ans_air-180814-979.

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Jackson, Armitra, Gary Sullivan, Joseph G. Sebranek, and James S. Dickson. Survival and Growth of Clostridium perfringens on Commercial No-Nitrate-or-Nitrite Added (Natural and Organic) Bacon and Ham. Ames (Iowa): Iowa State University, January 2010. http://dx.doi.org/10.31274/ans_air-180814-778.

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Rosser, Katy, Iulia Gherman, Erica Kintz, Paul Cook, and Anthony WIlson. Assessment of the risk to consumers as a result of disruption to the cold chain during direct supply of Qurbani meat and offal. Food Standards Agency, June 2022. http://dx.doi.org/10.46756/sci.fsa.nuc910.

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Qurbani is a religious practice that takes place during Eid al-Adha. Consumers practicing Qurbani typically wish to collect meat and red offal within a short time after slaughter, which means these products cannot complete normal chilling processes before leaving the slaughterhouse. This could permit greater growth of pathogens and has the potential to increase the risk of consumer illness. The FSA is working with industry and stakeholder groups to ensure that the risk to consumers under these conditions remains at an acceptable level. To help inform these discussions, the FSA commissioned this assessment to understand the difference in risk from allowing meat and offal to be provided to consumers without the normal chilling process. The microbiological team at the FSA have analysed scientific literature, expert opinion and business and consumer survey data to assess the effect of disrupting the cold chain on pathogens in Qurbani meat. The pathogens that were chosen for inclusion in this assessment are non-typhoidal Salmonella enterica, Shiga toxin-producing Escherichia coli, and Clostridium perfringens. Their growth characteristics and prevalence in beef, lamb and goat meat and offal are discussed. The assessment concluded that given the reported variation in the process, there were two important scenarios with distinct outcomes. In the typical scenario, which is the most likely outcome based on the collected data, there is no significant difference in risk to consumer health compared to normal chilling processes, and the risk level was established as Very Low (“very rare but cannot be excluded”). In a reasonably foreseeable worst-case scenario, Salmonella spp. and STEC levels may increase, presenting an increased risk to the consumer. This risk level was established as Low (“rare but does occur”). We also identified several areas where more evidence would be helpful, and as a result identified a High level of uncertainty in our conclusion.
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Holland, Darren, and Nazmina Mahmoudzadeh. Foodborne Disease Estimates for the United Kingdom in 2018. Food Standards Agency, January 2020. http://dx.doi.org/10.46756/sci.fsa.squ824.

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In February 2020 the FSA published two reports which produced new estimates of foodborne norovirus cases. These were the ‘Norovirus Attribution Study’ (NoVAS study) (O’Brien et al., 2020) and the accompanying internal FSA technical review ‘Technical Report: Review of Quantitative Risk Assessment of foodborne norovirus transmission’ (NoVAS model review), (Food Standards Agency, 2020). The NoVAS study produced a Quantitative Microbiological Risk Assessment model (QMRA) to estimate foodborne norovirus. The NoVAS model review considered the impact of using alternative assumptions and other data sources on these estimates. From these two pieces of work, a revised estimate of foodborne norovirus was produced. The FSA has therefore updated its estimates of annual foodborne disease to include these new results and also to take account of more recent data related to other pathogens. The estimates produced include: •Estimates of GP presentations and hospital admissions for foodbornenorovirus based on the new estimates of cases. The NoVAS study onlyproduced estimates for cases. •Estimates of foodborne cases, GP presentations and hospital admissions for12 other pathogens •Estimates of unattributed cases of foodborne disease •Estimates of total foodborne disease from all pathogens Previous estimates An FSA funded research project ‘The second study of infectious intestinal disease in the community’, published in 2012 and referred to as the IID2 study (Tam et al., 2012), estimated that there were 17 million cases of infectious intestinal disease (IID) in 2009. These include illness caused by all sources, not just food. Of these 17 million cases, around 40% (around 7 million) could be attributed to 13 known pathogens. These pathogens included norovirus. The remaining 60% of cases (equivalent to 10 million cases) were unattributed cases. These are cases where the causal pathogen is unknown. Reasons for this include the causal pathogen was not tested for, the test was not sensitive enough to detect the causal pathogen or the pathogen is unknown to science. A second project ‘Costed extension to the second study of infectious intestinal disease in the community’, published in 2014 and known as IID2 extension (Tam, Larose and O’Brien, 2014), estimated that there were 566,000 cases of foodborne disease per year caused by the same 13 known pathogens. Although a proportion of the unattributed cases would also be due to food, no estimate was provided for this in the IID2 extension. New estimates We estimate that there were 2.4 million cases of foodborne disease in the UK in 2018 (95% credible intervals 1.8 million to 3.1 million), with 222,000 GP presentations (95% Cred. Int. 150,000 to 322,000) and 16,400 hospital admissions (95% Cred. Int. 11,200 to 26,000). Of the estimated 2.4 million cases, 0.9 million (95% Cred. Int. 0.7 million to 1.2 million) were from the 13 known pathogens included in the IID2 extension and 1.4 million1 (95% Cred. Int. 1.0 million to 2.0 million) for unattributed cases. Norovirus was the pathogen with the largest estimate with 383,000 cases a year. However, this estimate is within the 95% credible interval for Campylobacter of 127,000 to 571,000. The pathogen with the next highest number of cases was Clostridium perfringens with 85,000 (95% Cred. Int. 32,000 to 225,000). While the methodology used in the NoVAS study does not lend itself to producing credible intervals for cases of norovirus, this does not mean that there is no uncertainty in these estimates. There were a number of parameters used in the NoVAS study which, while based on the best science currently available, were acknowledged to have uncertain values. Sensitivity analysis undertaken as part of the study showed that changes to the values of these parameters could make big differences to the overall estimates. Campylobacter was estimated to have the most GP presentations with 43,000 (95% Cred. Int. 19,000 to 76,000) followed by norovirus with 17,000 (95% Cred. Int. 11,000 to 26,000) and Clostridium perfringens with 13,000 (95% Cred. Int. 6,000 to 29,000). For hospital admissions Campylobacter was estimated to have 3,500 (95% Cred. Int. 1,400 to 7,600), followed by norovirus 2,200 (95% Cred. Int. 1,500 to 3,100) and Salmonella with 2,100 admissions (95% Cred. Int. 400 to 9,900). As many of these credible intervals overlap, any ranking needs to be undertaken with caution. While the estimates provided in this report are for 2018 the methodology described can be applied to future years.
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Cairo, Jessica, Iulia Gherman, and Paul Cook. The effects of consumer freezing of food on its use-by date. Food Standards Agency, July 2021. http://dx.doi.org/10.46756/sci.fsa.ret874.

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The current Food Standards Agency consumer guidance states that consumers can freeze pre-packed food right up to the “use-by” date and, once food has been defrosted, it should be consumed within 24 hours. This strategic review has collated relevant data to determine whether there is an increased risk in relation to freezing ready-to-eat and non-ready-to-eat foods on the use-by date compared to the day before the use-by date. The review has focused on how the shelf-life of a food is determined and the effects of freezing, thawing and refrigeration on foodborne pathogens, including Bacillus spp., Campylobacter spp., Clostridium botulinum, Clostridium perfringens, Listeria monocytogenes, Salmonella, pathogenic Escherichia coli and Shigella spp. In the UK, food business operators are responsible for setting the safe shelf-life of a food which, in practice, should take into consideration the consumer habits, as well as the factors affecting shelf-life, such as food product characteristics, food processing techniques, transport, retail and domestic food storage temperatures, and type of packaging. Some countries, such as Ireland, New Zealand and Canada specifically recommend including safety margins within shelf lives. This is used to maintain brand integrity because it ensures that the food is consumed in its optimum condition. The FSA has collaborated with other organisations in the production of several guidance documents; however, there is no explicit requirement for the consideration of a margin of safety when setting shelf-life. There is also no legal requirement in the UK to consider a safety margin when setting shelf-life. According to regulations, pathogens should not be present in sufficient levels to cause foodborne illness on the use-by date, as food should still be safe to eat on that day. Given that these requirements are met, the risk assessed in this report arises from the processes of freezing, thawing and subsequent refrigerated storage for a further 24 hours, and the potential for these to increase pathogen levels. In this review, it was found that there is a risk of additional growth of certain pathogens during the refrigerated storage period although the impact of freezing and thawing on the extent of this growth was not readily evident. This risk would relate specifically to ready-to-eat foods as cooking of non-ready-to-eat foods after defrosting would eliminate pathogens. This report explores the potential issues related to consumer freezing on the use-by date and identifies additional information or research required to understand the risks involved. Overall, there is little evidence to suggest a significant change in risk between consumers freezing ready-to-eat food on the use-by date compared to freezing the food on the day before the use-by date. Specific areas that merit further research include the risks due to low temperature survival and growth of L. monocytogenes. There is also a lack of research on the effects of freezing, defrosting and refrigeration on the growth and toxin production of non-proteolytic C. botulinum, and the growth of Salmonella during domestic freezing and thawing. Finally, more information on how food business operators set shelf-life would enable a better understanding of the process and the extent of the safety margin when determining shelf-life of ready-to-eat and non-ready-to-eat foods.
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