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Relevant bibliographies by topics / Clostridium difficile

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Academic literature on the topic 'Clostridium difficile'

Author: Grafiati

Published: 4 June 2021

Last updated: 9 March 2023

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Journal articles on the topic "Clostridium difficile"

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von Eichel-Streiber, Christoph, and Veit Braun. "Das difficile Clostridium / The difficile Clostridium." LaboratoriumsMedizin 32, no. 4 (January 1, 2008): 219–34. http://dx.doi.org/10.1515/jlm.2008.043.

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Adhikari, Sushil. "Clostridium Difficile." Nepalese Medical Journal 1, no. 1 (June 22, 2018): 43–46. http://dx.doi.org/10.3126/nmj.v1i1.20400.

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Clostridium difficile ; a group of spore forming, toxin forming, gram positive anerobel is implicated in hospital associated diarrhea and is the causative agent of infectious diarrhea. It is the most common hospital associated infection in Europe and North America, and is presumed to be as prevalent in the rest of the world.There has been emergence of new virulent strain of C. difficile, identified as BI, NAP1, and toxinotype III and ribotype 027 (subsequently known as BI/NAP1/027) by various typing method in recent years, implicated in dramatic increase in C. difficile infections.Diagnosis is established by presence of C. difficile toxin or C. difficile toxin gene in stool. Lab testing does not distinguish C. difficile infection and asymptomatic carriage. Clinical suspicion and positive stool study confirms a diagnosis.Clostridium Difficile infection, is most common health care associated infection in Europe and North America, and the available studies show it may have similar prevalence in Nepal. Literature review does not reveal any significant study being conducted in Nepal as of now. It warrants further study to exactly determine the incidence/prevalence and its impact in current health care in Nepal. Clinicians need increased awareness and prompt diagnosis to reduce morbidity and further prevention of transmission.Nepalese Medical Journal, vol.1, No. 1, 2018, page: 43-47

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Sander, Ruth. "Clostridium difficile." Nursing Older People 19, no. 7 (September 2007): 38. http://dx.doi.org/10.7748/nop.19.7.38.s23.

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Heywood, Suzy. "Clostridium difficile." Nursing Standard 22, no. 12 (November 28, 2007): 59. http://dx.doi.org/10.7748/ns.22.12.59.s44.

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Robinson, Scott. "Clostridium difficile." Nursing Standard 23, no. 12 (November 28, 2008): 59–60. http://dx.doi.org/10.7748/ns.23.12.59.s58.

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Kauffman, Myrna. "Clostridium Difficile." Gastroenterology Nursing 29, no. 2 (March 2006): 164. http://dx.doi.org/10.1097/00001610-200603000-00060.

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Bartlett, John G. "Clostridium difficile." Journal of Clinical Gastroenterology 41, Supplement 1 (May 2007): S24—S29. http://dx.doi.org/10.1097/mcg.0b013e31803d16ec.

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Martin, Jessica, Damian Mawer, and Mark H. Wilcox. "Clostridium difficile." Current Opinion in Infectious Diseases 26, no. 5 (October 2013): 454–60. http://dx.doi.org/10.1097/01.qco.0000433319.82618.8f.

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Stoddart, Bethan, and Mark H. Wilcox. "Clostridium difficile." Current Opinion in Infectious Diseases 15, no. 5 (October 2002): 513–18. http://dx.doi.org/10.1097/00001432-200210000-00010.

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Stoddart, Bethan, and Mark H. Wilcox. "Clostridium difficile." Current Opinion in Internal Medicine 2, no. 1 (February 2003): 12–17. http://dx.doi.org/10.1097/00132980-200302010-00003.

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Dissertations / Theses on the topic "Clostridium difficile"

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Hamdi, Cassandra. "Clostridium difficile : Rapid typing Clostridium difficile using MALDI-TOF MS analysis." Thesis, Högskolan i Skövde, Institutionen för biovetenskap, 2019. http://urn.kb.se/resolve?urn=urn:nbn:se:his:diva-17659.

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Caproni, Lisa J. "Antibiotics and Clostridium difficile." Thesis, University of Edinburgh, 2004. http://hdl.handle.net/1842/24132.

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The aims of this thesis were to investigate the antibiotic susceptibility patterns of C. difficile with relation to the S-type of the isolates over a period of 18 months. Detailed growth curves were performed on strains NCTC 11223, the sequenced strain 630 and an endemic isolate 338a. Toxin A was shown to be produced upon entry to stationary phase in agreement with other studies. OD₆₀₀ was found to be a good predictor of growth phase and allowed this measurement to be used for subsequent experiments. MICs were performed on 186 random isolates of C. difficile collected during an 18-month epidemiological study to investigate the patterns of sensitivity to six different antibiotics. No evidence of resistance was seen to the two treatment antibiotics and all strains were resistant to cefoxitin (MICs 64-256mg/ml), the antibiotic used in most selective media. Most strains (98.9%) had intermediate resistance to ceftriaxone. The MIC₅₀ and MIC₉₀ of the strains to amoxicillin and clindamycin were very close (8 and 16 for amoxicillin and 16 for clindamycin) but the range of MICs was great. Clindamycin resistance was common with 67% of strains resistant (MICs of > 8mg/ml), 25% with intermediate resistance (MIC > 4mg/ml) and only 8% sensitive (MICs of < 2mg/ml). Twelve isolates from six different patients had very high resistance to clindamycin with MICs > 128mg/ml. Multiple isolates from the same patient, taken at different times, showed changes in susceptibility patterns over time. The only major change in susceptibility over the time period was in clindamycin resistance; some strains appearing to become more resistant while others became less resistant. No differences were apparent in the MIC₅₀ and MIC₉₀ of the different S-types of C. difficile identified, although some S-types were present in very small numbers. No links between antibiotics prescribed and susceptibility patterns were found. Three strains (NCTC 11223, strain 630 and endemic isolate 338a) were cultured in sub-lethal concentrations of the six antibiotics (1/2,1/4 and1/8 of the MIC) over 104 hours and growth and toxin A measured three times a day. The effects varied between strain and antibiotic. The most common effect on the growth of the strains was to increase the initial lag period by ca. 4h compared to the antibiotic-free controls through the clindamycin resistant strain NCTC 11223, (MIC >512mg/ml) showed not lag whatsoever in comparison to the controls when grown in this antibiotic. The most common effect on toxin A production was in the onset of toxin elaboration. Normally toxin began to appear in low levels in early stationary phase before accumulating to high levels by the start of decline.

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Båverud, Viveca. "Clostridium difficile in horses /." Uppsala : Dept. of Veterinary Microbiology, Swedish Univ. of Agricultural Sciences ([Institutionen för veterinärmedicinsk mikrobiologi], Sveriges lantbruksuniv.), 2002. http://epsilon.slu.se/avh/2002/91-576-6378-5.pdf.

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Cairns, Michelle Dawn. "Evolution of Clostridium difficile." Thesis, University College London (University of London), 2018. http://discovery.ucl.ac.uk/10060221/.

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Clostridium difficile continues to be a leading cause of healthcare-associated infections in the developed world. Increased detection of C. difficile infection (CDI) and development of typing schemes to differentiate between strains is primarily due to the recognition of global outbreaks of a single strain, BI/NAP1/027 which is characterised by three common typing techniques; restriction endonuclease analysis (REA), pulsed-field gel electrophoresis (PFGE) and PCR ribotyping. Phylogenetic analysis using multilocus sequence typing (MLST) divides C. difficile into five phylogenetic lineages which align the well-known PCR ribotypes; 027, 023, 017, 078 and a lineage containing diverse PCR ribotypes. MLST data in this thesis confirmed the five phylogenetic lineages were maintained after testing a larger collection of isolates from varied sources with further micro-diversity within the individual lineages. MLST investigation did not identify a lineage exclusive to nonhuman strains or any correlation between sequence type and geographical location. Data in this thesis also supports the notion that PCR ribotyping and REA do not correspond as well as previously considered. This may result in phylogenetically similar strains being designated as a different type or variant. The toxin A-B+ PCR ribotype 017 strain that forms a predominant lineage is little investigated. Through whole genome sequencing (WGS) and single nucleotide polymorphism (SNP) analysis, a historical clone of PCR ribotype 017 was identified from a London hospital ward. Although no phenotype exclusive to the clonal strain was characterised, this is the first report in the UK investigating the phylohistory of isolates from hospitalised patients with CDI due to PCR ribotype 017. Further investigation of PCR ribotype 017 with a larger and global collection of strains revealed two distinct sub-lineages containing multiple independent clonal expansions, antimicrobial resistant SNP determinants, deletions and insertions which were well distributed geographically and temporally. The data suggests transmission between humans and animals and findings support a USA origin with multiple, global transmission events. The key findings of this thesis are that C. difficile as a species is continually evolving with the appearance of divergent sub-lineages. WGS is superior to routine typing methodologies for tracking this evolution and will have significant impacts for outbreak investigation, understanding the phylohistory and phylogeography of C. difficile and other pathogens that are a threat to human health.

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Wheeldon, Laura J. "Studies on Clostridium difficile." Thesis, Aston University, 2008. http://publications.aston.ac.uk/15406/.

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Clostridium difficile Is the major cause of nosocomial diarrhoea in the UK and is associated with high morbidity and mortality rates. There has been a large increase in cases of C. difficile associated disease (CDAD) in the last decade and It is thought that the emergence of the hypervirulent strain (ribotype 027) has contributed towards this rise. A major factor in the control and prevention of the disease is adequate cleaning of the clinical environment and disinfection, usually with chlorine based agents. However, the spores of C. difficile are highly resistant to many disinfectants.

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Karlsson, Sture. "Toxin production in Clostridium difficile /." Stockholm : Karolinska institutet, 2004. http://diss.kib.ki.se/2004/91-77349-812-2/.

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Permpoonpattana, Patima. "Clostridium difficile : infection and immunity." Thesis, Royal Holloway, University of London, 2013. http://repository.royalholloway.ac.uk/items/33009ec4-7815-0803-d39b-f968c8d9cdbb/7/.

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Clostridium difficile is a Gram positive pathogen of significant importance in the UK, Europe and the USA. No vaccine has been developed and current treatments are focused on hospital management and the use of antibiotics. The disease is spread in hospitals in the spore form and the role of spores in C. difficile infecton is poorly understood. In this project spores of C. difficile have been characterised. The proteins from the outermost layers of the spore were identified and the genes cloned. Three of these surface proteins have unique enzymatic properties that maybe important for symptoms of disease. The ability of C. difficile spores to adhere to intestinal cells was found to be far greater than with live cells and through this we have identified that the spore may play an important role in colonisation. The regulation of spore coat gene expression during sporulation was also examined and temporal phases of genes expression identified. A major part of this project was to develop a mucosal vaccine to C. difficile. The approach used was to clone the C-terminus of toxin A onto the surface of Bacillus subtilis spores and use these recombinant spores to immunise mice and hamsters. We found that oral delivery of these spores conferred 75% protection to C. difficile infection in a hamster model of infection. Further, parenteral immunisation of the same antigens (toxin A and B) failed to generate mucosal responses and this showed that mucosal immunisation is critical for good protection. Finally, we found that antibodies to the C-terminus of toxin A were cross reactive to the C-terminus of toxin B. This showed that mucosal delivery of just the C-terminus of toxin A is sufficient to confer protection in an animal model of infection. The outcome of this work is that we have shown the parameters for successful immunisation and vaccination against C. difficile.

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Underwood, Sarah. "Sporulation initiation in Clostridium difficile." Thesis, University of Leeds, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.505066.

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Clostridium difficile is a leading cause of hospital-acquired diarrhoea, responsible for over 30% of cases of antibiotic-associated colitis, nearly all cases of pseudomembranous colitis and costs the NHS over 200 million per year. This bacterium is able to persist in the hospital environment to cause recurrent infection by the formation of stable spores, refractile to current decontamination procedures. A more comprehensive understanding of the sporulation signal transduction pathway is essential for the design of a decontamination regime effective in removing the spores from the nosocomial environment and the logical design of novel antimicrobial agents. This project aimed to elucidate the mechanism of sporulation initiation . regulation and the role of sporulation-associated proteins in other C. difficile virulence processes, such as toxin production and colonisation. Analysis of sporulation in response to various hospital cleaning agents showed that the combination of a neutral detergent (such as Hospec) with EDTA is a more effective cleaning agent than the chlorine-based agents currently used, as the combination product is uniquely able to both kill vegetative cells and inhibit spore formation. A variety of molecular approaches were used to elucidate information regarding the C. difficile sporulation initiation pathway and the relationship between sporulation and toxin production. Three putative C. difficile sporulation-associated sensor histidine kinases (CD1A, CD2A and CD3B) were identified and shown to be independently involved in sporulation initiation. Furthermore, CD3B has been shown to directly phosphorylate the master response regulator SpoOA, strongly suggesting that this pathway is a two-component system, as opposed to the extended phosphore lay pathway found in B. subtilis. Previous studies on bacteria capable of both toxin production and endospore formation have described links between the two processes. Data presented here indicates SpoOA has a role in indirectly regulating C. difficile toxin A and B production, as the protein is capable of specifically binding promoter regions of the toxin regulatory genes tcdC and tcdD. Inoculation of a triple-stage continuous-culture chemostat that modelled the human gut with C. difficile spoDA- mutant provided further evidence that SpoDA has a key role in both colonisation a!1d toxin production. Overall, this work adds to the growing body of evidence that SpaDA is a master global regulator and has a crucial role in the pathogenicity of C. difficile, making it an excellent target for future novel antimicrobial therapies.

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Martins, Luís Filipe Pires. "Clostridium difficile uma ameaça renovada." Master's thesis, Instituto de Ciências Biomédicas Abel Salazar, 2008. http://hdl.handle.net/10216/21105.

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César, Artur Jorge Fernandes. "Clostridium difficile - prevenção e controlo." Master's thesis, Faculdade de Medicina da Universidade do Porto, 2009. http://hdl.handle.net/10216/50362.

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Books on the topic "Clostridium difficile"

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Roberts, Adam P., and Peter Mullany, eds. Clostridium difficile. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-6361-4.

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Aktories, Klaus, and Tracy D. Wilkins, eds. Clostridium difficile. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-662-06272-2.

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Mullany, Peter, and Adam P. Roberts, eds. Clostridium difficile. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-365-7.

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Clostridium difficile: Methods and protocols. New York: Humana Press, 2010.

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C. difficile treatments & remedies your doctor isn't telling you. Port Townsend, WA: Embrace Health, 2014.

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Mastrantonio, Paola, and Maja Rupnik, eds. Updates on Clostridium difficile in Europe. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-72799-8.

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Crowley, Dolores. Cloning virulence factors of clostridium difficile. [S.l: The Author], 1991.

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D, Rolfe Rial, and Finegold Sydney M. 1921-, eds. Clostridium difficile: Its role in intestinal disease. San Diego: Academic Press, 1988.

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Dubberke, Erik R. Contemporary diagnosis and management of Clostridium difficile infection. Newtown, Pa: Handbooks in Health Care Co., 2011.

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Great Britain. Commission for Healthcare Audit and Inspection. Investigation into outbreaks of Clostridium difficile at Stoke Mandeville Hospital, Buckinghamshire Hospitals NHS Trust. London: Commission for Healthcare Audit and Inspection, 2006.

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Book chapters on the topic "Clostridium difficile"

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Gerding, Dale N., and Stuart Johnson. "Clostridium difficile." In Bacterial Infections of Humans, 243–51. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-5327-4_13.

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Coia, John, and Heather Cubie. "Clostridium difficile." In The Immunoassay Kit Directory, 679–85. Dordrecht: Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-009-0359-3_7.

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Gale, Alexa R., and Matthew Wilson. "Clostridium Difficile." In Gastrointestinal Emergencies, 353–56. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-98343-1_101.

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Schütt-Gerowitt, Heidi. "Clostridium difficile." In Lexikon der Infektionskrankheiten des Menschen, 175–77. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-39026-8_199.

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Wilcox, Mark H. "Clostridium difficile." In Principles and Practice of Clinical Bacteriology, 557–66. Chichester, UK: John Wiley & Sons, Ltd, 2006. http://dx.doi.org/10.1002/9780470017968.ch46.

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Friedman, Gerald. "Clostridium difficile." In Mount Sinai Expert Guides, 411–21. Oxford, UK: John Wiley & Sons, Ltd, 2014. http://dx.doi.org/10.1002/9781118932759.ch39.

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Gerding, Dale N., and Stuart Johnson. "Clostridium difficile." In Bacterial Infections of Humans, 273–82. Boston, MA: Springer US, 2009. http://dx.doi.org/10.1007/978-0-387-09843-2_13.

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Magdesian, K. Gary. "Clostridium difficile." In Interpretation of Equine Laboratory Diagnostics, 197–201. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2017. http://dx.doi.org/10.1002/9781118922798.ch35.

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Andersen, Bjørg Marit. "Clostridium difficile." In Prevention and Control of Infections in Hospitals, 755–64. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-99921-0_53.

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Bhunia, Arun K. "Clostridium botulinum, Clostridium perfringens, Clostridium difficile." In Foodborne Microbial Pathogens, 209–28. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7349-1_12.

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Conference papers on the topic "Clostridium difficile"

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Lipman, L. J. A., N. E. M. Hopman, and E. C. Keessen. "Clostridium difficile in a farrowing pen." In Ninth International Conference on the Epidemiology and Control of Biological, Chemical and Physical Hazards in Pigs and Pork. Iowa State University, Digital Press, 2011. http://dx.doi.org/10.31274/safepork-180809-624.

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Berhane, F., L. Leys, L. Moeng, A. Thomas, and V. Poddar. "Atypical Presentation of Clostridium Difficile Infection." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a6921.

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Cama, Valeria, Luisa Pieragostini, Giovanna Fontanelli, Maria Rosa Velletri, and Calafiore Mariarosa. "P387 Clostridium difficile severe infection in a newborn." In Faculty of Paediatrics of the Royal College of Physicians of Ireland, 9th Europaediatrics Congress, 13–15 June, Dublin, Ireland 2019. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2019. http://dx.doi.org/10.1136/archdischild-2019-epa.733.

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Matos, Eduarda Syhara Rocha, Costa, D.V.S., Martins, C.S., Oliveira, L.C., Silva, A.M.H.P., Martins, D.S., Pimentel, P.V.S., and Brito, G.A.C. "Alterações neuroimunes induzidas por toxinas do Clostridium difficile." In II Encontro do Programa de Pós-Graduação em Ciências Farmacêuticas da Universidade Federal do Ceará e I Simpósio Norte-Nordeste de Ciências Farmacêuticas. Fortaleza - CE, Brazil: Galoa, 2017. http://dx.doi.org/10.17648/ppgcf-2017-64895.

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Thompson, Theresa, and Jay Pasquantonio. "High-intensity UV LED inactivation of Clostridium difficile spores." In Photonic Diagnosis, Monitoring, Prevention, and Treatment of Infections and Inflammatory Diseases 2019, edited by Tianhong Dai, Mei X. Wu, and Jürgen Popp. SPIE, 2019. http://dx.doi.org/10.1117/12.2507993.

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Harvey, Roger B., Keri N. Norman, Kathleen Andrews, Bo Norby, Michael E. Hume, and H. Morgan Scott. "Clostridium difficile in pork and retail meat in Texas." In Ninth International Conference on the Epidemiology and Control of Biological, Chemical and Physical Hazards in Pigs and Pork. Iowa State University, Digital Press, 2011. http://dx.doi.org/10.31274/safepork-180809-620.

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Prohaska, C., and M. F. Ragland. "A Toxic Combination: Clostridium Difficile Infection Leading to Miscarriage." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6573.

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Rabold, D., A. Schellin, J. Brombach, K. Teske, H. Pischon, L. Mundhenk, M. Fulde, C. Seyboldt, and A. Lübke-Becker. "Reptilien: Ein bislang unterschätztes zoonotisches Risiko für Clostridium difficile-Infektionen?" In 62. Jahrestagung der Fachgruppe Pathologie der Deutschen Veterinärmedizinischen Gesellschaft. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1688596.

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Harvey, Roger B., K. Norman, M. Hume, K. Andrews, H. Scott, B. Norby, C. Scanlan, Robin C. Anderson, and David J. Nisbet. "Survey of Clostridium difficile in Food Animals and Retail Meats." In Eighth International Symposium on the Epidemiology and Control of Foodborne Pathogens in Pork. Iowa State University, Digital Press, 2009. http://dx.doi.org/10.31274/safepork-180809-884.

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Allegretti, Jessica, and Karin Drooff. "Oral microbes effective for prevention of recurrent Clostridium difficile infections." In Digestive Disease Week 2022, edited by Rachel Giles. Baarn, the Netherlands: Medicom Medical Publishers, 2022. http://dx.doi.org/10.55788/4995cbe9.

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Reports on the topic "Clostridium difficile"

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Muldrow, Lycurgus L., and Joe Johnson. Genetic Engineering of Clostridium Difficile Toxin A Vaccine. Fort Belvoir, VA: Defense Technical Information Center, September 1991. http://dx.doi.org/10.21236/ada242265.

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Muldrow, Lycurgus L., and Joe Johnson. Genetic Engineering of Clostridium Difficile Toxin a Vaccine. Fort Belvoir, VA: Defense Technical Information Center, August 1990. http://dx.doi.org/10.21236/ada230411.

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Neumann, Charlotte, and Uzo Chukwuma. Clostridium difficile Infection in the Department of Defense (DOD): 2007-2013. Fort Belvoir, VA: Defense Technical Information Center, February 2015. http://dx.doi.org/10.21236/ada618664.

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Giesen, Christine, Angélica Ortega-Torres, Inmaculada López-Carrillo, Laura López-Vázquez, Carmen María Saa-Requejo, and Cristine García-Fernández. Evolución de la Detección de Clostridium Difficile en un Hospital de Segundo Nivel en Madrid, 2019-2021. Peeref, September 2022. http://dx.doi.org/10.54985/peeref.2209p4674900.

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Ciapponi, Agustín. What is the effectiveness of interventions to improve antibiotic prescribing practices for hospital inpatients? SUPPORT, 2016. http://dx.doi.org/10.30846/1610132.

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In-hospital infections caused by antibiotic-resistant bacteria and Clostridium difficile are associated with higher rates of death, illness and prolonged hospital stay which is a serious problem for patients and healthcare systems. These infections occur because antibiotics are used too often and incorrectly.

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Faecal transplant effectively treats recurrent or unresponsive Clostridium difficile. National Institute for Health Research, November 2017. http://dx.doi.org/10.3310/signal-000506.

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