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1

Jones, P. F. "Cloning and expression of tyrosine kinase genes." Thesis, University of Oxford, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.382626.

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2

Suen, Ki-Ling. "Cloning of protein kinase genes from a carrot cDNA library." Diss., Georgia Institute of Technology, 1991. http://hdl.handle.net/1853/25431.

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3

Schweighoffer, Edina. "The role of Syk protein tyrosine kinase in B cell development and function." Thesis, Open University, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.250493.

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4

Coffer, Paul James. "The identification, cloning and characterisation of novel mammalian protein-serine kinase genes." Thesis, Open University, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.293298.

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5

Moncrieff, Colin Lindsay. "Cloning and characterisation of a novel DMPK-related gene : CDC42BPB." Thesis, University of Glasgow, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.323439.

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6

Chen, Yi-Jun Grace. "Cloning and expression of three protein kinase genes of Colletotrichum gloeosporioides f. sp. malvae." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp03/MQ40402.pdf.

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7

Pylayeva, Yuliya. "Role of focal adhesion kinase in mammary gland tumorigenesis /." Access full-text from WCMC, 2008. http://proquest.umi.com/pqdweb?did=1528351831&sid=1&Fmt=2&clientId=8424&RQT=309&VName=PQD.

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8

Prata, Rodrigo Ferreira. "Papel do Tyrosine receptor Kinase B (TrkB) na regulação de genes do metabolismo hepático de colesterol e triglicerídeos." reponame:Repositório Institucional da UFABC, 2012.

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9

Summy, Justin Matthew. "Functional domain contributions to signaling specificity between the non-receptor tyrosine kinases c-src and c-yes." Morgantown, W. Va. : [West Virginia University Libraries], 2001. http://etd.wvu.edu/templates/showETD.cfm?recnum=2239.

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Thesis (Ph. D.)--West Virginia University, 2001.
Title from document title page. Document formatted into pages; contains vi, 195 p. : ill. (some col.). Vita. Includes abstract. Includes bibliographical references (p. 182-190).
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10

AGNES, FRANCOIS. "Etude de la structure des genes codant les recepteurs tyrosine kinase avec des domaines de type immunoglobuline : un modele d'evolution moleculaire." Paris 6, 1995. http://www.theses.fr/1995PA066005.

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Les recepteurs tyrosine kinase (rtk) possedant cinq, trois et sept domaines de type immunoglobuline dans leur region extracytoplasmique se repartissent respectivement dans les sous-classes iii, iv et v. Les genes humains qui codent les recepteurs des sous-classes iii et v sont groupes en paire ou en triplet sur trois chromosomes. Les genes kit et fms (rtkiii) ont une structure exon/intron identique dans la partie codant la region intracellulaire et similaire dans la partie codant la region extracellulaire ; ce sont deux genes homologues qui derivent probablement d'un ancetre commun. Sur la base de ces resultats et de la localisation en tandem des genes rtkiii, un modele d'evolution par duplication en cis et en trans concernant les genes de la sous-classe iii a ete postule. La structure exon/intron des genes flt3 et pdgfra (rtkiii) a ete determinee dans la partie codant la region intracellulaire. La comparaison de la structure de ces deux genes avec celle des genes kit et fms permet de confirmer le modele d'evolution par duplication en cis et en trans specifique des genes rtk de sous-classe iii. Elle montre que la structure exon/intron observee est une caracteristique des genes de la sous-classe iii. La structure genomique des genes flt4 (sous-classe v) et fgfr4 (sous-classe iv) a aussi ete determinee. Dans la partie codant la region intracellulaire, la comparaison de la structure de ces genes avec celle des genes de la sous-classe iii montre que les genes des sous-classes iii et v ont la meme structure exon/intron et que celle ci est similaire a celle des genes de la sous-classe iv. Ces resultats permettent d'elargir le modele d'evolution par duplications en cis et en trans aux genes de la sous-classe v et de proposer un origine ancestrale commune pour les genes des trois sous-classes. Dans la partie codant la region extracellulaire, la comparaison des genes des trois sous-classes montre qu'au sein de chaque sous-classe, la structure exon/intron est identique et que les genes appartenant aux sous-classes iii et v ont une structure similaire. Les analogies de structure mises en evidence entre des genes des trois sous-classes indiquent que l'ancetre commun etait probablement un gene codant un recepteur a trois domaines de type immunoglobuline. Sur la base de la conservation de la position d'un certain nombre d'introns, nous postulons que le gene ancestral etait deja partiellement morcele, qu'il contenait des exons plus grand et quelques introns, que les duplications ont concerne des genes entiers et que plusieurs introns se sont inseres, apres les duplications geniques, a des sites differents suivant les sous-classes
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11

朱江 and Jiang Zhu. "RET transcriptional regulation by HOXB5 in Hirschsprung's disease." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hdl.handle.net/10722/193397.

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Hirschsprung’s disease (HSCR) is the major enteric nervous system anomaly affecting newborns with high incidence in Asians. HSCR is a congenital complex genetic disorder characterized by a lack of enteric ganglia along a variable length of the intestine. The receptor tyrosine kinase gene (RET) is the major HSCR gene and cis-elements in the promoter and intron of RET gene are crucial for RET expression. Abnormal RET expression leading to insufficient RET activity causes defective development of the enteric nervous system and is implicated in the pathogenesis of the Hirschsprung’s disease. The human homeobox B5, HOXB5, has an important role in the development of enteric neural crest cells, and perturbation of HOXB5 signaling causes reduced RET expression and HSCR phenotypes in mice. To investigate the roles of HOXB5 in the regulation of RET expression and in the aetiology of HSCR, I sought to(i) elucidate the underlying mechanisms that HOXB5 mediates RET expression, and (ii) to examine the interactions between HOXB5 and other transcription factors including SOX10 and NKX2-1 that have been implicated in RET expression and HSCR. In this study, I demonstrated that HOXB5 binds to the RET promoter and regulates RET expression. HOXB5 and NKX2-1 forma protein complex and mediate RET expression in a synergistic manner. In contrast, HOXB5 cooperates in an additive manner with SOX10in trans-activation from RET promoter. ChIP assay further revealed that HOXB5 and NKX2-1 interact with the same chromatin region proximate to the transcription start site of RET, suggesting that these two factors may interact with each other and regulate the transcription of RET. In silico analysis, EMSA and ChIP analysis showed that HOXB5 also binds to an enhancer element (MCS+9.7)in the intron 1 of RET gene, and HSCR-associated SNPs have been identified in this enhancer element. To further access the HOXB5 trans-activity onMCS+9.7, RET mini-gene was constructed by ligating the RET promoter to the 5’and MCS+9.7 to the 3’of a luciferase gene. Luciferase assay indicated that MCS+9.7 enhances the HOXB5 trans-activation from the RET promoter. In addition, previously identified HSCR-associated SNPs inintron 1 markedly reduce the HOXB5 trans-activation from the RET mini-gene. Moreover, the result of IP-LC-MS/MS indicated that HOXB5 could form protein-protein complexes with nuclear proteins involved in the transcription initiation of genes with TATA-less promoter. This evidence suggested that HOXB5 may cooperate with other activators or co-factors in the remodeling of chromatin conformation, local histone modification and recruitment of essential transcription factors for RNA Polymerase II based transcription from TATA-less promoter, such as RET. My data indicated that HOXB5 in coordination with other transcription factors mediates RET expression. Therefore, defects in cis-or trans-regulation of RET by HOXB5 could lead to a reduction of RET expression and contribute to the manifestation of the HSCR phenotype.
published_or_final_version
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12

Barker, Andrew Bellamy. "Expression, Cloning, and Sequencing of Putative Insulin Signaling Genes Involved in Diapause in the Flesh Fly Sarcophaga crassipalpis." Digital Commons @ East Tennessee State University, 2005. https://dc.etsu.edu/etd/1041.

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Diapause is a programmed developmental arrest that allows insects to survive harsh environmental conditions. The diapause state has been linked to the insulin signaling pathway. Insulin signaling has been associated with many physiological processes including aging. It is a working hypothesis that the diapause and aging programs have a common set of gene expression pathways via insulin signaling. Analysis of a heterologous microarray indicated that two genes involved in the insulin pathway were down regulated during diapause. Both of these genes, Pi3K68d and Pde6, along with the Insulin receptor (InR) were targets for further investigation. Putative gene products have been isolated, cloned, and sequenced. RNA interference experimentation was conducted to characterize the role of the putative InR gene products obtained.
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13

Ma, Grace. "Heterodimeric deoxyguanosine/deoxyadenosine kinase from Lactobacillus acidophilus R-26 : affinity protein purification, molecular cloning, sequence of the genes, and expression in E. coli /." The Ohio State University, 1993. http://rave.ohiolink.edu/etdc/view?acc_num=osu148784197535822.

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14

Salsman, Scott J. "Redox regulation of protein tyrosine phosphatases in cell membrane receptor-mediated signal transduction." Oklahoma City : [s.n.], 2005.

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15

Junior, Euclides Matheucci. "Isolamento e análise funcional do gene que codifica uma proteína serina-treonina quinase que modula a expressão de genes regulados por carboidratos em Trichoderma reesei." Universidade de São Paulo, 2000. http://www.teses.usp.br/teses/disponiveis/46/46131/tde-18092015-163705/.

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O gene TrSNF1, homólogo aos membros da subfamília das proteínas serina-treonina quinases ativadas por AMP (AMPK) e relacionadas a SNF1, foi isolado do fungo filamentoso trichoderma reesei. A seqüência de aminoácidos putativa possui um domínio de quinase com 42% de identidade e 59% de similaridade com outras proteínas quinases da mesma subfamília. Em S. cerevisiae a SNFl é essencial para a expressão de genes reprimidos por glicose, em resposta a privação de glicose do meio de cultura. A expressão de TrSNFl em levedura mutante para SNF1, restaura a função de SNF1. A expressão de um antisense de TrSNFl em T. reesei causa um atraso na expressão do gene regulado por glicose, CBHI. Além disso, em experimento utilizando matrizes de DNA foi possível observar uma alteração da tendência global da expressão gênica entre a cepa selvagem e a cepa antisense. A observação da homologia estrutural com proteínas quinase da mesma subfamília, a similaridade funcional com SNFl de S. cerevisiae, e a alteração no padrão da expressão gênica in vivo, sugerem que TrSNFl pode estar envolvido na regulação do metabolismo de carboidratos em T. reesei.
A gene homologue to the members of the AMP-activated/SNFl protein kinase subfamily, TrSNF1, was isolated from the filamentous fungus Trichoderma reesei. The predicted protein of 692 amino acids has a kinase domain, that share 42 % identity and 59 % similarity to that of serine/threonine protein kinase of this family. In Saccharomyces cerevisiae, the SNFl protein kinase is required for expression of glucose repressed genes in response to withdrawal of glucose from the medium. Expression of the Trichoderma reesei SNF1-related sequence in yeast SNFl mutant restores SNFl function. The TrSNFl antisense expression in T. reesei causes a control alteration in the glucose-regulated gene CBHI. The observed structural identity with the AMP-activated/SNFl protein kinase subfamily, and the functional similarity to the yeast SNFl suggest that the TrSNFl may be involved in the regulation of sugar metabolism in Trichoderma reesei.
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16

Chen, Min. "Studies of the SH2- and SH3- containing adaptor, Nck /." 1999. http://wwwlib.umi.com/dissertations/fullcit/9934031.

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17

chen, Hui-Lin, and 陳慧玲. "Cloning and characterization of a FGFR tyrosine kinase from Aedes aegypti." Thesis, 2001. http://ndltd.ncl.edu.tw/handle/27772796482258781416.

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18

Liao, chen yee, and 廖千億. "Cloning,Expression , and Structure studies of SH2 Domain of Bruton's Tyrosine Kinase." Thesis, 1997. http://ndltd.ncl.edu.tw/handle/09038241850831693475.

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19

陳俊宏. "Molecular cloning and characterization of kinase genes from lily (Lilium longiflorum)." Thesis, 2000. http://ndltd.ncl.edu.tw/handle/44629284594846786695.

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碩士
國立中興大學
農業生物科技學研究所
88
Signal transduction pathway involving protein kinase is required for flower development in plants. To study the signal transduction pathway regulated by protein kinase in lily, the cloning and characterization of protein kinase genes from lily is necessary. Using RT-PCR and 5’-RACE strategies, a cDNA showed high homology to ATPK1/ATPK6, a serine/threonine protein kinase gene with unknown function from Arabidopsis, was isolated from lily floral buds and named LPK1 (lily protein kinase gene 1). LPK1, which showed 64% identity and 74% similarity to ATPK1/ATPK6 with the highest homology (about 87% identity) in the central catalytic domain (about 243 amino acids), encodes a hydrophilic polypeptide of 479 amino acids with the centrally located catalytic domain containing all 11 conserved subdomains of eukaryotic protein kinase, and have the greatest homology to p70 ribosomal S6 kinases, protein kinase C and protein kinase A family of protein-serine kinases. The catalytic domain also contains motifs characteristic of protein-serine/threonine kinases such as GXGXXG and HRDLKPEN. Other motifs such as putative autophosphorylation site (XRXXSX) and a lysine-rich domain (42% lysine) were also found in the catalytic domain of LPK1. Northern blot analysis indicated that LPK1 was developmental regulated. LPK1 RNA was strongly detected in young floral buds and its expression was absent in mature floral buds. Low expression for LPK1 was also observed in leaf and stem. Ectopic expression of LPK1 in transgenic Arabidopsis plants reduced the petal and stamen growth by inhibiting the cell expansion. The 35S::LPK1 transgenic plants were sterile due to the reduction of filaments elongation of stamens. A 1911 bp cDNA encoding 510 amino acids of a lily pyruvate kinase (LPYK) was also cloned and characterized in this research during the cloning of LPK1 cDNA.
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20

Lee, Tzuu-Fen, and 李祖芬. "Cloning and Analysis of the Ripening-related Genes Encoding Protein Kinase in Banana." Thesis, 1997. http://ndltd.ncl.edu.tw/handle/26601213171978414721.

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碩士
國立臺灣大學
園藝學系研究所
85
Reversible protein phosphorylation mechanism is thought to play a key role in diverse biological signal transduction systems in animal, yeast, and plant. To identify protein kinases which functions during fruit ripening, we have isola ted several cDNA clones from Musa spp. using PCR and degenerate oligonucleotid es designed based on the conserved regions of the catalytic domain of protein kinases. One of the cDNA clones (pBPK3) was completely sequenced and character ized.The cDNA clone is 1172bp in length and encodes a polypeptide of 266 amino acids with a calculated molecular mass of 29,814 Da. The putative BPK3 protei n shows extensive homology to MAPKK genes from Arabidopsis, yeast and vertebra tes. Major (1.35kb) and minor (2.5kb) transcripts were detected in bract, anth er, and most abundantly in ripened banana fruit. The mRNA level of BPK3 increa sed markedly at stage 2 fruit, remained nearly constant during ripening stages and slightly decreased at stage 7 fruit. To investigate the structure of pro tein kinase genes, a genomic library of Musa spp. was screened using four cDNA s (pBPK3、pBPK6、pBPK9 and pBPK10) as probes. Thirty-three positive clones wer e obtained by screening 1.5x106 plaques under high-stringency hybridization co ndition. One of the genomic clone lBPSK70 was characterized and revealed the p resence of only one exon. The gene is 3644 bp long, and the encoded protein co ntains an open reading frame of 348 amino acids with calculated molecular mass of 38,329 Da and predicted pI of 9.41 . Sequence comparison revealed that pBP K3 cDNA was derived from BPSK70 gene. The two protein kinase genes fragments ( BPK6 and PK5) were also sequenced, and the predicted polypeptides show closely related to receptor-like serine/threonine kinase / or Raf and ribosomal prote in kinase, respectively. Northern analysis revealed that the two genes express ed in ripening banana fruit.The existence of these protein kinase genes has in dicated that the signaling pathway during fruit ripening may resemble eukaryot ic MAPK signaling cascades.
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21

MOHAMED, AHMED. "Identification of Genes Involved in the C. elegans VAB-1 Eph Receptor Tyrosine Kinase Signaling Pathway." Thesis, 2011. http://hdl.handle.net/1974/6615.

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The generation of a functional nervous system requires that neuronal cells and axons navigate precisely to their appropriate targets. The Eph Receptor Tyrosine Kinases (RTKs) and their ephrin ligands have emerged as one of the important guidance cues for neuronal and axon navigation. However, the molecular mechanisms of how Eph RTKs regulate these processes are still incomplete. The purpose of this work was to contribute to the understanding of how Eph receptors regulate axon guidance by identifying and characterizing components of the Caenorhabditis elegans Eph RTK (VAB-1) signaling pathway. To achieve this objective I utilized a hyper active form of the VAB-1 Eph RTK (MYR-VAB-1) that caused penetrant axon guidance defects in the PLM mechanosensory neurons, and screened for suppressors of the MYR-VAB-1 phenotype. Through a candidate gene approach, I identified the adaptor NCK-1 as a downstream effector of VAB-1. Molecular and genetic analysis revealed that the nck-1 gene encodes for two isoforms (NCK-1A and NCK-1B) that share similar expression patterns in parts of the nervous system, but also have independent expression patterns in other tissues. Genetic rescue experiments showed that both NCK-1 isoforms can function in axon guidance, but each isoform also has specific functions. In vitro binding assays showed that NCK-1 binds to VAB-1 in a kinase dependent manner. In addition to NCK-1, WSP-1/N-WASP was also identified as an effector of VAB-1 signaling. Phenotypic analysis showed that nck-1 and wsp-1 mutants had PLM axon over extension defects similar to vab-1 animals. Furthermore, VAB-1, NCK-1 and WSP-1 formed a complex in vitro. Intriguingly, protein binding assays showed that NCK-1 can also bind to the actin regulator UNC-34/Ena, but genetic experiments suggest that unc-34 is an inhibitor of nck-1 function. Through various genetic and biochemical experiments, I provide evidence that VAB-1 can disrupt the NCK-1/UNC-34 complex, and negatively regulate UNC-34. Taken together, my work provides a model of how VAB-1 RTK signaling can inhibit axon extension. I propose that activated VAB-1 can prevent axon extension by inhibiting growth cone filopodia formation. This is accomplished by inhibiting UNC-34/Ena activity, and simultaneously activating Arp2/3 through a VAB-1/NCK-1/WSP-1 complex.
Thesis (Ph.D, Biology) -- Queen's University, 2011-07-28 16:20:31.957
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22

Parnell, Laurence David. "Cloning and characterization of a serine/threonine protein kinase of Dictyostelium discoideum, with a phylogenetic analysis of serine/threonine and tyrosine protein kinases." 1993. http://catalog.hathitrust.org/api/volumes/oclc/31338222.html.

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Thesis (Ph. D.)--University of Wisconsin--Madison, 1993.
Typescript. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references (leaves 398-488).
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23

Hsu, Yi-Hsuan, and 許翼軒. "Cloning of Deoxynucleoside Monophosphate Kinase and Deoxynucleoside Diphosphate Kinase Genes from Thermus thermophilus HB8 and Establishment of a New Enzymatic Method for Biosynthesis of Deoxynucleoside Triphosphates." Thesis, 2014. http://ndltd.ncl.edu.tw/handle/3t59rs.

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碩士
國立清華大學
分子醫學研究所
102
Deoxynucleoside triphosphates (dNTP) are essential biological materials for synthesizing DNA. The compounds are also used in molecular biotechnology such as the polymerase chain reaction (PCR) and other DNA polymerase-based applications. Nowadays, dNTPs are commercially produced by chemical synthesis due to their low concentrations in living organisms. However, the chemical synthesis method utilizes toxic solvents and the yield is low. To solve these problems, this study attempt to establish an in vitro enzymatic method for complete biosynthesis of dNTP. The process involves two stages of enzymatic phosphorylation procedures. The deoxynucleoside monophosphates (dNMPs) are used in the first step. The products of the first and second stages are deoxynucleoside diphosphates (dNDP) and dNTP, respectively. Five genes from the Thermus thermophilus HB8, which encode four kinds of deoxynucleoside monophosphate kinases (NMKs) and one nucleoside diphosphate kinase (NDK), were cloned and expressed in high quality in E. coli BL21 (DE3) and NovaBlue (DE3). We have performed functional and kinetic property analysis of NMKs and NDK. The results showed that the five enzymes exhibited optimum activity at the pH value of 7.0 to 8.0 and at the temperature between 75OC to 80 OC. Furthermore, all of them displayed excellent thermal stability. Approximate 80% and 50% of enzyme activities were retained after incubation at 75 OC for 3 hr and 24hr, respectively. The kinetic property analysis showed that five enzymes have higher catalytic efficiency under optimum environment as compared to the mesophilic bacteria. Later, we combined these crude enzymes preliminarily to produce dNTP. The PCR performed using the enzymes-synthesized dNTP showed the same efficiency compared to the commercial one. Finally, this system provides a clean and better method of preparation that has potential for commercialization.
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24

Tice, David Alan. "The role of C-SRC in tumorigenesis /." 1999. http://wwwlib.umi.com/dissertations/fullcit/9930109.

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25

VanDusen, Nathan J. "Hand2 function within non-cardiomyocytes regulates cardiac morphogenesis and performance." Thesis, 2014. http://hdl.handle.net/1805/6170.

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Indiana University-Purdue University Indianapolis (IUPUI)
The heart is a complex organ that is composed of numerous cell types, which must integrate their programs for proper specification, differentiation, and cardiac morphogenesis. During cardiac development the basic helix-loop-helix transcription factor Hand2 is dynamically expressed within the endocardium and extra-cardiac lineages such as the epicardium, cardiac neural crest cells (cNCCs), and NCC derived components of the autonomic nervous system. To investigate Hand2 function within these populations we utilized multiple murine Hand2 Conditional Knockout (H2CKO) genetic models. These studies establish for the first time a functional requirement for Hand2 within the endocardium, as several distinct phenotypes including hypotrabeculation, tricuspid atresia, aberrant septation, and precocious coronary development are observed in endocardial H2CKOs. Molecular analyses reveal that endocardial Hand2 functions within the Notch signaling pathway to regulate expression of Nrg1, which encodes a crucial secreted growth factor. Furthermore, we demonstrate that Notch signaling regulates coronary angiogenesis via Hand2 mediated modulation of Vegf signaling. Hand2 is strongly expressed within midgestation NCC and endocardium derived cardiac cushion mesenchyme. To ascertain the function of Hand2 within these cells we employed the Periostin Cre (Postn-Cre), which marks cushion mesenchyme, a small subset of the epicardium, and components of the autonomic nervous system, to conditionally ablate Hand2. We find that Postn-Cre H2CKOs die shortly after birth despite a lack of cardiac structural defects. Gene expression analyses demonstrate that Postn-Cre ablates Hand2 from the adrenal medulla, causing downregulation of Dopamine Beta Hydroxylase (Dbh), a gene encoding a crucial catecholaminergic biosynthetic enzyme. Electrocardiograms demonstrate that 3-day postnatal Postn-Cre H2CKO pups exhibit significantly slower heart rates than control littermates. In conjunction with the aforementioned gene expression analyses, these results indicate that loss of Hand2 function within the adrenal medulla results in a catecholamine deficiency and subsequent heart failure.
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