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Journal articles on the topic 'Clinical diagnosis'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 May 2024

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1

Edwards, I. Ralph. "Clinical Diagnosis." Drug Safety 35, no. 4 (April 2012): 261–64. http://dx.doi.org/10.2165/11632340-000000000-00000.

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2

Maurer, Brian T. "Clinical diagnosis." JAAPA 35, no. 4 (April 2022): 66. http://dx.doi.org/10.1097/01.jaa.0000823156.40176.e6.

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3

Burr, Michael. "Clinical Diagnosis." Journal of the American Geriatrics Society 35, no. 1 (January 1987): 77. http://dx.doi.org/10.1111/j.1532-5415.1987.tb01324.x.

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4

Knebel, U., H. J. Brambs, and J. F. Riemann. "Clinical Diagnosis." Zeitschrift für Gastroenterologie 42, no. 09 (September 2004): 988–93. http://dx.doi.org/10.1055/s-2004-813509.

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5

Dickson, Robert A. "Clinical diagnosis." Current Orthopaedics 4, no. 1 (January 1990): 3–8. http://dx.doi.org/10.1016/0268-0890(90)90025-b.

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6

Nouraeinejad, Ali. "Choroideremia – A clinical insight and differential diagnosis." Oftalmologicheskii Zhurnal 98, no. 3 (June 20, 2022): 50–53. http://dx.doi.org/10.31288/oftalmolzh202235053.

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7

Hegde, Shubha V., and Khaja Javed Khan. "Clinical Evaluation: An Essential Tool for Diagnosis." Indian Journal of Anesthesia and Analgesia 143, no. 149 (December 15, 2023): 205–8. http://dx.doi.org/10.21088/ijaa.2349.8471.10423.9.

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Low back pain is one of the commonest and leading causes of hospital visits, functional limitation and absence from work in the world.1 Low back pain can occur at any age, but its highest prevalence is in third decade of life. Avascular necrosis of Hip commonly presents at the age of 35-50 years with mean age being 36 years.3 Presenting complaints of both lumbar spine and hip pathologies are overlapping which include low back pain with associated buttock, groin, anterior thigh, and knee pain. Therefore, identifying the exact pain generator becomes crucial, where clinical evaluation plays a vital role.
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8

Hassannezhad, Reshad. "Hyperketonemia: Clinical features and diagnosis of Diabetic Ketoacidosis." Endocrinology and Disorders 2, no. 5 (November 12, 2018): 01–04. http://dx.doi.org/10.31579/2640-1045/098.

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Diets that boost ketone production are increasingly used for treating several neurological disorders. Elevation in ketones in most cases is considered favorable, as they provide energy and are efficient in fueling the body's energy needs.Several physiological and pathological triggers, such as fasting, ketogenic diet, and diabetes cause an accumulation and elevation of circulating ketones. Complications of the brain, kidney, liver, and microvasculature were found to be elevated in diabetic patients who had elevated ketones compared to those diabetics with normal ketone levels. Diabetic ketoacidosis is an acute metabolic complication of diabetes characterized by hyperglycemia, hyperketonemia, and metabolic acidosis. Hyperglycemia causes an osmotic diuresis with significant fluid and electrolyte loss. DKA occurs mostly in type 1 diabetes mellitus (DM). It causes nausea, vomiting, and abdominal pain and can progress to cerebral edema, coma, and death. DKA is diagnosed by detection of hyperketonemia and anion gap metabolic acidosis in the presence of hyperglycemia. Treatment involves volume expansion, insulin replacement, and prevention of hypokalemia. Diabetic ketoacidosis (DKA) is a rare yet potentially fatal hyperglycemic crisis that can occur in patients with both type 1 and 2 diabetes mellitus. Due to its increasing incidence and economic impact related to the treatment and associated morbidity, effective management and prevention is key. Elements of management include making the appropriate diagnosis using current laboratory tools and clinical criteria and coordinating fluid resuscitation, insulin therapy, and electrolyte replacement through feedback obtained from timely patient monitoring and knowledge of resolution criteria. In addition, awareness of special populations such as patients with renal disease presenting with DKA is important. During the DKA therapy, complications may arise and appropriate strategies to prevent these complications are required. DKA prevention strategies including patient and provider education are important. This review aims to provide a brief overview of DKA from its pathophysiology to clinical presentation with in depth focus on up-to-date therapeutic management.
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9

Hassannezhad, Reshad. "Hyperketonemia: Clinical features and diagnosis of Diabetic Ketoacidosis." Endocrinology and Disorders 2, no. 5 (August 27, 2018): 01–04. http://dx.doi.org/10.31579/2640-1045/033.

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Diets that boost ketone production are increasingly used for treating several neurological disorders. Elevation in ketones in most cases is considered favorable, as they provide energy and are efficient in fueling the body's energy needs.Several physiological and pathological triggers, such as fasting, ketogenic diet, and diabetes cause an accumulation and elevation of circulating ketones. Complications of the brain, kidney, liver, and microvasculature were found to be elevated in diabetic patients who had elevated ketones compared to those diabetics with normal ketone levels. Diabetic ketoacidosis is an acute metabolic complication of diabetes characterized by hyperglycemia, hyperketonemia, and metabolic acidosis. Hyperglycemia causes an osmotic diuresis with significant fluid and electrolyte loss. DKA occurs mostly in type 1 diabetes mellitus (DM). It causes nausea, vomiting, and abdominal pain and can progress to cerebral edema, coma, and death. DKA is diagnosed by detection of hyperketonemia and anion gap metabolic acidosis in the presence of hyperglycemia. Treatment involves volume expansion, insulin replacement, and prevention of hypokalemia. Diabetic ketoacidosis (DKA) is a rare yet potentially fatal hyperglycemic crisis that can occur in patients with both type 1 and 2 diabetes mellitus. Due to its increasing incidence and economic impact related to the treatment and associated morbidity, effective management and prevention is key. Elements of management include making the appropriate diagnosis using current laboratory tools and clinical criteria and coordinating fluid resuscitation, insulin therapy, and electrolyte replacement through feedback obtained from timely patient monitoring and knowledge of resolution criteria. In addition, awareness of special populations such as patients with renal disease presenting with DKA is important. During the DKA therapy, complications may arise and appropriate strategies to prevent these complications are required. DKA prevention strategies including patient and provider education are important. This review aims to provide a brief overview of DKA from its pathophysiology to clinical presentation with in depth focus on up-to-date therapeutic management.
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10

Ramírez-Madrigal, Miguel A., Patricia Flores Troche, Juan Francisco Medina Ledesma, Esmeralda Hernández Lerma, and Mario Eduardo Elias Medina. "Kerion celsi: Clinical Features, Diagnosis and Therapeutic Approaches." INTERNATIONAL JOURNAL OF MEDICAL SCIENCE AND CLINICAL RESEARCH STUDIES 03, no. 07 (July 22, 2023): 1336–39. http://dx.doi.org/10.47191/ijmscrs/v3-i7-020.

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Querion de Celso, also known as Celso abscess, is a rare and severe inflammatory condition that arises as a complication of folliculitis. It is characterized by the formation of a purulent and fluctuant abscess in the pilosebaceous region. This dermatological disorder is primarily associated with the invasion of pathogenic bacteria, most commonly Staphylococcus aureus, into the hair follicles and sebaceous glands. Clinically, kerion de Celso presents as a prominent, erythematous, warm, and fluctuant lesion, often accompanied by local pain, restricted movement, and potential systemic symptoms such as fever and malaise. The diagnosis of kerion de Celso relies on a comprehensive evaluation, including clinical assessment, histopathological findings, and microbiological analysis. These diagnostic modalities support the presence of an intense inflammatory response and aid in identifying the causative agent. Treatment of kerion de Celso necessitates a multidisciplinary approach. Systemic antibiotics are essential to eliminate the underlying bacterial infection, while analgesics and anti-inflammatory agents provide symptomatic relief. Surgical incision and drainage may be required in cases of extensive abscess formation. In more complex situations, immunosuppressive therapy may be considered to modulate the exaggerated inflammatory response. Adequate wound care and close follow-up are crucial to ensure successful resolution and prevent long-term complications. In summary, Celso's kerion is a rare but potentially severe condition that requires prompt recognition and appropriate management to minimize complications and promote healing. Understanding the clinical, etiological, and therapeutic aspects of this condition is essential for healthcare professionals to deliver optimal care and improve clinical outcomes.
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11

Holub, Michal, Martina Sekowská, Dagmar Smetanová, Monika Koudová, Kateřina Sobolová, Alexandra Šinská, and Hynek Heřman. "Diagnosis of thanatophoric dysplasia using clinical exome screening." Česká gynekologie 88, no. 5 (October 30, 2023): 376–79. http://dx.doi.org/10.48095/cccg2023376.

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Summary: Bone dysplasias are a broad, heterogeneous group of diseases. Thanatophoric dysplasia is a rare bone dysplasia, but it is the most common lethal skeletal dysplasias. The major role in diagnostics plays a high-quality ultrasound examination in the 2nd trimester and the latest methods of genetic testing, including clinical exome testing. Knowing the correct diagnosis is crucial for the future of the fetus and the couple. Key words: skletal dysplasia – whole exome – thanaphoric dysplasia
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12

Wilson, J. N. "Clinical orthopaedic diagnosis." Journal of Bone and Joint Surgery. British volume 82-B, no. 4 (May 2000): 621–22. http://dx.doi.org/10.1302/0301-620x.82b4.0820621b.

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13

Mollan, R. A. B. "Diagnosis—(i) clinical." Current Orthopaedics 1, no. 1 (September 1986): 7–14. http://dx.doi.org/10.1016/s0268-0890(86)80004-4.

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14

Penny, R. H. C. "Veterinary clinical diagnosis." British Veterinary Journal 141, no. 5 (September 1985): 548. http://dx.doi.org/10.1016/0007-1935(85)90050-8.

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15

Laurence, M. "Clinical orthopaedic diagnosis." Journal of Bone and Joint Surgery. British volume 79-B, no. 2 (March 1997): 345. http://dx.doi.org/10.1302/0301-620x.79b2.0790345a.

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16

Sydorko, Igor, Roman Baitsar, and Oksana Plakhtii. "VALIDATION AND VERIFICATION OF MEASUREMENT METHODS IN CLINICAL DIAGNOSIS." Measuring Equipment and Metrology 82, no. 3 (2021): 26–31. http://dx.doi.org/10.23939/istcmtm2021.03.026.

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The article investigates and analyzes the validation and verification of measurement methods in the clinical diagnostic laboratory. The content and features of validation and verification are revealed. Measurement methods are considered in detail. Each direction of validation and verification of measurement methods is analyzed. The difference between validation and verification is substantiated. Measuring systems are increasingly used in the laboratories of the clinical sector. This means that the responsibility for validation lies mainly with the manufacturer. The laboratory may operate a validated methodology, which, for example, is published as a standard, or purchase a ready-made measuring system from a manufacturer for a specific application. In both cases, the main validation operate has already been done, but the laboratory must still confirm its ability to apply the method.
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17

Ramírez-Madrigal, Miguel A., Patricia Flores Troche, Juan Francisco Medina Ledesma, Esmeralda Hernández Lerma, and Mario Eduardo Elias Medina. "Kerion of Celso: Clinical Features, Diagnosis and Therapeutic Approaches." INTERNATIONAL JOURNAL OF MEDICAL SCIENCE AND CLINICAL RESEARCH STUDIES 03, no. 07 (July 15, 2023): 1336–39. http://dx.doi.org/10.47191/ijmscrs/v3-i7-20.

Full text
Abstract:
Querion de Celso, also known as Celso abscess, is a rare and severe inflammatory condition that arises as a complication of folliculitis. It is characterized by the formation of a purulent and fluctuant abscess in the pilosebaceous region. This dermatological disorder is primarily associated with the invasion of pathogenic bacteria, most commonly Staphylococcus aureus, into the hair follicles and sebaceous glands. Clinically, kerion de Celso presents as a prominent, erythematous, warm, and fluctuant lesion, often accompanied by local pain, restricted movement, and potential systemic symptoms such as fever and malaise. The diagnosis of kerion de Celso relies on a comprehensive evaluation, including clinical assessment, histopathological findings, and microbiological analysis. These diagnostic modalities support the presence of an intense inflammatory response and aid in identifying the causative agent. Treatment of kerion de Celso necessitates a multidisciplinary approach. Systemic antibiotics are essential to eliminate the underlying bacterial infection, while analgesics and anti-inflammatory agents provide symptomatic relief. Surgical incision and drainage may be required in cases of extensive abscess formation. In more complex situations, immunosuppressive therapy may be considered to modulate the exaggerated inflammatory response. Adequate wound care and close follow-up are crucial to ensure successful resolution and prevent long-term complications. In summary, Celso's kerion is a rare but potentially severe condition that requires prompt recognition and appropriate management to minimize complications and promote healing. Understanding the clinical, etiological, and therapeutic aspects of this condition is essential for healthcare professionals to deliver optimal care and improve clinical outcomes.
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18

K, Tiwari. "Invasive Aspergillosis: Etiology, Clinical Presentation, Laboratory Diagnosis and Treatment." Journal of Natural & Ayurvedic Medicine 4, no. 3 (July 6, 2020): 1–5. http://dx.doi.org/10.23880/jonam-16000257.

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Aspergillosis is an opportunistic infection, usually seen in the patients having underlying diseases especially acquired immuno deficiency syndrome (AIDS). The present review article highlights the mechanism of infection, virulence factors, latest diagnostic methods and treatment strategies of aspergillosis in a precise manner.
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19

Agnihotri Upender Kumar, Aastha. "Annular Pancreas in Adults: Clinical Presentations, Diagnosis, and Management." International Journal of Science and Research (IJSR) 12, no. 8 (August 5, 2023): 1035–36. http://dx.doi.org/10.21275/sr23810134015.

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20

Aihole, Jayalaxmi Shripati. "Testicular torsion; clinical diagnosis or imaging diagnosis?" Radiology Case Reports 17, no. 8 (August 2022): 2665–67. http://dx.doi.org/10.1016/j.radcr.2022.05.010.

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21

Andreev, Maksim N., Ekaterina Y. Fedotova, Rodion N. Konovalov, and Sergey N. Illarioshkin. "Clinical polymorphism of multiple system atrophy: a clinical case series." Almanac of Clinical Medicine 50, no. 5 (December 8, 2022): 310–14. http://dx.doi.org/10.18786/2072-0505-2022-50-044.

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Multiple system atrophy (MSA) is a neurodegenerative disorder characterized by autonomous insufficiency and motor abnormalities, such as akinetic rigid syndrome and cerebellar ataxia. The diagnosis of this disorder is challenging, and quite frequently the patients are seen by other specialties or with other diagnoses. The paper presents three clinical observations of patients with various phenotypic MSA subtypes. In two of them, the diagnosis of MSA was clinically proven and in the third one, clinically probable. All patients were initially followed up with other diagnoses. The authors describe specifics of complaints and past history registration, neurological examination and typical signs of MSA at neuroimaging.
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22

Choi, Seong Hye. "Clinical Diagnosis of Dementia." Journal of Korean Diabetes 13, no. 3 (2012): 133. http://dx.doi.org/10.4093/jkd.2012.13.3.133.

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23

Vakharia, Kavita T. "Clinical Diagnosis and Classification." Clinics in Plastic Surgery 48, no. 4 (October 2021): 577–85. http://dx.doi.org/10.1016/j.cps.2021.06.006.

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24

Tsvelev, U. V., V. F. Bejenar, S. А. Povzun, and D. В. Fridman. "Clinical diagnosis of adenomyosis." Journal of obstetrics and women's diseases 54, no. 3 (November 1, 2005): 91–98. http://dx.doi.org/10.17816/jowd83488.

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Literature review is performed of modern adenomyosis diagnostics methods, morphologic changes of myometrium, producing diagnostic phenomena are described, comparative analysis of diagnostic value is performed.
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25

Rowsey, J. James, Harold Jensen, and Daniel J. Sexton. "CLINICAL DIAGNOSIS OF ENDOPHTHALMITIS." International Ophthalmology Clinics 27, no. 2 (1987): 82–88. http://dx.doi.org/10.1097/00004397-198702720-00004.

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26

Joob, Beuy, and Viroj Wiwanitkit. "Clinical diagnosis in Ayurveda." Ancient Science of Life 32, no. 4 (2013): 262. http://dx.doi.org/10.4103/0257-7941.131992.

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27

Peiris, Alan N., Dima Youssef, Tamra Ranasinghe, and Tammy Monk. "Spot Clinical Diagnosis (Revisited)." Southern Medical Journal 107, no. 1 (January 2014): 57. http://dx.doi.org/10.1097/smj.0000000000000045.

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28

Schutt, W. H. "THUMBS IN CLINICAL DIAGNOSIS." Developmental Medicine & Child Neurology 13, no. 3 (November 12, 2008): 393–94. http://dx.doi.org/10.1111/j.1469-8749.1971.tb03281.x.

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29

Bolodeoku, J., and D. Donaldson. "Urinalysis in clinical diagnosis." Journal of Clinical Pathology 49, no. 8 (August 1, 1996): 623–26. http://dx.doi.org/10.1136/jcp.49.8.623.

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30

Lovelock, James E. "Ultrasonics in Clinical Diagnosis." Radiology 154, no. 1 (January 1985): 178. http://dx.doi.org/10.1148/radiology.154.1.178.

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31

Coll de Tuero, Gabriel, Joan Bayó Llibre, Antonio Rodriguez Poncelas, Carme Roca Saumell, Marc Saez, Quintí Foguet Boreu, Narcís Salleres Marcó, Antoni Dalfó Baqué, M. Rosa Senán Sanz, and Mireia Ventura Fontanet. "Isolated clinical hypertension diagnosis." Blood Pressure Monitoring 16, no. 1 (February 2011): 11–15. http://dx.doi.org/10.1097/mbp.0b013e32834331c3.

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32

Widiger, Thomas A., and Timothy J. Trull. "Diagnosis and Clinical Assessment." Annual Review of Psychology 42, no. 1 (January 1991): 109–33. http://dx.doi.org/10.1146/annurev.ps.42.020191.000545.

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33

Jana, Atanu Kumar, and S. Sridhar. "Clinical diagnosis of sepsis." Journal of Neonatology 23, no. 1 (March 2009): 44–47. http://dx.doi.org/10.1177/0973217920090107.

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34

Yorke, Clifford. "Diagnosis in Clinical Practice." Psychoanalytic Study of the Child 51, no. 1 (January 1996): 190–214. http://dx.doi.org/10.1080/00797308.1996.11822427.

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35

de A. Nishioka, Sérgio. "Clinical diagnosis of malaria." Transactions of the Royal Society of Tropical Medicine and Hygiene 90, no. 4 (July 1996): 446. http://dx.doi.org/10.1016/s0035-9203(96)90542-8.

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36

Sherwood, R. "Clinical diagnosis in China." Trends in Biotechnology 4, no. 2 (February 1986): 27–28. http://dx.doi.org/10.1016/0167-7799(86)90147-2.

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37

Pugliese, Gina, and Martin S. Favero. "Clinical Diagnosis of Influenza." Infection Control & Hospital Epidemiology 22, no. 11 (November 2001): 731. http://dx.doi.org/10.1017/s0195941700072817.

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38

Bihari, D. J. "Septicaemia--the clinical diagnosis." Journal of Antimicrobial Chemotherapy 25, suppl C (January 1, 1990): 1–7. http://dx.doi.org/10.1093/jac/25.suppl_c.1.

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39

&NA;. "Clinical and Diagnosis Issues." Psychiatric Genetics 5, Supplement (August 1995): 107. http://dx.doi.org/10.1097/00041444-199508001-00034.

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40

Richardson, Malcolm, and Michael Ellis. "Clinical and laboratory diagnosis." Hospital Medicine 61, no. 9 (September 2000): 610–14. http://dx.doi.org/10.12968/hosp.2000.61.9.1416.

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41

Robillard, Alain. "Clinical diagnosis of dementia." Alzheimer's & Dementia 3, no. 4 (October 2007): 292–98. http://dx.doi.org/10.1016/j.jalz.2007.08.002.

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42

Kasagi, Kanji. "Clinical diagnosis: Muscular sarcoidosis." Annals of Nuclear Medicine 15, no. 4 (August 2001): 386. http://dx.doi.org/10.1007/bf02988249.

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43

Kawamoto, Masami. "Clinical diagnosis: Retroperitoneal fibrosis." Annals of Nuclear Medicine 17, no. 1 (February 2003): 68. http://dx.doi.org/10.1007/bf02988262.

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44

Ronai, Zeev, and Marina Yakubovskaya. "Pcr in clinical diagnosis." Journal of Clinical Laboratory Analysis 9, no. 4 (1995): 269–83. http://dx.doi.org/10.1002/jcla.1860090409.

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45

Cohen, Robert M. "Epidemiology and Clinical Diagnosis." PET Clinics 8, no. 4 (October 2013): 391–405. http://dx.doi.org/10.1016/j.cpet.2013.08.001.

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46

Loscalzo, Ingrid Llovera, James Ryan, John Loscalzo, Andrew Sama, and Stefan Cadag. "Tetanus: A clinical diagnosis." American Journal of Emergency Medicine 13, no. 4 (July 1995): 488–90. http://dx.doi.org/10.1016/0735-6757(95)90150-7.

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47

Perrotta, G. "Bipolar disorder: definition, differential diagnosis, clinical contexts and therapeutic approaches." Neuroscience and Neurological Surgery 5, no. 1 (November 18, 2019): 01–06. http://dx.doi.org/10.31579/2578-8868/097.

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Starting from the general concept of "bipolarity", the present work focuses on the essential aspects of the disorder defining the clinical and diagnostic contexts, laying the foundations for correct differential diagnosis, without neglecting the neural characteristics elaborated in the scientific community. The discussion concludes with the best therapeutic approaches suggested on the subject., hypothesizing that borderline disorder may be the evolution of bipolar disorder.
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48

Velnic, Andreea-Alexandra, Bianca Hanganu, Valentin Petre Ciudin, Dragoș Crauciuc, Irina Smaranda Manoilescu, and Beatrice Gabriela Ioan. "Clinical diagnosis versus autopsy diagnosis in head trauma." Romanian Neurosurgery 31, no. 4 (December 20, 2017): 455–64. http://dx.doi.org/10.1515/romneu-2017-0072.

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Abstract The correct and complete diagnosis is essential for the adequate care and the favourable clinical evolution of the patients with head trauma. Purpose: To identify the error rate in the clinical diagnosis of head injuries as shown in comparison with the autopsy diagnosis and to identify the most common sources of error. Material and method: We performed a retrospective study based on data from the medical files and the autopsy reports of patients with head trauma who died in the hospital and underwent forensic autopsy. We collected: demographic data, clinical and laboratory data and autopsy findings. To quantify the concordance rate between the clinical diagnosis of death and the autopsy diagnosis we used a 4 classes classification, which ranged from 100% concordance (C1) to total discordance (C4) and two classes of partial discordance: C2 (partial discordance in favour of the clinical diagnosis- missing injuries in the autopsy reports) and C3 (partial discordance in favor of the necroptic diagnosis- missing injuries in the medical files). Data were analyzed with SPSS version 20.0. Results: We analyzed 194 cases of death due to head injuries. We found a total concordance between the clinical death diagnosis and autopsy diagnosis in 30.4% of cases and at least one discrepancy in 69.6% of cases. Increasing the duration of hospitalization directly correlates with the amount of the imaging investigations and these in turn correlates with an increased rate of diagnosis concordance. Among the patients with stage 3 coma who associated a spinal cord injury, we found a partial diagnosis discordance in 50% of cases and a total discordance in 50% of cases, possibly due to the need for conducting emergency imaging investigation and the need for surgical treatment. In cases with partial and total discordant diagnosis, at least one lesion was omitted in 45.1% of the cases. The most commonly omitted injuries in C2 cases were subdural hematoma, intracerebral hematoma and ventricular hemorrhage (21.6%). In C3 cases the most frequently omitted injuries were subarachnoidian hemorrhage and skull base fractures (17.9%). Conclusions: The clinical cause of death is not always concordant with the autopsy diagnosis. Autopsy may identify the inconsistencies in diagnosis, the injuries frequently skipped and the factors favoring the discordance rate between the clinical death diagnosis and the autopsy diagnosis, making it a valuable tool for improving the clinical care of the patients with head trauma.
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49

Mallet, Jasmina, Caroline Dubertret, and Olivier Huillard. "Clinical Diagnosis of Mental Disorders Before Cancer Diagnosis." JAMA Oncology 3, no. 4 (April 1, 2017): 565. http://dx.doi.org/10.1001/jamaoncol.2016.5293.

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50

De Pangher Manzini, Vincenzo, Maria Gloria Revignas, and Alessandro Brollo. "Diagnosis of malignant tumor: Comparison between clinical and autopsy diagnoses." Human Pathology 26, no. 3 (March 1995): 280–83. http://dx.doi.org/10.1016/0046-8177(95)90058-6.

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