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Journal articles on the topic 'Clinical description'

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Published: 9 November 2024

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1

Spiegel, David. "Clinical Description Introduction." American Journal of Psychiatry 151, no. 6 (June 1994): 90–96. http://dx.doi.org/10.1176/appi.ajp.151.6.90.

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2

CHIZH, ROMAN S., and ALBINA R. KHAKIMOVA. "LUMBOSACRAL PLEXOPATHY: CLINICAL CASE DESCRIPTION." Bulletin of Contemporary Clinical Medicine 11, no. 5 (October 2018): 143–48. http://dx.doi.org/10.20969/vskm.2018.11(5).143-148.

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3

Pachman, Lauren M. "Clinical description and epidemiology data." Clinical Immunology Newsletter 18, no. 10 (October 1998): 105–18. http://dx.doi.org/10.1016/s0197-1859(00)89058-5.

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4

Scharer, L. "Clinical Description of Nail Clubbing." JAMA: The Journal of the American Medical Association 286, no. 16 (October 24, 2001): 1972–73. http://dx.doi.org/10.1001/jama.286.16.1972.

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Parakhina, T. V., and U. I. Pryimych. "Weber-Christian Disease (Clinical Case Description)." PAIN. JOINTS. SPINE, no. 3.23 (January 5, 2017): 74–78. http://dx.doi.org/10.22141/2224-1507.3.23.2016.85011.

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6

Smirnova, L. A. Smirnova, and O. V. Simonova Simonova. "PARANEOPLASTIC DERMATOMYOSITIS: DESCRIPTION OF CLINICAL CASE." Pharmateca s2_2018 (May 3, 2018): 72–74. http://dx.doi.org/10.18565/pharmateca.2018.s2.72-74.

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7

Hamer, P. W., R. H. Holloway, R. Heddle, P. G. Devitt, and S. K. Thompson. "Type III achalasia—a clinical description." Diseases of the Esophagus 30, no. 8 (June 15, 2017): 1–6. http://dx.doi.org/10.1093/dote/dox051.

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8

Westen, Drew, and Joel Weinberger. "When clinical description becomes statistical prediction." American Psychologist 59, no. 7 (October 2004): 595–613. http://dx.doi.org/10.1037/0003-066x.59.7.595.

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9

Shafik, Ahmed. "Deflation reflex: Description and clinical significance." Anatomical Record 249, no. 3 (November 1997): 405–8. http://dx.doi.org/10.1002/(sici)1097-0185(199711)249:3<405::aid-ar12>3.0.co;2-s.

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Bulatov, Al'bert R., Igor' V. Litvinenko, Nikolay V. Tsygan, Sergey N. Bardakov, Aleksandra A. Boykova, Tatyana A. Kolesnik, and Lyudmila S. Onishchenko. "Nodopathy: clinic, diagnosis, treatment. Clinical description." Russian Military Medical Academy Reports 42, no. 4 (December 9, 2023): 427–35. http://dx.doi.org/10.17816/rmmar611154.

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AIM: Evaluation of the significance and possibilities of laboratory-instrumental diagnostic methods in establishing the diagnosis and selection of targeted therapy in patients with nodopathies. MATERIALS AND METHODS: System analysis of data from foreign and domestic literature with the presentation of a clinical case. RESULTS: Polyneuropathies are classified as demyelinating or axonal based on electrophysiological studies. However, in 2015, in addition to axonal and demyelinating neuropathies, it was proposed to distinguish a separate pathophysiological group — nodopathies. The pathogenesis of nodopathies may differ depending on the type of ion channels involved in the process, but always leads to a loss of excitability of the axon membrane; in the nodal region the membrane becomes inexcitable. Such neuropathies are characterized by transient conduction blocks followed by the development of axonal degeneration. Typical examples of nodopathies are acute motor axonal neuropathy, as well as multifocal motor neuropathy. Current pathophysiological understanding of specialized nodal regions (nodes of Ranvier) and associated axoglial proteins is growing. Hypotheses have been put forward about their role in the pathogenesis of immune-mediated attack on the peripheral myelinated axon. Recently, high titers of antibodies directed against a number of key adhesion molecules have been identified in both acute and chronic inflammatory neuropathies. These facts add to the differences in differential diagnosis between axonal and demyelinating peripheral neuropathies. New disease classification schemes based on seropositivity, improved electrophysiological and ultrasound classification, and identification of putative underlying pathological targets and mechanisms are being rapidly developed. CONCLUSION: Using our clinical example, we demonstrated the capabilities of laboratory and instrumental diagnostic methods in establishing a diagnosis in a patient with one of the forms of nodopathies — multifocal motor neuropathy.
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11

Crout, J. Richard. "A description of clinical pharmacology and the clinical pharmacologist." Clinical Pharmacology and Therapeutics 39, no. 4 (April 1986): 439. http://dx.doi.org/10.1038/clpt.1986.73.

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12

Takarangi, Melanie K. T., Jacinta M. Oulton, and Deryn Strange. "Explaining Memory Amplification: Is It All About the Test Format?" Clinical Psychological Science 6, no. 3 (December 8, 2017): 394–406. http://dx.doi.org/10.1177/2167702617744326.

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Trauma-exposed people commonly exhibit a “memory amplification” effect, endorsing exposure to more traumatic events over time. Studies reporting this phenomenon have typically relied on checklists, where participants read event descriptions and indicate (yes/no) their exposure. We examined whether that approach is vulnerable to response biases and memory errors. In two experiments, participants viewed negative photos and completed an Old-New recognition test. In Experiment 1, participants completed either a photo recognition test or description test—composed of written descriptions of negative photos. In Experiment 2, we measured analogue PTSD symptoms and participants completed the description test twice, 24 hr apart. Those in the description test condition performed worse on the memory test and were more biased to endorse negative photos compared with the photo test condition. Furthermore, this bias to endorse negative photos increased over time and was related to analogue PTSD symptoms. Overall, our findings suggest that test format plays a role in memory amplification.
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13

Friedenberg, RM, JB Lightfoote, SP Wang, and MF Smolin. "Digital tomography: description and preliminary clinical experience." American Journal of Roentgenology 144, no. 3 (March 1985): 639–43. http://dx.doi.org/10.2214/ajr.144.3.639.

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14

Piccolo, Vincenzo, Teresa Russo, Lilian Mathias Delorenze, and Giuseppe Argenziano. "Perinevic dermatosis neglecta: clinical and dermoscopic description." Anais Brasileiros de Dermatologia 94, no. 3 (May 2019): 361–62. http://dx.doi.org/10.1590/abd1806-4841.20197980.

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15

Suryono, Suryono, and Nasronudin Nasronudin. "Clinical Description and Diagnosis of HIV/AIDS." Indonesian Journal of Tropical and Infectious Disease 5, no. 1 (July 6, 2015): 23. http://dx.doi.org/10.20473/ijtid.v5i1.212.

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16

Buchinskaya, Natalia V., Eugenia A. Isupova, and Mikhail M. Kostik. "Homocystinuria: Literature Review and Clinical Case Description." Current Pediatrics 18, no. 3 (September 18, 2019): 187–95. http://dx.doi.org/10.15690/vsp.v18i3.2036.

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Homocystinuria is rare autosomal-recessive monogenic disorder associated with disturbance of methionine metabolism due to liver enzyme cystathionine--synthetase (CBS) deficit. That in turn causes elevated concentration of homocystein and its metabolites in blood and urine. The main clinical manifestations of homocystinuria are: myopia, ectopia lentis, psychomotor retardation, learning difficulties, mental retardation, mental illnesses, behaviour problems, paroxysms, extrapyramidal symptoms, skeletal anomalies (body height), long limbs — dolichostenomelia and arachnodactylia (Marfan Phenotype), pectus carinatum, valgus lower limbs, scoliosis, osteoporosis, thromboembolic disorders. Diagnostics of homocystinuria is based on clinical findings and laboratory changes (increase of methionine and homocysteine levels in serum). There is prenatal and DNA-diagnostics (genetic variants in CBS gene). Revealing of homocystinuria demands examination of first-degree relatives. Therapy of patients with homocystinuria includes not only diet therapy but also pyridoxine, folic acid, betaine administration. Syndromic concomitant therapy is also used. The description of the patient with severe B6-resistant form of homocystinuria is given in this article.
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17

Comstock Barker, Paige, and Jennifer S. Scherer. "Illness Trajectories: Description and Clinical Use #326." Journal of Palliative Medicine 20, no. 4 (April 2017): 426–27. http://dx.doi.org/10.1089/jpm.2016.0554.

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18

Grandjean, Michael. "New Abdominal Acupuncture: Description With Clinical Examples." Medical Acupuncture 22, no. 3 (September 2010): 197–201. http://dx.doi.org/10.1089/acu.2009.0733.

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19

GRISSO, THOMAS, and JUDITH QUINLAN. "Juvenile Court Clinical Services: A National Description." Juvenile and Family Court Journal 56, no. 4 (September 2005): 9–20. http://dx.doi.org/10.1111/j.1755-6988.2005.tb00175.x.

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20

Lopez, F., D. Muñoz, A. Mendoza, P. Parra, C. Rojas, A. Hernandez, S. Witting, L. Troncoso, M. Troncoso, and E. Marquez. "Electric sleep status: clinical and electroencephalographic description." Journal of the Neurological Sciences 357 (October 2015): e444. http://dx.doi.org/10.1016/j.jns.2015.09.088.

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21

Rajaraman, Revathi, Prajna Lalitha, Anita Raghavan, Manikandan Palanisamy, and Namperumalsamy Venkatesh Prajna. "Traumatic Lenticular Abscess: Clinical Description and Outcome." American Journal of Ophthalmology 144, no. 1 (July 2007): 144–46. http://dx.doi.org/10.1016/j.ajo.2007.02.040.

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22

Hupp, James R. "Digital tomography: Description and preliminary clinical experience." Journal of Oral and Maxillofacial Surgery 43, no. 10 (October 1985): 829. http://dx.doi.org/10.1016/0278-2391(85)90350-7.

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23

Shannon, Daniel C., David W. Carley Phd, and Dorothy H. Kelly. "Periodic breathing: Quantitative analysis and clinical description." Pediatric Pulmonology 4, no. 2 (1988): 98–102. http://dx.doi.org/10.1002/ppul.1950040207.

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24

Nikolaeva, D. S., A. N. Suinalieva, M. R. Shaidullina, L. S. Sozaeva, and M. A. Kareva. "Hippel-Lindau syndrome: description of clinical cases." Endocrine Surgery 17, no. 4 (December 25, 2023): 71. http://dx.doi.org/10.14341/serg12893.

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25

KISELEVA, T. A., D. R. ISLAMOVA, and K. R. BAGAUTDINOVA. "Insulinoma: literature review and clinical case description." Practical medicine 22, no. 4 (2024): 36–40. http://dx.doi.org/10.32000/2072-1757-2024-4-36-40.

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Insulinoma is a neuroendocrine tumor arising from β-cells of the islets of Langerhans, causing hypoglycemic attacks due to endogenous hyperinsulinism. The worldwide prevalence is approximately 1 case per 250,000–1,000,000 population, the incidence is 1–4 cases per million population per year. In most cases, the formation of insulinoma is sporadic (90%), less often it can be detected as part of the multiple endocrine neoplasia syndrome type 1 (10%). This review discusses the pathogenetic mechanisms of hypoglycemic syndrome, criteria for differential diagnosis, modern laboratory and imaging methods, and approaches to the treatment of insulinoma. A clinical observation is presented that demonstrates the difficulties of timely diagnosis of insulinoma due to nonspecific manifestations of hypoglycemia. The time that elapses from the first manifestations of hypoglycemic syndrome to diagnosis directly affects the prognosis and quality of life of a patient with insulinoma. It requires vigilance from doctors of all specialties regarding the clinical features of this disease.
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26

Galvão, Márcio Antônio Moreira, Cláudio Mafra, Chequer Buffe Chamone, Simone Berger Calic, Jorge E. Zavala-Velazquez, and David Hughes Walker. "Clinical and laboratorial evidence of Rickettsia felis infections in Latin America." Revista da Sociedade Brasileira de Medicina Tropical 37, no. 3 (June 2004): 238–40. http://dx.doi.org/10.1590/s0037-86822004000300009.

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After the discovery and initial characterization of Rickettsia felis in 1992 by Azad and cols, and the subsequent first description of a human case of infection in 1994, there have been two communications of human rickettsiosis cases caused by Rickettsia felis in Latin America. The first one was published in 2000 by Zavala-Velazquez and cols in Mexico. In 2001 Raoult and cols described the occurrence of two human cases of Rickettsia felis rickettsiosis in Brazil. In the present discussion these two articles were compared and after the description of the principal signs and symptoms, it was concluded that more studies are needed with descriptions of a greater number of patients to establish the true frequency of the clinical signs and symptoms present in Rickettsia felis rickettsiosis.
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27

R. Diaz Aguila, Hector. "Clinical Ultrasound. Essential tool for health care." Radiology Research and Diagnostic Imaging 2, no. 1 (February 9, 2023): 01–05. http://dx.doi.org/10.58489/2836-5127/006.

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A brief description of the clinical iltrasound is presented; its definition; main indications; advantages as well as its weaknesses. The POCUS learning curve is pointed out for personnel without prior knowledge of it.
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28

Ingula, N. I. "Clinical management of patients with vertebral lumbosacral radiculopathy. Description clinical cases." East European Journal of Neurology, no. 3(3) (December 20, 2015): 49–53. http://dx.doi.org/10.33444/2411-5797.2015.3(3).49-53.

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Lumbosacral radiculopathy one of the most difficult choices vertebrogenic pain syndromes characterized especially intense and prolonged pain, usually accompanied by a sharp restriction of mobility, and is the most common cause of disability. Most of the destruction of roots of spinal nerves caused by vertebral reasons the presence of a herniated disc, degenerative changes in the intervertebral joints, narrow spinal canal. This article describes a clinical case of practice management of patients with chronic vertebral lumbosacral radiculopathy. The main causes of pain in the lower back, the main approaches to the diagnosis and treatment of chronic pain. Special attention is paid to the differential diagnosis of chronic pain in his back and leg.
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29

Palma, J.-A., and F. Palma. "A probable cluster headache case from a textbook of 1726: Francisco Suárez de Rivera’s description." Cephalalgia 31, no. 11 (July 18, 2011): 1232–35. http://dx.doi.org/10.1177/0333102411413161.

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Background: Few descriptions of cluster and cluster-like headache made before the 19th century have been reported. Case description: We present a previously unreported early description of a probable cluster headache case made by Francisco Suárez de Rivera (1686− c.1751), one of the main physicians of the Spanish Age of Enlightenment, writer of almost 40 textbooks about medicine, surgery, pharmacology, and therapeutics. Discussion: The depiction here reported of a woman with probable cluster headache is possibly one of the earliest known and, to our knowledge, the first in Hispanic literature. We also review other descriptions of cluster and cluster-like headache from the same time period.
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Stilidi, I. S., E. R. Charchyan, K. I. Zhordania, V. J. Bokhian, N. A. Kozlov, and J. G. Pajanidi. "Intracardiac intravenous leiomyomatosis: description of a clinical case." Clinical and Experimental Surgery. Petrovsky journal 9, no. 1 (2021): 70–76. http://dx.doi.org/10.33029/2308-1198-2021-9-1-70-76.

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31

Komarova, L. N., E. I. Berzina, S. A. Zvezda, U. U. Bakumenko, L. A. Ked, M. A. Kiseleva, and K. U. Nabieva. "Cold agglutinin disease. Description of the clinical case." Medical Science And Education Of Ural 21, no. 4 (December 30, 2020): 62–64. http://dx.doi.org/10.36361/1814-8999-2020-21-4-62-64.

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The article presents a clinical case of admission, observation and treatment of a senior patient with a diagnosis: Primary (idiopathic) cold agglutinin disease. Coronary heart disease. Exertional angina pectoris. Functional class 1. One of the rather rare and non-standard diagnoses in the work of a clinical doctor on duty is presented. A spectrum of examinations of the blood from patients with this disease was presented. Cold agglutinin disease (CAD) is an autoimmune hemolytic anemia in which immunoglobulin M antibodies bind to red blood cells and fix complement, which leads to the development of hemolysis. The study was conducted on the basis of the Chuz “Klinicheskaya Bol’nitsa “Rzhd-Meditsina” (private healthcare facility «Clinical Hospital «Russian Railways-Medicine») in Tyumen in April 2019, where in admission department a clinical case was registered which included admission, observation and treatment of a patient who have been diagnosed with: Primary cold agglutinin disease. Coronary heart disease. Exertional angina. Functional class 1. Observation and treatment of a patient with a diagnosis of Primary cold agglutinin disease was registered in the admission department. Coronary heart disease. Exertional angina. The patient did not know that she was suffering from primary cold agglutinin disease, and was never before diagnosed with it. Aim. Description of the identified case of primary (idiopathic) cold agglutinin disease for a more complete understanding of the problem due to the rarity of the disease; analysis of the possibility of early diagnosis based on the example of a specific case. Material and methods. A retrospective analysis of the medical history of a patient with a rare disease - primary (idiopathic) cold agglutinin disease was carried out. The anamnesis of life and disease, clinical, laboratory and instrumental data were assessed. An example is given from the practice of the admission department of the private healthcare facility «Clinical Hospital «Russian Railways-Medicine» in Tyumen of the clinical observation of the patient with diagnosed primary (idiopathic) cold agglutinin disease. Conclusion. This case clearly demonstrates the classic course of primary cold agglutinin disease. Due to the sudden deterioration of the condition associated with hypothermia, we see the importance of paying close attention to a thorough anamnesis taking to identify the relationship with hypothermia, and with exacerbation of the disease in cold conditions. It is also important to take into account clinical symptoms characteristic of this pathology, one of which is - livedo. Also, when planning a treatment for patients with CAD it is necessary to take into consideration the features of the course and manifestation of not only the underlying disease, but also concomitant pathology which will make it possible to carry out the corresponding laboratory and instrumental tests earlier and prescribe a rational complex therapy.
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Cherednichenko, M. M., T. G. Zadorkina, N. V. Nekrasova, and E. M. Ovechkina. "Hereditary pigmentary dermatopathy (description of the clinical case)." Смоленский медицинский альманах, no. 2 (2022): 148–52. http://dx.doi.org/10.37903/sma.2022.2.30.

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33

Evseeva, A. L., S. V. Koshkin, V. V. Ryabova, and O. S. Kovrova. "Eixtragenital primary syphilis: a description of clinical cases." Vestnik dermatologii i venerologii 94, no. 5 (February 12, 2019): 72–76. http://dx.doi.org/10.25208/0042-4609-2018-94-5-72-76.

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34

Dudnyk, V. M., V. G. Furman, O. V. Kutsak, and O. I. Izyumets. "Heritable amegacariocytic thrombocytopenia: description of a clinical case." Reports of Vinnytsia National Medical University 26, no. 1 (March 28, 2022): 27–30. http://dx.doi.org/10.31393/reports-vnmedical-2022-26(1)-04.

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Annotation. Features of the clinical course and differential diagnosis of hereditary thrombocytopenia, characterized by the development of pancytopenia in childhood, are described. It was found that the main manifestation of this pathology is hemorrhagic syndrome, accompanied by the presence of polymorphic, polychrome, asymmetric hemorrhagic rashes, frequent bleeding from the mucous membranes. The Mpl gene (1p34) was sequestered to confirm the diagnosis and establish the genomic mutation. Complex diagnosis of this disease requires a comprehensive and interdisciplinary approach involving a coordinated team of hematologists, geneticists, immunologists, neurosurgeons and ophthalmologists.
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Dudnyk, V. M., V. G. Furman, O. V. Kutsak, and O. I. Izyumets. "Heritable amegacariocytic thrombocytopenia: description of a clinical case." Reports of Vinnytsia National Medical University 26, no. 1 (March 28, 2022): 27–30. http://dx.doi.org/10.31393/reports-vnmedical-2022-26(1)-04.

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Annotation. Features of the clinical course and differential diagnosis of hereditary thrombocytopenia, characterized by the development of pancytopenia in childhood, are described. It was found that the main manifestation of this pathology is hemorrhagic syndrome, accompanied by the presence of polymorphic, polychrome, asymmetric hemorrhagic rashes, frequent bleeding from the mucous membranes. The Mpl gene (1p34) was sequestered to confirm the diagnosis and establish the genomic mutation. Complex diagnosis of this disease requires a comprehensive and interdisciplinary approach involving a coordinated team of hematologists, geneticists, immunologists, neurosurgeons and ophthalmologists.
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Kocabaş, Engin, Gülsüm Gençoğlan, Ferdi Öztürk, and Aylin Türel Ermertcan. "Halo angioma: A new clinical and dermoscopical description." European Journal of Dermatology 21, no. 4 (July 2011): 617–18. http://dx.doi.org/10.1684/ejd.2011.1328.

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АКЧУРИН, С. В., Г. П. ДЮЛЬГЕР, И. В. АКЧУРИНА, В. С. БЫЧКОВ, and Е. С. ЛАТЫНИНА. "RECOMMENDATIONS FOR THE DESCRIPTION OF VETERINARY CLINICAL CASES." VESTNIK RIAZANSKOGO GOSUDARSTVENNOGO AGROTEHNOLOGICHESKOGO UNIVERSITETA IM P A KOSTYCHEVA, no. 4(48) (December 29, 2020): 5–10. http://dx.doi.org/10.36508/rsatu.2020.48.4.001.

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Проблема и цель. Проблемой в системе высшего ветеринарного образования является дефицит ветеринарных клинических кейсов на русском языке, находящихся в свободном доступе, что оказывает негативное влияние на процесс непрерывного повышения качества подготовки студентов ветеринарных специальностей и повышения квалификации практикующих врачей. Библиометрический анализ публикаций, проведенный по материалам библиотеки eLIBRARY, позволил установить, что доля работ, посвященных использованию кейсов в ветеринарии, в общем количестве публикаций на тему кейсов составляет всего 0,04 %. Целью настоящего исследования является создание рекомендаций по разработке ветеринарных клинических кейсов. Методология. На основании результатов анализа отечественной и зарубежной литературы, в первую очередь, ветеринарного и медицинского профилей, личной практики авторов ими разработаны и представлены рекомендации по составлению ветеринарных клинических кейсов. Результаты. В результате проведенных исследований предложена структура ветеринарного клинического кейса, который включает в себя 12 разделов (заголовок, контактная информация, ключевые слова, аннотация, введение, кейс, обсуждение кейса, благодарности, ссылки, приложение, разрешения, конфликт интересов) и содержит рекомендации по написанию каждого из разделов. Разработанные рекомендации предлагают практикующим ветеринарным врачам, исследователям, преподавателям и студентам готовую структуру для написания и публикации кейсов, что будет способствовать увеличению числа публикуемых случаев из практики. Заключение. Большое количество и доступность качественных и актуальных кейсов в области ветеринарии, безусловно, будет способствовать повышению квалификации ветеринарных кадров, а, следовательно, и росту качества оказания ветеринарных услуг населению. Ключевые слова: кейс, кейс-метод, структура кейса, ветеринарная медицина, образование. Timiryazev Problem and purpose. A problem in the system of higher veterinary education is the lack of freely available veterinary clinical cases in Russian, which has a negative impact on the process of continuous improvement of the quality of training veterinary students and advanced training of practicing doctors. The bibliometric analysis of publications based on eLIBRARY made it possible to establish that the share of works devoted to the use of cases in veterinary medicine in the total number of publications on cases is only 0.04 %. The purpose of this study is to create recommendations for the development of veterinary clinical cases. Methodology. Based on the results of the analysis of domestic and foreign literature of primarily veterinary and medical profles, and personal practice of the authors, recommendations were developed to prepare veterinary clinical cases. Results. As a result of the research, the structure of the veterinary clinical case is proposed, which includes 12 sections (title, contact information, key words, abstract, introduction, case, case discussion, acknowledgments, links, appendix, permissions, confict of interest) and contains recommendations for writing each of the sections. The developed guidelines ofer practicing veterinarians, researchers, teachers and students a readymade structure for writing and publishing cases, which will contribute to an increase in the number of published cases from practice. Conclusion. At the same time, a large number and availability of high-quality and relevant cases in the feld of veterinary medicine will undoubtedly contribute to an increase in the qualifcations of veterinary personnel, and, consequently, the quality of veterinary services to the population
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PA, Leye, Malissin I, Mora P, Voicu S, Elgharbi F, and Megarbane B. "Refractory Myasthenia Gravis: Clinical Description and Literature Review." Scholars Journal of Applied Medical Sciences 08, no. 01 (January 25, 2020): 171–73. http://dx.doi.org/10.36347/sjams.2020.v08i01.033.

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Ferral, Hector. "Hydrogel-Coated Coils: Product Description and Clinical Applications." Seminars in Interventional Radiology 32, no. 04 (November 10, 2015): 343–48. http://dx.doi.org/10.1055/s-0035-1564809.

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Miller, Linda T., Cheryl A. Missiuna, Jennifer J. Macnab, Theresa Malloy-Miller, and Helene J. Polatajko. "Clinical Description of Children with Developmental Coordination Disorder." Canadian Journal of Occupational Therapy 68, no. 1 (February 2001): 5–15. http://dx.doi.org/10.1177/000841740106800101.

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Borsa, R., M. Governa, G. Aimé, D. Fraire, E. Baralis, and G. Fontana. "Penetrating bladder injuries: Description of a clinical case." Urologia Journal 62, no. 1_suppl (January 1995): 190–91. http://dx.doi.org/10.1177/039156039506201s51.

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— A penetrating bladder injury in a youth due to impalement through the scrotum was successfully operated. Perforation was at the bottom and on the dome and the absence of a clinical uro-peritoneal picture was due to the plugging of the latter rupture by the omentum. Retrograde urethrocystography was negative and only pelvic ultrasound characterised the diagnosis, leading to an immediate explorative surgical approach.
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Marszal, Elzbieta, Ewa Jamroz, Jacek Pilch, Ewa Kluczewska, Halina Jablecka-Deja, and Robert Krawczyk. "Agenesis of Corpus Callosum: Clinical Description and Etiology." Journal of Child Neurology 15, no. 6 (June 2000): 401–5. http://dx.doi.org/10.1177/088307380001500609.

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Lau, William, and David Pye. "A Clinical Description of Ocular Response Analyzer Measurements." Investigative Opthalmology & Visual Science 52, no. 6 (May 2, 2011): 2911. http://dx.doi.org/10.1167/iovs.10-6763.

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Weidman, Hazel H. "Stylistic Aspects of Clinical Anthropology: A Mirrored Description." Medical Anthropology Newsletter 16, no. 3 (May 1985): 63–64. http://dx.doi.org/10.1525/maq.1985.16.3.02a00040.

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Weidman, Hazel H. "Stylistic Aspects of Clinical Anthropology: A Mirrored Description." Medical Anthropology Quarterly 16, no. 3 (May 1985): 63–64. http://dx.doi.org/10.1111/j.1937-6219.1985.tb00982.x.

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Kudrow, L., and P. Esperanca. "Conversion Cephalgia. A Clinical Description Of 100 Patients." Cephalalgia 7, no. 6_suppl (September 1987): 187–88. http://dx.doi.org/10.1177/03331024870070s663.

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Dzeranova, Larisa K., Natalya S. Fedorova, Svetlana Yu Vorotnikova, and Ekaterina A. Pigarova. "Description of clinical portraits of patients with hyperprolactinemia." Obesity and metabolism 15, no. 3 (November 23, 2018): 65–69. http://dx.doi.org/10.14341/omet9866.

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Hyperprolactinemia is one of the most common endocrine diseases in the practice of different specialists. Interdisciplinary approach and unified principles of patient management are extremely relevant for this pathology due to the variety of clinical forms. The article demonstrates typical clinical manifestations of hyperprolactinemic syndrome on the example of three patients and shows the different approach to management and treatment.
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Bergman, Nils J. "Clinical description of Parabuthus transvaalicus scorpionism in Zimbabwe." Toxicon 35, no. 5 (May 1997): 759–71. http://dx.doi.org/10.1016/s0041-0101(96)00041-4.

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Poppas, Dix P., Gary E. Lemack, and David T. Mininberg. "Laparoscopic Orchiopexy: Clinical Experience and Description of Technique." Journal of Urology 155, no. 2 (February 1996): 708–11. http://dx.doi.org/10.1016/s0022-5347(01)66506-6.

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Lanska, D. J., J. M. Lanska, and B. F. Remler. "Description and clinical application of the Pulfrich effect." Neurology 84, no. 22 (June 1, 2015): 2274–78. http://dx.doi.org/10.1212/wnl.0000000000001646.

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