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1

Jadhav AK, Jadhav AK, Mulla NH Mulla NH, Prasad HB Prasad HB, and Kadam DB Kadam DB. "Study of Clinical Course and Prognostic Factors Affecting Course of Septicemia." International Journal of Scientific Research 3, no. 2 (June 1, 2012): 397–99. http://dx.doi.org/10.15373/22778179/feb2014/130.

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&NA;. "Clinical Esophagology Course." Journal of Clinical Gastroenterology 13, no. 6 (December 1991): 722. http://dx.doi.org/10.1097/00004836-199112000-00026.

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3

Lawler, W. R., A. E. Dobbie, and F. D. Schneider. "A clinical integration course." Academic Medicine 74, no. 5 (May 1999): 592. http://dx.doi.org/10.1097/00001888-199905000-00063.

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4

Kennedy, P. F. "Course in Clinical Neurophysiology." Psychiatric Bulletin 10, no. 12 (December 1, 1986): 369. http://dx.doi.org/10.1192/pb.10.12.369-a.

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5

Brandner, Sebastian, Isabel Kellermann, Nirjhar Hore, Yavor Bozhkov, and Michael Buchfelder. "Clinical Course Score (CCS)." Journal of Neurosurgical Anesthesiology 27, no. 1 (January 2015): 26–30. http://dx.doi.org/10.1097/ana.0000000000000083.

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6

Mahishale, G. S. "Clinical course of patients with Tombstoning ST-Elevation Myocardial Infarction." Journal of Cardiovascular Medicine and Surgery 4, no. 3 (2018): 217–20. http://dx.doi.org/10.21088/jcms.2454.7123.4318.4.

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7

Prikhodko, O. B., I. V. Kostrova, M. V. Pogrebnaya, S. A. Goryacheva, and E. I. Smorodina. "FEATURES OF BRONCHIAL ASTHMA CLINICAL COURSE IN CONJUNCTION WITH BRONCHIECTASIS." Amur Medical Journal, no. 3 (2017): 141–42. http://dx.doi.org/10.22448/amj.2017.3.141-142.

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8

Azamkulova, N. O., and Sevara Irgasheva. "CLINICAL COURSE OF PERIMENOPAUSAL PERIOD IN WOMEN WITH HYPERANDROGENISM SYNDROME." UZBEK MEDICAL JOURNAL 2, no. 5 (May 30, 2021): 25–29. http://dx.doi.org/10.26739/2181-0664-2021-5-5.

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Hyperandrogenism is a disorder of endocrine status caused by excess production of androgens. The syndrome is a consequence of increased androgen production both in the ovaries and adrenal glands. However, such a division is very arbitrary, as increased production of androgens in the adrenal glands may increase production in the ovaries and vice versa. Androgens in women are synthesized by ovaries, adrenal glands and peripheral tissues, which also participate in metabolism.The set of androgens in both womenand men includes dehydroepiandrosterone-sulfate, dehydroepiandrosterone, androstenedione, testosterone and 5-alpha-dihydrotestosterone (5-alpha-DHT). Still, unlike men, women have a higher concentration of the first three hormones than the lasttwo. Androgen synthesis in the adrenal glands in women is regulated by adrenocorticotropic and in the ovaries by luteinizing hormone (LH) and some other intraglandular autoparacrine mechanisms. According to recent studies, in addition to the basic biological, previously commonly known effects of androgens, their new mechanisms of influence on the female body have been discovered. Keywords: hyperandrogenism, hormones, ovaries, adrenal glands, reproductive disorders
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9

Pare, Vidyashambhava, Harsha C.R., Roopa Kulkarni, and Sheela G. Nayak. "ARTERY OF HAAS- ITS ORIGIN AND COURSE: A CLINICAL PERSPECTIVE." International Journal of Anatomy and Research 5, no. 2.1 (April 30, 2017): 3771–76. http://dx.doi.org/10.16965/ijar.2017.174.

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10

Upadhyaya Kafle, S., M. Singh, N. Kafle, A. Sinha, P. Guragain, and H. S. Rimal. "Introducing Clinical Pathology Course to Fourth Year Medical Students as a Bridge between Pre-clinical and Clinical Medical Sciences." Kathmandu University Medical Journal 20, no. 1 (March 31, 2022): 97–101. http://dx.doi.org/10.3126/kumj.v20i1.49964.

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There isn’t any vertical integration of pre-clinical and clinical sciences subjects in the existing Kathmandu University MBBS curriculum. Many of the graduates are not able to correlate the clinico-pathological aspects of various diseases as a result the rational use of investigations for diagnosing various diseases is compromised. There are few published examples of implementation of pathology instruction courses during the clinical years of medical training but it is not universally practiced. This lack of exposure to pathology may lead to poor understanding of laboratory testing and the role of pathologists in patient care. To set and implement an exemplary vertical integration of pre-clinical science with clinical science. A 12 credit hours clinical pathology education course comprising clinical hematology, cytopathology and histopathology was developed. Students belonging from the ongoing fourth year MBBS course of Birat Medical College were enrolled in the course. All of the interactive lecture sessions were delivered via an e-learning interface, using the Zoom platform as the main teaching methods. Evaluation of students’ achievement of learning objectives was conducted through distributing pre and post-test online multiple-choice questionnaires. Chi-square tests were used to compare the variables between pre-test and post-test questionnaire responses. Results suggested that the designed clinical pathology course is valuable. The pretest and post-test questionnaire responses revealed the positive impact regarding the importance of introducing clinical pathology courses within the clinical year of MBBS undergraduate curriculum. Response rate to the online session was 100%. The point of agreement between the pre-test and post-test questionnaire responses were highly achieved after intervention of the clinical pathology course. A statistically significant result (p < 0.05) between all of the pre-test and post-test questionnaire responses was noted. There was a strong positive recommendation for incorporating clinical laboratory medicine courses within the MBBS clinical science curriculum. The improvement observed among fourth-year MBBS students on learning the importance of clinical pathology courses was encouraging. This experience thus contributed to set and implement an exemplary vertical integration of pre-clinical science with clinical science.
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11

Karimovich, Samatov Dilshod, and Shodikulova Gulandom Zikriyayevna. "Clinical Course In Upper Gastrointestinal Patients With Connective Tissue Dysplasia Syndrome." American Journal of Medical Sciences and Pharmaceutical Research 03, no. 02 (February 28, 2021): 135–39. http://dx.doi.org/10.37547/tajmspr/volume03issue02-20.

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Connective tissue dysplasias (CTDs) are genetically determined conditions characterised by defects in fibrous structures and connective tissue basic substance, leading to organ and system malformations, having a progressive course, defining features of associated pathology, as well as pharmacokinetics and pharmacodynamics of drugs.
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12

Talmasovna, Kayumova Dilrabo. "THE MAIN ASPECTS OF MENOPAUSAL METABOLIC SYNDROME IN THE CLINICAL COURSE." International Journal of Psychosocial Rehabilitation 24, no. 02 (February 28, 2020): 2122–34. http://dx.doi.org/10.37200/ijpr/v24i4/pr201323.

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13

Koh, Young Min, Kyeong Jae Chung, Sang Joon Park, Kyeong Woo Kang, Gee Young Suh, Man Pyo Chung, Hojoong Kim, O. Jung Kwon, and Chong H. Rhee. "Clinical Course of Untreated Sarcoidosis." Tuberculosis and Respiratory Diseases 47, no. 6 (1999): 807. http://dx.doi.org/10.4046/trd.1999.47.6.807.

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14

OGURA, TOSHIHIRO. "CT Colonography (Clinical Technology Course)." Japanese Journal of Radiological Technology 57, no. 4 (2001): 411–17. http://dx.doi.org/10.6009/jjrt.kj00001357557.

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15

Cho, Hyun Kyu, Byeong-Ho Jeong, and Hojoong Kim. "Clinical course of tracheobronchopathia osteochondroplastica." Journal of Thoracic Disease 12, no. 10 (October 2020): 5571–79. http://dx.doi.org/10.21037/jtd-20-1898.

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16

Koyama, Yoshiko, Housaku Torii, Masao Imai, and Akira Tamai. "The Clinical Course of Prosopagnosia." Higher Brain Function Research 16, no. 2 (1996): 143–52. http://dx.doi.org/10.2496/apr.16.143.

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17

&NA;. "On Course for Clinical Excellence." Nurse Practitioner 26, no. 12 (December 2001): 11. http://dx.doi.org/10.1097/00006205-200112000-00002.

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18

Klineova, Sylvia, and Fred D. Lublin. "Clinical Course of Multiple Sclerosis." Cold Spring Harbor Perspectives in Medicine 8, no. 9 (January 22, 2018): a028928. http://dx.doi.org/10.1101/cshperspect.a028928.

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19

Brass, Eric P., and Ross D. Feldman. "Clinical Pharmacology Curriculum Review Course." Clinical Pharmacology and Therapeutics 52, no. 6 (December 1992): 668. http://dx.doi.org/10.1038/clpt.1992.205.

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20

Dosso, André Alexandre, and Elisabeth Rungger-Brändle. "Clinical Course of Epidemic Keratoconjunctivitis." Cornea 27, no. 3 (April 2008): 263–68. http://dx.doi.org/10.1097/ico.0b013e31815b7d7d.

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21

Cohen, Elisabeth J., Stephen E. Higgins, Juan J. Arentsen, Ralph C. Eagle, and Peter R. Laibson. "Clinical Course of Acanthamoeba Keratitis." Clinical Infectious Diseases 13, Supplement_5 (March 1, 1991): S426. http://dx.doi.org/10.1093/clind/13.supplement_5.s426.

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22

Cetnarskyg, Roseanne. "Clinical Genetics — A Short Course." Heredity 86, no. 2 (February 2001): 251. http://dx.doi.org/10.1046/j.1365-2540.2001.0902a.x.

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23

Heitz-Mayfield, Lisa J. A., Marc Schätzle, Harald Löe, Walter Bürgin, Åge Ånerud, Hans Boysen, and Niklaus P. Lang. "Clinical course of chronic periodontitis." Journal of Clinical Periodontology 30, no. 10 (October 2003): 902–8. http://dx.doi.org/10.1034/j.1600-051x.2003.00399.x.

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24

Schätzle, Marc, Harald Löe, Niklaus P. Lang, Lisa J. A. Heitz-Mayfield, Walter Bürgin, Åge Ånerud, and Hans Boysen. "Clinical course of chronic periodontitis." Journal of Clinical Periodontology 30, no. 10 (October 2003): 909–18. http://dx.doi.org/10.1034/j.1600-051x.2003.00401.x.

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25

Schätzle, Marc, Harald Löe, Walter Bürgin, Åge Ånerud, Hans Boysen, and Niklaus P. Lang. "Clinical course of chronic periodontitis." Journal of Clinical Periodontology 30, no. 10 (October 2003): 887–901. http://dx.doi.org/10.1034/j.1600-051x.2003.00414.x.

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26

RUTSTEIN, ROBERT P., and WENDY MARSH-TOOTLE. "Clinical Course of Accommodative Esotropia." Optometry and Vision Science 75, no. 2 (February 1998): 97–102. http://dx.doi.org/10.1097/00006324-199802000-00013.

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27

Evans, Bruce J. W. "Clinical Course of Accommodative Esotropia." Optometry and Vision Science 76, no. 2 (February 1999): 80. http://dx.doi.org/10.1097/00006324-199902000-00010.

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28

Gupta, P. N., M. S. Valiathan, K. G. Balakrishnan, C. C. Kartha, and M. K. Ghosh. "Clinical course of endomyocardial fibrosis." Heart 62, no. 6 (December 1, 1989): 450–54. http://dx.doi.org/10.1136/hrt.62.6.450.

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29

Cottone, M., D. Scimeca, F. Mocciaro, G. Civitavecchia, G. Perricone, and A. Orlando. "Clinical course of ulcerative colitis." Digestive and Liver Disease 40 (July 2008): S247—S252. http://dx.doi.org/10.1016/s1590-8658(08)60533-2.

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30

Dua, H. S. "Clinical course of hurricane keratopathy." British Journal of Ophthalmology 84, no. 3 (March 1, 2000): 285–88. http://dx.doi.org/10.1136/bjo.84.3.285.

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31

Bloch, Michael H., and James F. Leckman. "Clinical course of Tourette syndrome." Journal of Psychosomatic Research 67, no. 6 (December 2009): 497–501. http://dx.doi.org/10.1016/j.jpsychores.2009.09.002.

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32

Radulescu, E., E. Vlad, and D. Ionescu. "Clinical course patterns of schizophrenia." Schizophrenia Research 60, no. 1 (March 2003): 25. http://dx.doi.org/10.1016/s0920-9964(03)80073-7.

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33

Ganser, Arnold, and Dieter Hoelzer. "Clinical Course of Myelodysplastic Syndromes." Hematology/Oncology Clinics of North America 6, no. 3 (June 1992): 607–18. http://dx.doi.org/10.1016/s0889-8588(18)30331-9.

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34

Hočevar-Boltežar, I., M. Žargi, and N. Gale. "Clinical course of laryngeal papillomatosis." International Congress Series 1240 (October 2003): 769–71. http://dx.doi.org/10.1016/s0531-5131(03)00742-8.

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35

Fonseca, Cristina, Andreia Mendes Silva, Sandra Freire, and Rui Proença. "Ocular toxocariasis: atypical clinical course." BMJ Case Reports 12, no. 4 (April 2019): e228717. http://dx.doi.org/10.1136/bcr-2018-228717.

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A 17-year-old man presented to the emergency department with complaints of retro-orbital pain of the left eye and an altitudinal visual field defect for 2 weeks. Fundus examination revealed ipsilateral hyperaemic optic disc oedema, and the patient was admitted with the presumptive diagnosis of left optic neuritis. Subsequently, during follow-up, the patient developed a retinal granulomatous lesion in the superior temporal arcade with vitritis and fibrotic strands extending to the mid-periphery. Serum antibodies detection by ELISA and aqueous humour immunoblot were positive for Toxocara canis. Medical therapy with albendazole and oral steroids was instituted with satisfactory results. One year later, a new macular lesion developed with consequent vision loss.
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36

Greden, John F. "Developmental neurobiology and clinical course." Biological Psychiatry 31, no. 5 (March 1992): iii. http://dx.doi.org/10.1016/0006-3223(92)90349-5.

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37

Athanasou, N. A., and J. O'D. McGee. "The oxford clinical pathology course." Journal of Pathology 156, no. 3 (November 1988): 267–69. http://dx.doi.org/10.1002/path.1711560313.

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38

Chew, Emily Y. "Clinical Course of Macular Holes." Archives of Ophthalmology 117, no. 2 (February 1, 1999): 242. http://dx.doi.org/10.1001/archopht.117.2.242.

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39

Michalak, Arkadiusz, Marta Koptas, Aleksandra Świercz, Krystyna Wyka, Anna Hogendorf,, Agnieszka Szadkowska, Wojciech Młynarski, and Wojciech Fendler. "Coexisting psoriasis affects the clinical course of type 1 diabetes in children." Pediatric Endocrinology Diabetes and Metabolism 23, no. 3 (2017): 139–45. http://dx.doi.org/10.18544/pedm-23.03.0085.

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40

Maryem, E. S., D. Ibrahim, C. Abdelfetah, S. Mustapha, M. Latifa, and M. E. F. Kamal. "Pneumococcal meningitis: Clinical aspects, bacterial profile and clinical course." International Journal of Infectious Diseases 53 (December 2016): 79. http://dx.doi.org/10.1016/j.ijid.2016.11.200.

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41

Estey, Elihu, J. P. Issa, Guillermo Garcia-Manero, Stefan Faderl, and Hagop Kantarjian. "Use of Post-Treatment Clinical Data To Predict Response to Decitabine." Blood 110, no. 11 (November 16, 2007): 1448. http://dx.doi.org/10.1182/blood.v110.11.1448.1448.

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Abstract Best response of AML or MDS to decitabine (DAC) may require &gt; 3 courses. Identification of pts whose response to initial courses suggests they are unlikely to respond further to subsequent courses would allow such pts to receive other therapies more expeditiously. 104 pts, median age 69, received DAC (20 mg/m2 IV daily × 5 days every 5 weeks) for newly- diagnosed MDS (80, 76 IPSS int-2 or high) or AML (24) from 9/00-5/07. After 3 courses, 33 of the 104 were in CR, 6 were dead, and 32 had been removed from study, with the remaining 32 receiving more DAC. The probability of CR decreased to &lt; 10% on all courses after the 5th: Course# Pts Receiving CR Deaths/Off Study Received Next Course after course Course 1 104 7 (7%) 2/15 80 2 80 16 (20%) 4/12 48 3 48 10 (21%) 0/6 32 4 32 5 (16%) 0/5 22 5 22 3 (14%) 0/3 16 6 16 1 (6%) 1/3 11 7–15 11 1 (9%) 1/9 0 Results were qualitatively similar for AML and MDS. We divided the 32 pts who received ≥ 4 courses according to whether after course 3 their: marrow showed a response, defined as a p &lt; 0.05 difference between the pre-Rx and post course 3 blast count, platelet count increased &gt; 30, 000 if &lt; 100,000 pre-Rx, or neutrophil count increased &gt; 500 if &lt; 500 pre Rx; criteria 2 and 3 were adapted from those of the IWG. 8/14 pts with vs. 1/11 without a marrow response after course 3 entered CR on subsequent courses (p = .02, 6/10 vs. 1/10 considering only MDS). The corresponding figure for platelet response/no response was 5/8 vs. 2/11 (3/5 vs. 2/9 for MDS). The median number of courses after the 3rd given pts with marrow response was 2 (range 1–10) vs. 3 (range 1–12) for pts without marrow response. Our data suggest that if CR is the goal, pts should not receive more than 5 courses of DAC and no more than 3 if marrow response has not occurred after the 3rd.
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42

George, Marshall A., and Patricia Davis-wiley. "Team Teaching a Graduate Course: Case Study: A Clinical Research Course." College Teaching 48, no. 2 (April 2000): 75–80. http://dx.doi.org/10.1080/87567550009595818.

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43

Can, Aylin, Kursat Altinbas, Erhan Kurt, and Niyazi Uygur. "Delirious mania: Clinical course and treatment." Journal of Mood Disorders 5, no. 1 (2015): 40. http://dx.doi.org/10.5455/jmood.20140901035042.

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44

Kalbus, O. I., and V. M. Shkolnyk. "Clinical features of mystania’s gravis course." Ukrainian Neurological Journal, no. 1 (March 29, 2019): 10–16. http://dx.doi.org/10.30978/unj2019-1-10.

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45

Park, Soon Kew. "Diagnosis and Clinical Course of Tuberculosis." Tuberculosis and Respiratory Diseases 44, no. 6 (1997): 1209. http://dx.doi.org/10.4046/trd.1997.44.6.1209.

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46

Park, Joo Hun, M. Kitaichi, Ho Kee Yum, Tae Sun Shim, Chae Man Lim, Youn Suck Koh, Sang Do Lee, Woo Sung Kim, Won Dong Kim, and Dong Soon Kim. "Clinical Course of Usual Interstitial Pneumonia." Tuberculosis and Respiratory Diseases 49, no. 5 (2000): 601. http://dx.doi.org/10.4046/trd.2000.49.5.601.

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47

Ohta, Takashi, Hiroyuki Ishibashi, Ikuo Sugimoto, Hirohide Iwata, Jun Kawanishi, Tetsuya Yamada, Masao Tadakoshi, and Noriyuki Hida. "The Clinical Course of Buerger’s Disease." Annals of Vascular Diseases 1, no. 2 (2008): 85–90. http://dx.doi.org/10.3400/avd.avdct00208.

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48

Shkatova, Yanina, Andrey Budnevsky, Evgeniy Ovsyannikov, Galina Prozorova, Anna Volynkina, and Irina Olysheva. "Neuropeptide Y and Asthma Clinical Course." International Journal of Biomedicine 11, no. 4 (December 10, 2021): 410–13. http://dx.doi.org/10.21103/article11(4)_oa2.

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Background: The spectrum of clinical manifestations and pathogenetic mechanisms of bronchial asthma (BA) is very wide. Given the complex pathogenesis and syndromic nature of BA, it is not surprising that there is no single universal biomarker. The objective of this study was to evaluate levels of neuropeptide Y (NPY) and its association with levels of leptin, adiponectin, oxidative damage, antioxidant status, spirometry parameters, and asthma control in BA patients. Methods and Results: Overall, 140 patients [35(25%) men and 105(75%) women; mean age of 57.0±9.34 years] with moderate asthma participated in the study. According to body mass index, all patients were divided into three groups. The asthma diagnosis was based on the integral assessment of symptoms, medical history, health status, and spirometry values according to the Global Strategy for Asthma Management and Prevention. (GINA, 2017 REPORT). The Asthma Control Test (АСТ) was used to assess asthma control. NPY was measured in blood serum in EIA. The NPY level was significantly higher in overweight patients and patients with obesity than in patients with normal body weight. The NPY level significantly correlated with leptin (r=0.44; P<0.05), adiponectin (r=-0.24; P<0.05), ImanOx (r=-0.40; P<0.05), PerOx (r=0.58; P<0.05), ACT (r=-0.41; P<0.05), VC (r=-0.31; P<0.05), FEV1 (r=-0.41; P<0.05), FEF25% (r=-0.26; P<0.05), FVC (r=-0.23; P<0.05), Tiffno index (r=-0.36; P<0.05), FEF50% (r=-0.22; P<0.05), and PEF (r=-0.23; P<0.05) Conclusion: The severity of the asthma clinical course is associated with different factors, including oxidative stress, levels of leptin, adiponectin and NPY. NPY seems to be associated with worse asthma control and higher levels of leptin and oxidative damage.
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49

Hanyu, Haruo. "Clinical Course Of Diabetes-Related Dementia." Journal of Alzheimer’s & Neurodegenerative Diseases 6, no. 1 (April 30, 2020): 1–3. http://dx.doi.org/10.24966/and-9608/100036.

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50

Turner III, Norman S., Samuel Hoxie, Justin Strickland, and David Jacofsky. "Clinical Course of Open Metatarsal Fractures." Orthopedics 30, no. 8 (August 1, 2007): 662–65. http://dx.doi.org/10.3928/01477447-20070801-02.

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