Relevant bibliographies by topics / Chylomicron

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Academic literature on the topic 'Chylomicron'

Author: Grafiati
Published: 4 June 2021
Last updated: 11 February 2022

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Journal articles on the topic "Chylomicron"

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Redgrave, T. G. "Chylomicron metabolism." Biochemical Society Transactions 32, no. 1 (2004): 79–82. http://dx.doi.org/10.1042/bst0320079.

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Chylomicrons are the ‘orphans’ of the lipoprotein family. Difficulty of measurement has impeded understanding of their metabolism. Plasma concentrations of chylomicrons and chylomicron remnants give no insight into the magnitude of substrate flux through these pathways. A defect in clearance of chylomicron remnants is probably an indication of a more generalized defect in lipoprotein metabolism. Accumulating evidence supports a relationship between abnormalities in the clearance from plasma of chylomicron remnants and accelerated progression of atherosclerosis. Methods using stable isotopes in
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Phillips, Catherine, Claire Madigan, Daphne Owens, Patrick Collins, and Gerald H. Tomkin. "Defective Chylomicron Synthesis as a Cause of Delayed Particle Clearance in Diabetes?" International Journal of Experimental Diabetes Research 3, no. 3 (2002): 171–78. http://dx.doi.org/10.1080/15604280214277.

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Chylomicron metabolism is abnormal in diabetes and the chylomicron particle may play a very important role in atherosclerosis. The aim of this study was to examine the effect of diabetes on the metabolism of chylomicrons in cholesterol-fed alloxan diabetic and nondiabetic rabbits. Five diabetic rabbits and 5 control rabbits were given [C14]linoleic acid and [H3]cholesterol by gavage. Lymph was collected following cannulation of the lymph duct and radiolabelled chylomicrons were isolated by ultracentrifugation. The chylomicrons from each animal were injected into paired control and diabetic rec
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Tso, P., J. A. Barrowman, and D. N. Granger. "Importance of interstitial matrix hydration in intestinal chylomicron transport." American Journal of Physiology-Gastrointestinal and Liver Physiology 250, no. 4 (1986): G497—G500. http://dx.doi.org/10.1152/ajpgi.1986.250.4.g497.

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We have shown previously that lymph flow has a profound effect on intestinal chylomicron transport. However, since lymph flow both determines the rate of convective movement of chylomicrons within the interstitium and reflects the degree of hydration of the interstitial matrix, we were unable to determine which factor was more important for the inverse relation between the chylomicron appearance time and lymph flow. In this investigation, we measured the chylomicron appearance time in rats with a normal lymph flow and expanded matrix (study A), in rats with a reduced lymph flow but expanded ma
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James, AP, K. Slivkoff-Clark, and JCL Mamo. "New Insights into Cardiovascular Disease Risk in Subjects with Visceral Obesity." Asia Pacific Journal of Public Health 15, no. 1_suppl (2003): S37—S40. http://dx.doi.org/10.1177/101053950301500s10.

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Obese insulin resistant individuals often present with a dyslipidemic phenotype characterised by hypertriglyceridemia, low HDL cholesterol levels, essentially normal total- and LDL-cholesterol, but a propensity for smaller, denser LDL particles. We have reported that concentrations of chylomicrons are two to three folds greater than in age-matched lean controls. We have recently observed that in lean free-living subjects the flux of chylomicrons over a 12h period was just 25% greater in these subjects than basal chylomicron production. Constitutive secretion of chylomicrons appears to be of gr
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Tso, P., V. Pitts, and D. N. Granger. "Role of lymph flow in intestinal chylomicron transport." American Journal of Physiology-Gastrointestinal and Liver Physiology 249, no. 1 (1985): G21—G28. http://dx.doi.org/10.1152/ajpgi.1985.249.1.g21.

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In this study we investigated the influence of lymph flow on chylomicron transport. We examined the effects of varying the hydration of the interstitial matrix on chylomicron appearance time and on lymphatic lipid transport rate when a lipid test meal containing oleic acid and 1-monoolein was infused intraduodenally at a constant rate. The three groups of rats tested were control rats (normal interstitial hydration), rats receiving intravenous saline infusion (expanded interstitial matrix), and rats with an attenuated water absorption rate (dehydrated interstitial matrix). This study shows tha
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Irawati, Deasy, John C. L. Mamo, Karin M. Slivkoff-Clark, Mario J. Soares, and Anthony P. James. "Dietary fat and physiological determinants of plasma chylomicron remnant homoeostasis in normolipidaemic subjects: insight into atherogenic risk." British Journal of Nutrition 117, no. 3 (2017): 403–12. http://dx.doi.org/10.1017/s0007114517000150.

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AbstractTAG depleted remnants of postprandial chylomicrons are a risk factor for atherosclerosis. Recent studies have demonstrated that in the fasted state, the majority of chylomicrons are small enough for transcytosis to arterial subendothelial space and accelerate atherogenesis. However, the size distribution of chylomicrons in the absorptive state is unclear. This study explored in normolipidaemic subjects the postprandial distribution of the chylomicron marker, apoB-48, in a TAG-rich lipoprotein plasma fraction (Svedberg flotation rate (Sf>400), in partially hydrolysed remnants (Sf 20–
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Milan, Amber M., Anu Nuora, Shikha Pundir, et al. "Older adults have an altered chylomicron response to a high-fat meal." British Journal of Nutrition 115, no. 5 (2016): 791–99. http://dx.doi.org/10.1017/s000711451500505x.

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AbstractAgeing is associated with a prolonged and exaggerated postprandial lipaemia. This study aimed to examine the contribution of alterations in chylomicron synthesis, size and lipid composition to increased lipaemia. Healthy older (60–75 years; n 15) and younger (20–25 years; n 15) subjects consumed a high-fat breakfast. Chylomicron dynamics and fatty acid composition were analysed for 5 h in the postprandial state. Plasma TAG levels were elevated following the meal in the older subjects, relative to younger subjects (P<0·01). For older subjects compared with younger subjects, circulati
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Lambert, Marc S., Kathleen M. Botham, and Peter A. Mayes. "Modification of the fatty acid composition of dietary oils and fats on incorporation into chylomicrons and chylomicron remnants." British Journal of Nutrition 76, no. 3 (1996): 435–45. http://dx.doi.org/10.1079/bjn19960048.

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Possible changes in the fatty acid composition of dietary fats and oils which might occur during digestion, absorption and formation of chylomicrons and chylomicron remnants were investigated. Chylomicrons were collected from the thoracic duct of rats tube-fed with olive, maize, palm or fish oil or butter fat, and their fatty acid composition was determined and compared with that of their parent lipids. In turn, these lipoproteins were converted to chylomicron remnants infunctionally hepatectomized rats and their composition re-determined. The predominant fatty acids in each of the oils and fa
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Bowler, A., T. G. Redgrave, and J. C. L. Mamo. "Chylomicron-remnant clearance in homozygote and heterozygote Watanabe-heritable-hyperlipidaemic rabbits is defective. Lack of evidence for an independent chylomicron-remnant receptor." Biochemical Journal 276, no. 2 (1991): 381–86. http://dx.doi.org/10.1042/bj2760381.

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Lymph chylomicrons radiolabelled in triacylglycerol and cholesteryl ester were injected into control and Watanabe heritable-hyperlipidaemic (WHHL) rabbits. Clearance of chylomicrons was slower in heterozygote and homozygote WHHL rabbits. Slower remnant clearance in WHHL rabbits was confirmed by monitoring the clearance from plasma of preformed chylomicron remnants. Use of chylomicron-like lipid emulsions injected into control and WHHL rabbits also confirmed the defect in remnant clearance in heterozygote WHHL and homozygote WHHL groups. Clearance from plasma of emulsion triolein was delayed in
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Tsai, Michael Y., Angeliki Georgopoulos, James D. Otvos, et al. "Comparison of Ultracentrifugation and Nuclear Magnetic Resonance Spectroscopy in the Quantification of Triglyceride-Rich Lipoproteins after an Oral Fat Load." Clinical Chemistry 50, no. 7 (2004): 1201–4. http://dx.doi.org/10.1373/clinchem.2004.032938.

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Abstract Background: The measurement of triglyceride (TG)-rich particles after an oral fat challenge has been used to provide a measure of risk for coronary artery disease independent of the fasting plasma triglyceride concentration. The analytical “gold standard” for measuring TG-rich lipoproteins uses density gradient ultracentrifugation; however, this technique is labor-intensive. Because of our need to perform numerous postprandial analyses of TG-rich lipoproteins for a large interventional study (Genetics of Lipid Lowering Drugs and Diet Network), we evaluated the use of nuclear magnetic
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Dissertations / Theses on the topic "Chylomicron"

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Guldur, Tayfun. "Metabolism of chylomicrons and chylomicron remnants." Thesis, Royal Veterinary College (University of London), 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.522548.

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Grieve, David James. "The role of chylomicrons and chylomicron remnants in the initiation of atherosclerotic lesions in the artery wall." Thesis, Royal Veterinary College (University of London), 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.522544.

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Silva, K. D. Renuka R. "Markers of chylomicron metabolism in man." Thesis, University of Reading, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.394221.

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Cuerq, Charlotte. "Absorption de la vitamine E dans les hypocholestérolémies génétiques." Thesis, Lyon, 2016. http://www.theses.fr/2016LYSE1086.

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Les hypocholestérolémies génétiques représentent un ensemble de pathologies héréditaires du métabolisme caractérisées par une hypocholestérolémie inférieure au 5ème percentile pour l'âge et le sexe. Elles sont majoritairement dues à un défaut d'assemblage et de sécrétion des lipoprotéines entraînant, dès la période néonatale, une malabsorption des lipides et des vitamines liposolubles. Les conséquences à long terme peuvent être dramatiques (complications ophtalmologiques et neurologiques graves) si un traitement par vitamines liposolubles, en particulier vitamine E à fortes doses, n'est pas in
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Sethi, Sunil. "Chylomicron marker metabolism in health and disease." Thesis, University of Surrey, 1995. http://epubs.surrey.ac.uk/844052/.

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In this thesis, chylomicron (CM) and CM-remnant (CM-R) metabolism in humans was studied by the application of unique markers which label these lipoprotein particles from the stage of production by the enterocyte until, removal by hepatic receptor mediated processes. Retinyl palmitate (RP) is a vitamin A ester, which labels the CM/CM-R by behaving like the cholesterol ester (CE) which is carried in the core of these particles. Development of a mono-specific antibody to apolipoprotein (apo) B-48 and application of an enzyme linked immunosorbant assay (ELISA) enabled quantification of this apolip
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Field, Polly Ann. "The effects of insulin resistance on chylomicron metabolism." Thesis, University of Oxford, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.302120.

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Siddiq, Ajmal 1963. "Modeling the distribution of chylomicron cholesterol in the body." Thesis, The University of Arizona, 1991. http://hdl.handle.net/10150/277981.

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A physiologically-based multi-compartment model has been developed which makes a priori predictions of the chylomicron cholesterol distribution in the body of a rat. The model considers each organ to be a separate compartment which interacts with all the other organs/compartments through the blood stream. A particle approach has been used since chylomicron cholesterol distribution is regulated more by the particle concentrations than cholesterol concentrations. The chylomicron cholesterol distribution is coupled to the triglyceride contents of the particle. Unsteady nonlinear differential equa
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Nauli, Andromeda Margono. "Intestinal Lipid Uptake and Secretion of VLDL and Chylomicron." University of Cincinnati / OhioLINK, 2005. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1123866425.

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Sun, Yuxi. "Development of in vitro Chylomicron Assay Using Caco-2 Cells." Digital Commons @ East Tennessee State University, 2013. https://dc.etsu.edu/etd/1781.

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Dietary fats are mainly transported by the intestine in lipoproteins: chylomicrons (CMs) and very low density lipoproteins (VLDLs). Unfortunately, studies of the intestinal absorption of dietary fat have been hampered by the lack of an adequate in vitro model system. As an in vitro model Caco-2 cells are able to secrete lipoproteins. We investigated the possible factors that may affect the secretion of CMs through the ultracentrifugation technique. The dose-dependent effects of oleic acid, mono-olein, egg lecithin, collagen matrix, and the effect of cell differentiation on CM secretion were th
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Goulter, Andrew Barry. "Effects of chylomicron remnants on porcine coronary artery endothelial cell function." Thesis, Royal Veterinary College (University of London), 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.522537.

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Books on the topic "Chylomicron"

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Brahm, Amanda J., and Robert A. Hegele. Monogenic Chylomicronemia: Deficiency of Lipoprotein Lipase and Related Factors. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0033.

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Monogenic chylomicronemia is an autosomal recessive condition characterized by severely elevated fasting triglyceride that carries lifelong elevated risk of developing pancreatitis. The majority of cases are caused by mutations in the LPL gene encoding lipoprotein lipase, the enzyme primarily responsible for chylomicron clearance. Mutations in genes encoding associated proteins (APOC2, APOA5, GPIHBP1 and LMF1) may also present with a very similar phenotype. Current management, which includes restriction of dietary fat intake and standard pharmacologic interventions, has met with limited succes
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Skoutas, Demetrios. Responses of the chylomicron remnant marker, retinyl ester and apolipoprotein B-48 to meals ofvarying monousaturated fatty acid content in middle aged men. 1995.

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Wiklund, Olov, and Jan Borén. Pathogenesis of atherosclerosis: lipid metabolism. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0011.

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Lipids are carried in plasma as microparticles, lipoproteins, composed of a core of hydrophobic lipids and a surface of amphipathic lipids. In addition, the particles carry proteins (i.e. apolipoproteins). The proteins have key functions in the metabolism as receptor ligands, enzymes or activators. Lipoproteins are classified based on density into: chylomicrons, VLDL, IDL, LDL, and HDL. Retention of apoB-containing lipoproteins (LDL, IDL, and VLDL) in the arterial intima is the initiating event in the development of atherosclerosis. Retention is mediated by binding of apoB to structural proteo
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Book chapters on the topic "Chylomicron"

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Gooch, Jan W. "Chylomicron." In Encyclopedic Dictionary of Polymers. Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_13397.

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Peters, Nils, Martin Dichgans, Sankar Surendran, et al. "Chylomicron Retention Disease." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_352.

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Rodgers, John B. "Inhibitors of Chylomicron Formation and Secretion." In Intestinal Lipid Metabolism. Springer US, 2001. http://dx.doi.org/10.1007/978-1-4615-1195-3_18.

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Windler, E., and H. Greten. "Chylomicron Catabolism and Uptake by the Liver." In Expanding Horizons in Atherosclerosis Research. Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71753-6_26.

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Beisiegel, Ulrike, Annette Krapp, Jörg Heeren, and Wilfried Weber. "In Vitro Studies on Human Chylomicron Catabolism." In Drugs Affecting Lipid Metabolism. Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-009-0311-1_76.

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Windier, Eberhard E. T. "The Uptake of Chylomicron Remnants by the Liver." In Advances in Experimental Medicine and Biology. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-1268-0_19.

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Harris, W. S. "ω3 Fatty Acids and Human Chylomicron Metabolism." In Fatty Acids and Lipids - New Findings. KARGER, 2000. http://dx.doi.org/10.1159/000059777.

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Wickham, Nicholas, J. Cormak, G. M. Vercellotti, D. E. Hammerschmidt, and H. S. Jacob. "Post-Prandial Chylomicron Creaming, C-Reactive Protein and Enhanced Complement Activation." In Vascular Endothelium. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2437-3_55.

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Torbet, J. "Magnetic Birefringence Study of Fibrin Formation and Chylomicron Behaviour in Human Blood Plasma." In Springer Proceedings in Physics. Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-71526-6_5.

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Sultan, Fabrice, Dominique Lagrange, and Sabine Griglio. "In Vitro Binding and in Vivo Uptake of Chylomicron Remnants after their Hydrolysis by Hepatic Lipase." In Hypercholesterolemia, Hypocholesterolemia, Hypertriglyceridemia, in Vivo Kinetics. Springer US, 1990. http://dx.doi.org/10.1007/978-1-4684-5904-3_37.

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Conference papers on the topic "Chylomicron"

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Dixon, J. Brandon. "Engineering Tools for Studying the Interplay Between Mechanics and Biology in Lymphatic Lipid Transport." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19364.

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The lymphatic vasculature extends through most tissues of the body and plays an essential role in maintaining fluid balance, immune cell trafficking, and lipid transport. Nearly all dietary lipid is transported from the intestine to the circulation via the lymphatic system in the form of triglyceride-rich lipoproteins called chylomicrons. This process can be described through two different mechanisms: 1) entry of the chylomicron into the initial lymphatic vessels of the small intestine, known as lacteals, and 2) the transport of these chylomicrons through the larger collecting lymphatics by a
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Marcato, Larissa A., Diego V. S. Carvalho, Margarida M. Hamada, et al. "Internal Dosimetry of a Chylomicron-like Emulsion Labeled with [sup 14]C-CE in Humans." In XXXIII BRAZILIAN WORKSHOP ON NUCLEAR PHYSICS. AIP, 2011. http://dx.doi.org/10.1063/1.3608989.

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Chamulitrat, W., J. Seeßle, B. Javaheri-Haghighi, et al. "Intestinal deletion of fatty acid transport protein 4 in mice increases blood chylomicrons." In 36. Jahrestagung der Deutschen Arbeitsgemeinschaft zum Studium der Leber. Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0039-3402169.

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Ruf, Horst, and Barry J. Gould. "Use of dynamic light scattering for the determination of size distributions of chylomicrons from human lymph." In BiOS '97, Part of Photonics West, edited by Alexander V. Priezzhev, Toshimitsu Asakura, and Robert C. Leif. SPIE, 1997. http://dx.doi.org/10.1117/12.273655.

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Surya, I. E., and J. W. N. Akkerman. "HUMAN PLASMA PAF-ACETYLHYDROLASE, NORMALLY PRESENT IN LOW DENSITY LIPOPROTEINS, IS ASSOCIATED WITH HIGH DENSITY LIPOPROTEINS IN A PATIENT WITH LDL DEFICIENCY." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642882.

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Platelet Activating Factor (l-0-alkyl-2-acetyl-sn-glycerol-3-phosphocholine; PAF) plays an important role in allergic and inflammatory reactions and activates platelets in the nanomolar range. One of the main factors that controls PAF activity in blood is an enzyme that hydrolyzes the acetyl-chain thereby converting PAF to biologically inactive lyso-PAF. The enzyme is acid labile and normally associated with apo B-containing low density lipoproteins (LDL, density 1,006-1,063 g/ml).We investigated whether a deficiency in LDL would affect the enzyme activity. PAF-inactivating activity was measur
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