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Journal articles on the topic 'Chronic venous insufficiency'

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Author: Grafiati

Published: 4 June 2021

Last updated: 24 January 2023

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1

Panda, Dwijesh Kumar. "Chronic Venous Insufficiency Resemble Lymphedema Leg." Asian Pacific Journal of Health Sciences 6, no. 3 (September 2019): 52–55. http://dx.doi.org/10.21276/apjhs.2019.6.3.10.

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2

Kestler, Bri. "Chronic Venous Insufficiency." Physician Assistant Clinics 6, no. 2 (April 2021): 319–30. http://dx.doi.org/10.1016/j.cpha.2020.11.005.

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3

ÇINAR ÖZDEMİR, Özlem, Emre ALTINDAĞ, Fatma AVCI, and M. Fatih UYSAL. "Chronic Venous Insufficiency." Turkiye Klinikleri Journal of Health Sciences 1, no. 2 (2016): 125–38. http://dx.doi.org/10.5336/healthsci.2015-45121.

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4

Wilson, Paul. "Chronic venous insufficiency." Morecambe Bay Medical Journal 3, no. 3 (September 1, 1998): 85–89. http://dx.doi.org/10.48037/mbmj.v3i3.549.

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5

Stadnicki, Antoni, Martin Rusnák, and Anna Stadnicka. "CHRONIC VENOUS INSUFFICIENCY." Scientific Journal of Polonia University 32, no. 1 (April 3, 2019): 131–36. http://dx.doi.org/10.23856/3217.

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Chronic venous insufficiency (CVI) is a common, but underdiagnozed clinical disorder associated with a variety of signs and symptoms. The presence of leg edema in association with varicose veins, and venous leg ulcer in later disease stages defines the disease. The pathogenesis of chronic venous disease is based on venous reflux, obstruction, or a combination thereof. Prior postthrombotic syndrome is one of risk factor for CVI which may explain observed prevalence of thrombophilia in CVI. Color flow duplex ultrasound is the gold standard for nearly all diagnostic issues related to chronic venous disease. Compression stockings are the mainstay for conservative management. Earlier use of venous ablation therapy should be considered in symptomatic patients with superficial tortuous vein

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6

Miller, Wayne L. "Chronic venous insufficiency." Current Opinion in CARDIOLOGY 10, no. 5 (September 1995): 543. http://dx.doi.org/10.1097/00001573-199509000-00016.

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7

Kwun, Woo-Hyung. "Chronic Venous Insufficiency." Yeungnam University Journal of Medicine 24, no. 2 Suppl (December 31, 2007): S234–244. http://dx.doi.org/10.12701/yujm.2007.24.2s.s234.

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8

Reich-Schupke, S. "Chronic venous insufficiency." Phlebologie 46, no. 01 (January 2017): 34–36. http://dx.doi.org/10.12687/phleb2348-1-2017.

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9

Eberhardt, Robert T., and Joseph D. Raffetto. "Chronic Venous Insufficiency." Circulation 111, no. 18 (May 10, 2005): 2398–409. http://dx.doi.org/10.1161/01.cir.0000164199.72440.08.

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10

Eberhardt, Robert T., and Joseph D. Raffetto. "Chronic Venous Insufficiency." Circulation 130, no. 4 (July 22, 2014): 333–46. http://dx.doi.org/10.1161/circulationaha.113.006898.

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11

Earnshaw, Jonothan J. "Chronic venous insufficiency." Journal of Vascular Surgery 34, no. 5 (November 2001): 953. http://dx.doi.org/10.1067/mva.2001.117614.

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12

Capeheart, Jeanine Kunkel. "Chronic Venous Insufficiency." Journal of Wound, Ostomy and Continence Nursing 23, no. 4 (July 1996): 227–34. http://dx.doi.org/10.1097/00152192-199607000-00018.

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13

O’Donnell, Thomas F., C. Scott McEnroe, and Paula Heggerick. "Chronic Venous Insufficiency." Surgical Clinics of North America 70, no. 1 (February 1990): 159–80. http://dx.doi.org/10.1016/s0039-6109(16)45042-5.

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14

White, J. V. "Chronic Venous Insufficiency." Perspectives in Vascular Surgery and Endovascular Therapy 17, no. 4 (December 1, 2005): 319–27. http://dx.doi.org/10.1177/153100350501700406.

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15

Vernick, Sanford H., and Frank D. Shaw. "Chronic venous insufficiency." Archives of Physical Medicine and Rehabilitation 75, no. 9 (September 1994): 1023–24. http://dx.doi.org/10.1016/0003-9993(94)90688-2.

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16

Gujja, Karthik, Jose Wiley, and Prakash Krishnan. "Chronic Venous Insufficiency." Interventional Cardiology Clinics 3, no. 4 (October 2014): 593–605. http://dx.doi.org/10.1016/j.iccl.2014.07.001.

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17

Donaldson, Magruder C. "Chronic venous insufficiency." Current Treatment Options in Cardiovascular Medicine 2, no. 3 (May 2000): 265–72. http://dx.doi.org/10.1007/s11936-000-0021-2.

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18

Schainfeld, Robert M. "Chronic venous insufficiency." Current Treatment Options in Cardiovascular Medicine 5, no. 2 (April 2003): 109–19. http://dx.doi.org/10.1007/s11936-003-0019-7.

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19

Kim, Esther S. H., Claudiu Diaconu, Leasa Baus, Robert J. Fox, Alia Grattan, Irene Katzan, Larry Raber, Alexander Rae-Grant, and Mei Lu. "Chronic Cerebrospinal Venous Insufficiency." Journal of Ultrasound in Medicine 34, no. 6 (June 2015): 1097–106. http://dx.doi.org/10.7863/ultra.34.6.1097.

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20

Rasman, Alessandro. "Chronic Cerebrospinal Venous Insufficiency." Vascular Specialist International 31, no. 3 (September 30, 2015): 106–7. http://dx.doi.org/10.5758/vsi.2015.31.3.106.

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21

Dua, Anahita, and Jennifer A. Heller. "Advanced Chronic Venous Insufficiency." Vascular and Endovascular Surgery 51, no. 1 (December 26, 2016): 12–16. http://dx.doi.org/10.1177/1538574416682175.

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Introduction: Intervention for advanced chronic venous insufficiency is considered an appropriate standard of care. However, outcomes vary among patients who present in advanced clinical stages of disease. The main objectives of this study were to determine whether racial disparity exists at initial presentation and response to intervention. Methods: A retrospective database was created to include all radiofrequency ablation procedures performed by a single surgeon from January 14, 2009, through May 25, 2011. Demographics, clinical traits, race, procedure, and outcomes were analyzed. Stepwise model selection reduced candidate baseline factors to a final parsimonious model, which was analyzed using analysis of variance. Results: The database consisted of 300 patients with a predominant female (n = 215, 85%) base and 85 (15%) males, with a mean age distribution of 53 years. The mean body mass index was 30.2. Racial distribution revealed Asian (n = 9, 3.3%), Pacific Islander (n = 1, 0.4%), African American (n = 37, 13.6%), and Caucasian (CAU, n = 225, 82.7%). African Americans presented with more advanced clinical stages than the CAU group—C2: African American 21.6%, CAU 36.7%; C4: African American 35%, CAU 24.3%; and C6: African American 35.1%, CAU 7.5%. African Americans demonstrated a higher preoperative venous clinical severity score (VCSS) than their CAU counterparts. Postprocedural decrease in VCSS score was lower in African Americans than their CAU counterparts. Conclusion: African American patients present with more advanced venous insufficiency than CAUs. Postprocedural analysis reveals not only slower ulcer healing times but also higher ulcer recurrence rates.

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22

Sternberg, Zohara. "Chronic Cerebrospinal Venous Insufficiency." Journal of Endovascular Therapy 22, no. 4 (June 19, 2015): 647–49. http://dx.doi.org/10.1177/1526602815592204.

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23

Shami, S., S. Sarin, and J. Scurr. "Chronic venous insufficiency disease." International Journal of Angiology 6, no. 01 (April 23, 2011): 30–48. http://dx.doi.org/10.1007/bf01616230.

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24

Antel, Jack, Alan Thompson, and William Carroll. "Chronic cerebrospinal venous insufficiency." Multiple Sclerosis Journal 16, no. 7 (July 2010): 770. http://dx.doi.org/10.1177/1352458510374342.

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25

Barreto, Andrew D., Staley A. Brod, Thanh‐Tung Bui, James R. Jemelka, Larry A. Kramer, Kelly Ton, Alan M. Cohen, et al. "Chronic cerebrospinal venous insufficiency." Annals of Neurology 73, no. 6 (February 26, 2013): 721–28. http://dx.doi.org/10.1002/ana.23839.

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26

Taheri, Syde A., Julie Cullen, and Thomas Wormer. "Venous Reconstruction in Chronic Venous Insufficiency." Vascular Surgery 23, no. 6 (November 1989): 470–74. http://dx.doi.org/10.1177/153857448902300609.

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27

Alguire, Patrick C., and Barbara M. Mathes. "Chronic Venous Insufficiency and Venous Ulceration." Journal of General Internal Medicine 12, no. 6 (June 1997): 374–83. http://dx.doi.org/10.1046/j.1525-1497.1997.00063.x.

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28

Alguire, Patrick C., and Barbara M. Mathes. "Chronic Venous Insufficiency and Venous Ulceration." Journal of General Internal Medicine 12, no. 6 (June 1997): 374–83. http://dx.doi.org/10.1007/s11606-006-5087-4.

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29

Barajas Colón, José Ángel, Juan José Granados-Romero, Baltazar Barrera-Mera, Rodrigo Banegas-Ruiz, José Juan Vargas-Morales, Elvira Barrera-Calva, Francisco Fabián Gómez-Mendoza, et al. "Chronic venous insufficiency: a review." International Journal of Research in Medical Sciences 9, no. 6 (May 27, 2021): 1808. http://dx.doi.org/10.18203/2320-6012.ijrms20211928.

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Chronic venous insufficiency (CVI) comprises a complete spectrum of morphological and functional abnormalities of the venous system1 including any long-term functional and morphological alteration. CVI accounts for several abnormalities of the venous system. It is a highly prevalent disease that causes serious economic consequences, a decrease in the quality of life and can lead to serious complications. An exhaustive review was performed with the available literature, using the PubMed, ScienceDirect, Scopus and Cochrane databases from 2004 to 2021. The search criteria were formulated to identify reports related to chronic venous insufficiency. The pathophysiology of chronic venous insufficiency begins with chronic venous hypertension and the dilation of the vessel, this leads to a series of pathological changes in the venous wall and surrounding tissues, in advanced stages of CVI, skin lesions are associated with an increased proliferation of skin capillaries and microcirculatory abnormalities that may be the result of an altered level of factors responsible for the angiogenic response, such as vascular endothelial growth factor (VEGF), fibroblast growth factor 2 (FGF2) and angiostatin. In this review, updates on pathophysiology, clinic, diagnosis, classification and treatment of this disease are analyzed, with special emphasis on therapeutic options. Chronic venous insufficiency is a disease that affects the patient at several levels, mainly diminishing his/her quality of life. Currently there are various treatments ranging from habit modifications, pharmacological, to endovenous and surgical treatment.

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30

Valencia, Isabel C., Anna Falabella, Robert S. Kirsner, and William H. Eaglstein. "Chronic venous insufficiency and venous leg ulceration." Journal of the American Academy of Dermatology 44, no. 3 (March 2001): 401–24. http://dx.doi.org/10.1067/mjd.2001.111633.

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31

Steins, Anke, Hans-Martin Häfher, Martin Hahn, and Michael Jünger. "Microcirculation in Chronic Venous Insufficiency." Phlebology: The Journal of Venous Disease 17, no. 3-4 (December 2002): 115–20. http://dx.doi.org/10.1177/026835550201700307.

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Objective: To study the microcirculation of the skin of the leg in patients with chronic venous disease of the lower limb, and to assess the effect of compression treatment. Patients and Methods: Patients were recruited from the vascular clinic and investigated by direct capillary pressure, transcutaneous oxygen tension, intravital video capillaroscopy and fluorescence video microscopy. The microcirculation was observed over the healing period in patients with venous leg ulcers. The effects of compression therapy on microcirculatory changes were studied in patients with Widmer stage I and II chronic venous disease. Results: In patients suffering from lipodermatosclerosis or venous leg ulcers retrograde pressure waves were detected in the nutritive capillaries of the skin by the ‘servo nulling’ pressure measurement during simulated calf muscle contraction. A close correlation was found between the degree of trophic skin change and the microangiopathy observed. Healing of venous ulcers occurred only if the cutaneous microcirculation in the ulcer area improved. Capillary density in base of the ulcer and at the border predicted venous ulcer healing. Conclusions: Cutaneous microangiopathy precedes the development of trophic skin alterations due to chronic venous disease and microcirculatory changes are closely related to the clinical stage of the disease as well as to the outcome of treatment.

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32

Shcheglov, E. A. "Chronic Venous Insufficiency and Gonarthrosis." N.N. Priorov Journal of Traumatology and Orthopedics 19, no. 2 (June 15, 2012): 31–34. http://dx.doi.org/10.17816/vto20120231-34.

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The rate of previously not diagnosed chronic insufficiency in patients with knee osteoarthrosis was determined. The effect of treatment measures directed to elimination of venous insufficiency was evaluated. It was shown that in 48.8% of patients chronic venous insufficiency was not diagnosed by orthopaedic surgeons. Observance of therapeutic regimen, use of elastic compression and phlebotropic drugs contributed to the elimination of gonarthrosis symptoms and improved the quality of life (by KOOS scale).

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33

Mościcka, Paulina, Justyna Cwajda-Białasik, Maria Teresa Szewczyk, and Arkadiusz Jawień. "Chronic venous insufficiency – clinical manifestation." Leczenie ran 16, no. 3-4 (2019): 84–91. http://dx.doi.org/10.5114/lr.2019.94621.

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34

Nuzum, Donald S., Tsion T. Gebru, and Samir A. Kouzi. "Pycnogenol for chronic venous insufficiency." American Journal of Health-System Pharmacy 68, no. 17 (September 1, 2011): 1589–601. http://dx.doi.org/10.2146/ajhp100676.

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35

Fowkes, F. G. R. "Epidemiology of Chronic Venous Insufficiency." Phlebology: The Journal of Venous Disease 11, no. 1 (March 1996): 2–5. http://dx.doi.org/10.1177/026835559601100102.

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Objective: To determine the prevalence of chronic venous insufficiency in the general population and its association with varicose veins. Data sources: MEDLINE search 1980–94 plus scanning of reference lists in articles obtained. Study selection: Studies on venous disease in subjects not attending health services. Data synthesis: A formal systematic review of metaanalysis was not carried out because of the heterogeneity of the few available studies. Skin changes were found to occur in over 3% of adults, more so in women than men. The prevalence was higher in subjects with varicose veins and depended on the definition of skin changes and the severity of varicose veins. Approximately 0.3% of adults had an open varicose ulcer, and around 1% had an open or healed ulcer. Prevalence was higher in women and increased with age. Conclusions: The prevalence of chronic venous insufficiency was found to be common in the general population, but more studies of distribution and aetiology are required.

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36

Bauersachs, J., I. Fleming, and R. Busse. "Pathophysiology of Chronic Venous Insufficiency." Phlebology: The Journal of Venous Disease 11, no. 1 (March 1996): 16–22. http://dx.doi.org/10.1177/026835559601100105.

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Objective: To review the physiological mechanisms determining venous return to the heart and the pathophysiological events culminating in chronic venous insufficiency (CVI), focusing primarily on the role of alterations in nitric oxide (NO) production by the vascular endothelium. Background: Congenital valve incompetence, thrombotic damage or venous outflow obstruction result in the development of chronic venous hypertension which frequently leads to ulceration. One major aetiological factor of trophic changes in the skin of patients with CVI is the phenomenon of leucocyte trapping. Hypothesis: It has been suggested that endothelial dysfunction, effectively resulting in a decrease in cellular levels of NO, is a key event in the initiation of enhanced adhesion molecule expression. Data: P-selectin, monocyte chemoattractant protein-1 and vascular cell adhesion molecule-1 expression can be enhanced by attenuating endothelial NO production. The mechanism by which NO alters the expression of genes encoding these adhesion molecules would appear to involve an interaction with transcription factors, in particular NF*** xB. Conclusion: Impaired endothelial NO synthesis associated with CVI may enhance the expression of adhesion molecules and chemotactic factors and lead to leucocyte adhesion and extravasation.

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37

De Backer, G., and G. De Backer. "Epidemiology of Chronic Venous Insufficiency." Angiology 48, no. 7 (July 1997): 569–76. http://dx.doi.org/10.1177/000331979704800703.

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38

Abascal, Kathy, and Eric Yarnell. "Botanicals for Chronic Venous Insufficiency." Alternative and Complementary Therapies 13, no. 6 (December 2007): 304–11. http://dx.doi.org/10.1089/act.2007.13609.

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39

Kistner, Robert L. "Diagnosis of chronic venous insufficiency." Journal of Vascular Surgery 3, no. 1 (January 1986): 185–88. http://dx.doi.org/10.1067/mva.1986.avs0030185.

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40

Gschwandtner, Michael E., and Herbert Ehringer. "Microcirculation in chronic venous insufficiency." Vascular Medicine 6, no. 3 (August 2001): 169–79. http://dx.doi.org/10.1177/1358836x0100600308.

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41

Rathbun, Suman. "Book review: Chronic Venous Insufficiency." Vascular Medicine 16, no. 6 (November 29, 2011): 443. http://dx.doi.org/10.1177/1358863x11426616.

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42

BRUNK, DOUG. "Obesity Worsens Chronic Venous Insufficiency." Clinical Endocrinology News 6, no. 6 (June 2011): 21. http://dx.doi.org/10.1016/s1558-0164(11)70269-3.

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43

Hach, W., V. Hach-Wunderle, and F. Präve. "Staging of chronic venous insufficiency." Gefässchirurgie 5, no. 4 (November 15, 2000): 0255–61. http://dx.doi.org/10.1007/s007720000103.

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44

Steins, Anke, Hans-Martin Häfner, Martin Hahn, and Michael Jünger. "Microcirculation in chronic venous insufficiency." Phlebology 17, no. 3-4 (December 2002): 115–20. http://dx.doi.org/10.1007/bf02638603.

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45

Rathbun, Suman W., and Angelia C. Kirkpatrick. "Treatment of chronic venous insufficiency." Current Treatment Options in Cardiovascular Medicine 9, no. 2 (May 2007): 115–26. http://dx.doi.org/10.1007/s11936-007-0005-6.

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46

Kistner, Robert L. "Diagnosis of chronic venous insufficiency." Journal of Vascular Surgery 3, no. 1 (January 1986): 185–88. http://dx.doi.org/10.1016/0741-5214(86)90098-4.

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47

Partsch. "Varicose veins and chronic venous insufficiency." Vasa 38, no. 4 (November 1, 2009): 293–301. http://dx.doi.org/10.1024/0301-1526.38.4.293.

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Varicose veins are a very frequent disorder with prevalence in our adult population between 14 % for large varices and 59 % for small teleangiectasias. Subjective symptoms may be very non-specific. The term “chronic venous insufficiency (CVI)” defines functional abnormalities of the venous system producing advanced symptoms like oedema, skin changes or leg ulcers. Both entities, varicose veins and CVI, may be summarized under the term “chronic venous disorders” which includes the full spectrum of morphological and functional abnormalities of the venous system. A classification system to describe chronic venous disorders regarding clinical appearance, etiology, anatomical distribution and pathophysiology has been proposed under the acronym of CEAP. The revised version of the CEAP classification contains also definitions of clinical signs and suggests three levels of apparative investigations adjusted to the clinical stage. Concerning the etiology of venous disorders controversial theories exist leading to different therapeutic concepts. As a matter of fact there is a vicious circle between structural changes in valves and venous wall and hemodynamic forces leading to reflux and venous hypertension. Different methods for treating varicose veins are available producing satisfactory early outcome in most cases, but followed by a high recurrence rate after years. Chronic venous insufficiency requires “chronic management”. Compression therapy by bandages for initial treatment of severe stages and maintenance therapy using medical compression stockings is essential. In addition correction of venous refluxes by surgery or endovenous procedures including echo-guided foam sclerotherapy should be considered in every single case.

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48

Dörler, M., and M. Stücker. "Pain in chronic venous insufficiency (including venous claudication)." Phlebologie 43, no. 06 (November 2014): 287–89. http://dx.doi.org/10.12687/phleb2237-6-2014.

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SummaryPain in chronic venous insufficiency can indicate an acute complication in the form of superficial thrombosis or deep venous thrombosis of the leg or chronic symptoms associated with venous leg ulcers or venous claudication. Up to 80 % of patients with venous leg ulcers report pain. This pain is a principal cause of the reduced quality of life of such patients. A distinction must be made between the acute pain arising from dressing changes, wound cleaning and debridement and pain occurring between these procedures. The pain should be evaluated systematically using visual analogue scales, as any increase in pain can be a warning sign of wound infections, irritation or allergic reactions to the wound dressings or exacerbation of a vascular disorder (e.g. additional peripheral arterial occlusive disease). Venous claudication occurs in the form of leg pain with a sensation of constriction on physical exertion, which subsides with rest. In the majority of cases, it is a symptom of reduced iliofemoral venous outflow. It occurs in up to 43.6 % of patients after iliofemoral thromboses.

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49

Jones, G. T., C. Solomon, A. Moaveni, A. M. van Rij, I. A. Thomson, and I. Galvin. "Venous Morphology Predicts Class of Chronic Venous Insufficiency." European Journal of Vascular and Endovascular Surgery 18, no. 4 (October 1999): 349–54. http://dx.doi.org/10.1053/ejvs.1999.0902.

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50

Mosmiller, Lindsey T., Kelsey N. Steele, Carl D. Shrader, and Ashley B. Petrone. "Evaluation of inflammatory cell biomarkers in chronic venous insufficiency." Phlebology: The Journal of Venous Disease 32, no. 9 (April 5, 2017): 634–40. http://dx.doi.org/10.1177/0268355517701806.

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Objective Inflammation has been implicated as a factor that may contribute to chronic venous insufficiency. The purpose of this study is to compare readily available inflammatory cell biomarkers, with an emphasis on neutrophil count, lymphocyte count, and neutrophil lymphocyte ratio, in patients with chronic venous insufficiency. We hypothesized that circulating leukocyte counts would be higher in the peripheral blood of patients with severe compared to mild chronic venous insufficiency. Methods We performed a retrospective medical record review of patients discharged from Ruby Memorial Hospital (Morgantown, WV, USA) with a primary diagnosis of chronic venous insufficiency. Patients were organized into two groups—mild and severe chronic venous insufficiency—based on the Clinical, Etiologic, Anatomic, and Pathophysiological classification system, and inflammatory cell counts were compared between groups. Results We observed a significantly higher neutrophil count ( p = .002) and neutrophil-lymphocyte ratio ( p = .005) in patients with severe chronic venous insufficiency compared to mild. Further, the neutrophil–lymphocyte ratio may be a useful predictor of chronic venous insufficiency severity. Conclusions We reported significant differences in inflammatory cell biomarkers between mild and severe chronic venous insufficiency, as well as provided support for the use of the neutrophil–lymphocyte ratio as a predictor of chronic venous insufficiency severity. These results may provide clinicians with additional insight to manage chronic venous insufficiency in patients and provide a framework for the development of novel treatment options targeting the immune system in chronic venous insufficiency.

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