Books on the topic 'Chronic toxicity testing'

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1

Agency, Illinois Environmental Protection. IEPA mobile laboratory screening tests information sheet. Springfield, IL]: Illinois Environmental Protection Agency, Division of Laboratories, 1993.

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2

Rach, J. J. Acute and chronic toxicity of rotenone to Daphnia magna. Washington, D.C: U.S. Dept. of the Interior, Fish and Wildlife Service, 1988.

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3

Horning, William B. Short-term methods for estimating the chronic toxicity of effluents and receiving waters to freshwater organisms. Cincinnati, OH: U.S. Environmental Protection Agency, Environmental Monitoring and Support Laboratory, 1986.

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4

Horning, William B. Short-term methods for estimating the chronic toxicity of effluents and receiving waters to freshwater organisms. Cincinnati, OH: U.S. Environmental Protection Agency, Environmental Monitoring and Support Laboratory, 1986.

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5

Horning, William B. Short-term methods for estimating the chronic toxicity of effluents and receiving waters to freshwater organisms. Cincinnati, OH: U.S. Environmental Protection Agency, Environmental Monitoring and Support Laboratory, 1986.

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6

Horning, William B. Short-term methods for estimating the chronic toxicity of effluents and receiving waters to freshwater organisms. Cincinnati, OH: U.S. Environmental Protection Agency, Environmental Monitoring and Support Laboratory, 1986.

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7

Horning, William B. Short-term methods for estimating the chronic toxicity of effluents and receiving waters to freshwater organisms. Cincinnati, OH: U.S. Environmental Protection Agency, Environmental Monitoring and Support Laboratory, 1986.

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8

W, Moore David. Chronic sublethal effects of San Francisco Bay sediments on Nereis (Neanthes) arenaceodentata: Effect of food ration on sediment toxicity. Vicksburg, MS: US Army Corps of Engineers, Waterways Experiment Station, 1993.

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9

Moore, David W. Chronic sublethal effects of San Francisco Bay sediments on Nereis (Neanthes) arenaceodentata: Effect of storage time on sediment toxicity. Vicksburg, MS: US Army Corps of Engineers, Waterways Experiment Station, 1994.

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10

W, Moore David. Chronic sublethal effects of San Francisco Bay sediments on "Nereis (Neanthes) Arenaceodentata": Nontreatment factors. [Vicksburg, Miss: U.S. Army Engineer Waterways Experiment Station, 1992.

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11

Moses, Jennifer. Short term chronic and acute toxicity screening of water and sediment using fathead minnows, daphnids, rotifers (Rotox®) and light emitting bacterial (Microtox®), ambient stream monitoring, summers of 1990 and 1991. Muscle Shoals, Ala: Tennessee Valley Authority, Resource Group, River Basin Operations, Water Resources, 1992.

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12

W, Moore David. Chronic sublethal effects of San Francisco Bay sediments on Nereis (Neanthes) arenaceodentata: Full life-cycle exposure to bedded sediments. Vicksburg, Miss: U.S. Army Corps of Engineers, Waterways Experiment Station, 1993.

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13

W, Moore David. Chronic sublethal effects of San Francisco Bay sediments on Nereis (Neanthes) arenaceodentata: Interpretative guidance for a growth end point. Vicksburg, MS: US Army Corps of Engineers, Waterways Experiment Station, 1993.

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14

Programme, Canada Therapeutic Products, ed. Guidance for industry: Duration of chronic toxicity testing in animals (rodent and non rodent toxicity testing) ICH topic S4A. Ottawa, Ont: Therapeutic Products Programme, Health Canada, 2000.

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15

Statistical approach to predicting chronic toxicity of chemicals to fishes from acute toxicity test data. Gulf Breeze, FL: U.S. Environmental Protection Agency, Environmental Research Laboratory, Office of Research & Development, 1992.

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16

European Centre for Ecotoxicology and Toxicology of Chemicals., ed. Practical concepts for dose selection in chronic toxicity and carcinogenicity studies in rodents. Brussels: ECETOC, 1996.

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17

W, Hall Lenwood, and Chesapeake Bay Program (U.S.), eds. Standard operating procedures for conducting acute and chronic aquatic toxicity tests with Eurytemora affinis, a Calanoid copepod. Annapolis, Md: Printed by the U.S. Environmental Protection Agency for the Chesapeake Bay Program, 1998.

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18

I, Weber Cornelius, Environmental Monitoring Systems Laboratory (Cincinnati, Ohio), and United States. Environmental Protection Agency. Office of Modeling, Monitoring Systems, and Quality Assurance, eds. Short-term methods for estimating the chronic toxicity of effluents and receiving waters to freshwater organisms. 2nd ed. Cincinnati, Ohio: U.S. Environmental Protection Agency, Environmental Monitoring Systems Laboratory, 1989.

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19

A, Chapman Gary, Denton Debra L, Lazorchak James M, and National Exposure Research Laboratory (U.S.), eds. Short-term methods for estimating the chronic toxicity of effluents and receiving waters to west coast marine and estuarine organisms. Cincinnati, Ohio: National Exposure Research Laboratory, Office of Research and Development, U.S. Environmental Proection Agency, 1995.

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20

Short-term methods for estimating the chronic toxicity of effluents and receiving waters to west coast marine and estuarine organisms. Cincinnati, OH: U.S. EPA, Office of Research and Development, National Exposure Research Laboratory, 1995.

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21

Chronic sublethal effects of San Francisco Bay sediments on Nereis (Neanthes) arenaceodentata: Bioaccumulation from bedded sediments. Vicksburg, Miss: US Army Corps of Engineers, Waterways Experiment Station, 1993.

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22

Srivastava, Siddharth, and Jeffrey Chinsky. Methylmalonic Acidemia. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0062.

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Abstract:
Isolated methylmalonic acidemia (MMA) refers to a group of inborn errors of organic acid metabolism caused by impaired conversion of methylmalonyl-CoA to succinyl-CoA. Individuals with MMA experience both acute and chronic neurological complications. The pathophysiology likely reflects impaired energy metabolism in the mitochondria leading to neuronal toxicity in MMA. MMA presents with a spectrum of clinical phenotypes with onset of symptoms anytime from the neonatal period to adulthood. Once there is clinical suspicion for MMA, definitive diagnosis requires biochemical testing. The treatment of MMA centers on acute interventions when affected individuals are ill, as well as preventative measures when they are doing relatively well. Neuroimaging in MMA demonstrates a variety of findings.
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