Relevant bibliographies by topics / Chronic rhinosinusitus

Academic literature on the topic 'Chronic rhinosinusitus'

Author: Grafiati
Published: 10 December 2022
Last updated: 28 January 2023

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Journal articles on the topic "Chronic rhinosinusitus"

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Senior, Kathryn. "Is chronic rhinosinusitus a fungal problem?" Lancet Infectious Diseases 4, no. 5 (May 2004): 257. http://dx.doi.org/10.1016/s1473-3099(04)01018-7.

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Acharya, Rabin Chandra, B. Pradhan, and N. M. Thapa. "Outcome of functional endoscopic surgery for chronic rhinosinusitus." Nepalese Journal of ENT Head and Neck Surgery 5, no. 1 (February 28, 2017): 20–21. http://dx.doi.org/10.3126/njenthns.v5i1.16872.

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Objective: To measure the outcome of Functional Endoscopic Sinus Surgery (FESS) for Chronic Rhinosinusitis in early postoperative period.Materials and Methods: This is a prospective comparative study conducted in Ganesh Man Singh Memorial Academy of ENT-Head and Neck Studies, Tribhuvan University Teaching Hospital, Kathmandu, Nepal from November 2009 to March, 2011. Thirty cases of Chronic Rhinosinusitis diagnosed by clinical and radiological criteria were included in this study. Modified sinonasal outcome test was used to record the pre and postoperative scores and compared by using paired t- test.Results: All the cases showed significant improvement in postoperative scores in both physical and psychosocial domains of modified sinonasal outcome test. Two symptoms, concentration and misery of psychosocial domain didn’t improve significantly.Conclusion: This study attempts to measure the outcome of FESS in patients with CRS. Nepali version of SNOT-10 has been used in the Nepalese population. Significant improvement in quality of life score has been observed in early postoperative period.
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SARRAZIN, J. "627 Expression and synthesis of growth factors in chronic rhinosinusitus." Journal of Allergy and Clinical Immunology 105, no. 1 (January 2000): S212. http://dx.doi.org/10.1016/s0091-6749(00)91055-8.

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Ahn, Jae-Cheul, Jeong-Whun Kim, Chul Hee Lee, and Chae-Seo Rhee. "Prevalence and Risk Factors of Chronic Rhinosinusitus, Allergic Rhinitis, and Nasal Septal Deviation." JAMA Otolaryngology–Head & Neck Surgery 142, no. 2 (February 1, 2016): 162. http://dx.doi.org/10.1001/jamaoto.2015.3142.

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Gariuc, Lucia, Alexandru Sandul, and Lupoi Daniel. "Invasive fungal rhinosinusitis." Romanian Journal of Rhinology 9, no. 33 (March 1, 2019): 13–19. http://dx.doi.org/10.2478/rjr-2019-0001.

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Abstract Invasive fungal rhinosinusitides are a group of disorders with three subtypes (acute invasive fungal rhinosinusitis, chronic invasive fungal rhinosinusitis and granulomatous invasive fungal rhinosinusitis), requiring urgent diagnosis and early treatment due to the reserved vital and functional prognosis. This disorder occurs in immunocompromised patients, but it can also occur in immunocompetent people. Aspergillus and Mucormicosis species are the most common microorganisms found in invasive fungal rhinosinusites. The otorhinolaryngologic clinical examination and imaging techniques provide important diagnostic information in patients with risk factors for invasive fungal rhinosinusitis, including intracranial or orbital extension identification. The treatment of invasive fungal rhinosinusites (acute or chronic) consists of reversing immunosuppression, appropriate systemic antifungal therapy and aggressive and prompt surgical debridement of the affected tissues.
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Kochetkov, P. A., V. A. Svistushkin, and E. S. Shchennikova. "Intranasal glucocorticosteroids for the complex treatment of patients with chronic diseases of the nose and paranasal sinuses." Meditsinskiy sovet = Medical Council, no. 6 (May 27, 2020): 66–70. http://dx.doi.org/10.21518/2079-701x-2020-6-66-70.

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Intriduction. Chronic rhinitis and rhinosinusitis noticeably deteriorate the patients’ quality of life and lead to the accompanying upper airway pathology development. The main purpose of treatment of this group of patients is to decrease severity of symptoms and the second one to prevent complications. The optimal therapy will help patients to maintain their lifestyle. Intranasal glucocorticosteroids are first-line drugs to treat acute rhinosinusitis or exacerbations of chronic rhinosinusitis in adults (including the elderly) and adolescents aged 12 years and older as an auxiliary therapeutic agent if treated by antibiotics, and to treat acute rhinosinusitis with mild to moderate symptoms without signs of severe bacterial infection. In the number of trials, mometasone furoate effectiveness in regard to decreasing of prominent symptoms with no side effects development has been shown.Objective: this article reviews available data on the effectiveness of intranasal corticosteroids – mometasone furoate – in the treatment of different forms of chronic inflammatory diseases of the nose and paranasal sinuses.Methods: information for this review was identified through a RISC and MEDLINE databases applying key words.Conclusions: based on the available data, treatment of chronic rhinosinusites and rhinitis should be initiated by conservative therapy. Summarizing information from the available literature we can conclude that treatment by mometasone furoate improve quality of life decreasing clinical symptoms of chronic rhinosinusitis and rhinitis.
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Ivanov, M. O., N. M. Ivanova, M. V. Maksimenya, T. M. Karavaeva, E. V. Egorova, E. V. Fefelova, and N. N. Tsybikov. "Changed content of heat shock proteins and antibodies to them in blood and nasal mucosa cells in rhinites and rhinosinusites of different etiology." Perm Medical Journal 35, no. 6 (December 30, 2018): 23–28. http://dx.doi.org/10.17816/pmj35623-28.

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Aim. To determine the content of heat shock proteins with molecular weight 70 kDa (HSP 70) and antibo dies to them in blood and nasal secretion in patients with allergic rhinites and infectious rhinosinusites of different etiology. Materials and methods. The paper presents the results of examination of 10 patients with allergic rhinitis and 30 patients, infected with rhinosinusites(the age range 25–35 years). The patients with infectious rhinosinusites were divided into 3 groups according to nosologic form of disease. The control group included 10 practically healthy persons in the ratio, comparable by their gender and age with sick persons. Results.The analysis showed that in the nasal secretion of all patients, HSP 70 level significantly raised compared to the control. Maximum values were registered in patients with bacterial rhinosinusitis and were higher than in patients with viral and fungous ones by 1.9 times (p = 0.015) and 2.9 times (p = 0.001), respectively. In blood serum, HSP 70 concentration compared with the control increased in patients with allergic rhinitis and bacterial rhinosinusitis by 103.67 % (p = 0.015) and 32.11 % (p = 0.049), respectively; these values in the last two groups exceeded the latter in patients with fungous RS by 2.37 times (p = 0.01) and by 1.54 times (p = 0.035). Conclusions. It was detected that in the group of patients with allergic rhinitis and chronic bacterial rhinosinusitis in the nasal secretion and blood serum, HSP 70 values were the highest. In the nasal secretion, HSP 70 level was higher than in blood. The amount of autoantibodies to HSP 70 in blood grew in allergic rhinitis, fungous and viral forms of rhinosinusites that reflects the immunological effect of chaperone proteins.
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Ramadan, Hassan H., and Justin Douglas. "S246 – Chronic Sinusitis in Children: Which Sinuses Are Involved." Otolaryngology–Head and Neck Surgery 139, no. 2_suppl (August 2008): P157. http://dx.doi.org/10.1016/j.otohns.2008.05.421.

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Objectives 1) Learn which paranasal sinuses are involved in chronic rhinosinusitis in children. 2) Be able to customize surgical treatment based on those findings. Methods Retrospective review of children who had sinus surgery over a period of 10 years. 76 children whose age ranged between 3 and 14 years had their CT scans reviewed to determine which sinuses were diseased. All children had a CT scan because of failure of medical management and were considered for surgery. Outcome was to determine which sinuses were developed and of those, which ones were diseased and what was the severity of the disease. Results The maxillary sinuses were the most common sinuses involved in children with chronic rhinosinusits (92%). The ostiomeatal complex was next most common area involved in these children (88%). Conclusions The maxillary sinuses, followed by the ostiomeatal complex, were the most common areas involved in children with chronic rhinosinusitis. Initial surgical management of these children should then be based on those findings.
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Dotlic, Jelena, and Jelena Marinkovic. "Health-related quality of life in patients with chronic rhinosinusitis." Srpski arhiv za celokupno lekarstvo 137, no. 9-10 (2009): 470–74. http://dx.doi.org/10.2298/sarh0910470d.

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Introduction The need for assessing the quality of life in chronic rhinosinusitis is emphasized by the medical, social and economic importance of this pathological condition. Questionnaires have been employed in majority of studies while there are no available data that Q method has been used for quality of life research in chronic rhinosinusitis up to the present. Objective The aim of this study was to identify, group and analyze subjective perception of the impact of the disease on health-related quality of life in patients with chronic rhinosinusitis. Methods The study involved 36 consecutive patients of both sexes, older than 10 years of age, diagnosed with chronic rhinosinusitis in the tertiary health clinic in three successive weeks. They were selected for medicamentous treatment. Health-related quality of life was assessed by Q method. Through 34 representative statements, which were sorted by the examinees related to subjective priorities, 10 key quality of life domains were examined. The data were processed by factor analysis employing specialized software package PQMethod. Results Analyzing individual opinions of the examinees, five different opinion-types (factors) on the impact of chronic rhinosinusitis on quality of life were demonstrated. They were marked numerically and descriptively according to dominant characteristics of the group: Factor 1 - stable group (47.2% of individuals), Factor 2 - symptomatic group (11.1% of individuals), Factor 3 - mental group (5.6% of individuals), Factor 4 - physical group (11.1% of individuals) and Factor 5 - optimistic group (25.0% of individuals). Each group was systematically analyzed. Conclusion Majority of patients with chronic rhinosinusits (72.2%) were found to be satisfied with the quality of life, while just a small number (5.6%) heavily bore the illness which especially jeopardised their mental health. This study demonstrates the strength of Q method in analyzing and categorizing subjectivity and offers quality practical information which enables a more comprehensive approach and more adequate intervention in patients with chronic rhinosinusitis.
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Vokhidov, Ulugbek N. "IMMUNOHISTOCHEMICAL STUDY OF MESENCHYMAL FORMATIONS OF CHRONIC POLYPOID RHINOSINUSITIS." Oriental Journal of Medicine and Pharmacology 02, no. 01 (March 1, 2022): 144–52. http://dx.doi.org/10.37547/supsci-ojmp-02-01-11.

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: The aim of the study was to investigate the mesenchymal formations in the stroma of polyps of different forms of chronic polypoid rhinosinusitis. We carried out morphological and immunohistochemical study of paraffin blocks prepared from nasal polyps, which remote by endoscopic operation in 45 patients with chronic polypoid rhinosinusitis in 2013. The study showed that the observation of mesenchymal formations in nasal polyps, which could be regarded as a growth zone of polyps.
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Dissertations / Theses on the topic "Chronic rhinosinusitus"

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Al-Mot, Sawsan. "Molecular signatures as a new classification scheme for chronic rhinosinusitus." Thesis, McGill University, 2013. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=114268.

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Chronic Rhinosinusitis (CRS), an inflammation of the paranasal sinus cavities, is a common disorder of uncertain etiology that affects the upper airways and paranasal sinuses. Biopsy specimens had taken from CRS patients document disruption of the normal epithelial architecture, in addition to an intense infiltration of inflammatory cells, consisting mainly of eosinophils. Current clinical classification of CRS is based on the presence or absence of nasal polyposis; however, no consistent difference in histological aspect characterizes these two groups. Recently, we have identified distinct gene expression patterns in cultured epithelial cells obtained from surgical CRS subjects. These molecular signatures, which differ from clinical phenotype, may help better differentiate this disorder than clinical phenotype. In our current study we investigated the histological pattern associated with these two different molecular signatures in surgical biopsies obtained from CRS patients and control subjects. Cellular infiltrates were identified using immunohistochemistry (IHC) staining using three markers: neutrophil elastase (NE), CD68, Major basic protein (MBP). Macrophage activation status into classical and alternatively activated macrophages was verified by double-staining for CD68 and CD 206 markers. Results were reported both according to standard clinical criteria (CRSwNP and CRSsNP) and also according to their expression signature into two groups (CRS1, CRS2) and control subjects. Expression signatures were validated using immunohistochemical staining for the highest differentially expressed marker, CCL2. Results showed differences in the number of eosinophils, macrophages and neutrophils cells in CRS patients compared to the control subjects. Using conventional criterion, eosinophilia was higher in the CRSwNP group, but not greatly different for neutrophils or macrophages between the two groups. Using the molecular signatures to assign groups, eosinophilia was similar between both groups, however, there was a significant increase in the number of neutrophils and macrophages in CRS1 comparing to CRS2. The CRS2 group had a higher incidence of alternatively activated macrophages, supporting the concept of a less inflammatory, immunotolerant CRS2 phenotype. Validity of the molecular signature was supported by demonstration of increased levels of protein product of CCL2 expression in CRS1 compared to CRS2. Taken together, these results identify a molecular phenotype of CRS that is characterized by a marked neutrophilic infiltration, and a second one that is markedly less inflammatory, accompanied by alternative macrophage activation. This suggests that these expression signatures may identify novel mechanism-based phenotypes, which differ from the clinical phenotype, and can help in providing a better understanding of pathophysiologic mechanism and phenotypes of CRS.
La rhinosinusite chronique (RSC), une inflammation des sinus paranasaux, est un trouble commun avec une étiologie incertaine, qui affecte les voies respiratoires supérieures et les sinus paranasaux. Les biopsies des échantillons prélevés sur des patients atteints de RSC documentent la perturbation de l'architecture normale épithéliale, en plus d'une infiltration de cellules inflammatoires intense constituée principalement par des éosinophiles. La classification clinique actuelle de la RSC est basée sur la présence (CRSwNP) ou l'absence (CRSsNP) de polypose nasale, mais aucune différence consistente de l'aspect histologique caractérise ces deux groupes. Récemment, nous avons identifié des profils d'expression génique distincts dans des cultures de cellules épithéliales provenant de sujets atteints de la RSC ayant subis une chirurgie des sinus. Ces signatures moléculaires, qui diffèrent du phénotype clinique, peuvent aider à mieux différencier ce trouble que le phénotype clinique. Dans notre étude, nous avons étudié l'aspect histologique associé à ces deux différentes signatures moléculaires à partir de biopsies chirurgicales obtenus chez des patients atteints de la RSC et les sujets témoins. Les infiltrats cellulaires ont été identifiés par immunohistochimie (IHC), une coloration à l'aide de trois marqueurs: l'élastase de neutrophile (NE), le CD68 et la protéine basique majeure (MBP). L'état d'activation des macrophages dans les formes classiques et alternativement activés a été vérifié par une double-coloration pour les marqueurs CD68 et CD206. Les résultats ont été rapportés à la fois selon les critères cliniques habituels (CRSwNP et CRSsNP) et aussi en fonction de leur signature d'expression en deux groupes (CRS1, CRS2) et les sujets témoins. Les signatures d'expression ont été validées à l'aide de coloration immunohistochimique pour le marqueur le plus différentiellement exprimé, le CCL2.Les résultats ont montré des différences dans le nombre d'éosinophiles, macrophages et les cellules de neutrophiles chez les patients atteints de la RSC par rapport aux sujets témoins. Selon le critère classique, l'éosinophilie était plus élevée dans le groupe CRSwNP, mais pas très différent entre les deux groupes pour les neutrophiles ou les macrophages. En utilisant les signatures moléculaires pour assigner des groupes, l'éosinophilie était similaire entre les deux groupes, cependant, il y avait une augmentation significative du nombre de neutrophiles et de macrophages dans CRS1 comparativement à CRS2. Le groupe CRS2 avait une incidence plus élevée des macrophages alternativement activés, supportant le concept d'une inflammatoire basse, phénotype CRS2 immunotolérant. La validité de la signature moléculaire a été supportée par la démonstration du niveau accru de la protéine produite par l'expression de CCL2 dans CRS1 par rapport à CRS2.En somme, ces résultats mettent en évidence un phénotype moléculaire de la RSC qui se caractérise par une infiltration neutrophilique marquée, et une seconde qui est nettement moins inflammatoire, accompagnée par l'activation alternative des macrophages. Ceci suggère que ces signatures d'expression peuvent identifier de nouveaux mécanismes basés sur des phénotypes, qui diffèrent du phénotype clinique, et peuvent aider à fournir une meilleure compréhension du mécanisme physiopathologique et les phénotypes de la RSC.
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Vaidyanathan, Sriram. "Optimising therapeutic strategies for chronic rhinosinusitis." Thesis, University of Dundee, 2014. https://discovery.dundee.ac.uk/en/studentTheses/337b63fd-4590-4ef9-a55f-8bf447986906.

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The aim of this thesis is to evaluate and optimise current pharmacotherapeutic options in rhinosinusitis. There is often a marked variation in treatment response in those afflicted with chronic rhinosinusitis, both within and between patients, attributable in part to different disease phenotypes/endotypes, poor awareness of treatment optimization options, and trivialization of symptoms by patients and physicians. Characteristically, these factors contribute to a typical remitting and relapsing disease course. The objectives of this work are to improve the therapeutic index and reach of commonly used medications by boosting efficacy whilst reducing concomitant side effects. The third chapter explores the use of initial oral steroids in patients with chronic rhinosinusitis and nasal polyposis, focusing on the role of the ostiomeatal complex in the perpetuation of disease symptoms. Often a short course of oral steroids is used in patients with moderate to severe disease to achieve initial control before maintenance with intranasal steroids. This is termed as a ‘medical polypectomy’ and anecdotally is commonly used in patients with chronic rhinosinusitis with nasal polyposis (CRSwNP). However, the evidence for its efficacy is tenuous and there are no data to evaluate if it indeed re-establishes ostiomeatal sinus complex drainage which is a condicio sine qua non of ensuring long-term symptom resolution. Further, it is known that monotherapy with nasal steroids may result in loss of symptom control. We have therefore in a double-blind placebo controlled trial (Chapter 4) evaluated the effect of this initial induction with oral steroids on subsequent sequential intranasal therapy. Perhaps, however, more crucially we have for the first time comprehensively addressed the safety of both oral and topical steroids in patients with CRSwNP who have other concomitant steroid-dependent illnesses like asthma and COPD. A particularly refractory subset of those with CRSwNP also have aspirin intolerance and asthma. While recent guidelines have recommended more aspirin challenge testing in these patients, it is unclear what the significance of a positive test is in the absence of overt clinical symptoms or in patients with only moderate disease. This is addressed in Chapter 5, as this significant phenotype of aspirin intolerant rhinosinusitis need close monitoring, dose optimization, polytherapy, and in selected cases may be suitable for aspirin desensitization. Penultimately, we evaluate in a double-blind placebo controlled trial (Chapter 6) the tachyphylaxis and rebound congestion that blights the medium to long-term use of sympathomimetic nasal decongestant sprays like oxymetazoline and if this can be reversed by the concomitant use of nasal steroids. We also characterized nasal blood flow as an outcome to evaluate in these patients and its relation to other rhinological outcome measures (Chapter 7).
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Erskine, Sally. "The epidemiology and experience of chronic rhinosinusitis." Thesis, University of East Anglia, 2017. https://ueaeprints.uea.ac.uk/66950/.

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Chronic rhinosinusitis (CRS) is a common and debilitating disorder. There is a deficit of knowledge about the epidemiology of CRS or the experience of sufferers. The aims of the study were to identify differences in socio-economic variables and quality of life between patients with chronic rhinosinusitis and healthy controls, to identify any significant associations between CRS and other medical co-morbidities, psychiatric disease or environmental exposures and to explore the experience of CRS from the perspective of CRS sufferers. This study consisted of a self-reported questionnaire distributed from 30 ENT clinics across the UK, and qualitative interviews with 21 patients with CRS. Additional studies were undertaken to support this work including further qualitative interviews with patients who have disturbed olfaction, and studies to assess new or unproven treatment regimens including a feasibility study for Clarithromycin for CRS and a trial of sodium citrate for hyposmia. No clear differences in socioeconomic variables were identified between cases and controls. CRS was found to be strongly associated with asthma and inhaled allergies as well as significantly impairing quality of life. Quality of life issues were very important to sufferers, and had been poorly addressed, particularly with regards to sense of smell. Further research is needed to better understand and manage CRS although better adherence to current guidelines would improve care in the interim.
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Wallwork, Benjamin, and n/a. "The Anti-Inflammatory Effect of Macrolide Antibiotics in Chronic Rhinosinusitis." Griffith University. School of Biomolecular and Biomedical Science, 2006. http://www4.gu.edu.au:8080/adt-root/public/adt-QGU20070201.160023.

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Chronic rhinosinusitis is a common disorder of chronic inflammation of the upper respiratory tract. It is associated with significant symptoms and impairment of the quality of life of sufferers. Despite recent advances in the medical and surgical management of chronic rhinosinusitis, there remains a population of patients who fail to obtain relief from their symptoms. Chronic inflammation of the mucosa of the nasal cavity and paranasal sinuses is one of the hallmarks of chronic rhinosinusitis. This inflammation is demonstrated by an increased number of chronic inflammatory cells, elevated levels of pro-inflammatory cytokines, increased expression of adhesion molecules and metaplastic changes in the epithelium. The current medical treatments for chronic sinusitis aim to reduce this inflammation and consequently improve symptoms. In recent years, evidence has emerged that macrolide antibiotics have an anti-inflammatory effect that is separate from their anti-bacterial effect. This effect was first described in the treatment of diffuse panbronchiolitis, a disorder of chronic inflammation of the lower respiratory tract. Following the success of macrolides in treating this condition it was trialed in chronic rhinosinusitis. Several open-label trials have subsequently demonstrated a beneficial effect. Laboratory studies have investigated the mechanism of the anti-inflammatory effect of macrolides. These have shown that macrolides effect cytokine production, inflammatory cell apoptosis, expression of adhesion molecules, neutrophil oxidative burst, bacterial virulence and mucociliary function. In this thesis we report a series of experiments designed to further investigate the mechanism of action and clinical effect of macrolides. In vitro studies using whole sections of chronic rhinosinusitis mucosa cultured for 24 hours in macrolide, prednisolone or control showed that macrolide and prednisolone produced significant reductions in the production of interleukin-5, interleukin-8 and granulocyte-macrophage colony stimulating factor. The same cultured specimens also showed a reduction in expression of transforming growth factor-?. No reduction was seen in the expression of the key pro-inflammatory nuclear transcription factor Nuclear factor-?B. In our in vivo experiments, biopsies were taken from chronic rhinosinusitis patients who had received a 3-month course of macrolide. These biopsies showed a reduction in the number of neutrophils present following treatment. There was no reduction in the number of other inflammatory cells or in the expression of TGF-? and NK-?B. We have performed the first ever double-blinded, randomized, placebo-controlled trial of macrolide in the treatment of chronic rhinosinusitis. Patients receiving macrolide showed significant improvements in saccharine transit time, nasal endoscopic scoring and symptom scores following a 12 week course. Patients with low levels of serum immunoglobulin E showed significantly improved outcomes compared to those with high levels. Interleukin-8 levels in nasal lavage fluid were significantly reduced in the patients with low levels of IgE following macrolide treatment. No improvements in any of the objective or subjective outcome measures were seen in the placebo-treated patients. We have performed a series of experiments investigating the anti-inflammatory effect of macrolide antibiotics from 'the bench to the bedside'. These experiments have provided insight into the mechanism of action of macrolides in the laboratory setting and evidence of a beneficial effect in the treatment of chronic rhinosinusitis patients.
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Viswanathan, Harishnath. "Mucin Gene Expression and GastricReflux in Chronic Rhinosinusitis and Otitis Media with Effusion." Thesis, University of Newcastle Upon Tyne, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.499336.

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Ball, Stephen Leslie. "The role of epithelial cells and fibroblasts in the pathogenesis of chronic rhinosinusitis." Thesis, University of Newcastle upon Tyne, 2017. http://hdl.handle.net/10443/3726.

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Chronic rhinosinusitis without nasal polyps (CRSsNP) is a heterogeneous condition with common symptoms, clinical and radiological findings. CRSsNP is typified by inflammation of the sinonasal epithelium and development of fibrosis, yet its precise pathophysiology remains elusive. Recently stromal cells have been shown to act like immune effector cells in orchestrating chronic inflammation. Histological analysis of tissue biopsies from patients with CRSsNP demonstrates recruitment of circulating inflammatory cells, though the precise role of structural cells such as epithelial and fibroblast cells in CRSsNP remains to be discovered. Aims 1. (a) Recruit phenotyped cohorts of control & CRSsNP participants. (b) Characterise recruited CRSsNP participants’ tissue samples and isolated epithelial & fibroblast cells. 2. Assay the sinonasal environment to determine any association between, infection, inflammation and remodelling. 3. Identify clusters of genes differentially expressed in CRSsNP & control participants. Methods Cohorts of healthy control and CRSsNP participants were recruited. Matched tissue biopsy, epithelial and fibroblast cells were harvested together with clinical, radiological, microbiological and mucosal swab data. Tissue and cellular samples were characterised to confirm their identity and disease status. The sinonasal environment was characterised from mucosal swabs and analysed for a range of 40 human disease biomarkers. Transcriptome analysis was performed using microarrays and RNA sequencing with downstream bioinformatics investigation of the data. Results 47 age and sex matched CRSsNP and control participants were recruited, differing significantly in symptom and radiological scores. Histological analysis of tissue biopsy specimens was consistent with CRSsNP and control samples. Matched epithelial and fibroblast cells were generated. Assay of the sinonasal microenvironment identified 13 discriminant mediators separating CRSsNP samples from controls using a novel, non-invasive technique. Transcriptomics identified 239 differentially expressed genes in CRSsNP tissue biopsy samples. Cellular samples differed significantly from their matched tissue biopsies. Conclusions This thesis characterises a cohort of tightly defined CRSsNP patients and healthy controls to investigate the potential role of epithelial and fibroblast cells in CRSsNP. Transcriptomics has demonstrated clusters of genes upregulated in CRSsNP, however changes were not consistent in matched cellular samples questioning the validity of cellular models in CRSsNP. Additionally, a straightforward, non-invasive measure of the CRSsNP cytokine profile has been demonstrated. The mediators identified in these assays could potentially be developed as biomarkers of sinonasal inflammation as an adjunct in patient management.
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Migliavacca, Raphaella de Oliveira. "Modelo experimental de rinossinusite crônica em coelhos sem utilização de bactérias : comparação de técnicas de indução." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2012. http://hdl.handle.net/10183/61886.

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Os modelos experimentais têm um papel importante no conhecimento dos mecanismos envolvidos na patogênese da rinossinusite crônica (RSC). Objetivos: comprovar que sem inoculação de bactérias seria possível induzir alterações histológicas crônicas nos seios maxilares de coelhos através da obstrução do óstio de drenagem dos mesmos, produzindo um modelo experimental consistente e reproduzível para RSC. Secundariamente, comparar achados inflamatórios entre duas técnicas de oclusão do óstio do seio maxilar com N-butil cianocrilato: via transmaxilar (VTM) e via teto de fossa nasal (VTFN). Métodos: estudo experimental randomizado cego em animais de laboratório realizado na Unidade de Experimentação Animal do Centro de Pesquisa do Hospital de Clínicas de Porto Alegre, no qual foram sorteados dezesseis coelhos Nova Zelândia entre oclusão do seio maxilar direito VTM ou VTFN. Após 12 semanas de seguimento, os animaisforam anestesiados e sacrificados para análise histopatológica cegada da mucosa do seio maxilar. Resultados: apresentavam alterações histopatológicas compatíveis com RSC os oito (100%) seios maxilares intervindos através da técnica VTM e três (37,5%) através da técnica VTFN, com p 0,008 e 0,250, respectivamente, quando comparados lado direito com o lado controle. Comparando-se as duas técnicas de oclusão, a técnica VTM mostrou-se mais consistente em provocar alterações crônicas nas mucosas dos seios maxilares ocluídos (p 0,026). Conclusões: O modelo do presente trabalho obteve sucesso em provocar alterações histológicas compatíveis com RSC nos animais submetidos à técnica de oclusão VTM com seguimento de 12 semanas, podendo ser facilmente replicável para futuros estudos celulares na mucosa sinusal.
Experimental models have an important role in understanding the mechanisms involved in the pathogenesis of chronic rhinosinusitis (CRS). Objectives: To demonstrate that, without the inoculation of pathogenic bacteria, it is possible to induce chronic histological changes in the maxillary sinuses of rabbits secondary to sinus ostium obstruction, producing a consistent and reproducible experimental model for CRS. Secondly, to compare inflammatory findings between two techniques of experimental occlusion of the maxillary sinus ostium with N-butyl cyanoacrylate: transmaxillary and through the roof of the nasal cavity. Methods: In a randomized, blinded, experimental study, 16 New Zealand rabbits were assigned for occlusion of the right maxillary sinus through a transmaxillary approach or through the roof of the nasal cavity. The contralateral sinus was left undisturbed to serve as a control. After 12 weeks of follow-up, the animals were anesthetized and sacrificed for blinded histopathological analysis of the maxillary sinus mucosa. Results: Histopathological changes consistent with CRS were found in eight (100%) of the maxillary sinuses approached transmaxillary and three of thoseapproached through the roof of the nasal cavity (37.5%), p 0.008 and 0.250, respectively, comparing the right to the left control sinus. Comparing the occlusion techniques, the transmaxillary approach was more consistent in causing chronic mucosal changes (p 0.026). Conclusions: The proposed model was successful in causing histological changes compatible with CRS in animals subjected to sinus occlusion with a transmaxillary approach followed-up for 12 weeks. This experimental model can be easily replicated for future cellular studies of the sinus mucosa.
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Kuchai, Romana. "The effect of macrolides on allergic rhinitis versus chronic rhinosinusitis- an in-vitro study." Thesis, Queen Mary, University of London, 2009. http://qmro.qmul.ac.uk/xmlui/handle/123456789/568.

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Background The mechanisms of the rhinitic process are complex. Previous studies upon nasal epithelial cells have begun to investigate rhinitis. HNECs from turbinate explant tissue were taken from three patient groups (Normals, Chronic Rhinosinusitics and Rhinitics). Aims The study, firstly, aims to establish fundamental differences in cytokine activity between allergic rhinitis and chronic rhinosinusitis by analysing baseline levels of cytokines IL-6 and IL-8 and subsequent impact of bacterial endotoxin. Secondly the study analyses the affect of macrolides on activity in each subgroup. Methods HNECs were grown from the biopsy specimens as explant culture. Standardised exposures to LPS bacterial endotoxin and macrolide were carried out. The concentration of each mediator present in the medium at the end of incubation was assessed by ELISA). A final quantity of total cellular protein was obtained. 3 Results Baseline levels of IL-6 in unstimulated Allergic Rhinitics are significantly higher than in Normal patients. Baseline levels of IL-8, however, are lowest in Allergics. LPS significantly stimulates Allergics to increase production of both IL-6 and IL-8. Macrolides lower IL-6 and IL-8 in both stimulated and unstimulated AR cells. Baseline levels of IL-6 and IL-8 are higher in CRS than AR and Normals. LPS significantly raises IL-6 and IL-8 in CRS. Macrolides increase IL-6 and IL-8 in stimulated CRS cells however reduce levels of both in un-stimulated cells. Discussion Pre-existing neutrophilic and eosinophilic activity in CRS subjects may explain the increased baseline levels of both cytokines upon macrolide exposure. Whilst some studies have suggested macrolides act as antimicrobial, others have suggested that it is their anti-inflammatory effects that are more relevant. Treatment for Allergic Rhinitis needs to be effective long-term. The results here are novel and encourage further research to improve understanding of the effects of macrolides in a potentially pivotal role.
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Pilavakis, Yiannis [Verfasser]. "Occurence and characteristics of allergic rhinitis in 195 patients with chronic rhinosinusitis / Yiannis Pilavakis." Göttingen : Niedersächsische Staats- und Universitätsbibliothek Göttingen, 2020. http://d-nb.info/1223171612/34.

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Cordeiro, Daniel Loiola. "Doença respiratória exacerbada por aspirina: papel da periostina em pacientes com rinossinusite crônica e polipose nasossinusal." Universidade de São Paulo, 2018. http://www.teses.usp.br/teses/disponiveis/17/17138/tde-18072018-154146/.

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Doença respiratória exacerbada por aspirina, conhecida como AERD (Aspirin exacerbated respiratory disease) é caracterizada por rinossinusite crônica eosinofílica, polipose nasossinusal, asma e hipersensibilidade a aspirina e outros anti-inflamatórios não-esteroidais. Expressão aumentada do biomarcador periostina foi descrita em pacientes com AERD, em tecido nasossinusal, incluindo membrana basal, matriz extracelular e pólipo nasal. Avaliamos níveis de periostina sérica em pacientes com AERD e comparamos com níveis em pacientes com rinite alérgica perene (RAP) e indivíduos saudáveis. Foram selecionados 29 pacientes (20F/9M) com diagnóstico de AERD, dentre aqueles atendidos nos Ambulatórios de Alergia e de Otorrinolaringologia do Hospital das Clínicas da Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo (HCFMRP-USP). Estes pacientes realizaram exames confirmatórios, incluindo teste de provocação oral com aspirina, e foram submetidos a biópsia de pólipos nasais por nasofibroscopia. Como controles, foram selecionados 12 pacientes com RAP (9F/3M) e 23 indivíduos saudáveis (14F/9M). Eosinófilos foram quantificados em sangue periférico e em tecido de pólipo ou mucosa nasal. IgE total foi determinada por ImmunoCAP, e periostina sérica foi medida por ELISA. Número de eosinófilos teciduais por campo de grande aumento (CGA), número de eosinófilos por milímetro cúbico em sangue periférico, níveis de IgE total e de periostina sérica em pacientes com AERD foram comparados aos de pacientes com RAP e indivíduos saudáveis. Pacientes com AERD tinham idade maior (mediana 54 anos, faixa 22-60) que pacientes com RAP (mediana 30 anos, faixa 19-57, p=0,0001) e indivíduos saudáveis (mediana 29 anos, faixa 19-53, p=0,0001), sem diferença entre os sexos. Números de eosinófilos em sangue periférico e em tecido foram mais elevados em pacientes com AERD que em pacientes com RAP e indivíduos saudáveis. A mediana do número de eosinófilos em sangue periférico foi 640eos/µL (faixa 100-5.100); 200eos/µL (faixa 100-500); e 100 eos/µL (faixa 100-400) em pacientes com AERD, RAP e indivíduos saudáveis, respectivamente (AERD versus RAP, p=0,0003; AERD versus indivíduos saudáveis, p=0,01). A média do número de eosinófilos teciduais foi de 113,3células/CGA; 2,5células/CGA; e 0,7células/CGA, respectivamente (AERD versus RAP, p=0,017; AERD versus indivíduos saudáveis p=0,003). A média geométrica da IgE total foi de 290,18kU/L (faixa 59,5-8.140); 69,96kU/mL (faixa 5,5-898); e 43,14kU/mL (faixa 4- 1.328) em pacientes com AERD, RAP e indivíduos saudáveis, respectivamente, sem diferença entre os grupos. Periostina sérica foi mais elevada em pacientes com AERD quando comparados a indivíduos saudáveis. A mediana de periostina sérica foi de 602ng/ml (faixa 290,7-1.055); 535,6ng/mL (faixa 209-733,2); e 496,7mg/mL (faixa 327,4-713,4), em pacientes com AERD, RAP e indivíduos saudáveis, respectivamente (AERD versus indivíduos saudáveis, p=0,01). Em subgrupo de pacientes brasileiros com AERD, observamos elevado número de eosinófilos em sangue periférico e em tecido, quando comparados a pacientes com RAP e indivíduos saudáveis. Níveis mais elevados de periostina sérica foram observados em pacientes com AERD, quando comparados a indivíduos saudáveis, indicando forte resposta do tipo 2 em pacientes com AERD em nosso meio
Aspirin exacerbated respiratory disease (also known as AERD), is characterized by eosinophilic chronic hypertrophic rhinosinusitis, nasosinusal polyps, asthma and hypersensitivity to Aspirin or other non-steroidal anti-inflammatory drugs. A higher expression of the biomarker periostin has been described in patients with AERD, in nasosinusal tissue, including basal membrane, extracellular matrix and nasal polyps. We evaluated the levels of serum periostin in patients with AERD, and compare those levels with patients with perennial allergic rhinitis (PAR), and with healthy subjects. Twenty-nine patients (20F/9M) with AERD were selected from the Allergy and Otolaryngology Clinics, from the Clinical Hospital of the Ribeirão Preto Medicine School, University of São Paulo (HCFMRP-USP). Those patients underwent confirmatory exams, such as Oral Provocation test with aspirin, and were submitted to polyp biopsy through nasofibroscopy. As a control group, 12 patients (9F/3M) with PAR and 23 healthy subjects (14F/9M) were selected. Eosinophils were quantified in peripheral blood and in polyp tissue or nasal mucosa. Total IgE was determined by ImmunoCAP, and serum periostin was measured by ELISA. The number of tissue eosinophils by high magnification field (HMF), number of eosinophils by cubic milliliter in peripheral blood, total IgE levels and serum periostin levels in patients with AERD were compared with those from patients with PAR and healthy subjects. Patients with AERD were older (median 54 years, and range 22-60) than patients with PAR (median 30 years, range 19-57, p=0,0001) and healthy subjects (median 29 years, range 19-53, p=0,0001), with no difference between genders. The numbers of eosinophils in peripheral blood and in tissue were higher in patients with AERD than patients with PAR or healthy subjects. The median of eosinophil number in peripheral blood was 640eos/µL (range 100-5.100); 200eos/µL (range 100-500); e 100eos/µL (range 100-400) in patients with AERD, PAR and healthy subjects respectively (AERD vs PAR, p=0,0003; AERD vs healthy subjects, p=0,01). The average number of tissue eosinophils was 113,3cels/HMF; 2,5cels/HMF; e 0,7cels/HMF, respectively (AERD vs PAR, p=0,017; AERD vs healthy subjects, p=0,003). The geometric mean for total IgE was 290,18kU/mL (range 59,5-8.140); 69,96kU/mL (range 5,5-898); and 43,14kU/mL (range 4-1.328) in patients with AERD, PAR and healthy subjects respectively, with no difference between the groups. Serum periostin was higher in patients with AERD when compared with healthy subjects. The median for serum periostin was 602ng/ml (range 290,7-1.055); 535,6ng/mL (range 209-733,2); e 496,7ng/mL (range 327,4-713,4), in patients with AERD, PAR and healthy subjects respectively (AERD vs healthy subjects, p=0,01). In a Brazilian subgroup of patients with AERD, we observed an elevated number of eosinophils in peripheral blood and tissue, when compared with patients with PAR and healthy subjects. Higher levels of serum periostin were observed in patients with AERD, when compared with healthy subjects, indicating a strong type 2 response in patients with AERD in our environment.
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Books on the topic "Chronic rhinosinusitus"

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Zhang, Luo, and Claus Bachert, eds. Chronic Rhinosinusitis. Singapore: Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-0784-4.

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Batra, Pete S., and Joseph K. Han, eds. Practical Medical and Surgical Management of Chronic Rhinosinusitis. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-16724-4.

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Chronic rhinosinusitis. New York: Informa Healthcare, 2007.

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Hamilos, Daniel L., and Fuad M. Baroody, eds. Chronic Rhinosinusitis. CRC Press, 2007. http://dx.doi.org/10.3109/9781420014020.

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Chronic rhinosinusitis. Philadelphia, Pa: Saunders, 2004.

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Sood, V. P. Chronic Rhinosinusitis. Elsevier - Health Sciences Division, 2010.

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Dilmuratovna, Alimova Durdona. Chronic Rhinosinusitis in Children. Primedia eLaunch LLC, 2022.

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Bachert, Claus, and Luo Zhang. Chronic Rhinosinusitis: The Mucosal Concept. Springer Singapore Pte. Limited, 2021.

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Chronic Rhinosinusitis and Concomitant Medical Disorders. MDPI, 2020. http://dx.doi.org/10.3390/books978-3-03928-812-0.

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Baroody, Fuad M., and Daniel L. Hamilos. Chronic Rhinosinusitis: Pathogenesis and Medical Management. Taylor & Francis Group, 2007.

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Book chapters on the topic "Chronic rhinosinusitus"

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Kato, Atsushi, and Robert P. Schleimer. "T Cells and Group 2 Innate Lymphoid Cells 2." In Chronic Rhinosinusitis, 37–46. Singapore: Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-0784-4_6.

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Wang, Xiangdong, Ming Zheng, and Luo Zhang. "Epidemiology." In Chronic Rhinosinusitis, 3–8. Singapore: Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-0784-4_2.

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Halderman, Ashleigh, and Bradley F. Marple. "Surgical Procedures." In Chronic Rhinosinusitis, 371–81. Singapore: Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-0784-4_47.

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Sha, Jichao, and Dongdong Zhu. "Wegener’s Granulomatosis." In Chronic Rhinosinusitis, 287–89. Singapore: Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-0784-4_32.

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Bedoya, David, Cristóbal Langdon, Isam Alobid, José Antonio Castillo, and Joaquim Mullol. "Multimorbidities." In Chronic Rhinosinusitis, 187–99. Singapore: Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-0784-4_22.

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Zhu, Luping, and Lei Cheng. "Probiotics, Bacterial Lysates, and Proton Pump Inhibitors." In Chronic Rhinosinusitis, 361–68. Singapore: Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-0784-4_45.

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Gevaert, Elien. "Neutrophils." In Chronic Rhinosinusitis, 69–79. Singapore: Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-0784-4_9.

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Bachert, Claus, and Luo Zhang. "Introduction." In Chronic Rhinosinusitis, 1–2. Singapore: Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-0784-4_1.

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Bachert, Claus, and Nan Zhang. "Integrated Care Pathways." In Chronic Rhinosinusitis, 423–36. Singapore: Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-0784-4_53.

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Piao, Yingshi. "IgG4-Related Disorders." In Chronic Rhinosinusitis, 291–98. Singapore: Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-0784-4_33.

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Conference papers on the topic "Chronic rhinosinusitus"

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Send, T., KWG Eichhorn, M. Bertlich, P. Korsten, F. Bootz, and M. Jakob. "A rare cause of chronic rhinosinusitis." In Abstract- und Posterband – 89. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Forschung heute – Zukunft morgen. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1639956.

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Ferreira, Diogenes S., Sonia Kaushik, Shyamali Dharmage, Bruce Thompson, Geza Benke, and Michael J. Abramson. "Rhinitis and chronic rhinosinusitis in Melbourne, Australia." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa1141.

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Mulligan, Jennifer, Sarah Casey, Ryan Mulligan, Nick Reaves, Tucker Williamson, Gary Gilkeson, Rodney Schlosser, and Carl Atkinson. "Cigarette Smoke Exacerbates Inflammation Associated With Chronic Rhinosinusitis." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a4190.

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Bogaert, S., N. Suchonos, K. van Ackeren, S. Dazert, J. Park, and C. Bachert. "Mapping the inflammation in chronic rhinosinusitis with nasal polyps." In Abstract- und Posterband – 91. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Welche Qualität macht den Unterschied. © Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1711356.

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Mummoorthy, R., P. Thandi, P. Bhadoria, and D. P. Bhadoria. "Rhinosinusitis in Indian Patients of Chronic Obstructive Pulmonary Disease." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a4743.

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Kim, D. Y. "Distinct Microbial Composition of Nasal Polyp in Chronic Rhinosinusitis." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a2200.

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Pilavakis, Y., F. Ihler, M. Canis, C. Welz, and D. Beutner. "What is the role of allergy in chronic rhinosinusitis?" In Abstract- und Posterband – 89. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Forschung heute – Zukunft morgen. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1639802.

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Bogaert, S., N. Suchonos, K. van Ackeren, S. Dazert, J. Park, and C. Bachert. "Mapping der Entzündung bei chronisch polypöser Rhinosinusitis." In Abstract- und Posterband – 91. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Welche Qualität macht den Unterschied. © Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1711952.

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Kattar, Nrusheel, Basit Jawad, Muhib Haidari, and Ryan Winters. "Chronic Invasive Fungal Rhinosinusitis: A Systematic Review and Meta-analysis." In Special Virtual Symposium of the North American Skull Base Society. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1725473.

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Park, J., D. Seidel, S. Bogaert, K. van Ackeren, S. Dazert, C. Bachert, and K. Kostev. "Use of medication in patients with chronic rhinosinusitis in Germany." In Abstract- und Posterband – 90. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Digitalisierung in der HNO-Heilkunde. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1686740.

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Reports on the topic "Chronic rhinosinusitus"

1

Rabago, David. Nasal Irrigation for Chronic Rhinosinusitis and Fatigue in Patients with Gulf War Syndrome. Fort Belvoir, VA: Defense Technical Information Center, July 2012. http://dx.doi.org/10.21236/ada568066.

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Rabago, David. Nasal Irrigation for Chronic Rhinosinusitis and Fatigue in Patients with Gulf War Syndrome. Fort Belvoir, VA: Defense Technical Information Center, July 2013. http://dx.doi.org/10.21236/ada592220.

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Zheng, Peng-ju. Nasal nebulization inhalation of budesonide for chronic rhinosinusitis with nasal polyps: A protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review Protocols, April 2020. http://dx.doi.org/10.37766/inplasy2020.4.0108.

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