Relevant bibliographies by topics / Chronic Obstructive Pulmonary Disease

Academic literature on the topic 'Chronic Obstructive Pulmonary Disease'

Author: Grafiati
Published: 4 June 2021
Last updated: 29 July 2024

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Journal articles on the topic "Chronic Obstructive Pulmonary Disease"

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Herald, G. Peter Praveen, and H. Krishna Murthy. "EVALUATION OF PULMONARY HYPERTENSION IN CHRONIC OBSTRUCTIVE PULMONARY DISEASE." International Journal of Integrative Medical Sciences 6, no. 1 (February 20, 2019): 765–68. http://dx.doi.org/10.16965/ijims.2019.102.

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FAIVRE, M., and S. LACAN. "Bronchopneumopathie chronique obstructive." EXERCER 35, no. 199 (January 1, 2024): 38–40. http://dx.doi.org/10.56746/exercer.2024.199.38.

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Les recommandations internationales concernant la prise en charge de la bronchopneumopathie chronique obstructive (BPCO) ont été mises à jour par la Global Initiative for Chronic Obstructive Lung Disease (GOLD) en 20231. Cet article revient sur les modifications apportées par rapport à celles de 2022.
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Uryasev, O. M., S. V. Faletrova, and L. V. Korshunova. "Combination of asthma and chronic obstructive pulmonary disease: features of etiology, pathogenesis, diagnosis, pharmacotherapy." Kazan medical journal 97, no. 3 (June 15, 2016): 394–400. http://dx.doi.org/10.17750/kmj2016-394.

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Bronchial asthma and chronic obstructive pulmonary disease are the most common obstructive diseases of the respiratory system. 230 million people suffer from chronic obstructive pulmonary disease, from bronchial asthma - 300 million people worldwide. Annually 200-300 people in Europe and 2.74 million of world population die from chronic obstructive pulmonary disease, from asthma - 250 thousand people a year. The social and economic significance of these diseases determine the need for in-depth study of their combination in the same patient. Each disease has its own phenotypes, but in 10-20% of patients, there are symptoms of both chronic obstructive pulmonary disease and asthma. In spite of clear diagnostic criteria, in some cases it is difficult to distinguish these diseases. Morphological basis of these diseases is a chronic inflammation in the bronchial tree that causes damage to the epithelial continuity that initiates bronchoconstrictive reaction and leads to irreversible airway obstruction attributable for both severe bronchial obstruction and chronic obstructive pulmonary disease. However, the treatment strategy of bronchial asthma and chronic obstructive pulmonary disease has significant differences, it is important to have a clear diagnostic criteria to distinguish different phenotypes, including those of combined phenotype of asthma and chronic obstructive pulmonary disease. Rational starting therapy of asthma and chronic obstructive pulmonary disease overlap syndrome includes drugs acting on the pathogenic mechanisms of both diseases, and is a combination of inhaled corticosteroids with combined bronchodilator therapy - long-acting β2-agonists and long-acting anticholinergics.
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Ramraje, N. N. "Study of OSA in Chronic Obstructive Pulmonary Disease." Journal of Medical Science And clinical Research 05, no. 05 (May 12, 2017): 21712–14. http://dx.doi.org/10.18535/jmscr/v5i5.78.

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Ish, Pranav. "Obstructive Sleep Apnea and Chronic Obstructive Pulmonary Disease Overlap Syndrome - Double Trouble." Journal of Advanced Research in Medicine 05, no. 04 (December 13, 2018): 25–30. http://dx.doi.org/10.24321/2349.7181.201821.

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Mycroft, Katarzyna, and Katarzyna Górska. "Diagnosis and management of COPD in primary care." Medycyna Faktów 14, no. 4 (December 31, 2021): 350–55. http://dx.doi.org/10.24292/01.mf.0421.3.

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Chronic obstructive pulmonary disease is a significant health problem. However, a large proportion of cases remain undiagnosed. Early diagnosis of chronic obstructive pulmonary disease leads to earlier treatment initiation, and in consequence, to improvement of patients quality of life. The gold standard for chronic obstructive pulmonary disease diagnosis is spirometry and the presence of irreversible obstruction after a bronchodilator. One of the most important interventions in the treatment of chronic obstructive pulmonary disease is anti-smoking education. The main group of drugs used in chronic obstructive pulmonary disease treatment are the long-acting bronchodilators.
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Zakaria, Mohamed W., and Heba A. Moussa. "Chronic obstructive pulmonary disease in treated pulmonary tuberculous patients." Egyptian Journal of Bronchology 9, no. 1 (March 20, 2015): 10–13. http://dx.doi.org/10.4103/1687-8426.153519.

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Abstract Background/Aim To detect the prevalence of chronic obstructive pulmonary disease (COPD) as a sequel of treated pulmonary tuberculosis (PTB). Materials and methods A total of 50 adults, 28 men and 22 women, with a definite diagnosis of PTB and complete antituberculous therapy, with subsequent presentation of exertional dyspnea and/or cough, and expectorations for which no other alternative cause was found, were included in our study. All the patients underwent full history taking, full clinical examination, chest radiography, erythrocyte sedimentation rate, prebronchodilator and postbronchodilator forced vital capacity (FVC%), and forced expiratory volume (FEV1%) in the first second of FEV1/FVC%. Results Pulmonary function testing showed 22 patients (44%) with irreversible obstructive pattern denoting chronic obstructive pulmonary disease (COPD), seven patients had restrictive ventilatory defect, and three patients had mixed obstructive and restrictive pattern. Of those 22 patients with irreversible obstructive pattern (COPD), 11 patients (50%) had mild obstruction, nine patients (40.9%) had moderate obstruction, and two patients (9.1%) had severe obstruction. There is a positive correlation between dyspnea and post-tuberculous COPD patients, and a negative correlation between cough and post-tuberculous COPD patients. There is no correlation between the duration since the completion of antituberculous therapy and development of COPD. Conclusion COPD can be a sequel of PTB and should be overlooked, especially in those patients complaining of dyspnea even in the absence of any history of smoking. Post-tuberculous COPD as a cause of COPD in nonsmokers should be now more recognized in countries where the prevalence of PTB is still high.
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Kotolová, Hana, and Tomáš Hammer. "Chronic obstructive pulmonary disease." Praktické lékárenství 14, no. 4 (December 15, 2018): 156–60. http://dx.doi.org/10.36290/lek.2018.030.

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Schneider, Sandra M. "Chronic Obstructive Pulmonary Disease." Emergency Medicine Clinics of North America 7, no. 2 (May 1989): 237–54. http://dx.doi.org/10.1016/s0733-8627(20)30335-7.

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MacIntyre, Neil R. "Chronic Obstructive Pulmonary Disease." Pharmacotherapy 24, no. 5 Part 2 (May 2004): 33S—43S. http://dx.doi.org/10.1592/phco.24.7.33s.34756.

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Dissertations / Theses on the topic "Chronic Obstructive Pulmonary Disease"

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McAllister, David Anthony. "Chronic obstructive pulmonary disease, pulmonary function and cardiovascular disease." Thesis, University of Edinburgh, 2011. http://hdl.handle.net/1842/5615.

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Cardiovascular disease is common in Chronic Obstructive Pulmonary Disease (COPD), and forced expiratory volume in one second (FEV1) independently predicts cardiovascular morbidity and mortality. Pathological changes in the systemic vasculature have been proposed as potential mechanisms linking COPD to cardiovascular disease, and patients with COPD may be at increased risk of acute myocardial infarction during acute exacerbations. Notwithstanding causation, FEV1 may be a useful prognostic marker in patients undergoing cardiac surgery. This thesis examined these three aspects of cardiovascular co-morbidity in relation to COPD and FEV1. In 2,241 consecutive cardiac surgery patients, FEV1 was associated with length of hospital stay (p<0.001) and mortality (p<0.001) adjusting for age, sex, height, body mass index, socioeconomic status, smoking, cardiovascular risk factors, chronic pulmonary disease, and type/urgency of surgery. In a survey of Scottish Respiratory Consultants there was no consensus regarding the investigation and management of acute coronary syndrome in exacerbation of COPD. In a case-series of 242 patients with exacerbations 2.5% (95% CI 1.0 to 5.6%) had chest pain, raised serum troponin and serial electrocardiogram changes suggestive of acute coronary syndrome. However, over half reported chest pain, while raised troponin was not associated with chest pain or serial ECG changes. Carotid-radial pulse wave velocity (PWV), aortic distensibility, and aortic calcification were measured to assess the relationship of the systemic vasculature to FEV1 and emphysema severity on CT. In adjusted analyses, emphysema was associated with PWV in patients with COPD (p = 0.006) and, in population based samples, with extent of distal aortic calcification (p=0.02) but not with aortic distensibility (p=0.60). This thesis found that FEV1 was associated with mortality and length of hospital stay in patients undergoing cardiac surgery, and that chest pain and raised troponin were common but unrelated in exacerbation of COPD. In the vascular studies distal but not proximal vascular pathology was associated with FEV1, and if COPD is truly related to systemic arterial disease, the distal arterial tree is implicated.
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Murphy, Nicola. "Chronic obstructive pulmonary disease and anxiety." Thesis, Coventry University, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.368862.

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Roos-Engstrand, Ester. "T cells in chronic obstructive pulmonary disease." Doctoral thesis, Umeå : Umeå university, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-33677.

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Stevenson, Nicola Jane. "Lung mechanics in chronic obstructive pulmonary disease." Thesis, University of Liverpool, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.432977.

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Al-shair, Khaled. "Systemic Manifestations of Chronic Obstructive Pulmonary Disease." Thesis, University of Manchester, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.509061.

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Revill, Susan M. "Endurance exercise in chronic obstructive pulmonary disease." Thesis, Loughborough University, 1997. https://dspace.lboro.ac.uk/2134/15388.

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Polkey, Michael Iain. "Diaphragm function in chronic obstructive pulmonary disease." Thesis, King's College London (University of London), 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.286262.

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Donaldson, Anna. "Circulating microRNA in Chronic Obstructive Pulmonary Disease." Thesis, Imperial College London, 2014. http://hdl.handle.net/10044/1/24776.

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Skeletal muscle dysfunction in COPD is associated with increased morbidity. Novel agents might reverse skeletal muscle dysfunction through different mechanisms since the biopsy picture in COPD patients is heterogenous. Thus there is a need for a biomarker of skeletal muscle dysfunction which could both be measured in blood and obviate the need for a biopsy. Previous work has found that muscle-specific microRNA (miR-1, miR-499, miR-206 and miR-133) are down-regulated in the quadriceps of COPD patients. Tissue specific miRNA circulate in the blood at detectable levels and are currently under investigation as biomarkers of other diseases. I therefore hypothesised that muscle-specific microRNA might be clinically viable biomarkers of skeletal muscle dysfunction. The studies in this thesis found that: 1. Levels of circulating muscle-specific microRNA (miR-1, miR-133, miR-206 and miR-499) were increased in stable COPD patients. The increase in muscle-specific microRNA in the stable COPD patients suggests that continual muscle turnover occurs outside of times of disease exacerbation. 2. Plasma levels of muscle-specific miRNA were not different in COPD patients admitted to hospital for an exacerbation of their disease compared to stable COPD patients. 3. Most muscle-specific microRNA did not change in muscle with acute exercise. I demonstrated an increase in miR-181 (an miRNA not restricted to, but with known function in muscle) one hour after acute exercise in the quadriceps muscle of COPD patients. However when fold change was calculated for the COPD patients and controls, there was no statistical difference found. There were no detectable miR-181 changes measured in blood. 4. Finally, I used a microarray approach to investigate other circulating microRNA that might to be useful to separate patients based on their lean muscle-mass. COPD patients with a reduced skeletal muscle mass had a reduced number of microRNA associated with growth and cell pluripotency, further studies are required to validate these findings. Taken together, the results from this thesis suggest that the microRNA analysed were detectable in blood, but they could not be usefully used (based on current analysis) as biomarkers of quadriceps dysfunction.
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Taylor, Abigail Elizabeth. "Macrophage phagocytosis in chronic obstructive pulmonary disease." Thesis, Imperial College London, 2009. http://hdl.handle.net/10044/1/7374.

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The hypothesis examined in this thesis stated that macrophages from COPD subjects are defective in their ability to phagocytose. To investigate this hypothesis, engulfment by COPD macrophages was compared to that of cells from controls (smokers without COPD and non-smokers). Phagocytosis of polystyrene beads by monocytes-derived-macrophages (MDM) was comparable to that of alveolar macrophages, thus validating the MDM model. There was no difference in uptake of beads by any subject group, however, COPD MDM phagocytosed significantly less E. coli, H. influenzae and S. pneumoniae than cells from controls. This was not due to current medications as commonly prescribed therapies did not reduce phagocytosis of these bacteria. To identify the mechanism of this defeat, cytoskeletal arrangement was investigated. Actin polymerisation was not altered by disease, but COPD MDM had a greater susceptibility to microtubule disruptors and reduced levels of acetylated tubulin compared to control cells. Increasing microtubule acetylation enhanced uptake of H. influenzae, suggesting that this may be a novel mechanism for improving phagocytosis. Other mechanisms investigated included sphingosine-1-phosphate (S1P) signalling, as production of this molecule was increased by COPD MDM and inhibition of S1P signalling cascade increased uptake of H. influenzae. Microarray analysis demonstrated that following exposure to H. influenzae, COPD MDM expressed significantly more inflammatory genes than control cells, suggesting that these macrophages respond differently to bacteria. These findings suggest that COPD macrophages have a reduced phagocytic ability, which may result in bacterial colonisation, inflammation and exacerbations. Improving this macrophage function would offer therapeutic potential in this disease.
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Baldrick, Francina Rose. "Diet and chronic obstructive pulmonary disease (COPD)." Thesis, Queen's University Belfast, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.527657.

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Books on the topic "Chronic Obstructive Pulmonary Disease"

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Nakamura, Hiroyuki, and Kazutetsu Aoshiba, eds. Chronic Obstructive Pulmonary Disease. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-0839-9.

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Nici, Linda, and Richard ZuWallack, eds. Chronic Obstructive Pulmonary Disease. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-60761-673-3.

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Stockley, Robert A., Stephen I. Rennard, Klaus Rabe, and Bartolome Celli, eds. Chronic Obstructive Pulmonary Disease. Oxford, UK: Blackwell Publishing Ltd, 2007. http://dx.doi.org/10.1002/9780470755976.

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Calverley, P. M. A., and N. B. Pride, eds. Chronic Obstructive Pulmonary Disease. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4899-4525-9.

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A, Calverley P. M., ed. Chronic obstructive pulmonary disease. 2nd ed. London: Arnold, 2003.

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S, Cherniack Neil, ed. Chronic obstructive pulmonary disease. Philadelphia: Saunders, 1991.

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A, Stockley Robert, ed. Chronic obstructive pulmonary disease. Malden, Mass: Blackwell Pub., 2005.

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National Heart, Lung, and Blood Institute. Division of Lung Diseases and National Heart, Lung, and Blood Institute. Office of Prevention, Education, and Control, eds. Chronic obstructive pulmonary disease. [Bethesda, Md.?]: National Institutes of Health, National Heart, Lung, and Blood Institute, 1993.

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National Heart, Lung, and Blood Institute, ed. Chronic obstructive pulmonary disease. [Bethesda, Md.?]: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1986.

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National Heart, Lung, and Blood Institute., ed. Chronic obstructive pulmonary disease. [Bethesda, Md.?]: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1986.

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Book chapters on the topic "Chronic Obstructive Pulmonary Disease"

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Farver, Carol, Subha Ghosh, Thomas Gildea, and Charles D. Sturgis. "Chronic Obstructive Pulmonary Diseases." In Pulmonary Disease, 145–60. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-47598-7_11.

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Clark, C. J. "Pulmonary Rehabilitation." In Chronic Obstructive Pulmonary Disease, 527–45. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4899-4525-9_21.

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Troosters, Thierry. "Cardiovascular Disease." In Chronic Obstructive Pulmonary Disease, 47–59. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-60761-673-3_4.

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Rodriguez-Roisin, R., and J. Roca. "Pulmonary Gas Exchange." In Chronic Obstructive Pulmonary Disease, 161–84. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4899-4525-9_8.

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Morgan, M. D. L. "Bullous Lung Disease." In Chronic Obstructive Pulmonary Disease, 547–59. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4899-4525-9_22.

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Underwood, David C. "Chronic obstructive pulmonary disease." In In Vivo Models of Inflammation, 159–77. Basel: Birkhäuser Basel, 1999. http://dx.doi.org/10.1007/978-3-0348-7775-6_7.

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Bertholet, Renette, and Inessa McIntyre. "Chronic Obstructive Pulmonary Disease." In Patient Assessment in Clinical Pharmacy, 213–24. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-11775-7_16.

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Stucky, Kirk, and Jeff Greenblatt. "Chronic Obstructive Pulmonary Disease." In Comprehensive Handbook of Clinical Health Psychology, 277–300. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2012. http://dx.doi.org/10.1002/9781118269657.ch12.

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Kozora, Elizabeth, and Karin F. Hoth. "Chronic Obstructive Pulmonary Disease." In Encyclopedia of Clinical Neuropsychology, 782–85. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_545.

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Franco, S., and Marilyn Glassberg. "Chronic Obstructive Pulmonary Disease." In Encyclopedia of Women’s Health, 262–64. Boston, MA: Springer US, 2004. http://dx.doi.org/10.1007/978-0-306-48113-0_88.

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Conference papers on the topic "Chronic Obstructive Pulmonary Disease"

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Sokolovic, Irena, Jelena Knezevic, Sanja Popovic-Grle, Andrea Vukic Dugac, Gordana Bubanovic, Mateja Jankovic Makek, Ana Hecimovic, Gzim Redzepi, Miroslav Samarzija, and Marko Jakopovic. "Chronic obstructive pulmonary disease and comorbidities." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa1123.

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Nadia, Fettal, Beziou Yamina, and Taleb Abdessamed. "Diabetes and chronic obstructive pulmonary disease." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa4319.

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Sellami, Rahma, Sabrine Majdoub Fehri, Imen Ketata, and Hamida Kwass. "Chronic obstructive pulmonary disease in elderly." In ERS International Congress 2023 abstracts. European Respiratory Society, 2023. http://dx.doi.org/10.1183/13993003.congress-2023.pa957.

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Balasubramanian, A., T. M. Kolb, R. L. Damico, P. M. Hassoun, M. C. McCormack, and S. C. Mathai. "Characterization of Chronic Obstructive Pulmonary Disease-Pulmonary Hypertension." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a5961.

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Calancea, Valentin, Eudochia Terna, Tatiana Dumitras, Doina Barba, Irina Cosciug, and Sergiu Matcovschi. "Pulmonary haemodynamic disorders in chronic obstructive pulmonary disease." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa2464.

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Jakimova, Marina Artemovna, Natalya Karpina, Olga Gordeeva, and Rasul Asanov. "Comorbidity: pulmonary tuberculosis and chronic obstructive pulmonary disease." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa2969.

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Gupta, V., D. Singh, S. Pandit, P. Bhadoria, and D. P. Bhadoria. "Prevalence of Asthma Chronic Obstructive Pulmonary Disease Overlap Syndrome in Indian Patients of Chronic Obstructive Pulmonary Disease." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a4746.

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Walid, Feki, Chaabouni Malek, Ketata Wajdi, Moussa Nadia, Bahloul Najla, Msaad Sameh, Kotti Amina, et al. "Chronic pain in patients with chronic obstructive pulmonary disease." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa4012.

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Gill, K., and D. Lee. "More than Just Chronic Obstructive Pulmonary Disease." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6449.

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DeMeo, DL, CP Hersh, B. Klanderman, AA Litonjua, and EK Silverman. "Aging Genes and Chronic Obstructive Pulmonary Disease." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a3000.

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Reports on the topic "Chronic Obstructive Pulmonary Disease"

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Barros-Poblete, Marisol, Rodrigo Torres-Castro, Mauricio Henríquez, Anita Guequen, Isabel Blanco, and Carlos Flores. Dysbiosis as a prognostic factor for clinical worsening in chronic respiratory disease: A systematic review and metanalysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2022. http://dx.doi.org/10.37766/inplasy2022.4.0089.

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Review question / Objective: Is dysbiosis a prognostic factor for clinical worsening in patients with chronic respiratory diseases?. Condition being studied: Dysbiosis, defined as changes in the quantitative and qualitative composition of the microbiota. Eligibility criteria: Over 18 years old adult patients with chronic respiratory diseases clinical diagnosis (cystic fibrosis, chronic obstructive pulmonary disease, asthma, idiopathic pulmonary fibrosis, interstitial lung disease, sarcoidosis, bronchiectasis, non-CF bronchiectasis, pulmonary hypertension) according to the International Statistical Classification of Diseases and Related Health Problems (ICD) from OMS) and international guidelines of each disease.
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Czerwaty, Katarzyna, Karolina Dżaman, Krystyna Maria Sobczyk, and Katarzyna Irmina Sikrorska. The Overlap Syndrome of Obstructive Sleep Apnea and Chronic Obstructive Pulmonary Disease: A Systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2022. http://dx.doi.org/10.37766/inplasy2022.11.0077.

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Review question / Objective: To provide the essential findings in the field of overlap syndrome of chronic obstructive pulmonary disease and obstructive sleep apnea, including prevalence, possible predictors, association with clinical outcomes, and severity compared to both chronic obstructive pulmonary disease and obstructive sleep apnea patients. Condition being studied: OSA is characterized by complete cessation (apnea) or significant decrease (hy-popnea) in airflow during sleep and recurrent episodes of upper airway collapse cause it during sleep leading to nocturnal oxyhemoglobin desaturations and arousals from rest. The recurrent arousals which occur in OSA lead to neurocognitive consequences, daytime sleepiness, and reduced quality of life. Because of apneas and hypopneas, patients are experiencing hypoxemia and hypercapnia, which result in increasing levels of catecholamine, oxidative stress, and low-grade inflammation that lead to the appearance of cardio-metabolic consequences of OSA. COPD is a chronic inflammatory lung disease defined by persistent, usually pro-gressive AFL (airflow limitation). Changes in lung mechanics lead to the main clini-cal manifestations of dyspnea, cough, and chronic expectoration. Furthermore, patients with COPD often suffer from anxiety and depression also, the risk of OSA and insomnia is higher than those hospitalized for other reasons. Although COPD is twice as rare as asthma but is the cause of death eight times more often.
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Liang, R., D. Liu, HB Li, and ZG Zhai. The efficacy and safety of traditional Chinese medicine formulas in the treatment of chronic obstructive pulmonary disease complicated with pulmonary hypertension: a systematic review and meta-analysis study. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, October 2022. http://dx.doi.org/10.37766/inplasy2022.10.0041.

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Review question / Objective: This systematic review and meta-analysis was intended to evaluate the efficacy and safety of traditional Chinese medicine(TCM) formulas in the treatment of chronic obstructive pulmonary disease(COPD) complicated with pulmonaryhypertension (PH). Condition being studied: Chronic obstructive pulmonary disease(COPD) complicated with pulmonary hypertension(PH) is classified as the third group PH.According to epidemiology, the most common cause of PH associated with lung diseases and/or hypoxia is COPD, but the prevalence rate of COPD with PH range from 20% to 91% variously. In China, many TCM formulas are regularly used in COPD patients , thus TCM formulas therapy is worth considering.
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Yang, Hongkuan, Shuifeng He, Linbao Liang, and Junjie Pan. Efficacy of Nemiralisib in chronic obstructive pulmonary disease: A systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, December 2023. http://dx.doi.org/10.37766/inplasy2023.12.0040.

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Wang, Shanshan, Chengsen Cai, Jianjian Yu, Xuejie Si, Jun Wang, and Wenjing Chen. Meta-analysis of Self-Blood Acupoint Injection for Chronic Obstructive Pulmonary Disease (COPD). INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, December 2023. http://dx.doi.org/10.37766/inplasy2023.12.0056.

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6

Liu, Lu, Wenchuan Qi, Qian Zeng, Ziyang Zhou, Daohong Chen, Lei Gao, Bin He, Dingjun Cai, and Ling Zhao. Does acupuncture improve lung function in chronic obstructive pulmonary disease animal model?: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2022. http://dx.doi.org/10.37766/inplasy2022.3.0104.

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Abstract:
Review question / Objective: Chronic obstructive pulmonary disease (COPD) is a common, preventable, and treatable disease characterized by persistent respiratory symptoms and progressive airflow obstruction documented on spirometry. Acupuncture, as a safe and economical non-pharmacology therapy, has pronounced therapeutic effects in COPD patients. Several systematic reviews draw the conclusion that acupuncture could improve patients’ quality of life, exercise capacity and dyspnoea, however, the results about lung function were inconclusive. Recently, increasing number of animal studies has been published to illustrate the effects of acupuncture in improving lung function in COPD animal model. However, the efficacy of acupuncture for experimentally induced COPD have not been systematically investigated yet. A systematic review of animal experiments can benefit future experimental designs, promote the conduct and report of basic researches and provide some guidance to translate the achievements of basic researches to clinical application in acupuncture for COPD. Therefore, we will conduct this systematic review and meta-analysis to evaluate effects of acupuncture on COPD animal model.
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Wang, Yanping, Mingru Huang, Liping Tang, Lingxia Xu, Jiangfeng Wu, Fei Wang, and Ying Zhang. Moxibustion for stable chronic obstructive pulmonary disease: a protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2021. http://dx.doi.org/10.37766/inplasy2021.4.0047.

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Du, Dongru, Guangyue Zhang, Lei Chen, Yongchun Shen, and Fuqiang Wen. Prevalence and characteristics of restless leg syndrome in chronic obstructive pulmonary disease: a meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2023. http://dx.doi.org/10.37766/inplasy2023.3.0057.

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Feng, Zhenzhen, Xuanlin Li, Yang Xie, and Jiansheng Li. Effectiveness of Tai Chi for Chronic obstructive Pulmonary Disease: Overview of Systematic Reviews and Meta-analyses. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, October 2020. http://dx.doi.org/10.37766/inplasy2020.10.0114.

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Zhou, Wen. Does benralizumab effectively treat chronic obstructive pulmonary disease? a protocol of systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review Protocols, April 2020. http://dx.doi.org/10.37766/inplasy2020.4.0039.

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