Relevant bibliographies by topics / Chronic lung disease

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Chronic lung disease'

Author: Grafiati
Published: 4 June 2021
Last updated: 11 March 2023

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Journal articles on the topic "Chronic lung disease"

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Petty, Thomas L. "Chronic lung disease." Postgraduate Medicine 86, no. 6 (November 1989): 113. http://dx.doi.org/10.1080/00325481.1989.11704476.

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Falcão, Mário Cícero. "Neonatal chronic lung disease." Revista do Hospital das Clínicas 54, no. 6 (December 1999): 173–74. http://dx.doi.org/10.1590/s0041-87811999000600001.

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Filho, Luiz Vicente F. da Silva. "Neonatal chronic lung disease." Jornal de Pediatria 74, no. 4 (July 15, 1998): 265–74. http://dx.doi.org/10.2223/jped.433.

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Avent, Minyon, Diana Coile, and Letha Mathai. "Neonatal Chronic Lung Disease." Journal of Pharmacy Practice 14, no. 3 (June 2001): 181–206. http://dx.doi.org/10.1106/j5vj-evx8-19ru-7e0b.

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Chronic lung disease (CLD), formerly known as bronchopulmonary dysplasia, is presently defined as the need for oxygen therapy either at 28 days of age or greater than 36 weeks postmenstrual age. Clinical signs and symptoms include tachypnea, retractions, apnea, and radiographic findings of poorly inflated lungs with reticulogranular opacities. The disease develops as a result of chronic pulmonary inflammation and continuous lung injury induced by oxygen, positive pressure ventilation, and other causes. Fifty to sixty-five percent of neonates with CLD are rehospitalized with respiratory problems, and 21% of very low birth weight neonates are diagnosed with asthma or other respiratory disorders by the age of five. These infants are at risk of adverse neurodevelopmental sequelae as they have a more complicated neonatal course. Many studies have explored various preventive therapies including α1-proteinase inhibitors, superoxide dismutase, antioxidants, and ventilatory management. Although the results from these trials are promising, further studies are needed to define which patients are most likely to benefit from preventive therapy. Two preventive treatment approaches that have shown a decrease in morbidity and an improvement in mortality are antenatal steroids and surfactant therapy. Postnatal corticosteroid therapy continues to be the mainstay of treatment for CLD, however, there are a number of detrimental side effects associated with this treatment. Due to the increased incidence in periventricular leukomalacia, early treatment of steroid therapy cannot be recommended. The optimal time to start steroid therapy appears to be after the first week of life. In addition, the lowest dose and shortest duration of treatment needs to be implemented in order to minimize potential complications. Although bronchodilators and diuretics continue to be used extensively in infants with CLD, there are surprisingly few well-controlled studies that have evaluated the clinical impact of this therapy. Further trials are needed in order to support the routine use of these therapies in CLD. Unfortunately, inhaled steroids have not shown an improvement in long-term outcomes of CLD, however, they have shown a decrease in systemic steroid usage. CLD is a complex disease with many unanswered questions. Further studies are needed to evaluate the effects of various treatment modalities with particular focus on the long-term outcomes such as oxygen and ventilator dependency as well as the incidence of CLD.
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Brauer, Sandra. "Chronic obstructive lung disease." Journal of Physiotherapy 59, no. 4 (December 2013): 278. http://dx.doi.org/10.1016/s1836-9553(13)70212-1.

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Hilman, Bettina C. "Chronic Interstitial Lung Disease." Clinical Pediatrics 37, no. 11 (November 1998): 701–2. http://dx.doi.org/10.1177/000992289803701111.

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Hogg, James C. "A Brief Review of Chronic Obstructive Pulmonary Disease." Canadian Respiratory Journal 19, no. 6 (2012): 381–84. http://dx.doi.org/10.1155/2012/496563.

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A recent study, based on a combination of multidetector computed tomography scanning of an intact specimen with microcomputed tomography and histological analysis of lung tissue samples, reported that the number of terminal bronchioles were reduced from approximately 44,500/lung pair in control (donor) lungs to approximately 4800/lung pair in lungs donated by individuals with very severe (Global initiative for chronic Obstructive Lung Disease stage 4) chronic obstructive pulmonary disease (COPD) treated by lung transplantation. The present short review discusses the hypothesis that a rapid rate of terminal bronchiolar destruction causes the rapid decline in lung function leading to advanced COPD. With respect to why the terminal bronchioles are targeted for destruction, the postulated mechanisms of this destruction and the possibility that new treatments are able to either prevent or reverse the underlying cause of airway obstruction in COPD are addressed.
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Gilpin, Sarah E., and Darcy E. Wagner. "Acellular human lung scaffolds to model lung disease and tissue regeneration." European Respiratory Review 27, no. 148 (June 6, 2018): 180021. http://dx.doi.org/10.1183/16000617.0021-2018.

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Recent advances in whole lung bioengineering have opened new doors for studying lung repair and regeneration ex vivo using acellular human derived lung tissue scaffolds. Methods to decellularise whole human lungs, lobes or resected segments from normal and diseased human lungs have been developed using both perfusion and immersion based techniques. Immersion based techniques allow laboratories without access to intact lobes the ability to generate acellular human lung scaffolds. Acellular human lung scaffolds can be further processed into small segments, thin slices or extracellular matrix extracts, to study cell behaviour such as viability, proliferation, migration and differentiation. Recent studies have offered important proof of concept of generating sufficient primary endothelial and lung epithelial cells to recellularise whole lobes that can be maintained for several days ex vivo in a bioreactor to study regeneration. In parallel, acellular human lung scaffolds have been increasingly used for studying cell–extracellular environment interactions. These studies have helped provide new insights into the role of the matrix and the extracellular environment in chronic human lung diseases such as chronic obstructive pulmonary disease and idiopathic pulmonary fibrosis. Acellular human lung scaffolds are a versatile new tool for studying human lung repair and regeneration ex vivo.
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COOPER, JOEL D. "Lung Transplantation for Chronic Obstructive Lung Disease." Annals of the New York Academy of Sciences 624, no. 1 (May 1991): 209–11. http://dx.doi.org/10.1111/j.1749-6632.1991.tb17019.x.

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Wu, Yifan, Evan Li, Morgan Knight, Grace Adeniyi-Ipadeola, Li-zhen Song, Alan R. Burns, Ana Clara Gazzinelli-Guimaraes, Ricardo Fujiwara, Maria Elena Bottazzi, and Jill E. Weatherhead. "Transient Ascaris suum larval migration induces intractable chronic pulmonary disease and anemia in mice." PLOS Neglected Tropical Diseases 15, no. 12 (December 16, 2021): e0010050. http://dx.doi.org/10.1371/journal.pntd.0010050.

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Ascariasis is one of the most common infections in the world and associated with significant global morbidity. Ascaris larval migration through the host’s lungs is essential for larval development but leads to an exaggerated type-2 host immune response manifesting clinically as acute allergic airway disease. However, whether Ascaris larval migration can subsequently lead to chronic lung diseases remains unknown. Here, we demonstrate that a single episode of Ascaris larval migration through the host lungs induces a chronic pulmonary syndrome of type-2 inflammatory pathology and emphysema accompanied by pulmonary hemorrhage and chronic anemia in a mouse model. Our results reveal that a single episode of Ascaris larval migration through the host lungs leads to permanent lung damage with systemic effects. Remote episodes of ascariasis may drive non-communicable lung diseases such as asthma, chronic obstructive pulmonary disease (COPD), and chronic anemia in parasite endemic regions.
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Dissertations / Theses on the topic "Chronic lung disease"

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Stevenson, Nicola Jane. "Lung mechanics in chronic obstructive pulmonary disease." Thesis, University of Liverpool, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.432977.

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Roberts, Della Kim. "The family experience with chronic obstructive pulmonary disease." Thesis, University of British Columbia, 1985. http://hdl.handle.net/2429/24422.

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This study was designed to gain an understanding of the family experience when an adult member has chronic obstructive pulmonary disease (COPD). It is recognized that illness within the family affects the well-being of the family unit and the health of all members. To understand the impact of COPD upon the family, however, the literature provides only knowledge of the experience of the individual who has COPD and the spouse, not that of the family unit. Thus, the purpose of this study was to describe and explain the COPD experience from the perspective of the family unit. A qualitative method, phenomenology, was chosen for this investigation. Data were collected through semi-structured interviews with eight families who shared their experiences. From the content analysis of these data, three themes that were common throughout the families' accounts were identified and developed to describe and explain family life with COPD. The first theme, disease-dictated family life, describes four aspects of a common lifestyle that is imposed on the family by the characteristics of COPD. The second theme, isolation, describes the isolation that accompanies the illness experience, for the family group and the individual members within the group. The final theme, family work, describes the four primary challenges the families face and the coping strategies they use to deal with them. These findings revealed that COPD acts as an intense stressor within the family, requiring extensive family work to cope with COPD in a way that maintains the well-being of the family unit. Furthermore, it was found that living with COPD in many ways inhibits the resources within the family and those external sources of support that foster the family's ability to manage the stress associated with living with COPD. The implications for nursing practice and nursing research were delineated in light of the research findings.
Applied Science, Faculty of
Nursing, School of
Graduate
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Sze, Marc Alexander. "The lung microbiome in chronic obstructive pulmonary disease." Thesis, University of British Columbia, 2011. http://hdl.handle.net/2429/36343.

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Until recently the normal human lung was thought to be sterile below the larynx, but recent reports from other laboratories indicate that a diverse microbiome exists and becomes less diverse in smokers. These reports led naturally to the hypothesis that pathogens emerging from the abnormal microbiome in smokers could drive the innate and adaptive immune response that has been associated with the pathology of peripheral lung abnormalities observed in Chronic Obstructive Pulmonary Disease (COPD). The purpose of the present study was to examine this hypothesis in human lung tissue. This began with a preliminary experiment in which DNA isolated from 2 samples from a control lung were compared to DNA isolated from 5 different samples of a severe COPD lung, using 75 based pair-end tag sequencing (metagenomic sequencing). For bacteria, a weighted average genome size representing bacterial species identified was applied and the results validated using PCR and qPCR assays. This preliminary experiment was followed by a qPCR, T-RFLP, and targeted sequencing analysis of the bacterial 16S rRNA gene in DNA isolated from single samples of frozen lung tissue obtained from 8 non-smoking and 8 smoking controls, 8 COPD (GOLD 4), and 8 cystic fibrosis patients. The metagenomic sequencing conducted in the preliminary study showed that the 5 samples from a single COPD patient had an average of 2.4 ± 0.7 bacteria/1000 human genomes while the smoking control had 1.6 ± 0.8 bacteria/1000 human genomes. The qPCR results obtained from a single sample from 32 different subjects showed that on average the 8 samples/group of non-smokers, smokers, and COPD (GOLD 4) patients had 34.5 ± 21.8, 44.3 ± 47.0, and 24.1 ± 36.9 bacteria/1000 human cells, respectively, while cystic fibrosis patients had (20 ± 54) x 10 4 bacteria /1000 human cells. T-RFLP analysis showed three distinct community compositions: smokers and non-smokers, cystic fibrosis, and COPD (GOLD 4) patients. These results confirm the presence of a small number of bacteria within the human lung of non-smoker and smoker controls and in COPD patients with a shift in bacterial composition in lungs of those with COPD (GOLD 4).
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Dhillon, Satvir Singh. "Physical activity measurement strategies in advanced chronic lung disease." Thesis, University of British Columbia, 2014. http://hdl.handle.net/2429/50830.

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Background: Physical activity may reduce mortality risk in advanced chronic lung disease by optimizing functional capacity, which is a major prognostic indicator in lung transplantation candidates. There is uncertainty as to the optimal method to measure physical activity in this patient population. We assessed different commercially-available physical activity measurement techniques (flex heart rate monitoring (FHR); pedometry; tri-axial accelerometry; and multi-sensor technology) by investigating their agreement with indirect calorimetry (IC) in adult lung disease patients (chronic obstructive pulmonary disease (COPD), interstitial lung disease (ILD), and cystic fibrosis (CF)) with advanced pulmonary impairment. Methods: This is a cross-sectional method comparison study conducted on two separate days. We recruited consecutive COPD, ILD, and CF patients with physician diagnosis of advanced pulmonary impairment. On day one, participants performed cardiopulmonary exercise testing until exhaustion with measurements of oxygen uptake (VO₂) and heart rate (HR) collected. On day two, subjects had their VO₂ and HR measured during standardized resting and sub-maximal activity. Simultaneous VO₂ and HR measures from both days were used to develop individual regressions for FHR-derived energy expenditure (EE). We then simultaneously measured each subject’s EE using a variety of index measures of physical activity and IC during standardized “free-living” type activities and varying intensities of sub-maximal cycle exercise. Results: In a sample of eight participants (CF, n=5; COPD, n=2; ILD, n=1), Flex HR methods using submaximal (FMSUB) and CPET-derived (FMCPX) calibrations showed the best agreement and interchangeability with IC during free-living and cycling activities compared to the SenseWear (SW) and ActiCal (AC) devices as evidenced by lower mean differences with IC and widths of limit of agreement (LOA) + 95% confidence interval (CI). For the secondary index methods assessed, the Tractivity and DigiWalker devices significantly over and underestimated IC EE respectively (p<0.05), whereas the Dynaport device did not differ from IC (p>0.05) over the entire protocol. Conclusion: Our study found that the Flex HR method for EE estimation had the lowest bias and variability during free-living activities and exercise. EE estimation using Flex HR methods may be potentially useful clinical tools to ensure metabolic energy balance and activity monitoring in advanced lung disease groups.
Medicine, Faculty of
Graduate
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Toma, Tudor Paul. "Endoscopic lung volume reduction in chronic obstructive pulmonary disease." Thesis, Imperial College London, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.428589.

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Sweet, D. G. "Studies on the pathogenesis of neonatal chronic lung disease." Thesis, Queen's University Belfast, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.395367.

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Kotecha, Sailesh. "The role of cytokines in chronic lung disease of prematurity." Thesis, Imperial College London, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.244032.

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Soler, Artigas María. "Genetic epidemiology of lung function and chronic obstructive pulmonary disease." Thesis, University of Leicester, 2015. http://hdl.handle.net/2381/31980.

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Chronic obstructive pulmonary disease (COPD) is a leading cause of death worldwide. Lung function measures obtained through spirometry play a key role in the diagnosis of COPD. Both COPD and lung function are affected by genetic factors, and identifying genetic variants that have an effect on lung function or COPD risk has the potential to lead to improved treatment and prevention of COPD. This thesis is structured in five chapters, an introductory, a concluding chapter and three main chapters which present different approaches that aim to bring insights into the genetics of COPD and lung function. Chapter 2 tests the association with COPD risk of genetic variants previously associated with lung function, and tests their combined effect on lung function and COPD risk, in order to explore the role of risk prediction. Chapter 3 aims to identify new genetic variants associated with lung function and tests the association of genetic variants genome-wide. Chapter 4 focuses on the analysis of low frequency variants using different approaches and methodologies, and includes two studies. One study assesses associations of low frequency variants genome-wide, and the other focuses on genetic regions associated with lung function, in order to improve the localization of association signals that often comprise broad regions and several genes. These studies overall have identified 16 new genetic variants associated with lung function, have shown the association with COPD of 4 genetic variants previously associated with lung function, and present suggestive evidence of association with COPD for low frequency variants within regions associated with lung function.
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Brandén, Eva. "Chronic infection with Chlamydia pneumoniae in COPD and lung cancer." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-344-2/.

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Steele, Bonnie Gail. "Dimensions of dyspnea in chronic obstructive pulmonary disease : a nociceptive model /." Thesis, Connect to this title online; UW restricted, 1991. http://hdl.handle.net/1773/7347.

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Books on the topic "Chronic lung disease"

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1932-, Petty Thomas L., ed. Chronic obstructive pulmonary disease. 2nd ed. New York: Dekker, 1985.

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Rochester, Carolyn L. Chronic obstructive pulmonary disease. Philadelphia: W.B. Saunders Co., 2000.

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F, Voelkel Norbert, and MacNee William, eds. Chronic obstructive lung diseases. Hamilton, Ont: BC Decker, 2002.

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Hodgkin, John E. Chronic obstructive pulmonary disease. Philadelphia, Pa: W.B. Saunders, 1990.

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A, Calverley P. M., ed. Chronic obstructive pulmonary disease. 2nd ed. London: Arnold, 2003.

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Bhatt, Surya P., ed. Cardiac Considerations in Chronic Lung Disease. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-43435-9.

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Chamba, Anita. Neutrophil function in chronic lung disease. Birmingham: University of Birmingham, 1991.

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Romain, Pauwels, Postma D. S, and Weiss Scott T, eds. Long-term intervention in chronic obstructive pulmonary disease. New York: Marcel Dekker, 2004.

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1939-, Hodgkin John E., and Petty Thomas L. 1932-, eds. Chronic obstructive pulmonary disease: Current concepts. Philadelphia: Saunders, 1987.

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Stockley, Robert A. Serum and secretion proteins in chronic lung disease. Birmingham: University of Birmingham, 1986.

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Book chapters on the topic "Chronic lung disease"

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Abbasi, Adeel, Francis DeRoos, José Artur Paiva, J. M. Pereira, Brian G. Harbrecht, Donald P. Levine, Patricia D. Brown, et al. "Chronic Lung Disease." In Encyclopedia of Intensive Care Medicine, 556. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_1349.

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Morgan, M. D. L. "Bullous Lung Disease." In Chronic Obstructive Pulmonary Disease, 547–59. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4899-4525-9_22.

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Corris, P. "Lung Transplantation." In Chronic Obstructive Pulmonary Disease, 561–70. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4899-4525-9_23.

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Pride, N. B., and J. Milic-Emili. "Lung Mechanics." In Chronic Obstructive Pulmonary Disease, 135–60. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4899-4525-9_7.

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Trigaux, J. P., L. Goncette, and P. DeCoster. "Chronic Obstructive Lung Disease." In Radiologic Diagnosis of Chest Disease, 221–39. New York, NY: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4684-0347-3_18.

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Peters, Nils, Martin Dichgans, Sankar Surendran, Josep M. Argilés, Francisco J. López-Soriano, Sílvia Busquets, Klaus Dittmann, et al. "Chronic Obstructive Lung Disease." In Encyclopedia of Molecular Mechanisms of Disease, 358. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_7820.

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Jehan Peerzada, Kaiser. "Chronic Obstructive Pulmonary Disease: An Update on Therapeutics and Pathophysiological Understanding." In Chronic Lung Diseases, 157–80. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-3734-9_8.

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Paré, P. D., and J. C. Hogg. "Lung Structure-Function Relationships." In Chronic Obstructive Pulmonary Disease, 35–45. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4899-4525-9_3.

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Peters, Nils, Martin Dichgans, Sankar Surendran, Josep M. Argilés, Francisco J. López-Soriano, Sílvia Busquets, Klaus Dittmann, et al. "Chronic Lung Allograft Rejection." In Encyclopedia of Molecular Mechanisms of Disease, 357. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_7363.

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Burchell, Richard K. "Interstitial Lung Diseases." In Chronic Disease Management for Small Animals, 281–84. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2017. http://dx.doi.org/10.1002/9781119201076.ch32.

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Conference papers on the topic "Chronic lung disease"

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Robbins, Peter A., James Mountain, David O'Neill, Luca Ciaffoni, John Couper, Jon Whiteley, Gus Hancock, and Grant Ritchie. "Measuring lung inhomogeneity in early chronic lung disease." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa2265.

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Weng, Tingting, Mesias Pedroza, Jose Molina, and Michael R. Blackburn. "Deoxyadenosine Mediated Apoptosis In Chronic Lung Disease." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a2119.

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Murphy, Raymond, and Andrey Vyshedskiy. "Acoustic Biomarkers Of Chronic Obstructive Lung Disease." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a5895.

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KIM, C. Y., Y. N. Kim, and B. K. Kim. "lung function decline in chronic kidney disease." In ERS International Congress 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/13993003.congress-2022.4599.

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Mendoza, N., S. Casas-Recasens, F. Hernandez, N. Albacar, G. Noell, A. Agustí, J. Sellares, and R. Faner. "Immunological profiling of Chronic Obstructive Airways Disease and Idiopathic Pulmonary Fibrosis." In ERS Lung Science Conference 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/23120541.lsc-2020.37.

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Hedhli, A., A. Slim, M. Mjid, S. Hadj Taieb, Y. Ouahchi, S. Cheikh Rouhou, S. Merai, S. Toujani, M. Feki, and B. Dhahri. "Association of testosterone with lung function in patients with chronic obstructive lung disease." In ERS Lung Science Conference 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/23120541.lsc-2021.38.

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Hlapčić, I., A. Hulina-Tomašković, M. Grdić Rajković, S. Popović-Grle, A. Vukić Dugac, and L. Rumora. "Association of extracellular Hsp70 with the severity of chronic obstructive pulmonary disease." In ERS Lung Science Conference 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/23120541.lsc-2020.60.

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Haefner, Verena, Lianyong Han, David Kutschke, Beatrix Steer, Ali Oender Yildirim, Heiko Adler, and Tobias Stoeger. "Reactivation of latent virus infection links air pollutants to chronic lung disease." In ERS Lung Science Conference 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/23120541.lsc-2022.157.

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Güzelkaş, Ismail, Nagehan Emiralioğlu, Dilber Ademhan Tural, Beste Özsezen, Birce Sunman, Halime Nayır Büyükşahin, Ebru Yalçın, Deniz Doğru, Uğur Özçelik, and Nural Kiper. "Case Series of COVID-19 Disease in Pediatric Chronic Lung Diseases." In ERS International Congress 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/13993003.congress-2021.pa3154.

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Bollenbecker, S., B. Czaya, J. Garth, M. Easter, D. Kentrup, J. W. Barnes, C. Faul, and S. Krick. "Elevated Serum Phosphate Levels Exacerbate Chronic Lung Disease." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a4748.

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Reports on the topic "Chronic lung disease"

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Barros-Poblete, Marisol, Rodrigo Torres-Castro, Mauricio Henríquez, Anita Guequen, Isabel Blanco, and Carlos Flores. Dysbiosis as a prognostic factor for clinical worsening in chronic respiratory disease: A systematic review and metanalysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2022. http://dx.doi.org/10.37766/inplasy2022.4.0089.

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Review question / Objective: Is dysbiosis a prognostic factor for clinical worsening in patients with chronic respiratory diseases?. Condition being studied: Dysbiosis, defined as changes in the quantitative and qualitative composition of the microbiota. Eligibility criteria: Over 18 years old adult patients with chronic respiratory diseases clinical diagnosis (cystic fibrosis, chronic obstructive pulmonary disease, asthma, idiopathic pulmonary fibrosis, interstitial lung disease, sarcoidosis, bronchiectasis, non-CF bronchiectasis, pulmonary hypertension) according to the International Statistical Classification of Diseases and Related Health Problems (ICD) from OMS) and international guidelines of each disease.
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Ly, Lena, Jennifer Philip, Peter Hudson, and Natasha Smallwood. Singing for people with advance chronic respiratory diseases: a qualitative meta-synthesis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, August 2022. http://dx.doi.org/10.37766/inplasy2022.8.0017.

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Review question / Objective: This study undertook a meta-synthesis of qualitative data with the aim of collating, synthesizing, and evaluating the current evidence regarding the experiences of singing for people with advanced chronic respiratory disease. Condition being studied: Advanced respiratory illnesses are disorders that impact the airways and other structures of the lung. People with lung cancer, chronic obstructive pulmonary disease (COPD) and interstitial lung disease (ILD) frequently experience progressive, frightening breathlessness, cough and fatigue, which affect their quality of life. Furthermore, people with advanced chronic respiratory disease (CRD) and their carers experience a high prevalence of loneliness and uncertainty, especially if breathlessness is felt to herald death and thus, require both psychological and practical supportive care to cope with their symptoms.
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Czerwaty, Katarzyna, Karolina Dżaman, Krystyna Maria Sobczyk, and Katarzyna Irmina Sikrorska. The Overlap Syndrome of Obstructive Sleep Apnea and Chronic Obstructive Pulmonary Disease: A Systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2022. http://dx.doi.org/10.37766/inplasy2022.11.0077.

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Review question / Objective: To provide the essential findings in the field of overlap syndrome of chronic obstructive pulmonary disease and obstructive sleep apnea, including prevalence, possible predictors, association with clinical outcomes, and severity compared to both chronic obstructive pulmonary disease and obstructive sleep apnea patients. Condition being studied: OSA is characterized by complete cessation (apnea) or significant decrease (hy-popnea) in airflow during sleep and recurrent episodes of upper airway collapse cause it during sleep leading to nocturnal oxyhemoglobin desaturations and arousals from rest. The recurrent arousals which occur in OSA lead to neurocognitive consequences, daytime sleepiness, and reduced quality of life. Because of apneas and hypopneas, patients are experiencing hypoxemia and hypercapnia, which result in increasing levels of catecholamine, oxidative stress, and low-grade inflammation that lead to the appearance of cardio-metabolic consequences of OSA. COPD is a chronic inflammatory lung disease defined by persistent, usually pro-gressive AFL (airflow limitation). Changes in lung mechanics lead to the main clini-cal manifestations of dyspnea, cough, and chronic expectoration. Furthermore, patients with COPD often suffer from anxiety and depression also, the risk of OSA and insomnia is higher than those hospitalized for other reasons. Although COPD is twice as rare as asthma but is the cause of death eight times more often.
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Liu, Lu, Wenchuan Qi, Qian Zeng, Ziyang Zhou, Daohong Chen, Lei Gao, Bin He, Dingjun Cai, and Ling Zhao. Does acupuncture improve lung function in chronic obstructive pulmonary disease animal model?: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2022. http://dx.doi.org/10.37766/inplasy2022.3.0104.

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Review question / Objective: Chronic obstructive pulmonary disease (COPD) is a common, preventable, and treatable disease characterized by persistent respiratory symptoms and progressive airflow obstruction documented on spirometry. Acupuncture, as a safe and economical non-pharmacology therapy, has pronounced therapeutic effects in COPD patients. Several systematic reviews draw the conclusion that acupuncture could improve patients’ quality of life, exercise capacity and dyspnoea, however, the results about lung function were inconclusive. Recently, increasing number of animal studies has been published to illustrate the effects of acupuncture in improving lung function in COPD animal model. However, the efficacy of acupuncture for experimentally induced COPD have not been systematically investigated yet. A systematic review of animal experiments can benefit future experimental designs, promote the conduct and report of basic researches and provide some guidance to translate the achievements of basic researches to clinical application in acupuncture for COPD. Therefore, we will conduct this systematic review and meta-analysis to evaluate effects of acupuncture on COPD animal model.
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du, xinying, xinying du, xianjun fu, kuo xu, and kejian li. Meta analysis on the treatment of acute exacerbation of chronic obstructive pulmonary disease with lung dispersing therapy. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2022. http://dx.doi.org/10.37766/inplasy2022.11.0094.

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Liang, R., D. Liu, HB Li, and ZG Zhai. The efficacy and safety of traditional Chinese medicine formulas in the treatment of chronic obstructive pulmonary disease complicated with pulmonary hypertension: a systematic review and meta-analysis study. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, October 2022. http://dx.doi.org/10.37766/inplasy2022.10.0041.

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Review question / Objective: This systematic review and meta-analysis was intended to evaluate the efficacy and safety of traditional Chinese medicine(TCM) formulas in the treatment of chronic obstructive pulmonary disease(COPD) complicated with pulmonaryhypertension (PH). Condition being studied: Chronic obstructive pulmonary disease(COPD) complicated with pulmonary hypertension(PH) is classified as the third group PH.According to epidemiology, the most common cause of PH associated with lung diseases and/or hypoxia is COPD, but the prevalence rate of COPD with PH range from 20% to 91% variously. In China, many TCM formulas are regularly used in COPD patients , thus TCM formulas therapy is worth considering.
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Ma, He, Jifu Zhao, and Zhilei Wang. Efficacy and safety of HuaYu TongFu Method combined with acupuncture in the treatment of Acute Exacerbation of Chronic Obstructive Pulmonary Disease：A protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2022. http://dx.doi.org/10.37766/inplasy2022.9.0114.

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Review question / Objective: This study is the protocol for a systematic review to evaluate the efficacy and safety of HuaYu TongFu Method combined with acupuncture in the treatment of Acute Exacerbation of Chronic Obstructive Pulmonary Disease. we conducted a systematic review and meta-analysis of published randomized clinical trials (RCTs) of such combined therapy in the treatment of AECOPD, It provides a reliable scientific basis for clinicians to use this approach to treat AECOPD. Condition being studied: Chronic obstructive pulmonary disease is the third leading cause of death worldwide. AECOPD is the most common cause of hospitalization and death in patients with COPD. As lung function deteriorates and the disease progresses, the risk of alveolar hypoxia and consequent hypoxemia increases. Inflammation plays an important role in the progression of AECOPD. Modern medicine mainly treats AECPD by anti-inflammatory, relief of airway spasm, glucocorticoids, inhalants and other methods. Long-term application can easily lead to bacterial flora imbalance and drug resistance in patients. Comparatively, traditional Chinese medicine and acupuncture therapy are safe and effective.To assess the therapeutic efficacy and safety of HuaYu TongFu Method combined with acupuncture in AECOPD, we created a protocol for a systematic review to inform future clinical applications.
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Appleyard, Bruce, and Tim Garrett. Incorporating Public Health into Transportation Decision Making. Mineta Transportation Institute, January 2023. http://dx.doi.org/10.31979/mti.2023.2150.

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Investments in transportation have the potential to significantly affect public health outcomes. Decisions to build highways, transit, or bikeways, for example, influence how residents and visitors move around a metropolitan area. Personal travel habits and proximity to transportation infrastructure play a role in how likely people are to be physically active or be exposed to dangerous traffic and toxic pollution. For this study, the research team reviewed the literature that links transportation infrastructure, the surrounding built environment context, and public health outcomes such as chronic heart and lung diseases, obesity, and death. The team then researched publicly available data that planners could use to inform decision-makers about the public health effects of funding certain investments. Finally, the team reviewed the guidelines of existing discretionary grant programs administered by the California Transportation Commission (CTC), and proposed improvements that would better incorporate available data on public health for consideration. These steps can positively influence funding decision-making for better public health outcomes in California.
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Questionnaires directed at smokers improve detection of chronic lung disease in general practice. National Institute for Health Research, October 2016. http://dx.doi.org/10.3310/signal-000311.

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Study of the prevalence of chronic, non-specific lung disease and related health problems in the grain handling industry. U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control, National Institute for Occupational Safety and Health, October 1986. http://dx.doi.org/10.26616/nioshpub86117.

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