Relevant bibliographies by topics / Chronic fatigue syndrome

Academic literature on the topic 'Chronic fatigue syndrome'

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Published: 4 June 2021
Last updated: 29 July 2024

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Journal articles on the topic "Chronic fatigue syndrome"

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Blitshteyn, Svetlana, and Pradeep Chopra. "Chronic Fatigue Syndrome: From Chronic Fatigue to More Specific Syndromes." European Neurology 80, no. 1-2 (2018): 73–77. http://dx.doi.org/10.1159/000493531.

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In the last decade, a group of chronic disorders associated with fatigue (CDAF) emerged as the leading cause of chronic fatigue, chronic pain, and functional impairment, all of which have been often labeled in clinical practice as chronic fatigue syndrome (CFS) or fibromyalgia. While these chronic disorders arise from various pathophysiologic mechanisms, a shared autoimmune or immune-mediated etiology could shift the focus from symptomatic treatment of fatigue and pain to targeted immunomodulatory and biological therapy. A clinical paradigm shift is necessary to reevaluate CFS and fibromyalgia diagnoses and its relationship to the CDAF entities, which would ultimately lead to a change in diagnostic and therapeutic algorithm for patients with chronic fatigue and chronic pain. Rather than uniformly apply the diagnoses of CFS or fibromyalgia to any patient presenting with unexplained chronic fatigue or chronic pain, it may be more beneficial and therapeutically effective to stratify these patients into more specific diagnoses in the CDAF group.
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DIENER, ROBERT B. "Chronic Fatigue and Related Immune Deficiency Syndromes, Chronic Fatigue Syndrome." American Journal of Psychiatry 153, no. 1 (January 1996): 125—a—126. http://dx.doi.org/10.1176/ajp.153.1.125-a.

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BOMBARDIER, CHARLES H., and DEDRA BUCHWALD. "Chronic Fatigue, Chronic Fatigue Syndrome, and Fibromyalgia." Medical Care 34, no. 9 (September 1996): 924–30. http://dx.doi.org/10.1097/00005650-199609000-00005.

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White, P. D. "Chronic unexplained fatigue." Acta Neuropsychiatrica 11, no. 4 (December 1999): 130–33. http://dx.doi.org/10.1017/s0924270800035870.

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SummaryFatigue is a common symptom in the community and the commonest associations are with stress or mood disturbance. One in a hundred people complain of unexplained and prolonged fatigue, with half that number meeting the strictest criteria for the chronic fatigue syndrome (CFS). Discrete fatigue syndromes have been described, particularly after Epstein Barr virus infection. The majority of patients with CFS have a syndrome similar to the ICD-10 definition of neurasthenia. Mood and somatoform disorders are common comorbid or differential diagnoses.The prognosis is poor, particularly in patients attending hospitals and those with comorbid psychiatric disorders. The aetiology of both CFS and chronic unexplained fatigue are essentially unknown, perhaps reflecting the heterogenenous natures of both the symptom and syndrome. There is reasonable evidence to suggest that particular infections may trigger both prolonged fatigue and CFS. Maintaining factors are different from triggering factors and include mood and sleep disorders, illness beliefs and behaviours, and possibly inactivity. Treatments aimed at reversing these maintaining factors show promise.
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Brkic, Snezana, Slavica Tomic, Maja Ruzic, and Daniela Maric. "Chronic fatigue syndrome." Srpski arhiv za celokupno lekarstvo 139, no. 3-4 (2011): 256–61. http://dx.doi.org/10.2298/sarh1104256b.

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Chronic fatigue syndrome (CFS) is defined by a profound, debilitating fatigue, lasting for at least 6 months and resulting in a substantial reduction of occupational, personal, social and educational status. CFS is a relatively poorly recognized clinical entity, although everyday experience shows that there are many patients with CFS symptoms. The incidence and prevalence of CFS remain unknown in most countries; however, the working population is most affected with predominantly female patients in generative period. Although, CFS was first mentioned four centuries ago, mysterious aethiopathogensis of CFS still intrigues scientists as hundreds of studies are still published every year on the subject. About 80 different aetiological CFS factors are mentioned, which can be classified into five basic groups: genetics, immunology, infectious diseases, endocrinology and neuropsychiatry-psychology. Even today the condition is passed established based on the diagnosis by exclusion of organic and psychiatric disorders, which demands u multidisciplinary approach. As the syndrome is often misdiagnosed and mistreated, self-medication is not uncommon in CFS patients?. In addition, such patients usually suffer for years tolerating severe fatigue. Thus, at the moment there are three priorities regarding CFS; understanding pathogenesis, development of diagnostic tests and creating efficient treatment program.
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Sue Graves, B., and Sigourney Kame. "Chronic Fatigue Syndrome." WSEAS TRANSACTIONS ON BIOLOGY AND BIOMEDICINE 18 (March 17, 2021): 17–23. http://dx.doi.org/10.37394/23208.2021.18.2.

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Chronic fatigue syndrome is an enduring disease, characterized by a level of persistent fatigue for 6 months or a longer time period. At this time, the etiology is unknown. The other symptoms individuals effected by chronic fatigue syndrome, may experience are sore throat, headaches, impaired cognition, depression, sleep disturbances, and many others. While the diagnosis of chronic fatigue syndrome can be challenging, the Center for Disease Control (CDC) has a set of guidelines to help characterize the presence of this condition in patients. Chronic fatigue syndrome has far-reaching consequences impacting an individual’s physical and mental wellbeing. The best approach in helping these individuals to still engage in physical activity is through gentle, lowlevel exercise program with the use of patient feedback to individualize the treatment as well as providing guidance and support through support groups and therapy as prescribed by a medical professional.
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Datieva, Veronika K., Achcha Sh Chimagomedova, and Oleg S. Levin. "Chronic fatigue syndrome." Psychiatry 80 (2018): 74–81. http://dx.doi.org/10.30629/2618-6667-2018-80-74-81.

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Fuller, Nancy S., and Robert E. Morrison. "Chronic fatigue syndrome." Postgraduate Medicine 103, no. 1 (January 1998): 175–84. http://dx.doi.org/10.3810/pgm.1998.01.278.

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Mooney, Tracy. "Chronic fatigue syndrome." Nursing Standard 27, no. 4 (September 26, 2012): 57–58. http://dx.doi.org/10.7748/ns.27.4.57.s58.

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Mooney, Tracy. "Chronic fatigue syndrome." Nursing Standard 27, no. 4 (September 26, 2012): 57. http://dx.doi.org/10.7748/ns2012.09.27.4.57.c9315.

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Dissertations / Theses on the topic "Chronic fatigue syndrome"

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Nairn, Carron. "Enteroviruses and chronic fatigue syndrome." Thesis, University of Glasgow, 1999. http://theses.gla.ac.uk/39026/.

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A prospective study was initiated to follow a group of patients with chronic fatigue syndrome (CFS) and examine their serum for evidence of enteroviral sequences over time and to compare any positive PCR products by sequence analysis. Additionally, a questionnaire was developed to determine the clinical nature of the illness and to try to correlate this with the enteroviral status of the patient at a particular time. Initial phylogenetic analysis of sequence derived from twenty serum positive samples showed that the sequences derived from CFS patients grouped apart from the known enteroviruses and from sequences derived from other clinical cases on a phylogenetic tree (Galbraith et al., 1995). The sequences were approximately 69-84% similar to the closest sequence of a coxsackievirus B3. However, the sequences could have been from known enteroviruses with no sequence data available, from known enteroviruses with variant 5' NTRs or from previously unknown enteroviruses. Further analysis at a later date with other available sequence data showed that the sequences grouped within the coxsackie/echovirus group although some were still 10-13% different. The variation observed was of the order of that seen between clinical isolates of the same serotype, thus the CFS patient sequences may have represented known enteroviruses with variant 5' NTRs. Without corroboration from other regions this tentative group could not be described further. The capsid region was chosen as an alternative target region since it is relatively conserved among the enteroviruses, containing the neutralizing regions which form the basis of the serotypes. Semi-nested priming could amplify the majority of the known enteroviruses to a sensitivity of 1 TCID50, which was 100-fold less than the standard PCR. Testing of 55 samples previously positive by the standard PCR did not generate any positives. The use of other techniques (inverse PCR, long PCR and production of a cDNA library) to acquire additional sequence were also unsuccessful. Either the assays were not sensitive enough to detect the low levels of RNA present in the serum (as assessed by the standard PCR) or the genome is so different or deleted (perhaps due to the presence of defective-interfering particles) that amplification was not possible. Indeed, the discovery that 85% of enteroviral RNA samples lacked a poly-A tail may also point to there being atypical sequence present. Limited sample volume prevented this from being pursued further. Persistence was examined by analyzing the 5' NTR sequence of two sequential samples obtained from sixteen patients. Many sequences, however, were more similar to those isolated in the same year than to their respective pair. Additionally, correlation with clinical data did not suggest a role for enteroviral persistence in the maintenance of the syndrome, although a positive correlation between severity of symptoms (based on ability to work and time spent walking for example) and the presence of enteroviral sequence was noted on analysis of first questionnaires from a cohort of 130 patients. This was not present on analysis of the second questionnaire from each patient. The average duration of fatigue for this group of patients was 3.9 years and thus questionnaire analysis proved difficult in terms of patients recalling features prior to their fatigue. In a preliminary study, a group of patients who had a history of fatigue of approximately six months was described. Enteroviral sequences were detected in the serum of 42% of CFS patients compared to 27% of acutely-ill patients and 2% of healthy control individuals. Additionally, a number of positives in the CFS patient group were detected after one round of PCR only, perhaps indicating a higher titre of enterovirus in these samples. While this study was being carried out there was an outbreak of echovirus 4 and this suggested therefore that the virus played a direct role in triggering the syndrome. Sequence analysis of PCR positives was not performed at this time to confirm this theory. Thus it may be useful to test patients shortly after the onset of fatigue where there is a history of a flu-like illness, when there is a greater chance of recovering a possible triggering agent, even though a formal diagnosis has not been made. Consistently, a higher proportion of CFS patients than comparison individuals were positive for enteroviral sequences throughout this study (except in 1997) and thus this work does not rule out the enteroviruses as being involved in CFS. However, the methods employed did not elucidate any further sequence information nor provide a definitive answer regarding the role of these viruses in individuals with a long history of illness.
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Dogolich, O. I. "Chronic fatigue syndrome – an illness or a syndrome?" Thesis, БДМУ, 2020. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/18048.

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White, Christine M. "Chronic fatigue syndrome : a qualitative investigation /." [St. Lucia, Qld.], 2004. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe17733.pdf.

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Coryell, Virginia Tai. "Orthostatic Intolerance in Chronic Fatigue Syndrome." Scholarly Repository, 2008. http://scholarlyrepository.miami.edu/oa_theses/94.

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Persons with chronic fatigue syndrome (CFS) often complain of an inability to maintain activity levels and experience a variety of orthostatic symptoms such as dizziness, trembling, nausea, postural hypotension with bradycardia or tachycardia, sweating, palpitations, paleness, and syncope. Orthostatic intolerance (OI) may be defined as an inability to maintain systolic blood pressure (SBP) within 20 mmHg of resting level upon moving from a supine to upright posture. The primary objective of this study is to determine whether men and women with CFS are more susceptible to OI during a 3-stage head-up tilt (HUT) than non CFS, sedentary subjects matched by age, sex, and ethnicity. The secondary objective is to examine whether possible underlying mechanisms may be predictively associated with OI susceptibility in CFS. Possible causes of OI include autonomic nervous system (ANS) dysfunction and altered hematological profile. Thus, specific aims included within this objective are: 1) to determine whether there are differences in resting cardiovascular function {i.e., blood pressure [BP], heart rate [HR], stroke volume [SV], cardiac output [CO], total peripheral resistance [TPR], and contractility [i.e., ejection fraction (EF), fractional shortening (FS), and the velocity of circumferential shortening corrected by HR (VCFc)]}, ANS function {i.e., beta1-, beta2-, and alpha-receptor sensitivities, baroreceptor sensitivity [BRS], and vagal function [i.e., respiratory sinus arrhythmia (RSA), RSA envelope (RSAE), high frequency (HF) spectral component, and HR range]}, and hematological profile [i.e., red blood cell volume (RBCV), plasma volume (PBV), and total blood volume (TBV)] between CFS and non-CFS groups; and 2) to determine whether cardiovascular, ANS, and hematological measures differentially predicted OI during HUT. The results indicate that OI susceptibility does not occur with greater prevalence in persons with CFS than non-CFS sedentary persons. However, power analyses revealed that with a much larger sample size group differences in OI susceptibility would be found. The CFS group was distinguished from the control group only by differences in blood volume measures. There appears to be no substantive group differences in a range of cardiovascular and ANS measures; moreover, none of these measures, including the blood volume measures, accounted for differences in OI susceptibility. Compensatory mechanisms may be present in CFS for the diminished blood volume that could explain the lack of group differences in OI susceptibility. In addition, future research may find some clues relevant to CFS pathophysiology in the assessment of hemodynamic responses during orthostatic challenge in the present subjects.
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Brocki, Joanna Mary. "Significant others and chronic fatigue syndrome." Thesis, University of Manchester, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.491148.

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Chronic fatigue syndrome is a symptomatically defined condition primarily characterised by a minimum of 6 months severe and debilitating fatigue of new onset which cannot be explained by any other medical causes. CFS is considered a controversial condition as aetiology is unclear, prognosis poor and symptom patterns commonly fluctuate. Families may be particularly important in CFS as the loss of wider social networks may make CFS patients more reliant on close family members. What little work there is suggests that significant others and CFS patients hold largely similar beliefs about the illness and that significant others' responses may be associated with functional outcome in CFS (Heijmans, de Ridder, & Bensing, 1999; Schmaling, Smith, & Buchwald, 2000). The primary purpose of this study was to explore the illness cognitions, including spontaneously made causal explanations for CFS onset and symptoms (causal attributions), of significant others of patients with CFS in relation to a number of outcomes including significant others' burden, distress and their behavioural responses to the CFS patient. A secondary feature of this study is the use of a variety of different quantitative and qualitative research methods, combined to obtain a detailed understanding of significant others' beliefs. Participants were 30 CFS patients and their nominated 'significant other'. Participants were interviewed at home using a semistructured interview examining illness cognitions. They also completed a battery of questionnaires measuring distress and the illness perceptions of both patient and significant other. Patient functioning was assessed from the viewpoints of both patient and significant other. Significant others also completed a measure of behavioural responses to the patient, and a measure of burden. Additionally, eighty five undergraduate students completed questionnaire measures to assess their knowledge of and beliefs about CFS. CFS patients and students reported less coherent models of the condition than did significant others. Significant others' illness cognitions were found to be predictive of burden and distress experienced, and their behavioural responses were associated with a number of aspects of patient functioning. Significant other distress was associated with personal and global attributions. Rejecting-hostile responses towards the patient were associated with attributing CFS to causes personal to the patient. Where significant others made more controllability attributions, they were less likely to make responses encouraging the patient to rest. Additionally, there were significant associations between causal attributions and a number of illness cognition dimensions which are discussed. The use of qualitative analysis techniques (thematic analysis and interpretative phenomenological analysis) identified issues of identity as important in terms of illness beliefs and behavioural response. The present study demonstrates that illness cognitions, whether measured using standard questionnaires, from spontaneous attributions, or qualitatively, are related to important outcomes for both the significant other and the CFS patient. However, the use of different methods demonstrates that what appear to be similar or the same constructs derived from different methodologies may actually be subtly different. In terms of formulating models of illness, the present study highlights the importance of clarity when utilising terminology in terms of research in this area. Further research in this area might usefully focus on an examination of the way in which two partners thinking about and experiencing the impact of chronic illness together formulate a joint story with a particular focus on how such a joint story might develop.
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Chaudhuri, Abhijit. "Metabolic changes in chronic fatigue syndrome." Thesis, University of Glasgow, 2003. http://theses.gla.ac.uk/6542/.

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Metabolic functions are one of the principal determinants of energy expenditure and are exquisitely susceptible to the effects of circulating hormones and chemical changes. Consequently, clinical experiments based on energy expenditure and metabolic functions were considered to be valid approaches to the present research. Significant abnormalities were found in the proton magnetic resonance spectroscopy of basal ganglia in CFS patients. Automatic cardiovascular responses to exercise are also impaired in a subset of CFS patients. Finally, plasma membrane injury appears to be a possible explanation for a range of observations made in this research. Subjective fatigue is a complex symptom. It is the outcome of a variable combination of physiological and neuropsychological changes induced by the primary disease process. Downstream links between brain, neuromuscular and the cardiorespiratory functions are implicated in the neural control of force output during exercises in health and disease. Higher perceived fatigue in CFS is probably caused by the central mechanisms while the sensory input to these neural regulatory mechanisms may limit endurance to maximal and submaximal exercises. Based on these findings and more indirect evidence from other studies, changes in cell membrane properties affecting neuronal signalling in the basal ganglia seem to emerge as one of the likely pathophysiological mechanisms in CFS. There is also evidence of an imbalance of the central autonomic tone in a subset of CFS patients. Surely, research in CFS has the potential to unravel the biology of central fatigue and may bridge the gap that exists between the borderland of neurology and psychiatry.
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Majeed, Tahir. "Neuroendocrine alterations in chronic fatigue syndrome." Thesis, University of Glasgow, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.295330.

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Dendy, Catherine. "Cognitive aspects of chronic fatigue syndrome." Thesis, Open University, 1997. http://oro.open.ac.uk/57681/.

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Recent interest in cognitive behavioural therapy techniques for treatment of Chronic Fatigue Syndrome (CFS) has highlighted the contribution of psychological approaches to alleviating the debilitating symptoms of this illness. In previous research sufferers from CFS have been compared with depressed patients and patients with neuromuscular disease, as they share similar symptoms, but not diagnosis. This study attempts to compare four groups including a normal working group. A new measures was developed and piloted, designed to measure interpretations of symptoms in CFS. In addition standard instruments were used to focus on the measurement of high personal standards, perfectionism, emotional control and conscientiousness and levels of autonomy. Results showed the CFS group were similar to the normal working group on all standard scales and scored low on autonomous personality traits. Reasons for this result, and the clinical implications for treating such a heterogeneous patient group are discussed. Measures on the symptom interpretation scale show CFS patients are less likely to give an emotional explanation for their symptoms than the other participant groups. This has implications for communication between physician and patient, and the treatment of CFS with a psychological model.
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Karfakis, Nikolaos. "The biopolitics of chronic fatigue syndrome." Thesis, University of Leicester, 2013. http://hdl.handle.net/2381/27969.

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This thesis approaches Chronic Fatigue Syndrome (CFS) as a biopolitical problem, that is as a shifting scientific object which needs to be studied, classified and regulated. Assemblages of authorities, knowledges, and techniques make CFS subjects and shape their everyday conduct in an attempt to increase their supposed autonomy, wellbeing and health. CFS identities are, however, made not only through government, scientific and medical interventions but also by the patients themselves, a biosocial community that collaborates with scientists, educates itself about the intricacies of biomedicine, and contests psychiatric truth claims. CFS is a socio-medical disorder, an illness trapped between medicine, psychology and society, an illness that is open to debate, and therefore difficult to manage and standardise. CFS is, thus, more than a fixed and defined medical category; it is a performative and multiple category, it is a heterogeneous world. This thesis studies that performative complexity by assembling different pieces of empirical data that constitute its heterogeneity: medical and psychiatric journals and monographs, self-help books, CFS organisations’ magazines, newsletters and websites, illness narratives and social studies of CFS, CFS blogs, and qualitative interviews with diagnosed CFS patients and CFS activists. The thesis delineates different interventions by medicine, science, the state and the patients themselves and concludes that CFS remains elusive, only partially standardised, in an on-going battle between all the different actors that want to define it for their own situated interests.
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Fairhurst, Denise Jocelyn. "Cognitive deficits in chronic fatigue syndrome." Thesis, University of Leeds, 1999. http://etheses.whiterose.ac.uk/1393/.

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This thesis focuses on cognition in Chronic Fatigue Syndrome (CFS). Many patients report difficulties such as `fuzzy' thinking, poor memory, reduced attention, and slowness of thought. Whilst much of the earlier literature presented conflicting results, more recently the theory that there is slowed processing has been suggested. Standard neuropychological tests have been used and been helpful in the description of this slowing, however an explanation for this slowing has not been postulated. This thesis proposes a slowed processing theory of cognition in CFS focusing on representational weakness and global reductions in cortical activity as possible mediators of slowing. Sixty-eight CFS patients (tertiary care clinic attendees) and 63 healthy controls participated in the study. They completed standard neuropsychological tests from the Weschler Memory Scale-R and measures of comorbid symptomatology, specifically the Hospital Anxiety and Depression Scale, The Fatigue Scale and the Profile for Fatigue Related Symptoms. They also completed a battery of tests designed to assess whether CFS patients had slowed performance; whether these problems could be attributed to representational weakness; whether there were differences in CFS and control participants' performance on perceptually and conceptually processed tasks, and tasks requiring conscious and non-conscious processing. The role of non-novel versus novel stimuli and interference is also discussed. The The results suggested that CFS patients' recall was worse than control participants' on the following measures: Paired Associate Learning - hard pairs, Logical Memory, and explicit memory. They were slower than controls for all levels of processing graded from perceptual to conceptual, and for semantic judgements of word pair relatedness. The results are discussed as support for a theory of cognition in fatigue which is dependent on 2 factors; firstly, representational weakness and secondly global slowing of cortical activity. It is proposed that these two factors interact, and the performance of CFS patients on what may initially appear to be similar tests can be quite discrepant.
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Books on the topic "Chronic fatigue syndrome"

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Yehuda, Shlomo, and David I. Mostofsky, eds. Chronic Fatigue Syndrome. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-5953-5.

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Straus, Stephen E. Chronic fatigue syndrome. Bethesda, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1991.

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Fisher, Gregg Charles. Chronic Fatigue Syndrome. New York: Grand Central Publishing, 2009.

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1930-, Dawson David M., and Sabin Thomas D, eds. Chronic fatigue syndrome. Boston: Little, Brown, 1993.

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Straus, Stephen E. Chronic fatigue syndrome. Bethesda, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1991.

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Abrams, Liesa. Chronic fatigue syndrome. San Diego: Lucent Books, 2003.

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Shlomo, Yehuda, Mostofsky David I, and Farber Center International Conference on Chronic Fatigue Syndrome (2nd : 1995 : Ramat-Gan, Israel), eds. Chronic fatigue syndrome. New York: Plenum Press, 1997.

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Straus, Stephen E. Chronic fatigue syndrome. Bethesda, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1991.

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Engdahl, Sylvia. Chronic fatigue syndrome. Detroit: Gale, Cengage Learning, 2012.

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Stoff, Jesse A. Chronic fatigue syndrome. New York, NY: HarperPerennial, 1992.

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Book chapters on the topic "Chronic fatigue syndrome"

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Theisler, Charles. "Fatigue/Chronic Fatigue Syndrome." In Adjuvant Medical Care, 127–28. New York: CRC Press, 2022. http://dx.doi.org/10.1201/b22898-137.

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Newton, Julia. "Chronic Fatigue Syndrome." In Postural Tachycardia Syndrome, 191–92. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-54165-1_23.

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Besdine, Richard W. "Chronic Fatigue Syndrome in the Elderly." In Chronic Fatigue Syndrome, 15–27. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-5953-5_1.

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Fukuda, Keiji. "Development of the 1994 Chronic Fatigue Syndrome Case Definition and Clinical Evaluation Guidelines." In Chronic Fatigue Syndrome, 29–50. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-5953-5_2.

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Salit, Irving E. "The Chronic Fatigue Syndrome." In Chronic Fatigue Syndrome, 51–72. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-5953-5_3.

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Christodoulou, Christopher, John DeLuca, Susan K. Johnson, Gudrun Lange, and Benjamin H. Natelson. "Efforts to Reduce Heterogeneity in Chronic Fatigue Syndrome Research." In Chronic Fatigue Syndrome, 73–94. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-5953-5_4.

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Moldofsky, Harvey. "Nonrestorative Sleep, Musculoskeletal Pain, Fatigue, and Psychological Distress in Chronic Fatigue Syndrome, Fibromyalgia, Irritable Bowel Syndrome, Temporal Mandibular Joint Dysfunction Disorders (CFIT)." In Chronic Fatigue Syndrome, 95–117. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-5953-5_5.

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Chaudhuri, Abhijit, Walter S. Watson, and Peter O. Behan. "Arguments for a Role of Abnormal Ionophore Function in Chronic Fatigue Syndrome." In Chronic Fatigue Syndrome, 119–30. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-5953-5_6.

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Sredni, D. "Cytokine Patterns Associated with Chronic Fatigue Syndrome." In Chronic Fatigue Syndrome, 131–59. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-5953-5_7.

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Kastin, Abba J., Richard D. Olson, J. Martin Martins, Gayle A. Olson, James E. Zadina, and William A. Banks. "Chronic Fatigue Syndrome: Possible Integration of Hormonal and Immunological Observations." In Chronic Fatigue Syndrome, 161–92. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-5953-5_8.

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Conference papers on the topic "Chronic fatigue syndrome"

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Russo Macchi, María Verónica, Vanessa Félix Nascimento Coelho, Thais de Campos Ferreira Pinto, Lucas Eduardo Pedri, and Flavia Regina Andrade. "POST-COVID-19 CHRONIC FATIGUE SYNDROME." In Congresso Brasileiro de Reumatologia 2020. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2020.17076.

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Kruglova, Marina, Elena Stolyarchuk, and Irina Panteenko. "Ergonomic predictors of chronic fatigue syndrome in teachers." In Personal resourse of human agency at work in changing Russia. ScientificWorld, 2018. http://dx.doi.org/10.30888/978-5-6041451-4-2.1.16.

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Moeda, S., L. Gamper, A. Gregorowski, and T. Segal. "P27 When chronic fatigue syndrome leads to mutism." In RCPCH and SAHM Adolescent Health Conference; Coming of Age, 18–19 September 2019. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/bmjpo-2019-rcpch-sahm.32.

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Zupanc, Timon. "Myalgic Encephalomyelitis/Chronic Fatigue Syndrome – Etiology, Pathophysiology, Diagnosis and Treatment." In Socratic lectures 10. University of Lubljana Press, 2024. http://dx.doi.org/10.55295/psl.2024.ii1.

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Abstract: Myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) is a complex condition characterized by a broad spectrum of overlapping symptoms and manifesting in multiple systems of the body. Patients experience symptoms such as profound fatigue, post-exertional malaise (PEM), unrefreshing sleep, and cognitive impairments. It affects millions worldwide, yet much is still unknown about its etiology and pathophysiology. The condition's onset is frequently linked to infectious triggers, including viral infections, suggesting a dysregulated immune response as a central component. Diagnosing ME/CFS poses significant challenges due to many unspecific symptoms that overlap with various other conditions. Current treatment strategies focus primarily on symptomatic relief and lifestyle modifications to manage disease impact. The COVID-19 pandemic has further spotlighted ME/CFS, drawing parallels between long COVID and ME/CFS symptomatology and underscoring the urgent need for comprehensive research. Keywords: Myalgic Encephalomyelitis, Chronic Fatigue Syndrome, Infectious trigger, Post-Exertional Malaise, COVID-19, Post-COVID syndrome
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Wu, Yi-Zhi, Yong-Sheng Ding, Hong-An Xu, Jin-Lan Shi, and Bo-Hui Zhu. "SVM Based Chronic Fatigue Syndrome Evaluation for Intelligent Garment." In 2008 2nd International Conference on Bioinformatics and Biomedical Engineering (ICBBE '08). IEEE, 2008. http://dx.doi.org/10.1109/icbbe.2008.816.

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Moeda, S., L. Gamper, A. Gregorowski, and TY Segal. "G615(P) When chronic fatigue syndrome leads to mutism." In Royal College of Paediatrics and Child Health, Abstracts of the RCPCH Conference and exhibition, 13–15 May 2019, ICC, Birmingham, Paediatrics: pathways to a brighter future. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2019. http://dx.doi.org/10.1136/archdischild-2019-rcpch.595.

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Dibnah, Beth, Emmanuella Traianos, Jessica Tarn, Dennis Lendrem, and Wan Fai Ng. "AB0061 INVESTIGATING THE ROLE OF TGF-B AND FATIGUE IN CHRONIC FATIGUE SYNDROME." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.4690.

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Davies, Tabby, Simon L. Jones, and Ryan M. Kelly. "Patient Perspectives on Self-Management Technologies for Chronic Fatigue Syndrome." In CHI '19: CHI Conference on Human Factors in Computing Systems. New York, NY, USA: ACM, 2019. http://dx.doi.org/10.1145/3290605.3300452.

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Kruglova, Marina Anatolyevna, Elena Stanislavovna Starchenkova, Vladimir Georgievich Kruglov, Natalia Evgenievna Vodopyanova, Oleg Valentinovich Leontiev, and Vladimir Anatolyevich Kruglov. "Chronic Fatigue Syndrome Among Teachers in the Conditions of Distance Learning." In 2020 3rd International Seminar on Education Research and Social Science (ISERSS 2020). Paris, France: Atlantis Press, 2021. http://dx.doi.org/10.2991/assehr.k.210120.031.

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Rincon, Alejandro Lopez, Aletta D. Kraneveld, and Alberto Tonda. "Batch correction of genomic data in chronic fatigue syndrome using CMA-ES." In GECCO '20: Genetic and Evolutionary Computation Conference. New York, NY, USA: ACM, 2020. http://dx.doi.org/10.1145/3377929.3389947.

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Reports on the topic "Chronic fatigue syndrome"

1

Yue, Quang H. Neural Mechanism of Chronic Fatigue Syndrome. Fort Belvoir, VA: Defense Technical Information Center, April 2005. http://dx.doi.org/10.21236/ada522800.

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Yue, Guang H. Neural Mechanisms of Chronic Fatigue Syndrome. Fort Belvoir, VA: Defense Technical Information Center, April 2006. http://dx.doi.org/10.21236/ada625338.

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Bi, Yunpeng, Xi Li, Huixin Yan, Xiaomei Zhang, Hongyi Guan, Haiyu Zhu, Tingwei Ding, and Bailin Song. Acupoint massage for chronic fatigue syndrome：A protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2023. http://dx.doi.org/10.37766/inplasy2023.4.0083.

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Review question / Objective: With changes in lifestyle and rhythm, chronic fatigue syndrome (CFS) is becoming increasingly common in the population. Many randomized controlled clinical studies have shown that acupoint massage has significant advantages in improving symptoms such as fatigue. However, there is no systematic review and meta-analysis published on the treatment of chronic fatigue syndrome with acupoint massage, which is worthy of our team's research. Condition being studied: Chronic fatigue syndrome (CFS) is characterized by persistent or recurrent conscious fatigue, accompanied by accompanying symptoms such as sleep disorders, subjective cognitive impairment, or diffuse muscle and bone pain. Its symptoms usually persist for six months or more, and fatigue cannot be relieved after rest. The average prevalence of CFS in the global general population ranges from 1.40 to 1.57%. However, the impact of acupoint massage on chronic vibration fatigue syndrome is still controversial. Therefore, a current systematic review and meta-analysis will be conducted to investigate the role of acupoint massage in the management of chronic fatigue syndrome.
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Smith, M. E. Beth, Heidi D. Nelson, Elizabeth Haney, Miranda Pappas, Monica Daeges, Ngoc Wasson, and Marian McDonagh. Diagnosis and Treatment of Myalgic Encephalomyelitis/Chronic Fatigue Syndrome. Agency for Healthcare Research and Quality, December 2014. http://dx.doi.org/10.23970/ahrqepcerta219.

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Montoya, Jose, Ian Valencia, Holden Maecker, Michael Mindrinos, and Rosemary Fernandez. Subgrouping Chronic Fatigue Syndrome Patients by Genetic and Immune Profiling. Fort Belvoir, VA: Defense Technical Information Center, October 2014. http://dx.doi.org/10.21236/ada608724.

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Vahratian, Anjel, Jin-Mann S. Lin, Jeanne Bertolli, and Elizabeth R. Unger. Myalgic Encephalomyelitis/Chronic Fatigue Syndrome in Adults: United States, 2021-2022. Hyattsville, MD: National Center for Health Statistics (U.S.). National Health Interview Survey (NHIS), December 2023. http://dx.doi.org/10.15620/cdc:134504.

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Liu, Zhen, Zhizhen Lv, Jiao Shi, Shuangwei Hong, Huazhi Huang, and Lijiang Lv. Efficacy of traditional Chinese exercise in patients with chronic fatigue syndrome: a protocol for a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2022. http://dx.doi.org/10.37766/inplasy2022.9.0022.

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Review question / Objective: Chronic fatigue syndrome (CFS) is a disease in which fatigue strikes or lasts for more than 6 months, accompanied by pain, sleep disturbance, anxiety, and depression. Moreover, it brings a heavy economic burden to society. Traditional Chinese exercises (TCEs) are a traditional Chinese medical treatment and have good efficacy on CFS, therefore, this systematic evaluation is to accurately evaluate the efficacy of TCEs on CFS. P: Patients with chronic fatigue syndrome. I: Traditional Chinese exercises. C: conventional exercise, acupuncture, physiotherapy, and other physical therapy methods. O: quality of life, fatigue, pain, sleep, anxiety, and depression. S: randomized controlled trials. Condition being studied: Chronic fatigue syndrome (CFS) is a disease in which fatigue strikes or lasts for more than 6 months, accompanied by pain, sleep disturbance, anxiety, and depression. Moreover, it brings a heavy economic burden to society. Traditional Chinese exercises (TCEs) are a traditional Chinese medical treatment and have good efficacy on CFS. Therefore, this systematic evaluation is to accurately evaluate the efficacy of TCEs on CFS, to provide an alternative therapy for clinical treatment of CFS.
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Tang, Cheng, and Min Fang. Tuina for Chronic Fatigue syndrome: a protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, December 2020. http://dx.doi.org/10.37766/inplasy2020.12.0014.

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Rabago, David. Nasal Irrigation for Chronic Rhinosinusitis and Fatigue in Patients with Gulf War Syndrome. Fort Belvoir, VA: Defense Technical Information Center, July 2012. http://dx.doi.org/10.21236/ada568066.

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Rabago, David. Nasal Irrigation for Chronic Rhinosinusitis and Fatigue in Patients with Gulf War Syndrome. Fort Belvoir, VA: Defense Technical Information Center, July 2013. http://dx.doi.org/10.21236/ada592220.

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