Relevant bibliographies by topics / Chronic diseases

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Chronic diseases'

Author: Grafiati
Published: 4 June 2021
Last updated: 29 July 2024

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Journal articles on the topic "Chronic diseases"

1

Gupta, Neeraj. "Chronic diseases." Indian Journal of Research in Homoeopathy 9, no. 4 (2015): 285. http://dx.doi.org/10.4103/0974-7168.172870.

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Glick, Michael. "Chronic diseases." Journal of the American Dental Association 137, no. 9 (September 2006): 1204–6. http://dx.doi.org/10.14219/jada.archive.2006.0362.

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Miranda, J. J. "Chronic Diseases." International Journal of Epidemiology 44, suppl_1 (September 23, 2015): i29. http://dx.doi.org/10.1093/ije/dyv097.096.

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Ferrando, Stephen J. "Chronic Diseases." Journal of Nervous &amp Mental Disease 186, no. 1 (January 1998): 64. http://dx.doi.org/10.1097/00005053-199801000-00016.

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Montfort-Cabello, H. "Chronic diseases:." Homeopathy 93, no. 2 (April 2004): 88–93. http://dx.doi.org/10.1016/j.homp.2004.01.001.

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Yach, Derek, Stephen R. Leeder, John Bell, and Barry Kistnasamy. "Global Chronic Diseases." Science 307, no. 5708 (January 21, 2005): 317. http://dx.doi.org/10.1126/science.1108656.

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Lee, Young-Sok. "Chronic Liver Diseases." Journal of the Korean Medical Association 47, no. 1 (2004): 10. http://dx.doi.org/10.5124/jkma.2004.47.1.10.

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Ackerman, Nicola. "Managing chronic diseases." Veterinary Nursing Journal 20, no. 2 (February 2005): 30–31. http://dx.doi.org/10.1080/17415349.2005.11013320.

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Fried, Linda P. "Countering Chronic Diseases." Science 339, no. 6115 (January 3, 2013): 35.1–35. http://dx.doi.org/10.1126/science.1230826.

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MAYER, R. J., M. LANDON, G. LENNOX, J. LOW, and F. DOHERTY. "CHRONIC DEGENERATIVE DISEASES." Alzheimer Disease & Associated Disorders 2, no. 3 (1988): 198. http://dx.doi.org/10.1097/00002093-198802030-00047.

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Dissertations / Theses on the topic "Chronic diseases"

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Ye, Ping. "Autoimmunity in chronic periodontitis." Thesis, The University of Sydney, 2003. http://hdl.handle.net/2123/4256.

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Profound perturbation of epithelial structure is a characteristic feature of the immunopatholoical response to bacterial antigens considered to be central in the pathogenesis of the destructive lesion of periodontitis. The pathological basis for the disturbance of epithelial structure is not understood. It was demonstrated that the structural integrity and functional differentiation of the lining epithelium is compromised in relation to inflammatory changes associated with destructive periodontitis. In the pathological lining epithelium of the periodontal pocket there was a marked reduction of epithelial cadherin important in intercellular adhesion, of involucrin, a marker of terminal differentiation, and of the gap junction connexions that form intercellular communication channels. These changes were associated with alterations of filamentous actin expression, collectively indicating profound perturbation of epithelial structure. The data reported support the concept that the ability of the pathological lining epithelium to function as an effective barrier against the ingress of microbial products into the tissues is severely compromised (Ye et al., 2000). In addition, a recent study (Ye et al., 2003) by Western analysis of serum IgG from all 22 patients with chronic periodontitis tested indicated recognition of multiple epithelial components in individual patterns. In contrast, subjects with a healthy periodontium displayed only trace recognition of epithelial antigens. Levels of epithelial-reactive antibodies were significantly correlated with attachment loss as an indication of disease activity. To investigate a possible relationship between the bacterial flora adjacent to the diseased sites and the presence of epithelial-reactive antibodies, subgingival plague samples were taken from deep periodontal pockets and cultured anaerobically. Gram positive bacteria containing antigens potentially cross-reactive with epithelial cells were reproducibly isolated by probing membrane colony lifts with affinity-isolated (epitheial-specific) antibodies. The bacteria were identified as streptococci (S. mitis, S. constellatus and two S. intermedius strains) and Actinomyces (A. georgiae, and A. sp. oral clone) by 16S rDNA sequence homology. Recognition by affinity-isolated antibodies of antigens from the captured organisms was confirmed by Western analysis. Conversely, absorption of affinity-isolated antibodies with bacterial species specifically reduced subsequent recognition of epithelial antigens. To identify the auto-antigens, a human keratinocyte cDNA expression library in Lambda phage was probed using a pooled sera. Groups of responders were detected for CD24 (a recently described adhesion molecule also known as P-selectin ligand), antioxidant protein 2 (a newly recognised member of the thiol-dependment anti-oxidant proteins), lavtate dehydrogenase A, the transcription factor NFAT5, and for three genes encoding novel proteins. Six identified bacteria, especially S intermedius were demonstrated to absorb antibodies reaching with identified auto-antigens in patterns varying between individuals. This evidence indicated that during the course of periodontits, subjects develop increased levels of antibodies to common oral bacteria amongst which are included tissue cross-reactive antigens. Periodontitis could therefore present a risk for the subsequent initiation or exacerbation of a broad spectrum of disease processes including autoimmune, inflammatory, proliferative and degenerative disorders.
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Ye, Ping. "Autoimmunity in chronic periodontitis." University of Sydney, 2003. http://hdl.handle.net/2123/4256.

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Doctor of Philosophy
Profound perturbation of epithelial structure is a characteristic feature of the immunopatholoical response to bacterial antigens considered to be central in the pathogenesis of the destructive lesion of periodontitis. The pathological basis for the disturbance of epithelial structure is not understood. It was demonstrated that the structural integrity and functional differentiation of the lining epithelium is compromised in relation to inflammatory changes associated with destructive periodontitis. In the pathological lining epithelium of the periodontal pocket there was a marked reduction of epithelial cadherin important in intercellular adhesion, of involucrin, a marker of terminal differentiation, and of the gap junction connexions that form intercellular communication channels. These changes were associated with alterations of filamentous actin expression, collectively indicating profound perturbation of epithelial structure. The data reported support the concept that the ability of the pathological lining epithelium to function as an effective barrier against the ingress of microbial products into the tissues is severely compromised (Ye et al., 2000). In addition, a recent study (Ye et al., 2003) by Western analysis of serum IgG from all 22 patients with chronic periodontitis tested indicated recognition of multiple epithelial components in individual patterns. In contrast, subjects with a healthy periodontium displayed only trace recognition of epithelial antigens. Levels of epithelial-reactive antibodies were significantly correlated with attachment loss as an indication of disease activity. To investigate a possible relationship between the bacterial flora adjacent to the diseased sites and the presence of epithelial-reactive antibodies, subgingival plague samples were taken from deep periodontal pockets and cultured anaerobically. Gram positive bacteria containing antigens potentially cross-reactive with epithelial cells were reproducibly isolated by probing membrane colony lifts with affinity-isolated (epitheial-specific) antibodies. The bacteria were identified as streptococci (S. mitis, S. constellatus and two S. intermedius strains) and Actinomyces (A. georgiae, and A. sp. oral clone) by 16S rDNA sequence homology. Recognition by affinity-isolated antibodies of antigens from the captured organisms was confirmed by Western analysis. Conversely, absorption of affinity-isolated antibodies with bacterial species specifically reduced subsequent recognition of epithelial antigens. To identify the auto-antigens, a human keratinocyte cDNA expression library in Lambda phage was probed using a pooled sera. Groups of responders were detected for CD24 (a recently described adhesion molecule also known as P-selectin ligand), antioxidant protein 2 (a newly recognised member of the thiol-dependment anti-oxidant proteins), lavtate dehydrogenase A, the transcription factor NFAT5, and for three genes encoding novel proteins. Six identified bacteria, especially S intermedius were demonstrated to absorb antibodies reaching with identified auto-antigens in patterns varying between individuals. This evidence indicated that during the course of periodontits, subjects develop increased levels of antibodies to common oral bacteria amongst which are included tissue cross-reactive antigens. Periodontitis could therefore present a risk for the subsequent initiation or exacerbation of a broad spectrum of disease processes including autoimmune, inflammatory, proliferative and degenerative disorders.
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Ye, Ping. "Autoimmunity In Chronic Periodontitis." Thesis, The University of Sydney, 2003. http://hdl.handle.net/2123/4872.

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Palma, Durán Susana Alejandra. "Protein glycation & chronic diseases." Thesis, University of Glasgow, 2017. http://theses.gla.ac.uk/8640/.

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Cexus, Olivier. "Immunological mechanisms controlling chronic inflammatory diseases." Thesis, University of Southampton, 2009. https://eprints.soton.ac.uk/67634/.

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Autoimmune diseases (AID) are chronic inflammatory diseases (CID) mediated by selfreactive T and B cells and are generally the results of the breakdown of T cell tolerance to self-antigen and failure of peripheral regulatory mechanisms. In this thesis I studied different mechanisms controlling the development of CIDs. I investigated the initial events involved in the activation of self-reactive CD4+ T cells which mediate the destruction of the thyroid in a mouse model of spontaneous thyroiditis. TAZ10 transgenic mice express a human T cell receptor (TCR) specific for a cryptic epitope of thyroid peroxidise (TPO) generated upon endogenous processing by thyroid epithelial cells (TEC), and a naturally occurring antagonistic epitope presented by dendritic cells (DC) upon exogenous processing of TPO. I have characterized the function of myeloid derived suppressor cells (MDSCs) in TAZ10 mice. MDSCs accumulate in lymphoid and non-lymphoid organs of TAZ10 mice during acute phases of inflammation and their number decrease as inflammation is fading. Despite their strong inhibitory function on T cell function and proliferation, MDSCs fail to prevent the activation of self-reactive T cells. I showed that the manipulation of MDSCs generated DCs that efficiently promoted the activation of T cells from TAZ10 mice. By contrast, peripheral T cells from patients with rheumatoid arthritis (RA) and lupus had a high proliferative activity compared to controls. Further analysis revealed that RA patients had reduced amounts of inhibitory MDSCs in peripheral blood. I showed that in TAZ10 mice TEC upregulate MHC class II molecules and present the cryptic epitope to TAZ10 T cells inducing their activation. I have demonstrated that DCs are responsible for the spreading of the TPO cryptic epitope from the thyroid to draining lymphnodes (DLN) resulting in the strong activation of transgenic T cells from TAZ10 mice. By adoptive transfer experiments, I showed that the activation of naive TAZ10 T cells occurs within days both in the thyroid and draining lymph-nodes (DLN) and resulted in the destruction of the thyroid. Altogether, this work shows for the first time that in a model devoid of any environmental insults, the normal turnover of TEC is sufficient to induce the activation of self-reactive T cells and the development of AID. In this thesis, I have highlighted the potential role of tissue transglutaminse 2 (TG2) in the treatment of CIDs. TG2 contributes to the pathogenesis of celiac disease and I have showed that TG2 activity promotes inflammation in patients with cystic fibrosis (CF). Mutation of the cystic fibrosis transmembrane regulator gene (CFTR) in CF patients is associated with increased TG2 expression and activity. In CF, TG2 promoted the crosslinking of the antiinflammatory peroxisome proliferator-activated receptor (PPAR) into perinuclear agresomes. The functional sequestration of PPAR was leading to increased inflammation. The finding of this function of TG2 in CF was relevant in TAZ10 mice as in-vivo inhibition of TG2 downregulated common markers of inflammation.
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Amoo, A., and Igor Antonovuch Plesh. "Anthroposophic art therapy in chronic diseases." Thesis, МАТЕРІАЛИ міжнародної науково-практичної інтернет-конференції CHERNIVTSI INTERNATIONAL MEDICAL CONFERENCE (СІМЕС). - м. Чернівці 02-03 червня 2017 р, 2017. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/12882.

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Kirsch, Florian [Verfasser]. "Economic aspects of disease management programs in chronic diseases / Florian Kirsch." München : Verlag Dr. Hut, 2018. http://d-nb.info/1164293648/34.

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Verma, Anju. "Ontology based personalized modeling for chronic disease risk evaluation and knowledge discovery an integrated approach : a thesis submitted to Auckland University of Technology in fulfilment of the requirements for [the] degree of Doctor of Philosophy (PhD), 2009 /." Click here to access this resource online, 2009. http://hdl.handle.net/10292/784.

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Populations are aging and the prevalence of chronic disease, persisting for many years, is increasing. The most common, non-communicable chronic diseases in developed countries are; cardiovascular disease (CVD), type 2 diabetes, obesity, arthritis and specific cancers. Chronic diseases such as cardiovascular disease, type 2 diabetes and obesity have high prevalence and develop over the course of life due to a number of interrelated factors including genetic predisposition, nutrition and lifestyle. With the development and completion of human genome sequencing, we are able to trace genes responsible for proteins and metabolites that are linked with these diseases. A computerized model focused on organizing knowledge related to genes, nutrition and the three chronic diseases, namely, cardiovascular disease, type 2 diabetes and obesity has been developed for the Ontology-Based Personalized Risk Evaluation for Chronic Disease Project. This model is a Protégé-based ontological representation which has been developed for entering and linking concepts and data for these three chronic diseases. This model facilitates to identify interrelationships between concepts. The ontological representation provides the framework into which information on individual patients, disease symptoms, gene maps, diet and life history can be input, and risks, profiles, and recommendations derived. Personal genome and health data could provide a guide for designing and building a medical health administration system for taking relevant annual medical tests, e.g. gene expression level changes for health surveillance. One method, called transductive neuro-fuzzy inference system with weighted data normalization is used to evaluate personalized risk of chronic disease. This personalized approach has been used for two different chronic diseases, predicting the risk of cardiovascular disease and predicting the risk of type 2 diabetes. For predicting the risk of cardiovascular disease, the National Nutrition Health Survey 97 data from New Zealand population has been used. This data contains clinical, anthropometric and nutritional variables. For predicting risk of type 2 diabetes, data from the Italian population with clinical and genetic variables has been used. It has been discovered that genes responsible for causing type 2 diabetes are different in male and female samples. A framework to integrate the personalized model and the chronic disease ontology is also developed with the aim of providing support for further discovery through the integration of the ontological representation in order to build an expert system in genes of interest and relevant dietary components.
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Pereira, Filipa Alexandra Antunes Vences de Matos. "Patient associations: raising awareness on chronic diseases." Master's thesis, Universidade de Aveiro, 2015. http://hdl.handle.net/10773/16582.

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Mestrado em Biomedicina Farmacêutica
The patient associations assume a significant role in representing the patients’ rights, sharing information on the diseases, accessing the available resources, maintaining important interactions with other relevant stakeholders and facing challenges. These organizations provide an important support not only to the patients, but also to their families and friends, who are most of the times their direct caregivers. Currently, the patient empowerment has been highly discussed. This project aims to demonstrate in five different clinical disorders what are the motivations, the objectives, the activities, the involvement with other stakeholders and the challenges assumed by the correspondent patient associations. The result of this project allowed to conclude that the significant prevalence of chronic diseases has been introducing changes into the healthcare systems and national disease programmes. Simultaneously, the patients’ ownership has been increasing, contributing to a more participated role near the healthcare professionals and to a more conscious decision regarding the available therapeutics. Patient associations represent these patients and act in critical areas, such as, social, clinical, research, training, education and advocacy.
As associações de doentes assumem um papel fundamental na representação dos direitos dos pacientes, na divulgação de informação acerca das doenças, no acesso aos recursos disponíveis, no relacionamento com os vários intermediários e nos desafios a enfrentar. Estas associações prestam não só um grande apoio aos doentes, como também aos seus familiares e amigos, que são na maior parte das vezes os seus cuidadores diretos. Atualmente, a capacitação de doentes tem vindo a ser bastante discutida. Este projeto pretende demonstrar em cinco quadros clínicos distintos, quais as motivações presentes, os objetivos, as atividades realizadas, o envolvimento com outros stakeholders e os desafios enfrentados pelas respetivas associações de doentes. O resultado deste trabalho permitiu concluir que a prevalência significativa de doenças crónicas tem introduzido alterações nos sistemas de saúde e nos planos nacionais de doenças. Em simultâneo, a responsabilidade dos pacientes tem vindo a aumentar, contribuindo para um posicionamento cada vez mais participativo junto dos profissionais de saúde e para uma tomada de decisão mais consciente em relação às terapêuticas disponíveis. As associações de doentes dão voz a estes pacientes e atuam em áreas críticas, tais como, a área social, emocional, clínica, de investigação, de formação, de educação e de defesa e representatividade de direitos e benefícios.
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White, Joanna D. "Investigations into feline chronic kidney disease." Thesis, The University of Sydney, 2010. https://hdl.handle.net/2123/28931.

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Chronic kidney disease (CKD) is arguably the most common disease of older cats. A disproportionate number of younger, male cats with CKD was identified among a cohort of cats with CKD and was hypothesised to be due to membranous glomerulopathy or an FIV associated nephropathy. Examination of epidemiologic, histologic, immunohistologic and survival data revealed an association between the presence of CKD and FIV infection among young cats and an adverse effect of FIV infection on survival among cats with CKD, but no specific histological changes were seen among FIV positive cats. Glomerulopathies were identified among 16% of cats with CKD but more female than male cats were diagnosed with glomerulopathies and proliferative rather than membranous glomerulopathies were diagnosed more commonly. ' While glomerulopathies are the cause of CKD in a proportion of cats, membranous glomerulopathy is unlikely to be the predominant glomerulopathy and more work is required to define the both the pathophysiology and histology of feline glomerulopathies. A novel familial glomerulopathy was identified among young Abyssinian cats which was characterised by the presence of haematuria. Ultrastructural and immunhistochemical studies will be required to further characterise these glomerulopathies. Routine histologic examination of 95 cats with kidney disease confirmed the results of earlier studies regarding the proportions of cats with CKD with glomerulopathies, chronic tubulointerstitial nephritis (TIN) and pyelonephritis. A new observation was the significant number of cats had pathologic changes in the inner medulla and renal crest including necrosis and epithelial dysplasia. Bacteriuria was common among cats with CKD, in particular among older, female cats. There was no association between a positive urine culture and disease severity, assessed by creatinine concentration, and a treated episode of bacteriuria had no adverse influence on survival. Bacteria were infrequently identified in cats with neutrophilic TIN, including cats with a histologic diagnosis consistent with pyelonephritis. Further work is required to distinguish cats with asymptomatic bacteriuria from those with urinary tract infections and at risk of pyelonephritis. In addition, causes of neutrophilic TIN other than bacterial infection should be evaluated.
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Books on the topic "Chronic diseases"

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Marvin, Stein, and Baum Andrew, eds. Chronic diseases. Mahwah, N.J: L. Erlbaum Associates, 1995.

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Rayees, Sheikh, Inshah Din, Gurdarshan Singh, and Fayaz A. Malik, eds. Chronic Lung Diseases. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-3734-9.

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Lauerman, John F. Chronic diseases of aging. Waltham, MA: Decision Resources, 1996.

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Parliament, Great Britain. Prescriptions (Chronic Diseases) Bill. London: Stationery Office, 2002.

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F, Voelkel Norbert, and MacNee William, eds. Chronic obstructive lung diseases. Hamilton, Ont: BC Decker, 2002.

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Ghana, University of, ed. Chronic non-communicable diseases in Ghana: Multidisciplinary perspectives. Legon, Accra, Ghana: For the University of Ghana by Sub-Saharan Publishers, 2013.

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Washington (State). Dept. of Fish and Wildlife., ed. Chronic wasting disease. [Olympia, Wash.]: Washington Dept. of Fish and Wildlife, 2000.

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Washington (State). Dept. of Fish and Wildlife., ed. Chronic wasting disease. [Olympia, Wash.]: The Dept., 2000.

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Simoes, Eduardo J. Chronic disease report. Jefferson City, MO: Missouri Dept. of Health, Division of Chronic Disease Prevention and Health Promotion, 2000.

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Vercellini, Paolo. Chronic pelvic pain. Chichester, West Sussex: Wiley-Blackwell, 2011.

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Book chapters on the topic "Chronic diseases"

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Ramirez, Amelie G., Ian M. Thompson, and Leonel Vela. "Chronic Diseases." In The South Texas Health Status Review, 73–83. Cham: Springer International Publishing, 2013. http://dx.doi.org/10.1007/978-3-319-00233-0_7.

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Chuck, Emil T., and Ledric D. Sherman. "Chronic diseases." In Men’s Health, 79–91. Abingdon, Oxon ; New York, NY : Routledge, 2020.: Routledge, 2020. http://dx.doi.org/10.4324/9781351022620-9.

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Fry, John. "Chronic Bronchitis." In Common Diseases, 106–19. Dordrecht: Springer Netherlands, 1985. http://dx.doi.org/10.1007/978-94-009-4924-9_12.

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Millikan, Keith W. "Chronic Pancreatitis." In Common Surgical Diseases, 152–55. New York, NY: Springer New York, 1998. http://dx.doi.org/10.1007/978-1-4757-2945-0_34.

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Paswan, Shravan Kumar, Vishal Kumar Vishwakarma, Chetan Rastogi, Pritt Verma, Ch V. Rao, and Sajal Srivastava. "Chronic Pneumonia." In Chronic Lung Diseases, 75–86. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-3734-9_4.

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Broucek, Joseph, and Jonathan A. Myers. "Chronic Pancreatitis." In Common Surgical Diseases, 143–45. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1565-1_35.

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Nagaratnam, Nages, Kujan Nagaratnam, and Gary Cheuk. "Chronic Liver Disease." In Geriatric Diseases, 1–12. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-32700-6_22-1.

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Nagaratnam, Nages, Kujan Nagaratnam, and Gary Cheuk. "Chronic Liver Disease." In Geriatric Diseases, 203–15. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-33434-9_22.

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Billo, Nils E., Nick Banatvala, Pascal Bovet, and Asma El Sony. "Chronic respiratory diseases." In Noncommunicable Diseases, 118–24. London: Routledge, 2023. http://dx.doi.org/10.4324/9781003306689-18.

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Garcia-Martinez, Rita, Raquel Diaz-Ruiz, Jesus Millan, and Rafael Bañares. "Chronic Liver Failure and Acute-on-Chronic Liver Failure." In Liver Diseases, 381–94. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-24432-3_33.

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Conference papers on the topic "Chronic diseases"

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Liu, Renxuan, and Duan Wu. "Re-establishing the balance: A New Community-based Chronic Disease Management Service Model in China." In 14th International Conference on Applied Human Factors and Ergonomics (AHFE 2023). AHFE International, 2023. http://dx.doi.org/10.54941/ahfe1003492.

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As China's aging process accelerates, chronic diseases such as diabetes and high blood pressure gradually become hidden dangers that endanger the health of the elderly. Based on this, China has formulated a hierarchical medical system for chronic diseases and proposed a community-based chronic disease management plan. However, there are some problems, such as insufficient service resources and unreasonable satisfaction of patients' needs in the actual implementation process. Based on the Kano model, this study analyzes the demands of patients with chronic diseases in the Chinese community at this stage. It matches their existing service subjects according to the priority of demands and then constructs a community-based chronic disease management service model. This study aims to accurately identify the demands of patients with chronic diseases, redistribute and reuse existing facilities and resources, and balance the supply and demand relationship among service subjects and patients. It can provide more humane health management services for chronic disease patients in the community context.
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Rebouças Filho, Pedro Pedrosa, Suane Pires Pinheiro Da Silva, Jefferson Silva Almeida, Elene Firmeza Ohata, Shara Shami Araujo Alves, and Francisco Dos Santos Hercules Silva. "An Approach to Classify Chronic Kidney Diseases using Scintigraphy Images." In XXXII Conference on Graphics, Patterns and Images. Sociedade Brasileira de Computação - SBC, 2019. http://dx.doi.org/10.5753/sibgrapi.est.2019.8318.

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Chronic kidney diseases cause over a million deaths worldwide every year. One of the techniques used to diagnose the diseases is renal scintigraphy. However, the way that is processed can vary depending on hospitals and doctors, compromising the reproducibility of the method. In this context, we propose an approach to process the exam using computer vision and machine learning to classify the stage of chronic kidney disease. An analysis of different features extraction methods, such as Gray-Level Co-occurrence Matrix, Structural Co-occurrence Matrix, Local Binary Patters (LBP), Hu's Moments and Zernike's Moments in combination with machine learning methods, such as Bayes, Multi-layer Perceptron, k-Nearest Neighbors, Random Forest and Support Vector Machines (SVM), was performed. The best result was obtained by combining LBP feature extractor with SVM classifier. This combination achieved accuracy of 92.00% and F1-score of 91.00%, indicating that the proposed method is adequate to classify chronic kidney disease in two stages, being a high risk of developing end-stage renal failure and other outcomes, and otherwise.
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Almansa, Luciana, Gabriel Rubio, and Alessandra Macedo. "A Question Answering System over Chronic Diseases and Epigenetics Knowledge." In Anais Principais do Simpósio Brasileiro de Computação Aplicada à Saúde. Sociedade Brasileira de Computação - SBC, 2020. http://dx.doi.org/10.5753/sbcas.2020.11514.

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Medical records describe patients’ health conditions and help experts to decide on treatments. The scientific biomedical knowledge can improve the prevention and treatment of diseases and promote innovation and discovery in health. However, healthcare professionals may have difficulty in searching for relevant scientific information due to lack time and constant literature update. The present work proposes a Question Answering (Q&A) architecture to support a more focused search for information about chronic diseases. A user question in natural language initiates the search for answering and promoting knowledge such as a learning healthcare system. To evaluate the system, we employ a reference collection on epigenetics and chronic disease and calculate performance measures like precision, recall and F-measure.
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Wang, Yingzi, Xiao Zhou, Anastasios Noulas, Cecilia Mascolo, Xing Xie, and Enhong Chen. "Predicting the Spatio-Temporal Evolution of Chronic Diseases in Population with Human Mobility Data." In Twenty-Seventh International Joint Conference on Artificial Intelligence {IJCAI-18}. California: International Joint Conferences on Artificial Intelligence Organization, 2018. http://dx.doi.org/10.24963/ijcai.2018/497.

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Chronic diseases like cancer and diabetes are major threats to human life. Understanding the distribution and progression of chronic diseases of a population is important in assisting the allocation of medical resources as well as the design of policies in preemptive healthcare. Traditional methods to obtain large scale indicators on population health, e.g., surveys and statistical analysis, can be costly and time-consuming and often lead to a coarse spatio-temporal picture. In this paper, we leverage a dataset describing the human mobility patterns of citizens in a large metropolitan area. By viewing local human lifestyles we predict the evolution rate of several chronic diseases at the level of a city neighborhood. We apply the combination of a collaborative topic modeling (CTM) and a Gaussian mixture method (GMM) to tackle the data sparsity challenge and achieve robust predictions on health conditions simultaneously. Our method enables the analysis and prediction of disease rate evolution at fine spatio-temporal scales and demonstrates the potential of incorporating datasets from mobile web sources to improve population health monitoring. Evaluations using real-world check-in and chronic disease morbidity datasets in the city of London show that the proposed CTM+GMM model outperforms various baseline methods.
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Chaban, Victor V. "Chronic Pain Associated with Functional Diseases." In 6th Annual Global Healthcare Conference (GHC 2017). Global Science & Technology Forum (GSTF), 2017. http://dx.doi.org/10.5176/2251-3833_ghc17.63.

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Ganiger, Shweta, and K. M. M. Rajashekharaiah. "Chronic Diseases Diagnosis using Machine Learning." In 2018 International Conference on Circuits and Systems in Digital Enterprise Technology (ICCSDET). IEEE, 2018. http://dx.doi.org/10.1109/iccsdet.2018.8821235.

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Chen, Chih-Yang, Chun-Nan Chou, and I. Wen Wu. "Toward Personalized Treatment of Chronic Diseases." In MM '17: ACM Multimedia Conference. New York, NY, USA: ACM, 2017. http://dx.doi.org/10.1145/3132635.3132646.

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Roca, J., Diego A. Rodríguez, Francesco Falciani, Peter Aronsson, Esther Barreiro, John Brozek, Jan Brugard, et al. "Systems Medicine In Complex Chronic Diseases: Chronic Obstructive Pulmonary Disease (COPD) As A Use Case." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a5132.

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Uribarri, Jaime. "Exogenous Ages, Microbiota and Their Role in Chronic Diseases." In The 1st International Electronic Conference on Nutrients - Nutritional and Microbiota Effects on Chronic Disease. Basel, Switzerland: MDPI, 2020. http://dx.doi.org/10.3390/iecn2020-06976.

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Farré, R., A. Papadopoulos, G. Munaro, and R. Rosso. "An Open, Ubiquitous and Adaptive Chronic Disease Management Platform for Chronic Respiratory and Renal Diseases (CHRONIOUS)." In 2009 International Conference on eHealth, Telemedicine, and Social Medicine (eTELEMED). IEEE, 2009. http://dx.doi.org/10.1109/etelemed.2009.12.

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Reports on the topic "Chronic diseases"

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Barros-Poblete, Marisol, Rodrigo Torres-Castro, Mauricio Henríquez, Anita Guequen, Isabel Blanco, and Carlos Flores. Dysbiosis as a prognostic factor for clinical worsening in chronic respiratory disease: A systematic review and metanalysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2022. http://dx.doi.org/10.37766/inplasy2022.4.0089.

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Review question / Objective: Is dysbiosis a prognostic factor for clinical worsening in patients with chronic respiratory diseases?. Condition being studied: Dysbiosis, defined as changes in the quantitative and qualitative composition of the microbiota. Eligibility criteria: Over 18 years old adult patients with chronic respiratory diseases clinical diagnosis (cystic fibrosis, chronic obstructive pulmonary disease, asthma, idiopathic pulmonary fibrosis, interstitial lung disease, sarcoidosis, bronchiectasis, non-CF bronchiectasis, pulmonary hypertension) according to the International Statistical Classification of Diseases and Related Health Problems (ICD) from OMS) and international guidelines of each disease.
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Shilova, L. N., and S. S. Spitsina. LIPID EXCHANGE DISORDERS IN PATIENTS WITH CHRONIC INFLAMMATORY JOINT DISEASES. DOI CODE, 2021. http://dx.doi.org/10.18411/wco-iof-esceo-2021-386.

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Lampley, Katrice, and Nicole Therrien. "Geisinger Ambulatory Pharmacy Care Program Field Notes". National Center for Chronic Disease Prevention and Health Promotion (U.S.)., 2023. http://dx.doi.org/10.15620/cdc:126232.

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These Field Notes summarize the Geisinger Ambulatory Care Program’s care coordination work with pharmacists alongside other health care team members to manage chronic diseases including cardiovascular disease.
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Chatterji, Pinka, Heesoo Joo, and Kajal Lahiri. Beware of Unawareness: Racial/Ethnic Disparities in Awareness of Chronic Diseases. Cambridge, MA: National Bureau of Economic Research, December 2010. http://dx.doi.org/10.3386/w16578.

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Gandelman, Néstor. Titling and Chronic Diseases: Evidence from A Natural Experiment in Uruguay. Inter-American Development Bank, December 2008. http://dx.doi.org/10.18235/0011274.

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In this paper the author exploits a natural experiment to study the effects of granting formal property rights on health for households in Uruguay. Titling could improve health outcomes through various channels: housing investment, income or family cohesion. The author found that titling diminishes the probability of suffering several chronic diseases (hypertension, diabetes, sinusitis and rheumatism) but could not find supporting evidence for the housing channel.
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Ly, Lena, Jennifer Philip, Peter Hudson, and Natasha Smallwood. Singing for people with advance chronic respiratory diseases: a qualitative meta-synthesis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, August 2022. http://dx.doi.org/10.37766/inplasy2022.8.0017.

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Review question / Objective: This study undertook a meta-synthesis of qualitative data with the aim of collating, synthesizing, and evaluating the current evidence regarding the experiences of singing for people with advanced chronic respiratory disease. Condition being studied: Advanced respiratory illnesses are disorders that impact the airways and other structures of the lung. People with lung cancer, chronic obstructive pulmonary disease (COPD) and interstitial lung disease (ILD) frequently experience progressive, frightening breathlessness, cough and fatigue, which affect their quality of life. Furthermore, people with advanced chronic respiratory disease (CRD) and their carers experience a high prevalence of loneliness and uncertainty, especially if breathlessness is felt to herald death and thus, require both psychological and practical supportive care to cope with their symptoms.
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Macinko, James, Inês Dourado, and Frederico C. Guanais. Chronic Diseases, Primary Care and Health Systems Performance: Diagnostics, Tools and Interventions. Inter-American Development Bank, November 2011. http://dx.doi.org/10.18235/0007980.

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Growing exposure to risk factors in combination with low levels of access to preventive care are increasing unmet health needs. LAC has been experiencing a "nutrition transition" towards less healthy diets. Thirty to sixty percent of the region's population does not achieve the minimum recommended levels of physical activity and obesity is rising rapidly. Inadequate access to high quality health services, including clinical prevention and diagnostic services and difficult access to essential medicines are significant contributing factors to the growing burden of chronic disease.
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Arrieta, Alejandro, and Ariadna García Prado. Series of Avoidable Hospitalizations and Strengthening Primary Health Care: The Case of Chile. Inter-American Development Bank, December 2012. http://dx.doi.org/10.18235/0006952.

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This paper studies the effect of ambulatory and hospital coinsurance rates on HACSC among individuals with private insurance in Chile. During the last decade, Chile's private health sector has experienced a dramatic increase in its hospitalization rates, growing at four times the rate of ambulatory visits (see graph 1). Such evolution has raised concern among policy makers, interested in promoting more preventive services, and a major use of ambulatory care. The growth on the prevalence of chronic diseases has also set up the alarm. A burden disease study made in 2007 shows that 84% of the total diseases in the country were non-communicable diseases (Universidad Católica de Chile, 2008). The 2003 National Health Survey showed that only a small fraction of those affected by a chronic disease had their condition under control (Bitrán et al, 2010). In this context, coinsurance can be a valuable tool for dealing with cost escalating problems in the health system while, at the same time, promoting more ambulatory visits and preventive services and less HCSC.
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Zhang, Mingzhu, Wujisiguleng Bao, Luying Sun, Zhi Yao, and Xiyao Li. Efficacy and safety of finerenone in chronic kidney disease associated with type 2 diabetes: meta-analysis of randomized clinical trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2022. http://dx.doi.org/10.37766/inplasy2022.3.0020.

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Review question / Objective: To assess the beneficial effect and safety of finerenone for patients with chronic kidney disease associated with type 2 diabetes. Condition being studied: Chronic kidney disease (CKD) is a major contributor to morbidity and mortality from non-communicable diseases, affecting almost 700 million people worldwide. Approximately 40% of patients with diabetes have CKD, which exposes them to a 3-fold higher risk of cardiovascular death versus those with T2D alone. Strategies to protect the kidneys of patients with CKD and T2D may reduce their risk of cardiovascular events. Finerenone, a nonsteroidal, selective mineralocorticoid receptor antagonist, reduced composite kidney and cardiovascular outcome in trials involving patients with chronic kidney disease. Recently, quite a few clinical studies have been conducted to compare finerenone and placebo. Our meta-analysis aimed to investigate the efficacy and safety of finerenone in chronic kidney disease associated with T2D. 1st author* - Mingzhu Zhang and Wujisiguleng Bao contributed equally to this study.
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Liang, R., D. Liu, HB Li, and ZG Zhai. The efficacy and safety of traditional Chinese medicine formulas in the treatment of chronic obstructive pulmonary disease complicated with pulmonary hypertension: a systematic review and meta-analysis study. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, October 2022. http://dx.doi.org/10.37766/inplasy2022.10.0041.

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Review question / Objective: This systematic review and meta-analysis was intended to evaluate the efficacy and safety of traditional Chinese medicine(TCM) formulas in the treatment of chronic obstructive pulmonary disease(COPD) complicated with pulmonaryhypertension (PH). Condition being studied: Chronic obstructive pulmonary disease(COPD) complicated with pulmonary hypertension(PH) is classified as the third group PH.According to epidemiology, the most common cause of PH associated with lung diseases and/or hypoxia is COPD, but the prevalence rate of COPD with PH range from 20% to 91% variously. In China, many TCM formulas are regularly used in COPD patients , thus TCM formulas therapy is worth considering.
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