Journal articles on the topic 'Chronic disease'

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1

S, Memon. "Hyponatremia in Chronic Kidney Disease." Open Access Journal of Urology & Nephrology 7, no. 2 (April 4, 2022): 1–7. http://dx.doi.org/10.23880/oajun-16000202.

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Introduction: There is adequate literature written on hyponatremia, but not much seen in context of Chronic Kidney Disease (CKD). Patients become prone for this electrolyte derangement when they are afflicted with organ failure especially CKD. This vulnerability becomes even worse in ageing due to impaired sensitivity to fluid intake and often burdened with multiple comorbid. For the adequate management of hyponatremia, knowledge of volume status, age of patients and associated comorbid, and duration of hyponatremia are very important. Patients and Methods: This observational study was conducted in adult CKD admitted patients. Demographic information, history, and examination finding were noted. Then each patient underwent investigation i.e., serum sodium, urea, creatinine, spot urine sodium, chloride, potassium, urine and serum osmolality, random blood sugar and echocardiography and noted in questionnaire. CKD staging was done with the help of Modified of diet and Renal disease (MDRD) equation. Diagnosis/ Impression of patient and need of hypertonic saline (3%saline) were all noted along with final outcome whether sodium improved/unimproved, discharge/expired were noted in pre-formed questionnaire. Results: Analysis was done on 171 CKD patients with female to male ratio: 1.19/1 and mean age 55.8 ± 15.16. Hypertension was most prevalent comorbid. Hypovolemia was the most common volume status seen along with moderate hyponatremia and hypertonicity were frequently observed features. 23 out of 171 patients were symptomatic, 44(25.7%) had low Left Ventricular function. Mortality was noted at 9.4%. Conclusion: Overall outcome of patient remained satisfactory despite of presence of CKD and significant number of patients were severe hyponatremic. Predominant management for hyponatremia remained conservative along with treatment for primary disease. Symptomatic hyponatremia and low LV function were found to be contributing to bad outcomes while severity of hyponatremia did not influence badly.
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2

Parmar, Dr Jigar A., Anant G. Joshi, and Dr Manish Chakrabarti. "Dyslipidemia and Chronic Kidney Disease." International Journal of Scientific Research 3, no. 5 (June 1, 2012): 396–97. http://dx.doi.org/10.15373/22778179/may2014/123.

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3

L Pathan, Fayaj. "Nonalcoholic Fatty Liver Disease (NAFLD) A Chronic Liver Disease." Acta Scientifci Nutritional Health 4, no. 1 (December 9, 2019): 58. http://dx.doi.org/10.31080/asnh.2020.04.nonalcoholic-fatty-liver-disease-nafld-a-chronic-liver-disease.

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4

Hague, Andrew. "CellSonic cures chronic disease in a person. Sapiens Shield stops chronic disease in a population." Endocrine System and Diabetes 1, no. 1 (January 26, 2023): 01–03. http://dx.doi.org/10.58489/2836-502x/001.

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All humans and many other species get cancer. It happens daily. The body’s cells replicate on average every six weeks. With billions of cells, some of the new cells fail to be exact copies of the previous cells. The new ones are different and are mutations that we call cancer. Survival of the species depends on removing the wrong cells and this is done by the immune system at night during proper sleep.
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5

singh, Jagroop. "Inflammatory Markers in Chronic Kidney Disease." Journal of Virology and Vaccination 1, no. 1 (February 27, 2023): 01–05. http://dx.doi.org/10.58489/2836-6387/004.

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Background: Despite recent advances in chronic kidney disease (CKD) and end-stage renal disease (ESRD) management, morbidity and mortality in this population remain exceptionally high. Persistent, low-grade inflammation has been recognized as an important component of CKD, playing a unique role in its pathophysiology and being accountable in part for cardiovascular and all-cause mortality, as well as contributing to the development of protein-energy wasting. Material and methods; This study was conducted on CKD patients attending OPD & IPD of Civil Hospital tarn taran. The participants having age more than 18 years & less than 60 years. We assessed 120 individuals out of 60 are normal healthy individuals comprising the control groups & rest 60 is of CKD cases. Serum CRP (mg/dl) concentration was measured by Latex agglutination test & ESR (mm/hour) was measured by Wintrobe’s method. Serum Creatinine (mg/dl), Urea (mg/dl) & Uric acid (mg/dl) concentration was measured by Modified Jaffe’s method, Urease & Uricase method respectively. Results: - In the present study, serum CRP & ESR was increased in CKD patients. The mean serum CRP levels of CKD patients & controls were 33.55 ± 22.8 & 2.07 ± 6121 respectively (p< 0.001), highly significant result was observed. Mean level of serum ESR (40.25 ± 14.93) of cases shows statistical significant differences as compared with the mean of serum ESR of controls (13.50 ± 3.421). Conclusion: - CRP & ESR are the markers used to evaluate kidney disease, however, each of these has its own limitation. The use of these inflammatory biomarkers may better assess overall patients’ risk & be able to stage patients more appropriately.
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6

Mal, Pooran, Muhammad Nadeem Ahsan, Mehwish Bukhari, and Abdul Manan Junejo. "CHRONIC KIDNEY DISEASE." Professional Medical Journal 25, no. 09 (September 9, 2018): 1380–85. http://dx.doi.org/10.29309/tpmj/18.4360.

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7

Kent, Elizabeth. "Chronic Cardiac Disease Chronic Cardiac Disease." Nursing Standard 17, no. 22 (February 12, 2003): 28. http://dx.doi.org/10.7748/ns2003.02.17.22.28.b331.

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8

Fatima, Tanveer, Aurangzeb Afzal, and Sania Ashraf. "CHRONIC KIDNEY DISEASE." Professional Medical Journal 25, no. 06 (June 9, 2018): 887–91. http://dx.doi.org/10.29309/tpmj/18.4418.

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9

Nishi, Shinichi. "Chronic Kidney Disease and Cardiovascular Disease: Progression of Arterial Diseases in Chronic Kidney Disease." Nihon Naika Gakkai Zasshi 105, no. 5 (2016): 791–92. http://dx.doi.org/10.2169/naika.105.791.

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10

TONELLI, Marcello, and Miguel RIELLA. "Chronic Kidney Disease and the Aging Population." Turkish Nephrology Dialysis Transplantation 23, no. 01 (January 21, 2014): 3–7. http://dx.doi.org/10.5262/tndt.2014.1001.02.

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11

Kumar, Santosh. "Chronic Kidney Disease Pre and Post Hemodylasis." International Journal of Science and Research (IJSR) 12, no. 2 (February 5, 2023): 356–58. http://dx.doi.org/10.21275/sr23204172603.

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12

Rakesh, R., Dr G. S. Nishanthi, and Dr S. Kevin Andrews. "Chronic Kidney Disease Cast Using SML Technique." International Journal of Research Publication and Reviews 5, no. 5 (May 2, 2024): 1884–93. http://dx.doi.org/10.55248/gengpi.5.0524.1137.

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13

khatal, Sujata, Priya Nair, and U. B. Shetkar U.B.Shetkar. "Case Report of Acute on Chronic Pancreatitis with Hyperparathyroidism with Chronic Kidney Disease." Indian Journal of Applied Research 4, no. 3 (October 1, 2011): 397–99. http://dx.doi.org/10.15373/2249555x/mar2014/124.

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14

Dale, Marita T., and Mark R. Elkins. "Chronic disease." Journal of Physiotherapy 67, no. 2 (April 2021): 84–86. http://dx.doi.org/10.1016/j.jphys.2021.02.001.

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15

Watkins, Peter. "Chronic disease." Clinical Medicine 4, no. 4 (July 1, 2004): 297–98. http://dx.doi.org/10.7861/clinmedicine.4-4-297.

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16

Beech, Bettina M. "Chronic Disease." Family & Community Health 36, no. 2 (2013): 95–96. http://dx.doi.org/10.1097/fch.0b013e3182867034.

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17

Porche, Demetrius J. "Chronic Disease." American Journal of Men's Health 5, no. 6 (November 2011): 465. http://dx.doi.org/10.1177/1557988311402052.

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18

C Allen, M. Palta T LeCaire, G.-H. Huang, P. Brazy, and D. D'Alessio. "Chronic disease." Annals of Epidemiology 12, no. 7 (October 2002): 498. http://dx.doi.org/10.1016/s1047-2797(02)00310-1.

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19

Russell, Richard, and Jayne Norcliffe. "Chronic obstructive pulmonary disease: management of chronic disease." Medicine 36, no. 4 (April 2008): 218–22. http://dx.doi.org/10.1016/j.mpmed.2008.01.010.

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20

Russell, Richard, Jayne Norcliffe, and Mona Bafadhel. "Chronic obstructive pulmonary disease: management of chronic disease." Medicine 40, no. 5 (May 2012): 262–66. http://dx.doi.org/10.1016/j.mpmed.2012.02.009.

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21

Hambleton, Kirsty, Mona Bafadhel, and Richard Russell. "Chronic obstructive pulmonary disease: management of chronic disease." Medicine 44, no. 5 (May 2016): 310–13. http://dx.doi.org/10.1016/j.mpmed.2016.02.019.

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22

Ramakrishnan, Sanjay, Mona Bafadhel, and Richard Russell. "Chronic obstructive pulmonary disease: management of chronic disease." Medicine 48, no. 5 (May 2020): 333–36. http://dx.doi.org/10.1016/j.mpmed.2020.02.002.

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23

Gluhovschi, Gh, Ligia Petrica, I. Sporea, Manuela Curescu, Silvia Velciov, Cristina Gluhovschi, and R. Timar. "Chronic Kidney Disease – Chronic Liver Disease. An Immunologic Cross-talk." Romanian Journal Of Internal Medicine 53, no. 1 (March 1, 2015): 3–12. http://dx.doi.org/10.1515/rjim-2015-0001.

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Abstract The relationship between the kidney and other organs is notable. The bestknown is the relation with the cardiovascular system. Relationships with other organs are less studied, although their involvement sometimes dominates the clinical picture and the outcome of disease. The paper analyzes the kidney-liver relationship, namely chronic kidney disease and chronic liver disease from an immune viewpoint. The immune system operates as a unitary whole. There is an interdependence between the immune system of the liver, considered a lymphoid organ, and the kidney, whose participation in immune processes is well-known. The most important chronic liver diseases are viral hepatitis B and C. Infection with these viruses can lead to renal involvement, producing mainly glomerular disease. At the same time, secondary glomerulonephritis can cause an unfavorable outcome of the primary disease. The relationship between chronic liver disease and chronic kidney disease during chronic B and C hepatitis occurs via circulating immune complexes or complexes formed in situ. Cell-mediated immunity is also involved. The antiviral treatment of B and C hepatitis is also aimed at secondary glomerular disease. The participation of immune mechanisms raises the question of administering immunomodulating medication, a type of medication that influences viral replication - this is why it is associated with antiviral medication. Other two chronic liver diseases, namely liver cirrhosis, in which the main mechanism is a toxic one, and non-alcoholic steatohepatitis can produce via immune mechanisms glomerular involvement. In its turn, chronic kidney disease in advanced stages causes lipid metabolism disturbances with hypertriglyceridemia, which can influence fatty loading of the liver in the above-mentioned liver diseases. One can speak about a cross-talk between the liver and the kidney, in which immune mechanisms play an important role.
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24

M S, Champa, and Rekha B S. "Chronic Kidney Disease Detection Using Micro-service Architecture." International Journal of Innovative Research in Computer Science & Technology 7, no. 3 (May 2019): 84–89. http://dx.doi.org/10.21276/ijircst.2019.7.3.10.

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25

Shivraj, Shivraj, and Venugopal K. "Gastrointestinal Manifestations in Patients with Chronic Kidney Disease." Academia Journal of Medicine 2, no. 2 (July 24, 2019): 54–59. http://dx.doi.org/10.21276/ajm.2019.2.2.15.

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26

VN, Karan, and Vishwanath VN. "Clinical Profile of Patients with Chronic Kidney Disease." Academia Journal of Medicine 2, no. 2 (July 24, 2019): 175–78. http://dx.doi.org/10.21276/ajm.2019.2.2.45.

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27

(Medicine), Dr Ashok Kumar M. D. "Observation on Serum Magnesium in Chronic Kidney Disease." Journal of Medical Science And clinical Research 05, no. 05 (June 2, 2017): 20570–72. http://dx.doi.org/10.18535/jmscr/v5i5.211.

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28

Ramraje, N. N. "Study of OSA in Chronic Obstructive Pulmonary Disease." Journal of Medical Science And clinical Research 05, no. 05 (May 12, 2017): 21712–14. http://dx.doi.org/10.18535/jmscr/v5i5.78.

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29

Tokgöz Akyıl, Fatma, Tülin Sevim, Safa Barış, Emine Aksoy, Dilem Anıl Tokyay, Yasemin Bodur, and Oğuz Aktaş. "Two Chronic Granulomatous Disease Diagnosed in Adult Age." Turkish Thoracic Journal 17, no. 4 (October 10, 2016): 163–67. http://dx.doi.org/10.5578/ttj.30515.

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30

Peco-Antić, Amira. "How to grow up with chronic kidney disease?" Central European Journal of Paediatrics 14, no. 1 (March 7, 2018): 37–46. http://dx.doi.org/10.5457/p2005-114.197.

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31

G.K., Rakhmatullayeva, Artykova M.A., and Khudayarova S.M. "NEUROIMAGING CHARACTERISTICS OF ENCEPHALOPATHY IN CHRONIC KIDNEY DISEASE." American Journal of Medical Sciences and Pharmaceutical Research 05, no. 05 (May 1, 2023): 24–29. http://dx.doi.org/10.37547/tajmspr/volume05issue05-07.

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According to WHO, vascular diseases of the brain in the developed countries of the world account for 15% of the total mortality. At the same time, acute and chronic encephalopathies occupy a special place in the structure of vascular diseases. In recent years, special attention has been paid to encephalopathies developing as a result of renal pathology, since kidney diseases, namely chronic kidney disease (CKD) is a huge socio-economic problem that leads to cerebral complications, with a significant deterioration in the quality of life, a sharp loss of working capacity, and a high percentage of mortality. In the diagnosis of various encephalopathies and other disorders of cerebral circulation, an invaluable role is played by MRI, MSCT, brain angiography with their wide diagnostic capabilities.
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32

..., Vishakha, Pragya Sinha, Bandhu Sharma, and S. P. Subashini. "Efficacy of Growth Hormone in Chronic Kidney Disease." Indian Journal of Genetics and Molecular Research 11, no. 1 (June 15, 2022): 29–30. http://dx.doi.org/10.21088/ijgmr.2319.4782.11122.3.

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Children with chronic kidney disease (CKD) have difficulty achieving normal growth due to severe alterations in the growth hormone (GH) and insulin-like growth factor (IGF-1) axis. Recombinant human growth hormone (rhGH) therapy (GH). It encourages long-term growth and allows children with CKD to reach normal height.It has been demonstrated to be both safe and effective. After a kidney transplant, growth retardation may remain. Multiple variables, such as steroid use, impaired renal function, and an aberrant GH axis, have contributed to this condition.
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33

Amrelevich, Akhmedov Ibrat. "CHRONIC KIDNEY DISEASE IN PATIENTS WITH ARTERIAL HYPERTENSION." American Journal of Medical Sciences and Pharmaceutical Research 6, no. 5 (May 1, 2024): 15–17. http://dx.doi.org/10.37547/tajmspr/volume06issue05-04.

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The article presents the results of assessing the state of kidney function in patients with arterial hypertension. The study used a method for calculating the glomerular filtration rate according to the expanded formula for determining creatinine in the blood.
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34

Parfenov, V. A. "Ménière’s disease and chronic cerebrovascular diseases." Meditsinskiy sovet = Medical Council, no. 19 (December 1, 2021): 35–40. http://dx.doi.org/10.21518/2079-701x-2021-19-35-40.

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Ménière’s disease (MD) is an idiopathic inner ear disease, which is characterized by noise in the ears, periodic attacks of vertigo and the development of sensorineural hearing loss. MD is characterized by endolymphatic hydrops – an increase in the volume of endolymph that fills the membered labyrinth of the inner ear. Currently highlighted subtypes MD. The first subtype meets most often and is characterized by classical manifestations of MD. The second subtype is characterized by the development of sensorineural hearing loss, to which only after a long time are joined by the episodes of dizziness. The third subtype of MD includes family cases of the disease. The fourth and fifth subtypes of MD is observed in patients with migraine and autoimmune diseases. The diagnosis of definite MD is based on the 2 or more spontaneous episodes of vertigo with each lasting 20 minutes to 12 hours, low-to medium-frequency sensorineural hearing loss in one ear, fluctuating aural symptoms (fullness, hearing, tinnitus) located in the affected ear, and lack of data for other reasons for dizziness. There are no effective treatment for auditory disorders MD, therapy is aimed at preventing dizziness attacks. The first line of MD’s therapy includes a dietary salt restriction, the use of betahistine and diuretics. Betahistine (Betaserc) is usually used in a daily dose of 48 mg for 3–6 months to reduce the frequency of vertigo. For long-term treatment, it is convenient to use a betahistine modified-released (Betaserc Long) 48 mg, taken once a day. With the ineffectiveness of conservative therapy, other methods of therapy are possible: intratympanic administration of corticosteroids or gentamicin, labyrinthectomy or vestibular neurectomy. Unfortunately, many patients suffering from BM mistakenly makes a diagnosis of cerebrovascular disease, vertebrobasilar insufficiency, cervical osteochondrosis. Diagnostic errors are usually caused by the fact that the patients with MD are not conducted audiometry, vestibular tests, and the signs of cerebral microangiopathy identified when MRI brain are mistakenly regarded as confirmation of vascular dizziness genesis.
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35

Sanai, Toru, Ken Okamura, Tsuyoshi Takashima, Motoaki Miyazono, and Yuji Ikeda. "Chronic kidney disease and thyroid diseases." Nihon Toseki Igakkai Zasshi 52, no. 11 (2019): 615–23. http://dx.doi.org/10.4009/jsdt.52.615.

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36

FAIVRE, M., and S. LACAN. "Bronchopneumopathie chronique obstructive." EXERCER 35, no. 199 (January 1, 2024): 38–40. http://dx.doi.org/10.56746/exercer.2024.199.38.

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Les recommandations internationales concernant la prise en charge de la bronchopneumopathie chronique obstructive (BPCO) ont été mises à jour par la Global Initiative for Chronic Obstructive Lung Disease (GOLD) en 20231. Cet article revient sur les modifications apportées par rapport à celles de 2022.
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37

Galil, A. G. D. S., B. A. B. B. Andrade, M. M. Gusmão, L. R. Araújo, M. M. Mello, A. S. Gomes, M. G. Bastos, and E. F. C. Banhato. "PO393 Chronic Kidney Disease in Smokers with Multiple Chronic Diseases." Global Heart 13, no. 4 (December 2018): 461. http://dx.doi.org/10.1016/j.gheart.2018.09.303.

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38

Betancourt, M. T., K. C. Roberts, T.-L. Bennett, E. R. Driscoll, G. Jayaraman, and L. Pelletier. "Monitoring chronic diseases in Canada: the Chronic Disease Indicator Framework." Chronic Diseases and Injuries in Canada 34, Supplement 1 (June 2014): 1–30. http://dx.doi.org/10.24095/hpcdp.34.s1.01.

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39

van Eeden, Stephan F., and Don D. Sin. "Chronic Obstructive Pulmonary Disease: A Chronic Systemic Inflammatory Disease." Respiration 75, no. 2 (November 28, 2007): 224–38. http://dx.doi.org/10.1159/000111820.

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40

Rathore, Hemlata, Brijesh Rathore, and Seema Singh. "RHEUMATOID ARTHRITIS: A CHRONIC INFLAMMATORY DISEASE AND ITS PATHOPHYSIOLOGY." Era's Journal of Medical Research 6, no. 1 (June 2019): 88–92. http://dx.doi.org/10.24041/ejmr2019.113.

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41

Herald, G. Peter Praveen, and H. Krishna Murthy. "EVALUATION OF PULMONARY HYPERTENSION IN CHRONIC OBSTRUCTIVE PULMONARY DISEASE." International Journal of Integrative Medical Sciences 6, no. 1 (February 20, 2019): 765–68. http://dx.doi.org/10.16965/ijims.2019.102.

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42

Vasylchenko, V. S., L. V. Korol, O. B. Kuchmenko, and N. M. Stepanova. "The oxidative status in patients with chronic kidney disease." Ukrainian Biochemical Journal 92, no. 5 (November 13, 2020): 70–77. http://dx.doi.org/10.15407/ubj92.05.070.

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43

Bhardwaj, Diwakar, and Anjani Rai. "Heterogeneous Clustering Network Chronic Kidney Disease Progression Mining (HCNCKPM)." Journal of Advanced Research in Dynamical and Control Systems 11, no. 11-SPECIAL ISSUE (November 29, 2019): 215–22. http://dx.doi.org/10.5373/jardcs/v11sp11/20192950.

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44

Madhavi Sarkari, Sushant. "Assessment of Malnutrition in Patients with Chronic Liver Disease." International Journal of Science and Research (IJSR) 12, no. 5 (May 5, 2023): 2287–93. http://dx.doi.org/10.21275/sr23520160136.

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45

H Mali Tejaswini, Basavaraj. "Left Ventricular Functions in Patients with Chronic Kidney Disease." International Journal of Science and Research (IJSR) 12, no. 7 (July 5, 2023): 1130–32. http://dx.doi.org/10.21275/mr23716144000.

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46

Chen, Jacqueline, and Ria Patel. "Chronic Kidney Disease." Meducator 1, no. 44 (January 15, 2024): 20–21. http://dx.doi.org/10.15173/m.v1i44.3620.

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Chronic kidney disease (CKD) is defined as low filtration function in the kidneys, protein in the urine, or functionally-important structural abnormalities. Low filtration function is associated with waste build-up in the bloodstream and difficulty in excreting salt and water, leading to fluid build-up. CKD may worsen over time, and some individuals with CKD may experience kidney failure, at which point dialysis or transplantation is required to replace kidney function. CKD may be caused by diabetes, renovascular disease, glomerulonephritis, polycystic kidney disease, and various genetic and environmental factors. In 2023, approximately four million Canadians live with and 11-13% of the global population are affected by CKD. This disorder has a 50% five-year mortality rate and is associated with lower quality of life compared to other chronic diseases, including sickle cell anemia, cancer, and cystic fibrosis. CKD’s comorbidities, such as diabetes, are increasing in prevalence and can increase the risk of developing CKD. Thus, the rates of CKD are also set to rise.3 Annual CKD management costs across Canada total $40 billion, with dialysis treatment for people with end-stage kidney disease (ESKD) costing $100,000 per patient.
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47

Bu, Jia, and Cui-Ping Xu. "Chronic constipation and chronic disease management." World Chinese Journal of Digestology 24, no. 30 (2016): 4162. http://dx.doi.org/10.11569/wcjd.v24.i30.4162.

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48

Gálvez, Alyshia, Megan Carney, and Emily Yates‐Doerr. "Chronic Disaster: Reimagining Noncommunicable Chronic Disease." American Anthropologist 122, no. 3 (August 24, 2020): 639–40. http://dx.doi.org/10.1111/aman.13437.

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49

Ball, Timothy, Kevin Wheelan, and Peter A. McCullough. "Chronic Anticoagulation in Chronic Kidney Disease∗." Journal of the American College of Cardiology 64, no. 23 (December 2014): 2483–85. http://dx.doi.org/10.1016/j.jacc.2014.09.052.

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50

Zainudeen, Zarina Thasneem, Ilie Fadzilah Hashim, and Intan Juliana Abd Hamid. "Chronic Granulomatous Disease." Malaysian Journal of Paediatrics and Child Health 25, no. 2 (February 4, 2020): 28–29. http://dx.doi.org/10.51407/mjpch.v25i2.17.

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