Relevant bibliographies by topics / Chronic disease

Academic literature on the topic 'Chronic disease'

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Published: 4 June 2021
Last updated: 31 August 2024

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Journal articles on the topic "Chronic disease"

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S, Memon. "Hyponatremia in Chronic Kidney Disease." Open Access Journal of Urology & Nephrology 7, no. 2 (April 4, 2022): 1–7. http://dx.doi.org/10.23880/oajun-16000202.

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Introduction: There is adequate literature written on hyponatremia, but not much seen in context of Chronic Kidney Disease (CKD). Patients become prone for this electrolyte derangement when they are afflicted with organ failure especially CKD. This vulnerability becomes even worse in ageing due to impaired sensitivity to fluid intake and often burdened with multiple comorbid. For the adequate management of hyponatremia, knowledge of volume status, age of patients and associated comorbid, and duration of hyponatremia are very important. Patients and Methods: This observational study was conducted in adult CKD admitted patients. Demographic information, history, and examination finding were noted. Then each patient underwent investigation i.e., serum sodium, urea, creatinine, spot urine sodium, chloride, potassium, urine and serum osmolality, random blood sugar and echocardiography and noted in questionnaire. CKD staging was done with the help of Modified of diet and Renal disease (MDRD) equation. Diagnosis/ Impression of patient and need of hypertonic saline (3%saline) were all noted along with final outcome whether sodium improved/unimproved, discharge/expired were noted in pre-formed questionnaire. Results: Analysis was done on 171 CKD patients with female to male ratio: 1.19/1 and mean age 55.8 ± 15.16. Hypertension was most prevalent comorbid. Hypovolemia was the most common volume status seen along with moderate hyponatremia and hypertonicity were frequently observed features. 23 out of 171 patients were symptomatic, 44(25.7%) had low Left Ventricular function. Mortality was noted at 9.4%. Conclusion: Overall outcome of patient remained satisfactory despite of presence of CKD and significant number of patients were severe hyponatremic. Predominant management for hyponatremia remained conservative along with treatment for primary disease. Symptomatic hyponatremia and low LV function were found to be contributing to bad outcomes while severity of hyponatremia did not influence badly.
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Parmar, Dr Jigar A., Anant G. Joshi, and Dr Manish Chakrabarti. "Dyslipidemia and Chronic Kidney Disease." International Journal of Scientific Research 3, no. 5 (June 1, 2012): 396–97. http://dx.doi.org/10.15373/22778179/may2014/123.

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L Pathan, Fayaj. "Nonalcoholic Fatty Liver Disease (NAFLD) A Chronic Liver Disease." Acta Scientifci Nutritional Health 4, no. 1 (December 9, 2019): 58. http://dx.doi.org/10.31080/asnh.2020.04.nonalcoholic-fatty-liver-disease-nafld-a-chronic-liver-disease.

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Hague, Andrew. "CellSonic cures chronic disease in a person. Sapiens Shield stops chronic disease in a population." Endocrine System and Diabetes 1, no. 1 (January 26, 2023): 01–03. http://dx.doi.org/10.58489/2836-502x/001.

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All humans and many other species get cancer. It happens daily. The body’s cells replicate on average every six weeks. With billions of cells, some of the new cells fail to be exact copies of the previous cells. The new ones are different and are mutations that we call cancer. Survival of the species depends on removing the wrong cells and this is done by the immune system at night during proper sleep.
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singh, Jagroop. "Inflammatory Markers in Chronic Kidney Disease." Journal of Virology and Vaccination 1, no. 1 (February 27, 2023): 01–05. http://dx.doi.org/10.58489/2836-6387/004.

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Background: Despite recent advances in chronic kidney disease (CKD) and end-stage renal disease (ESRD) management, morbidity and mortality in this population remain exceptionally high. Persistent, low-grade inflammation has been recognized as an important component of CKD, playing a unique role in its pathophysiology and being accountable in part for cardiovascular and all-cause mortality, as well as contributing to the development of protein-energy wasting. Material and methods; This study was conducted on CKD patients attending OPD & IPD of Civil Hospital tarn taran. The participants having age more than 18 years & less than 60 years. We assessed 120 individuals out of 60 are normal healthy individuals comprising the control groups & rest 60 is of CKD cases. Serum CRP (mg/dl) concentration was measured by Latex agglutination test & ESR (mm/hour) was measured by Wintrobe’s method. Serum Creatinine (mg/dl), Urea (mg/dl) & Uric acid (mg/dl) concentration was measured by Modified Jaffe’s method, Urease & Uricase method respectively. Results: - In the present study, serum CRP & ESR was increased in CKD patients. The mean serum CRP levels of CKD patients & controls were 33.55 ± 22.8 & 2.07 ± 6121 respectively (p< 0.001), highly significant result was observed. Mean level of serum ESR (40.25 ± 14.93) of cases shows statistical significant differences as compared with the mean of serum ESR of controls (13.50 ± 3.421). Conclusion: - CRP & ESR are the markers used to evaluate kidney disease, however, each of these has its own limitation. The use of these inflammatory biomarkers may better assess overall patients’ risk & be able to stage patients more appropriately.
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Mal, Pooran, Muhammad Nadeem Ahsan, Mehwish Bukhari, and Abdul Manan Junejo. "CHRONIC KIDNEY DISEASE." Professional Medical Journal 25, no. 09 (September 9, 2018): 1380–85. http://dx.doi.org/10.29309/tpmj/18.4360.

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Kent, Elizabeth. "Chronic Cardiac Disease Chronic Cardiac Disease." Nursing Standard 17, no. 22 (February 12, 2003): 28. http://dx.doi.org/10.7748/ns2003.02.17.22.28.b331.

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Fatima, Tanveer, Aurangzeb Afzal, and Sania Ashraf. "CHRONIC KIDNEY DISEASE." Professional Medical Journal 25, no. 06 (June 9, 2018): 887–91. http://dx.doi.org/10.29309/tpmj/18.4418.

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Nishi, Shinichi. "Chronic Kidney Disease and Cardiovascular Disease: Progression of Arterial Diseases in Chronic Kidney Disease." Nihon Naika Gakkai Zasshi 105, no. 5 (2016): 791–92. http://dx.doi.org/10.2169/naika.105.791.

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TONELLI, Marcello, and Miguel RIELLA. "Chronic Kidney Disease and the Aging Population." Turkish Nephrology Dialysis Transplantation 23, no. 01 (January 21, 2014): 3–7. http://dx.doi.org/10.5262/tndt.2014.1001.02.

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Dissertations / Theses on the topic "Chronic disease"

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Williams, Stacey L., and E. Fekete. "Chronic Disease." Digital Commons @ East Tennessee State University, 2012. https://dc.etsu.edu/etsu-works/8145.

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Jaishankar, Gayatri. "Chronic Granulomatous Disease." Digital Commons @ East Tennessee State University, 2009. https://dc.etsu.edu/etsu-works/8869.

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Ye, Ping. "Autoimmunity in chronic periodontitis." Thesis, The University of Sydney, 2003. http://hdl.handle.net/2123/4256.

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Profound perturbation of epithelial structure is a characteristic feature of the immunopatholoical response to bacterial antigens considered to be central in the pathogenesis of the destructive lesion of periodontitis. The pathological basis for the disturbance of epithelial structure is not understood. It was demonstrated that the structural integrity and functional differentiation of the lining epithelium is compromised in relation to inflammatory changes associated with destructive periodontitis. In the pathological lining epithelium of the periodontal pocket there was a marked reduction of epithelial cadherin important in intercellular adhesion, of involucrin, a marker of terminal differentiation, and of the gap junction connexions that form intercellular communication channels. These changes were associated with alterations of filamentous actin expression, collectively indicating profound perturbation of epithelial structure. The data reported support the concept that the ability of the pathological lining epithelium to function as an effective barrier against the ingress of microbial products into the tissues is severely compromised (Ye et al., 2000). In addition, a recent study (Ye et al., 2003) by Western analysis of serum IgG from all 22 patients with chronic periodontitis tested indicated recognition of multiple epithelial components in individual patterns. In contrast, subjects with a healthy periodontium displayed only trace recognition of epithelial antigens. Levels of epithelial-reactive antibodies were significantly correlated with attachment loss as an indication of disease activity. To investigate a possible relationship between the bacterial flora adjacent to the diseased sites and the presence of epithelial-reactive antibodies, subgingival plague samples were taken from deep periodontal pockets and cultured anaerobically. Gram positive bacteria containing antigens potentially cross-reactive with epithelial cells were reproducibly isolated by probing membrane colony lifts with affinity-isolated (epitheial-specific) antibodies. The bacteria were identified as streptococci (S. mitis, S. constellatus and two S. intermedius strains) and Actinomyces (A. georgiae, and A. sp. oral clone) by 16S rDNA sequence homology. Recognition by affinity-isolated antibodies of antigens from the captured organisms was confirmed by Western analysis. Conversely, absorption of affinity-isolated antibodies with bacterial species specifically reduced subsequent recognition of epithelial antigens. To identify the auto-antigens, a human keratinocyte cDNA expression library in Lambda phage was probed using a pooled sera. Groups of responders were detected for CD24 (a recently described adhesion molecule also known as P-selectin ligand), antioxidant protein 2 (a newly recognised member of the thiol-dependment anti-oxidant proteins), lavtate dehydrogenase A, the transcription factor NFAT5, and for three genes encoding novel proteins. Six identified bacteria, especially S intermedius were demonstrated to absorb antibodies reaching with identified auto-antigens in patterns varying between individuals. This evidence indicated that during the course of periodontits, subjects develop increased levels of antibodies to common oral bacteria amongst which are included tissue cross-reactive antigens. Periodontitis could therefore present a risk for the subsequent initiation or exacerbation of a broad spectrum of disease processes including autoimmune, inflammatory, proliferative and degenerative disorders.
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Ye, Ping. "Autoimmunity in chronic periodontitis." University of Sydney, 2003. http://hdl.handle.net/2123/4256.

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Doctor of Philosophy
Profound perturbation of epithelial structure is a characteristic feature of the immunopatholoical response to bacterial antigens considered to be central in the pathogenesis of the destructive lesion of periodontitis. The pathological basis for the disturbance of epithelial structure is not understood. It was demonstrated that the structural integrity and functional differentiation of the lining epithelium is compromised in relation to inflammatory changes associated with destructive periodontitis. In the pathological lining epithelium of the periodontal pocket there was a marked reduction of epithelial cadherin important in intercellular adhesion, of involucrin, a marker of terminal differentiation, and of the gap junction connexions that form intercellular communication channels. These changes were associated with alterations of filamentous actin expression, collectively indicating profound perturbation of epithelial structure. The data reported support the concept that the ability of the pathological lining epithelium to function as an effective barrier against the ingress of microbial products into the tissues is severely compromised (Ye et al., 2000). In addition, a recent study (Ye et al., 2003) by Western analysis of serum IgG from all 22 patients with chronic periodontitis tested indicated recognition of multiple epithelial components in individual patterns. In contrast, subjects with a healthy periodontium displayed only trace recognition of epithelial antigens. Levels of epithelial-reactive antibodies were significantly correlated with attachment loss as an indication of disease activity. To investigate a possible relationship between the bacterial flora adjacent to the diseased sites and the presence of epithelial-reactive antibodies, subgingival plague samples were taken from deep periodontal pockets and cultured anaerobically. Gram positive bacteria containing antigens potentially cross-reactive with epithelial cells were reproducibly isolated by probing membrane colony lifts with affinity-isolated (epitheial-specific) antibodies. The bacteria were identified as streptococci (S. mitis, S. constellatus and two S. intermedius strains) and Actinomyces (A. georgiae, and A. sp. oral clone) by 16S rDNA sequence homology. Recognition by affinity-isolated antibodies of antigens from the captured organisms was confirmed by Western analysis. Conversely, absorption of affinity-isolated antibodies with bacterial species specifically reduced subsequent recognition of epithelial antigens. To identify the auto-antigens, a human keratinocyte cDNA expression library in Lambda phage was probed using a pooled sera. Groups of responders were detected for CD24 (a recently described adhesion molecule also known as P-selectin ligand), antioxidant protein 2 (a newly recognised member of the thiol-dependment anti-oxidant proteins), lavtate dehydrogenase A, the transcription factor NFAT5, and for three genes encoding novel proteins. Six identified bacteria, especially S intermedius were demonstrated to absorb antibodies reaching with identified auto-antigens in patterns varying between individuals. This evidence indicated that during the course of periodontits, subjects develop increased levels of antibodies to common oral bacteria amongst which are included tissue cross-reactive antigens. Periodontitis could therefore present a risk for the subsequent initiation or exacerbation of a broad spectrum of disease processes including autoimmune, inflammatory, proliferative and degenerative disorders.
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Philips, L. G. "Disease management in chronic kidney disease /." abstract and full text PDF (free order & download UNR users only), 2005. http://0-wwwlib.umi.com.innopac.library.unr.edu/dissertations/fullcit/1430446.

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Thesis (M.B.A.)--University of Nevada, Reno, 2005.
"May, 2005." Includes bibliographical references (leaves 92-97). Online version available on the World Wide Web. Library also has microfilm. Ann Arbor, Mich. : ProQuest Information and Learning Company, [2005]. 1 microfilm reel ; 35 mm.
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Ye, Ping. "Autoimmunity In Chronic Periodontitis." Thesis, The University of Sydney, 2003. http://hdl.handle.net/2123/4872.

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Ormarsdóttir, Sif. "Osteoporosis in chronic liver disease." Doctoral thesis, Uppsala University, Department of Medical Sciences, 2001. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-660.

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Ormarsdóttir, S. 2001. Osteoporosis in Chronic Liver Disease. Acta Universitatis Upsaliensis. Comprehensive Summaries of Uppsala Dissertations from the Faculty of Medicine 1037. 60 pp. Uppsala. ISBN 91-554-5021-0.

Osteoporosis is a well-known and frequently reported complication of chronic liver disease (CLD) with a high fracture rate contributing to significant morbidity after liver transplantation. The pathogenesis is unknown and controversy exists about many risk factors for osteoporosis in CLD.

In the present thesis, bone mineral density (BMD) was found to be significantly lower at the lumbar spine (p<0.01) in a cohort of patients with CLD compared with age- and gender -matched individuals. Osteoporosis was found in 30% of the patients and 15% of the controls, respectively. Low body mass index (BMI), corticosteroid treatment, prothrombin time, age and female gender were independent risk factors for osteoporosis in the patients.

In a follow-up study, 43 of 72 patients were available for a second BMD measurement 25 months (median) after the first. Bone loss at the femoral neck was 1.5 ± 2.4% in females and 2.9 ± 2.0% in males with a significant decrease in BMD Z-score over time (p=0.005 and p=0.02 for females and males, respectively), indicating increased bone loss at this site. Hyperbilirubinaemia and low circulating levels of 25-hydroxy vitamin D3 predicted increased bone loss at the femoral neck. These findings suggest that cortical bone, in addition to trabecular bone, may be affected in CLD and bilirubin and vitamin D3 may be involved in the pathophysiology of osteoporosis in CLD.

In order to elucidate the suggested role of insulin-like growth factors (IGFs) and leptin in the pathophysiology of osteoporosis in CLD, we studied the relationship between these factors and BMD. Levels of IGFs were extremely low (p<0.0001 compared with the controls) and related to liver function but no correlation was found between the IGFs and BMD. Serum leptin adjusted for BMI correlated negatively with BMD in female patients (p=0.003 and p=0.04 at the lumbar spine and the femoral neck, respectively) and in male patients at the femoral neck (p=0.04). Thus, the IGFs appear not to be involved in the pathophysiology of osteoporosis in CLD but a role of circulating leptin is possible.

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Ormarsdóttir, Sif. "Osteoporosis in chronic liver disease /." Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2001. http://publications.uu.se/theses/91-554-5021-0/.

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Begbuna, Veronica. "Haemodynamics in chronic venous disease." Thesis, Imperial College London, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.401816.

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Leslie, Wilma S. "Weight management and chronic disease." Thesis, University of Glasgow, 2009. http://theses.gla.ac.uk/1300/.

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Abstract Background: Obesity, in addition to being a serious condition in its own right, is causally associated with many chronic non-communicable diseases, and its prevention, identification and treatment is a public health priority. Results: The main findings of the present thesis were that 1) many drugs, used in the management of chronic disease, have an adverse effect on body weight with weight change of +10kg observed as a real side effect of some. 2) Identification and management of obesity is not a formal part of current practice in many secondary care clinics. While acknowledging the adverse health effects of obesity within their specialist areas, clinicians felt under-skilled and insufficiently resourced to provide effective management. 3) Improvements in iron status in pre-menopausal women can be achieved during weight loss, using eating plans that either include or exclude red meat. The data while in-conclusive suggest that a diet including red meat may confer greater benefits on iron status. Discussion: Weight gain is an adverse effect of many drugs used to treat chronic diseases. This should be discussed with patients prior to treatment and advice provided on how to avoid or minimise weight gain. NHS secondary care consultants are concerned about obesity and its impact on their patient’s health. Most have no weight management strategy and would like one. This will require additional training and resources. Excluding red meat did not adversely affect iron status in pre-menopausal women. A larger study is required for definitive health promotion advice. Conclusion: Pharmacotherapy is a significant factor in the rising prevalence of obesity. Weight management is not an integral part of patient care in secondary care clinic settings. The exclusion of red meat during weight management does not compromise iron status in pre-menopausal women with low iron stores.
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Books on the topic "Chronic disease"

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Yang, Junwei, and Weichun He, eds. Chronic Kidney Disease. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-32-9131-7.

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Nuovo, Jim. Chronic Disease Management. New York, NY: Springer New York, 2007. http://dx.doi.org/10.1007/978-0-387-49369-5.

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Cummings, Nancy Boucot, and Saulo Klahr, eds. Chronic Renal Disease. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-4826-9.

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Washington (State). Dept. of Health., ed. Chronic disease prevention. Olympia, Wash: The Department, 1994.

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Washington (State). Dept. of Fish and Wildlife., ed. Chronic wasting disease. [Olympia, Wash.]: Washington Dept. of Fish and Wildlife, 2000.

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Wildlife, Ohio Division of, ed. Chronic wasting disease. [Columbus, Ohio?]: Division of Wildlife, Ohio Dept. of Natural Resources, 2005.

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Oliver, Susan. Chronic Disease Nursing. New York: John Wiley & Sons, Ltd., 2006.

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Washington (State). Dept. of Fish and Wildlife., ed. Chronic wasting disease. [Olympia, Wash.]: The Dept., 2000.

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S, Cappell Mitchell, ed. Chronic kidney disease. Philadelphia: Saunders, 2005.

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Wildlife, Ohio Division of, ed. Chronic wasting disease. [Columbus, Ohio?]: Division of Wildlife, Ohio Dept. of Natural Resources, 2007.

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Book chapters on the topic "Chronic disease"

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Fekete, Erin M., and Stacey L. Williams. "Chronic Disease." In Encyclopedia of Immigrant Health, 432–38. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-5659-0_145.

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Gooch, Jan W. "Chronic Disease." In Encyclopedic Dictionary of Polymers, 882. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_13394.

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Schmidt, Harald H. H. W. "Chronic Disease?" In The end of medicine as we know it - and why your health has a future, 31–38. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-95293-8_3.

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Wu, Jining, Wenjin Liu, and Hongdi Cao. "Cardiovascular Disease in Chronic Kidney Disease." In Chronic Kidney Disease, 111–21. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-32-9131-7_9.

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Fry, John, Gerald Sandler, and David Brooks. "Chronic Bronchitis." In Disease Data Book, 88–111. Dordrecht: Springer Netherlands, 1986. http://dx.doi.org/10.1007/978-94-009-4149-6_5.

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Nuovo, Jim. "Chronic Pain." In Chronic Disease Management, 346–58. New York, NY: Springer New York, 2007. http://dx.doi.org/10.1007/978-0-387-49369-5_13.

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Hawkins, Alexander T., and Liliana Bordeianou. "Chronic Anal Pain." In Anorectal Disease, 243–62. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-23147-1_11.

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Meurer, Lindsay, and Anthony Post. "Chronic Hepatitis B." In Liver Disease, 61–73. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-98506-0_6.

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Cohen, Stanley Martin. "Chronic Hepatitis C." In Liver Disease, 75–87. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-98506-0_7.

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Cai, Ting, and Junwei Yang. "Diabetic Kidney Disease." In Chronic Kidney Disease, 33–43. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-32-9131-7_3.

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Conference papers on the topic "Chronic disease"

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Allam, Zyad, Reem Jamal, Fathan Shajrah, and Sawsan Hilal. "Chronic Kidney Disease Detection: A Comparative study." In 2023 4th International Conference on Data Analytics for Business and Industry (ICDABI), 18–22. IEEE, 2023. http://dx.doi.org/10.1109/icdabi60145.2023.10629400.

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Liu, Renxuan, and Duan Wu. "Re-establishing the balance: A New Community-based Chronic Disease Management Service Model in China." In 14th International Conference on Applied Human Factors and Ergonomics (AHFE 2023). AHFE International, 2023. http://dx.doi.org/10.54941/ahfe1003492.

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As China's aging process accelerates, chronic diseases such as diabetes and high blood pressure gradually become hidden dangers that endanger the health of the elderly. Based on this, China has formulated a hierarchical medical system for chronic diseases and proposed a community-based chronic disease management plan. However, there are some problems, such as insufficient service resources and unreasonable satisfaction of patients' needs in the actual implementation process. Based on the Kano model, this study analyzes the demands of patients with chronic diseases in the Chinese community at this stage. It matches their existing service subjects according to the priority of demands and then constructs a community-based chronic disease management service model. This study aims to accurately identify the demands of patients with chronic diseases, redistribute and reuse existing facilities and resources, and balance the supply and demand relationship among service subjects and patients. It can provide more humane health management services for chronic disease patients in the community context.
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Periyasamy, Kasi, and Venkateswaran Iyer. "Chronic Kidney Disease Helper." In 2020 IEEE International Conference on Healthcare Informatics (ICHI). IEEE, 2020. http://dx.doi.org/10.1109/ichi48887.2020.9374299.

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Pilli, Venkata Sai, Kumar Pamidi, and Poovammal E. "Chronic Kidney Disease Prediction." In 2023 International Conference for Advancement in Technology (ICONAT). IEEE, 2023. http://dx.doi.org/10.1109/iconat57137.2023.10080709.

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Rani, Palak, Palak Patel, and Babu S. "Chronic Kidney Disease Prediction." In 2024 2nd International Conference on Networking and Communications (ICNWC). IEEE, 2024. http://dx.doi.org/10.1109/icnwc60771.2024.10537461.

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Belawati, Yeny Ristaning, Didik Gunawan Tamtomo, and Bhisma Murti. "Meta-Analysis the Effect of Chronic Disease on Catastrophic Health Expenditure." In The 7th International Conference on Public Health 2020. Masters Program in Public Health, Universitas Sebelas Maret, 2020. http://dx.doi.org/10.26911/the7thicph.04.49.

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ABSTRACT Background: The growing prevalence of chronic diseases contributed to high financial risks of health care. High total out-of-pocket health expenditure relative to income can result in catastrophic health expenditure. This meta-analysis was performed to assess the effect of chronic disease on catastrophic health expenditure. Subjects and Method: Meta-analysis and systematic review was conducted by collecting articles from Google Scholar, PubMed, Springer Link databases, which published from year 2000 to 2020. Keywords to collect the articles including,” chronic disease” OR “chronic illness” AND “catastrophic health expenditure” OR “financial burden” AND “cross sectional” AND “adjusted odds ratio”. The inclusion criteria were full text, in English language, using cross-sectional study design, and reporting adjusted odds ratio. Catastrophic health expenditure criteria if capacity to pay was ≥40% (excluding primary needs). The study population was households. The intervention was chronic disease with comparison non chronic disease. The study outcome was catastrophic health expenditure. The articles were selected by PRISMA flow chart. The quantitative data were analyzed using Revman 5.3. Results: 9 studies from Tanzania, Korea, China, Ghana, and India were selected for this study. This study showed that chronic disease increased catastrophic health expenditure (aOR= 1.94; 95% CI= 1.45 to 2.54; p<0.001). Conclusion: Chronic disease increases catastrophic health expenditure. Keywords: chronic disease, catastrophic health expenditure Correspondence: Yeny Ristaning Belawati. Masters Program in Public Health, Universitas Sebelas Maret. Jl. Ir. Sutami 36A, Surakarta 57216, Central Java. Email: belawatiyeni@gmail.com. Mobile: 082243302740. DOI: https://doi.org/10.26911/the7thicph.04.49
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Roccetti, Marco, Alice Casari, and Gustavo Marfia. "Inside Chronic Autoimmune Disease Communities." In ASONAM '15: Advances in Social Networks Analysis and Mining 2015. New York, NY, USA: ACM, 2015. http://dx.doi.org/10.1145/2808797.2808813.

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Hoque, Md Rakibul, and Mohammed Sajedur Rahman. "Predictive Modelling for Chronic Disease." In ICCDA 2020: 2020 The 4th International Conference on Compute and Data Analysis. New York, NY, USA: ACM, 2020. http://dx.doi.org/10.1145/3388142.3388174.

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Muvva, Bhuvan Chandra, Shiva Sankar Modala, Sandeep Mallikarjuna, Revanth Manohar Muppa, and Lipika Goel. "Diagnosis of chronic kidney disease." In LOW RADIOACTIVITY TECHNIQUES 2022 (LRT 2022): Proceedings of the 8th International Workshop on Low Radioactivity Techniques. AIP Publishing, 2023. http://dx.doi.org/10.1063/5.0169531.

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Charlie, Chumani, and Liezel Cilliers. "Introducing the Chronic Disease mHealth App Quality Evaluation Framework (CHRONIQ)." In 2024 Conference on Information Communications Technology and Society (ICTAS). IEEE, 2024. http://dx.doi.org/10.1109/ictas59620.2024.10507109.

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Reports on the topic "Chronic disease"

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Aranovich, Gabriel, Jay Bhattacharya, Alan Garber, and Thomas MaCurdy. Coping with Chronic Disease? Chronic Disease and Disability in Elderly American Population 1982-1999. Cambridge, MA: National Bureau of Economic Research, March 2009. http://dx.doi.org/10.3386/w14811.

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2

Lee, S. Chronic Beryllium Disease Prevention Program Report. Office of Scientific and Technical Information (OSTI), March 2012. http://dx.doi.org/10.2172/1047244.

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3

Gordon, Terry. Transgenic Mouse Model of Chronic Beryllium Disease. Office of Scientific and Technical Information (OSTI), May 2009. http://dx.doi.org/10.2172/953218.

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4

Nelson, Julie. Feline chronic kidney disease: Pathophysiology and diagnosis. Ames (Iowa): Iowa State University, January 2021. http://dx.doi.org/10.31274/cc-20240624-721.

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5

Barros-Poblete, Marisol, Rodrigo Torres-Castro, Mauricio Henríquez, Anita Guequen, Isabel Blanco, and Carlos Flores. Dysbiosis as a prognostic factor for clinical worsening in chronic respiratory disease: A systematic review and metanalysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2022. http://dx.doi.org/10.37766/inplasy2022.4.0089.

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Abstract:
Review question / Objective: Is dysbiosis a prognostic factor for clinical worsening in patients with chronic respiratory diseases?. Condition being studied: Dysbiosis, defined as changes in the quantitative and qualitative composition of the microbiota. Eligibility criteria: Over 18 years old adult patients with chronic respiratory diseases clinical diagnosis (cystic fibrosis, chronic obstructive pulmonary disease, asthma, idiopathic pulmonary fibrosis, interstitial lung disease, sarcoidosis, bronchiectasis, non-CF bronchiectasis, pulmonary hypertension) according to the International Statistical Classification of Diseases and Related Health Problems (ICD) from OMS) and international guidelines of each disease.
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6

Paskaran, S., M. LeBlanc, S. Petrie, and P. Peters. Infographic: eHealth Chronic Disease Management for Older Adults. Spatial Determinants of Health Lab, Carleton University, November 2019. http://dx.doi.org/10.22215/sdhlab/kt/2019.5.

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7

Dickerman, Joel L. Chronic Disease Management in Family Practice: Clinical Note. Fort Belvoir, VA: Defense Technical Information Center, March 1998. http://dx.doi.org/10.21236/ada364106.

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8

Gordon, Terry. Development of Biomarkers for Chronic Beryllium Disease in Mice. Office of Scientific and Technical Information (OSTI), January 2013. http://dx.doi.org/10.2172/1060004.

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9

Boone, Claire, Pablo Celhay, Paul Gertler, and Tadeja Gracner. Encouraging Preventative Care to Manage Chronic Disease at Scale. Cambridge, MA: National Bureau of Economic Research, August 2023. http://dx.doi.org/10.3386/w31643.

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10

Tomko, Lainey. Current Perspectives on Chronic Kidney Disease in Veterinary Medicine. Ames (Iowa): Iowa State University, May 2024. http://dx.doi.org/10.31274/cc-20240624-460.

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You might also be interested in the extended bibliographies on the topic 'Chronic disease' for particular source types:
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