Relevant bibliographies by topics / Chronic and acute diseases

Academic literature on the topic 'Chronic and acute diseases'

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Published: 11 March 2023

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Journal articles on the topic "Chronic and acute diseases"

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Senok, A. C. "Chronic Versus Acute Diseases." Science 309, no. 5733 (July 15, 2005): 380b—381b. http://dx.doi.org/10.1126/science.309.5733.380b.

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Zafrani, Lara, and Can Ince. "Microcirculation in Acute and Chronic Kidney Diseases." American Journal of Kidney Diseases 66, no. 6 (December 2015): 1083–94. http://dx.doi.org/10.1053/j.ajkd.2015.06.019.

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V. Vernekar, Pradeep. "HYPOPHOSPHATEMIA IN ACUTE & CHRONIC LIVER DISEASES." Journal of Evolution of Medical and Dental Sciences 2, no. 43 (October 26, 2013): 8372–78. http://dx.doi.org/10.14260/jemds/1469.

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Pfeiffer, H. "Acute infections in children with chronic diseases." British Homoeopathic journal 82, no. 2 (April 1993): 134. http://dx.doi.org/10.1016/s0007-0785(05)81063-4.

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Turk, Dennis C. "Are Pain Syndromes Acute or Chronic Diseases?" Clinical Journal of Pain 16, no. 4 (December 2000): 279–80. http://dx.doi.org/10.1097/00002508-200012000-00001.

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Floege, Jürgen. "Models for acute and chronic kidney diseases." Drug Discovery Today: Disease Models 7, no. 1-2 (March 2010): 1–2. http://dx.doi.org/10.1016/j.ddmod.2010.11.002.

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Lin, Tien-An, Victor Chien-Chia Wu, and Chao-Yung Wang. "Autophagy in Chronic Kidney Diseases." Cells 8, no. 1 (January 16, 2019): 61. http://dx.doi.org/10.3390/cells8010061.

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Autophagy is a cellular recycling process involving self-degradation and reconstruction of damaged organelles and proteins. Current evidence suggests that autophagy is critical in kidney physiology and homeostasis. In clinical studies, autophagy activations and inhibitions are linked to acute kidney injuries, chronic kidney diseases, diabetic nephropathies, and polycystic kidney diseases. Oxidative stress, inflammation, and mitochondrial dysfunction, which are implicated as important mechanisms underlying many kidney diseases, modulate the autophagy activation and inhibition and lead to cellular recycling dysfunction. Abnormal autophagy function can induce loss of podocytes, damage proximal tubular cells, and glomerulosclerosis. After acute kidney injuries, activated autophagy protects tubular cells from apoptosis and enhances cellular regeneration. Patients with chronic kidney diseases have impaired autophagy that cannot be reversed by hemodialysis. Multiple nephrotoxic medications also alter the autophagy signaling, by which the mechanistic insights of the drugs are revealed, thus providing the unique opportunity to manage the nephrotoxicity of these drugs. In this review, we summarize the current concepts of autophagy and its molecular aspects in different kidney cells pathophysiology. We also discuss the current evidence of autophagy in acute kidney injury, chronic kidney disease, toxic effects of drugs, and aging kidneys. In addition, we examine therapeutic possibilities targeting the autophagy system in kidney diseases.
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khatal, Sujata, Priya Nair, and U. B. Shetkar U.B.Shetkar. "Case Report of Acute on Chronic Pancreatitis with Hyperparathyroidism with Chronic Kidney Disease." Indian Journal of Applied Research 4, no. 3 (October 1, 2011): 397–99. http://dx.doi.org/10.15373/2249555x/mar2014/124.

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Suvada, Jozef, Irad Bejdjebel, Vladimir Krcmery, Zuzana Dudlova, Premyslaw Ulmann, Alexandra Mamova, Michael Leisten, et al. "The Most Common Diseases Among Syrian and Palestinian Refugees to Lebanon: Acute and Chronic Stress Related Diseases are Prevalent." Clinical Social Work and Health Intervention 8, no. 1 (March 27, 2017): 50–53. http://dx.doi.org/10.22359/cswhi_8_1_11.

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Ramonet, David, Marco Pugliese, Manuel J. Rodrı́guez, Lluı̈sa de Yebra, Carmen Andrade, Rosa Adroer, Teresa Ribalta, Joan Mascort, and Nicole Mahy. "Calcium precipitation in acute and chronic brain diseases." Journal of Physiology-Paris 96, no. 3-4 (May 2002): 307–12. http://dx.doi.org/10.1016/s0928-4257(02)00020-7.

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Dissertations / Theses on the topic "Chronic and acute diseases"

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Stewart, Simon. "Optimising therapeutic efficacy in acute and chronic cardiac disease states /." Title page, contents and abstract only, 1999. http://web4.library.adelaide.edu.au/theses/09PH/09phs851.pdf.

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White, David James. "Haemophilus in acute and chronic respiratory disease." Thesis, Liverpool John Moores University, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.304425.

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Wei, Jin. "Acute Kidney Injury and Chronic Kidney Disease." Scholar Commons, 2017. http://scholarcommons.usf.edu/etd/6780.

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Ischemia and reperfusion are natural steps during kidney transplantation, and IRI is considered one of the most important nonspecific factors affecting allograft dysfunction. Transplanted organs experience several episodes of ischemia, in which cold ischemia occurs during allograft storage in preservation solutions. Even though cold ischemia has been studied extensively, all of the studies have been carried out in vitro and ex vivo models. There is no in vivo model available to examine renal IRI induced solely by cold ischemia. In the present study, we developed an in vivo mouse model to study renal IRI induced exclusively by cold ischemia through clamping the renal pedicle for 1 to 5 hours. During the ischemic phase, blood was flushed from the kidney with cold saline through a small opening on the renal vein. The kidney was kept cold in a kidney cup with circulating cooled saline, while the body temperature was maintained at 37℃ during the experiment. The level of kidney injury was evaluated by plasma creatinine, KIM-1, NAGL, GFR, and histology. Plasma creatinine was significantly increased from 0.15±0.04 mg/dl in the sham group to 1.14±0.21 and 2.65±0.14 mg/dl in 4 and 5-hours ischemia groups at 24 hours after cold IRI. The plasma creatinine in mice with ischemic time <3 hours demonstrated no significant increase compared with sham mice. Changes in KIM-1, NAGL, GFR and histology were similar to plasma creatinine. 65 In summary, we developed and characterized a novel in vivo IRI-induced AKI mouse model exclusively produced by cold ischemia.
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Rota, Cinzia. "Effect of foetal and adult stem cells in acute and chronic kidney diseases." Thesis, Open University, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.594841.

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Acute kidney injury (AKl) and chronic kidney disease (CKD) are serious illnesses associated to high mortality and unsatisfactory therapeutic treatments. In search for new therapies, it has become evident that stem cells could be a possible option for patients with AKI and CKD. The evidence of the reno protective effect of bone marrow-mesenchymal stem cells (BM-MSCs) in experimental model of AKl, prompted us to study the effect of stem cells isolated from sources that are more accessible as cord blood (CB) and amniotic fluid. Infusion of hCB-MSCs in inununodeficient mice with AKI ameliorated renal function and tubular structure, prolonging survival. Moreover, transplanted hCB-MSCs localized in peritubular areas, limiting oxidative stress and apoptosis. By virtue of stem cell capacity to produce growth factors, hCB-MSCs were able- to induce the pro-survival factor Akt in tubular cells and subsequently their proliferation. Using the well-established model of AKI in immunodeficient mice, we studied the pro-regenerative effect of amniotic fluid stem (hAFS) cells. Infusion of hAPS cells in cisplatin-mice improved renal function and limited tubular damage, although not to control level, and prolonged animal survival. These cells engrafted injured kidney predominantly in peri tubular region and through a paracrine mechanism are able to exert an anti-apoptotic effect, to activate AId and stimulate proliferation of tubular cells. We enhanced the therapeutic potential of hAFS cells by cell pretreatment with GDNF, which markedly ameliorated renal function and tubular injury by increasing stem cell homing to the tubulointerstitial compartnent. In AKI models, the renoprotective effect of BM-MSCs is well established, however the role of these stem cells in model of eKD is controversial and not demonstrated so far. Therefore, we tested the effect of BM-MSCs in a model of adriarnycin-induced nephropathy. Repeated infusions of BMMSCs limited podocyte loss, and normalized distribution of parietal epithelial cells along the Bowman's capsule, reducing glomerulosclerosis. Moreover, through the local release of growth factors as VEGF, BM-MSCs were able to provide a local pro-survival environment that limited glomerular inflanunation and microvascular rarefaction.
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何美美 and Mai-mai Ho. "A study on the acute and chronic effects of morphine on rat stomachs." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1986. http://hub.hku.hk/bib/B31230611.

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Bevan, Damon. "Cytokine involvement in ultraviolet (UV) B induced chronic and acute inflammation in porcine skin." Thesis, Royal Veterinary College (University of London), 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.300963.

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Phan, Thanh Huyen. "Development of therapeutic extracellular vesicles for the treatment of acute and chronic lung diseases." Thesis, The University of Sydney, 2022. https://hdl.handle.net/2123/28760.

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Acute and chronic lung diseases are among the most common causes of disability and death worldwide, which consequently contribute a substantial burden for health and economic system. Chronic obstructive pulmonary disease (COPD) – one type of chronic lung diseases – is a complicated and life-threatening disease. Despite considerable efforts toward developing treatments for COPD, there are currently no effective treatments to resolve this disease. The only available treatments are supportive, and they either allow for temporary relief of several symptoms of COPD such as dyspnoea, cough, and sputum, or reduce exacerbations. Hence, there is an urgent need for a new regenerative medicine approach that can promote the repair/regeneration process in lungs. In the context of tissue regeneration, extracellular vesicles (EVs) are considered as the next generation therapeutics, which can regenerate and restore the function of lung tissue that has been damaged by a disease or injury. However, very few EV-therapeutics can progress to clinical trials due to (a) the lack of standardisation of EV production, and (b) the lack of appropriate lung models to validate the therapeutic effects of EVs. Hence, to accelerate the clinical translation of EV-therapeutics to resolve COPD, the presented work in this thesis was divided into two stages: 1. Development of EV-therapeutics and establishing quality control protocol for EVs to ensure their safety and efficacy in treating COPD. 2. Establishing pre-clinical models to enable testing and validation of the efficacy of EV-therapeutics in treating COPD. This research provided an innovative methodology to produce and comprehensively characterise EVs, which offers a new means to understand the role of EVs in numerous biological processes and disease mechanisms. Moreover, this research would bring significant benefits to global healthcare by accelerating the development of EV-therapeutics for the treatment of lung diseases.
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Brocca, Alessandra. "Characterization of molecular pathways involved in acute and acute-on-chronic liver disease." Doctoral thesis, Università degli studi di Padova, 2017. http://hdl.handle.net/11577/3425355.

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Introduction: Acute decompensation was defined as the acute development of one or more major complications of liver disease and it was the main cause of hospitalization in patients with cirrhosis. The acute-on-chronic liver failure (ACLF) was characterized by acute decompensation of cirrhosis, organ failure and high 28-day mortality. ACLF displayed key features of systemic inflammation and its poor outcome was closely associated with exacerbated systemic inflammatory responses. Inflammasomes were multiprotein complexes which proteolytically activates the cytokines IL-1β and IL-18. These substrates might have an effect on the development of liver disease. Extracellular vesicles (EVs) were involved in many important biological processes as well as in disease pathogenesis. The dynamics of EVs secretion by different cell types and how the secreted EVs interact to advance the pathogenesis of liver disease were still unknown. Aims: The aim of this study was to characterize the molecular pathways involved in acute decompensation of cirrhosis and ACLF through: the characterization of the inflammatory profile of patients, the in vitro evaluation of cytotoxic effects of plasma from patients on renal tubular cells, the expression of Inflammasome in these treated cells and in Peripheral Blood Mononuclear Cells (PBMC) of patients, the characterization of EVs from patients and the study of in vitro effects of isolated EVs in renal tubular cells. Material and Methods: We enrolled patients with compensated cirrhosis, acute decompensation in cirrhosis, ACLF and healthy subjects as control population. Plasma levels of IL-6, IL-1β and IL-18 were detected by ELISA assay. Cytotoxic effects of plasma on renal tubular cells were assayed by annexin V and propidium iodide kit. Inflammasomes expression was detected both in renal tubular cells treated and in PBMC extracted from patients by Real Time PCR. Plasma EVs were extracted by ultracentrifugation and concentration was measured by Nanosight. Characterization of EVs was performed by FACS analysis. Cytotoxic effects of plasma EVs on renal tubular cells were assayed by XTT assay. Results: Plasma levels of pro-inflammatory cytokines measure in the firsts patients enrolled did not differed between the groups of compensated cirrhosis, acute decompensation and ACLF. Also viability and death rate did not change in a way statistically significant in cell stimulated with plasma from the three groups of patients. Furthermore, Inflammasome gene expression in these cells did not underlines the activation of this protein complex. In PBMC from patients, gene expression of Tool-like receptor 2 (TLR-2) was significantly higher in patients with compensated cirrhosis compare to acute decompensation of cirrhosis (p=0.036). Albumin added to cell medium reduced cytotoxic effects of plasma on renal tubular cells. Plasma EVs of patients enrolled were more concentrated in ACLF groups compare to healthy subjects. EVs did not expressed selected platelets (CD41, CD42b) and monocyte markers (CD14) in their surface but they expressed marker of platelets activated endothelium (CD62E). The levels of CD62E were significantly higher in patients with ACLF compare to healthy subjects and patients with compensated cirrhosis (p=0.0041 and p=0.0111, respectively). CD40L levels were significantly higher in all patients' groups compare to healthy subjects (p<0.02). Plasma EVs from patients with acute and acute-on-chronic liver failure exerted a higher cytotoxic effects compare to healthy subjects and patients with compensated cirrhosis on renal tubular cells (p<0.0001). Cells incubated with EVs from acute and acute-on-chronic liver failure underwent to apoptosis (p<0.0001), to ROS production (p<0.0001), to lose albumin intake capabilities (p<0.0001) and reduction of Zonula Occludens-1 (ZO-1) expression (p=0.0166) compare to healthy subjects and patients with compensated cirrhosis. Instead, megalin and PGC1α expression did not change. Conclusions: The role of EVs in decompensated cirrhosis and ACLF need to be invastigated and study their hypothetic role as vehicle of mediator of extrahepatic organ injury and complications of cirrhosis.
Introduzione: Lo scompenso acuto in cirrosi è definito come la progressione acuta di una o più gravi complicanze della patologia epatica ed è la principale causa di ospedalizzazione nei pazienti con cirrosi. L'insufficienza epatica acuta su cronica (ACLF) è caratterizzata da uno scompenso acuto della cirrosi, da danno d'organo e da un elevato tasso di mortalità a 28 giorni. L'ACLF è caratterizzato da infiammazione sistemica e l'outcome infausto è strettamente associato con l'eccessiva risposta infiammatoria che si attiva nel paziente. L'inflammasoma è un complesso multi-proteico che attiva, mediante taglio proteolitico, citochine pro-infiammatorie come IL-1β e IL-18. Queste, hanno un ruolo nello sviluppo della patologia epatica. Le vescicole extracellulari (EVs) sono coinvolte in molti processi biologici importanti, sia fisiologici che patologici. Il processo di secrezione delle EVs da diversi tipi di cellule e la loro azione nel modulare l'avanzamento della patogenesi nella malattia epatica, non sono ancora completamente chiariti. Scopo: Lo scopo di questo studio è caratterizzare la via del segnale coinvolta nello scompenso acuto della cirrosi e dell'insufficienza epatica acuta-su-cronica attraverso: la caratterizzazione del profilo infiammatorio dei pazienti arruolati, lo studio in vitro dell'effetto citotossico nel plasma dei pazienti nelle cellule tubulari renali (RTC), lo studio dell'espressione dell'inflammasoma nelle cellule trattate e nelle cellule mononucleate del sangue periferico (PBMC) estratte dai pazienti, la caratterizzazione delle EVs estratte dal plasma dei pazienti arruolati e lo studio del loro effetto in vitro su colture di RTC. Materiali e Metodi: sono stati arruolati pazienti con cirrosi compensata, scompenso acuto in cirrosi, insufficienza epatica acuta-su-cronica e una popolazione di volontari sani come controllo. I livelli plasmatici di IL-6, IL-1β e IL-18 sono stati misurati con kit ELISA. L'effetto citotossico del plasma nelle RTC è stato testato con il kit costituito da annexina V e propidio ioduro. Il livello di espressione delle molecole dell'inflammasoma è stato determinato sia nelle cellule stimolate con il plasma, sia nei PBMC estratti dai pazienti, attraverso Real Time Poly Chain Reaction (RT-PCR). Le EVs plasmatiche sono state estratte mediante ultracentrifugazione e la loro concentrazione determinata con il Nanosight. La loro caratterizzazione è stata eseguita mediante analisi al FACS. L'effetto citotossico delle EVs sulle RTC è stato determinato mediante saggio XTT. Risultati: I livelli plasmatici delle citochine pro-infiammatorie, misurati nei primi pazienti arruolati, non mostra differenze tra i gruppi di pazienti con cirrosi compensata, scompensata e insufficienza epatica acuta-su-cronica. Anche la vitalità e la morte cellulare nelle colture stimolate con i plasma dei pazienti arruolati non hanno mostrato un profilo peculiare dei gruppi testati e non è stata riscontrata attivazione della trascrizione delle molecole dell'inflammasoma. L'espressione del Tool-like receptor 2 (TLR-2) nei PBMC si è dimostrata significativamente elevata nei pazienti con cirrosi compensata rispetto a quelli con scompenso acuto (p=0,036). Aggiungendo albumina al mezzo di coltura cellulare si è notata una riduzione dell'effetto citotossico del plasma dei pazienti nelle RTC. La concentrazione plasmatica di EVs risulta maggiore nei pazienti con insufficienza epatica acuta-su-cronica rispetto ai controlli sani. Le vescicole non esprimono i marcatori tipici piastrinici (CD41 e CD42b) e monocitari (CD14) sulla loro superficie ma esprimono il marcatore dell'epitelio attivato da piastrine (CD62E). I livelli di CD62E sono significativamente elevati nei pazienti con insufficienza eparica acuta-su-cronica rispetto ai controlli sani e ai pazienti con cirrosi compensata (p=0,0041 e p=0,0111, rispettivamente). I livelli di CD40L sono significativamente elevati in tutti i gruppi di pazienti rispetto ai controlli sani (p<0,02). Le EVs isolate dei pazienti con insufficienza epatica acuta-su-cronica hanno un effetto citotossico superiore rispetto a quelle dei controlli sani e dei pazienti con cirrosi compensata nelle RTC (p<0,0001). Le cellule incubate con le EVs da pazienti con scompenso acuto in cirrosi e insufficienza epatica acuta-su-cronica vanno incontro ad apoptosi (p<0,0001), a produzione massiccia di ROS (p<0,0001), alla perdita della capacità di internalizzare l'albumina (p<0,0001) e alla riduzione dell'espressione di Zonula Occludens-1 (ZO-1) (p=0,0166) rispetto ai soggetti sani e ai pazienti con cirrosi compensata. Non si osserva invece un cambiamento nell'espressione cellulare di megalina e PGC1α. Conclusioni: Il ruolo delle EVs nella cirrosi scompensata necessita di essere approfondito perché potrebbero rappresentare il veicolo su cui viaggiano i mediatori del danno d’organo extraepatico.
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Lo, Iek-long, and 羅奕龍. "Impacts of cognitive impairment on acute exacerbations of chronic obstructive pulmonary disease among Chinese elderly patients." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B45830770.

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Chen, Roy Yu-Wei. "Biomarkers for acute exacerbation of chronic obstructive pulmonary disease." Thesis, University of British Columbia, 2016. http://hdl.handle.net/2429/57759.

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Rationale: There are currently no generally accepted and validated blood tests available for diagnosing acute exacerbations of chronic obstructive pulmonary disease (AECOPD). There is an urgent need of biomarkers that can guide therapeutic management in AECOPD. Based on literature review, systemic inflammation and mild cardiac dysfunction are often associated with AECOPD. We hypothesized that certain protein markers can indeed be useful in tracking and diagnosing AECOPD progression. Methods: The study cohort consisted of 368 patients recruited in the chronic obstructive pulmonary disease (COPD) Rapid Transition Program who were hospitalized with a primary diagnosis of AECOPD, and 76 stable COPD patients who served as controls. We first determined the relationship of AECOPD of C-reactive protein (CRP) and the N-terminal of the prohormone brain natriuretic peptide (NT-proBNP). We then performed a discriminatory analysis using receiver-operating characteristics (ROC) curve in a logistic regression model. We compared the area under the curve (AUC) of 4 different combinations of CRP and NT-proBNP models. Lastly, we examined several potential biomarkers that were implicated in AECOPD. Results: The demographic data of the cohort and the controls were well matched, with an average age of 68 versus 65 years old, 64% versus 77% male, and a forced expiratory volume in 1 second (FEV1) % predicted of 52% versus 58%. The CRP and NT-proBNP levels at exacerbation onset were found to be the highest and progressively decreased over time. Of the 4 models of ROC curves, the leave-one-out cross-validated model including both CRP and NT-proBNP had an AUC of 0.80. This model replicated well in an external LEUKO dataset. On the ii other hand, D-Dimer, pulmonary and activation-regulated chemokine (PARC) and troponin I, showed minimal or no temporal changes during hospitalization and were no different than those with stable COPD. Conclusions: In summary, this thesis demonstrated that biomarkers such as CRP and NT-proBNP are significantly elevated during AECOPD and decreased with recovery. Secondly, a combination of CRP and NT-proBNP could discriminate patients who were hospitalized for their AECOPD from stable patients. Together, these two biomarkers show promise in diagnosing and tracking AECOPD.
Medicine, Faculty of
Medicine, Department of
Experimental Medicine, Division of
Graduate
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Books on the topic "Chronic and acute diseases"

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M, Siafakas N., Anthonisen N. R, and Georgopoulos Dimitris, eds. Acute exacerbations of chronic obstructive pulmonary disease. New York: M. Dekker, 2004.

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Shipton, E. A. Pain: Acute & chronic. Johannesburg: Witwatersrand University Press, 1993.

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G, Edwards Scott, ed. Acute and chronic elbow instability. Philadelphia, Pa: Saunders, 2008.

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Acute exacerbation of respiratory diseases. New Delhi: Jaypee Brothers Medical Publishers, 2012.

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Edward, Redington Anthony, and Morice Alyn H, eds. Acute and chronic cough. Boca Raton: Taylor & Francis, 2005.

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Pain: Acute and chronic. London: Arnold, 1999.

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Cazzola, Mario. Acute exacerbations in COPD. Oxford: Clinical Pub., 2009.

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Jean-Philippe, Derenne, Similowski Thomas 1961-, and Whitelaw William A. 1941-, eds. Acute respiratory failure in chronic obstructive pulmonary disease. New York: M. Dekker, 1996.

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E, Naschitz Jochanan, ed. Challenges in acute geriatric care. Hauppauge, NY: Nova Science Publishers, 2009.

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1955-, Ferguson Berrylin J., and Johnson Jonas T, eds. Complications of acute and chronic sinus disease. 4th ed. Alexandria, VA: American Academy of Otolaryngology--Head and Neck Surgery Foundation, 2007.

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Book chapters on the topic "Chronic and acute diseases"

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Thabet, Asalim, Rhonda Philopena, and Joseph Domachowske. "Acute and Chronic Lymphadenitis." In Introduction to Clinical Infectious Diseases, 25–34. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-91080-2_3.

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Domachowske, Joseph, and Manika Suryadevara. "Acute and Chronic Lymphadenitis." In Clinical Infectious Diseases Study Guide, 15–20. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-50873-9_3.

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Garcia-Martinez, Rita, Raquel Diaz-Ruiz, Jesus Millan, and Rafael Bañares. "Chronic Liver Failure and Acute-on-Chronic Liver Failure." In Liver Diseases, 381–94. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-24432-3_33.

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Vellingiri, Vigneshwaran, Prabhu Thirusangu, and Inshah Din. "Acute Respiratory Distress Syndrome: Therapeutics, Pathobiology, and Prognosis." In Chronic Lung Diseases, 143–56. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-3734-9_7.

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Brunner, H., and W. Weidner. "Acute and chronic prostatitis." In Clinical Problems in Sexually Transmitted Diseases, 37–59. Dordrecht: Springer Netherlands, 1985. http://dx.doi.org/10.1007/978-94-009-5014-6_3.

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Wong, Florence. "Acute-on-Chronic Liver Failure." In Schiff's Diseases of the Liver, 432–59. Oxford, UK: John Wiley & Sons, Ltd, 2017. http://dx.doi.org/10.1002/9781119251316.ch18.

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Wang, Cenyi, Michael Kirberger, and Ning Chen. "Acute and Chronic Exercise on Autophagy." In Exercise, Autophagy and Chronic Diseases, 29–46. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-4525-9_2.

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Apfel, Tehilla, and Nikolaos T. Pyrsopoulos. "Liver Transplantation for Acute and Chronic Liver Failure." In Liver Diseases, 727–39. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-24432-3_68.

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Giovagnoni, Andrea, and Federico Crusco. "Imaging of Acute and Chronic Pancreatitis." In Surgical Treatment of Pancreatic Diseases, 63–81. Milano: Springer Milan, 2009. http://dx.doi.org/10.1007/978-88-470-0856-4_5.

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Kjellstrand, C. M. "Acute Problems during Hemodialysis." In Chronic Renal Disease, 417–26. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-4826-9_42.

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Conference papers on the topic "Chronic and acute diseases"

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Evcik, D., and A. Yücel. "FRI0241 Lumbar lordosis in acute and chronic low back pain patients." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.545.

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Héroult, M., F. Lioté, R. Champy, D. Barritault, and J. Courty. "THU0052 Heparin affin regulatory peptide (harp) in acute and chronic articular diseases." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.849.

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Bernardes, Leonardo de Sousa, Marina Trombin Marques, Matheus Gonçalves Maia, Edson Junior Gonçalves Bechara, Eduardo dos Santos Sousa, Juliana Rodrigues Dias Primo, and Francisco Tomaz Meneses de Oliveira. "Acute Vogt-Koyanagi-Harada: a case report." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.037.

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Context: Vogt-Koyanagi-Harada disease is an inflammatory disorder, which presents with intraocular, auditory and central nervous system involvement. It has two distinct courses: acute onset and chronic recurrence, whose differential diagnoses are, respectively, diseases of the optic neuromyelitis spectrum, and chronic meningitis. The diagnostic criteria developed by the international disease committee in 2001 classify patients into: probable disease (ocular findings only), incomplete (ocular plus cutaneous system or neurological manifestations) and complete (when the three forms occur together). Methods: Report the case of a patient seen at the emergency room of Santa Casa de São Paulo, diagnosed with Vogt-Koyanagi-Harada disease. Case report: 43-year-old woman, reporting occipital headache, with irradiation to the retro-orbital region, progressed to sudden bilateral amaurosis, in addition to conjunctival hyperemia. Neurological physical examination presented bilateral visual acuity (Snellen)> 20/200, poorly delimited optical discs. Uveitis and scleritis were also found. Brain and orbit MRI showed: bilateral retinal detachment, with small subretinal collections; regular thickening and impregnation of the choroid; tenuous episcleral impregnation; alteration of the sign of the inner ears, more evident in the cochleae; tenuous linear leptomeningeal impregnation at the level of the cerebellobulbar cistern. Liquor: 21 cells (99% lymphocytes), 31 proteins and 47 glucose, negative culture for bacteria. We proceeded to infusion 1000mg of methylprednisolone for 3 days, with daily use, subsequently, of prednisone 60mg, with significant improvement of the condition. Conclusions: It is a rare disease, but it must be recognized by every neurologist, since it is treatable and can leave serious visual sequelae.
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Dragun, K. V., and E. P. Zhivitskaya. "EPIDEMIOLOGICAL ANALYSIS OF THE MORBIDITY OF THE POPULATION OF KRUPKI DISTRICT WITH RESPIRATORY DISEASES." In SAKHAROV READINGS 2022: ENVIRONMENTAL PROBLEMS OF THE XXI CENTURY. International Sakharov Environmental Institute of Belarusian State University, 2022. http://dx.doi.org/10.46646/sakh-2022-2-61-64.

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Respiratory diseases are one of the most common pathologies in the structure of general and primary morbidity. The analysis of the primary morbidity of respiratory diseases of the adult population of Krupki district for the period 2014-2020 was carried out and the trends of morbidity were determined. In the structure of the primary morbidity of the adult population, respiratory diseases occupy the first rank place. There is a decrease in the primary incidence of respiratory diseases of the adult population of Krupki district. Acute respiratory infections of the respiratory tract, pneumonia, chronic rhinitis, nasopharyngitis, pharyngitis, sinusitis, asthma and asthmatic status, vasomotor and allergic rhinitis made the main contribution to the structure of the primary incidence of respiratory diseases by nosological forms of the adult population in 2020. Acute respiratory viral infections, vasomotor and allergic rhinitis are characterized by a decrease in primary morbidity, and for pneumonia, chronic rhinitis, nasopharyngitis, pharyngitis and sinusitis - an increase in indicators.
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Mertnoff, Rosa, Cecilia Vindrola Padros, Eugenia Brague, Walter Cattaneo, Lucila Falcone, Maria Belen Ayala Torales, and Mariangeles Pirchi. "P22 Improving the comprehensive care of people with advanced chronic diseases in acute hospitals." In Crafting the future of qualitative health research in a changing world abstracts. British Medical Journal Publishing Group, 2019. http://dx.doi.org/10.1136/bmjopen-2019-qhrn.57.

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Santos, Justino Duarte, Romuere R. V. Silva, and Rodrigo M. S. Veras. "Application of geostatistical functions and deep features to kidney biopsy images to differentiate focal segmental glomerulosclerosis from minimal change disease." In Conference on Graphics, Patterns and Images. Sociedade Brasileira de Computação, 2020. http://dx.doi.org/10.5753/sibgrapi.est.2020.12984.

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Chronic kidney diseases arise from acute or intermittent pathologies that have not been adequately treated, such as minimal change disease (MCD) and focal segmental glomerulosclerosis (FSGS). The accurate identification of these two diseases is of paramount importance, because their treatments and prognoses are different. Thus, we propose a method that is capable of differentiating MCD from FSGS based on images from pathological examinations. In the proposed method, we use four pre-trained convolutional neural networks and geostatistical functions to extract image features. Of the 8,720 extracted features, we selected 94 based on mutual information criteria, and in the classification step, we used a random forest classifier. The proposed method obtained an accuracy of 94.3% and Kappa index of 87.9%, a level that is regarded as “almost perfect”, confirming that our method is very promising.
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Miller, T. V., S. S. Dikunina, and E. P. Kotelnikova. "ANALYSIS OF ANTIBACTERIAL PROPERTIES CHELIDONIUM AND YODOPYRON TINCTURES." In "International Scientific and Practical Conference" THEORY AND PRACTICE OF VETERINARY PHARMACY, ECOLOGY AND TOXICOLOGY IN AIC ", dedicated to the centenary of the Department of Pharmacology and Toxicology, SPbSUVM. FSBEI HE St. Petersburg SUVM, 2021. http://dx.doi.org/10.52419/3006-2021-2-167-168.

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One of the promising and dynamically developing areas of modern medicine and pharmacy is the development of new effective means of herbal or natural origin for the treatment and prevention of acute and chronic diseases caused by various microorganisms. A pronounced antibacterial activity was shown by celandine tincture for all four gram-negative microorganisms. Recommended external use of celandine tincture in the treatment and prevention of skin diseases, in the pathogenesis of which are involved gram-negative microorganisms Enterobacter cloacae, Proteus mirabilis, Proteus vulgaris and Pseudomonas aeruginosа.
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Ghallab, A., M. Maiju, R. Hassan, A. Zaza, U. Hofmann, EM Buhl, KM Schneider, et al. "A mouse model of chronic liver disease progression and acute-on-chronic liver failure." In 35. Jahrestagung der Deutschen Arbeitsgemeinschaft zum Studium der Leber. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0038-1677072.

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Jo-Avila, Miguel, Kevin Roos, Ahmed Al-Jumaily, and Jun Lu. "Super Imposed Length Oscillations (SILO) and Their Effect on Asthmatic Models (Acute and Chronic) During an Asthmatic Attack." In ASME 2014 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2014. http://dx.doi.org/10.1115/imece2014-37633.

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The main driving mechanism during an asthmatic attack is the hyperconstriction of airway smooth muscle (ASM), which reduces the airway lumen and makes normal breathing difficult. The contraction can be relieved using bronchodilator drugs such as Isoproterenol, which induce temporary relaxation of the constricted airways. Pharmacological treatments are widely used in asthma, but their effectiveness varies from one subject to another, as do their side effects. Studies have shown that mechanical oscillations equivalent to physiological patterns such as breathing and deep inspiration in healthy airways can induce airway relaxation, but this type of response is not observed in asthmatics. Length oscillations seem to be a non-medicinal approach to treat ASM hyperconstriction present in many respiratory diseases such as asthma. Currently little is known about the effect of other oscillations’ patterns and their combination with breathing and deep inspiration on healthy and asthmatic airways during an asthmatic attack. Preliminary results obtained from in vitro and in vivo experiments in our laboratory indicate that the use of super imposed length oscillations (SILO) over normal breathing patterns can induce relaxation during an induced asthmatic attack on healthy and asthmatic subjects. These tests have been carried out using animal models which have been prepared under an acute protocol for the disease (new asthmatics), but these oscillations still remain to be tested in chronic asthmatic models (chronic asthmatics).
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Rufino, Rogério L., Mateus Bettencourt, and Cláudia H. Costa. "Longitudinal Study Of Acute Exacerbation In Chronic Obstructive Pulmonary Disease." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a3051.

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Reports on the topic "Chronic and acute diseases"

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Research, Gratis. Regenerative Medicine: A Breakthrough in the Branch of Medicine. Gratis Research, November 2020. http://dx.doi.org/10.47496/gr.blog.04.

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Regenerative medicine, being an interdisciplinary field, applies the principle of engineering and life science to promote regeneration. Regenerative medicine supports the treatment of chronic diseases and acute insults
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Spahn, Joanne, Charlotte Bahnfleth, Marlana Bates, Natasha Cole, Molly Higgins, Julie Obbagy, and Sara Scinto-Madonich. A Series of Evidence Scans on Acute Adverse Health Effects, Chronic Disease Risk, and Daily Requirements. U.S. Department of Agriculture, Food and Nutrition Service, Center for Nutrition Policy and Promotion, Nutrition Evidence Systematic Review, 2022. http://dx.doi.org/10.52570/nesr.dri2022.es01.

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du, xinying, xinying du, xianjun fu, kuo xu, and kejian li. Meta analysis on the treatment of acute exacerbation of chronic obstructive pulmonary disease with lung dispersing therapy. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2022. http://dx.doi.org/10.37766/inplasy2022.11.0094.

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Ji, Zile, Xuanlin Li, Siyuan Lei, and Yang Xie. A pooling analysis of the risk prediction models for mortality in acute exacerbation of chronic obstructive pulmonary disease. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2022. http://dx.doi.org/10.37766/inplasy2022.4.0151.

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Ji, Zile, Xuanlin Li, Siyuan Lei, and Yang Xie. A pooling analysis of the risk prediction models for mortality in acute exacerbation of chronic obstructive pulmonary disease. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2022. http://dx.doi.org/10.37766/inplasy2022.4.0151.

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Liu, Ying, Hanyu Fang, Zheng Hong, Yu Ming, and Hongchun Zhang. Effectiveness and safety of Suhuang Zhike Capsule for acute exacerbations of Chronic Bronchitis or Chronic Obstructive Pulmonary Disease in adults :a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, August 2022. http://dx.doi.org/10.37766/inplasy2022.8.0084.

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Hong, Zheng, Ying Liu, HanYu Fang, Hongchun Zhang, and Bang Yu. Effectiveness and safety of Qingjin Huatan Capsule for acute exacerbation of Chronic Bronchitis or Chronic Obstructive Pulmonary Disease in adults: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2022. http://dx.doi.org/10.37766/inplasy2022.9.0075.

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LEI, XING, and HONGMIN GUO. The effects of Su-Zi-Jiang-Qi decoction (SZJQ) in acute exacerbation of chronic obstructive pulmonary disease: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2021. http://dx.doi.org/10.37766/inplasy2021.6.0115.

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Chen, Dan, Jian Sun, and Ya-nan Chu. The diagnostic value of serum amyloid A in patients with acute exacerbation of chronic obstructive pulmonary disease: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2022. http://dx.doi.org/10.37766/inplasy2022.4.0143.

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Hu, Yang Yang, Xing Zhang, Yue Luo, and Yadong Wang. Systematic review and Meta analysis of the efficacy and safety of rifaximin in the prevention and treatment of hepatic encephalopathy. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, February 2023. http://dx.doi.org/10.37766/inplasy2023.2.0061.

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Review question / Objective: P：Liver cirrhosis patients with risk factors associated with HE attack;HE patients caused by chronic liver diseases represented by cirrhosis. I: Rifaximin treatment. C: Other drugs or placebo. O：HE incidence; HE improvement; All-cause mortality; Blood ammonia level; PSE index; mental state; NCT-A; NCT-B; Adverse events. Condition being studied: Hepatic encephalopathy(HE) is a neuropsychiatric disorder syndrome based on metabolic disorders, which is caused by severe acute and chronic liver dysfunction or various abnormalities of portosystemic shunt (hereinafter referred to as portosystemic shunt). The research data shows that the prevalence of OHE in patients with cirrhosis is 10-14%, and the prevalence of HE in patients with decompensated cirrhosis is 16-21%. HE can lead to 60-80% of patients with liver cirrhosis with mild cognitive impairment, affecting their ability of daily life and quality of life. When OHE occurs, the one-year mortality rate of patients with liver cirrhosis is 64%, which brings a heavy economic burden to patients and public health resources. Therefore, the prevention and early management of HE is very important.
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