Relevant bibliographies by topics / Chordoid Glioma

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters

Academic literature on the topic 'Chordoid Glioma'

Author: Grafiati
Published: 16 November 2024

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Journal articles on the topic "Chordoid Glioma"

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Thavaratnam, LK, ST Loy, A. Gupta, I. Ng, and JF Cullen. "Chordoid glioma." Singapore Medical Journal 56, no. 11 (November 2015): 641–43. http://dx.doi.org/10.11622/smedj.2015175.

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Liu, Wei-ping, Jin-xiang Cheng, Xi-cai Yi, Hai-ning Zhen, Zhou Fei, Qing Li, and Xiang Zhang. "Chordoid Glioma." Neurologist 17, no. 1 (January 2011): 52–56. http://dx.doi.org/10.1097/nrl.0b013e3181e7db67.

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Leeds, Norman E., Frederick F. Lang, Teresa Ribalta, Raymond Sawaya, and Gregory N. Fuller. "Origin of Chordoid Glioma of the Third Ventricle." Archives of Pathology & Laboratory Medicine 130, no. 4 (April 1, 2006): 460–64. http://dx.doi.org/10.5858/2006-130-460-oocgot.

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Abstract Context.—Chordoid glioma is a relatively recently described unique glial neoplasm that has been formally codified by the World Health Organization in Pathology and Genetics of Tumours of the Nervous System, in which it is included along with astroblastoma and gliomatosis cerebri under the rubric “Tumors of Uncertain Origin.” Many examples of chordoid glioma come to clinical attention only at a relatively large size and occupy a large portion of the third ventricle. Accordingly, the anatomic origin of chordoid glioma has been unclear and debated. Objective.—To examine the regional anatomic origin of chordoid glioma. Data Sources.—The clinical, imaging, histologic, immunophenotypic, and ultrastructural data in previously published case series and individual case reports of chordoid glioma were reviewed in conjunction with the study of a new case of chordoid glioma that presented at a relatively small size, thereby facilitating neuroanatomic localization. Conclusions.—Chordoid glioma exhibits features of specialized ependymal differentiation on ultrastructural examination, and all examples reported in the literature to date have displayed a highly stereotypical suprasellar anatomic localization and an ovoid shape, as seen on neuroimaging studies and gross anatomy. Neuroanatomic, radiologic, and clinical evidence supports an anatomic origin for chordoid glioma from the vicinity of the lamina terminalis.
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Ricoy, J. R., R. D. Lobato, B. Báez, A. Cabello, M. A. Martínez, and G. Rodríguez. "Suprasellar chordoid glioma." Acta Neuropathologica 99, no. 6 (June 9, 2000): 699–703. http://dx.doi.org/10.1007/s004010051183.

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Brat, D. J., B. W. Scheithauer, S. C. Cortez, K. Brecher, and P. C. Burger. "THIRD VENTRICULAR “CHORDOID GLIOMA”." Journal of Neuropathology and Experimental Neurology 56, no. 5 (May 1997): 586. http://dx.doi.org/10.1097/00005072-199705000-00072.

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Brat, Daniel J. "Chordoid glioma further defined." Advances in Anatomic Pathology 9, no. 1 (January 2002): 77. http://dx.doi.org/10.1097/00125480-200201000-00016.

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Buccoliero, Anna Maria, Adele Caldarella, Pasquale Gallina, Nicola Di Lorenzo, Antonio Taddei, and Gian Luigi Taddei. "Chordoid Glioma: Clinicopathologic Profile and Differential Diagnosis of an Uncommon Tumor." Archives of Pathology & Laboratory Medicine 128, no. 11 (November 1, 2004): e141-e145. http://dx.doi.org/10.5858/2004-128-e141-cgcpad.

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Abstract Chordoid glioma is an uncommon low-grade brain neoplasm arising in the third ventricular region, predominantly in middle-aged women. It characteristically shows chordoma-like histologic features and glial fibrillary acidic protein immunoreactivity. We present a case of chordoid glioma in a previously healthy 56-year-old woman admitted to our hospital because of a cranial trauma subsequent to an incidental fall. Radiologic examinations revealed a well-demarcated, partially cystic, enhancing mass at the level of the lamina terminalis. The lesion was surgically removed. The patient remained alive and well 8 months after the surgery. Histologically, the tumor consisted of clusters and cords of epithelioid cells embedded in a mucinous matrix. Lymphoplasmacytic infiltrates and Russell bodies were prominent. Immunohistochemically, the tumor cells were positive for glial fibrillary acidic protein, neurofilaments, and neuron-specific enolase, suggesting a divergent neuronal and glial differentiation. The Ki-67 index was low. The clinicopathologic profile and the differential diagnosis of this tumor are discussed.
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Sanda, Nicolae, Claudiu-Nicolae Mircea, Michèle Bernier, Avinoam B. Safran, and Sorin Aldea. "Chordoid Glioma Infiltrating Optic Structures." Journal of Neuro-Ophthalmology 39, no. 3 (September 2019): 408–10. http://dx.doi.org/10.1097/wno.0000000000000757.

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Castellano-Sanchez, Amilcar Antonio, Erwin Schemankewitz, Claire Mazewski, and Daniel J. Brat. "Pediatric Chordoid Glioma withChondroid Metaplasia." Pediatric and Developmental Pathology 4, no. 6 (November 1, 2001): 564–67. http://dx.doi.org/10.1007/s10024001-0087-1.

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Brat, Daniel J. "The Elusive Origin of Chordoid Glioma." Archives of Pathology & Laboratory Medicine 130, no. 4 (April 1, 2006): 437–38. http://dx.doi.org/10.5858/2006-130-437-teoocg.

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Dissertations / Theses on the topic "Chordoid Glioma"

1

Bellamy, Charlotte. "Functional characterization of a novel mutation in PRKCA, the major driver of Chordoid glioma." Electronic Thesis or Diss., université Paris-Saclay, 2024. http://www.theses.fr/2024UPASL052.

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Le gliome chordoïde (ChG) est une tumeur cérébrale rare de bas grade, caractérisée par une nouvelle mutation ponctuelle récurrente PRKCA p.D463H, une substitution dans le domaine kinase de la protéine kinase C alpha (PKCα). Cette étude démontre le rôle de cette kinase mutée dans le développement des ChG. Ici, nous montrons l'inactivation et l'effet négatif dominant de PKCαD463H via des tests d'activité in vitro et in cellulo. Nos résultats montrent que la mutation affecte la structure tertiaire, ce qui entraîne une protéine ouverte et instable. Les données de spectrométrie de masse phosphoprotéomique et de co-immunoprécipitation des cellules HEK surexprimant PKC⍺D463H montrent une diminution de phosphorylation et interaction avec les protéines impliquées dans l'adhésion cellulaire. Nous confirmons génétiquement par le RNAseq à noyau unique que les ChG dérivent de tanycytes spécialisés. En comprenant le contexte cellulaire de la tumorigenèse ainsi que les changements fonctionnels de cette mutation sur l'activité et l'interactome de PKCα, nous avons élaboré un modèle de développement des ChGs parallèlement à l'identification d'une approche thérapeutique
Chordoid glioma (ChG) is a rare low-grade brain tumor, characterised by a novel recurrent point mutation PRKCA p.D463H, a substitution in the kinase domain of Protein kinase C alpha (PKCα). This study demonstrates the role of this mutated kinase in the development of ChGs. Here we show the inactivation and dominant negative effect of PKCαD463H via in vitro and In cellulo activity assays. Our results show the mutation affects the tertiary structure, resulting in an open, unstable protein. Phosphoproteomic and Co-Immunoprecipitation mass spectrometry data from HEK cells overexpressing PKC⍺D463H show decreased phosphorylation and interaction with proteins involved in cell adhesion. We confirm genetically through single nuclei RNAseq that ChGs derive from specialised tanycytes. By understanding the cell context of tumorigenesis alongside the functional changes of this mutation on the activity and interactome of PKCα, we elaborated a model of the development of ChGs alongside the identification of a therapeutic approach
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Books on the topic "Chordoid Glioma"

1

van den Bent, Martin J., Frederic Dhermain, and Walter Stummer. Other neuroepithelial tumours: astroblastoma, angiocentric glioma, and chordoid glioma. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199651870.003.0007.

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This chapter contains a description of three rare entities: astroblastoma, angiocentric glioma, and chordoid glioma. Because these tumours are so rare, the evidence on how to best treat them is anecdotal and essentially consists of case series, but it is the best guidance available and the presentation of larger series with new cases remain unlikely. If one is confronted with such a case, a meticulous review of the clinical, radiological, and pathological characteristics of the case is indicated, to minimize the risk of an erroneous diagnosis. If the case does not fit in with the descriptions of previous cases, alternative diagnoses should be considered.
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Book chapters on the topic "Chordoid Glioma"

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Liu, Dongyou. "Chordoid Glioma, Angiocentric Glioma, and Diffuse Midline Glioma." In Tumors and Cancers, 31–36. Boca Raton : Taylor & Francis, 2018. | Series: Pocket guides to biomedical sciences | “A CRC title, part of the Taylor & Francis imprint, a member of the Taylor & Francis Group, the academic division of T&F Informa plc.”: CRC Press, 2017. http://dx.doi.org/10.1201/9781315120522-6.

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"Chordoid Glioma of 3rd Ventricle." In Diagnostic Pathology: Neuropathology, 150–53. Elsevier, 2016. http://dx.doi.org/10.1016/b978-0-323-44592-4.50027-5.

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"Chordoid Glioma of the Third Ventricle." In Diagnostic Imaging: Brain, 478–79. Elsevier, 2016. http://dx.doi.org/10.1016/b978-0-323-37754-6.50140-8.

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Gallia, Gary L., Martin G. Pomper, and Alessandro Olivi. "Chordoid Glioma of the Third Ventricle." In Textbook of Neuro-Oncology, 218–21. Elsevier, 2005. http://dx.doi.org/10.1016/b978-0-7216-8148-1.50032-2.

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Brat, Daniel. "Chordoid glioma of the third ventricle." In Russell & Rubinstein's Pathology of Tumors of the Nervous System 7Ed, 229–33. CRC Press, 2006. http://dx.doi.org/10.1201/b13439-17.

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You might also be interested in the extended bibliographies on the topic 'Chordoid Glioma' for particular source types:
Journal articles
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