Academic literature on the topic 'Choc hémorragique traumatique'
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Journal articles on the topic "Choc hémorragique traumatique":
Werner, Marie, and Anatole Harrois. "Cas clinique commenté: choc hémorragique traumatique." Anesthésie & Réanimation 8, no. 2 (March 2022): 188–92. http://dx.doi.org/10.1016/j.anrea.2022.01.003.
Sharifian, Léa, Samir Tine, Georges Sebbane, Robin Dhote, Chitra Sharifian, Stéphanie Clémencon, and Frédéric Pamoukdjian. "41. Un choc hémorragique post-traumatique." Soins Gérontologie 24, no. 139 (September 2019): 43–44. http://dx.doi.org/10.1016/j.sger.2019.07.009.
Morel, N., M. Biais, F. Delaunay, V. Dubuisson, O. Cassone, F. Siméon, O. Morel, and G. Janvier. "Érythrocytes et tonus microvasculaire au cours du choc hémorragique traumatique." Annales Françaises d'Anesthésie et de Réanimation 32, no. 5 (May 2013): 339–46. http://dx.doi.org/10.1016/j.annfar.2013.02.025.
Martin, C., and R. Domergue. "Prise en charge préhospitalière et hospitalière précoce d'un état de choc hémorragique d'origine traumatique." Annales Françaises d'Anesthésie et de Réanimation 16, no. 8 (December 1997): 1030–36. http://dx.doi.org/10.1016/s0750-7658(97)82152-1.
Ait Tamlihat, Y., J. E. Bourcier, V. Brisseau, and P. Lazzerini. "Choc hémorragique sur hématome rétropéritonéal post-traumatique révélant un rein en fer à cheval." Annales Françaises d'Anesthésie et de Réanimation 32, no. 10 (October 2013): 725–27. http://dx.doi.org/10.1016/j.annfar.2013.07.817.
Jost, Daniel. "Administration préhospitalière de plasma lyophilisé « PLYO » au cours d’un choc hémorragique post-traumatique : étude « PREHO-PLYO »." Transfusion Clinique et Biologique 25, no. 4 (November 2018): 303. http://dx.doi.org/10.1016/j.tracli.2018.08.090.
Perez, Pauline, Anatole Harrois, Mathieu Raux, Sophie Hamada, Catherine Paugam-Burtz, and Tobias Gauss. "Évaluation de la performance du théorème de Bayés pour prédire la survenue d’un choc hémorragique post-traumatique." Anesthésie & Réanimation 1 (September 2015): A5—A6. http://dx.doi.org/10.1016/j.anrea.2015.07.008.
Morel, N., O. Morel, L. Chimot, V. Lortet, B. Julliac, A. Lelias, L. Merson, and Ph Dabadie. "Prise en charge transfusionnelle du choc hémorragique d’origine traumatique à la phase aiguë : quoi de neuf en 2009 ?" Annales Françaises d'Anesthésie et de Réanimation 28, no. 3 (March 2009): 222–30. http://dx.doi.org/10.1016/j.annfar.2008.12.023.
Bacus, Morgane, Benoît Tavernier, Sophie Susen, Eric Kipnis, Anne Guidat, and Delphine Garrigue. "Audit clinique ciblé sur la prise en charge des 24 premières heures d’un patient en choc hémorragique d’origine traumatique." Anesthésie & Réanimation 1 (September 2015): A165. http://dx.doi.org/10.1016/j.anrea.2015.07.255.
Deras, P., M. Villiet, P. Latry, X. Capdevila, and J. Charbit. "Évaluation des effets adverses d’une stratégie d’administration précoce de concentrés de complexe prothrombinique chez les patients en choc hémorragique d’origine traumatique." Transfusion Clinique et Biologique 22, no. 4 (September 2015): 202. http://dx.doi.org/10.1016/j.tracli.2015.06.255.
Dissertations / Theses on the topic "Choc hémorragique traumatique":
Poloujadoff, Marie-Pierre. "Evaluation de différentes stratégies thérapeutiques pour la prise en charge initiale du choc hémorragique traumatique." Paris 13, 2008. http://www.theses.fr/2008PA132022.
Patients with profound hypotension in the pre-hospital phase have a high mortality. Patients who recover their blood pressure before hospital admission have better outcome, which emphasizes the importance of pre-hospital resuscitation. Our experimental model consists in an uncontrolled hemorrhagic shock (UHS) associated or not with a lateral fluid percussion trauma head injury (LFP) in rats. During USH, an infusion of hypertonic-hyperoncotic solution may not increase the rate of survival compared to conventional treatment, and appears to be associated with renal toxicity. Using norepinephrine alows a normotensive resuscitation, and is beneficial on survival, bleeding, and resuscitation volum. When USH is associated with LFP, using isotonic saline in a normotensive resuscitation leads to an increase in mortality. However, the early use of norepinephrine significantly improves survival in normotensive resuscitation
Dufour-Gaume, Frédérique. "Enjeux, préparation et évaluation de produits sanguins labiles innovants adaptés aux blessés de guerre." Electronic Thesis or Diss., université Paris-Saclay, 2023. http://www.theses.fr/2023UPASQ076.
War casualties associate multiple injuries with shock hemorrhage. Despite the therapeutic progress of recent years, hemorrhage is the leading cause of preventable deaths and secondary multiple organ failure can lead to vital and functional prognosis. Management of war-injured patients based on damage control resuscitation and massive transfusion of whole blood reduced considerably the number of deaths. Nevertheless, during foreign operations whole blood is sometimes lacking because of logistic limitations and massive casualties. Modified blood products that are free from constraints could help war-injured patients to survive. To achieve this objective, we developed two French hyper-concentrated lyophilized plasmas with (FLYP-H/LP) or not (FLYP-H) lyophilized platelets. The production of these products is of a high quality. FLYP-H and FLYP-H/LP are high protein products, especially albumin, which confer them a hyperosmolarity twice that of plasma. In FLYP-H/LP, platelets were lysed during the manufacturing process and liberated high quantities of coagulation factors, such as fibrinogen. The therapeutic effects of FLYP-H and FLYP-H/LP were evaluated thanks to our war-injured porcin model. Statistical analysis highlighted the beneficial effects of FLYP-H and FLYP-H/LP on cardiovascular function and hemostasis. These results open the door to more analysis but on human FLYP-H and FLYP-H/LP
Avaro, Jean-Philippe. "Intérêts et limites du clampage endovasculaire de l'aorte thoracique en situation de choc hémorragique non contrôlé lié à un traumatisme abdominal sur un modèle animal." Thesis, Aix-Marseille 2, 2011. http://www.theses.fr/2011AIX20667.
Trauma is the leading cause of mortality in industrialized countries for people aged below 40 years. Fifty percent of the pre hospital and in hospital mortality from severe blunt and penetrating abdominal traumas is due to an hemorrhagic shock. Peritoneal bloody effusion is the main reason to under estimate the seriousness of trauma.Damage control resuscitation (DCR) and damage control surgery (DCS) typify the current paradigm of hemorrhagic torso trauma management. Damage control includes a basic pre operative management before a short surgical control of bleeding followed by intensive resuscitation care based on massive blood transfusion, palliation of hypothermia and correction of biological coagulation disorders. According to this strategy, the curative surgical treatment is postponed until the patient has been stabilized.Some authors have reported on the efficacy of resuscitation thoracotomy with aortic crossclamping in the emergency room in patients with severe abdominal trauma . However, the end results of such a procedure are contrasted and its use is still debated. More recently, endovascular approach has emerged in the management algorithm of some vascular emergencies. We hypothesized that an endovascular retrograde occlusion of the thoracic aorta would be a safe and efficient to preserve hemodynamic profile in cardiac and cerebral area, and to improve survival in case of uncontrolled hemorrhagic shock caused by an abdominal trauma.Our results sustain this hypothesis, even if its benefits seem time-limited, according to the medullar and visceral side-effects of ischemia/reperfusion
Prunet, Bertrand. "Contusion pulmonaire : aspects physiopathologiques et conséquences thérapeutiques." Thesis, Aix-Marseille, 2015. http://www.theses.fr/2015AIXM5001.
Pulmonary contusion is often associated with hemorrhagic shock, constituting a challenge in trauma care. For patients who have sustained lung contusions, fluid resuscitation should be carefully performed, because injured lungs are particularly vulnerable to massive fluid infusions with an increased risk of pulmonary edema and compliance impairment. Fluid administration should be included in an optimized and goal directed resuscitation, based on blood pressure objectives and hemodynamical monitoring. The use of fluids with high volume-expanding capacities (hypertonic saline, colloids) is probably interesting, as well as early introduction of vasopressors. Hemodynamic monitoring will allow to conduct resuscitation on blood pressure objectives, on preload parameters and on extravascular lung water measurement.Our work, based on experimental and clinical studies, objective to characterize the current modalities of ventilatory and hemodynamical aspect of pulmonary contusion care
Khazoom, François. "Rôle de l’acide urique dans la défaillance d’organes suite au choc hémorragique : une avenue thérapeutique?" Thesis, 2020. http://hdl.handle.net/1866/24497.
While hemorrhagic shock is the first cause of early mortality among severe trauma patients, organ failure leads to late mortality and morbidity in this population. Alarmins, molecules released after ischemia-reperfusion, are able to activate local and systemic inflammatory pathways and potentially represent a therapeutic target to minimize organ failure. Uric acid is a pro-inflammatory and pro-apoptotic molecule released after hemorrhagic shock and its role pertaining to organ failure is incompletely studied. The first part of this thesis presents a proof of concept that uric acid plays a key role in liver and intestinal damage in an animal model of hemorrhagic shock; it will be presented in the format of a submitted article. The second part of this thesis presents preliminary data from a prospective observational clinical study evaluating uric acid kinetics in a cohort of trauma patients. Animal study Hemorrhagic shock was induced with blood withdrawal among Wistar rats for a target mean arterial blood pressure of 30-35 mmHg for 60 minutes. Animals were resuscitated with a 1 :1 mix of Ringer Lactate and drawn blood with or without Uricase, a recombinant enzyme that metabolizes uric acid. Results show a statistically significant decrease in hepatocellular damage (plasma AST and ALT), inflammatory markers (ICAM-1, MPO, TNF-alpha, IL-1, Caspase-1) and apoptotic markers (Caspase-3, -8, Bax/BCL-2, pAKT/AKT) among the Uricase group. The intervention on uric acid also prevented increased intestinal permeability and bacterial product (LPS) translocation. Clinical study Twenty patients sustaining major trauma with hemorrhagic shock were prospectively recruited at Montreal Sacré-Cœur Hospital, in the context of a pilot study funded by the FRSQ trauma consortium. Uric acid concentration was determined serially for 7 days after trauma. Feasibility criteria, notably consent rate (95%), sampling observance rate (90% for first sample, 65% for samples every 4 hours, and 73% for samples every 8 hours) were considered acceptable. Uric acid kinetics were reproducible among the entire cohort (R2 = 0.87). The area under the curve was significantly increased among patients with higher sequential organ failure assessment score at 72h (SOFA³6). Conclusions Although mechanisms remain to be elucidated, these studies show that uric acid is an important mediator for the development of organ damage after hemorrhagic shock. This molecule potentially represents a therapeutic target with the ultimate goal of minimizing organ failure after hemorrhagic shock.