Relevant bibliographies by topics / Childhood smoking

Academic literature on the topic 'Childhood smoking'

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Published: 4 June 2021
Last updated: 31 January 2023

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Journal articles on the topic "Childhood smoking"

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HipoLito, Junior, J. J. Jorge, Almeida de, C. Scully, and J. Bowden. "Childhood tobacco smoking." British Dental Journal 171, no. 7 (October 1991): 195. http://dx.doi.org/10.1038/sj.bdj.4807658.

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Landau, Louis I. "Smoking and childhood asthma." Medical Journal of Australia 154, no. 11 (June 1991): 715–16. http://dx.doi.org/10.5694/j.1326-5377.1991.tb121304.x.

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LANDAU, L. "Passive smoking in childhood." Journal of Paediatrics and Child Health 24, no. 5 (October 1988): 271–72. http://dx.doi.org/10.1111/j.1440-1754.1988.tb01359.x.

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Di Benedetto, G. "Passive Smoking in Childhood." Journal of the Royal Society of Health 115, no. 1 (February 1995): 13–16. http://dx.doi.org/10.1177/146642409511500105.

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Rosenberg, Karen. "Childhood and Adolescent Smoking Linked to Adult Smoking." AJN, American Journal of Nursing 120, no. 8 (August 2020): 49. http://dx.doi.org/10.1097/01.naj.0000694576.07706.ec.

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Taryaka, Apriyani, and Evi Afifah Hurriyati. "Mengapa Late Childhood Merokok?" Humaniora 2, no. 1 (April 30, 2011): 405. http://dx.doi.org/10.21512/humaniora.v2i1.3050.

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This research is set to determine the cause of late childhood smoking and whether the factor behind late child hood smoking is the same with teenagers. The background of this research are the findings of Tobacco Control Support Center that 3 out of 10 students are found to be smoking before reaching the age of 10. This research uses qualitative case study research through observation and in-depth direct interview towards 3 male subjects aged 11 who smokes every day. Results show that the 3 subjects smoke due to personal factor, friends, family and cigarette advertisements. Most of the factor behind the smoking behavior are found to be in the sociogenic motive category. Therefore, it could be concluded that the smoking behavior of the 3 subjects is not purely from the personal factor, but more of the environmental factor having big part in creating smoking behavior in the 3 subjects. Factors behind smoking behavior of the three late childhood subjects and teenagers have a lot in common. Friend factor is the first driving factor of smoking behavior on both late childhood and teenager.
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Whatling, Jill. "Childhood asthma and passive smoking." Nursing Standard 8, no. 46 (August 10, 1994): 25–27. http://dx.doi.org/10.7748/ns.8.46.25.s44.

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Millichap, J. Gordon. "Maternal Smoking and Childhood Hyperactivity." Pediatric Neurology Briefs 17, no. 7 (July 1, 2003): 56. http://dx.doi.org/10.15844/pedneurbriefs-17-7-11.

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Weitzman, Michael, Steven Gortmaker, Deborah Klein Walker, and Arthur Sobol. "Maternal Smoking and Childhood Asthma." Pediatrics 85, no. 4 (April 1, 1990): 505–11. http://dx.doi.org/10.1542/peds.85.4.505.

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According to a substantial literature, passive smoking by children is associated with an increased incidence of lower respiratory illness and diminished pulmonary function. The relationship between passive smoking and childhood asthma, however, is not clear. Data from the Child Health Supplement to the 1981 National Health Interview Survey were analyzed with information about 4331 children aged 0 to 5 years to study the relationship between maternal smoking and (1) the prevalence of childhood asthma, (1) the likelihood of taking asthma medication, (3) the age of onset of children's asthma, and (4) the number of hospitalizations among children with and without asthma. An odds ratio for asthma of 2.1 was shown by multivariate logistic regressions among children whose mothers smoke 0.5 packs of cigarettes or more per day compared with children of nonsmokers (P = .001). In similar analyses maternal smoking of 0.5 packs per day was identified as an independent risk for children's use of asthma medications (odds ratio 4.6, P = .0006) and for asthma developing in the first year of life (odds ratio 2.6, P = .0006). Maternal smoking is also associated with increased numbers of hospitalizations by its association with an increased risk of asthma as well as by contributing to hospitalizations independently of a child having asthma. Among children with asthma, however, maternal smoking is not associated with increased numbers of hospitalizations. It was concluded that maternal smoking is associated with higher rates of asthma, an increased likelihood of using asthma medications, and an earlier onset of the disease. These findings have implications for renewed efforts to discourage smoking in families, especially during pregnancy and the first 5 years of children's lives.
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Willers, S., E. Svenonius, and G. Skarping. "Passive smoking and childhood asthma." Allergy 46, no. 5 (July 1991): 330–34. http://dx.doi.org/10.1111/j.1398-9995.1991.tb00595.x.

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Dissertations / Theses on the topic "Childhood smoking"

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Woodward, Alistair. "Passive smoking and acute respiratory illness in childhood." Title page, contents and summary only, 1988. http://web4.library.adelaide.edu.au/theses/09PH/09phw899.pdf.

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Lee, So-lun. "Foetal exposure to passive maternal smoking and childhood asthma." View the Table of Contents & Abstract, 2006. http://sunzi.lib.hku.hk/hkuto/record/B36887468.

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Lee, So-lun, and 李素輪. "Foetal exposure to passive maternal smoking and childhood asthma." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2006. http://hub.hku.hk/bib/B39724840.

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WANG, Calvin, and c. wang@ecu edu au. "EXPLORING YOUNG PEOPLE'S CONCEPTS OF SMOKING ADDICTION: PERCEIVED OPPORTUNITIES TO TRY SMOKING WITHOUT BECOMING ADDICTED." Edith Cowan University. Business And Law: School Of, 2006. http://adt.ecu.edu.au/adt-public/adt-ECU2006.0033.html.

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This study explores how young people conceptualise addiction to smoking and, also the relationship between young people's addiction beliefs and intentions to smoke cigarettes. Addiction to smoking is a major health problem, not just for adults, but also for young smokers, up to 60% of whom are dependent on nicotine. However, anti-smoking prevention efforts targeted at young people generally emphasise ill-health effects and little attention is paid to addiction education which is generally considered relevant only to adult smoking and cessation efforts. Perhaps as a consequence, young people appear to have many misconceptions and unrealistic ideas about addiction, and these may possibly have influenced initial decisions to take up smoking. For example, between 50% and 60% of young smokers believe that it would be easy or very easy to stop smoking altogether if and when they choose to and the majority of daily smokers mistakenly believe that they will not be smoking for more than five years. For these young smokers, becoming addicted is often an unforeseen consequence and most are surprised to find that they cannot give up smoking as easily as they thought. The majority of addicted smokers regret ever taking up smoking but nevertheless continue to smoke cigarettes for perhaps 30 to 40 years because they find it very difficult to stop. This backdrop provides the impetus for the present study.
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Wang, Calvin. "Exploring young people's concepts of smoking addiction: Perceived opportunities to try smoking without becoming addicted." Thesis, Edith Cowan University, Research Online, Perth, Western Australia, 2006. https://ro.ecu.edu.au/theses/102.

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This study explores how young people conceptualise addiction to smoking and, also the relationship between young people's addiction beliefs and intentions to smoke cigarettes. Addiction to smoking is a major health problem, not just for adults, but also for young smokers, up to 60% of whom are dependent on nicotine. However, anti-smoking prevention efforts targeted at young people generally emphasise ill-health effects and little attention is paid to addiction education which is generally considered relevant only to adult smoking and cessation efforts. Perhaps as a consequence, young people appear to have many misconceptions and unrealistic ideas about addiction, and these may possibly have influenced initial decisions to take up smoking. For example, between 50% and 60% of young smokers believe that it would be easy or very easy to stop smoking altogether if and when they choose to and the majority of daily smokers mistakenly believe that they will not be smoking for more than five years. For these young smokers, becoming addicted is often an unforeseen consequence and most are surprised to find that they cannot give up smoking as easily as they thought. The majority of addicted smokers regret ever taking up smoking but nevertheless continue to smoke cigarettes for perhaps 30 to 40 years because they find it very difficult to stop. This backdrop provides the impetus for the present study.
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Kauffman, Ross M. "Smoking and Tobacco in Ohio Prisons." The Ohio State University, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=osu1243363292.

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Jaakkola, Niina. "Passive smoking during pregnancy and early childhood : occurrence, determinants, health effects and prevention." Helsinki : University of Helsinki, 2002. http://ethesis.helsinki.fi/julkaisut/laa/kansa/vk/jaakkola/.

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Strine, Tara Wynn. "The mediating role of psychological distress in the relationship between adverse childhood experiences and adult smoking." ScholarWorks, 2010. https://scholarworks.waldenu.edu/dissertations/791.

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While research has indicated that impaired mental health partially mediates the relationship between adverse childhood experiences (ACEs) and alcohol and illicit drug use, little research has examined potential mediators in the relationship between ACEs and smoking, the number one cause of preventable mortality in the United States. Accordingly, this study examined the potential mediating effect of psychological distress on the relationship between ACES and smoking using data from Wave II of the ACE Study, a cross-sectional study completed between June and October of 1997 on a sample of adult health maintenance organization members (N = 7,211). The theoretical underpinnings for this study were grounded in the developmental psychopathological perspective which examines both environmental and biological influences as they interact to promote or impede social, emotional, and behavioral development. Mediation modeling employing both linear and logistic regression techniques indicated that, after adjusting by select covariates, psychological distress (as assessed using the SF-36 Mental Component Summary score) partially mediated the relationship between several of the ACEs examined and smoking in women. These same relationships were not found in men. This research contains several key findings with social change implications. First, additional research should be conducted to examine the causes, developmental paths, and critical points that link ACEs and psychological distress to smoking among women. Second, given the gender differences in the association between ACEs and smoking, gender-specific intervention programs that build resiliency, increase positive social support, and provide tools for developing alternative coping strategies may be important adjuncts to smoking cessation programs, particularly for women with a history of ACEs.
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Pugmire, Juliana. "Health Effects of Childhood Exposure to Environmental Tobacco Smoke in Children followed to Adulthood." Diss., The University of Arizona, 2011. http://hdl.handle.net/10150/202985.

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Background A significant proportion of children are exposed to environmental tobacco smoke (ETS) throughout the world. This is mainly because of exposure to parental smoking. It is unknown to what extent the negative effects of ETS on respiratory symptoms track from childhood into adulthood. Methods TESAOD (Tucson Epidemiologic Study of Airway Obstructive Disease) is a large population-based prospective study that was initiated in 1972. Participants were followed prospectively with questionnaires and pulmonary function tests (PFTs) completed about every two years in 12 follow-up surveys up to 1996. Skin prick tests and blood samples for IgE measurements were collected at surveys 1, 6, and 11. We identified subjects who entered the study as children (<15 years old) and were followed to adulthood (>18 years) during the study follow-up. Based on questionnaire data, active asthma, wheeze, cough, and chronic cough (cough for three consecutive months) were coded as never (never reported in childhood or adulthood), incident (never reported in childhood, but ≥ one positive report in adulthood), remittent (≥ one positive report in childhood, but not in adulthood), and persistent (≥ one positive report both in childhood and adulthood). PFTs measurements included forced expiratory volume in 1 second, forced vital capacity, and forced expiratory flow at 25-75%. Parent information on smoking status was collected simultaneously at child visits. ETS exposure status was assessed as “ever” or “never” between birth and 15 years. Results Information on parental ETS exposure in childhood and outcomes in adulthood was available for 444 non-Hispanic white participants (51.4% male) with mean age at initial survey of 7.7 years. Total mean follow-up time was 19.0 years (8.8 years in adulthood). Between birth and 15 years, 53.4% of children were exposed to ETS. After adjusting for sex, age at enrollment, years of follow-up, and personal smoking status (assessed at age 15 and above), combined parental ETS exposure in childhood was significantly associated with persistent wheeze (RR(adj) 1.9, p=0.026), persistent cough (RR(adj) 5.9, p<0.001), and persistent (RR(adj) 3.7, p=0.030) and incident chronic cough (RR(adj) 2.3, p=0.040). Paternal ETS exposure in childhood was associated with persistent wheeze (RR(adj) 2.3, p=0.002), persistent cough (RR(adj) 3.9, p<0.001), persistent (RR(adj) 4.8, p=0.004) and incident chronic cough (RR(adj) 2.2, p=0.031), and persistent asthma (RR(adj) 2.3, p=0.016). Maternal ETS exposure was associated with persistent (RR(adj) 1.9, p=0.029) and incident cough (RR(adj) 2.5, p=0.006). Maternal ETS exposure was associated with an increased percent predicted FVC in adulthood (coefficient, 3.75; p=0.019). No other effects on lung function were seen. There were no effects of ETS exposure on total serum IgE or allergic sensitization. ETS exposure was associated with respiratory symptoms in adulthood among both never and current smokers. Conclusions ETS exposure in childhood has long term health effects on lung function and respiratory symptoms. These effects do not appear to be IgE-mediated. ETS exposure, especially paternal ETS exposure, seems to influence the persistence of respiratory symptoms from childhood to adulthood and to affect women more than men. These effects are independent of personal smoking and also seen in never smokers. Both smoking mothers and fathers should be targeted when attempting to reduce ETS exposure among children.
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Yahaya, Ismail. "Childhood Sexual Abuse Against Girls in Sub-Saharan Africa : Individual and Contextual Risk Factors." Doctoral thesis, Mittuniversitetet, Avdelningen för hälsovetenskap, 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:miun:diva-21919.

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Background and objectives: Childhood sexual abuse (CSA) is a substantial public health and human rights problem, as well as a growing concern in sub-Saharan Africa (SSA). It has both short and long term effects on girls: physical and psychological, including negative sexual outcomes. Up to one-third of adolescent girls report their first sexual experience as being forced. Despite growing evidence supporting a link between contextual factors and violence, no studies have investigated the connection between CSA and contextual factors. It is therefore important to identify the extent of CSA and understand factors associated with it in SSA in order to develop interventions aimed to address the scale of the problem. Aim: The overall aim of this thesis is to assess the individual and contextual factors associated with CSA. In addition, the thesis aims to quantify the magnitude of CSA and describe the factors associated with CSA among women from SSA (Study I). This thesis also examines the independent contribution of individual and community socio-economic status on CSA (Study II). Moreover, it scrutinises the effect of social disorganisation on CSA (Study III) and explores the relationship between CSA and sexual risk behaviours as well as potential mediators (Study IV). Methods: This thesis used the Demographic and Health Survey (DHS) datasets conducted between 2006 and 2008 from six SSA countries. The thesis used multiple logistic regression models to describe and explore factors associated with CSA among 69,977 women (Study I).  It used multivariable multilevel logistic regression analysis to explore the effect of contextual level variables (neighbourhood socio-economic status) on CSA among 6,351 girls (Study II). Neighbourhood socio-economic status was operationalized with a principal component analysis using the proportion of respondents who were unemployed, illiterates, living below poverty level and rural residents. Study III applied multivariable multilevel logistic regression analysis on 6,351 girls and considered five measures of social disorganisation at the community level: neighbourhood poverty, female-headed households, residential mobility, place of residence, population density, and ethnic diversity. In study IV, 12,800 women from the Nigerian DHS were used. Structural equation modelling was applied using a two-step approach. The first step used a confirmatory factor analysis to develop an acceptable measurement model while the second step involved modifying the measurement model to represent the postulated causal model framework. Results: In study I, the reported prevalence of CSA ranged from 0.3% in Liberia to 4.3% in Zambia when the prevalence was based on all respondents aged between 15 and 49 years and who were present during the survey. None of the socio-economic factors were associated with CSA. In study II, where the data was restricted to permanent residents aged between 15 and 18 years, the prevalence ranged between 1.04% in Liberia to 5.8% in Zambia. At the individual level, there was no significant association between CSA and wealth status while at the community level, there was no significant association between CSA and socio-economic position. However, 22% of the variation in CSA was attributed to the community level factors. In study III, there was significant variation in the odds of reporting CSA across the communities, with community level factors accounting for 18% of the variation. In addition, respondents from communities with a high family disruption rate were 57% more likely to have reported sexual abuse in childhood. Study IV showed that there was a significant association between CSA and sexual risk behaviours and the association was mediated by alcohol and cigarette use. Conclusions: The study provides evidence that adolescents in the same community were subjected to common contextual influences. It also highlighted the significance of mediators in the relationship between CSA and sexual risk behaviours. It is therefore important that effective preventive strategies are developed and implemented that will cut across all socio-economic spheres in a context that both permits and encourages disclosure as well as identifying predisposing circumstances for recurrence.
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Books on the topic "Childhood smoking"

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Cheung, Philip. Childhood smoking: Incidence, interrelationships and interventions. London: Smith-Gordon, 2001.

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Cheung, P., and D. Wooff. Childhood Smoking. Smith-Gordon & Co Ltd, 2002.

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J, Moss Abigail, and National Center for Health Statistics (U.S.), eds. Prenatal smoking and childhood morbidity. Hyattsville, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Center for Health Statistics, 1987.

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J, Moss Abigail, and National Center for Health Statistics (U.S.), eds. Prenatal smoking and childhood morbidity. Hyattsville, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Center for Health Statistics, 1987.

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J, Moss Abigail, and National Center for Health Statistics (U.S.), eds. Prenatal smoking and childhood morbidity. Hyattsville, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Center for Health Statistics, 1987.

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Jin, Cui. The effects of passive smoking on respiratory illness in early childhood in Shanghai, P.R.China. 1993.

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Levy, David. Adolescence and emerging adulthood. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198766452.003.0009.

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Adolescence and emerging childhood forms an increasing proportion of the lifespan of urbanized individuals. Glycaemic control worsens during adolescence; physiology and psychology contribute. A1C levels peak around 9% (75 mmol/mol) before declining from late teens onwards. However, unchanging glycaemia (tracking) is common. Glycaemia has generally improved in the past 10–15 years, but significant differences between and within countries persist. Microvascular complications are prevalent at this stage, but have probably also decreased with time. During this important period, the stage can be set for premature macrovascular disease (early onset hypertension, arterial stiffening, dyslipidaemia, and smoking). Exercise reduces the risk of microvascular complications. Smoking is as common in young Type 1 patients than in the general population. Efforts at smoking cessation need reinforcing. Glycaemic control during university does not improve. Transition from paediatric to adult diabetes services is often unsatisfactory; clinics should implement simple procedures focusing on accessibility, flexibility, and improved communications.
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Wilens, Timothy, Nicholas Carrellas, and Joseph Biederman. ADHD and substance misuse. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198739258.003.0023.

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There has been great interest in the overlap between ADHD and substance use disorder (SUD). ADHD is a common neurobehavioural disorder of childhood that places the individual at elevated risk for later SUD. Studies have shown that 25–40% of adults and adolescents with SUD have ADHD. Although the exact link between the two disorders is still unclear, it appears their connection is complex, and involves the interplay between various biological, behavioural, and genetic factors. Early pharmacotherapy of ADHD does not increase SUD, and, in fact, appears to reduce cigarette smoking and SUD. In individuals with ADHD and SUD, stabilization of SUD is recommended initially, with consideration of adjunct non-stimulant and extended release stimulant medications. More research on the mechanisms of overlap between the disorders, preventative effects of early ADHD treatment on SUD, and concurrent treatments for ADHD and SUD are necessary.
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Puttler, Leon I., Robert A. Zucker, and Hiram E. Fitzgerald. Developmental Science, Alcohol Use Disorders, and the Risk–Resilience Continuum. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190676001.003.0001.

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The origins and expression of addiction are best understood within the context of developmental processes and dynamic systems organization and change. For some individuals, these dynamic processes lead to risk cumulative or cascade effects that embody adverse childhood experiences that exacerbate risk; predict early onset of drinking, smoking, or other substance use; and often lead to a substance use disorder (SUD) during the transitions to adolescence and emergent adulthood. In other cases, protective factors within or outside of the individual’s immediate family enable embodiment of normative stress regulatory systems and neural networks that support resilience and prevention of SUDs. A case study is provided to illustrate these processes and principles of the organization of addictive behavior. Finally, a model of risk to resilience captures the flow of development and the extent to which individual-experience relationships contribute to risk and resilience.
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Book chapters on the topic "Childhood smoking"

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Chang, Jeffrey S. "Parental Smoking and Childhood Leukemia." In Methods in Molecular Biology, 103–37. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-492-0_5.

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Wojcik, Katherine J., and Elizabeth J. Mayer-Davis. "Maternal Determinants of Childhood Obesity: Weight Gain, Smoking, and Breastfeeding." In Pediatric Obesity, 93–102. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-60327-874-4_7.

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Stanton, Warren R. "The Dunedin Study of Childhood and Adolescent Smoking: Selected Findings." In Tobacco and Health, 391–99. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4615-1907-2_82.

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Hollis, Jenna, Hazel Inskip, and Siân Robinson. "Maternal Determinants of Childhood Obesity: Maternal Obesity, Weight Gain and Smoking." In Contemporary Endocrinology, 205–13. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-68192-4_11.

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Yeoman, Kristin, Thomas Safranek, Bryan Buss, Betsy Cadwell, and David Mannino. "Adverse Childhood Experiences and Adult Smoking, Nebraska, 2011." In The Societal Burden of Child Abuse, 149–60. Apple Academic Press, 2015. http://dx.doi.org/10.1201/b18768-13.

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"Adverse Childhood Experiences and Adult Smoking, Nebraska, 2011." In The Societal Burden of Child Abuse, 179–90. Apple Academic Press, 2015. http://dx.doi.org/10.1201/b18768-17.

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Tyc, Vida L. "Prevention and Cessation of Tobacco Use and Exposure to Environmental Tobacco Smoke." In Comprehensive Handbook of Childhood Cancer and Sickle Cell Disease. Oxford University Press, 2006. http://dx.doi.org/10.1093/oso/9780195169850.003.0029.

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Tobacco use remains the single most important preventable cause of premature death and disability in the United States and is a critical health issue for our nation’s youths. Cigarette smoking is the most common form of tobacco use among adolescents (Centers for Disease Control and Prevention, 2001), with over 90% of adult smokers initiating smoking at or before age 19 years (Mowery, Brick, & Farrelly, 2000). Consequently, reduction of tobacco use during adolescence is especially critical before lifelong smoking habits are established. Current national health objectives for children and adolescents focus on reducing health risks related to tobacco use and exposure to secondhand smoke (U.S. Department of Health and Human Services, 2000). Specific objectives include reducing the initiation of tobacco use among children and adolescents, reducing their average age of first use of tobacco products, increasing cessation attempts by current smokers, and reducing the proportion of children who are regularly exposed to tobacco smoke in the home. These health objectives are especially important for children and adolescents with cancer, who may be at even greater risk than their healthy peers for tobacco-related health problems because of their compromised health status (Hollen & Hobbie, 1996). Exposure to environmental tobacco smoke (ETS) has similar serious consequences for the child with cancer (Alligne & Stoddard, 1997; Cook & Strachan, 1999). Interventions that attempt to prevent, reduce, or terminate tobacco use and ETS exposure could therefore contribute to a decrease in the morbidity and mortality of patients treated for cancer. This chapter reviews the prevalence of tobacco use, the magnified health effects associated with tobacco use, and some of the correlates associated with tobacco use among young patients treated for cancer. We also describe tobacco interventions that have been conducted with this population and discuss how health care providers involved in the treatment or long-term care of childhood cancer patients can assist their high-risk patients in making healthy lifestyle choices, including the decision to abstain from, reduce, or quit smoking and to avoid environmental tobacco exposures. Tobacco use is a significant behavioral health problem that poses serious health risks for young patients treated for cancer.
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Sockrider, Marianna, and Harold J. Farber. "Secondhand Tobacco Smoke Exposure and Active Smoking in Childhood and Adolescence." In Pediatric Pulmonology, 1017–33. American Academy of Pediatrics, 2011. http://dx.doi.org/10.1542/9781581104936-10-ch52.

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Charakida, Marietta, and John Deanfield. "Arterial disease in the young." In ESC CardioMed, 775–78. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0179.

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The growing adult population with congenital heart disease (CHD) are getting older and their clinical presentation and outcomes are likely to be determined not only by their underlying CHD, but also by the development of acquired cardiovascular diseases such as atherosclerosis. This begins in childhood. The adult CHD population have multiple cardiovascular risk factors, as found in the general population, including obesity, smoking, and sedentary behaviour. Adults with complex congenital heart circulation are likely to be particularly vulnerable to the development of acquired arterial disease. Maintenance of ‘ideal cardiovascular health’ from childhood, is a key target, as leveraged gains can be achieved by early intervention to reduce risk factor exposure.
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Pitchforth, Jacqueline, and Dougal Hargreaves. "Adolescent non-communicable diseases." In Oxford Textbook of Global Health of Women, Newborns, Children, and Adolescents, edited by Delan Devakumar, Jennifer Hall, Zeshan Qureshi, and Joy Lawn, 65–69. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198794684.003.0013.

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Four non-communicable diseases (NCDs): cardiovascular disease, cancer, diabetes, and chronic respiratory conditions, are responsible for 63% of deaths worldwide. Most of these deaths (86%) occur in low and middle-income countries, where the highest proportion of adolescents live. Four shared behavioural risk factors for NCDs (tobacco use, unhealthy diet, physical inactivity, and harmful use of alcohol) are usually acquired during adolescence and persist throughout life. For example, globally 100,000 young people start smoking each day and over 90% of adults who smoke started during childhood or adolescence. This chapter will explore each of these risk factors, the impact on adolescent health and what steps are being taken to address these problems, as well as the contribution of chronic disease to the NCDs.
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Conference papers on the topic "Childhood smoking"

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Ayubi, Anwar, MAS Ahmed, and Olajumoke Davies. "P533 Childhood obesity and cigarette smoking during pregnancy." In Faculty of Paediatrics of the Royal College of Physicians of Ireland, 9th Europaediatrics Congress, 13–15 June, Dublin, Ireland 2019. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2019. http://dx.doi.org/10.1136/archdischild-2019-epa.868.

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Kasteler, Rahel, Fabiën Belle, Fabienne Gumy-Pause, Eva M. Tinner, and Claudia Kuehni. "Prevalence and reasons for smoking in adolescent Swiss childhood cancer survivors." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa2995.

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Haberg, SE, YE Bentdal, SJ London, H. Stigum, KJ Kvaerner, W. Nystad, and P. Nafstad. "Pre- and Postnatal Parental Smoking and Acute Otitis Media in Early Childhood." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a4804.

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Fouzas, Sotirios, Panagiotis Lambropoulos, Styliani Malliori, Olga Lagiou, Kostas Priftis, and Michael Anthracopoulos. "Parental smoking habits and prevalence trends of childhood asthma: four surveys during 1998-2013." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa1858.

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Wu, Pingsheng, William Dupont, Kecia Carroll, Tebeb S. Gebretsadik, Edward Mitchel, and Tina V. Hartert. "Infant Bronchiolitis Modifies The Effect Of Maternal Smoking During Pregnancy On The Risk Of Childhood Asthma." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a2504.

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Soares, Felipe A. L., Efrem E. O. Lousada, Tiago B. Silveira, Raquel A. F. Mini, Luis E. Zárate, and Henrique C. Freitas. "Analysis and Prediction of Childhood Pneumonia Deaths using Machine Learning Algorithms." In Symposium on Knowledge Discovery, Mining and Learning. Sociedade Brasileira de Computação - SBC, 2021. http://dx.doi.org/10.5753/kdmile.2021.17456.

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Acute Respiratory Tract Infections are among the leading causes of child mortality worldwide. Specifically, community-acquired pneumonia has different causes, such as: passive smoking, air pollution, poor hygiene, cardiac insufficiency, oropharyngeal colonization, nutritional deficiency, immunosuppression, and environmental, economic and social factors. Due to the variation of these causes, knowledge discovery in this area of health has been a great challenge for researchers. Thus, this paper presents the steps for the construction of a database and evaluation results applied to the analysis and prediction of potential deaths caused by childhood pneumonia using the Pictorea method. For this, the Random Forest and Artificial Neural Network algorithms were used, and after comparison, the Neural Network algorithm showed higher accuracy by up to 87.57%. This algorithm was used to analyze and predict the number of deaths from pneumonia in children up to 5 years old, and the results were presented using Root Mean Square Error and scatter plots. A domain specialist validated the results and defined that the pattern found is relevant for future studies in the medical field, helping to analyze the behavior of countries and predict future scenarios.
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Smedengaard, Nanna, Morten Dahl, and Eskild Landt. "Exposure to second-hand smoking during childhood and/or adulthood as a risk factor for respiratory disease." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.1884.

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Seror, R., G. Gusto, MC Boutron-Ruault, and X. Mariette. "OP0253 Passive smoking in childhood and history of chronic diarrhea increases the risk of developing rheumatoid arthritis (RA)." In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.3853.

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Metayer, Catherine S., Luoping Zhang, Joe L. Wiemels, Karen Bartley, Joshua Schiffman, Xiaomei Ma, Melinda Aldrich, Steve Selvin, Martyn Smith, and Patricia A. Buffler. "Abstract 662: Effect of tobacco smoking on the risks of childhood acute lymphocytic and myeloid leukemias, by phenotypic and cytogenetic subtype." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-662.

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Peppas, I., EJA Fitchett, W. Kenworthy, P. Kimkool, and CR Fertleman. "G185(P) Designing transformative osces: lessons from an adolescent smoking cessation station." In Royal College of Paediatrics and Child Health, Abstracts of the Annual Conference, 13–15 March 2018, SEC, Glasgow, Children First – Ethics, Morality and Advocacy in Childhood, The Journal of the Royal College of Paediatrics and Child Health. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2018. http://dx.doi.org/10.1136/archdischild-2018-rcpch.180.

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Reports on the topic "Childhood smoking"

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Gender-specific pathways mediate the risk of substance use in adolescents with ADHD. ACAMH, December 2019. http://dx.doi.org/10.13056/acamh.10679.

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Data suggest that children with attention-deficit/hyperactivity disorder (ADHD) are more likely to start smoking tobacco and/or marijuana earlier in childhood than unaffected children, and then escalate use during adolescence. Now, a study by researchers at the University of Minnesota has examined the mediating pathways underlying this association between childhood ADHD and later substance-abuse problems.
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