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Books on the topic 'Childhood deafness'

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Author: Grafiati

Published: 10 December 2022

Last updated: 28 January 2023

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1

Luterman, David. Deafness in the family. Boston: College-Hill Press, 1987.

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2

Davis, Lennard J. My sense of silence: Memoirs of a childhood with deafness. Urbana: University of Illinois Press, 2000.

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3

Central auditory processing disorder: Strategies for use with children and adolescents. Tucson, Ariz: Communication Skill Builders, 1995.

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4

Löwe, Armin. Hörerziehung für hörgeschädigte Kinder: Geschichte, Methoden, Möglichkeiten : eine Handreichung für Eltern, Pädagogen und Therapeuten. Heidelberg: HVA, Edition Schindele, 1991.

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5

Psychological development of deaf children. New York: Oxford University Press, 1993.

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6

Kratz, Laura E. Movement and fundamental motor skills for sensory deprived children. Springfield, Ill., U.S.A: Thomas, 1987.

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7

Evelyn, Cherow, Matkin Noel D, Trybus Raymond J, American Speech-Language-Hearing Association, and United States. Administration on Developmental Disabilities., eds. Hearing-impaired children and youth with developmental disabilities: An interdisciplinary foundation for service. Washington, D.C: Gallaudet College Press, 1985.

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8

1936-, Powers Stephen, and Gregory Susan 1945-, eds. Deaf and hearing impaired pupils in mainstream schools. London: David Fulton, 1999.

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9

Boothroyd, Arthur. Hearing impairments in young children. Washington, D.C: Alexander Graham Bell Association for the Deaf, 1988.

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10

(Illustrator), Simon Smith, ed. Understanding Childhood Deafness. Thorsons Publishers, 1996.

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11

(Editor), Ellen Kurtzer-White, and David Luterman (Editor), eds. Early Childhood Deafness. Not Avail, 2001.

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12

(Editor), Ellen Kurtzer-White, and David Luterman (Editor), eds. Early Childhood Deafness. York Press, 2001.

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13

Ellen, Kurtzer-White, and Luterman David, eds. Early childhood deafness. Austin, Tex: Pro-Ed., 2006.

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14

Turner, Gary. Understand Deafness During Childhood. Lulu Press, Inc., 2018.

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15

Society, National Deaf Children's, ed. Childhood deafness: A guide for students. [London]: National Deaf Children's Society, 1999.

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16

Luterman, David. Deafness in the Family. Taylor & Francis Group, 1987.

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17

My Sense Of Silence Memoirs Of A Childhood With Deafness. University of Illinois Press, 2008.

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18

Davis, Lennard J. My Sense of Silence: Memoirs of a Childhood with Deafness. University of Illinois Press, 2015.

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19

Meadow, Kathryn P. Deafness and Child Development. University of California Press, 2022.

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20

Meadow, Kathryn P. Deafness and Child Development. University of California Press, 2022.

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21

Liben, Lynn S. Deaf Children: Developmental Perspectives. Elsevier Science & Technology Books, 2013.

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22

Jonsson, David. You are deaf, congratulations!: My childhood journey and understanding audism. 2015.

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23

Davis, Lennard J. My Sense of Silence: Memoirs of a Childhood with Deafness (Creative Nonfiction Series). University of Illinois Press, 1999.

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24

Kelly, Dorothy A. Central Auditory Processing Disorder: Strategies for Use With Children and Adolescents. Psychological Corp, 1999.

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25

Elmer, Owens, and Kessler Dorcas K. 1942-, eds. Cochlear implants in young deaf children. Boston: College-Hill Press, 1989.

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26

Educational Resources Information Center (U.S.), ed. Antecedents of language competence and social-emotional adjustment of young deaf children. Atlanta, Ga: Dept. of Educational Foundations, Georgia State University, 1990.

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27

Marschark, Marc. Psychological Development of Deaf Children. Oxford University Press, USA, 1997.

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28

Marschark, Marc. Psychological Development of Deaf Children. 2nd ed. Oxford Univ Pr (Txt), 2007.

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29

Lightstone, A. H., and Abraham Robinson. Nonarchimedean Fields and Asymptotic Expansions. Elsevier Science & Technology Books, 2016.

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30

Cohen, Jeffrey A., Justin J. Mowchun, Victoria H. Lawson, and Nathaniel M. Robbins. A 20-year-Old Male with Acute Lower-Extremity Weakness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190491901.003.0025.

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A metabolic myopathy may be an overlooked etiology of recurrent exercised-induced weakness. This chapter discusses the clinical features and diagnostic considerations for adult patients with suspected metabolic myopathy. Adult patients with carbohydrate or fatty acid disorders typically have a history since childhood of exercise-induced myalgias, cramps, and fatigue. Episodes of myoglobinuria tend to present later, usually in the second decade. For any unexplained myopathy, especially if there is concern for a metabolic myopathy, there should also be consideration of a mitochondrial disease. Many mitochondrial disorders present with multiple neurological problems, such as deafness, seizures, encephalopathy, ptosis, or ophthalmoplegia. This chapter will discuss the complexities of genetic testing and muscle biopsy for the evaluation of a possible metabolic myopathy.

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31

Hall, Andrew, and Shamima Rahman. Mitochondrial diseases and the kidney. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0340.

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Mitochondrial disease can affect any organ in the body including the kidney. As increasing numbers of patients with mitochondrial disease are either surviving beyond childhood or being diagnosed in adulthood, it is important for all nephrologists to have some understanding of the common renal complications that can occur in these individuals. Mitochondrial proteins are encoded by either mitochondrial or nuclear DNA (mtDNA and nDNA, respectively); therefore, disease causing mutations may be inherited maternally (mtDNA) or autosomally (nDNA), or can arise spontaneously. The commonest renal phenotype in mitochondrial disease is proximal tubulopathy (Fanconi syndrome in the severest cases); however, as all regions of the nephron can be affected, from the glomerulus to the collecting duct, patients may also present with proteinuria, decreased glomerular filtration rate, nephrotic syndrome, water and electrolyte disorders, and renal tubular acidosis. Understanding of the relationship between underlying genotype and clinical phenotype remains incomplete in mitochondrial disease. Proximal tubulopathy typically occurs in children with severe multisystem disease due to mtDNA deletion or mutations in nDNA affecting mitochondrial function. In contrast, glomerular disease (focal segmental glomerulosclerosis) has been reported more commonly in adults, mainly in association with the m.3243A<G point mutation. Co-enzyme Q10 (CoQ10) deficiency has been particularly associated with podocyte dysfunction and nephrotic syndrome in children. Underlying mitochondrial disease should be considered as a potential cause of unexplained renal dysfunction; clinical clues include lack of response to conventional therapy, abnormal mitochondrial morphology on kidney biopsy, involvement of other organs (e.g. diabetes, cardiomyopathy, and deafness) and a maternal family history, although none of these features are specific. The diagnostic approach involves acquiring tissue (typically skeletal muscle) for histological analysis, mtDNA screening and oxidative phosphorylation (OXPHOS) complex function tests. A number of nDNA mutations causing mitochondrial disease have now been identified and can also be screened for if clinically indicated. Management of mitochondrial disease requires a multidisciplinary approach, and treatment is largely supportive as there are currently very few evidence-based interventions. Electrolyte deficiencies should be corrected in patients with urinary wasting due to tubulopathy, and CoQ10 supplementation may be of benefit in individuals with CoQ10 deficiency. Nephrotic syndrome in mitochondrial disease is not typically responsive to steroid therapy. Transplantation has been performed in patients with end-stage kidney disease; however, immunosuppressive agents such as steroids and tacrolimus should be used with care given the high incidence of diabetes in mitochondrial disease.

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32

El Deafo. New York: Amulet Books, an imprint of Abrams, 2014.

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33

El Deafo. Harry N. Abrams, 2014.

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