Relevant bibliographies by topics / Childhood cancers

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Childhood cancers'

Author: Grafiati
Published: 18 May 2024

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Journal articles on the topic "Childhood cancers"

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Smith, Helin, and Bob Phillips. "Childhood Cancer." InnovAiT: Education and inspiration for general practice 5, no. 10 (September 14, 2012): 595–603. http://dx.doi.org/10.1093/innovait/ins133.

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Childhood cancers are uncommon, accounting for only 0.5% of all cancers in the UK. Approximately, 1500 children are diagnosed with cancer in the UK every year. Despite it being a rare occurrence, cancer still remains the largest cause of death in the 1–14 year age group, amongst whom it counts for 20% of all deaths. Although most adult cancers affect the lung, breast, bowel and prostate, the majority of childhood cancers are haematological and central nervous system (CNS) tumours. The primary care physician's role is vital across the disease trajectory, requiring them to recognize the signs and symptoms of childhood cancer, understand treatment, provide support to children and families, and finally consider the issues affecting survivors of childhood cancer.
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Manirakiza, A. "The Role of Childhood Cancer Civil Society on Cancer Control on Low-Income Countries." Journal of Global Oncology 4, Supplement 2 (October 1, 2018): 136s. http://dx.doi.org/10.1200/jgo.18.71600.

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Background and context: Over 250,000 new pediatric cancer cases are diagnosed yearly worldwide. In the developing countries, the childhood cancer burden is estimated to increase even more. Rwanda Children's Cancer Relief (RCCR) is a nonprofit organization with a mission to ensure that children with cancers access high standards of treatment and support. After realizing that majority in our community lack information on childhood cancers, our efforts since 2014 has been concentrated on raising awareness of childhood cancers. Aim: Raise awareness on childhood cancer among community members. Strategy/Tactics: During a massive walk, trained volunteers with informative brochures, posters in both local and international languages comprising information on early symptoms of childhood cancers, ways to navigate referral system among others are used during community outreaches. These activities are aired on national radios and TVs, newspapers and social media are also used to spread information. Program/Policy process: Many activities are carried out in September during the RCCR Annual Childhood Cancers Awareness Month. We involve community health workers, private sectors and academic institutions. We focus on talks, community outreaches and childhood cancers awareness activities. Outcomes: 2017 campaign included the aforementioned activities and was concluded by a walk dubbed (#KidsCancerWalk2016) which attracted more than 600 participants. Two local TVs, 6 radios and 7 online diaries covered the campaign in its different phases. More than 1000 posters, 1100 brochures and 500 flyers were distributed followed by intensive online campaign with more than 100 Facebook posts, 350 Tweets, and 700 Retweets. Strong partnerships with private, public sectors were created and Ministry of Health approved September as the national childhood cancer awareness month following RCCR campaigns. What was learned: Childhood cancers are fatal when left untreated but treatable when they are detected at an early stage. Activities that RCCR carry out on annual basis increase the general population knowledge about childhood cancers. Civil societies like RCCR play a crucial role in addressing the growing burden of childhood cancers and the disparities in access to and quality of care. They can also influence policy changes and address the specific patients' and community needs.
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Brooks, G. A., J. E. Stopfer, J. Erlichman, R. Davidson, K. L. Nathanson, and S. M. Domchek. "Childhood cancer in families with and without germline BRCA1/2 mutations ascertained at a high-risk breast cancer clinic." Journal of Clinical Oncology 24, no. 18_suppl (June 20, 2006): 10013. http://dx.doi.org/10.1200/jco.2006.24.18_suppl.10013.

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10013 Background: Germline mutations in the BRCA1 and BRCA2 genes (BRCA1/2) are associated with breast cancer, ovarian cancer and other malignancies. Biallelic mutations of BRCA2 are a cause of Fanconi anemia and characteristic childhood cancers. We undertook this study to evaluate the contribution of familial heterozygous BRCA1/2 mutations to childhood cancer in hereditary breast cancer families. Methods: We conducted a retrospective cohort study to compare the prevalence of childhood cancers among 379 families with BRCA1/2 mutations and 426 families without known mutations. All families were ascertained at a high-risk breast cancer clinic. Our study and comparison cohorts included first- through fourth-degree relatives of cancer-affected mutation carriers and cancer-affected individuals with negative full sequencing for BRCA1/2 mutations. The primary endpoint was any case of childhood cancer (diagnosed <age 21) as recorded in the family history. Results were analyzed using Fisher’s exact test. Results: 20 cases of childhood cancer were observed in 379 families with BRCA1/2 mutations and 35 cases of childhood cancer were observed in 426 families with negative mutation testing (p = 0.12). Apart from two very early-onset breast cancers, the types of childhood cancers occurring in the BRCA1/2 mutation positive families were similar to those occurring in the mutation negative families and in the general population. Nine childhood cancers occurred in 240 families with BRCA1 mutations, and 11 childhood cancers occurred in 141 families with BRCA2 mutations (p=0.1). 13 of 18 BRCA1/2 mutation positive families with cases of childhood cancer (72%) and 13 of 31 families with childhood cancer cases and negative mutation testing (42%) met the Birch clinical criteria for Li-Fraumeni like syndrome (LFL); no individual has undergone testing for germline TP53 mutations. Conclusions: Heterozygous BRCA1/2 mutations do not appear to be a significant risk factor for childhood cancer in hereditary breast cancer families. This observation supports the current practice of delaying BRCA1/2 mutation testing until candidates reach adulthood. Although many families in both groups met criteria for LFL, the clinical significance of this finding is uncertain. No significant financial relationships to disclose.
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Cole, Catherine. "Registering childhood cancers." Lancet 364, no. 9451 (December 2004): 2074–76. http://dx.doi.org/10.1016/s0140-6736(04)17561-2.

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Herzog, Cynthia E., Richard J. Andrassy, and Farzin Eftekhari. "Childhood Cancers: Hepatoblastoma." Oncologist 5, no. 6 (December 2000): 445–53. http://dx.doi.org/10.1634/theoncologist.5-6-445.

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Houghton, Peter J. "Developing novel treatments for childhood solid cancers." Open Access Government 39, no. 1 (July 10, 2023): 112–13. http://dx.doi.org/10.56367/oag-039-10780.

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Developing novel treatments for childhood solid cancers With a particular focus on glioma, Dr Peter J Houghton from Greehey Children’s Cancer Research Institute outlines the barriers that have hindered the development of effective therapies for childhood solid cancers. Childhood cancer represents less than 1% of human cancers, thus, is not a priority for drug development in the pharmaceutical industry. However, the consequences of having a child diagnosed with a malignant disease can be devastating for families. Developing new therapies for childhood solid cancers presents certain constraints that are seldom encountered with neoplastic diseases diagnosed in adults. Childhood tumours are rare, which has therefore restricted large-scale drug evaluations or randomised clinical trials. For example, of the new Phase I agents evaluated in adult malignancies, less than 30% receive an adequate assessment in children.
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Robison, L. L. "Second primary cancers after childhood cancer." BMJ 312, no. 7035 (April 6, 1996): 861–62. http://dx.doi.org/10.1136/bmj.312.7035.861.

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Houghton, Peter J. "Developing novel therapies for childhood cancers." Open Access Government 40, no. 1 (October 25, 2023): 154–55. http://dx.doi.org/10.56367/oag-040-10780.

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Developing novel therapies for childhood cancers Dr Peter J Houghton from Greehey Children’s Cancer Research Institute discusses the obstacles in developing new treatments for childhood cancers and new approaches in preclinical testing. In my previous article for Open Access Government, I focused on the challenges of developing novel therapies for children with glioma, the most prevalent brain cancer in children. Here, I want to discuss the challenges at the preclinical stage of drug development, an area of research that will be altered significantly by the Research to Accelerate Cures and Equity for Children Act (RACE for Children Act). This U.S. law requires the Food and Drug Agency (FDA) to develop a list of molecular targets and molecular targets of new drugs and biologics in development that are determined to be relevant to pediatric cancers. The objective is to facilitate a more rapid introduction of new drugs into the pediatric cancer armamentarium. The FDA may now require pediatric assessments when molecular targets under FDA review are considered relevant to childhood cancer.
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MacDonald, Tamara. "Pediatric Cancer: A Comprehensive Review. Part I: Biology, Epidemiology, Common Tumours, Principles of Treatment and Late Effects." Canadian Pharmacists Journal / Revue des Pharmaciens du Canada 143, no. 4 (July 2010): 176–83. http://dx.doi.org/10.3821/1913-701x-143.4.176.

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The incidence rates of pediatric cancer, like adult cancer, are increasing, though to a lesser degree. Options for the treatment of childhood cancers are continually changing and improving and overall survival has increased dramatically over the last 60 years. This paper discusses the incidence and survival trends of childhood cancer. The biology and epidemiology of the most common cancers seen in children and the late effects of treatment for childhood cancer will also be discussed. A basic understanding of childhood cancer is important for both hospital and community pharmacists, since many young adults in North America are now survivors of childhood cancer and may experience long-term consequences of chemotherapy.
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Smith, Malcolm A., Nita L. Seibel, Sean F. Altekruse, Lynn A. G. Ries, Danielle L. Melbert, Maura O'Leary, Franklin O. Smith, and Gregory H. Reaman. "Outcomes for Children and Adolescents With Cancer: Challenges for the Twenty-First Century." Journal of Clinical Oncology 28, no. 15 (May 20, 2010): 2625–34. http://dx.doi.org/10.1200/jco.2009.27.0421.

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Purpose This report provides an overview of current childhood cancer statistics to facilitate analysis of the impact of past research discoveries on outcome and provide essential information for prioritizing future research directions. Methods Incidence and survival data for childhood cancers came from the Surveillance, Epidemiology, and End Results 9 (SEER 9) registries, and mortality data were based on deaths in the United States that were reported by states to the Centers for Disease Control and Prevention by underlying cause. Results Childhood cancer incidence rates increased significantly from 1975 through 2006, with increasing rates for acute lymphoblastic leukemia being most notable. Childhood cancer mortality rates declined by more than 50% between 1975 and 2006. For leukemias and lymphomas, significantly decreasing mortality rates were observed throughout the 32-year period, though the rate of decline slowed somewhat after 1998. For remaining childhood cancers, significantly decreasing mortality rates were observed from 1975 to 1996, with stable rates from 1996 through 2006. Increased survival rates were observed for all categories of childhood cancers studied, with the extent and temporal pace of the increases varying by diagnosis. Conclusion When 1975 age-specific death rates for children are used as a baseline, approximately 38,000 childhood malignant cancer deaths were averted in the United States from 1975 through 2006 as a result of more effective treatments identified and applied during this period. Continued success in reducing childhood cancer mortality will require new treatment paradigms building on an increased understanding of the molecular processes that promote growth and survival of specific childhood cancers.
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Dissertations / Theses on the topic "Childhood cancers"

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Watkins, Sharon Minnich. "Increased Risk of Childhood Cancers Among Children With Congenital Anomalies: A Florida Birth Cohort Experience /." The Ohio State University, 1996. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487933245536724.

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Kroll, Mary Eileen. "Time trends in childhood cancer : Britain 1966-2005." Thesis, University of Oxford, 2009. http://ora.ox.ac.uk/objects/uuid:8be887be-36e7-4b77-a7af-5887f3a1df8c.

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Increasing time trends in the recorded incidence of childhood cancer have been reported in many different settings. The extent to which these trends reflect real changes in incidence, rather than improvements in methods for diagnosis and registration, is controversial. Using data from the National Registry of Childhood Tumours (NRCT), this thesis investigates time trends in cancer diagnosed under age 15 in residents of Britain during 1966-2005 (54650 cases), and considers potential sources of artefact in detail. Several different methods are used to estimate completeness of NRCT registration. The history of methods for diagnosis and registration of childhood cancers in Britain is described, and predictions are made for effects on recorded incidence. For each of the 12 main diagnostic groups, Poisson regression is used to fit continuous time trends and ‘step’ models to the annual age-sex-standardised rates by year of birth and year of diagnosis. Age-specific rates by period, and quinquennial standardised rates for diagnostic subgroups, are shown graphically. For three broad groups (leukaemia, CNS tumours and other cancer), geographical variation is compared by period of diagnosis. The results of these analyses are discussed in relation to the predicted artefacts. The evidence for a positive association between affluence and recorded incidence of childhood leukaemia is briefly reviewed. A special form of diagnostic artefact, the ‘fatal infection’ hypothesis, is proposed as an explanation of both this association and the leukaemia time trend. This hypothesis is examined in a novel test based on clinical data. The recorded incidence of childhood cancer in Britain increased in each of 12 diagnostic groups during 1966-2005 (from 0.5% per year for bone cancer to 2.5% for hepatic cancer, with 0.7% for leukaemia). Evidence presented here suggests that these increases are probably artefacts of diagnosis and registration. The potential implications for epidemiological studies of childhood cancer should be considered.
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Grèze, Victoria. "Cancers dans l'enfance et fertilité à l'âge adulte." Thesis, Université Clermont Auvergne‎ (2017-2020), 2019. http://www.theses.fr/2019CLFAS011.

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Les progrès dans le traitement des cancers de l’enfant et de l’adolescent ont permis de réelles améliorations puisque la survie à long terme est maintenant de plus de 80%. De ce fait, la qualité de vie de ces futurs adultes est une préoccupation majeure des professionnels impliqués dans la prise en charge de ces patients. Notre travail se focalise en particulier sur le développement pubertaire et la fertilité, au travers de la recherche épidémiologique et de la recherche de transfert, chez les enfants et adolescents guéris d’un cancer.Sur le versant « recherche épidémiologique », le développement pubertaire et la fertilité chez la fille ont été étudiés à partir de la cohorte L.E.A. (Leucémies de l’Enfant et Adolescent), qui recueille des données concernant le devenir des patients traités pour une leucémie au cours de leur enfance. Bien qu’elle soit plus importante chez les femmes ayant bénéficié d’une greffe de cellules souches hématopoïétiques et/ou ayant rechuté, la gonadotoxicité atteint également celles n’ayant reçu que de la chimiothérapie en première ligne.En parallèle, l’accès à la préservation de la fertilité des adolescents et jeunes adultes pris en charge pour des pathologies malignes en Auvergne a été analysé. Les garçons bénéficient davantage de consultations au CECOS et d’une préservation de leur fertilité. Des progrès devraient se faire grâce à la création de plateformes clinico-biologiques telles que PREFERA (PREservation FERtilité Auvergne) qui assurent une meilleure coordination entre les différentes équipes prenant en charge ces patients. Sur le versant « recherche de transfert », nous nous sommes intéressés à la cryoconservation des tissus gonadiques, seule option envisageable chez les enfants prépubères notamment, mais comportant un risque potentiel de réintroduction de cellules néoplasiques en cas d’utilisation ultérieure. Nous avons mis au point des techniques sensibles et spécifiques de détection de la maladie résiduelle pour deux tumeurs solides pédiatriques dont les traitements actuels sont potentiellement stérilisants : le neuroblastome et la tumeur d’Ewing. L’intérêt étant de disposer d’un test diagnostic utilisable pour les adultes guéris de ces cancers, dont la fertilité a été compromise par les traitements et qui ont bénéficié d’une cryoconservation de tissu ovarien ou testiculaire
Advances in the treatment of childhood and adolescent cancers have led to real improvements as long-term survival is now over 80%. As a result, quality of life of these future adults is a major concern for the professionals involved in the care of these patients. Our work focuses on pubertal development and fertility, through epidemiological research and transfer research, in childhood cancer survivor.Concerning the "epidemiological research", female pubertal development and fertility were studied using the L.E.A cohort (Childhood and Adolescent Leukemias), which collects data about the outcome of patients treated for childhood leukemia. Although it is more important in women who received hematopoietic stem cell transplantation and/or relapsed, gonadotoxicity also affects those who received only first-line chemotherapy.In parallel, access to fertility preservation for adolescents and young adults treated for malignant diseases in Auvergne was analyzed. Boys benefit more from fertility consultation and preservation of their fertility. Progress should be made thanks the creation of clinico-biological platforms such as PREFERA (PREservation FERtilité Auvergne), which ensure better coordination between the different teams that support these patients.Concerning the "transfer research", we addressed the cryopreservation of gonadal tissues, only option for prepubertal children in particular, but with a potential risk of neoplastic cells reintroduction in case of future use. We developed sensitive and specific techniques of residual disease detection for two solid pediatric tumors whose current treatments are potentially sterilizing: neuroblastoma and Ewing's tumor. The interest is to have a diagnostic test usable for adults cured of these cancers, whose fertility has been compromised by the treatments and who have benefited from ovarian or testicular tissue cryopreservation
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Vangile, Kirsten M. "Childhood cancer survivorship patient characteristics /." restricted, 2008. http://etd.gsu.edu/theses/available/etd-12042008-133347/.

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Thesis (M.P.H.)--Georgia State University, 2008.
Title from file title page. Russ Toal, committee chair; Karen Wasilewski-Masker, committee member. Description based on contents viewed July 7, 2009. Includes bibliographical references (p. 68-72).
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Vangile, Kirsten M. "Childhood Cancer Survivors: Patient Characteristics." Digital Archive @ GSU, 2008. http://digitalarchive.gsu.edu/iph_theses/51.

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Survivors of childhood cancer are a relatively new phenomenon in the medical world. The introduction of treatment protocols in the 1970s started a trend in curing children of cancer that historically had been a death sentence. Under these treatment protocols children were given different treatment regimens based on past research that helped remove cancerous cells from their bodies, but were later found to be the cause of treatment related morbidities years into the future; for most survivors roughly ten to 20 years post treatment. These morbidities, commonly called late-effects, are the prime reason that survivors of childhood cancer need to participate in survivorship care. Survivors of childhood cancer are particularly vulnerable to late-effects because the majority of them receive their treatment at a time when their bodies are still growing and developing. Survivorship care services vary by site, but all maintain the common goals of providing long-term follow up for the survivor and education about the ways in which treatments may affect a survivors’ health as they age. Similar to many other facets of healthcare and medicine, there are many populations who do not participate in survivorship care. The purpose of this research is to identify possible barriers to care, assess the level of impact these barriers have upon the survivor’s potential for participation and provide suggestions as to how these barriers can be mitigated. Additionally, this research highlights areas that need further research and analysis.
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Janson, Christopher M. "Marriage and Divorce in Survivors of Childhood Cancer: A Report from the Childhood Cancer Survivor Study." Yale University, 2008. http://ymtdl.med.yale.edu/theses/available/etd-08092007-145913/.

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In this report from the Childhood Cancer Survivor Study (CCSS), we described marriage and divorce rates in survivors of childhood cancer, as compared to a sibling control group and the general U.S. population. We also sought to identify patient and treatment characteristics that were associated with survivor marital status. This study included 8,930 five-year survivors of childhood malignancy and 2,855 sibling controls participating in the CCSS. Data on marital status, sociodemographic factors, and current health status were obtained from questionnaires; detailed disease and treatment histories were available from medical records. Marital status of the U.S. population was obtained from the 2002 Current Population Survey of the U.S. Census. We found that survivors were more likely to have never married than both sibling (odds ratio [OR] = 1.79; 95 % CI = 1.65-1.94; p < 0.0001) and population controls (OR = 2.29; 95 % CI = 2.19-2.38; p < 0.0001), with persistence of trends across age and gender strata. Once married, survivors divorced at rates equivalent to controls. In adjusted analysis, we found that several survivor characteristics predicted never-married status, including treatment involving cranial radiation (OR = 2.41; p < 0.0001), CNS tumor diagnosis (OR = 2.05; p < 0.0001), history of growth hormone deficiency (OR = 2.02; p < 0.0001), and unemployment secondary to disability (OR = 1.78; p = 0.0001). Survivor characteristics predictive of divorce included unemployment (OR = 1.91; p < 0.0001, for unemployed or disabled), lower educational achievement (OR = 1.74; p < 0.0001, for non-college graduates), and psychological distress (OR = 1.60; p < 0.0001). This study confirms prior reports of lower marriage rates in survivors of childhood cancer, providing further evidence that this population struggles with psychosocial adjustment to adult life.
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Mutalima, Nora. "Infections and childhood cancer in Malawi." Thesis, University of Oxford, 2007. http://ora.ox.ac.uk/objects/uuid:9d0c9045-34eb-426c-acec-9c1ffa269417.

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The causes of childhood cancers are not well understood. That infections are believed to play an important role in childhood cancer development is of particular interest in sub-Saharan Africa, where infectious diseases are common. The objectives of this thesis were to identify childhood cancers associated with HIV, malaria, EBV and HHV-8, and to investigate child and maternal factors associated with Burkitt lymphoma and Kaposi sarcoma. In Blantyre, Malawi, 305 children diagnosed with cancer and 212 of their mothers, were recruited. Risk factor data were collected using a brief questionnaire and blood samples tested for infections. Case-control analyses were conducted to compare 148 Burkitt lymphoma cases and 22 Kaposi sarcoma case with a control group comprising 104 children with cancers other than those known to be associated with HIV. The prevalence of HIV was 6% among children with Burkitt lymphoma and 2% in controls (OR=12.4, 95% CI 1.3 to 116.2). Tumour risk increased with increasing litres of antibodies against EBV and malaria. In comparison with those who had low titres against both EBV and malaria, the highest risk of Burkitt lymphoma was among those with high titres against both infections (OR=13.2, 95% CI 3.8 to 46.6). Reported use of mosquito nets was protective against Burkitt lymphoma. The prevalence of HIV was 81% among children with Kaposi sarcoma (OR=762.7, 95% CI 44 to 13376), and risk increased with increasing HHV-8 antibodies. Prevalence of infections was also examined among children with other cancer types and no associations were identified, although the number of cases was small. Few maternal factors were found to be associated with cancer in children. This research demonstrates that infections play a particularly important role in increasing the risk of Burkitt lymphoma and Kaposi sarcoma in children in sub- Saharan Africa. Prevention or early treatment of these infections may be vital in the control of childhood cancer.
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Sylvester, Dianne. "Genes underpinning predisposition to childhood cancer." Thesis, The University of Sydney, 2020. https://hdl.handle.net/2123/22458.

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The genetic changes underpinning cancer predisposition in children are not clearly defined and warrant further research, as identification of a genetic contribution to a patient’s disease can be beneficial for patients and their families. With technological advances, it is now possible to discover a broad spectrum of genetic changes implicated in cancer predisposition by sequencing the genome. An analytical review of six publications utilising germline genome sequencing in paediatric oncology found that these studies differed in the selected childhood cancer diagnoses and the genes considered for interpretation, resulting in differences in the proportions of childhood cancer patients that were reported to carry clinically relevant pathogenic germline variants. In this study, childhood cancer patients that presented with phenotypes indicative of a genetic susceptibility to cancer, such as multiple cancer diagnoses, a family history of cancer and/or a genetic diagnosis, underwent germline exome sequencing. Sequencing data were analysed for rare germline variants in over 1000 cancer predisposition, cancer associated and DNA repair genes, that were predicted to cause a loss of function or to be deleterious. Almost one quarter of childhood cancer patients with features suggestive of a genetic predisposition to cancer were found to carry pathogenic or likely pathogenic germline variant/s in 12 known cancer predisposition genes. A rare variant burden analysis of 31 autosomal dominant cancer predisposition genes found that deleterious germline variants were significantly enriched in a cohort of 63 childhood cancer patients compared to a cohort of 1107 genetically matched healthy aged controls. Novel germline variants not previously associated with cancer predisposition were also detected in 10 genes in 16 childhood cancer patients. This study has expanded our understanding of cancer predisposition in children, by discovering the diagnostic potential of sequencing patients with defined phenotypic features, and by linking pathogenic or likely pathogenic germline variants in known predisposition genes with new cancer diagnoses. Ultimately, by combining the analysis of family pedigrees with functional gene studies and data-sharing, the significance of novel germline variants associated with the onset of cancer in childhood will be established. As more childhood cancer predisposition genes are identified and characterised, screening processes may be more routinely incorporated into paediatric clinical care.
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Luk, Yin-ching, and 陸燕青. "Evidence-based psychosocial intervention for families with childhood cancer patients." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B44625698.

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Prouty, Diana Frances Ward-Smith Peggy. "The lived experience of adult survivors of childhood cancer." Diss., UMK access, 2005.

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Thesis (Ph. D.)--School of Nursing. University of Missouri--Kansas City, 2005.
"A dissertation in nursing." Advisor: Peggy Ward-Smith. Typescript. Vita. Title from "catalog record" of the print edition Description based on contents viewed June 26, 2006. Includes bibliographical references (leaves 142-147). Online version of the print edition.
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Books on the topic "Childhood cancers"

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Janes-Hodder, Honna. Childhood cancer: A parent's guide to solid tumor cancers. 2nd ed. Beijing: O'Reilly, 2002.

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1930-, Sluyser M., and Voûte P. A. 1906-, eds. Molecular biology and genetics of childhood cancers: Approaches to neuroblastoma. Chichester [England]: E. Horwood, 1988.

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Gilman, E. A. The spatial and temporal distribution of childhood cancers in Britain. Birmingham: University of Birmingham, 1992.

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Gilman, E. A. Oxford survey of childhood cancers: A description of the first nationwide case/control study of childhoodcancers in Britain. Orkney: International Associates for Community Health, 1992.

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Ronald D. [et al.] Barr. Childhood cancer. 2nd ed. Hamilton, Ont: B.C. Decker, 2001.

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Steen, R. G. Childhood Cancer. Cambridge, MA: Harper Collins Trade, 2000.

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National Cancer Institute (U.S.), ed. Meeting of the President's Cancer Panel: The molecular characterization of childhood cancers and its application to innovative approaches to cancer therapy : 9:00 a.m., Friday, April 11, 1986, the Auditorium, St. Jude Children's Research Hospital, Memphis Tennessee. [Bethesda, Md?]: The Institute, 1986.

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Godden, John O., ed. Cancer in Childhood. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4684-2070-8.

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International Agency for Research on Cancer., ed. Epidemiology of childhood cancer. Lyon, France: International Agency for Research on Cancer, 1999.

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Woznick, Leigh A. Living with childhood cancer. Washington, DC: American Psychological Association, 2002.

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Book chapters on the topic "Childhood cancers"

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Sherman, C. D., K. C. Calman, S. Eckhardt, I. Elsebai, D. Firat, D. K. Hossfeld, J. P. Paunier, and B. Salvadori. "Childhood Cancers." In Manual of Clinical Oncology, 305–18. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-96995-9_31.

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Ward, Evelyn. "Childhood Cancers." In Clinical Paediatric Dietetics, 654–67. Chichester, UK: John Wiley & Sons, Ltd, 2014. http://dx.doi.org/10.1002/9781118915349.ch21.

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Harif, Mhamed, and Daniela Cristina Stefan. "Rare Childhood Cancers." In Pediatric Cancer in Africa, 229–36. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-17936-0_19.

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Divarci, Emre, and Ahmet Çelik. "Childhood Thyroid Cancers." In Endocrine Surgery, 155–62. Boca Raton: CRC Press, 2021. http://dx.doi.org/10.1201/9780429197338-20.

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Stehbens, James A., and Valerie A. Cool. "Neuropsychological Sequelae of Childhood Cancers." In Advances in Child Neuropsychology, 55–84. New York, NY: Springer New York, 1994. http://dx.doi.org/10.1007/978-1-4612-2608-6_2.

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Smith, Stephanie M., Lauren Jimenez-Kurlander, Lindsay Schwartz, Tara O. Henderson, and Danielle Novetsky Friedman. "Adult Survivors of Childhood Cancers." In Essentials of Cancer Survivorship, 6–23. Boca Raton: CRC Press, 2021. http://dx.doi.org/10.1201/9781003055426-3.

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Watson, Stuart B. "Melanoma and Other Skin Cancers." In The Surgery of Childhood Tumors, 395–411. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-48590-3_22.

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Pappo, Alberto S., and Gregory H. Reaman. "Introduction to Rare Cancers of Childhood." In Textbook of Uncommon Cancer, 847–49. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2012. http://dx.doi.org/10.1002/9781118464557.ch63.

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Wollina, Uwe. "Disabling Pansclerotic Morphea of Childhood." In Clinical Cases in Pediatric Skin Cancers, 99–102. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-93666-2_22.

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Gaze, Mark N., and John A. Kalapurakal. "Radiotherapy for Wilms’ Tumour and Other Childhood Renal Cancers." In Renal Tumors of Childhood, 207–27. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-662-44003-2_12.

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Conference papers on the topic "Childhood cancers"

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Zierhut, Heather A., Michael A. Murati, Tara Holm, Eric Hoggard, and Logan G. Spector. "Abstract 873: Association of rib anomalies and childhood cancers." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-873.

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Haskins, William E. "Abstract A58: Super SILAC proteomics for childhood blood cancers." In Abstracts: AACR International Conference on the Science of Cancer Health Disparities‐‐ Sep 30-Oct 3, 2010; Miami, FL. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1055-9965.disp-10-a58.

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Caron, Huib N., Ellen M. Westerhout, Jan J. Molenaar, Monique L. Boer den, Steve C. Clifford, Olivier Delattre, Birgit Geoerger, et al. "Abstract C116: The KidsCancerKinome: Validation of drug targets for high risk childhood cancers." In Abstracts: AACR-NCI-EORTC International Conference: Molecular Targets and Cancer Therapeutics--Nov 15-19, 2009; Boston, MA. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/1535-7163.targ-09-c116.

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Milosevic, Jelena, Nina Eissler, Diana Treis, Malin Wicktröm, Susanne Fransson, Baldur Sveinbjornsson, Ninib Baryawno, et al. "Abstract 1945: PPM1D/Wip1, promising new target in childhood cancers neuroblastoma and medulloblastoma." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-1945.

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Kalra, Anjali, David Schneider, Bhaskar Thakur, and Abigail Grant. "Adverse Childhood Experiences (ACEs) and the Development of Human Papilloma Virus (HPV) Related Cancers." In NAPCRG 51st Annual Meeting — Abstracts of Completed Research 2023. American Academy of Family Physicians, 2023. http://dx.doi.org/10.1370/afm.22.s1.4993.

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Magnusson, Susanne, Thomas Wiebe, Ulf Kristoffersson, Helena Jernström, and Håkan Olsson. "Abstract 5512: Increased incidence of adult cancers in relatives of childhood cancer patients and difference in tumor spectra between families with single and multiple cases of childhood tumors." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-5512.

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Adeloye, Davies, Asa Auta, Jhonathan P. Dos Santos, and Michael O. Harhay. "Abstract 1196: Epidemiology of childhood hematologic cancers in Africa: A systematic review of the evidence." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-1196.

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Langlois, Sylvie, Jasmine Healy, Mathieu Lajoie, Sophie Dumoucel, Fida Khater, Thomas Sontag, Pascal St-Onge, et al. "Abstract A41: TRICEPS: A feasibility study of personalized targeted therapy in relapsed/refractory childhood cancers." In Abstracts: AACR Special Conference: Advances in Pediatric Cancer Research: From Mechanisms and Models to Treatment and Survivorship; November 9-12, 2015; Fort Lauderdale, Florida. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.pedca15-a41.

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Chen, Kenneth S., Dinesh Rakheja, Yangjian Liu, Abhay A. Shukla, Joshua T. Mendell, and James F. Amatruda. "Abstract IA12: Mechanisms of tumorigenesis due to somatic mutations in DICER1 and DROSHA in childhood kidney cancers." In Abstracts: AACR Special Conference on Noncoding RNAs and Cancer: Mechanisms to Medicines; December 4-7, 2015; Boston, MA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.nonrna15-ia12.

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Tabori, Uri. "Abstract IA06: The role of hypermutation and replication repair deficiency in response of childhood cancers to immune checkpoint inhibitors." In Abstracts: AACR Special Conference: Pediatric Cancer Research: From Basic Science to the Clinic; December 3-6, 2017; Atlanta, Georgia. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.pedca17-ia06.

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Reports on the topic "Childhood cancers"

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Gregory Reaman. National Childhood Cancer Foundation. Office of Scientific and Technical Information (OSTI), November 2007. http://dx.doi.org/10.2172/920297.

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Snyder, Claire, Christina T. Yuan, Renee F. Wilson, Katherine Smith, Youngjee Choi, Paul C. Nathan, Allen Zhang, and Karen A. Robinson. Models of Care That Include Primary Care for Adult Survivors of Childhood Cancer: A Realist Review. Agency for Healthcare Research and Quality (AHRQ), February 2022. http://dx.doi.org/10.23970/ahrqepcrealistmodelsofcare.

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Objectives. We had two aims: (1) identify and analyze models of survivorship care for adult survivors of childhood cancer that include primary care, and (2) identify available tools, training, and other resources for adult survivors of childhood cancer. Methods. For each aim, we used realist synthesis to provide insights on how and for whom, in what contexts, and via what mechanisms the models of care and resources we identified can be effective for adult survivors of childhood cancer. We developed an initial program theory through searches of the literature and discussions with Stakeholders. We then identified and summarized quantitative evidence that supported or refuted the theory and developed specific hypotheses about how contexts and mechanisms may interact to produce outcomes (i.e., “CMO” hypotheses). The final program theory and CMO hypotheses were presented to Stakeholders for feedback. Results. Our final refined theory describes how, within the overall environment, survivor and provider characteristics and facilitators/barriers interact to produce intermediate and final outcomes. We focus on the role of models of care and resources (e.g., care plans) in these interactions. The program theory variables seen most consistently in the literature include oncology care versus primary care, survivor and provider knowledge (i.e., survivor risks and needs), provider comfort treating childhood cancer survivors, communication and coordination between and among providers and survivors, and delivery/receipt of prevention and surveillance of late effects of original cancer treatment. In turn, these variables played the most prominent role in the seven CMO hypotheses (4 focused on survivors and 3 focused on providers) regarding what works for whom and in what circumstances. Conclusions. To enable models of care that include primary care for adult survivors of childhood cancer, there needs to be communication of knowledge to both survivors and primary care providers. Our program theory provides guidance on the ways this knowledge could be shared, including the role of resources in doing so, and our CMO hypotheses suggest how the relationships illustrated in our theory could be associated with survivors living longer and feeling better through high-value care.
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Hendricks, Sam. Video: Better care for childhood cancer survivors. Monash University, February 2024. http://dx.doi.org/10.54377/696b-2665.

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Bancalari, Antonella, Samuel Berlinski, Giancarlo Buitrago, María Fernanda García, Dolores de la Mata, and Marcos Vera-Hernández. Health Inequalities in Latin American and the Caribbean: Child, Adolescent, Reproductive, Metabolic Syndrome and Mental Health. Inter-American Development Bank, October 2023. http://dx.doi.org/10.18235/0005208.

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Health constitutes a fundamental aspect of our well-being. It is also a key factor in determining our contribution to market and non-market output. Health inequality refers to the unequal realization of health outcomes between different groups in the population. Systematic disparities in health outcomes and in access to health resources not only undermine basic principles of fairness and social justice but also contributes towards perpetuating poverty and disadvantage. In this chapter, we start by presenting evidence on how the burden of disease in Latin America and the Caribbean (LAC) has changed during the last 30 years. Consistent with the fall in fertility and population aging, the region has shifted from a burden of disease dominated by maternal, neonatal, and communicable disease in the 1990s to one dominated by cardiovascular disease, cancers, diabetes, and increasingly by mental health disorders. The poorest in the region are burdened by worst access to maternal care and higher levels of infant mortality and stunting. Despite being knowledgeable about contraceptive methods, young women in Latin America and the Caribbean have very high levels of teenage pregnancy with a steep socio-economic gradient. Noncommunicable diseases also affect the poor disproportionately in many countries. Finally, mental health is a growing source of lost days of healthy living among women and the poor. Overall, our results highlight that despite the epidemiological transition which is underway, socio-economic health disparities in the LAC region are still more important on early childhood and teenagerhood than in adulthood, at least as it pertains to the outcomes analyzed in this chapter. At the same time, we show that while socio-economic inequalities in child health are smaller in the richest countries, the contrary happens with inequalities in some adult outcomes.
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Mobley, Erin M., Diana J. Moke, Joel Milam, Carol Y. Ochoa, Julia Stal, Nosa Osazuwa, Maria Bolshakova, et al. Disparities and Barriers to Pediatric Cancer Survivorship Care. Agency for Healthcare Research and Quality (AHRQ), March 2021. http://dx.doi.org/10.23970/ahrqepctb39.

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Objectives. Survival rates for pediatric cancer have dramatically increased since the 1970s, and the population of childhood cancer survivors (CCS) exceeds 500,000 in the United States. Cancer during childhood and related treatments lead to long-term health problems, many of which are poorly understood. These problems can be amplified by suboptimal survivorship care. This report provides an overview of the existing evidence and forthcoming research relevant to disparities and barriers for pediatric cancer survivorship care, outlines pending questions, and offers guidance for future research. Data sources. This Technical Brief reviews published peer-reviewed literature, grey literature, and Key Informant interviews to answer five Guiding Questions regarding disparities in the care of pediatric survivors, barriers to cancer survivorship care, proposed strategies, evaluated interventions, and future directions. Review methods. We searched research databases, research registries, and published reviews for ongoing and published studies in CCS to October 2020. We used the authors’ definition of CCS; where not specified, CCS included those diagnosed with any cancer prior to age 21. The grey literature search included relevant professional and nonprofit organizational websites and guideline clearinghouses. Key Informants provided content expertise regarding published and ongoing research, and recommended approaches to fill identified gaps. Results. In total, 110 studies met inclusion criteria. We identified 26 studies that assessed disparities in survivorship care for CCS. Key Informants discussed subgroups of CCS by race or ethnicity, sex, socioeconomic status, and insurance coverage that may experience disparities in survivorship care, and these were supported in the published literature. Key Informants indicated that major barriers to care are providers (e.g., insufficient knowledge), the health system (e.g., availability of services), and payers (e.g., network adequacy); we identified 47 studies that assessed a large range of barriers to survivorship care. Sixteen organizations have outlined strategies to address pediatric survivorship care. Our searches identified only 27 published studies that evaluated interventions to alleviate disparities and reduce barriers to care. These predominantly assessed approaches that targeted patients. We found only eight ongoing studies that evaluated strategies to address disparities and barriers. Conclusions. While research has addressed disparities and barriers to survivorship care for childhood cancer survivors, evidence-based interventions to address these disparities and barriers to care are sparse. Additional research is also needed to examine less frequently studied disparities and barriers and to evaluate ameliorative strategies in order to improve the survivorship care for CCS.
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Rapti, Christina, Petros C. Dinas, Costas Chryssanthopoulos, Alexandra Mila, and Anastassios Philippou. The effects of exercise and physical activity during and after childhood cancer: an umbrella review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, July 2022. http://dx.doi.org/10.37766/inplasy2022.7.0035.

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Camarillo, Ignacio, and Maxine Nichols. Breast Cancer and Early Onset Childhood Obesity: Cell Specific Gene Expression in Mammary Epithelia and Adipocytes. Fort Belvoir, VA: Defense Technical Information Center, July 2006. http://dx.doi.org/10.21236/ada483660.

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Kwan, Johnson, Adolf Baumgartner, Chun-Mei Lu, Mei Wang, Jingly F. Weier, Horst F. Zitzelsberger, and Heinz-Ulrich G. Weier. BAC-FISH assays delineate complex chromosomal rearrangements in a case of post-Chernobyl childhood thyroid cancer. Office of Scientific and Technical Information (OSTI), March 2009. http://dx.doi.org/10.2172/983040.

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Merrill, D. W., S. Selvin, E. R. Close, and H. H. Holmes. Use of density equalizing map projections (DEMP) in the analysis of childhood cancer in four California counties. Office of Scientific and Technical Information (OSTI), January 1995. http://dx.doi.org/10.2172/10117532.

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