Relevant bibliographies by topics / Cerebral Blood Flow Maps

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Cerebral Blood Flow Maps'

Author: Grafiati
Published: 6 September 2023

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Journal articles on the topic "Cerebral Blood Flow Maps"

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Hachaj, Tomasz, and Marek R. Ogiela. "Automatic Detection and Lesion Description in Cerebral Blood Flow and Cerebral Blood Volume Perfusion Maps." Journal of Signal Processing Systems 61, no. 3 (February 4, 2010): 317–28. http://dx.doi.org/10.1007/s11265-010-0454-0.

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Bergonzi, Karla M., Adam Q. Bauer, Patrick W. Wright, and Joseph P. Culver. "Mapping Functional Connectivity Using Cerebral Blood Flow in the Mouse Brain." Journal of Cerebral Blood Flow & Metabolism 35, no. 3 (March 2015): 367–70. http://dx.doi.org/10.1038/jcbfm.2014.211.

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Brain function can be assessed from resting-state functional connectivity (rs-fc) maps, most commonly created by analyzing the dynamics of cerebral hemoglobin concentration. Here, we develop the use of Laser Speckle Contrast Imaging (LSCI) for mapping rs-fc using cerebral blood flow (CBF) dynamics. Because LSCI is intrinsically noisy, we used spatial and temporal averaging to sufficiently raise the signal-to-noise ratio for observing robust functional networks. Although CBF-based rs-fc maps in healthy mice are qualitatively similar to simultaneously-acquired [HbO2]-based maps, some quantitative regional differences were observed. These combined flow/concentration maps might help clarify mechanisms involved in network disruption during disease.
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Johannsen, P., J. Jakobsen, and A. Gjedde. "Statistical maps of cerebral blood flow deficits in Alzheimer’s disease." European Journal of Neurology 7, no. 4 (July 2000): 385–92. http://dx.doi.org/10.1046/j.1468-1331.2000.00088.x.

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Scremin, Oscar U., Daniel P. Holschneider, Kevin Chen, Mingen G. Li, and Jean C. Shih. "Cerebral cortical blood flow maps are reorganized in MAOB-deficient mice." Brain Research 824, no. 1 (April 1999): 36–44. http://dx.doi.org/10.1016/s0006-8993(99)01167-1.

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Lacalle-Aurioles, María, José M. Mateos-Pérez, Juan A. Guzmán-De-Villoria, Javier Olazarán, Isabel Cruz-Orduña, Yasser Alemán-Gómez, María-Elena Martino, and Manuel Desco. "Cerebral Blood Flow is an Earlier Indicator of Perfusion Abnormalities than Cerebral Blood Volume in Alzheimer's Disease." Journal of Cerebral Blood Flow & Metabolism 34, no. 4 (January 15, 2014): 654–59. http://dx.doi.org/10.1038/jcbfm.2013.241.

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The purpose of this study was to elucidate whether cerebral blood flow (CBF) can better characterize perfusion abnormalities in predementia stages of Alzheimer's disease (AD) than cerebral blood volume (CBV) and whether cortical atrophy is more associated with decreased CBV or with decreased CBF. We compared measurements of CBV, CBF, and mean cortical thickness obtained from magnetic resonance images in a group of healthy controls, patients with mild cognitive impairment (MCI) who converted to AD after 2 years of clinical follow-up (MCI-c), and patients with mild AD. A significant decrease in perfusion was detected in the parietal lobes of the MCI-c patients with CBF parametric maps but not with CBV maps. In the MCI-c group, a negative correlation between CBF values and cortical thickness in the right parahippocampal gyrus suggests an increase in CBF that depends on cortical atrophy in predementia stages of AD. Our study also suggests that CBF deficits appear before CBV deficits in the progression of AD, as CBV abnormalities were only detected at the AD stage, whereas CBF changes were already detected in the MCI stage. These results confirm the hypothesis that CBF is a more sensitive parameter than CBV for perfusion abnormalities in MCI-c patients.
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Taylor, Stephan F., Satoshi Minoshima, and Robert A. Koeppe. "Instability of Localization of Cerebral Blood Flow Activation Foci with Parametric Maps." Journal of Cerebral Blood Flow & Metabolism 13, no. 6 (November 1993): 1040–41. http://dx.doi.org/10.1038/jcbfm.1993.134.

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Adam, Jean-François, Hélène Elleaume, Géraldine Le Duc, Stéphanie Corde, Anne-Marie Charvet, Irène Troprès, Jean-François Le Bas, and François Estève. "Absolute Cerebral Blood Volume and Blood Flow Measurements Based on Synchrotron Radiation Quantitative Computed Tomography." Journal of Cerebral Blood Flow & Metabolism 23, no. 4 (April 2003): 499–512. http://dx.doi.org/10.1097/01.wcb.0000050063.57184.3c.

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Synchrotron radiation computed tomography opens new fields by using monochromatic x-ray beams. This technique allows one to measure in vivo absolute contrast-agent concentrations with high accuracy and precision, and absolute cerebral blood volume or flow can be derived from these measurements using tracer kinetic methods. The authors injected an intravenous bolus of an iodinated contrast agent in healthy rats, and acquired computed tomography images to follow the temporal evolution of the contrast material in the blood circulation. The first image acquired before iodine infusion was subtracted from the others to obtain computed tomography slices expressed in absolute iodine concentrations. Cerebral blood volume and cerebral blood flow maps were obtained after correction for partial volume effects. Mean cerebral blood volume and flow values (n = 7) were 2.1 ± 0.38 mL/100 g and 129 ± 18 mL · 100 g–1 · min–1 in the parietal cortex; and 1.92 ± 0.32 mL/100 g and 125 ± 17 mL · 100 g–1 · min–1 in the caudate putamen, respectively. Synchrotron radiation computed tomography has the potential to assess these two brain-perfusion parameters.
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Ohta, Shinsuke, Ernst Meyer, Hitoshi Fujita, David C. Reutens, Alan Evans, and Albert Gjedde. "Cerebral [15O]Water Clearance in Humans Determined by PET: I. Theory and Normal Values." Journal of Cerebral Blood Flow & Metabolism 16, no. 5 (September 1996): 765–80. http://dx.doi.org/10.1097/00004647-199609000-00002.

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When used to measure blood flow in the brain, water leaves a residue in the vascular bed that influences the estimation of blood flow by current methods. To assess the magnitude of this influence, we developed a two-compartment model of blood flow with separate parameters for transport and vascular distribution of brain water. Maps of the water clearance, K1 into brain tissue, separated from the circulation by a measurably resistant blood–brain barrier (BBB), were generated by time-weighted integration. Depending on the validity of the assumptions underlying the two-compartment model presented here, the maps revealed a significant overestimation of the clearance of water when the vascular residue was ignored. Maps of Vo the estimate of the apparent vascular distribution volume of tracer H215O, clearly revealed major cerebral arteries. Thus, we claim that the accumulation of radioactive water in brain tissue also reflects the volume of the arterial vascular bed of the brain.
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Arteaga, Daniel F., Megan K. Strother, L. Taylor Davis, Matthew R. Fusco, Carlos C. Faraco, Brent A. Roach, Allison O. Scott, and Manus J. Donahue. "Planning-free cerebral blood flow territory mapping in patients with intracranial arterial stenosis." Journal of Cerebral Blood Flow & Metabolism 37, no. 6 (July 20, 2016): 1944–58. http://dx.doi.org/10.1177/0271678x16657573.

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A noninvasive method for quantifying cerebral blood flow and simultaneously visualizing cerebral blood flow territories is vessel-encoded pseudocontinuous arterial spin labeling MRI. However, obstacles to acquiring such information include limited access to the methodology in clinical centers and limited work on how clinically acquired vessel-encoded pseudocontinuous arterial spin labeling data correlate with gold-standard methods. The purpose of this work is to develop and validate a semiautomated pipeline for the online quantification of cerebral blood flow maps and cerebral blood flow territories from planning-free vessel-encoded pseudocontinuous arterial spin labeling MRI with gold-standard digital subtraction angiography. Healthy controls (n = 10) and intracranial atherosclerotic disease patients (n = 34) underwent 3.0 T MRI imaging including vascular (MR angiography) and hemodynamic (cerebral blood flow-weighted arterial spin labeling) MRI. Patients additionally underwent catheter and/or CT angiography. Variations in cross-territorial filling were grouped according to diameters of circle of Willis vessels in controls. In patients, Cohen’s k-statistics were computed to quantify agreement in perfusion patterns between vessel-encoded pseudocontinuous arterial spin labeling and angiography. Cross-territorial filling patterns were consistent with circle of Willis anatomy. The intraobserver Cohen's k-statistics for cerebral blood flow territory and digital subtraction angiography perfusion agreement were 0.730 (95% CI = 0.593–0.867; reader one) and 0.708 (95% CI = 0.561–0.855; reader two). These results support the feasibility of a semiautomated pipeline for evaluating major neurovascular cerebral blood flow territories in patients with intracranial atherosclerotic disease.
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Kato, Naoki, Vincent Prinz, Julius Dengler, and Peter Vajkoczy. "Blood Flow Assessment of Arteriovenous Malformations Using Intraoperative Indocyanine Green Videoangiography." Stroke Research and Treatment 2019 (March 17, 2019): 1–8. http://dx.doi.org/10.1155/2019/7292304.

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Intraoperative indocyanine green (ICG) videoangiography is widely used in patients undergoing neurosurgery. FLOW800 is a recently developed analytical tool for ICG videoangiography to assess semi-quantitative flow dynamics; however, its efficacy is unknown. In this study, we evaluated its functionality in the assessment of flow dynamics of arteriovenous malformation (AVM) through ICG videoangiography under clinical settings. ICG videoangiography was performed in the exposed AVM in eight patients undergoing surgery. FLOW800 analysis was applied directly, and gray-scale and color-coded maps of the surgical field were obtained. After surgery, a region of interest was placed on the individual vessels to obtain time-intensity curves. Parameters of flow dynamics, including the maximum intensity, transit time, and cerebral blood flow index, were calculated using the curves. The color-coded maps provided high-resolution images; however, reconstruction of colored images was restricted by the depth, approach angle, and brain swelling. Semi-quantitative parameters were similar among the feeders, niduses, and drainers. However, a higher cerebral blood flow index was observed in the feeders of large AVM (>3 cm) than in those of small AVM (P < 0.05). Similarly, the cerebral blood flow index values were positively correlated with the nidus volume (P < 0.01). FLOW800 enabled visualization of the AVM structure and safer resection, except in case of deep-seated AVM. Moreover, semi-quantitative values in the individual vessels through using ICG intensity diagram showed different patterns according to size of the AVM. ICG videoangiography showed good performance in evaluating flow dynamics of the AVM in patients undergoing surgery.
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Dissertations / Theses on the topic "Cerebral Blood Flow Maps"

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Alosco, Michael L. "A Prospective Examination of the Effects of Obesity on Cerebral Perfusion and Cognition in Heart Failure." Kent State University / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=kent1395155368.

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Poulin, Marc J. "Aspects of cerebral blood flow in humans." Thesis, University of Oxford, 1998. http://ora.ox.ac.uk/objects/uuid:a2af655f-9198-4cd0-a126-57c070f6399d.

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The technique of transcranial Doppler ultrasound (TCD) was used to assess cerebral blood flow (CBF) in humans. Studies were performed at rest and during dynamic submaximal exercise. In the resting experiments, TCD was combined with the technique of dynamic end-tidal forcing to study the dynamics of the CBF response to step changes in end-tidal (i.e. arterial) PC02 and PO2 In the resting and exercise experiments, the degree of consistency was examined between three indices of CBF that can be extracted from the TCD spectrum. Finally, the ventilatory and the CBF responses to acute isocapnic hypoxia were examined to try to quantify the possible reduction in ventilation that could be attributed to changes in CBF with hypoxia. In the studies performed at rest, during either hypoxia and/or hypercapnia (Chapter 2), the three indices of CBF extracted from the TCD spectrum were all consistent. However, during submaximal exercise (Chapter 5), the indices were less consistent and results suggest that the increase in CBF with exercise that has been reported with TCD needs to be treated with caution. The dynamic studies of the CBF response to step changes in end-tidal PC02 and PO2 in humans revealed that the CBF response to hypercapnia (Chapter 3) is characterised by a significant asymmetry, with a slower on-transient than off-transient, and also by a degree of undershoot following the relief of hypercapnia. The CBF response to hypocapnia (Chapter 4) is also characterised by a significant asymmetry, with a faster on-transient than off-transient. Furthermore, there is a slow progressive adaptation throughout the hypocapnic period. These studies show that the CBF responses to hypercapnia and hypocapnia are much faster than previously been thought. Finally, the work described in Chapter 6 attempts to quantify the possible reduction in ventilation that could be attributed to changes in CBF with hypoxia to determine whether it could be of sufficient magnitude to underlie hypoxic ventilatory decline (HVD). The results suggest that, in awake humans, changes in CBF during acute isocapnic hypoxia are quantitatively insufficient to underlie HVD.
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Mazzeo, M. D. "Lattice-Boltzmann simulations of cerebral blood flow." Thesis, University College London (University of London), 2010. http://discovery.ucl.ac.uk/19357/.

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Computational haemodynamics play a central role in the understanding of blood behaviour in the cerebral vasculature, increasing our knowledge in the onset of vascular diseases and their progression, improving diagnosis and ultimately providing better patient prognosis. Computer simulations hold the potential of accurately characterising motion of blood and its interaction with the vessel wall, providing the capability to assess surgical treatments with no danger to the patient. These aspects considerably contribute to better understand of blood circulation processes as well as to augment pre-treatment planning. Existing software environments for treatment planning consist of several stages, each requiring significant user interaction and processing time, significantly limiting their use in clinical scenarios. The aim of this PhD is to provide clinicians and researchers with a tool to aid in the understanding of human cerebral haemodynamics. This tool employs a high performance fluid solver based on the lattice-Boltzmann method (coined HemeLB), high performance distributed computing and grid computing, and various advanced software applications useful to efficiently set up and run patient-specific simulations. A graphical tool is used to segment the vasculature from patient-specific CT or MR data and configure boundary conditions with ease, creating models of the vasculature in real time. Blood flow visualisation is done in real time using in situ rendering techniques implemented within the parallel fluid solver and aided by steering capabilities; these programming strategies allows the clinician to interactively display the simulation results on a local workstation. A separate software application is used to numerically compare simulation results carried out at different spatial resolutions, providing a strategy to approach numerical validation. This developed software and supporting computational infrastructure was used to study various patient-specific intracranial aneurysms with the collaborating interventionalists at the National Hospital for Neurology and Neuroscience (London), using three-dimensional rotational angiography data to define the patient-specific vasculature. Blood flow motion was depicted in detail by the visualisation capabilities, clearly showing vortex fluid ow features and stress distribution at the inner surface of the aneurysms and their surrounding vasculature. These investigations permitted the clinicians to rapidly assess the risk associated with the growth and rupture of each aneurysm. The ultimate goal of this work is to aid clinical practice with an efficient easy-to-use toolkit for real-time decision support.
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Willie, Christopher Kenneth. "Cerebral blood flow in man : regulation by arterial blood gases." Thesis, University of British Columbia, 2014. http://hdl.handle.net/2429/47074.

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Due to the high metabolic rate of brain tissue and nominal substrate storage, brain perfusion must be precisely regulated to ensure continuous delivery of oxygen and substrates. Cerebral blood flow (CBF) is principally regulated by tissue metabolism, perfusion pressure, autonomic nervous activity, and the partial pressures of arterial oxygen (PaO₂)and carbon dioxide (PaCO₂) – an integrative process thus involving the marked influence of pulmonary gas exchange and cardiovascular function, in addition to intracranial mediators of cerebrovascular resistance. This thesis explicates the roles of PaO₂ and PaCO₂ in human regulation of regional CBF. In study 1, to elucidate their discrete roles, PaO₂ and PaCO₂ were independently manipulated at sea level through the widest range tolerated in humans. Flow reactivity to hypocapnia (low PaCO₂) and hypoxia (low PaO₂) was greater in the vertebral (VA) than internal carotid (ICA) artery, whereas similar reactivity was observed during hypercapnia (high PaCO₂) and hyperoxia (high PaO2₂. Cerebral oxygen delivery was well protected except in cases of extreme hypocapnia. The ventilatory response to hypoxia mitigates falling PaO₂ and reduces PaCO₂, particularly during initial exposure to high altitude. Study 2 assessed regional CBF during ascent to 5050m and every 12 hours during the first 3 days of acclimatization. Although total CBF increased by ~50% and was modestly related to reductions in oxygen saturation of hemoglobin, no regional CBF differences were observed. To extend these findings, Study 3 aimed to determine if cerebrovascular responses to changes in PaO₂ and PaCO₂ differed at 5050m compared to sea level. Despite respiratory alkalosis and partial metabolic compensation at 5050m restoration of PaO₂ to sea level values decreased CBF, and CBF sensitivity to acutely altered PaCO₂ remained similar to sea level. To elucidate the interactive effect on CBF of profound hypoxemia and hypercapnia, study 4 examined the temporal changes in elite breath-hold divers during maximum apneas. Despite 40-50% reductions in arterial oxygen content, CBF elevations were regionally similar (up to +100%) thereby facilitating maintenance of brain oxygen delivery throughout apnea. Although the regulation of CBF is multifaceted, the cerebrovasculature prioritizes oxygen delivery and adjusts to chronic changes in arterial blood gases.
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Shi, Yulu. "Cerebral blood flow and intracranial pulsatility in cerebral small vessel disease." Thesis, University of Edinburgh, 2018. http://hdl.handle.net/1842/29625.

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Cerebral small vessel disease (SVD) is associated with increased risks of stroke and dementia, however the mechanisms remain unclear. Low cerebral blood flow (CBF) has long been suggested and accepted, but clinical evidence is conflicting. On the other hand, growing evidence suggests that increased intracranial pulsatility due to vascular stiffening might be an alternative mechanism. Pulse-gated phase-contrast MRI is an imaging technique that allows measuring of CBF contemporaneously with pulsatility in multiple vessels and cerebrospinal fluid (CSF) spaces. The overall aim of this thesis was to provide an overview of existing clinical evidence on both hypotheses, to test the reproducibility of CBF and pulsatility measures in phase-contrast MRI, and to explore the relationship between CBF and intracranial pulsatility and SVD features in a group of patients with minor stroke and SVD changes on brain imaging. I first systematically reviewed and meta-analysed clinical studies that have assessed CBF or intracranial pulsatility in SVD patients. There were 38 studies (n=4006) on CBF and 27 (n=3356) on intracranial pulsatility. Most were cross-sectional, and longitudinal studies were scarce. There were large heterogeneities in patient characteristics and indices used particularly for measuring and calculating pulsatility. Methods to reduce bias such as blinding and the expertise of structural image readers were generally poorly reported, and many studies did not account for the impact of confounding factors (e.g. age, vascular risk factors and disease severity) on CBF or pulsatility. Evidence for falling CBF predating SVD was not supported by longitudinal studies; high pulsatility in one large artery such as internal carotid arteries (ICA) or middle cerebral arteries might be related to SVD, but studies that measured arteries, veins and CSF in the same patients were very limited and the reliability of some pulsatility measures, especially in CSF, needs to be tested. In order to test the reproducibility of the CBF and intracranial pulsatility measures, I repeated 2D phase-contrast MRI scans of vessels and CSF on healthy volunteers during two visits. I also compared the ICA pulsatility index derived from the MRI flow waveform to that from the Doppler ultrasound velocity waveform in patients with minor stroke and SVD features. In 10 heathy volunteers (age 35.2±9.78 years), the reproducibility of CBF and vascular pulsatility indices was good, with within-subject coefficients of variability (CV) less than 10%; whereas CSF flow and pulsatility measures were generally less reproducible (CV > 20%). In 56 patients (age 67.8±8.27 years), the ICA pulsatility indices in Doppler ultrasound and MRI were acceptably well-correlated (r=0.5, p < 0.001) considering the differences in the two techniques. We carried out a cross-sectional study aiming to recruit 60 patients with minor stroke and SVD features. We measured CBF and intracranial pulsatility using phase-contrast MRI, as well as aortic augmentation index (AIx) using a SphygmoCor device. I first investigated the relationship between intracranial measures, and systemic blood pressure or aortic AIx, and then focused on how the intracranial haemodynamic measures related to two main SVD features (white matter hyperintensities (WMH) and perivascular spaces (PVS)). We obtained usable data from 56/60 patients (age 67.8±8.27 years), reflecting a range of SVD burdens. After the adjustment for age, gender, and history of hypertension, higher pulsatility in the venous sinuses was associated with lower diastolic blood pressure and lower mean arterial pressure (e.g. diastolic blood pressure on straight sinus pulsatility index (PI): β=-0.005, P=0.029), but not with aortic AIx. Higher aortic AIx was associated with low ICA PI (β=-0.011, P=0.040). Increased pulsatility in the venous sinuses, not low CBF, was associated with greater WMH volume (e.g. superior sagittal sinus PI: β=1.29, P=0.005) and more basal ganglia PVS (e.g. odds ratio=1.379 per 0.1 increase in superior sagittal sinus PI) after the adjustment for age, gender and blood pressure. The thesis is the first to summarise the literature on CBF and intracranial pulsatility in SVD patients, addressed the major limitations of current clinical studies of SVD, and also assessed CBF and intracranial pulsatility contemporaneously in well-characterised patients with SVD features. The overall results of the thesis challenge the traditional hypothesis of the cause and effect between low CBF and SVD, and suggest that increased cerebrovascular pulsatility, which might be due to intrinsic cerebral small vessel pathologies rather than just aortic stiffness, is important for SVD. More importantly, this pilot study also provides a reliable methodology for measuring intracranial pulsatility using phase-contrast MRI for future longitudinal or larger multicentre studies, and shows that intracranial pulsatility could be used as a secondary outcome in clinical trials of SVD. However, future research is required to elucidate the implication of venous pulsatility and to fully explore the passage of pulse wave transmission in the brain. Overall, this thesis advances knowledge and suggest potential targets for future SVD studies in terms of mechanisms, prevention and treatment.
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吳志萍 and Chi-ping Ng. "Cerebral blood flow monitoring of brain injured patients." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1996. http://hub.hku.hk/bib/B31214484.

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Ng, Chi-ping. "Cerebral blood flow monitoring of brain injured patients /." Hong Kong : University of Hong Kong, 1996. http://sunzi.lib.hku.hk/hkuto/record.jsp?B18777077.

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Bathula, Rajaram. "Ethnic differences in cerebral blood flow and its determinants." Thesis, Imperial College London, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.522845.

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Khlunovska, L. Yu. "Peculiarities of cerebral blood flow in children with headache." Thesis, БДМУ, 2017. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/17135.

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Appaji, Abhisara Apoorva. "Estimating Cerebral Blood Flow from a Flow from a Rotatinal Angiographic System." Thesis, University of Oxford, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.504287.

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Books on the topic "Cerebral Blood Flow Maps"

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Pinsky, Michael R. Cerebral Blood Flow. Berlin, Heidelberg: Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/978-3-642-56036-1.

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Rescigno, Aldo, and Andrea Boicelli, eds. Cerebral Blood Flow. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4684-5565-6.

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Schmiedek, Peter, Karl Einhäupl, and Carl-Martin Kirsch, eds. Stimulated Cerebral Blood Flow. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-77102-6.

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Mitagvariia, N. P. Cerebral blood flow regulation. New York: Nova Science Publishers, 2009.

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Edvinsson, Lars. Cerebral blood flow and metabolism. New York: Raven Press, 1993.

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N, Krause Diana, ed. Cerebral blood flow and metabolism. 2nd ed. Philadelphia: Lippincott Williams & Wilkins, 2002.

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Hartmann, Alexander, and Siegfried Hoyer, eds. Cerebral Blood Flow and Metabolism Measurement. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70054-5.

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W, Phillis J., ed. The Regulation of cerebral blood flow. Boca Raton: CRC Press, 1993.

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Cold, Georg Emil. Cerebral Blood Flow in Acute Head Injury. Vienna: Springer Vienna, 1990. http://dx.doi.org/10.1007/978-3-7091-9101-9.

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Kreipke, Christian W., and Jose A. Rafols, eds. Cerebral Blood Flow, Metabolism, and Head Trauma. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-4148-9.

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Book chapters on the topic "Cerebral Blood Flow Maps"

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Luo, Qingming, and Zheng Wang. "Temporal Clustering Analysis of Cerebral Blood Flow Activation Maps Measured by Laser Speckle Contrast Imaging." In Biophotonics, 73–84. Boston, MA: Springer US, 2005. http://dx.doi.org/10.1007/0-387-24996-6_6.

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Hayashi, Nariyuki, and Dalton W. Dietrich. "Cerebral Blood Flow." In Brain Hypothermia Treatment, 27. Tokyo: Springer Japan, 2004. http://dx.doi.org/10.1007/978-4-431-53953-7_17.

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Roth, Elliot J. "Cerebral Blood Flow." In Encyclopedia of Clinical Neuropsychology, 731. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_2165.

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Hoyer, Daniel, Eric P. Zorrilla, Pietro Cottone, Sarah Parylak, Micaela Morelli, Nicola Simola, Nicola Simola, et al. "Cerebral Blood Flow." In Encyclopedia of Psychopharmacology, 277. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_4115.

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Roth, Elliot J. "Cerebral Blood Flow." In Encyclopedia of Clinical Neuropsychology, 526–27. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-79948-3_2165.

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Roth, Elliot J. "Cerebral Blood Flow." In Encyclopedia of Clinical Neuropsychology, 1. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-56782-2_2165-2.

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McGrath, Marie C. "Cerebral Blood Flow." In Encyclopedia of Child Behavior and Development, 323. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-0-387-79061-9_498.

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Suzuki, Jiro. "Cerebral Blood Flow." In Treatment of Cerebral Infarction, 54–88. Vienna: Springer Vienna, 1987. http://dx.doi.org/10.1007/978-3-7091-8861-3_4.

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Müller, Edgar. "Flow Studies." In Cerebral Blood Flow, 215–43. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4684-5565-6_12.

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Rodriguez, G., F. De Carli, G. Novellone, S. Marenco, and G. Rosadini. "Regional Cerebral Blood Flow Measurements Using the 133-Xenon Inhalation Method." In Cerebral Blood Flow, 121–43. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4684-5565-6_7.

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Conference papers on the topic "Cerebral Blood Flow Maps"

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Fueanggan, Santichai, Somchart Chokchaitam, and Sombat Muengtaweepongsa. "Automatic detection of ischemic stroke area from CT perfusion maps Cerebral Blood Volume and Cerebral Blood Flow." In 2011 International Symposium on Intelligent Signal Processing and Communications Systems (ISPACS 2011). IEEE, 2011. http://dx.doi.org/10.1109/ispacs.2011.6146202.

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Fueanggan, Santichai, Somchart Chokchaitam, and Sombat Muengtaweepongsa. "Ischemic stroke analysis of CT perfusion maps Cerebral Blood Volume and Cerebral Blood Flow based on Digital Image Processing techniques." In 2011 Biomedical Engineering International Conference (BMEiCON) - Conference postponed to 2012. IEEE, 2012. http://dx.doi.org/10.1109/bmeicon.2012.6172041.

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Wang, Zheng, and Qingming Luo. "Temporal clustering analysis of cerebral blood flow activation maps measured by laser speckle contrast imaging." In Biomedical Optics 2004, edited by Valery V. Tuchin. SPIE, 2004. http://dx.doi.org/10.1117/12.535380.

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Lima, Iana Campinho Braga de Araújo, Lavínia Flávia Xavier de Souza, Clara Wilma Fernandes Rosendo, Letícia de Freitas Barradas, Emerson Kennedy Ribeiro de Andrade Filho, Pedro Vilar de Oliveira Villarim, Gianluca Gomes Siebra, Ródio Luis Brandão Câmara, and Moisés Felipe da Costa Fernandes. "Cerebral venous thrombosis simulating cerebral arterial thrombosis: Late complication of COVID-19?" In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.345.

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Context: Brain venous thrombosis (BVT) is uncommon and usually has a different clinic and treatment from cerebral arterial thrombosis. In this context, COVID-19 correlates with thrombogenesis with varied clinical repercussions. This report describes an unusual BVT case as a possible late complication of COVID-19. Case report: Male, 68 years old, athlete and healthy. April/2020: COVID-19 mild symptoms. February/2021: in road-running, he fell due to sudden left hemiparesis. Upon hospital admission: contacting, persistent headache. A week after, low level of consciousness and coma, when underwent right hemicraniectomy. Remains hospitalized. On examination: weak gestural communication, tracheostomy, enteral tube feeding, voluntary blinking. Maintains neutral cervical posture, masticatory automatisms, photoreactive isocoria, generalized rigidity, decorticated right hemiparesis, left hemiplegia. On imaging: hemorrhagic infarction on the right and mass effect due to obstruction of the Basal Rosenthal and Labbé veins and transverse sinus on the right, with venous blood flow in the rest of the hemisphere diverted to the ipsilateral internal jugular vein, by anastomotic veins of the occipital foramen and suboccipital venous plexus. Obstructed left internal jugular vein, with venous collateral flow from the left hemisphere via posterior intercavernous sinus and basilar plexus to the right internal jugular vein. Conclusions: To diagnose the venous etiology that resembled segmental occlusion of the right middle cerebral artery, CT angiography was required. Late evolution of COVID-19 has been identified by the persistence of symptoms for months. Although physical activity and possible dehydration may have contributed to BVT, a prothrombotic state correlated to COVID-19 cannot be discarded.
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Lieber, Baruch B., Chander Sadasivan, Matthew J. Gounis, and Ajay K. Wakhloo. "The Role of Blood Impulse in Cerebral Aneurysm Coil Compaction: Effect of Aneurysm Neck Size." In ASME 2003 International Mechanical Engineering Congress and Exposition. ASMEDC, 2003. http://dx.doi.org/10.1115/imece2003-43099.

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Endovascular occlusion of cerebral aneurysms with bare platinum detachable coils is now recognized as preferable to surgical clipping (ISUIA Group, 2003, ISAT Group, 2002, Bavinzski et al, 1995, Thornton et al, 2002). Dependent on coil packing density (the ratio of the coil volume deposited in an aneurysm to that of the aneurysm volume), aneurysm location, size and neck width, coil compaction with recanalization of the aneurysm remains in the long-term a major concern. The aneurysm neck size is reported to be the main predictor for aneurysm recanalization (Fernandez-Zubillaga et al, 1994). The forces exerted on the coil mass at the aneurysm neck due to blood pulsatility are larger for wide neck aneurysms as compared to small neck aneurysms (Bavinzski et al, 1995). However, impingement forces have not been evaluated. We evaluated the force impinging on the aneurysm neck in a simplified aneurysm (basilar top) geometry utilizing the impulse-momentum equation and Womersley’s flow. Maximum impingement force as a function of aneurysm neck to parent lumen diameter ratio varies as a sigmoid curve. Analysis of the hemodynamic forces affecting coil compaction in cerebral aneurysms shows that the coil mass at the aneurysm neck may be subjected to cyclic impulse impingement due to redirection of blood momentum. Orientation of the aneurysm neck and the main axis of the aneurysm in relation to the oncoming parent vessel flow may help clinicians predict the risk of coil compaction and the location of subsequent aneurysm recanalization.
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Sadasivan, C., B. B. Lieber, D. K. Lopes, A. J. Ringer, and L. N. Hopkins. "Modeling of Angiographic Dye Washout From Cerebral Aneurysms Before and After Stenting: An Index for Stent Efficacy." In ASME 2001 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2001. http://dx.doi.org/10.1115/imece2001/bed-23130.

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Abstract The ultimate goal in the treatment of cerebral aneurysms is to exclude them from the intracranial circulation while preserving the parent artery. Recently, in vivo and in vitro experiments and clinical studies demonstrated that endovascular stenting is a significant and viable treatment option for cerebral aneurysms. Stents reduce the mass and momentum transport of blood from the parent artery into the aneurysm and alter both intra-aneurysmal flow and inflow-outflow patterns. The reduction of vorticity and flow stasis within the sac leads to thrombus formation and eventual exclusion of the aneurysm from the circulation. Digital subtraction angiography (DSA) has become an essential clinical tool for the diagnosis and treatment of aneurysms and is an important adjunct to stenting procedures.
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Atochin, Dmitriy. "CEREBRAL BLOOD FLOW REGULATION DURING STROKE." In XV International interdisciplinary congress "Neuroscience for Medicine and Psychology". LLC MAKS Press, 2019. http://dx.doi.org/10.29003/m307.sudak.ns2019-15/72.

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Aoi, M., P. Gremaud, H. T. Tran, V. Novak, and M. S. Olufsen. "Modeling cerebral blood flow and regulation." In 2009 Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2009. http://dx.doi.org/10.1109/iembs.2009.5334057.

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Balberg, Michal, Thao Pham, Giles Blaney, Angelo Sassaroli, and Sergio Fantini. "Depth Profiles of Cerebral Blood Flow Modulation." In Optical Tomography and Spectroscopy. Washington, D.C.: OSA, 2020. http://dx.doi.org/10.1364/ots.2020.stu4d.2.

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Balberg, Michal, and Revital Shechter. "Acousto Optics for Cerebral Blood Flow Monitoring." In Optics and the Brain. Washington, D.C.: OSA, 2019. http://dx.doi.org/10.1364/brain.2019.bw1a.1.

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Reports on the topic "Cerebral Blood Flow Maps"

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Bodo, Michael, Frederick Pearce, Stephen Van Albert, and Rocco Armonda. Rheoencephalogram Reflects Cerebral Blood Flow Autoregulation in Pigs. Fort Belvoir, VA: Defense Technical Information Center, January 2007. http://dx.doi.org/10.21236/ada473927.

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Kovitaya, Manaswee, Lloyd D. Tripp, Chelette Jr., and Tamara L. Middle Cerebral Artery Blood Flow Velocity After Exposure to Sustained +Gz. Fort Belvoir, VA: Defense Technical Information Center, June 1997. http://dx.doi.org/10.21236/ada337565.

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Bednarczyk, Edward M. A Comparison of Cerebral Blood Flow in Migraineurs during Headache-Free and Treatment Periods. Fort Belvoir, VA: Defense Technical Information Center, October 1996. http://dx.doi.org/10.21236/ada328296.

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Bednarczyk, Edward M. A Comparison of Cerebral Blood Flow in Migraineurs During Headache, Headache-Free and Treatment Periods. Fort Belvoir, VA: Defense Technical Information Center, January 1999. http://dx.doi.org/10.21236/ada374068.

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Liao, Lu, Yong Fu, Xiong Jun, Haif Zhang, Xiaoq Li, and Wen Yu. A meta-analysis of acupuncture and moxibustion used to improve migraine attack symptoms and cerebral blood flow velocity. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2020. http://dx.doi.org/10.37766/inplasy2020.6.0066.

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Jones, A., A. Packard, S. Treves, and A. Davison. Studies in technetium chemistry, Project 1: Evaluation of technetium acetylacetonates as potential cerebral blood flow agents, Project 2. Office of Scientific and Technical Information (OSTI), January 1990. http://dx.doi.org/10.2172/7002570.

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