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Journal articles on the topic 'Cerebral arteriovenous malformations'

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Published: 4 June 2021

Last updated: 23 February 2023

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1

Janicijevic, Milos. "Surgery of the arteriovenous cerebral malformations." Acta chirurgica Iugoslavica 55, no. 2 (2008): 11–16. http://dx.doi.org/10.2298/aci0802011j.

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According to the present technical possibilities modalities of the treatment of the arteriovenous cerebral malformations (surgery, embolization, radiosurgery) for direct neurosurgical excision of malformations remains of cases. Decision to operate is made separately for each malformation and is based on anticipation of the natural course of the illness, precise estimate of the risk from operation and on the estimate of the condition of the patient. Surgical technique is also chosen for each malformation separately, depending on its size, angioarchitecture, hemodynamic characteristics and localization. The following techniques are used: resection of the malformation "en blocque", coagulation of the lesion in situ, gradual marginal coagulation in the lesion cleavage, "backword technique", perivenous approach and excision of the complex malformations in several acts. The safety of the surgical excision is increased by preoperative and perioperative actions already described. Intraoperative problems and postoperative complications (brain edema, uncontrolled hemorrhage, intracerebral hematoma and others) were discussed.

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2

Aguiar, Paulo Henrique, Marco Antonio Stefani, Gustavo Rassier Isolan, Carlos Alexandre Zicarelli, and Apio Claudio Martins Antunes. "Cerebral Arteriovenous Malformations." JBNC - JORNAL BRASILEIRO DE NEUROCIRURGIA 23, no. 4 (March 29, 2018): 301–15. http://dx.doi.org/10.22290/jbnc.v23i4.1215.

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A significant improvement of central nervous system arteriovenous vascular malformations (AVM) outcome has been observed due to the advances in all modalities of diagnosis and treatment. The authors report the advances in diagnosis and integrated global treatment of AVM`s.

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3

Millichap, J. Gordon. "Cerebral Arteriovenous Malformations." Pediatric Neurology Briefs 8, no. 9 (September 1, 1994): 70. http://dx.doi.org/10.15844/pedneurbriefs-8-9-10.

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4

Venes, Joan. "CEREBRAL ARTERIOVENOUS MALFORMATIONS." Neurosurgery 30, no. 4 (April 1, 1992): 652. http://dx.doi.org/10.1097/00006123-199204000-00036.

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5

H??llerhage, H.-G. "Cerebral Arteriovenous Malformations." Neurosurgery 31, no. 3 (September 1992): 603???604. http://dx.doi.org/10.1097/00006123-199209000-00030.

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6

Tamaki, Norihiko, and Kazumasa Ehara. "Cerebral Arteriovenous Malformations." Neurosurgery 31, no. 3 (September 1992): 605. http://dx.doi.org/10.1097/00006123-199209000-00031.

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7

Warren, Daniel James, Nigel Hoggard, Lee Walton, Matthias Walter Richard Radatz, Andras A. Kemeny, David Martin Campbell Forster, Iain David Wilkinson, and Paul David Griffiths. "CEREBRAL ARTERIOVENOUS MALFORMATIONS." Neurosurgery 61, suppl_1 (July 1, 2007): 973–83. http://dx.doi.org/10.1227/01.neu.0000279215.07763.a1.

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8

Farhat, Hamad I. "Cerebral Arteriovenous Malformations." Disease-a-Month 57, no. 10 (October 2011): 625–37. http://dx.doi.org/10.1016/j.disamonth.2011.08.021.

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9

Venes, Joan. "CEREBRAL ARTERIOVENOUS MALFORMATIONS." Neurosurgery 30, no. 4 (April 1992): 652. http://dx.doi.org/10.1227/00006123-199204000-00036.

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10

Höllerhage, H.-G. "Cerebral Arteriovenous Malformations." Neurosurgery 31, no. 3 (September 1, 1992): 603–4. http://dx.doi.org/10.1227/00006123-199209000-00030.

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11

Tamaki, Norihiko, and Kazumasa Ehara. "Cerebral Arteriovenous Malformations." Neurosurgery 31, no. 3 (September 1, 1992): 605. http://dx.doi.org/10.1227/00006123-199209000-00031.

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12

Meder, J. F., C. Oppenheim, J. Blustajn, F. Nataf, L. Merienne, D. Lefkoupolos, A. Laurent, J. J. Merland, M. Schlienger, and D. Fredy. "Cerebral Arteriovenous Malformations." Journal of Neuro-Ophthalmology 19, no. 2 (June 1999): 117. http://dx.doi.org/10.1097/00041327-199906000-00045.

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13

Gold, Arnold P. "CEREBRAL ARTERIOVENOUS MALFORMATIONS." Developmental Medicine & Child Neurology 15, no. 1 (November 12, 2008): 84–87. http://dx.doi.org/10.1111/j.1469-8749.1973.tb04874.x.

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14

Jervis, S., and D. Skinner. "Screening for arteriovenous malformations in hereditary haemorrhagic telangiectasia." Journal of Laryngology & Otology 130, no. 8 (July 5, 2016): 734–42. http://dx.doi.org/10.1017/s0022215116008422.

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AbstractObjective:To determine whether patients with hereditary haemorrhagic telangiectasia were being screened according to international guidelines, and to review recent evidence in order to provide up-to-date guidelines for the initial systemic management of hereditary haemorrhagic telangiectasia.Methods:A retrospective case note analysis was conducted, assessing patients in terms of screening for: genetics, cerebral arteriovenous malformations, pulmonary and hepatic arteriovenous malformations, and gastrointestinal telangiectasia. Databases searched included Medline, the Cumulative Index to Nursing and Allied Health Literature, and Embase.Results:Screening investigations were most frequently performed for hepatic arteriovenous malformations and least frequently for genetics. Recent data suggest avoiding routine genetic and cerebral arteriovenous malformation screening because of treatment morbidities; performing high-resolution chest computed tomography for pulmonary arteriovenous malformation screening; using capsule endoscopy (if possible) to reduce complications from upper gastrointestinal endoscopy; and omitting routine liver enzyme testing in favour of Doppler ultrasound.Conclusion:Opportunities for systemic arteriovenous malformation screening are frequently overlooked. This review highlights the need for screening and considers the form in which it should be undertaken.

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15

Rodríguez-Hernández, Ana, Helen Kim, Tony Pourmohamad, William L. Young, and Michael T. Lawton. "Cerebellar Arteriovenous Malformations." Neurosurgery 71, no. 6 (September 24, 2012): 1111–24. http://dx.doi.org/10.1227/neu.0b013e318271c081.

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ABSTRACT BACKGROUND: Anatomic diversity among cerebellar arteriovenous malformations (AVMs) calls for a classification that is intuitive and surgically informative. Selection tools like the Spetzler-Martin grading system are designed to work best with cerebral AVMs but have shortcomings with cerebellar AVMs. OBJECTIVE: To define subtypes of cerebellar AVMs that clarify anatomy and surgical management, to determine results according to subtypes, and to compare predictive accuracies of the Spetzler-Martin and supplementary systems. METHODS: From a consecutive surgical series of 500 patients, 60 had cerebellar AVMs, 39 had brainstem AVMs and were excluded, and 401 had cerebral AVMs. RESULTS: Cerebellar AVM subtypes were as follows: 18 vermian, 13 suboccipital, 12 tentorial, 12 petrosal, and 5 tonsillar. Patients with tonsillar and tentorial AVMs fared best. Cerebellar AVMs presented with hemorrhage more than cerebral AVMs (P < .001). Cerebellar AVMs were more likely to drain deep (P = .04) and less likely to be eloquent (P < .001). The predictive accuracy of the supplementary grade was better than that of the Spetzler-Martin grade with cerebellar AVMs (areas under the receiver-operating characteristic curve, 0.74 and 0.59, respectively). The predictive accuracy of the supplementary system was consistent for cerebral and cerebellar AVMs, whereas that of the Spetzler-Martin system was greater with cerebral AVMs. CONCLUSION: Patients with cerebellar AVMs present with hemorrhage more often than patients with cerebral AVMs, justifying an aggressive treatment posture. The supplementary system is better than the Spetzler-Martin system at predicting outcomes after cerebellar AVM resection. Key components of the Spetzler-Martin system such as venous drainage and eloquence are distorted by cerebellar anatomy in ways that components of the supplementary system are not.

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16

Shakur, Sophia, and Seon-Kyu Lee. "Pediatric cerebral arteriovenous malformations." Journal of Pediatric Neuroradiology 02, no. 03 (July 29, 2015): 277–81. http://dx.doi.org/10.3233/pnr-13066.

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17

Cockroft, Kevin M. "Unruptured Cerebral Arteriovenous Malformations." Stroke 37, no. 5 (May 2006): 1148–49. http://dx.doi.org/10.1161/01.str.0000220514.28847.ac.

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18

Tsitsopoulos, P., J. Andrew, and M. J. Harrison. "Occult cerebral arteriovenous malformations." Journal of Neurology, Neurosurgery & Psychiatry 50, no. 2 (February 1, 1987): 218–20. http://dx.doi.org/10.1136/jnnp.50.2.218.

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19

Gomez, C. R. "Hereditary Cerebral Arteriovenous Malformations." Archives of Neurology 46, no. 11 (November 1, 1989): 1168. http://dx.doi.org/10.1001/archneur.1989.00520470018016.

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20

Heros, Roberto C. "Multimodality Treatment of Cerebral Arteriovenous Malformations: Modern Treatment of Cerebral Arteriovenous Malformations." World Neurosurgery 82, no. 1-2 (July 2014): 46–48. http://dx.doi.org/10.1016/j.wneu.2013.03.025.

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21

Lv, Xianli, Wei Li, Hongwei He, Chuhan Jiang, and Youxiang Li. "Known and unknown cerebral arteriovenous malformations in pregnancies: haemorrhage risk and influence on obstetric management." Neuroradiology Journal 30, no. 5 (June 21, 2017): 437–41. http://dx.doi.org/10.1177/1971400917712264.

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Objective The objective of this study was to evaluate the haemorrhage risk of known and unknown cerebral arteriovenous malformations and their obstetric management. Methods A retrospective review was performed and analysed 67 consecutive cases of arteriovenous malformation with pregnancy history. Results Sixty-seven cases of arteriovenous malformation with pregnancy histories were identified. In 14 cases (20.9%) of arteriovenous malformation diagnosed before pregnancy, 11 cases were treated (10 embolisation and one surgery), there was no haemorrhage in 14 pregnancies, 14 healthy babies were delivered by caesarean section in 12 pregnancies (85.7%) and vaginal delivery in two pregnancies (14.3%). In 53 cases (89.1%) of arteriovenous malformation diagnosed during/after pregnancy, there was one (1.6%) case of subarachnoid haemorrhage at 38 weeks’ gestation in 64 pregnancies, 64 healthy babies were delivered by caesarean section in 11 pregnancies (17.2%) and vaginal delivery in 53 pregnancies (82.8%). This resulted in 1.6% (95% confidence interval 0–4.6%) haemorrhage rate per pregnancy in unknown arteriovenous malformations. Known arteriovenous malformation gravida was prone to caesarean section; however, vaginal delivery did not increase the haemorrhage risk in unknown arteriovenous malformation gravidas (1.8% vs. 0%, P = 1.000). Conclusion Prior treatment for ruptured arteriovenous malformation could prevent its haemorrhage during pregnancy and the haemorrhage risk of unruptured arteriovenous malformation in pregnancies is low. Although known arteriovenous malformation gravida is prone to caesarean section, vaginal delivery seems not to increase the haemorrhage risk in unknown arteriovenous malformation gravidas.

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22

Fahed, Robert, Tim E. Darsaut, Charbel Mounayer, René Chapot, Michel Piotin, Raphaël Blanc, Vitor Mendes Pereira, et al. "Transvenous Approach for the Treatment of cerebral Arteriovenous Malformations (TATAM): Study protocol of a randomised controlled trial." Interventional Neuroradiology 25, no. 3 (February 4, 2019): 305–9. http://dx.doi.org/10.1177/1591019918821738.

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Background Transvenous embolisation is a promising technique but the benefits remain uncertain. We hypothesised that transvenous embolisation leads to a higher rate of arteriovenous malformation angiographic occlusion than transarterial embolisation. Methods The Transvenous Approach for the Treatment of cerebral Arteriovenous Malformations (TATAM) is an investigator initiated, multicentre, prospective, phase 2, randomised controlled clinical trial. To test the hypothesis that transvenous embolisation is superior to transarterial embolisation for arteriovenous malformation obliteration, 76 patients with arteriovenous malformations considered curable by up to two sessions of endovascular therapy will be randomly allocated 1:1 to treatment with either transvenous embolisation (with or without transarterial embolisation) (experimental arm) or transarterial embolisation alone (control arm). The primary endpoint of the trial is complete arteriovenous malformation occlusion, assessed by catheter cerebral angiography. Complete occlusions will be confirmed at 3 months, while incompletely occluded arteriovenous malformations, considered treatment failures, will then be eligible for complementary treatments by surgery, radiation therapy, or even transvenous embolisation. Standard procedural safety outcomes will also be assessed. Patient selection will be validated by a case selection committee, and participating centres with limited experience in transvenous embolisation will be proctored. Discussion The TATAM trial is a transparent research framework designed to offer a promising but still unvalidated treatment to selected arteriovenous malformation patients. Clinical Trial Registration-URL: http://www.clinicaltrials.gov . Unique identifier: NCT03691870.

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23

Shchehlov, D. V., O. Ye Svyrydiuk, S. V. Chebanyuk, and M. B. Vyval. "Endovascular embolization of cerebral arteriovenous malformations." Ukrainian Interventional Neuroradiology and Surgery 37, no. 3 (February 3, 2022): 69–76. http://dx.doi.org/10.26683/2786-4855-2021-3(37)-69-76.

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Endovascular embolization is a critical component in the treatment of cerebral arteriovenous malformations. It can be used as an independent treatment modality or as an adjunct to microurgery or radiosurgery. The published literature in the PubMed database until September 2021 was reviewed with reference to the results of cerebral arteriovenous malformations embolization using liquid embolic agents. More scientific data reporting about total embolization of the cerebral arteriovenous malformations with a final cure. Although complications and mortality after arteriovenous malformations embolization have decreased significantly, but they still exist, and decisions about it usage should be weighed against its benefits during the planning phase. Treatment of arteriovenous malformations of the brain requires a multidisciplinary approach involving vascular neurosurgeons, endovascular interventionists and radiation oncologists, with a deep understanding of the natural course and combination of risks of multimodal treatment. Only such approach can increase the likelihood of a positive outcome of the cerebral arteriovenous malformations treatment.

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24

Kim, Louis J., Felipe C. Albuquerque, Robert F. Spetzler, and Cameron G. McDougall. "Postembolization Neurological Deficitsin Cerebral Arteriovenous Malformations: Stratification By Arteriovenous Malformation Grade." Neurosurgery 59, no. 1 (July 1, 2006): 53–59. http://dx.doi.org/10.1227/01.neu.0000219219.97287.91.

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25

Eliahu, Karen, Florence Hofman, and Steven Giannotta. "A Systematic Review of Cerebral Arteriovenous Malformation Management." International Journal of Medical Students 5, no. 2 (August 31, 2017): 74–80. http://dx.doi.org/10.5195/ijms.2017.13.

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Cerebral Arteriovenous Malformation is a neurovascular lesion characterized by an abnormal connection between arterial and venous systems, resulting in a tangle of blood vessels lacking intervening capillaries. The goal of treatment is to prevent catastrophic hemorrhage, neurological injury, or death. Despite the availability of multiple cutting-edge treatment options there is little consensus on the most promising approaches for treatment due to the novelty of each Arteriovenous Malformation case. This analysis will link the various angioarchitectural characteristics and associated presentations of Arteriovenous Malformation to treatment modalities. In the era of personalized medicine, genomics-driven research to normalize by drawing parallels between Cerebral Cavernous Malformation and Arteriovenous Malformation, both of which are characterized by hemorrhage-prone vascular malformations, may provide insight for the development of pharmacological therapy. Understanding the underlying mechanisms and genes responsible for the symptoms will allow us to better treat patients in a non-invasive manner and paves future directions in Arteriovenous Malformation treatment.

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26

Woodall, M. Neil, Melissa McGettigan, Ramon Figueroa, James R. Gossage, and Cargill H. Alleyne. "Cerebral vascular malformations in hereditary hemorrhagic telangiectasia." Journal of Neurosurgery 120, no. 1 (January 2014): 87–92. http://dx.doi.org/10.3171/2013.10.jns122402.

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Object Hereditary hemorrhagic telangiectasia (HHT) is a hereditary disorder characterized by mucocutaneous telangiectasias, frequent nosebleeds, and visceral arteriovenous malformations (AVMs). Few reports have outlined the prevalence of the various cerebral vascular malformations found in patients with HHT. The authors set out to define the prevalence of cerebral vascular malformations in a population of HHT patients who underwent imaging with 3-T imaging (MRI/MR angiography [MRA]) of the brain. Methods A retrospective review of prospectively collected data was carried out using a database of 372 HHT patients who were seen and examined at the Georgia Regents University HHT Center and screened with 3-T MRI/MRA. Data were tabulated for numbers and types of vascular malformations in this population. Results Arteriovenous malformations were identified in 7.7%, developmental venous anomalies in 4.3%, and cerebral aneurysms in 2.4% of HHT patients. The HHT AVMs tended to be supratentorial, small, and cortical in this series, findings consistent with other recent studies in the literature. An arteriovenous fistula, cavernous malformation, and capillary telangiectasia were identified in 0.5%, 1%, and 1.9% of HHT patients, respectively. Conclusions Few studies have investigated the prevalence of the various vascular malformations found in HHT patients screened with 3-T MRI/MRA of the brain. Hereditary hemorrhagic telangiectasia AVMs are more likely to be multiple and have a tendency toward small size and cortical location. As such, they are often treated using a single-modality therapy.

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27

Prirodov, A. V., E. Yu Bakharev, R. M. Kozlova, M. Yu Sinkin, and A. A. Grin. "Case report: Surgical treatment of combined dural arteriovenous fistula and angiographically occult arteriovenous malformation complicated by intracerebral hemorrhage." Russian journal of neurosurgery 24, no. 4 (December 27, 2022): 85–94. http://dx.doi.org/10.17650/1683-3295-2022-24-4-85-94.

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Background. Cerebral arteriovenous malformations and dural arteriovenous fistulas are relatively rare pathologies with frequency of 1–1.5 and 0.1–0.2 cases per 100 000 people per year, respectively. Probability of a combination of these pathologies in a single patient is extremely low. The authors were not able to find publications describing a combination of these pathologies of the cerebral vasculature.Aim. To present a clinical case of successful surgical treatment of combined cerebral pathology – dural arteriovenous fistula and angiographically occult arteriovenous malformation – in a patient with intracranial hemorrhage.Materials and methods. Analysis of the results of treatment of patient with dural arteriovenous fistula with hemorrhagic disease course at the N.V. Sklifosovsky Research Institute of Emergency Medicine (Moscow) was performed. The patient underwent osteoplastic cranial trepanation, resection of the dural arteriovenous fistula. Intraoperatively an angiographically occult arteriovenous malformation was detected which caused the hemorrhage.Results. Surgical treatment allowed to achieve a satisfactory outcome. Intraoperatively diagnosed angiographically occult arteriovenous malformation was confirmed histologically.Conclusions. The presented clinical observation shows the necessity of careful revision of hematoma cavity in the presence of isolated intraparenchymal hemorrhage from dural arteriovenous fistula.

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28

Adroos, N. "Cerebral and pulmonary arteriovenous malformations." South African Journal of Radiology 10, no. 1 (February 14, 2006): 21. http://dx.doi.org/10.4102/sajr.v10i1.192.

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29

Hashimoto, Nobuo, Kazuhiko Nozaki, Yasushi Takagi, Ken-ichiro Kikuta, and Nobuhiro Mikuni. "SURGERY OF CEREBRAL ARTERIOVENOUS MALFORMATIONS." Neurosurgery 61, suppl_1 (July 1, 2007): SHC—375—SHC—389. http://dx.doi.org/10.1227/01.neu.0000255491.95944.eb.

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Abstract Despite remarkable progress, the microsurgical extirpation of cerebral arteriovenous malformations (AVMs) even by experienced neurosurgeons is not always easy or safe. This article focuses on how to render AVM surgery safer, and offers strategies and tactics for avoiding perilous bleeding and preserving postoperative neurological function. Our treatment strategies and surgical techniques are offered from the operating surgeon's perspective. An understanding of pathophysiology of cerebral AVMs is important for their appropriate surgical treatment. Sophisticated neuroimaging techniques and scrupulous neurophysiological examinations alert to possible complications, and improved surgical approaches help to minimize the sequelae of unanticipated complications. At the early stage of cerebral AVM surgery, extensive dissection of the sulci, fissures, and subarachnoid cistern should be performed to expose feeders, nidus, and drainers. Problems with the surgery of large and/or deep-seated lesions are exacerbated when arterial bleeding from the nidus continues even after all major feeders are thought to have been occluded. We routinely place catheters for angiography at the surgery of complex AVMs to find missing feeding arteries or to identify the real-time hemodynamic status of the lesion. Temporary clip application on feeders and less coagulation of the nidus is necessary to control intranidal pressure and to avoid uncontrollable bleeding from the nidus and adjacent brain. Intraoperative navigation images superimposed on tractography images can provide us with valuable information to minimize neurological deficits. Deeper insight into AVM nature and into events that occur during AVM surgery as well as the inclusion of molecular biological approaches will open new horizons for the safe and effective treatment of AVMs.

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30

Zamorano, Lucia, Amgad Matter, Laurie Caspar, Arturo Saenz, Razvan Buciuc, James Fontanesi, Azucena Garzon, and Fernando Diaz. "Radiosurgery for Cerebral Arteriovenous Malformations." Journal of Radiosurgery 1, no. 3 (September 1998): 225–31. http://dx.doi.org/10.1023/b:jora.0000015198.05410.96.

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31

Fuwa, Isao, Yutaka Kai, and Hidetaka Wada. "Cerebral arteriovenous malformations in elderly." Nosotchu 12, no. 5 (1990): 501–3. http://dx.doi.org/10.3995/jstroke.12.501.

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32

Choudhri, Omar, and Steven Daniel Chang. "Orbital and Cerebral Arteriovenous Malformations." New England Journal of Medicine 365, no. 13 (September 29, 2011): e28. http://dx.doi.org/10.1056/nejmicm1010443.

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Kurokawa, Toru, Akinobu Matsuzaki, Kanehiro Hasuo, Masashi Fukui, Shigeru Tomita, Makoto Matsuo, Yung-Jung Chen, and Charnchai Kasemkosolsri. "Cerebral arteriovenous malformations in children." Brain and Development 7, no. 4 (January 1985): 408–13. http://dx.doi.org/10.1016/s0387-7604(85)80138-8.

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Dasenbrock, Hormuzdiyar H., and Rose Du. "Presentation of Cerebral Arteriovenous Malformations." World Neurosurgery 89 (May 2016): 694–96. http://dx.doi.org/10.1016/j.wneu.2015.11.078.

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35

Willinsky, R. A., P. Lasjaunias, K. Terbrugge, and P. Burrows. "Multiple cerebral arteriovenous malformations (AVMs)." Neuroradiology 32, no. 3 (1990): 207–10. http://dx.doi.org/10.1007/bf00589113.

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Willinsky, R., P. Lasjaunias, J. Comoy, and P. Pruvost. "Cerebral micro arteriovenous malformations (mAVMs)." Acta Neurochirurgica 91, no. 1-2 (March 1988): 37–41. http://dx.doi.org/10.1007/bf01400525.

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37

Di Rocco, C., G. Tamburrini, and M. Rollo. "Cerebral Arteriovenous Malformations in Children." Acta Neurochirurgica 142, no. 2 (February 14, 2000): 145–58. http://dx.doi.org/10.1007/s007010050017.

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KUWABARA, Satoshi, Tohru UOZUMI, Kazunori ARITA, Takashi YANO, Akihiko TAKECHI, Shinji OHBA, Kunyu HARADA, Kuniki EGUCHI, and Zainal MUTTAQIN. "Cerebral Hemodynamics in Arteriovenous Malformations." Surgery for Cerebral Stroke 21, no. 3 (1993): 177–83. http://dx.doi.org/10.2335/scs1987.21.3_177.

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NEMOTO, Shigeru, Shunsuke KAWAMOTO, Tomio SASAKI, Kintomo TAKAKURA, Naoshi YAMADA, Hideaki IWASA, Eiju WATANABE, Waro TAKI, Hiroo IWATA, and Akira TAKAHASHI. "Embolization of Cerebral Arteriovenous Malformations." Surgery for Cerebral Stroke 21, no. 6 (1993): 443–47. http://dx.doi.org/10.2335/scs1987.21.6_443.

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40

Luessenhop, Alfred J. "Cerebral Arteriovenous Malformations—Part I." Contemporary Neurosurgery 11, no. 14 (1989): 1–6. http://dx.doi.org/10.1097/00029679-198911140-00001.

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Luessenhop, Alfred J., and Louis Rosa. "Cerebral Arteriovenous Malformations—Part II." Contemporary Neurosurgery 11, no. 15 (1989): 1–6. http://dx.doi.org/10.1097/00029679-198911150-00001.

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42

Ham, H., P. E. Burrows, O. Flodmark, K. Terbrugge, and R. Humphreys. "Neonatal Superficial Cerebral Arteriovenous Malformations." Pediatric Neurosurgery 20, no. 2 (1994): 126–36. http://dx.doi.org/10.1159/000120775.

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43

de León, Fernando Chico-Ponce, Luis Felipe Gordillo-Domínguez, Eduardo Castro-Sierra, and Patrick Dhellemmes. "Cerebral Arteriovenous Malformations in Childhood." Contemporary Neurosurgery 31, no. 5 (March 2009): 1–5. http://dx.doi.org/10.1097/01.cne.0000348471.20436.6c.

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de León, Fernando Chico-Ponce, Luis Felipe Gordillo-Domínguez, Eduardo Castro-Sierra, and Patrick Dhellemmes. "Cerebral Arteriovenous Malformations in Childhood." Contemporary Neurosurgery 31, no. 6 (March 2009): 1–7. http://dx.doi.org/10.1097/01.cne.0000348473.66178.ca.

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de León, Fernando Chico-Ponce, Luis Felipe Gordillo-Domínguez, Eduardo Castro-Sierra, and Patrick Dhellemmes. "Cerebral Arteriovenous Malformations in Childhood." Contemporary Neurosurgery 31, no. 7 (April 2009): 2–5. http://dx.doi.org/10.1097/01.cne.0000348540.42424.4a.

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de León, Fernando Chico-Ponce, Luis Felipe Gordillo-Domínguez, Eduardo Castro-Sierra, and Patrick Dhellemmes. "Cerebral Arteriovenous Malformations in Childhood." Contemporary Neurosurgery 31, no. 8 (April 2009): 1–5. http://dx.doi.org/10.1097/01.cne.0000348542.50048.07.

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Millar, Carol, Bruno Bissonnette, and R. P. Humphreys. "Cerebral arteriovenous malformations in children." Canadian Journal of Anaesthesia 41, no. 4 (April 1994): 321–31. http://dx.doi.org/10.1007/bf03009913.

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Lasjaunias, Pierre, Francis Hui, Michel Zerah, Ricardo Garcia-Monaco, Veronique Malherbe, Georges Rodesch, Akio Tanaka, and Hortensia Alvarez. "Cerebral arteriovenous malformations in children." Child's Nervous System 11, no. 2 (February 1995): 66–79. http://dx.doi.org/10.1007/bf00303807.

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Sirakov, Stanimir S., Alexander Sirakov, Krasimir Minkin, Hristo Hristov, Kristian Ninov, Marin Penkov, Vasil Karakostov, et al. "Initial experience with precipitating hydrophobic injectable liquid in cerebral arteriovenous malformations." Interventional Neuroradiology 25, no. 1 (September 17, 2018): 58–65. http://dx.doi.org/10.1177/1591019918798808.

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Abstract:

Background Precipitating hydrophobic injectable liquid is a newly introduced liquid embolic agent for endovascular embolization with some technical advantages over other liquid embolic agents. We present our initial experience with precipitating hydrophobic injectable liquid in the endovascular treatment of cerebral arteriovenous malformations. Methods From October 2015 to January 2018, 27 patients harboring cerebral arteriovenous malformations underwent endovascular embolization with precipitating hydrophobic injectable liquid 25. Clinical features, angiographic results, procedural details, complications, and follow-up details were retrospectively analyzed. Results Twenty-seven patients with cerebral arteriovenous malformations were included. Total obliteration in one endovascular session was confirmed for 14/27 (52%) patients. Partial embolization was attained in 13 patients (48%) in whom staged treatment with following radiosurgery or surgery was planned. No mortality was recorded in this series. Complications during or after the embolization occurred in six of 27 (22.2%) patients. Conclusion In our initial experience, precipitating hydrophobic injectable liquid has acceptable clinical outcome comparable to other liquid embolic agents. Although this is the largest reported study in arteriovenous malformation treatment with precipitating hydrophobic injectable liquid, further studies are needed to validate its safety and efficacy.

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Shchehlov, D. V., O. E. Svyrydiuk, S. V. Chebanyuk, O. V. Slobodian, and M. B. Vyval. "Spontaneous occlusion of the cerebral arteriovenous malformations." Ukrainian Interventional Neuroradiology and Surgery 39, no. 1 (September 27, 2022): 34–39. http://dx.doi.org/10.26683/2786-4855-2022-1(39)-34-39.

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Spontaneous occlusion of arteriovenous malformations (AVM), characterized by complete disappearance of the AVM nidus and early venous drainage, and is a rare. The frequency of this phenomenon varied from 0.1 to 1.3 %.We analyzed two cases of spontaneous occlusion of cerebral AVMs with a non-hemorrhagic debut after 3 and 13 years in women aged 28 and 40 years, respectively. In both cases AVM was diagnosed during routine neuroimaging because of headache. Both patients had superficial small malformations. After discussing the risks of surgical treatment, both patients refused to perform any intervention. Scheduled angiography was performed 3 years after the diagnosis to follow-up the course of the disease and revealed complete disappearance of the AVM. Another patient noted persistent regression of headache after 3 years. Follow-up angiography was performed 13 years after diagnosis and confirmed spontaneous occlusion of the AVM. Given the data on the recurrence of the disease after spontaneous occlusion, such patients require long-term follow-up.When an AVM ruptures, hemodynamic changes may explain the thrombosis of the malformation, but the mechanisms of spontaneous occlusion in non-ruptured AVM remain unclear.

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