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Books on the topic 'Cerebral arteriovenous malformations – Treatment'

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Author: Grafiati

Published: 10 December 2022

Last updated: 29 January 2023

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1

Samson, Duke S., H. Hunt Batjer, and Philip E. Stieg. Intracranial arteriovenous malformations. New York: Informa Healthcare, 2007.

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2

Shokei, Yamada, ed. Arteriovenous malformations in functional areas of the brain. Armonk, NY: Futura Pub. Co., 1999.

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3

Yaşargil, Mahmut Gazi. AVM of the brain. Stuttgart: Thieme, 1987.

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4

J, Teddy P., Valavanis A, and Duvernoy Henri M, eds. AVM of the brain, histology, embryology, pathological considerations, hemodynamics, diagnostic studies, microsurgical anatomy. Stuttgart: Georg Thieme, 1987.

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5

1953-, Steiger Hans-Jakob, ed. Neurosurgery of arteriovenous malformations and fistulas: A multimodal approach. Wien: Springer, 2002.

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6

Cavernous malformations of the nervous system. Houndmills, Basingstoke, Hampshire: Cambridge University Press, 2011.

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7

Kathie's miracle. [Place of publication not identified]: Xlibris, 2010.

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8

Maciunas, Robert J. Endovascular neurological intervention. Park Ridge, Ill: American Association of Neurological Surgeons, 1995.

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9

Spinelli, Allison, and Liang Huang. Arteriovenous Malformation. Edited by David E. Traul and Irene P. Osborn. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190850036.003.0006.

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Arteriovenous malformation (AVM) is an abnormal connection between arteries and veins, bypassing the capillary system, which may occur in the brain or the spinal cord. AVMs are challenging to diagnose, and the symptoms may be subtle or dramatic. The location and extent of the lesion will determine its potential for morbidity as these lesions also affect a young population. Most importantly, the technology and evolution of treatment for AVMs has changed dramatically over the past two decades and now incorporates interventional neuroradiology and stereotactic radiation. Management options include conservative management, surgical resection, endovascular embolization, and stereotactic surgery. This chapter’s discussion will cover surgical and interventional modes of treatment of cerebral AVMs.

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10

Renfrow, Jaclyn J., Aqib H. Zehri, Kyle M. Fargen, Jasmeet Singh, John A. Wilson, and Stacey Q. Wolfe. Management of Intracranial Vascular Lesions During Pregnancy. Edited by Emma Ciafaloni, Cheryl Bushnell, and Loralei L. Thornburg. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190667351.003.0016.

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Management of cerebral vascular lesions in pregnancy requires special consideration to an altered natural history in the pregnant patient, such as a higher rupture rate of arteriovenous malformations. Additionally, treatment challenges exist including radiation exposure, medication selection, optimal treatment timing, and modalities. If identified prior to a pregnancy most vascular lesions warrant a definitive treatment discussion to circumvent the risks associated with an intracranial hemorrhage during pregnancy. The treatment team consists of a multidisciplinary approach involving neurosurgeons, anesthesiologists, neurointensivists, and obstetricians.

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11

Awad, Issam. Dural Arteriovenous Malformations. Not Avail, 1993.

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12

Batjer, H. Hunt, Philip E. Stieg, and Duke Samson. Intracranial Arteriovenous Malformations. Taylor & Francis Group, 2006.

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13

(Editor), Philip E. Stieg, H. Hunt Batjer (Editor), and Duke Samson (Editor), eds. Intracranial Arteriovenous Malformations. Informa Healthcare, 2006.

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14

Kevin Luk, K. H., and Deepak Sharma. Subarachnoid Hemorrhage. Edited by David E. Traul and Irene P. Osborn. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190850036.003.0024.

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Subarachnoid hemorrhage (SAH) is commonly caused by rupture of an intracranial aneurysm, arteriovenous malformation, or due to trauma. Prompt diagnosis and intervention are required to control intracranial pressure, maintain cerebral perfusion, and prevent rebleeding. Clinical grading of the bleed predicts morbidity and mortality, whereas imaging grading predicts risk of cerebral vasospasm. Hydrocephalus can occur as a result of SAH, which requires treatment with an external ventricular drain. Endovascular and open microsurgical procedures are available for securing the vascular abnormalities. Patients are typically monitored in a neurocritical care unit for up to 21 days post-bleed to monitor for the development of cerebral vasospasm/delayed cerebral ischemia (DCI). Mainstay of treatment for DCI includes induced hypertension, balloon angioplasty, and intraarterial vasodilator therapy. In addition, patient may experience significant derangement in their cardiac, pulmonary, and endocrine systems, requiring inotropic support, mechanical ventilation, or insulin infusion therapy.

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15

Yamada, Shokei. Arteriovenous Malformations in Functional Areas of the Brain. Wiley & Sons, Incorporated, John, 2008.

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16

Cavernous Malformations. American Association of Neurological Surgeons, 1992.

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17

Brain Arteriovenous Malformations: Pathogenesis, Epidemiology, Diagnosis, Treatment and Outcome. Springer, 2017.

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18

Beneš, Vladimír, and Ondřej Bradáč. Brain Arteriovenous Malformations: Pathogenesis, Epidemiology, Diagnosis, Treatment and Outcome. Springer, 2018.

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19

Schmid-Elsaesser, Robert, Alexander Muacevic, Hartmut Brückmann, and Berndt Wowra. Neurosurgery of Arteriovenous Malformations and Fistulas: A Multimodal Approach. Springer London, Limited, 2012.

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20

Parikh, Roshni A., and David M. Williams. Proximal Arterial Occlusion During Treatment of Pelvic High-Flow Arteriovenous Malformations. Edited by S. Lowell Kahn, Bulent Arslan, and Abdulrahman Masrani. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199986071.003.0029.

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Pelvic arteriovenous malformations (AVMs) are a cause of significant morbidity. Surgical management of AVMs poses a risk of massive intraoperative hemorrhage, surrounding organ injury, and incomplete removal of the nidus. Unfortunately, treatment is associated with high recurrence rates. Endovascular treatment is the preferred method of treatment; however, the high-flow nature of these lesions poses a challenge, risking nontarget treatment. It is important to provide adequate proximal arterial occlusion before injecting the sclerosant. This chapter outlines the steps involved in creating temporary stasis proximally within an arterial feeder to extend the contact time between the sclerosant and the recipient vessel wall while simultaneously accessing the arterial feeder more distally to deliver the sclerosant.

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21

J, Jafar Jafar, Awad Issam A, and Rosenwasser Robert H, eds. Vascular malformations of the central nervous system. Philadelphia: Lippincott Williams & Wilkins, 1999.

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22

(Editor), Jafar J. Jafar, Issam A. Awad (Editor), and Robert H. Rosenwasser (Editor), eds. Vascular Malformations of the Central Nervous System. Lippincott Williams & Wilkins, 1999.

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23

Pasqualin, A., and R. Da Pian. New Trends in Management of Cerebro-Vascular Malformations: Proceedings of the International Conference, Verona, Italy, June 8-12, 1992. Springer-Verlag Telos, 1994.

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24

1928-, Pasqualin A., Da Pian R. 1951-, Scienza R, and International Conference "New Trends in Management of Cerebro-vascular Malformations" (1992 : Verona, Italy), eds. New trends in management of cerebro-vascular malformations: Proceedings of the international conference, Verona, Italy, June 8-12, 1992. Wien: Springer-Verlag, 1994.

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25

Pellettieri, L., S. Grevsten, G. Norlen, C. Uhlemann, and C. A. Carlsson. Surgical Versus Conservative Treatment of Intracranial Arteriovenous Malformations: A Study in Surgical Decision-Making. Springer, 2012.

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26

Mehta, Nikhil, and Bulent Arslan. Techniques for Treating Visceral Aneurysms and High-Flow Arteriovenous Malformations of the Renal and Visceral Vasculature. Edited by S. Lowell Kahn, Bulent Arslan, and Abdulrahman Masrani. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199986071.003.0028.

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Techniques for treating visceral aneurysms are based on location and anatomic region and also on whether an aneurysm is a true aneurysm or a pseudoaneurysm. Visceral artery aneurysms typically require treatment if they are greater than 2 cm. Aneurysms that are favorable for endovascular therapy include saccular aneurysms preferably with a narrow neck and/or aneurysms that have good collateral blood flow to the target organ. Endovascular techniques for treating arteriovenous malformations (AVMs) are multifaceted and require appropriate identification of the AVM using multiple imaging modalities in addition to angiography. AVMs can be defined as slow flow, intermediate flow, and high flow.

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27

Maciunas, Robert J. Endovascular Neurological Intervention. American Association of Neurological Surgeons, 1995.

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28

Prout, Jeremy, Tanya Jones, and Daniel Martin. Neuroanaesthesia and neurocritical care. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199609956.003.0022.

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This chapter describes the general conduct of anaesthesia for neurosurgery with particular reference to techniques for reducing intracranial pressure, safe positioning, and recognition and management of air embolus. Management for specific common procedures such as shunt surgery, haematomas, traumatic brain injury and pituitary surgery is described. Neurosurgical conditions such as cerebral aneurysms and arteriovenous malformations may be managed in neuroradiology and the special considerations for the provision of anaesthesia for these cases are detailed. The principles of management of traumatic brain injury in critical care which aim to reduce secondary brain injury are explained.

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29

Chong, Ji Y., and Michael P. Lerario. An Unusual Hemorrhage. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190495541.003.0032.

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A young patient without hypertension should have an evaluation for underlying cause of intracerebral hemorrhage. Arteriovenous malformations may cause intracerebral hemorrhage. Treatment can be challenging, and multimodal treatment is often required. The options include surgery, endovascular embolization, and radiotherapy.

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30

Harley, Kim, and Sue Jones. Neurological and spinal surgery. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642663.003.0023.

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Neurological assessment is performed on admission for surgery, as a routine part of medical examination. This is done to diagnose whether symptoms of illness in the patient are due to neurological conditions and, if so, where in the nervous system the pathological lesions are located. Hydrocephalus is either an acute or chronic condition whereby the cerebrospinal fluid pressure rises, causing symptoms of raised intracranial pressure. Patients at risk of raised intracranial pressure should be nursed by staff trained and experienced in neurological assessment using the Glasgow coma scale. This chapter looks at neurological assessment, raised intracranial pressure, head injuries, and brain and spinal tumours. This chapter also discusses the management of subarachnoid haemorrhage, cerebral aneurysm, arteriovenous malformations, and epilepsy. Finally, the chapter provides an overview of degenerative diseases of the spine and peripheral nerve injury.

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31

Karen, Bellenir, ed. Congenital disorders sourcebook: Basic information about disorders acquired during gestation, including spina bifida, hydrocephalus, cerebral palsy, heart defects, craniofacial abnormalities, fetal alcohol syndrome, and more, along with current treatment options and statistical data. Detroit, MI: Omnigraphics, 1997.

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32

Laureno, Robert. Foundations for Clinical Neurology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190607166.001.0001.

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Part 1 of this book deals with the practice of neurology. It comments on neurologic history-taking, including observations of patient gestures. It also discusses the neurologic examination and its quantification. Magnetic resonance and computerized tomographic brain imaging are compared and contrasted. The importance of gravity, timing of scans, and plane of section of scans are emphasized. Neurologic diagnosis and its pitfalls are discussed. Neurologic treatment, empirical approaches to therapy, and the provision of hope and reassurance are emphasized. In Part 2, symmetry in neurologic disease as a clue to metabolic cause is a major topic. Selective vulnerability of brain areas to trauma, anoxia, toxins, and other diseases are discussed. Diseases of rapid normalization including myelinolysis are described. Carotid stenosis, berry aneurysm, arteriovenous malformations and other examples of asymptomatic disease comprise a final chapter in this section. Part 3 comments on special topics in neurology. Major attention is given to neurologic terminology, its origins, evolution, eponyms, and politics. The rise, fall, and evolution of neurologic concepts are described. Consideration is given to classifications and causation in neurology. The phenomena of decussation and asymmetry in neurology are discussed.

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