Relevant bibliographies by topics / Cerebral arteriovenous malformations

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Academic literature on the topic 'Cerebral arteriovenous malformations'

Author: Grafiati
Published: 4 June 2021
Last updated: 23 February 2023

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Journal articles on the topic "Cerebral arteriovenous malformations"

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Janicijevic, Milos. "Surgery of the arteriovenous cerebral malformations." Acta chirurgica Iugoslavica 55, no. 2 (2008): 11–16. http://dx.doi.org/10.2298/aci0802011j.

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According to the present technical possibilities modalities of the treatment of the arteriovenous cerebral malformations (surgery, embolization, radiosurgery) for direct neurosurgical excision of malformations remains of cases. Decision to operate is made separately for each malformation and is based on anticipation of the natural course of the illness, precise estimate of the risk from operation and on the estimate of the condition of the patient. Surgical technique is also chosen for each malformation separately, depending on its size, angioarchitecture, hemodynamic characteristics and localization. The following techniques are used: resection of the malformation "en blocque", coagulation of the lesion in situ, gradual marginal coagulation in the lesion cleavage, "backword technique", perivenous approach and excision of the complex malformations in several acts. The safety of the surgical excision is increased by preoperative and perioperative actions already described. Intraoperative problems and postoperative complications (brain edema, uncontrolled hemorrhage, intracerebral hematoma and others) were discussed.
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Aguiar, Paulo Henrique, Marco Antonio Stefani, Gustavo Rassier Isolan, Carlos Alexandre Zicarelli, and Apio Claudio Martins Antunes. "Cerebral Arteriovenous Malformations." JBNC - JORNAL BRASILEIRO DE NEUROCIRURGIA 23, no. 4 (March 29, 2018): 301–15. http://dx.doi.org/10.22290/jbnc.v23i4.1215.

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A significant improvement of central nervous system arteriovenous vascular malformations (AVM) outcome has been observed due to the advances in all modalities of diagnosis and treatment. The authors report the advances in diagnosis and integrated global treatment of AVM`s.
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Millichap, J. Gordon. "Cerebral Arteriovenous Malformations." Pediatric Neurology Briefs 8, no. 9 (September 1, 1994): 70. http://dx.doi.org/10.15844/pedneurbriefs-8-9-10.

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Venes, Joan. "CEREBRAL ARTERIOVENOUS MALFORMATIONS." Neurosurgery 30, no. 4 (April 1, 1992): 652. http://dx.doi.org/10.1097/00006123-199204000-00036.

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H??llerhage, H.-G. "Cerebral Arteriovenous Malformations." Neurosurgery 31, no. 3 (September 1992): 603???604. http://dx.doi.org/10.1097/00006123-199209000-00030.

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Tamaki, Norihiko, and Kazumasa Ehara. "Cerebral Arteriovenous Malformations." Neurosurgery 31, no. 3 (September 1992): 605. http://dx.doi.org/10.1097/00006123-199209000-00031.

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Warren, Daniel James, Nigel Hoggard, Lee Walton, Matthias Walter Richard Radatz, Andras A. Kemeny, David Martin Campbell Forster, Iain David Wilkinson, and Paul David Griffiths. "CEREBRAL ARTERIOVENOUS MALFORMATIONS." Neurosurgery 61, suppl_1 (July 1, 2007): 973–83. http://dx.doi.org/10.1227/01.neu.0000279215.07763.a1.

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Farhat, Hamad I. "Cerebral Arteriovenous Malformations." Disease-a-Month 57, no. 10 (October 2011): 625–37. http://dx.doi.org/10.1016/j.disamonth.2011.08.021.

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Venes, Joan. "CEREBRAL ARTERIOVENOUS MALFORMATIONS." Neurosurgery 30, no. 4 (April 1992): 652. http://dx.doi.org/10.1227/00006123-199204000-00036.

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Höllerhage, H.-G. "Cerebral Arteriovenous Malformations." Neurosurgery 31, no. 3 (September 1, 1992): 603–4. http://dx.doi.org/10.1227/00006123-199209000-00030.

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Dissertations / Theses on the topic "Cerebral arteriovenous malformations"

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Grieve, Joan Patricia. "Novel structural and functional imaging in cerebral arteriovenous malformations." Thesis, University College London (University of London), 2004. http://discovery.ucl.ac.uk/1446575/.

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Clarification of the angio-architecture and haemodynamic effects of cerebral arteriovenous malformations (AVMs) using non-invasive imaging may advance our knowledge and understanding of their natural history, resulting in improvements in the management of patients with these lesions. The aims of the work in this dissertation was to investigate the haemodynamic effects of AVMs and to determine whether newer non-invasive imaging techniques allow an accurate enough assessment of the angio-architecture of AVMs to be able to replace conventional digital subtraction angiography (DSA) in some clinical situations. The hypotheses were: 1. Non-invasive structural imaging techniques, such as CT angiography (CTA) and MR angiography (MRA), provide adequate structural and volumetric information to replace the more invasive technique of DSA.2. MR perfusion imaging is able to demonstrate the alteration of cerebral haemodynamics by AVMs. 6 Rapid frame rate DSA (RFRDSA) provides useful quantitative data on the blood flow within cerebral AVMs. In this work CT and MR angiography (CTA and MRA) were compared with conventional digital subtraction angiography (DSA). Twenty patients were examined with CTA and ten with MRA. Both techniques were able to detect most of the important angio-architectural features, but were not as accurate as DSA as decision-making tools, in particular because temporal resolution and nidus definition were poor. The use of gadolinium enhancement during MRA improved the visualisation of both nidus and draining veins. The nidal volume of ten AVMs was calculated from DSA and three magnetic resonance (MR) sequences. With biplanar DSA, an ellipsoid volume was calculated using orthogonal projections. MR images showed potential but were difficult to interpret due to varied appearances of flowing blood and, on gadolinium-enhanced MRA, the enhancement of abnormal brain. The cerebral haemodynamics of fifteen patients with AVMs were examined with contrast bolus tracking. This semi-quantitative technique was able to demonstrate consistent differences in cerebral blood flow and volume, mean transit time and time to minimum signal intensity in brain distant from the AVM. Changes in the perinidal regions were dominated by the presence of draining veins. A vascular phantom was calibrated to allow calculation of flow rates from rapid frame rate DSA. The technique for quantifying flow was assessed in five patients and compared with values measured by transcranial doppler. It was not possible to calculate accurately flow and velocity for AVM feeding vessels. These imaging modalities allowed improved appreciation of the structure and haemodynamic effects of cerebral AVMs but further development is needed before they will be of use as reliable clinical tools.
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Storer, Kingsley Paul School of Medicine UNSW. "Cerebral arteriovenous malformations: molecular biology and enhancement of radiosurgical treatment." Awarded by:University of New South Wales. School of Medicine, 2006. http://handle.unsw.edu.au/1959.4/31942.

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Object Rupture of intracranial arteriovenous malformations is a leading cause of stroke in children and young adults. Treatment options include surgery and highly focused radiation (stereotactic radiosurgery). For large and deep seated lesions, the risks of surgery may be prohibitively high, while radiosurgery has a disappointingly low efficacy and long latency. Radiosurgery carries the most promise for significant advances, however the process by which radiosurgery achieves obliteration is incompletely understood. Inflammation and thrombosis are likely to be important in the radiation response and may be amenable to pharmacological manipulation to improve radiosurgical efficacy. Materials and methods Immunohistochemistry and electron microscopy were used to study normal cerebral vessels, cavernous malformations and AVMs, some of which had previously been irradiated. An attempt was made to culture AVM endothelial cells to study the immediate response of AVM endothelium to radiosurgery. The effects of radiosurgery in a rat model of AVM were studied using immunohistochemistry and the results used to determine the choice of a pharmacological strategy to enhance the thrombotic effects of radiosurgery. Results Vascular malformations have a different endothelial inflammatory phenotype than normal cerebral vessels. Radiosurgery may cause long term changes in inflammatory molecule expression and leads to endothelial loss with exposure of pro-thrombotic molecules. Ultrastructural effects of irradiation include widespread cell loss, smooth muscle cell (SMC) proliferation and thrombosis. Endothelial culture from AVMs proved difficult due to SMC predominance in initial cultures. Radiosurgery upregulated several endothelial inflammatory molecules in the animal model and may induce pro-thrombotic cell membrane alterations. The administration of lipopolysaccharide and soluble tissue factor to rats following radiosurgery led to selective thrombosis of irradiated vessels. Conclusions Inflammation and thrombosis are important in the radiosurgical response of AVMs. Lumen obliteration appears to be mediated by proliferation of cells within the vessel wall and thrombosis. Upregulation of inflammatory molecules and perhaps disruption of the normal phospholipid asymmetry of the endothelial and SMC membranes are some of the earliest responses to radiosurgery. The alterations induced by radiation may be harnessed to selectively initiate thrombus formation. Stimulation of thrombosis may improve the efficacy of radiosurgery, increasing treatable lesion size and reducing latency.
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Söderman, Michael. "Volume determination and predictive models in the management of cerebral arteriovenous malformations /." Stockholm, 2000. http://diss.kib.ki.se/2000/91-628-4136-X/.

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Verlaan, Dominique Jacqueline. "Genetic investigation of cerebrovascular disorders : cerebral cavernous malformations and intracranial aneurysms." Thesis, McGill University, 2007. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=103306.

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Cerebral Cavernous Malformations (CCM) and Intracranial Aneurysms (IA) are cerebrovascular disorders that can lead to a hemorrhagic stroke and other neurological problems. CCMs are characterized by abnormally enlarged capillary cavities while IAs are saccular outpouchings of intracranial arteries. CCM is found in approximately 0.4% to 0.9% of the population, while IA is more common (3-6%).
This dissertation aimed to add to the body of research for CCM and IA and was divided into two parts. Initial work focused on the characterization and identification of the genes involved in CCM; the second phase focused on the identification of a susceptibility gene for IA.
In the first phase, the CCM1, CCM2 and CCM3 genes were characterized in families and in sporadic cases of CCM. In both cohorts, a causative mutation was identified in 71% of the cases. Subsequent MLPA analysis of subjects with no CCM mutations revealed that large genomic deletions and duplications are a common cause of CCM. In addition, investigation of CCM1 point mutations revealed that these were not simple missense mutations but that they rather activated cryptic splice-donor sites and caused aberrant splicing. Furthermore, the genetic predisposition to CCM in sporadic cases with a single lesion was determined to be different from sporadic cases with multiple malformations. Investigation into the loss of heterozygosity demonstrated a plausible mechanism for CCM pathogenesis involving a second somatic hit at the site of the lesion, suggesting that CCM may be caused by a complete loss of CCM protein function.
In the second phase, a genome-wide scan of a large family and subsequent linkage analysis using a monogenic approach identified a susceptibility locus for IA (ANIB4).
As a result of this research, we have greatly contributed to the field of CCM, most specifically to its clinical diagnosis. A greater understanding of the genetics involved in CCM will facilitate and permit better management care for patients. Furthermore, the possibility of identification of a gene with a major effect for IA will give us more insight into which pathways are involved in IA formation.
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Lindvall, Peter. "Hypofractionated conformal stereotactic radiotherapy in the treatment of AVMs and cerebral metastases." Doctoral thesis, Umeå : Univ, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-864.

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Souza, Evandro César de. "Resultados do tratamento radiocirúrgico de doentes com malformações arteriovenosas encefálicas classificadas como graus 3A, 3B, 4 ou 5 previamente submetidos ou não à embolização." Universidade de São Paulo, 2010. http://www.teses.usp.br/teses/disponiveis/5/5138/tde-20092010-120333/.

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Introdução: O risco do tratamento das malformações arteriovenosas encefálicas (MAVEs) é proporcional à sua graduação. O processo de seleção da técnica empregada depende, além da natureza e localização das MAVEs, das condições clínicas e idade dos doentes e da disponibilidade de profissionais treinados e de equipamentos apropriados no ambiente onde o doente é tratado. Objetivo: Avaliar a eficácia da radiocirurgia e da embolização prévia ao tratamento radiocirúrgico das MAVEs classificadas como graus 3A, 3B, 4 ou 5. Métodos e Resultados: O trabalho baseou-se na observação retrospectiva de prontuários clínicos e de arquivos de imagens de 90 doentes com diagnóstico de MAVEs classificadas como graus 3A, 3B, 4 ou 5 submetidos a tratamento com radiocirurgia precedida ou não de embolização no Departamento de Radioterapia e Radioterapia Estereotáctica, Neuroradiologia Intervencionista e Neurocirugia da Real e Benemérita Associação Portuguesa de Beneficência de São Paulo, de outubro de 1993 a outubro de 2008. As idades dos doentes variaram de sete a 60 anos (média de 30,6 anos ± 11,59 anos, mediana de 28 anos). Eram do sexo feminino 46 (51,1%) doentes. Todos foram submetidos a três exames de ressonância magnética (RM) ou tomografia computadorizada (TC) no primeiro ano de acompanhamento e a um exame de RM ou TC do encéfalo no segundo e terceiro anos. Quando um dos exames sugeriu oclusão da MAVE ou o período de acompanhamento foi maior que três anos, os doentes foram submetidos à angiografia digital do encéfalo. Em 51 (56,7%) dos 90 doentes tratados ocorreu oclusão completa da MAVE após a primeira radiocirurgia (1ª Rc), em 21 (23,3%) foram evidenciadas complicações clínica em 36 (40,0%), foram visibilizadas anormalidades no exame de RM. Em 30 (33,3%) doentes evidenciou-se hipersinal no encéfalo em T2 na RM xvi e, em seis (6,7%), radionecrose. Trinta e dois(82,0%) dos 39 doentes que não apresentaram oclusão completa da MAVE após a 1ª Rc foram submetidos à segunda radiocirurgia (2ª Rc). Ocorreu oclusão completa em 12 (37,5%) destes doentes, instalaram-se complicações clínicas em cinco (15,6%) e, anormalidades no exame de RM em oito (25,0%), ou seja, hipersinal em T2 na RM em três (9,5%) e radionecrose em cinco (15,6%). Conclusão: Concluiu-se que a radiocirurgia foi eficaz no tratamento das MAVEs graus 3A, 3B, 4 e 5, que as MAVEs classificadas como grau 3B devem tratadas, preferencialmente, apenas com radiocirurgia e que as MAVEs classificadas como 3A, 4 e 5 devem ser submetidas à embolização previamente à radiocirugia para reduzir-se sua graduação e seu fluxo sanguíneo
Introduction: The risk of treatment of the cerebral arteriovenous malformations (AVM) is proportional to their grade and is affected by the method used. The selection of the AVM therapeutic method depends of the vascular pattern and anatomical site, clinical condition and age of the patient, experience of the treating team and of the equipment available. Objectives: This study aimed the evaluation of the efficacy of the radiosurgical treatment of Grade 3A, 3B, 4 and 5 cerebral AVMs in patients previosly treated or not with embolization. Methods and Results: The data of the clinical notes and the computed tomography (CT) and magnetic ressonance (RM) images of the brain of 90 patients with Grade 3A, 3B, 4 or 5 cerebral embolised or not AVMs treated with radiosurgery at the Department of Radiotherapy, Stereotactic Radiotherapy, Interventional Neuroradiology and Neurosurgery at Real e Benemerita Associação Portuguesa de Beneficência de São Paulo were retrospectively reviewed. The ages of the patients ranged from 7 to 60 years of (average = 30.6 ± 11.59 years; median = 28 years) and 46 (51.1%) were female. During the first year after treatment three MR or CT scans of the brain were evaluated and one at the end of the 2nd and 3rd years after the treatment one MR or CT scan were re-evaluated respectively. When the brain scans suggest AVM occlusion, cerebral angiography was performed. In 51 of the 90 patients (56.7%) there was complete occlusion of the AVM after one radiosurgical treatment; 21 of the patients (23.3%) had clinical complications. In 36 patients (40.0%) new abnormalities of the MR became evident. Thirty (33.3%) presented T2 hypersignal and six (6.7%), had radionecrosis. From the 39 patients who did not had complete occlusion of the AVM, 32 (82.0%) had a second radiosurgical course of treatment; in 12 xviii (37.5%) the AVM became completely occluded but five (15.6%) had clinical complications. Eight (25.0%) of these patients presented new abnormalities at the MR scan of the brain; three (9.5%) had T2 hypersignal and five (15.6%) radionecrosis. Conclusions: Radiosurgery was effective in the treatment of Grade 3A, 3B, 4 and 5 cerebral AVMs , Grade 3B cerebral AVM should be treated only with radiosurgery without previous embolization, and Grade 3A, 4 and 5 AVMs should be treated with embolization to reduce their AVM grade and blood flow prior to radiosurgery
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Torné, Torné Ramon. "Indicadores clínico-radiológicos de valor pronóstico en la patología vascular cerebral." Doctoral thesis, Universitat Autònoma de Barcelona, 2015. http://hdl.handle.net/10803/314189.

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El ictus cerebral hemorrágico es una causa frecuente de mortalidad y gran discapacidad funcional en nuestra población. Profundizar en el conocimiento de la evolución clínica de estos pacientes permite ofrecer expectativas reales a los familiares y una correcta optimización de los recursos sanitarios. Los indicadores con valor pronóstico en la fase aguda deben ser utilizados en la toma de decisiones en el tratamiento de estos pacientes. Esta tesis por compendio de artículos, pretende identificar diversos indicadores clínicos y neuroradiológicos con valor pronóstico que se encuentran presentes en la patología vascular cerebral con el objetivo de poder predecir con mayor precisión la evolución clínica de estos pacientes. Nuestro trabajo se centra en las dos patologías más habituales que trata el neurocirujano vascular: la hemorragia subaracnoidea aneurismática y las malformaciones arteriovenosas cerebrales. El compendio se fundamenta en cuatro trabajos publicados; tres de ellos realizados en el “Hospital Vall d’Hebron” y el cuarto en la “University of California SFO”. En referencia a la hemorragia subaracnoidea evaluamos aquellas variables clínicas y neuroradiológicas que nos pueden ayudar a predecir el pronóstico funcional en una enfermedad reumatológica (lupus eritematoso sistémico) y en el estudio de la evolución clínica de aquellos pacientes que concomitantemente presentaron un hematoma subdural. En referencia a las malformaciones arteriovenosas, nos centramos en determinar si la rotura intraoperatoria de una lesión vascular influye sobre el pronóstico funcional y cuales son los indicadores que predisponen a esta complicación. También evaluamos la evolución clínica de aquellos pacientes que presentaron un subtipo poco frecuente de malformación arteriovenosa como son las localizadas en la fosa posterior. Los resultados de nuestro trabajo describen tres patrones clínicos en relación con el lupus eritematoso sistémico previamente no descritos en la literatura; así como una peor evolución clínica en estos pacientes. Por lo contario, la presencia de un predictor radiológico como es un hematoma subdural en pacientes en coma no parece estar en relación con una peor evolución clínica. Si que presentan una peor estado funcional aquellos pacientes que padecieron una rotura intraoperatoria de una malformación arteriovenosa; describimos las tres causas más frecuentes e intentamos identificar la presencia de indicadores que aumenten el riesgo de evolucionar a dicha complicación. Por último, el estudio de las malformaciones localizadas en la fosa posterior nos aporta información sobre la mayor probabilidad de sangrado e hidrocefalia de estas lesiones; en relación con el pronóstico , se observa una buena evolución clínica de los supervivientes a pesar de presentar una elevada mortalidad inicial.
Brain haemorrhagic stroke is a common cause of mortality and major functional disability in our population. Deepening our understanding of clinical course of these patients lets us offer realistic expectations to their families and a proper optimization of health resources. Furthermore, by knowing prognostic value indicators during the acute phase we are able to improve our treatment decision-making. In order to predict the outcome of these patients, this thesis by compendium of articles sets out to identify several clinical and neuroradiological prognostic variables that could be defined in cerebrovascular pathology. Our work is focused on the two most common diseases in which vascular neurosurgeons are involved: aneurysmal subarachnoid haemorrhage and cerebral arteriovenous malformations (AVM). This compendium is based on four published papers, three of which were written from a neurosurgical twenty-years database at Vall d'Hebron Hospital and the fourth in collaboration with the University of California SFO. Regarding subarachnoid haemorrhage, we considered several clinical and neuroradiological variables that could help us to predict functional outcome in a rheumatological disease such as systemic lupus erythematous. Secondly, we investigated outcomes of patients who concomitantly had a subdural hematoma. Moreover, our study into AVMs was focused on whether AVMs intraoperative rupture could be related to a negative final outcome. We also analysed variables that predispose patients to this complication. In the last article we evaluated the clinical course of patients who showed an unusual subtype of arteriovenous malformation such as those located anatomically in the cerebral posterior fossa. Results of our work describe three new clinical patterns in systemic lupus erythematous not previously reported in the literature. In addition, these patients show a worse clinical outcome compared with simple subarachnoid haemorrhage. Furthermore, the presence of a radiological predictor as a subdural hematoma in coma patients does not seem to correlate to a negative functional outcome. AVMs Intraoperative rupture negatively impacts patient’s prognosis; we claim three common causes for this severe complication. Finally, our reviews of malformations anatomically located in posterior fossa strongly confirm a higher risk of bleeding and hydrocephalus. We find that unless the high first day’s mortality ratio in posterior fossa AVMs, survivors achieve a reasonably good long-term outcome.
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Peres, Carlos Michel Albuquerque. "Malformações arteriovenosas encefálicas: impacto da angioarquitetura nidal no resultado do tratamento radiocirúrgico isolado ou precedido de embolização." Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/5/5151/tde-16112017-110424/.

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Aspectos morfológicos do nido e embolização parcial neoadjuvante sem intenção de cura de malformações arteriovenosas encefálicas, precedendo a radiocirurgia, podem ter influência no resultado final do tratamento. Métodos: série consecutiva de 47 pacientes submetidos à radiocirurgia (1 a 5 sessões), precedida ou não por embolização com cianoacrilato. Acompanhamento clínico e radiológico mínimo de 36 meses. Resultados: a apresentação hemorrágica ocorreu em 68,1% dos pacientes tratados; destes, 62,5% portavam fístula arteriovenosa dentro da malformação arteriovenosa; 83,3% ectasia venosa e 90% restrição à drenagem venosa. A taxa de oclusão de embolização seguida de radiocirurgia foi de 46,1% e da radiocirurgia isolada foi de 52,4% (p=0,671). Foram identificados como fatores favoráveis à oclusão: baixo volume nidal, ausência de fístula arteriovenosa intranidal, maior dose de radiação e baixo grau na classificação das malformações arteriovenosas encefálicas baseadas na radiocirurgia (RBAS). Conclusões: o menor volume nidal (p < 0,001), o menor grau na escala RBAS (p=0,047), a ausência de fístula arteriovenosa intranidal (p=0,001) e a maior dose prescrita (p=0,001) tiveram correlação com resultado favorável no tratamento. Embolização seguida de radiocirurgia não foi superior à radiocirurgia isolada (p=0,772). A eliminação de fístulas arteriovenosas intranidais pela embolização pode aumentar a eficácia da radiocirurgia
Partial nidal embolization preceding radiosurgery of brain arteriovenous malformations (AVM) and some morphological nidal features may be related to final results. Methods: Analysis of a longitudinal cohort of 47 consecutive patients who underwent radiosurgery preceded or not by embolization. Embolizations were performed exclusively with n-butyl cyanoacrylate. Radiosurgery was delivered either as a single or divided up to 5 equal fractions. Clinical and radiological follow up of at least 36 months was obtained. Results: Hemorrhagic presentation was seen in 68.1% of the cases; 62.5% harbored intranidal arteriovenous fistulas (AVF), 83.3% had venous ectasias and 90% had venous outflow stenosis. The occlusion rate of embolization plus radiosurgery was 46.1% and radiosurgery alone was 52.4% (p = 0.671). Variables significantly associated with obliteration were lower nidus volume, lack of intranidal arteriovenous fistula, higher radiosurgical dose and lower grades in radiosurgical-based AVM scale (RBAS). Conclusions: a small nidus (p < 0.001), a lower RBAS grade (p = 0.047), no intranidal AVF (p = 0.001) and greater radiosurgical dose (p = 0.001) were associated to better results. Embolization followed by radiosurgery was not superior to radiosurgery alone (p = 0.772). Endovascular elimination of intranidal AVF\'s may help to promote radiosurgical occlusion
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"Middle cerebral artery (MCA) stenosis: genetic, pathological and imaging characterization." Thesis, 2006. http://library.cuhk.edu.hk/record=b6074252.

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According to the literature, MRI has been applied to characterize the atherosclerosis in coronary and extracranial internal carotid artery. The cross-sections of MCA were scanned by Magnetic Resonance Imaging (MRI) to assess the accuracy of MRI in identifying MCA stenosis with histopathology as a golden standard, which was performed in the same post-mortem brains as in the second part. The sensitivity and specificity of MRI in detecting more than 30% MCA stenosis were 38.6% and 92.2%, with a positive predictive value of 87.2% and negative predictive value of 52.2%, and the corresponding values of MRI in identifying more than 50% MCA stenosis were 57.1%, 90.8%, 50% and 83.0%, respectively. Stenotic lesions >30% and >50% identified by MRI were found to be associated with infarctions in corresponding MCA territory.
After verification of potential relationship between ischemic stroke and intracranial artery calcification, the incidence of intracranial artery calcification was assessed in the ischemic stroke. One hundred and seventy-five ischemic stroke patients and 182 controls were enrolled. There was a higher prevalence of intracranial artery calcification in ischemic stroke patients than in controls. Hypertension, diabetes, smoking, intracranial artery calcification, hyperlipidemia, and atrial fibrillation were found to be independently associated with ischemic stroke.
Atherosclerotic stenosis is a heterogeneous disorder. The studies performed in extracranial carotid artery and coronary artery showed that the genes associated with lipoprotein metabolism may be associated with atherosclerosis. Thus, we speculated that the genes concerned with lipid metabolism may also be risk factors for MCA atherosclerotic stenosis. In the part of genetic analysis, clinical parameters and the genotypes of polymorphisms in the apolipoprotein E (ApoE), lipoprotein lipase (LPL), and paraoxonase (PON1) genes were compared in patients with and without MCA stenosis. Two hundred and ninety-four ischemic stroke patients were recruited, 136 cases with and 158 without MCA stenosis. Systolic blood pressure (SBP), rather than ApoE, LPL, and PON1 polymorphism was found to be a risk factor of MCA stenosis.
Calcification of intracranial artery, as a common complication of atherosclerosis, was investigated by multi-detector-row computed tomography (MDCT). By this advanced technique, the prevalence and location of calcification in intracranial arteries were determined, and its potential risk factors were also investigated. Four hundred and ninety patients were recruited. The incidence of intracranial artery calcification was 69.4%. The highest prevalence of intracranial artery calcification was seen in internal carotid artery (60%), followed by vertebral artery (20%), middle cerebral artery (5%) and basilar artery (5%). Age, a history of ischemic stroke, and white blood cell count were shown to be independently associated with intracranial artery calcification.
In the present study, genetic, pathological, imaging characterizations and prognosis of MCA stenosis were investigated. The effect of candidate genes has not been confirmed in the present study, but SBP and hypertension appears to contribute a lot to the occurrence of MCA stenosis among Chinese populations. As for the pathology of MCA atherosclerotic plaques, luminal stenosis and also the morphology of atherosclerotic plaque seem to play a cooperative role in leading to ischemic stroke. Imaging studies demonstrated the agreement between ex vivo MRI and histopathology in identifying MCA stenosis, and the correlation between the MCA stenosis identified by MRI and ischemic events. Calcification of intracranial artery, as a common complication of atherosclerosis, may be associated with age, history of ischemic stroke. High incidence of ischemic stroke has been demonstrated in Chinese type 2 diabetes patients. The presence of asymptomatic MCA stenosis plays an important role in the occurrence of ischemic stroke.
Lastly, using a cohort-study, we aimed to investigate stroke incidence of asymptomatic MCA stenosis and its risk factors in Chinese type II diabetic population. Transcranial Doppler was performed to define MCA stenosis. Incident strokes between 1996 and 2006 were ascertained by the database of Clinical Management System of the Hong Kong Hospital Authority. Anthropometric parameters (waist circumference and body mass index), blood pressure, and baseline plasma biochemical profile (lipid and glucose) were recorded to find the risk factors of ischemic stroke in asymptomatic MCA stenosis patients. Totally, 2,197 type II diabetic patients without symptoms of cerebrovascular disease were recruited. The evidence of MCA stenosis was identified in 272 subjects (12.4%), including 146 (53.7%) subjects with single-vessel involvement. Ischemic stroke occurred in one hundred and eighty-four (8.4%, 184/2197) patients. History of ischemic heart disease, MCA stenosis, the presence of retinopathy, lipid total cholesterol and age were independently associated with ischemic stroke.
Secondly, the pathological features of MCA stenosis and their relationship with cerebral infarcts were investigated in a series of post-mortem adults aged 45 years or above. The morphological features of the MCA atherosclerotic plaques were described in detail. The results demonstrated that the degree of luminal stenosis, the percentage of the plaques containing more than 40% lipid area, the values of cap-lipid, cap-lipid-stenosis, and the prevalence of intraplaque hemorrhage, neovasculature and thrombus were higher in the group of plaques associated with infarction. And the mean index of both CD45RO and CD68 were higher in the group of plaques associated with infarction. Binary logistic regression showed that stenosis, lipid area and presence of neovasculature were independent risk factors of MCA infarcts.
Stroke is one of the leading causes of death, disability, and dementia throughout the world. The stenosis of the intracranial large artery, especially the middle cerebral artery (MCA), is common in Chinese, Hispanic, and African populations. But MCA stenosis has been understudied due to its infrequency in the white population and its relative inaccessibility and invasiveness involved in its investigations. The purpose of the study was to investigate the genetic, pathological, imaging characteristics and prognosis of MCA atherosclerotic stenosis in Chinese population.
Chen Xiang-yan.
"September 2006."
Adviser: MH Ng.
Source: Dissertation Abstracts International, Volume: 68-03, Section: B, page: 1460.
Thesis (Ph.D.)--Chinese University of Hong Kong, 2006.
Includes bibliographical references (p. 193-212).
Electronic reproduction. Hong Kong : Chinese University of Hong Kong, [2012] System requirements: Adobe Acrobat Reader. Available via World Wide Web.
Electronic reproduction. [Ann Arbor, MI] : ProQuest Information and Learning, [200-] System requirements: Adobe Acrobat Reader. Available via World Wide Web.
Abstracts in English and Chinese.
School code: 1307.
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Lin, Yue-Hsin, and 林岳欣. "Machine Learning Application in Classification of Cerebral Arteriovenous Malformation Hemorrhage." Thesis, 2019. http://ndltd.ncl.edu.tw/handle/s4f35q.

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Books on the topic "Cerebral arteriovenous malformations"

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Samson, Duke S., H. Hunt Batjer, and Philip E. Stieg. Intracranial arteriovenous malformations. New York: Informa Healthcare, 2007.

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Shokei, Yamada, ed. Arteriovenous malformations in functional areas of the brain. Armonk, NY: Futura Pub. Co., 1999.

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Yaşargil, Mahmut Gazi. AVM of the brain. Stuttgart: Thieme, 1987.

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J, Teddy P., Valavanis A, and Duvernoy Henri M, eds. AVM of the brain, histology, embryology, pathological considerations, hemodynamics, diagnostic studies, microsurgical anatomy. Stuttgart: Georg Thieme, 1987.

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1953-, Steiger Hans-Jakob, ed. Neurosurgery of arteriovenous malformations and fistulas: A multimodal approach. Wien: Springer, 2002.

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Cavernous malformations of the nervous system. Houndmills, Basingstoke, Hampshire: Cambridge University Press, 2011.

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Kathie's miracle. [Place of publication not identified]: Xlibris, 2010.

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Maciunas, Robert J. Endovascular neurological intervention. Park Ridge, Ill: American Association of Neurological Surgeons, 1995.

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Awad, Issam. Dural Arteriovenous Malformations. Not Avail, 1993.

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Batjer, H. Hunt, Philip E. Stieg, and Duke Samson. Intracranial Arteriovenous Malformations. Taylor & Francis Group, 2006.

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Book chapters on the topic "Cerebral arteriovenous malformations"

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Sellar, Robert J. "Cerebral Arteriovenous Malformations." In Endovascular Neurosurgery, 73–96. London: Springer London, 2000. http://dx.doi.org/10.1007/978-1-4471-3659-0_5.

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Ganz, Jeremy C. "Cerebral Arteriovenous Malformations." In Gamma Knife Surgery, 97–111. Vienna: Springer Vienna, 1993. http://dx.doi.org/10.1007/978-3-7091-3313-2_10.

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Mankowitz, Scott, and Suzanne K. W. Mankowitz. "Cerebral Arteriovenous Malformations." In Consults in Obstetric Anesthesiology, 125–28. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-59680-8_34.

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Fornezza, Umberto, Zeno Perini, Gianpaolo Zambon, Mariano Zanusso, and Leopoldo S. Casentini. "Cerebral Arteriovenous Malformations." In CyberKnife NeuroRadiosurgery, 483–96. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-50668-1_42.

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Ganz, Jeremy C. "Cerebral Arteriovenous Malformations." In Gamma Knife Surgery, 97–111. Vienna: Springer Vienna, 1997. http://dx.doi.org/10.1007/978-3-7091-6831-8_10.

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Lindegaard, K. F., R. Aaslid, and H. Nornes. "Cerebral Arteriovenous Malformations." In Transcranial Doppler Sonography, 86–105. Vienna: Springer Vienna, 1986. http://dx.doi.org/10.1007/978-3-7091-8864-4_6.

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Al Awar, Omar, and Umang Jash Patel. "Pathophysiological Principles of Cerebral Arteriovenous Malformations." In Brain Arteriovenous Malformations, 51–59. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-63964-2_5.

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Ducruet, Andrew F., R. Webster Crowley, Cameron G. McDougall, and Felipe C. Albuquerque. "Embolization of Cerebral Arteriovenous Malformations." In PanVascular Medicine, 2537–50. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-37078-6_94.

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Flores, Bruno C., Alfred P. See, Andrew F. Ducruet, and Felipe C. Albuquerque. "Embolization of Cerebral Arteriovenous Malformations." In Textbook of Catheter-Based Cardiovascular Interventions, 1171–87. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-55994-0_69.

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Michelsen, W. Jost, and Sadek K. Hilal. "Embolization of Cerebral Arteriovenous Malformations." In Cerebrovascular Surgery, 1151–60. New York, NY: Springer New York, 1985. http://dx.doi.org/10.1007/978-1-4613-8516-5_5.

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Conference papers on the topic "Cerebral arteriovenous malformations"

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Dholke, Harshal, Mohan Rao, and Manas Panigrahi. "23. Intraoperative management of cerebral arteriovenous malformations: Our experience." In 15th Annual Conference of the Indian Society of Neuroanaesthesiology and Critical Care. Thieme Medical and Scientific Publishers Private Ltd., 2014. http://dx.doi.org/10.1055/s-0038-1646102.

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Hassan, Ahmed Attia Ahmed, Ali Hassan Elmokadem, Ahmed Bahaa Elden Elserwi, Mohamed Metwally Abo El Atta, and Talal Ahmed Youssef Amer. "Endovascular Management of Cerebral Arteriovenous Malformations: Technical and Clinical Outcome." In PAIRS Annual Meeting. Thieme Medical and Scientific Publishers Pvt. Ltd., 2020. http://dx.doi.org/10.1055/s-0041-1729057.

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Demyanovskaya, M., N. Strelnikov, A. Moskalev, and K. Orlov. "E-089 Deep cerebral arteriovenous malformations: comparison between transvenous and transarterial approaches." In SNIS 15TH ANNUAL MEETING, July 23–26, 2018, Hilton San Francisco Union Square San Francisco, CA. BMA House, Tavistock Square, London, WC1H 9JR: BMJ Publishing Group Ltd., 2018. http://dx.doi.org/10.1136/neurintsurg-2018-snis.165.

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Ni Bhuachalla, CF, A. Brady, TM O'Connor, N. Colwell, and M. Murphy. "Low Prevalence of Cerebral Arteriovenous Malformations in Irish Patients with Hereditary Haemorrhagic Telangiectasia." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a5780.

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Kiselev, V., A. Perfilev, A. Sosnov, and R. Gafurov. "E-091 Transvenous approach in the endovascular embolization of the cerebral arteriovenous malformations." In SNIS 15TH ANNUAL MEETING, July 23–26, 2018, Hilton San Francisco Union Square San Francisco, CA. BMA House, Tavistock Square, London, WC1H 9JR: BMJ Publishing Group Ltd., 2018. http://dx.doi.org/10.1136/neurintsurg-2018-snis.167.

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Fatania, G., M. Patel, JE Jackson, and CL Shovlin. "P175 Burden of cerebral infarcts identified by screening cerebral mri scans in patients with pulmonary arteriovenous malformations." In British Thoracic Society Winter Meeting 2017, QEII Centre Broad Sanctuary Westminster London SW1P 3EE, 6 to 8 December 2017, Programme and Abstracts. BMJ Publishing Group Ltd and British Thoracic Society, 2017. http://dx.doi.org/10.1136/thoraxjnl-2017-210983.317.

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Lahmiri, Salim, Mounir Boukadoum, and Antonio Di Ieva. "Detrended fluctuation analysis of brain hemisphere magnetic resonnance images to detect cerebral arteriovenous malformations." In 2014 IEEE International Symposium on Circuits and Systems (ISCAS). IEEE, 2014. http://dx.doi.org/10.1109/iscas.2014.6865658.

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Mayercik, V., R. Taiwo, M. Marks, R. Dodd, J. Heit, H. Do, and N. Telischak. "O-023 Arterial spin labeling (ASL) MRI assists in identification of cerebral micro-arteriovenous malformations." In SNIS 18TH ANNUAL MEETING. BMA House, Tavistock Square, London, WC1H 9JR: BMJ Publishing Group Ltd., 2021. http://dx.doi.org/10.1136/neurintsurg-2021-snis.23.

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Lieber, B. B., A. K. Wakhloo, A. Divani, and S. Rudin. "Determination of Vascular Geometry and Flow Velocity in Cerebral Arteriovenous Malformations (AVMs) Using Double Contrast and High-Speed Digital Subtraction Angiography." In ASME 1998 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 1998. http://dx.doi.org/10.1115/imece1998-0027.

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Abstract Successful therapeutic embolization, i.e., catheter-based occlusion of cerebral arteriovenous malformations (AVMs) using liquid embolic agents requires precise knowledge of highly variant AVM architecture, blood flow velocity, and transit times through the AVM fistulae. In this study we tried to visualize both the AVM vascular substructure and dynamics of discrete microdroplets traveling through the AVM using a single injection composed of both soluble and non-soluble contrast material. The contrast injection is traced with high-speed digital subtraction angiography. This novel technique serves as a valuable diagnostic tool to the interventionist prior to embolization of the AVM.
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Calvo, William J., Baruch B. Lieber, Ajay K. Wakhloo, and L. Nelson Hopkins. "Improved Histologic Analysis of Component Distribution and Wall Behavior in Cyanoacrylate-Embolized Vessels." In ASME 2001 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2001. http://dx.doi.org/10.1115/imece2001/bed-23131.

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Abstract Use of embolic agents is currently an accepted treatment modality among neurointerventional clinicians in order to completely occlude abnormal shunts in the cerebral vasculature known as arteriovenous malformations or AVMs. The first cyanoacrylate polymer recently approved by the US Food and Drug Administration for use in humans is n-butyl 2-cyanoacrylate (NBCA). In order to control the polymerization time of NBCA, as well as to opacify the mixture for angiographic visualization, an iodized poppyseed oil-based contrast agent (Lipiodol) is added to the NBCA. Glacial acetic acid is also used to delay polymerization further by decreasing the pH of the mixture.
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Reports on the topic "Cerebral arteriovenous malformations"

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Sirakov, Stanimir, Alexander Sirakov, Krasimir Minkin, Hristo Hristov, and Vasil Karakostov. The Bulgarian Experience in Endovascular Treatment of Cerebral Arteriovenous Malformations. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, January 2018. http://dx.doi.org/10.7546/crabs.2018.01.15.

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Sirakov, Stanimir, Alexander Sirakov, Krasimir Minkin, Hristo Hristov, and Vasil Karakostov. The Bulgarian Experience in Endovascular Treatment of Cerebral Arteriovenous Malformations. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, January 2018. http://dx.doi.org/10.7546/grabs2018.1.15.

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