Relevant bibliographies by topics / Central nervous system in rats

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Central nervous system in rats'

Author: Grafiati
Published: 4 June 2021
Last updated: 7 February 2022

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Journal articles on the topic "Central nervous system in rats"

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Lang, Frederick F., Fred J. Epstein, Joseph Ransohoff, Jeffrey C. Allen, Jeffrey Wisoff, I. Richmond Abbott, and Douglas C. Miller. "Central nervous system gangliogliomas." Journal of Neurosurgery 79, no. 6 (December 1993): 867–73. http://dx.doi.org/10.3171/jns.1993.79.6.0867.

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The records of 58 patients with gangliogliomas surgically treated between January 1, 1980, and June 30, 1990, were retrospectively reviewed in order to determine long-term survival, event-free survival, and functional outcome resulting after radical resection and to assess the impact of histological grading on outcome. Tumors were located in the cerebral hemisphere in 19 cases, the spinal cord in 30, and the brain stem in nine. Forty-four patients had gross total resection and 14 had radical subtotal resection. Only six patients underwent postoperative irradiation or chemotherapy and, therefore, the outcome was generally related to surgery alone. Of the 58 gangliogliomas, 40 were classified as histological grade I, 16 were grade II, and two were grade III. The median follow-up period was 56 months. There were no operative deaths, and the operative morbidity rate was 5%, 37%, and 33% for cerebral hemisphere, spinal cord, and brain-stem gangliogliomas, respectively. The 5-year actuarial survival rates for cerebral hemisphere, spinal cord, and brain-stem gangliogliomas were 93%, 84%, and 73%, respectively (p = 0.7). The event-free survival rate at 5 years was 95% for cerebral hemisphere gangliogliomas and 36% for spinal cord gangliogliomas (p < 0.05); for brain-stem gangliogliomas the event-free survival rate at 3 years was 53% (p < 0.05). Neurological function at recent follow-up evaluation was stable or improved in 81% of patients. Multivariate analysis (Cox linear regression) revealed tumor location to be the only variable predictive of outcome, with spinal cord and brain-stem gangliogliomas having a 3.5- and 5-fold increased relative risk of recurrence, respectively, compared to cerebral hemisphere gangliogliomas. Histological grade was not predictive of outcome, although in each location there was a trend for higher-grade tumors to have a shorter time to recurrence. It is concluded that radical surgery leads to long-term survival of patients with gangliogliomas, regardless of location, and adjuvant therapy can probably be reserved for special cases.
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Hamidi, Mehrdad. "Central nervous system distribution kinetics of indinavir in rats." Journal of Pharmacy and Pharmacology 59, no. 8 (August 2007): 1077–85. http://dx.doi.org/10.1211/jpp.59.8.0004.

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Parham, D., A. Tereba, P. J. Talbot, D. P. Jackson, and V. L. Morris. "Analysis of JHM Central Nervous System Infections in Rats." Archives of Neurology 43, no. 7 (July 1, 1986): 702–8. http://dx.doi.org/10.1001/archneur.1986.00520070058019.

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Arieli, R., and G. Hershko. "Prediction of central nervous system oxygen toxicity in rats." Journal of Applied Physiology 77, no. 4 (October 1, 1994): 1903–6. http://dx.doi.org/10.1152/jappl.1994.77.4.1903.

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Cumulative O2 toxicity (K) can be calculated using the expression K = t2 x PO2c, where t is exposure time and the power c is to be determined; the phenomenon is liable to occur when K reaches Kc, the threshold value of K at which a symptom is manifested. Six rats were each exposed six times to 6 ATA O2 at 2-day intervals until the first electrical discharge (FED) was noted in an electroencephalogram. There was no difference in latency to FED in the series of six exposures. Thirteen rats were exposed to O2 until FED was noted in an electroencephalogram. They were exposed to four constant PO2's of 5, 6, 7, and 8 ATA and to two combined profiles of 1) 5 min at 7 ATA followed by 5 ATA and 2) 15 min at 5 ATA followed by 7 ATA. The solution of the equation for each rat was used to predict its latency to FED on the combined profile. The correlation of predicted to measured latency was significant (P < 0.0001), and the slope was not different from 1. Solving for these parameters using the combination of all the data, we obtained Kc = 5.71 x 10(6) and c = 5.39, which correctly predicted the mean latency but failed to predict individual latency. It is preferable to use each rat as its own control. The significance of the correlation supports the validity of the power equation for calculating K.
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Fisher, L. A., C. R. Cave, and M. R. Brown. "Central nervous system cardiovascular effects of bombesin in conscious rats." American Journal of Physiology-Heart and Circulatory Physiology 248, no. 4 (April 1, 1985): H425—H431. http://dx.doi.org/10.1152/ajpheart.1985.248.4.h425.

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The effects of intracerebroventricular administration of bombesin on mean arterial pressure and heart rate were studied in conscious, freely moving rats. Injection of bombesin produced dose-dependent elevations of mean arterial pressure and reductions of heart rate. These effects were not caused by leakage of bombesin into the peripheral circulation. Adrenalectomy abolished the pressor action of bombesin but did not alter bombesin-induced bradycardia. Systemic phentolamine pretreatment prevented bombesin-induced changes of mean arterial pressure, whereas rats treated intravenously with captopril or a vasopressin antagonist still exhibited pressor responses to bombesin administration. Bombesin-induced bradycardia was partially antagonized by intravenous atropine methyl nitrate administration, whereas systemic injections of propranolol did not modify this response. It is concluded that bombesin acts within the central nervous system to elevate mean arterial pressure through an adrenal-dependent mechanism involving alpha-adrenergic receptors and to reduce heart rate through an adrenal-independent mechanism involving, at least in part, cardiac parasympathetic nervous activation.
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Rykaczewska-Czerwińska, Monika, Piotr Oleś, Michał Oleś, Mariola Kuczer, Danuta Konopińska, and Andrzej Plech. "Effect of alloferon 1 on central nervous system in rats." Pharmacological Reports 62 (September 2010): 62–63. http://dx.doi.org/10.1016/s1734-1140(10)71168-3.

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Szenohradszky, János, Anthony J. Trevor, Philip Bickler, James E. Caldwell, Manohar L. Sharma, Ira J. Rampil, and Ronald D. Miller. "Central Nervous System Effects of Intrathecal Muscle Relaxants in Rats." Anesthesia & Analgesia 76, no. 6 (June 1993): 1304–9. http://dx.doi.org/10.1213/00000539-199306000-00020.

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Szenohradszky, János, Anthony J. Trevor, Philip Bickler, James E. Caldwell, Manohar L. Sharma, Ira J. Rampil, and Ronald D. Miller. "Central Nervous System Effects of Intrathecal Muscle Relaxants in Rats." Anesthesia & Analgesia 76, no. 6 (June 1993): 1304–9. http://dx.doi.org/10.1213/00000539-199376060-00020.

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Miyaguchi, Hideki, Ineko Kato, Tadashi Sano, Hisanori Sobajima, Shinji Fujimoto, and Hajime Togari. "Dopamine penetrates to the central nervous system in developing rats." Pediatrics International 41, no. 4 (August 1999): 363–68. http://dx.doi.org/10.1046/j.1442-200x.1999.01084.x.

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Crabb, David W., Sandra L. Morzorati, Jay R. Simon, and Ting-Kai Li. "Central nervous system control of alcohol dehydrogenase activity in rats." Life Sciences 37, no. 25 (December 1985): 2381–87. http://dx.doi.org/10.1016/0024-3205(85)90105-5.

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Dissertations / Theses on the topic "Central nervous system in rats"

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Sevilla, Elenita L. "The influence of 5-HT3 receptor antagonism on aspects of CNS activity in morphine-dependent rats." Thesis, [Hong Kong] : University of Hong Kong, 1991. http://sunzi.lib.hku.hk/hkuto/record.jsp?B13019211.

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Silva, Kelly Regina Torres da. "Estudo da distribuição das proteínas relacionadas às teneurinas no sistema nervoso central de primatas não-humanos (Sapajus spp) e ratos (Rattus norvegicus)." Botucatu, 2016. http://hdl.handle.net/11449/139450.

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Orientador: Cláudio Aparecido Casatti
Resumo: As teneurinas (TENs) representam uma família de proteínas transmembrana preservada entre as espécies, presente principalmente no sistema nervoso central (SNC). Nos vertebrados essa família é composta por quatro homólogos, denominados de teneurina-1 a -4 (Ten-1, Ten-2, Ten-3 e Ten-4). Estudos mostraram a presença das TENs em vias motoras, olfatórias e visuais, especialmente durante a neurogênese em aves e roedores. A análise da distribuição neuroanatômica das TENs em primatas poderia ampliar o conhecimento destas proteínas, contribuindo com achados funcionais recentes. Portanto, os propósitos deste estudo foram: 1) avaliar a distribuição dos neurônios que exibem imunorreatividade relacionada às TENs-“like immunoreactivity” (TENs-LI), em particular Ten-2-LI, Ten-3-LI e Ten-4-LI no SNC do primata não-humano (Sapajus spp); 2) realizar análise comparativa dos sítios de distribuição da proteína Ten-3 entre o SNC de primatas (Sapajus spp) e roedores (Rattus norvegicus), uma vez que a Ten-3 apresentou distribuição significante no SNC de primatas; 3) correlacionar a distribuição das TENs com seus ligantes endógenos denominados de latrofilinas (LPHNs-1, 2 e 3) em áreas do SNC de primatas. Para isso, cortes coronais do SNC de macacos (n=3) e de ratos (n=4) foram submetidos à técnica de imuno-histoquímica e analisados em microscopia de luz ou confocal. Os resultados demonstraram a distribuição de neurônios e fibras nervosas exibindo TENs-LI em todo o neuroeixo de primatas. Neurônios e... (Resumo completo, clicar acesso eletrônico abaixo)
Abstract: Teneurins (TENs) represent a transmembrane protein family preserved along animal species, mainly in the central nervous system (CNS). This protein family is constituted by four homologues, named as teneurin 1 to 4 (Ten-1, Ten-2, Ten-3 and Ten-4). Previous studies pointed out presence of TENs in motor, olfactory and visual systems in chicken and rodents, especially during neurogenesis. The neuroanatomic distribution analysis of TENs in the primate brain could provide additional information on this protein system, as well as support functional data from recent studies. Therefore, the purposes of the present study were: 1) to evaluate the distribution of neurons exhibiting TENs-like immunoreactivity (TENs-LI), in particular, Ten- 2-LI, Ten-3-LI and Ten-4-LI in the CNS of non-human primates (Sapajus spp); 2) to comparatively analyze the main brain regions exhibiting Ten-3-LI between primates (Sapajus spp) and rodents (Rattus norvegicus), since Ten-3-LI showed significant distribution in the CNS of primates; 3) To correlate TENsLI neurons with latrophilins (LPHNs-1, 2 and 3), an endogenous TENs ligand, in the CNS of primates. For this purpose, coronal histological sections of the CNS of non-human primates (n=3) and rats (n=4) were submitted to immunohistochemistry techniques and analyzed under light or confocal microscopes. Neurons and nerve fibers exhibiting TENs-LI were observed in all parts of the CNS in primates. Neurons showing Ten-2-LI were present mainly in the brainstem, s... (Complete abstract click electronic access below)
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Hughes, Rhome. "Immunohistochemical characterization of neuronal cilia in the rat central nervous system." Thesis, University of North Texas, 2002. https://digital.library.unt.edu/ark:/67531/metadc3136/.

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An anti-G"11 antibody was used to label neuronal cilia throughout the rat central nervous system. Immunoreactive cilia were observed in every examined region of the rat CNS, but not in monkey or mouse tissue. Antibodies to G"q and G"q/11 failed to label cilia. Immunoreactive cilia were observed as early as postnatal day 0 in spinal tissue, and postnatal day 3 in hypothalamic tissue. There was a statistically significant negative correlation between a region's mean cilium length and that region's distance to the nearest ventricle; regions nearest ventricles were those with the longest cilia. This correlation suggests neuronal cilia may function as chemosensors, detecting substances as they move out from the cerebrospinal fluid and into the extracellular space of the brain.
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McClure-Sharp, Jilliane Mary, and mikewood@deakin edu au. "Regulation of corticotropin-releasing factor concentration and overflow in the rat central nervous system." Deakin University. School of Biological and Chemical Sciences, 1998. http://tux.lib.deakin.edu.au./adt-VDU/public/adt-VDU20060802.143911.

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Corticotropin-releasing factor (CRF) is the primary hormone of the hypothalamo-pituitary adrenal axis (HPA-axis). In addition to its endocrine function, it has been proposed that CRF acts as a neurotransmitter. The widespread distribution of CRF immunoreactivity and CRF receptors in the rat central nervous system (CNS) supports this theory. Immunohistochemical studies have demonstrated high levels of CRF immunoreactivity the rat hypothalamus, a brain region involved in the regulation and integration of a variety of endocrine and autonomic homeostatic mechanisms. CRF has been shown to be involved in a number of these activities such as blood pressure control, food and water intake, behaviour and emotional integration. Many of these activities demonstrate progressive dysfunction as ageing proceeds. The aim of this thesis was to investigate the regulation of CRF in the rat CNS, particularly over the period of maturation and ageing. Tissue extraction and peptide radioimmunoassay (RIA) techniques were developed in order to measure regional CRF concentrations as a function of age in the rat CNS. Seven brain regions were examined including the hypothalamus, pituitary, medulla oblongata, pons, cerebral cortex, cerebellum and midbrain. Three age ranges were investigated: 3 – 4 weeks, 4 – 5 months and 14 – 18 months, representing young, mature and old age groups. Data for the tissues of individual rats from each age group were analysed using one-way analysis of variance (ANOVA) with post-hoc Scheffé tests (SPSS Release 6 for Windows, 1989 – 1993). CRF were detected in measurable quantities in all brain regions examined. Different age-related patterns of change were observed in each brain region. CRF concentrations (ng/g tissue) were highest in the pituitaries of young rats and were significantly reduced over the period of maturation (P< 0.05). However, the high CRF concentration of the young rat pituitary was likely to be a factor of the smaller tissue mass. Although the absolute CRF content (ng/tissue) of this tissue appeared to decline with maturation and ageing, the reduction was not significant (P>0.05). Therefore the pituitary of the young rat was relatively enriched with CRF per gram tissue. The highest CRF concentration in mature and aged rats was measured in the hypothalamus, in accordance with previous immunohistochemical studies. Hypothalamic CRF concentrations (ng/g tissue) demonstrated no significant alterations with maturation and ageing. The absolute CRF content (ng/tissue) of the hypothalamus was significantly less in the young rat compared to mature and aged animals, however this was accompanied by a smaller tissue mass (P<0.05). The CRF concentrations (ng/g tissue) of the rat cerebral cortex and medulla oblongata demonstrated significant reduction with advancing age (P<0.05), however in both cases this appeared to be due to significant increases in mean tissue mass. The absolute CRF content of these tissues (ng/tissue) were not significantly different over the period of maturation and ageing (P>0.05). CRF concentration (ng/g tissue) and absolute content (ng/tissue) of the pons demonstrated a trend to increase with advanced age in the rat, however this was not significant in both cases (P>0.05). Of interest were the significant increases observed in the CRF concentrations of the cerebellum and midbrain (ng/g tissue with advanced ageing (P<0.05). Significant increases were also observed in the mean tissue mass and absolute CRF content (ng/tissue) of these regions in aged rats (P<0.05). These findings perhaps indicate increased CRF synthesis and or decreased CRF turnover in these tissues with advancing age. The second stage of these studies examined age-related alterations in basal and potassium-stimulated hypothalamic CRF and overflow over the period of maturation and ageing in the rat, and required the preliminary development of an in vitro tissue superfusion system. The concomitant release of the co-modulatory compound, neuropeptide Y (NPY) was also measured. NPY has been shown to positively regulate CRF release and gene expression in the hypothalamus. In addition, NPY has been demonstrated to be involved in a number of hypothalamic activities, including blood pressure control and food intake regulation. Hypothalamic superfusion data were analysed using one factor repeated measures ANOVA (SPSS Release 6 for Windows, 1989-1993) followed by least significant difference tests ( Snedecor and Cochran, 1967) to enable both time and age comparisons. Basal hypothalamic CRF overflow was unaltered with maturation and ageing in the rat. Potassium stimulation (56 mM) elicted a significant 2 – 3 fold increase in hypothalamic CRF overflow across age groups (P<0.05). Stimulated hypothalamic CRF overflow was significantly greater in the young rat compared to the mature and aged animals (P<0.05). The enhanced response to depolarizing stimulus was observed at an age when the absolute CRF content of the hypothalamus was significantly less that of other age groups. It is possible that the enhanced responsiveness of the young rat may be of survival advantage in life threatening situations. Basal hypothalamic NPY overflow was much less than that of CRF, and potassium stimulation resulted in a very different age-related profile. The hypothalamic NPY response to depolarization was significantly reduced in the young rat and declined significantly with advanced ageing (P<0.05). The contrasting profiles of stimulated CRF and NPY overflow may indicate the activity of alternative regulatory factors present in the hypothalamus, whose activity may also be affected in an age-related manner. The final stage of these studies examined the nature of NPY modulation of hypothalamic CRF overflow in the mature rat. The facilitatory effect of NPY on hypothalamic CRF overflow was confirmed. The application of NPY (0.1 µM) significantly increased CRF overflow approximately 4 fold of basal (P<0.05). In addition, the role of the NPY-Y1 receptor was investigated by the prior application of Y1 receptor antagonists, GW1229 (0.05 µM). At this concentration GW1229 significantly reduced hypothalamic CRF overflow induced by perfusion with NPY (0.1 µm), P<0.05. It was concluded the Y1 receptor does have a role in the regulation of hypothalamic CRF overflow by NPY.
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Davies, M. "5-hydroxytryptamine receptors in the central nervous system." Thesis, Bucks New University, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.382505.

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Yick, Leung-wah. "Promotion of neuronal survival and axonal regeneration in Clarke's nucleus after spinal cord injury in adult rats /." Hong Kong : University of Hong Kong, 1999. http://sunzi.lib.hku.hk/hkuto/record.jsp?B21090099.

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Alrtemi, Milod M. Ahmed. "Radioprotective effects of rooibos herbal tea on the developing central nervous system of wistar rats." University of the Western Cape, 2018. http://hdl.handle.net/11394/6532.

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Magister Scientiae - MSc
Background: Early postnatal radiation exposure from environmental, diagnostic or therapeutic sources is potentially deleterious to the developing nervous system resulting in oxidative stress, structural damage, altered neurochemistry, DNA damage, inflammatory stresses as well as correlating cognitive impairment during adult life. Numerous studies in literature have investigated the radioprotective effects of medicinal plants and beverages. However, only a few studies have focused on the radioprotective effects of rooibos, an indigenous South African herbal tea, well known for its many acclaimed health benefits. Aims: This study was done to investigate the diverse radioprotective potential of fermented Rooibos herbal tea (FRHT) consumed ad libitum by pregnant rats on the adult offspring rats exposed to a once-off 6 Gy dose of gamma irradiation on postnatal day 3. Methods: Twenty-four (24) adult female rats were equally divided into four groups (6 per group) as control (NS), radiation (X), tea (RT) and their combination. On PND 30, offspring rats were subjected to neurobehavioural assessment for open field and novel object recognition parameters and later sacrificed, the brain tissues removed and processed for histological, immunohistochemical and neurochemical analyses, using standard techniques. Results: Pre-treatment with FRHT showed overall protection against radiation-induced distortions in offspring rats by significantly improving exploratory activity, the frequency of central square entry, rearing episodes, cumulative freezing time and memory retention as indicated by a relatively higher recognition index. FRHT was also found to significantly improve the antioxidant defence mechanisms in the offspring rats by reversing lowered FRAP levels, increasing superoxide dismutase and catalase enzyme activities and reducing lipid peroxidation. Histological and immunohistochemical analyses showed that morphological alterations were generally attenuated in the RTX group and the high number of caspase-3 and Glial fibrillary acidic protein (GFAP)-positive cells was significantly reduced, indicating protective effects against apoptosis and gliosis. Conclusion: Taken together, our findings tend to suggest that the potential radioprotective effects of FRHT are multimodal, possibly executed through the anti-apoptotic, antioxidative, anti-gliosis and other mechanisms, as observed in this study, and this is often attributed to the high polyphenol content in Rooibos tea.
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Totola, Leonardo Tedesco. "Envolvimento da região comissural do núcleo do trato solitário nas respostas cardiovasculares e simpáticas promovidas pela injeção do anti-hipertensivo de ação central moxonidina em ratos." Universidade de São Paulo, 2013. http://www.teses.usp.br/teses/disponiveis/42/42137/tde-14032014-130320/.

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O objetivo central do presente estudo foi avaliar se os agonistas adrenérgicos a2 e imidazólicos, importantes drogas de ação anti-hipertensiva utilizadas na clínica médica, podem atuar também na região comissural do núcleo do trato solitário (NTSc), o qual constituí uma importante região do bulbo envolvida no controle cardiovascular. Em ratos Wistar adultos, observamos que a hipotensão produzida pela injeção de moxonidina no 4º V foi reduzida após a lesão eletrolítica do NTSc. Ademais, a injeção de moxonidina no NTSc reduziu a pressão arterial média (PAM), a frequência cardíaca (FC) e a atividade simpática (AS). A injeção de antagonistas adrenérgicos (ioimbina ou RX821002) no NTSc foi capaz de bloquear as respostas hipotensora e de simpatoinibição produzida pela moxonidina no NTSc. A injeção bilateral de moxonidina na região RVL/C1 reduziu PAM e AS de maneira mais intensa do que as injeções no NTSc. Em concordância com os resultados apresentados, mostramos que a atividade elétrica dos neurônios da região do RVL/C1 foi reduzida após a injeção de moxonidina no NTSc. Concluímos que a moxonidina pode produzir os seus efeitos anti-hipertensivos atuando também sobre o NTSc.
The main objective of this study was to evaluate whether the a2 adrenergic and imidazoline agonists, important antihypertensive drugs used in clinical medicine, may also act in the commissural region of the nucleus of the solitary tract (cNTS), which constitutes an important region of brainstem involved in cardiovascular control. In adult rats, the hypotension elicited by central injections of moxonidine was reduced after electrolytic lesion of cNTS. Furthermore, injection of moxonidine into the cNTS reduced mean arterial pressure (MAP), heart rate (HR) and sympathetic activity (SNA). Injection of the a2 adrenergic antagonist (RX821002 or yohimbine) into the cNTS completely blocked the hypotension and sympathoinhibition responses produced by moxonidine into the cNTS. Bilateral injection of moxonidine in the RVLM/C1 produced huge effects on MAP and SNA in comparison of cNTS injections. In agreement with our results, moxonidine-injected into the cNTS also elicited a reduction in the activity of RVLM/C1 neurons. Our conclusion is that moxonidine may produce their antihypertensive effects also acting on cNTS neurons.
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Pook, P. C. K. "Ligand binding and electrophysiological studies of excitatory amino acid receptors in the rat central nervous system." Thesis, University of Bristol, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.381675.

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Hu, Ying. "Optic nerve regeneration in adult rat /." Connect to this title, 2006. http://theses.library.uwa.edu.au/adt-WU2007.0080.

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Books on the topic "Central nervous system in rats"

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Central nervous system. Cambridge: Cambridge University Press, 2009.

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Central nervous system angiitis. Boston: Butterworth-Heinemann, 2000.

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Central nervous system infections. Philadelphia, Pennsylvania: Elsevier, 2013.

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Emerich, Dwaine F., Reginald L. Dean, and Paul R. Sanberg, eds. Central Nervous System Diseases. Totowa, NJ: Humana Press, 2000. http://dx.doi.org/10.1007/978-1-59259-691-1.

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Ahluwalia, Manmeet, Philippe Metellus, and Riccardo Soffietti, eds. Central Nervous System Metastases. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-23417-1.

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Ramakrishna, Rohan, Rajiv S. Magge, Ali A. Baaj, and Jonathan P. S. Knisely, eds. Central Nervous System Metastases. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-42958-4.

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Kryzhanovsky, G. N. Central Nervous System Pathology. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-7870-9.

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Allen, Deborah Hutchinson, and Laurie L. Rice. Central nervous system cancers. Pittsburgh, Pa: Oncology Nursing Society, 2010.

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1933-, Voogd J., and Huijzen Chr van, eds. The human central nervous system. 4th ed. New York: Springer, 2008.

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Lacruz, César R., Javier Saénz de Santamaría, and Ricardo H. Bardales. Central Nervous System Intraoperative Cytopathology. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-98491-9.

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Book chapters on the topic "Central nervous system in rats"

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Huang, Ming-Chao, and Yi-Lo Lin. "Kringle 1-5 Reduces Growth of Malignant Gliomas in Rats." In Tumors of the Central Nervous System, 25–34. Dordrecht: Springer Netherlands, 2015. http://dx.doi.org/10.1007/978-94-017-7224-2_3.

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Lewis, S. J., and B. Jarrott. "The Effects of Continuous Clonidine Infusion on the Circadian Rhythms of Arterial Blood Pressure, Heart Rate and Spontaneous Locomotor Activity in Normotensive Wistar-Kyoto Rats." In Circadian Rhythms in the Central Nervous System, 213–16. London: Palgrave Macmillan UK, 1985. http://dx.doi.org/10.1007/978-1-349-07837-0_19.

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Lattanzi, Davide, David Savelli, Michael Di Palma, Stefano Sartini, Silvia Eusebi, Dasiel O. Borroto-Escuela, Riccardo Cuppini, Kjell Fuxe, and Patrizia Ambrogini. "Electrophysiological Approach to GPCR–RTK Interaction Study in Hippocampus of Adult Rats." In Receptor-Receptor Interactions in the Central Nervous System, 71–90. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8576-0_6.

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Flores-Burgess, Antonio, Carmelo Millón, Belén Gago, José Angel Narváez, Kjell Fuxe, and Zaida Díaz-Cabiale. "Small Interference RNA Knockdown Rats in Behavioral Functions: GALR1/GALR2 Heteroreceptor in Anxiety and Depression-Like Behavior." In Receptor-Receptor Interactions in the Central Nervous System, 133–48. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8576-0_9.

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Savolainen, K., H. Hervonen, H. Komulainen, and P. Kurttio. "Peripheral and Central Nervous System Effects of Nabam and Ethylenethiourea in Rats." In Archives of Toxicology, 345. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-71248-7_62.

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Dési, I., L. Nagymajtényi, R. Lorencz, and Z. Molnár. "The Effects of Organophosphorous Compounds on the Central Nervous System of Rats." In Archives of Toxicology, 33–37. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-74936-0_6.

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Rossi, E., and G. F. Rossi. "Heart Rate Behaviour During Partial Epileptic Seizures. An Electroclinical Study." In Central Nervous System Control of the Heart, 187–201. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4613-2327-3_15.

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Hashimoto, Naoya, and Toshiki Yoshimine. "Incidentally Discovered Meningiomas: Growth Rates and Patterns." In Tumors of the Central Nervous System, Volume 7, 87–94. Dordrecht: Springer Netherlands, 2012. http://dx.doi.org/10.1007/978-94-007-2894-3_10.

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Van Dam, A. M., S. R. Ruuls, C. Marquette, J. Bauer, C. J. A. de Groot, H. Tsiang, C. D. Dijkstra, and F. J. H. Tilders. "Nitric Oxide in the Central Nervous System of Rats Suffering from Rabies Virus Infection or Experimental Allergic Encephalomyelitis." In Neurochemistry, 967–72. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-5405-9_162.

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Imrich, H., S. Schwender, A. Hein, and R. Dörries. "Phenotypic and Functional Characterization of CD4+ T-Cells Infiltrating the Central Nervous System of Rats Infected with Coronavirus MHV IV." In Coronaviruses, 437–42. Boston, MA: Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-2996-5_69.

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Conference papers on the topic "Central nervous system in rats"

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Hill, Jonathan M., Ronald S. Adrezin, and Leonard Eisenfeld. "Wireless Central Apnea Response System for Neonatal Intensive Care." In ASME 2008 3rd Frontiers in Biomedical Devices Conference. ASMEDC, 2008. http://dx.doi.org/10.1115/biomed2008-38105.

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An apnea event is defined as the cessation of breathing for 10 to 20 seconds, generally accompanied by bradycardia (decrease in heart rate), cyanosis, or both. Unlike apnea in adults, which is usually caused by an obstruction in the airway, central apnea events appear in premature babies because the autonomic nervous system is not yet fully developed.
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Charpak, Serge. "Two-photon imaging of neuronal activity and vascular flow in the rat central nervous system." In Frontiers in Optics. Washington, D.C.: OSA, 2004. http://dx.doi.org/10.1364/fio.2004.fwf2.

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Di Guoqing, Zheng Yue, and Xu Jing. "The effects of high-speed train noise on tissue morphology of the rat central nervous system." In 2011 International Conference on Electric Technology and Civil Engineering (ICETCE). IEEE, 2011. http://dx.doi.org/10.1109/icetce.2011.5775746.

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Prokopova, Alena, Alena Gostyukhina, Tatyana Zamoshchina, Olga Doroshenko, Oksana Zhukova, and Konstantin Zaitsev. "BEHAVIORAL ACTIVITY AND LEVELS OF CORTICOSTERON IN THE SERUM OF LABORATORY RATS WITH DIFFERENT CENTRAL NERVOUS SYSTEM REACTIVITY AFTER LIGHT DESYNCHRONOSIS AND PHYSICAL OVERFATCH." In XVII INTERNATIONAL INTERDISCIPLINARY CONGRESS NEUROSCIENCE FOR MEDICINE AND PSYCHOLOGY. LCC MAKS Press, 2021. http://dx.doi.org/10.29003/m2285.sudak.ns2021-17/308-309.

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Smith, Katisha D., and Liang Zhu. "In Vivo Experimental Study of Rat Brain and Spinal Temperatures During Non-Invasive Spinal Cord Hypothermia Using a Cooling Pad." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53129.

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Traumatic injury causes mechanical tissue disruption that immediately follows a traumatic event. After the initial event, secondary injury often occurs. It is a cellular and molecular response to external trauma, including ischemia, inflammation, apoptosis, necrosis, and edema in the central nervous system (CNS). Since secondary injuries can lead to paralysis and permanent neurological damage, most current treatment are devoted to delaying or preventing secondary neurological injury.
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Kolesnikova, Inna, Natalia Budennaya, Yurii Severiukhin, Kristina Lyakhova, Dina Utina, and Victor Gaevsky. "THE EFFECT ON MORPHOLOGICAL CHANGES IN THE CENTRAL NERVOUS SYSTEM AND PHYSIOLOGICAL MEASUREMENTS OF AGED MICE AFTER TOTAL IRRADIATION BY GAMMA RAYS." In XVII INTERNATIONAL INTERDISCIPLINARY CONGRESS NEUROSCIENCE FOR MEDICINE AND PSYCHOLOGY. LCC MAKS Press, 2021. http://dx.doi.org/10.29003/m2165.sudak.ns2021-17/193-194.

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Kim, Jung Hwan, Garrett W. Astary, Thomas H. Mareci, and Malisa Sarntinoranont. "A Computational Model of Direct Infusion Into the Rat Brain: Corpus Callosum and Hippocampus." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-205945.

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Despite the high therapeutic potential of many macromolecular drugs, it has proven difficult to apply them to treatment of cancer and other degenerative diseases of the central nervous system (CNS) due to low capillary permeability and low diffusivity. To overcome these barriers, recent experimental studies have shown local infusion, i.e., convection-enhanced delivery (CED), to be a promising delivery technique in the brain and spinal cord [1–3]. Predictive models of extracellular fluid flow and transport during CED would be useful for treatment optimization and planning.
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Smith, Joshua H., and Jose Jaime García. "A Nonlinear Biphasic Model for Fluid Transport and Tissue Deformation During Constant Flow Rate Infusion Into Brain Tissue." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-204506.

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The delivery of therapeutic agents into the brain is impeded by the blood-brain barrier, preventing adequate treatment of diseases of the central nervous system. Convection enhanced delivery was developed as a means to deliver therapeutic agents directly into brain tissue and to transport the drugs in the extracellular space using convective flow. Poroelastic or biphasic models have been used to study the concomitant fluid transport and tissue deformation that occurs during infusion, however previous studies have been limited by the assumption of linear elasticity of the solid phase [1].
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Tanishita, Kazuo, Kazuto Masamoto, Iwao Kanno, and Hirosuke Kobayashi. "Biotransport to the Cerebral Tissues Related to the Vascular Diseases." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192501.

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Brain is a highly oxidative organ and its consumption rate of oxygen accounts for 20 percent of that of the whole body. This large consumption rate must be met by continuous supply of oxygen, because lack of oxygen rapidly causes irreversible damage to central nervous system. Acute hypoxic episodes cause a certain pattern of regional damage. Cerebral cortex (e.g., layers III, V, and VI) is one of the most susceptible regions to hypoxia, and damage to sensorimotor function is particularly severe in humans that survive hypoxic/ischemic episodes. However, little is known about whether oxygen transport in intracortical regions relates to such selective vulnerability to hypoxia.
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Begonia, Mark G. T., Jun Liao, Mark F. Horstemeyer, and Lakiesha N. Williams. "Strain Rate Dependence in the Structure Property Relationship of Porcine Brain." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206371.

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The brain is the control center for the central nervous system (CNS), and it is composed of specialized divisions that are attributed to a vast assortment of structural, homeostatic, and cognitive functions. These distinct regions are surrounded by supportive tissue and comprised of a complex arrangement of neurons that can be further categorized as either gray or white matter. The cerebrum constitutes the larger surrounding portion of the forebrain and includes sinuous ridges called gyri that are separated by grooves or fissures called sulci. The intermediate layer of the cerebrum primarily consists of white matter tracts that are responsible for integrating various regions throughout the cerebrum. The innermost and outermost layers of tissue mainly contain gray matter and are collectively known as the subcortical nuclei and cerebral cortex, respectively, which are crucial integrating components of the CNS [1]. An investigation into the mechanical properties of this vital organ coupled with microstructural characterization of its constituents under varying deformation levels is therefore crucial for implementing more accurate prediction and prevention of traumatic brain injury (TBI).
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Reports on the topic "Central nervous system in rats"

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Ridgway, Sam H. The Cetacean Central Nervous System. Fort Belvoir, VA: Defense Technical Information Center, January 1999. http://dx.doi.org/10.21236/ada381704.

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Albquerque, Edson X. Molecular Targets for Organophosphates in the Central Nervous System. Fort Belvoir, VA: Defense Technical Information Center, June 2004. http://dx.doi.org/10.21236/ada426356.

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Rowland, Vernon, and Henry Gluck. Attention and Preparatory Processes in the Central Nervous System. Fort Belvoir, VA: Defense Technical Information Center, August 1986. http://dx.doi.org/10.21236/ada171316.

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Butler, F. K., and Jr. Central Nervous System Oxygen Toxicity in Closed-Circuit Scuba Divers. Fort Belvoir, VA: Defense Technical Information Center, March 1986. http://dx.doi.org/10.21236/ada170879.

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Clark, J. M., and C. J. Lambertsen. Extension of Central Nervous and Visual System Oxygen Tolerance in Physical Work. Fort Belvoir, VA: Defense Technical Information Center, December 1990. http://dx.doi.org/10.21236/ada239160.

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Mery, Laura, Matthew Wayner, John McQuade, and Erica Anderson. Characterization of the Effects of Fatigue on the Central Nervous System (CNS) and Drug Therapies. Fort Belvoir, VA: Defense Technical Information Center, November 2007. http://dx.doi.org/10.21236/ada489794.

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Catlin, Kristen M. Role of Cytokines and Neurotrophins in the Central Nervous System in Venezuelan Equine Encephalitis Pathogenesis. Fort Belvoir, VA: Defense Technical Information Center, February 2001. http://dx.doi.org/10.21236/ad1012369.

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Li, Yanming, Zhigang Zhao, and Yuanbo Liu. Combined chemotherapy in new diagnosed primary central nervous system lymphoma: a systematic review and network meta‑analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2020. http://dx.doi.org/10.37766/inplasy2020.9.0084.

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Carpenter, A. V., W. D. Flanders, E. L. Frome, D. J. Crawford-Brown, and S. A. Fry. Radiation exposure and central nervous system cancers: A case-control study among workers at two nuclear facilities. Office of Scientific and Technical Information (OSTI), March 1987. http://dx.doi.org/10.2172/6646019.

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Qu, Chunrun, Yu Chen, Yuzhen Ouyang, Ruoyu Lu, Yu Zeng, and Zhixiong Liu. Metagenomics Next Generation Sequencing for the Diagnosis of Central Nervous System Infection: A Systematic Review and Meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, February 2021. http://dx.doi.org/10.37766/inplasy2021.2.0002.

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