Relevant bibliographies by topics / Cellular rejection

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Cellular rejection'

Author: Grafiati
Published: 4 June 2021
Last updated: 26 April 2022

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Journal articles on the topic "Cellular rejection"

1

Willicombe, Michelle, Candice Roufosse, Paul Brookes, Adam G. McLean, Jack Galliford, Tom Cairns, Terry H. Cook, and David Taube. "Acute Cellular Rejection." Transplantation Journal 97, no. 4 (February 2014): 433–39. http://dx.doi.org/10.1097/01.tp.0000437431.97108.8f.

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Wecker, Hermann, and Hugh Auchincloss. "Cellular mechanisms of rejection." Current Opinion in Immunology 4, no. 5 (January 1992): 561–66. http://dx.doi.org/10.1016/0952-7915(92)90026-b.

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Korsgren, Olle. "Acute cellular xenograft rejection." Xenotransplantation 4, no. 1 (February 1997): 11–19. http://dx.doi.org/10.1111/j.1399-3089.1997.tb00159.x.

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Subherwal, S., J. A. Kobashigawa, G. Cogert, J. Patel, M. Espejo, and B. Oeser. "Incidence of acute cellular rejection and non-cellular rejection in cardiac transplantation." Transplantation Proceedings 36, no. 10 (December 2004): 3171–72. http://dx.doi.org/10.1016/j.transproceed.2004.10.048.

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Bolton, E. M., J. A. Gracie, J. D. Briggs, J. Kampinga, and J. A. Bradley. "Cellular requirements for renal allograft rejection in the athymic nude rat." Journal of Experimental Medicine 169, no. 6 (June 1, 1989): 1931–46. http://dx.doi.org/10.1084/jem.169.6.1931.

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This study has examined the ability of adoptively transferred CD4+ and CD8+ T cells to mediate rejection of a fully allogeneic DA renal graft in the PVG nude rat. Transfer, at the time of transplantation, of naive CD4+ T cells caused rapid graft rejection and primed CD4+ cells were several times more potent. In contrast, naive or specifically sensitized CD8+ cells were entirely ineffective at mediating renal allograft rejection. Whereas nonrejecting grafts showed only a mild cellular infiltrate, rejecting grafts in CD4+ reconstituted animals showed a substantial infiltrate and many of the infiltrating cells had a phenotype (MRC OX8+, MRC OX19-), consistent with NK cells. Experiments using a mAb (HIS 41) against an allotypic determinant of the leukocyte common antigen confirmed that the majority (greater than 80%) of the cellular infiltrate in rejecting grafts derived from the host rather than from the CD4+ inoculum. Infiltrating mononuclear cells, obtained from rejecting allografts 7 d after transplantation in CD4+-injected PVG nude hosts, showed high levels of in vitro cytotoxicity against not only kidney donor strain Con A blasts but also third-party allogeneic Con A blasts, as well as against both NK and LAK susceptible targets. When splenocytes from nontransplanted nude PVG rats were tested in vitro they also demonstrated high levels of lytic activity against both NK and LAK susceptible targets as well as allogeneic Con A blasts, which were not susceptible to lysis by spleen cells from euthymic rats. These findings suggest that injected CD4+ cells may cause renal allograft rejection by the recruitment of extrathymically derived, widely alloreactive cells into the kidney in this model of graft rejection.
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Pinchuk, A. V., R. V. Storozhev, I. V. Dmitriev, N. V. Shmarina, G. A. Nefedova, R. Sh Muslimov, and Yu S. Teterin. "Cellular rejection of pancreaticoduodenal graft." Russian Journal of Transplantology and Artificial Organs 20, no. 3 (September 17, 2018): 80–86. http://dx.doi.org/10.15825/1995-1191-2018-3-80-86.

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Aim. The evaluation of donor’s duodenum histological examination in diagnosis of pancreaticoduodenal graft rejection.Materials and methods. The 35-yo patient with terminal diabetic nephropathy undergone simultaneous retroperitoneal kidney and pancreas transplantation with enteric exocrine drainage of the graft via inter-duodenal anastomosis. When performing the esophagogastroduodenoscopy 2 years posttransplant we implemented histologic examination of the duodenum of the graft.Results. We diagnosed and verified severe cellular rejection of pancreaticoduodenal graft. Successful etiopathogenetic treatment of acute rejection of the graft (pulse therapy with glucocorticoids) was performed.Discussion. The diagnostic value of donor’s duodenum morphological examination in the diagnosis of pancreaticoduodenal graft rejection, the efficacy of anti-rejection treatment were performed in this case.
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Hutchinson, Ian V. "Cellular mechanisms of allograft rejection." Current Opinion in Immunology 3, no. 5 (October 1991): 722–28. http://dx.doi.org/10.1016/0952-7915(91)90103-8.

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Nikaein, Afzal. "Cellular Basis of Allograft Rejection." Baylor University Medical Center Proceedings 1, no. 2 (April 1988): 39–48. http://dx.doi.org/10.1080/08998280.1988.11929670.

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Hamdorf, Matthias, Satoru Kawakita, and Matthew Everly. "The Potential of MicroRNAs as Novel Biomarkers for Transplant Rejection." Journal of Immunology Research 2017 (2017): 1–12. http://dx.doi.org/10.1155/2017/4072364.

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The control of gene expression by microRNAs (miRNAs, miR) influences many cellular functions, including cellular differentiation, cell proliferation, cell development, and functional regulation of the immune system. Recently, miRNAs have been detected in serum, plasma, and urine and circulating miR profiles have been associated with a variety of diseases. Rejection is one of the major causes of allograft failure and preventing and treating acute rejection are the central task for clinicians working with transplant patients. Invasive biopsies used in monitoring rejection are burdensome and risky to transplant patients. Novel and easily accessible biomarkers of acute rejection could make it possible to detect rejection earlier and make more fine-tuned calibration of immunosuppressive or new target treatment possible. In this review, we discuss whether circulating miRNA can serve as an early noninvasive diagnostic biomarker and an expression fingerprint of allograft rejection and transplant failure. Understanding the regulatory interplay of relevant miRNAs and the rejecting allograft will result in a better understanding of the molecular pathophysiology of alloimmune injury.
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Manyeruke, F., T. Pennel, R. Roberts, and G. L. Calligaro. "Acute cellular rejection in lung transplantation." African Journal of Thoracic and Critical Care Medicine 25, no. 2 (July 31, 2019): 55. http://dx.doi.org/10.7196/sarj.2019.v25i2.010.

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Dissertations / Theses on the topic "Cellular rejection"

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Sleater, Michelle Leigh. "Cellular and molecular effector mechanisms of islet allograft rejection /." Connect to full text via ProQuest. IP filtered, 2006.

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Thesis (Ph.D. in Immunology) -- University of Colorado at Denver and Health Sciences Center, 2006.
Typescript. Includes bibliographical references (leaves 151-168). Free to UCDHSC affiliates. Online version available via ProQuest Digital Dissertations;
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Chen, Raymond Hsin-Chih. "Cellular infiltration in transplanted organs : detection of cytotoxic granule-associated proteins." Thesis, University of Oxford, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.357410.

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Bathgate, Andrew John. "The pre-transplant prediction of acute cellular rejection following liver transplantation." Thesis, University of Edinburgh, 2000. http://hdl.handle.net/1842/22544.

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The aim of this thesis was to investigate the effect of various parameters in patients with chronic liver disease pretransplant which may influence the occurrence of acute rejection post transplant. This may be useful in tailoring immunosuppression to avoid adverse effects in patients less likely to develop acute rejection. The role of cytokines in acute rejection is not clear but animal and human studies had suggested that tumour necrosis factor alpha (TNF-a) played some role. Polymorphisms in the genes encoding TNFa, interleukin 10 and transforming growth factor beta1 (TGFb1) which influence in vitro production of cytokines were examined in transplant patients. This showed an increase in the TNFa 2 polymorphism at position -308 in patients with acute rejection but no association with IL-10 or TGFb1 polymorphisms. Pretransplant levels of TNFa and IL-10 were measured following stimulation of peripheral blood mononuclear cells with lipopolysaccharide from patients with chronic liver disease. PBMC were preincubated with different immunosupressants. There was increased production of stimulated TNFa pretransplant in patients who went on to develop acute rejection. No relationship was found between IL-10 production and acute rejection. There were differences in the effects of tacrolimus, cyclosporin and dexamethasone on the production of both cytokines. The pretransplant immune status of patients was assessed by contact sensitisation to diphenylcyclopropenone (DPC). This demonstrated that patients unable to mount an immune response to DPC did not require treatment for acute rejection following liver transplantation. It also demonstrated a correlation between the strength of reaction to DPC and the severity of acute rejection.
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Flynn, T. H. "Clinical and experimental studies on the cellular mediators of corneal allograft rejection." Thesis, University College London (University of London), 2011. http://discovery.ucl.ac.uk/1306706/.

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Despite significant advances in our knowledge of the cellular and molecular elements of transplant immunology the 10 year survival probability for all human corneal grafts is 0.73. In some "high-risk" recipients it is as low as 0.37. To date almost all our knowledge about the cellular events during acute corneal graft rejection comes from animal models. In mice, the presence of pre-existing host corneal vascularisation confers "high-risk" status on a graft and has been shown to accelerate rejection. In the first part of this thesis the effect on survival of grafting to an inflamed conjunctival bed was investigated. Using a mouse model of allergic conjunctivitis significantly reduced survival was seen in graft recipients with perioperative conjunctival inflammation. This appeared to be due to the local effects of conjunctivitis rather than systemic effects of allergy/ atopy. Subsequent experiments investigated the effect of perioperative allergic conjunctivitis on the cellular components of both early (surgical trauma-induced, alloantigen-independent) and late (alloantigen-dependent; rejection) post-keratoplasty anterior segment inflammation and demonstrated significant effects on both. Grafts recipients with allergic conjunctivitis had significantly greater early post-operative corneal inflammation and associated corneal and conjunctival lymphangiogenesis. Analysis of graft infiltrating cells during rejection in mice confirmed that large numbers of CD4+ cells, CD8+ cells and macrophages were recruited. Flow cytometric analysis of human aqueous during acute endothelial rejection demonstrated for the first time the presence of CD4+ cells, CD8+ cells and a surprisingly high proportion of macrophages therein. In mouse recipients with allergic conjunctivitis eosinophils were found in both the graft itself and the ciliary body during rejection although the role of these cells during rejection is uncertain. Chemokine analysis during both murine and human corneal graft rejection demonstrated increased expression of the chemokine IP-10 (CXCL-10) suggesting a potentially important role for this protein in the rejection process.
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McLean, Adam George. "Patterns of graft infiltration and cytokine gene expression during the first ten days of kidney transplantation." Thesis, University of Oxford, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.390513.

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Wilson, Nicole K. "Borderline Lesions Exhibit Clinical and Graft Survival Characteristics Common to Acute Cellular Rejection." University of Cincinnati / OhioLINK, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1627665576477761.

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Salah, Adeeb Ahmed Kassim. "Application of Complement Component 4d Immunohistochemistry to ABO-Compatible and ABO-Incompatible Liver Transplantation." Kyoto University, 2015. http://hdl.handle.net/2433/199180.

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Webley, Kayonne. "INTERFERENCE REJECTION PERFORMANCE AS A MEANS OF FREQUENCY OPTIMISATION IN A MIXED CELLULAR/MANET NETWORK." International Foundation for Telemetering, 2006. http://hdl.handle.net/10150/604076.

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ITC/USA 2006 Conference Proceedings / The Forty-Second Annual International Telemetering Conference and Technical Exhibition / October 23-26, 2006 / Town and Country Resort & Convention Center, San Diego, California
Research at Morgan State University shows a means of enabling both a mobile ad-hoc network (MANET) and a cellular network to operate simultaneously in the same spectrum. This enhanced frequency efficiency would facilitate the creation of a hybrid or Mixed Cellular/MANET network (MCMN) in which each of the MCMN sub-networks would have access to the entire allotted spectrum. Interference rejection and excision have been identified as a means of distinguishing between and isolating the two different kinds of signals. This paper shows the promising performance of such techniques within the MCMN environment as a part of the integrated Network Enhanced Telemetry (iNET) project.
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He, Rong. "AMPS co-channel interference rejection techniques and their impact on system capacity." Diss., This resource online, 1996. http://scholar.lib.vt.edu/theses/available/etd-10022008-063026/.

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Bodez, Diane. "Etude des profils transcriptionnels myocardiques et sanguins du rejet aigu de greffe cardiaque." Thesis, Paris Est, 2017. http://www.theses.fr/2017PESC0009.

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La greffe cardiaque est le traitement ultime de l’insuffisance cardiaque. Le rejet aigu pose plusieurs problématiques, en particulier sa survenue imprévisible même sous traitement immunosuppresseur, et un diagnostic histologique qui nécessite des biopsies endomyocardiques (BEM) invasives répétées, et qui souffre de nombreuses limites. Le besoin de critères diagnostiques et prédictifs, idéalement non invasifs, nous a conduits à étudier le rejet aigu de greffe cardiaque sur le plan moléculaire. Nous avons caractérisé les profils d’expression génique (PEG) myocardiques et sanguins lors de différentes phases du rejet cellulaire (RC) et du rejet médié par les anticorps (RMA), par analyse sans a priori des transcriptomes sur puce à ADN. Par une première étude des PEG myocardiques menée sur une collection historique de BEM, nous avons montré la modification des PEG tissulaires lors du RC. Pour le même grade histologique, deux profils de RC aux degrés d’activation immunitaire différents ont été identifiés. De plus, les PEG myocardiques étaient modifiés dès un mois avant la survenue d’un RC, quand l’analyse histologique ne montrait encore aucune anomalie. Par une seconde étude conduite sur une collection prospective de BEM et échantillons sanguins, nous avons confirmé les résultats de la première étude, et de plus montré l’existence de modulations des PEG également dans le sang périphérique, aussi bien pendant un épisode de RC qu’un mois avant. Enfin pour la première fois la modulation tissulaire et périphérique des PEG a été montrée dans le RMA en transplantation cardiaque. L’existence de voies modulées dans les deux types de rejet devrait conduire à la recherche de biomarqueurs
Heart transplantation is the last treatment in case of terminal heart failure. Acute rejection after heart transplantation raises several issues due to its occurrence despite immunosuppressive therapies and the requirement of invasive and repeated endomyocardial biopsies (EMB) that have several histological grading limitations. The need of non-invasive diagnostic and predictive criteria led us to study the acute rejection of cardiac allograft using a molecular approach. We characterized myocardial and peripheral blood gene expression profiles (GEP) during acute cellular rejection (CR) and antibody-mediated rejection (AMR) by mean of microarray analyses. By a retrospective study conducted on a historical EMB collection, we first showed a strong immunologic modulation during CR. For the same CR histological grading, two transcriptional profiles were identified according to the inflammation level. Moreover, myocardial GEP modifications were observed one month before the occurrence of CR, while histological characteristics showed no abnormality. A second study conducted on a prospective collection of both EMB and peripheral blood samples confirmed the results obtained on EMB and showed peripheral blood GEP modulations during both CR and even one month earlier. Finally, we have also shown for the first time in heart transplantation, myocardial and peripheral GEP modulations in AMR. Identification of modulated pathways in both types of rejection should allow for the determination of rejection biomarkers
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Books on the topic "Cellular rejection"

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Yang, Chun-Ping. Cellular mechanisms of blood transfusion induced-tolerance preventing rejection of cardiac allografts. Manchester: University of Manchester, 1995.

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Smith, Douglas Matthew. Cellular pathways for rejection of class I MHC disparte allografts. 1988.

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(Editor), Lawrence Rosenberg, and William P. Duguid (Editor), eds. Cellular Inter-Relationships in the Pancreas: Implications for Islet Transplantation (Medical Intelligence Unit). Chapman & Hall, 1996.

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Book chapters on the topic "Cellular rejection"

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Mayo, George L., Andrew M. Posselt, Luis Campos, Barbara C. Deli, Sean P. Mayo, Clyde F. Barker, and Ali Naji. "Induction of Donor-Specific Tolerance by Intrathymic Cellular Transplantation." In Rejection and Tolerance, 227–44. Dordrecht: Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-011-0802-7_23.

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Rose, Marlene L., Sudhir Kushwaha, and Deirdre Cunningham. "Immunology of Cellular and Humoral Rejection after Cardiac Transplantation." In Cardiac Allograft Rejection, 21–47. Boston, MA: Springer US, 2001. http://dx.doi.org/10.1007/978-1-4615-1649-1_2.

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Moses, R. D., and H. Auchincloss. "Mechanism of Cellular Xenograft Rejection." In Xenotransplantation, 101–20. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-97323-9_7.

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Moses, R. D., and H. Auchincloss. "Mechanism of Cellular Xenograft Rejection." In Xenotransplantation, 140–74. Berlin, Heidelberg: Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60572-7_12.

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Neil, Desley A. H., and Alexandra J. Rice. "Pathology of Acute Cellular Rejection." In The Pathology of Cardiac Transplantation, 199–217. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-46386-5_13.

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Havryk, Adrian. "Cellular Rejection: Is it Still Relevant?" In Essentials in Lung Transplantation, 67–77. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-90933-2_6.

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Häyry, Pekka. "Cellular and molecular mechanisms of allograft rejection." In Organ Transplantation 1990, 5–9. Dordrecht: Springer Netherlands, 1991. http://dx.doi.org/10.1007/978-94-011-3386-9_2.

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Roden, Anja C., and Henry D. Tazelaar. "Pathology of Lung Rejection: Cellular and Humoral Mediated." In Lung Transplantation, 209–30. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-91184-7_13.

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Jastrzebska, Agnieszka, and Rafael Toro Sluzhenko. "Pattern Classification with Rejection Using Cellular Automata-Based Filtering." In Computer Information Systems and Industrial Management, 3–14. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-59105-6_1.

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Lease, Erika D., and Ganesh Raghu. "Management of Cellular and Humoral Rejection: Prevention, Diagnosis, and Treatment." In Lung Transplantation, 195–207. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-91184-7_12.

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Conference papers on the topic "Cellular rejection"

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Tanes, C., M. Brown, K. Bittinger, F. D. Bushman, J. D. Christie, R. G. Collman, and J. M. Diamond. "Microbial and Proteomic Signature of Acute Cellular Rejection in Lung Transplants." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a7454.

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Arthur, Lindsay, Dala Zakaria, and Kenneth Knecht. "Echocardiographic Predictors of Acute Cellular Rejection in Pediatric Heart Transplant Recipients." In AAP National Conference & Exhibition Meeting Abstracts. American Academy of Pediatrics, 2021. http://dx.doi.org/10.1542/peds.147.3_meetingabstract.347.

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Tsai, Ming-Da, Chien-Wei Tseng, Kuen-Jou Tsai, Shuja Andrabi, Pin-Cheng Huang, Federico Beffa, Yangjian Chen, and Bernard Tenbroek. "10.6 A 4G/5G Cellular Transmitter in 12nm FinFET with Harmonic Rejection." In 2020 IEEE International Solid- State Circuits Conference - (ISSCC). IEEE, 2020. http://dx.doi.org/10.1109/isscc19947.2020.9063145.

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Vasileva, A., D. Abelson, J. Matelski, J. Wu, E. deHaas, Q. Huang, R. Nadj, et al. "Airway Oscillometry for Early Detection of Acute Cellular Rejection Following Lung Transplantation." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a7576.

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Atat, Rachad, Muhammad Zeeshan Shakir, and Mohamed-Slim Alouini. "On the interference rejection capabilities of triangular antenna array for cellular base stations." In MELECON 2012 - 2012 16th IEEE Mediterranean Electrotechnical Conference. IEEE, 2012. http://dx.doi.org/10.1109/melcon.2012.6196375.

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Alsaghayer, A., A. Rabie, and P. S. Garcha. "Epidemiology of Acute Cellular Rejection (ACR) Post-Lung Transplant - A Single Center Experience." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a1779.

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Pewekar, Mihir Mangesh, Pranit Pravin Sandye, and Kiran Chaudhari. "Investigation of Non-Pneumatic Tires Based on Helical Hexagonal Cellular Structure." In ASME 2018 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/imece2018-87631.

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Non-pneumatic tires (NPTs) have drawn attention mainly due to low contact pressure and low rolling resistance due to use of hyper-elastic materials in their construction. In this paper, an attempt to innovate the conventional design of NPT with hexagonal honeycomb cellular structure is made by creating the boundary planar geometries of the tire, skew to each other at a certain angle. Adding to the functionality as a tire, this modified structure increases the performance of automobile components by rejection of heat through convection (forced) at the expense of engine power. The primary investigation includes study of the effects of variation in degree of skewness with the strength and flow of air through the tire. The flow parameters are computed for rotational case and the heat transfer is computed for flow over a brake disk. The secondary investigation consists of finding an optimum range of the degree of skewness. The validation for strength is computed through Finite Element Analysis. The fluid flow is computed through Computational Fluid Dynamics approach in ANSYS Fluent. This modified structure improves the aerodynamic condition near the brake rotor that increases the rate of heat rejection by forced convection from the brake rotor surface.
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Beck, Sungho, Jeong-Cheol Lee, Seungyup Yoo, Kyoohyun Lim, Hyosun Jung, Jaekyung Han, Munkyung Ahn, et al. "A Low-Power Cellular/PCS/WCDMA Direct-Conversion Transmitter with Enhanced VCO Remodulation Rejection." In 2006 IEEE Asian Solid-State Circuits Conference. IEEE, 2006. http://dx.doi.org/10.1109/asscc.2006.357857.

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Yousef, I., and F. C. Cordova. "Efficacy of Corticosteroids in the Treatment of Acute Cellular Rejection in Lung Transplant Patients." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a5124.

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Byeon, Chul Woo, Jeong Ho Lee, Dae Young Lee, Mee-Ran Kim, and Ju Ho Son. "A high linearity, image/LO-rejection I/Q up-conversion mixer for 5G cellular communications." In 2015 European Microwave Conference (EuMC 2015). IEEE, 2015. http://dx.doi.org/10.1109/eumc.2015.7345983.

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