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Books on the topic 'Cell surface antigen receptors'

Author: Grafiati

Published: 4 June 2021

Last updated: 7 February 2022

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1

Cell surface and differentiation. London: Chapman and Hall, 1990.

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2

Recognition receptors in biosensors. New York: Springer, 2010.

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3

Cell surface receptors: A short course on theory & methods. 3rd ed. New York: Springer, 2004.

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4

BLyS ligands and receptors. New York: Humana Press, 2010.

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5

Cell surface receptors: A short course on theory and methods. Boston: Nijhoff, 1986.

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6

Limbird, Lee E. Cell surface receptors: A short course on theory and methods. 2nd ed. Boston: Kluwer Academic Publishers, 1996.

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7

Limbird, Lee E. Cell Surface Receptors: A Short Course on Theory and Methods. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4757-1882-9.

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8

Limbird, Lee E. Cell Surface Receptors: A Short Course on Theory and Methods. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4613-1255-0.

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9

Van den Elsen, Peter J., 1951-. The human T-cell receptor repertoire and transplantation. New York: Springer Verlag, 1995.

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10

Z, Atassi M., and Abbott Laboratories, eds. Immunobiology of proteins and peptides IV: T-cell recognition and antigen presentation. New York: Plenum Press, 1987.

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11

Harvey, Amanda. Cancer cell signalling. Chichester, West Sussex: John Wiley & Sons, Inc., 2013.

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12

Gesellschaft für Topochemie und Elektronenmikroskopie der DDR. Verhandlungen. Surface receptors and cell function: Verhandlungen der Gesellschaft für Topochemie und Elektronenmikroskopie der DDR vom 21. bis 24. November 1984 in Bad Berka. Jena: G. Fischer, 1986.

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13

V, Švorčík, ed. Cell colonization control by physical & chemical modification of materials. New York: Nova Science Pub., 2008.

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14

Iismaa, Tiina P. G protein-coupled receptors. Austin: R.G. Landes Co., 1995.

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15

J, Biden Trevor, and Shine John, eds. G protein-coupled receptors. New York: Springer-Verlag, 1995.

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16

NATO Advanced Study Institute on Receptors, Membrane Transport, and Signal Transduction (1988 Island of Spetsai, Greece). Receptors, membrane transport, and signal transduction. Berlin: Springer-Verlag, 1989.

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17

Wu, Yongjian. Molecular analysis of the signaling and transport differences of the B cell antigen receptors of the IgM and IgD classes. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1997.

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18

Masaru, Taniguchi, Akira S, and Nakayama Toshinori, eds. The innate immune system: Strategies for disease control : proceedings of the Uehara Memorial Foundation Symposium on the Innate Immune System ..., held in Tokyo, Japan between 11 and 13 July 2005. Boston: Elsevier, 2005.

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19

Alharbi, Yousef, Manish S. Patankar, and Rebecca J. Whelan. Antibody-Based Therapy for Ovarian Cancer. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190248208.003.0006.

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With their role in connecting disease-associated antigens to the cellular immune response, antibodies hold considerable promise as therapeutic agents. This chapter discusses three classes of therapeutic antibodies that have been developed for use in ovarian cancer therapy. The first includes antibodies selected against tumor-associated antigens such as MUC16/CA125, mesothelin, epithelial cell adhesion molecule, and folate receptor α‎. Antibodies in the second class target proteins such as CTLA-4 and PD1 that act as immune response checkpoint receptors. The third class of antibodies target secreted factors that promote tumor growth: targets in this class include vascular endothelial growth factor, cytokines, and chemokines. The development of each of these is described. The chapter also discusses the complications presented by soluble antigens, which serve to limit the applicability of antigens (such as MUC16/CA125) that are both cell-surface associated and circulating and the prospects for the combination of antibody-based immunotherapy and chemotherapy.

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20

Marchalonis. Antigen-Specific T-Cell Receptors. CRC Press Inc, 1987.

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21

Marchalonis. Antigen-Specific T-Cell Receptors. CRC Press Inc, 1987.

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22

Christopher, Garcia K., ed. Cell surface receptors. San Diego: Academic Press, 2004.

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23

Cell Surface Receptors. Boston: Kluwer Academic Publishers, 2005. http://dx.doi.org/10.1007/b100572.

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24

Cell Surface Receptors. Elsevier, 2004. http://dx.doi.org/10.1016/s0065-3233(00)x0016-2.

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25

1945-, Mak Tak W., ed. The T-cell receptors. New York: Plenum Press, 1988.

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26

I, Bell John, Owen M. J, and Simpson Elizabeth Dr, eds. T cell receptors. Oxford: Oxford University Press, 1995.

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27

1940-, Marchalonis John J., ed. Antigen-specific T cell receptors and factors. Boca Raton, Fla: CRC Press, 1987.

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28

1932-, Haber Edgar, ed. Antigen binding molecules: Antibodies and T-cell receptors. San Diego: Academic Press, 1996.

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29

Cattran, Daniel C., and Heather N. Reich. Membranous glomerulonephritis. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0064_update_001.

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It has been clear for several decades from comparison with the rodent model disease Heymann nephritis that membranous glomerulonephritis (MGN) is an immune condition in which antibodies, usually autoantibodies, bind to targets on the surface of podocytes. However, the antigen in Heymann nephritis, megalin, is not present on human podocytes. The first potential antigen was identified by studying rare examples of maternal alloimmunization, leading to congenital membranous nephropathy in the infant caused by antibodies to neutral endopeptidase. More recently, the target of autoantibody formation in most patients with primary MGN has been identified to be the phospholipase A2 receptor, PLA2R. Genome-wide association studies identify predisposing genetic loci at HLADQ and at the locus encoding the autoantigen itself. So antibodies to at least two different molecular targets can cause MGN, and it seems likely that there may be other targets in secondary types of MGN, and possibly haptenized or otherwise modified molecules are implicated in drug- and toxin-induced MGN. Once antibodies are fixed, animal models and human observations suggest that complement is involved in mediating tissue damage. However, immunoglobulin G4, not thought to fix complement, is the predominant isotype in human MGN, and the mechanisms are not fully unravelled. Podocyte injury is known to cause proteinuria. In MGN, antibody fixation or cell damage may stimulate production of extracellular matrix to account for the increased GBM thickness with ‘podocyte type’ basement membrane collagen isoforms, and ultimately cell death and glomerulosclerosis.

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30

R, Oksenberg Jorge, ed. The antigen T cell receptor: Selected protocols and applications. New York: R.G. Landes, 1997.

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31

Michael, Conn P., ed. Receptors: Model systems and specific receptors. San Diego, Calif: Academic Press, 1993.

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32

Recombinant Cell Surface Receptors (Biotechnology Intelligence Unit). Academic Press, 1997.

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33

Alexander, D. J., N. Phin, and M. Zuckerman. Influenza. Edited by I. H. Brown. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0037.

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Influenza is a highly infectious, acute illness which has affected humans and animals since ancient times. Influenza viruses form the Orthomyxoviridae family and are grouped into types A, B, and C on the basis of the antigenic nature of the internal nucleocapsid or the matrix protein. Infl uenza A viruses infect a large variety of animal species, including humans, pigs, horses, sea mammals, and birds, occasionally producing devastating pandemics in humans, such as in 1918 when it has been estimated that between 50–100 million deaths occurred worldwide.There are two important viral surface glycoproteins, the haemagglutinin (HA) and neuraminidase (NA). The HA binds to sialic acid receptors on the membrane of host cells and is the primary antigen against which a host’s antibody response is targeted. The NA cleaves the sialic acid bond attaching new viral particles to the cell membrane of host cells allowing their release. The NA is also the target of the neuraminidase inhibitor class of antiviral agents that include oseltamivir and zanamivir and newer agents such as peramivir. Both these glycoproteins are important antigens for inducing protective immunity in the host and therefore show the greatest variation.Influenza A viruses are classified into 16 antigenically distinct HA (H1–16) and 9 NA subtypes (N1–9). Although viruses of relatively few subtype combinations have been isolated from mammalian species, all subtypes, in most combinations, have been isolated from birds. Each virus possesses one HA and one NA subtype.Last century, the sudden emergence of antigenically different strains in humans, termed antigenic shift, occurred on three occasions, 1918 (H1N1), 1957 (H2N2) and 1968 (H3N2), resulting in pandemics. The frequent epidemics that occur between the pandemics are as a result of gradual antigenic change in the prevalent virus, termed antigenic drift. Epidemics throughout the world occur in the human population due to infection with influenza A viruses, such as H1N1 and H3N2 subtypes, or with influenza B virus. Phylogenetic studies have led to the suggestion that aquatic birds that show no signs of disease could be the source of many influenza A viruses in other species. The 1918 H1N1 pandemic strain is thought to have arisen as a result of spontaneous mutations within an avian H1N1 virus. However, most pandemic strains, such as the 1957 H2N2, 1968 H3N2 and 2009 pandemic H1N1, are considered to have emerged by genetic re-assortment of the segmented RNA genome of the virus, with the avian and human influenza A viruses infecting the same host.Influenza viruses do not pass readily between humans and birds but transmission between humans and other animals has been demonstrated. This has led to the suggestion that the proposed reassortment of human and avian influenza viruses takes place in an intermediate animal with subsequent infection of the human population. Pigs have been considered the leading contender for the role of intermediary because they may serve as hosts for productive infections of both avian and human viruses, and there is good evidence that they have been involved in interspecies transmission of influenza viruses; particularly the spread of H1N1 viruses to humans. Apart from public health measures related to the rapid identification of cases and isolation. The main control measures for influenza virus infections in human populations involves immunization and antiviral prophylaxis or treatment.

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34

Barros, Rodrigo José Saraiva de, Tereza Cristina de Brito Azevedo, Carla de Castro Sant’Anna, Marianne Rodrigues Fernandes, Leticia Martins Lamarão, and Rommel Mario Rodríguez Burbano. Grupos sanguíneos e anticorpos anti-eritrocitários de importância transfusional. Brazil Publishing, 2020. http://dx.doi.org/10.31012/978-65-5861-112-7.

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Immunohematology is an area dedicated to the study of the interactions of the immune system and blood cells in transfusion practice. Blood transfusion is a therapeutic technique that has been widely used since the 17th century. The transfusion medicine aims to repair the pathological needs of blood components in the living organism, be it red blood cells, plasma, platelets, clotting factors, among others. Despite being a therapeutic means, transfusion of blood components can be considered at risk because it is a biological material and due to the transfusion immunological reactions that can be caused during or after the moment of transfusion. In the surface structure of red blood cells, numerous molecules of a protein, glycoprotein or glycolipid nature are found, which are also called membrane antigens that make up structures and perform transport functions, as receptors, as adhesion, enzymatic and / or complement regulatory molecules. The formation of these antigens occurs by an approximate amount of 39 genes involved in their production, of which 282 different antigens are organized in more than 30 blood group systems. This antigenic diversity is a major cause of the formation of irregular anti-erythrocyte antibodies. Therefore, with the increase in blood transfusions in surgeries, transplants and clinical treatment of cancer and other chronic diseases, a significant increase in the occurrence of alloimmunizations in polytransfused patients began to be observed. Such biological phenomena motivated us to carry out this study and the antigenic diversity motivated us to elaborate this small compendium where we also describe the main blood groups.

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35

Zourob, Mohammed. Recognition Receptors in Biosensors. Springer, 2016.

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36

E, Reif Arnold, and Schlesinger Michael 1932-, eds. Cell surface antigen Thy-1: Immunology, neurology, and therapeutic applications. New York: Dekker, 1989.

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37

Leung. Superantigens: Molecular Biology, Immunology, and Relevance to Human Disease. CRC, 1997.

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38

Leung, Donald Y. M., 1949-, Huber Brigitte T, and Schlievert Patrick M. 1949-, eds. Superantigens: Molecular biology, immunology, and relevance to human disease. New York: M. Dekker, 1997.

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39

Receptors: Model Systems and Specific Receptors (Methods in Neurosciences, Vol 11). Academic Pr, 1993.

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40

C, Sealfon Stuart, ed. Receptor molecular biology. San Diego: Academic Press, 1995.

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41

Ph, D. K. Christopher Garcia. Cell Surface Receptors, Volume 68 (Advances in Protein Chemistry). Academic Press, 2004.

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42

Ph.D, K. Christopher Garcia. Cell Surface Receptors, Volume 68 (Advances in Protein Chemistry). Academic Press, 2004.

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43

(Editor), Edgar Haber, Frederic M. Richards (Series Editor), David S. Eisenberg (Series Editor), and Peter S. Kim (Series Editor), eds. Advances in Protein Chemistry, Volume 49: Antigen Binding Molecules and T-Cell Receptors (Advances in Protein Chemistry). Academic Press, 1996.

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44

Cell Surface Receptors:: A Short Course on Theory and Methods. 2nd ed. Springer, 1995.

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45

Limbird, Lee E. Cell Surface Receptors:: A Short Course on Theory and Methods. Springer, 1985.

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46

Limbird, Lee E. Cell Surface Receptors: A Short Course on Theory and Methods. Springer, 2013.

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47

Nanomagnetic Actuation of Cell Surface Receptors: Basic Principles and Applications. Taylor & Francis Group, 2018.

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48

Limbird, Lee E. E. Cell Surface Receptors: A Short Course on Theory and Methods. Springer, 2010.

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49

Reif, Arnold E. Cell Surface Antigen Thy-1: Immunology, Neurology, and Therapeutic Applications (Immunology Series). Marcel Dekker, 1989.

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50

G, Trist D., ed. Receptor classification: The integration of operational, structural, and transductional information. New York, N.Y: New York Academy of Sciences, 1997.

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