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Books on the topic 'Cell niche'

Author: Grafiati

Published: 24 April 2022

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1

Turksen, Kursad, ed. Stem Cell Niche. New York, NY: Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-9508-0.

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Turksen, Kursad, ed. Stem Cell Niche. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-508-8.

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3

Roy, Krishnendu, ed. Biomaterials as Stem Cell Niche. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-13893-5.

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4

Turksen, Kursad, ed. Biology in Stem Cell Niche. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-21702-4.

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5

Turksen, Kursad, ed. Tissue-Specific Stem Cell Niche. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-21705-5.

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6

Schaible, Ulrich E., and Haas Albert. Intracellular niches of microbes: A pathogens guide through the host cell. Weinheim: Wiley-VCH, 2009.

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7

Rice, Jerry M. The bone marrow niche, stem cells, and leukemia: Impact of drugs, chemicals, and the environment. Hoboken, NJ: published on behalf of the New York Academy of Sciences by Wiley Subscription Services, Inc., 2014.

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8

Sugao no Yamanaka Shin'ya: Kisha ga otta 2500-nichi. Kyōto-shi: Nakanishiya Shuppan, 2013.

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9

E.C. Photovoltaic Solar Energy Conference (13th 1995 Nice, France). Thirteenth European Photovoltaic Solar Energy Conference: Proceedings of the International Conference, held at Nice, France, 23-27 October 1995. Bedford, UK: H.S. Stephens & Associates, 1995.

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10

Kagaku Gijutsu no Mirai o Tenbōsuru Senryaku Wākushoppu (2004 Tokyo, Japan). Kagaku Gijutsu no Mirai o Tenbōsuru Wākushoppu: Suiso enerugī shisutemu no kanōsei to kadai (Heisei 16-nen 4-gatsu 10-nichi--11nichi kaisai) : hōkokusho. Tōkyō: Kagaku Gijutsu Shinkō Kikō Kenkyū Kaihatsu Senryaku Sentā Inoue Jōseki Ferō Gurūpu, 2004.

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11

Kagaku, Gijutsu no Mirai o. Tenbōsuru Senryaku Wākushoppu (2004 Tokyo Japan). Kagaku Gijutsu no Mirai o Tenbōsuru Wākushoppu: Suiso enerugī shisutemu no kanōsei to kadai (Heisei 16-nen 4-gatsu 10-nichi--11nichi kaisai) : hōkokusho. Tōkyō: Kagaku Gijutsu Shinkō Kikō Kenkyū Kaihatsu Senryaku Sentā Inoue Jōseki Ferō Gurūpu, 2004.

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12

Stem Cell Niche Methods And Protocols. Humana Press Inc., 2013.

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13

Intestinal Stem Cell Niche. Elsevier, 2018. http://dx.doi.org/10.1016/s2468-5097(18)x0002-5.

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14

Epidermal Stem Cell Niche. Elsevier, 2019. http://dx.doi.org/10.1016/s2468-5097(19)x0002-0.

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15

Bonnet, Dominique. Hematopoietic Stem Cell Niche. Elsevier Science & Technology, 2017.

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16

Nilsson, Susie Prof. Stem Cell Niche During Ageing. Elsevier Science & Technology, 2020.

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17

Nilsson, Susie Prof. Stem Cell Niche During Ageing. Elsevier Science & Technology Books, 2020.

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18

Roy, Krishnendu. Biomaterials as Stem Cell Niche. Springer, 2012.

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19

Biomaterials As Stem Cell Niche. Springer, 2010.

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20

Turksen, Kursad. Biology in Stem Cell Niche. Springer, 2015.

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21

The Cancer Stem Cell Niche. Elsevier, 2021. http://dx.doi.org/10.1016/s2468-5097(21)x0002-4.

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22

The Stem Cell Niche during Ageing. Elsevier, 2020. http://dx.doi.org/10.1016/s2468-5097(20)x0002-9.

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23

Kawaguchi, Takanori, ed. Cancer Metastasis and Cancer Stem Cell/Niche. BENTHAM SCIENCE PUBLISHERS, 2016. http://dx.doi.org/10.2174/97816810834761160101.

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24

Turksen, Kursad. Cell Biology and Translational Medicine, Volume 6 : Stem Cells: Their Heterogeneity, Niche and Regenerative Potential. Springer, 2020.

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25

Rammah, Mayyasa, Francesca Rochais, and Robert G. Kelly. Incorporation of myocardial progenitors at the arterial pole of the heart. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, José Luis de la Pompa, David Sedmera, Cristina Basso, and Deborah Henderson. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0007.

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Abstract:

The arterial pole of the heart is a hotspot for life-threatening forms of congenital heart defects (CHDs). It is formed by progressive addition of myocardium from epithelial progenitor cells in the second heart field (SHF). SHF cells contribute successively to the right ventricle and proximal and distal outflow tract myocardial walls which, after neural crest influx and cardiac septation, give rise to myocardium at the base of the aorta and pulmonary trunk. SHF cells are characterized by continued proliferation and differentiation delay controlled by an array of transcriptional regulators and signalling pathways which define the SHF progenitor cell niche in pharyngeal mesoderm. Failure of normal SHF deployment leads to a shortened outflow tract and failure of ventriculo-arterial alignment, resulting in a spectrum of conotruncal CHD. We discuss the origins of the SHF in cardiopharyngeal mesoderm and focus on the mechanisms driving SHF deployment, summarizing current understanding of critical signalling pathways and transcription factors.

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26

Wislet-Gendebien, Sabine, ed. Adult Stem Cell Niches. InTech, 2014. http://dx.doi.org/10.5772/57290.

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27

Biology and Engineering of Stem Cell Niches. Elsevier Science & Technology Books, 2017.

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28

Haas, Albert, and Ulrich E. Schaible. Intracellular Niches of Microbes: A Microbes Guide Through the Host Cell. Wiley & Sons, Incorporated, John, 2009.

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29

Intracellular Niches of Microbes: A Microbes Guide Through the Host Cell. Wiley-Interscience, 2009.

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30

Isaacs, John D., and Philip M. Brown. Rituximab and abatacept. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0083.

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Two biologics that target cells have been licensed to treat rheumatoid arthritis (RA). Rituximab is a chimeric monoclonal antibody (mAb) against CD20 that depletes B cells; abatacept is a soluble form of CTLA-4 that blocks costimulation and interferes with T-cell function. Both drugs alleviate signs and symptoms of RA and have been shown to retard radiographic progression. Rituximab is licensed for use following failure of tumour necrosis factor (TNF) blockade whereas abatacept's licence permits it use as a first-line biologic. In the United Kingdom, however, the National Institute for Health and Clinical Excellence (NICE) restricts the use of abatacept to patients who develop adverse effects with rituximab or in whom rituximab is contraindicated. As with other biologics, the use of either drug is associated with an enhanced risk of serious infections; additionally, rituximab in particular can cause infusion reactions, requiring prophylaxis. By targeting cells that are central to RA pathogenesis, these drugs provide important additional therapeutic options for patients with RA.

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31

Have a Nice DNA (Enjoy Your Cells, 3). Cold Spring Harbor Laboratory Press, 2002.

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32

Have a Nice DNA (Enjoy Your Cells, 3). Cold Spring Harbor Laboratory Press, 2002.

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33

Mantel, Gerhard. Cello üben. Eine Methodik des Übens nicht nur für Streicher. Schott, Mainz, 1999.

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34

Lattanzi, Wanda, and Maria Concetta Geloso, eds. Crosstalk between the Osteogenic and Neurogenic Stem Cell Niches: How Far are They from Each Other? Frontiers Media SA, 2016. http://dx.doi.org/10.3389/978-2-88919-777-4.

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35

(Editor), W. Freisesleben, W. Palz (Editor), H. A. Ossenbrink (Editor), and P. Helm (Editor), eds. Proceedings of the Thirteenth European Photovoltaic Solar Energy Conference: Nice, France, 23-27 October 1995. James & James Science Publishers, 1996.

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36

Hube, Bernhard, and Oliver Kurzai. Candida species. Edited by Christopher C. Kibbler, Richard Barton, Neil A. R. Gow, Susan Howell, Donna M. MacCallum, and Rohini J. Manuel. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755388.003.0011.

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Most pathogenic Candida species are members of the microbiota, but also cause superficial or invasive infections. C. albicans is predominant, followed by C. glabrata, C. parapsilosis, and C. tropicalis. C. albicans is polymorphic and grows as yeast, pseudohyphae, or hyphae. The cell wall has multiple functions in pathogenesis. Metabolism and nutrient up-take strategies facilitate growth in multiple niches within the host. Drug resistance is an intrinsic property of C. glabrata and C. krusei, but can be developed by C. albicans and other Candida species during antifungal therapy. Pathogenicity mechanisms include host cell attachment, invasion, and destructive activities; immune evasion; and biofilm production. A disbalanced microbiota and impaired immunity favour superficial infections, and disturbance of the mucosal barriers, together with compromised immunity, enables Candida to invade the human bloodstream and cause invasive infection. Even with antifungal therapy (e.g. azoles or echinocandins), disseminated candidiasis has a high mortality (40–50%).

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37

Lennon, Rachel, and Neil Turner. The molecular basis of glomerular basement membrane disorders. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0320_update_001.

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The glomerular basement membrane (GBM) is a condensed network of extracellular matrix molecules which provides a scaffold and niche to support the function of the overlying glomerular cells. Within the glomerulus, the GBM separates the fenestrated endothelial cells, which line capillary walls from the epithelial cells or podocytes, which cover the outer aspect of the capillaries. In common with basement membranes throughout the body, the GBM contains core components including collagen IV, laminins, nidogens, and heparan sulphate proteoglycans. However, specific isoforms of these proteins are required to maintain the integrity of the glomerular filtration barrier.Across the spectrum of glomerular disease there is alteration in glomerular extracellular matrix (ECM) and a number of histological patterns are recognized. The GBM can be thickened, expanded, split, and irregular; the mesangial matrix may be expanded and glomerulosclerosis represents a widespread accumulation of ECM proteins associated with loss of glomerular function. Whilst histological patterns may follow a sequence or provide diagnostic clues, there remains limited understanding about the mechanisms of ECM regulation and how this tight control is lost in glomerular disease. Monogenic disorders of the GBM including Alport and Pierson syndromes have highlighted the importance of both collagen IV and laminin isoforms and these observations provide important insights into mechanisms of glomerular disease.

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38

Gifting, Unique. Address Book with Large Spaces to Write-In / Nice Leaves Design / Pocket Size / Perfect for Keeping Private Information: Addresses, Email, Cell, Home and Work/Fax Phone Number, Birthday Dates / Alphabetical Tabs for Fast Searching. Independently Published, 2020.

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39

Jolles, G. New Trends in Genetic Risk Assessment: Based on the Proceedings of the Fifth International Round Table of the Rhone-Poulenc Sante Foundation, Nice 1. Academic Press, 1989.

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