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Relevant bibliographies by topics / Cell motilty

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Journal articles

Academic literature on the topic 'Cell motilty'

Author: Grafiati

Published: 14 March 2023

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Journal articles on the topic "Cell motilty"

1

Schmidt-Tanguy, Aline, Annette Romanski, Mathilde Hunault-Berger, and Oliver G. Ottmann. "Different Roles of Two Autotaxin Isoforms in Proliferation, Migration and Adhesion in the Non-Mutational Tyrosine Kinase Inhibitor Resistant Acute Lymphoblastic Leukemia Cell Line SupB15." Blood 112, no. 11 (2008): 1915. http://dx.doi.org/10.1182/blood.v112.11.1915.1915.

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Abstract The Bcr-Abl oncogene is present in 30–40% of adult patients with acute lymphoblastic leukemia (ALL). The Abl kinase inhibitor imatinib-based therapy has become standard for this subset ALL. Acquired resistance to imatinib occurs frequently and is associated with mutations in the tyrosine kinase domain (TKD) approximately in about 80% of patients. In contrast, TKD mutations are uncommon in primary imatinib resistance which appears to be multifactorial, although the underlying mechanisms have been incompletely elucidated. We have established a Ph+ cell line for the analysis of non-mutat

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2

An, Xingyue, Gabrielle Romain, Melisa Martinez-Paniagua, et al. "CAR+ T cell anti-tumor efficacy revealed by multi-dimensional single-cell profiling." Journal of Immunology 202, no. 1_Supplement (2019): 134.2. http://dx.doi.org/10.4049/jimmunol.202.supp.134.2.

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Abstract T cells engineered to express chimeric antigen receptor (CAR) targeting CD19 have shown promising clinical responses in patients with certain hematologic malignancies, however, it is desirable to be able to enrich cells with enhanced anti-tumor efficacy prior to infusion. We utilized a suite of high-throughput technologies with single-cell resolution, including Timelapse Imaging Microscopy In Nanowell Grids (TIMING) that integrates cytokine profiling to reveal that persistent motility of CD19- specific CAR T cells is correlated to desirable polyfunctionality (elimination of tumor cell

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3

Cramer, Louise P., Timothy J. Mitchison, and Julie A. Theriot. "Actin-dependent motile forces and cell motility." Current Opinion in Cell Biology 6, no. 1 (1994): 82–86. http://dx.doi.org/10.1016/0955-0674(94)90120-1.

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4

Murakami, Shinya, Yo Otsuka, Manabu Sugimoto, and Toshiyuki Mitsui. "3H1010 Controlled cell migration with ultrasound(Cell Biology III:Cytoskeleton & Motility,Oral Presentation)." Seibutsu Butsuri 52, supplement (2012): S70. http://dx.doi.org/10.2142/biophys.52.s70_4.

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5

Kolobov, A. V., A. A. Polezhaev, and G. I. Solyanik. "The Role of Cell Motility in Metastatic Cell Dominance Phenomenon: Analysis by a Mathematical Model." Journal of Theoretical Medicine 3, no. 1 (2000): 63–77. http://dx.doi.org/10.1080/10273660008833065.

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Metastasis is the outcome of several selective sequential steps where one of the first and necessary steps is the progressive overgrowth or dominance of a small number of metastatic cells in a tumour. In spite of numerous experimental investigations concerning the growth advantage of metastatic cells, the mechanisms resulting in their dominance are still unknown. Metastatic cell overgrowth occurs even if doubling time of the metastatic subpopulation is shorter than that of all others subpopulations in a heterogeneous tumour. In order to examine the hypothesis that under conditions of competiti

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6

Rezvan, Ali, Gabrielle Romain, Mohsen Fathi, et al. "Multiomic dynamic single-cell profiling of CAR T cell populations associated with efficacy." Journal of Immunology 208, no. 1_Supplement (2022): 54.18. http://dx.doi.org/10.4049/jimmunol.208.supp.54.18.

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Abstract T-cell therapy with specificity redirected through chimeric antigen receptors (CARs) has shown efficacy for the treatment of hematologic malignancies. Although treatment with CAR T cell can result in high response rates, the properties of the cells that comprise the cellular infusion product, associated with clinical benefit are incompletely understood. We utilized a suite of high-throughput single-cell assays including single-cell RNA-sequencing (scRNA-seq); confocal microscopy; and Timelapse Imaging Microscopy In Nanowell Grids (TIMING). TIMING profiling of a cohort of 16 patients s

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7

Marth, W., S. Praetorius, and A. Voigt. "A mechanism for cell motility by active polar gels." Journal of The Royal Society Interface 12, no. 107 (2015): 20150161. http://dx.doi.org/10.1098/rsif.2015.0161.

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We analyse a generic motility model, with the motility mechanism arising by contractile stress due to the interaction of myosin and actin. A hydrodynamic active polar gel theory is used to model the cytoplasm of a cell and is combined with a Helfrich-type model to account for membrane properties. The overall model allows consideration of the motility without the necessity for local adhesion. Besides a detailed numerical approach together with convergence studies for the highly nonlinear free boundary problem, we also compare the induced flow field of the motile cell with that of classical squi

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8

Breier, Rebekka E., Cristian C. Lalescu, Devin Waas, Michael Wilczek, and Marco G. Mazza. "Emergence of phytoplankton patchiness at small scales in mild turbulence." Proceedings of the National Academy of Sciences 115, no. 48 (2018): 12112–17. http://dx.doi.org/10.1073/pnas.1808711115.

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Phytoplankton often encounter turbulence in their habitat. As most toxic phytoplankton species are motile, resolving the interplay of motility and turbulence has fundamental repercussions on our understanding of their own ecology and of the entire ecosystems they inhabit. The spatial distribution of motile phytoplankton cells exhibits patchiness at distances of decimeter to millimeter scales for numerous species with different motility strategies. The explanation of this general phenomenon remains challenging. Furthermore, hydrodynamic cell–cell interactions, which grow more relevant as the de

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9

Alexandre, Gladys. "Chemotaxis Control of Transient Cell Aggregation." Journal of Bacteriology 197, no. 20 (2015): 3230–37. http://dx.doi.org/10.1128/jb.00121-15.

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Chemotaxis affords motile cells the ability to rapidly respond to environmental challenges by navigating cells to niches favoring growth. Such a property results from the activities of dedicated signal transduction systems on the motility apparatus, such as flagella, type IV pili, and gliding machineries. Once cells have reached a niche with favorable conditions, they often stop moving and aggregate into complex communities termed biofilms. An intermediate and reversible stage that precedes commitment to permanent adhesion often includes transient cell-cell contacts between motile cells. Chemo

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10

Cozzolino, Mauro, Venturina Stagni, Laura Spinardi, et al. "p120 Catenin Is Required for Growth Factor–dependent Cell Motility and Scattering in Epithelial Cells." Molecular Biology of the Cell 14, no. 5 (2003): 1964–77. http://dx.doi.org/10.1091/mbc.e02-08-0469.

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Cadherin-mediated cell–cell adhesion is dynamically modulated during epithelial–mesenchymal transition triggered by activation of receptor tyrosine kinases (RTK) in epithelial cells. Several cadherin-binding proteins have been identified that control cell–cell adhesion. However, the mechanisms by which intercellular adhesion and cell motility are coregulated are still unknown. Here, we delineate a hitherto uncharted cooperation between RTKs, RhoA GTPase, and p120 catenin in instructing a motile behavior to epithelial cells. We found that expression of an N-terminus–deleted p120 catenin in a va

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