Books on the topic 'Cell membranes Effect of drugs on'

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1

Alcohol and biological membranes. New York: Guilford Press, 1985.

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2

C, Aloia Roland, Curtain Cyril C, and Gordon Larry M, eds. Drug and anesthetic effects on membrane structure and function. New York: Wiley-Liss, 1991.

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3

C, Srivastava R. Surface activity in drug action. Amsterdam: Elsevier, 2005.

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4

Kasumov, Kh M. Molekuli͡a︡rnyĭ mekhanizm vzaimodeĭstvii͡a︡ polienovykh antibiotikov s lipidnymi membranami. Baku: "Ėlm", 1986.

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5

Robert, Lu D., and Øie Svein, eds. Cellular drug delivery: Principles and practice. Totowa, N.J: Humana Press, 2004.

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6

Polivoda, B. I. Biofizicheskie aspekty radiat͡s︡ionnogo porazhenii͡a︡ biomembran. Moskva: Ėnergoatomizdat, 1990.

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7

Sungurov, A. I͡U. Radiobiologii͡a kletochnoĭ poverkhnosti. Moskva: Akademii͡a nauk SSSR, Vses. in-t nauch. i tekhn. informat͡sii, 1988.

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8

Ryskulova, S. T. Radiat͡s︡ionnai͡a︡ biologii͡a︡ plazmaticheskikh membran. Moskva: "Ėnergoatomizdat", 1986.

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9

International School of Electromagnetic Fields and Biomembranes (1st 1986 Pleven, Bulgaria). Electromagnetic fields and biomembranes. New York: Plenum Press, 1988.

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10

E, Steele Vernon, ed. Cellular and molecular targets for chemoprevention. Boca Raton, Fla: CRC Press, 1992.

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11

Dockery, Dee. T cell responses to OspA, a candidate Lyme Disease vaccine. [New Haven, Conn: s.n.], 1993.

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12

1927-, Segawa Tomio, ed. Physiology and pharmacology of transmembrane signalling: Proceedings of the Uehara Memorial Foundation Symposium on the Mechanism of Transmembrane Signalling, Tokyo, Japan May 12-14, 1988. Amsterdam: Excerpta Medica, 1989.

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13

Filaretova, L. P. (Li︠u︡dmila Pavlovna) and Takeuchi K. (Koji), eds. Cell/tissue injury and cytoprotection/organoprotection in the gastrointestinal tract: Mechanisms, prevention, and treatment. Basel: Karger, 2012.

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14

M, Kasumov Kh. Struktura i membrannai︠a︡ funkt︠s︡ii︠a︡ polienovykh makrolidnykh antibiotikov. Moskva: Nauka, 2009.

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15

MD, Giordano Antonio, and Soprano Kenneth J, eds. Cell cycle inhibitors in cancer therapy: Current strategies. Totowa, N.J: Humana Press, 2003.

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16

Dr, Wiese Michael, ed. Drug-membrane interactions: Analysis, drug distribution, modeling. Weinheim: Wiley-VCH, 2002.

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17

C, Berman Mervyn, Gevers Wieland, Opie Lionel H, and International Union of Biochemistry, eds. Membranes and muscle: Proceedings of an international symposium, Cape Town, Republic of South Africa, March 18-21, 1985. Oxford, U.K: Published for the ICSU Press by IRL Press, 1985.

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18

Jean-François, Bach, ed. T-cell-directed immunointervention. Oxford [England]: Blackwell Scientific Publications, 1993.

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19

1931-, Chambers P. L., Tuomisto Jouko, and Chambers C. M, eds. Toxic interfaces of neurones, smoke, and genes: Proceedings of the European Society of Toxicology Meeting held in Kuopio, June 16-19, 1985. Berlin: Springer-Verlag, 1986.

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20

Coșofreț, Vasile V. Pharmaceutical applications of membrane sensors. Boca Raton: CRC Press, 1992.

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21

The membrane hypothesis of aging. Boca Raton: CRC Press, 1994.

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22

Silvio, Gutkind J., ed. Signaling networks and cell cycle control: The molecular basis of cancer and other diseases. Totowa, N.J: Humana Press, 2000.

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23

Ėĭdus, L. Kh. Membrannyĭ mekhanizm biologicheskogo deĭstvii͡a︡ malykh doz: Novyĭ vzgli͡a︡d na problemu. Moskva: In-t teoreticheskoĭ i ėksperimentalʹnoĭ biofiziki Rossiĭskoĭ akademii nauk, 2001.

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24

Chemokine receptors as drug targets. Weinheim, Germany: Wiley-VCH, 2011.

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25

K, Sen Amar, and Lee Tyrone, eds. Receptors and ligands in psychiatry. Cambridge [England]: Cambridge University Press, 1988.

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26

Ruth, Porter, Bock Gregory, Clark Sarah, Ciba Foundation, and Symposium on Depression, Antidepressants, and Receptor Sensitivity (1985 : Ciba Foundation), eds. Antidepressants and receptor function. Chichester: Wiley, 1986.

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27

Wood, W. Gibson, Grace Y. Sun, Christer Alling, Ivan Diamond, and Steven W. Leslie. Alcohol, Cell Membranes, and Signal Transduction in Brain. Springer, 2012.

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28

Christer, Alling, and Marcus Wallenberg Symposium on Alcohol, Cell Membranes, and Signal Transduction in Brain (1993 : Lund, Sweden), eds. Alcohol, cell membranes, and signal transduction in brain. New York: Plenum Press, 1993.

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29

Wood, W. Gibson, Grace Y. Sun, Christer Alling, Ivan Diamond, and Steven W. Leslie. Alcohol, Cell Membranes, and Signal Transduction in Brain. Springer London, Limited, 2012.

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30

Cornell, Svante. Membrane Structure in Disease and Drug Therapy. Taylor & Francis Group, 2000.

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31

Cornell, Svante. Membrane Structure in Disease and Drug Therapy. Taylor & Francis Group, 2000.

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32

Cornell, Svante. Membrane Structure in Disease and Drug Therapy. Taylor & Francis Group, 2000.

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33

Cornell, Svante. Membrane Structure in Disease and Drug Therapy. Taylor & Francis Group, 2000.

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34

Cornell, Svante. Membrane Structure in Disease and Drug Therapy. Taylor & Francis Group, 2000.

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35

1935-, Strata Piergiorgio, Carbone E. 1948-, and European Neuroscience Association Meeting, eds. Mg²⁺ and excitable membranes. Berlin: Springer-Verlag, 1991.

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36

Zimmer. Membrane Structure in Disease and Drug Therapy. CRC, 2000.

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37

Alois, Roland C., and Cyril C. Curtain. Drug and Anesthetic Effects on Membrane Structure and Function (Advances in Membrane Fluidity, Vol 5). John Wiley & Sons, 1991.

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38

T, Frangopol P., Morariu V. V, and Institutul Central de Fizicǎ (Romania), eds. Seminar on some Romanian original drugs, procaine-based drugs, Gerovital H₃, and Aslavital, Trofopar and Boicil: Analytical methods and effects on cell membranes : Bucharest-Măgurele, November 27, 1984. Bucharest: Central Institute of Physics, 1985.

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39

R.C. C. Srivastava (Editor) and A.N. N. Nagappa (Editor), eds. Surface Activity in Drug Action, Volume 21 (Studies in Interface Science). Elsevier Science, 2005.

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40

Membrane Structure in Disease and Drug Therapy. New York: Marcel Dekker, Inc., 2003.

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41

R, Watson Ronald, ed. Alcohol and neurobiology: Receptors, membranes, and channels. Boca Raton: CRC Press, 1992.

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42

Hopkins, Philip M. Neuromuscular physiology in anaesthetic practice. Edited by Jonathan G. Hardman. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0007.

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The pharmacological interventions that constitute general anaesthesia are targeted at producing unconsciousness and an immobile patient even in response to noxious stimuli. Surgical anaesthesia also requires skeletal muscle relaxation, the degree of which depends on the site and nature of the surgical procedure. The anaesthetist therefore needs an advanced level of knowledge and understanding of the function of nerves, synapses, and muscle in order to understand, from first principles, how the drugs they use every day mediate their effects. Nerves and muscle cells are termed excitable cells because the electrical potential across their cell membranes (membrane potential) can be rapidly and profoundly altered because of the presence of specialized ion channels. Some drugs, such as local anaesthetics, act on ion channels involved in nerve conduction while many others act on synaptic transmission, the neurochemical communication between neurons or between a neuron and its effector organ. The neuromuscular junction is a synapse of specific interest to anaesthetists because it is the site of action of neuromuscular blocking drugs. This chapter covers the fundamentals of cellular electrophysiology, structure and function of key ion channels, and the physiology of nerves, synapses, and skeletal muscle.
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43

Woywodt, Alexander, and Diana Chiu. Drug-induced and toxic glomerulopathies. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0082.

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Glomerulopathies induced by particular exogenous compounds or molecules include those attributable to toxicity, and those caused by inducing an immune or autoimmune response. Tubules are more commonly the target of toxicity as they absorb and concentrate components of filtrate. Damage to endothelial cells may account for thrombotic microangiopathy in response to calcineurin inhibitors. Endothelial cells are also likely to be the target in drug-induced small vessel vasculitis. Toxicity to podocytes accounts for focal segmental glomerulosclerosis caused by pamidronate and other agents. Chloroquine can cause a remarkable pseudo-storage disorder with inclusions in podocytes that resemble those seen in Fabry disease. The mechanism by which drugs cause minimal change disease, another podocyte disorder, is not known. Membranous nephropathy may be caused by exposure to gold, mercury, and some other drugs; this is antibody mediated and presumably the targets are altered podocyte surface molecules. Inhibitors of the mammalian target of rapamycin (mTOR) cause proteinuria, possibly through effects on vascular endothelial growth factor, inhibitors of which are associated with not only proteinuria (an expected podocyte effect) but also thrombotic microangiopathy (endothelial cell effect). This latter may be through disturbing podocyte-endothelium cross-signaling.
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44

Kurachi, Y., M. Mishina, and M. Endo. Pharmacology of Ionic Channel Function: Activators and Inhibitors. Springer London, Limited, 2012.

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45

Kurachi, Y., Masayoshi Mishina, and M. Endo. Pharmacology of Ionic Channel Function: Activators and Inhibitors. Springer Berlin / Heidelberg, 2012.

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46

(Editor), M. Endo, Y. Kurachi (Editor), and M. Mishina (Editor), eds. Pharmacology of Ionic Channel Function: Activators and Inhibitors (Handbook of Experimental Pharmacology). Springer, 2000.

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47

Cellular Drug Delivery: Principles and Practice. Humana Press, 2004.

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48

Montgomery, Erwin B. Principles of Electrophysiology. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190259600.003.0003.

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In many ways, post-operative DBS programming is “prescribing electricity” in much the same sense as “prescribing medications.” The principles of pharmacokinetics and pharmacodynamics that guide the rational use of medications find parallels in DBS. Many drugs have their effect by binding to ligand-gated channels, particularly channels that control the flow of electrical charges, in the form of ions across the cell membrane of the neuron in the soma. The binding of drugs to receptors can open the receptor to approximate the normal opening by endogenous neurotransmitters, or to block the channel from opening when endogenous neurotransmitters are released. In the case of DBS, the electrical charges manipulated in the nervous system similarly affect neuronal membrane channels; however, these initially and primarily are voltage gated ionic conductance channels, which are described in detail in this chapter.
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49

Peter, Eyer, ed. Metabolic aspects of cell toxicity. Mannheim: Wissenschaftsverlag, 1994.

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50

Cell Survival Programs And Ischemiareperfusion Hormesis Preconditioning And Cardioprotection. Biota Publishing, 2013.

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