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Books on the topic 'Cell cycle protein'

Author: Grafiati

Published: 4 June 2021

Last updated: 20 February 2023

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1

Pontus, Aspenstrøm, ed. The pombe Cdc 15 homology proteins. Austin, Tex: Landes Bioscience, 2009.

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2

L, Chen K., ed. Progress in cell cycle control research. New York: Nova Science Publishers, 2008.

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3

1949-, Lee R. C., Despa Florin, Hamann Kimm Jon, and New York Academy of Sciences., eds. Cell injury: Mechanisms, responses, and repair. New York, N.Y: New York Academy of Sciences, 2005.

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4

H, Leroy Nathan, and Fournier Noah T, eds. Cell cycle control: New research. New York: Nova Science Publishers, 2008.

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5

Calcium, cell cycles, and cancer. Boca Raton, Fla: CRC Press, 1990.

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6

Whitfield, James F. Calcium in cell cycles and cancer. 2nd ed. Boca Raton, Fla: CRC Press, 1995.

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7

Siddik, Zahid H. Checkpoint controls and targets in cancer therapy. Totowa, N.J: Humana Press, 2010.

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8

Bradshaw, Ralph A. Regulation of organelle and cell compartment signaling. Amsterdam: Elsevier/Academic Press, 2011.

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9

1950-, Karmazyn M., Avkiran M, and Fliegel Larry 1956-, eds. The sodium-hydrogen exchanger: From molecule to its role in disease. Boston: Kluwer Academic Publishers, 2003.

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10

Checkpoint controls and targets in cancer therapy. Totowa, N.J: Humana Press, 2010.

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11

Silencing, heterochromatin, and DNA double strand break repair. Boston: Kluwer Academic Publishers, 2001.

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12

Wagemakers, Alexandre. Physics of complex systems and life sciences. Kerala, India: Research Signpost, 2007.

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13

Pelech, Steven Leif. Protein Kinase Circuitry in Cell Cycle Control. Van Nostrand Reinhold, 1996.

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14

Hoffman, Barbara, and Dan A. Liebermann. Gadd45 Stress Sensor Genes. Springer, 2013.

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15

Zaidi, M. Raza, and Dan A. Liebermann. Gadd45 Stress Sensor Genes. Springer International Publishing AG, 2022.

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16

Hoffman, Barbara, and Dan A. Liebermann. Gadd45 Stress Sensor Genes. Springer London, Limited, 2013.

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17

Hoffman, Barbara, and Dan A. Liebermann. Gadd45 Stress Sensor Genes. Springer, 2013.

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18

Hoffman, Barbara, and Dan A. Liebermann. Gadd45 Stress Sensor Genes. Springer, 2016.

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19

Gross, Michael Karl. Thymidine kinase mRNA and protein levels during myogenic with drawal from the cell cycle: Identification of an mRNA-independent regulatory mechanism. 1988.

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20

B, Kastan M., and Imperial Cancer Research Fund (Great Britain), eds. Checkpoint controls and cancer. Plainview, NY: Cold Spring Harbor Laboratory Press, 1997.

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21

Despa, Florin, and Kimm Jon Hamann. Cell Injury: Mechanisms, Responses, and Therapeutics (Annals of the New York Academy of Sciences). Blackwell Publishing Limited, 2006.

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22

(Editor), R. C. Lee, Florin Despa (Editor), and Kimm Jon Hamann (Editor), eds. Cell Injury: Mechanisms, Responses, And Repair (Annals of the New York Academy of Sciences, V. 1066). New York Academy of Sciences, 2005.

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23

Ho, Yuen. Proteins physically interacting with the Swi6 cell cycle regulatory transcription factor. 1999.

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24

Siddik, Zahid H. Checkpoint Controls and Targets in Cancer Therapy. Humana, 2012.

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25

(Editor), Antonio Giordano, and Gaetano Romano (Editor), eds. Cell Cycle Control and Dysregulation Protocols (Methods in Molecular Biology). Humana Press, 2004.

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26

Mohamed, Sherif. Cell Cycle-Related Proteins in Mediastinal Lymph Nodes of Patients with N2-Nsclc Obtained by Ebus-Tbna. Independently Published, 2018.

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27

The Rvs167 and Pho85 proteins of S. cerevisiae: A link between nutrient sensing, morphogenesis and the cell cycle. Ottawa: National Library of Canada, 1995.

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28

(Editor), Morris Karmazyn, Metin Avkiran (Editor), and Larry Fliegel (Editor), eds. The Sodium-Hydrogen Exchanger: From Molecule to its Role in Disease. Springer, 2003.

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29

Voll, Reinhard E., and Barbara M. Bröker. Innate vs acquired immunity. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0048.

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The innate and the adaptive immune system efficiently cooperate to protect us from infections. The ancient innate immune system, dating back to the first multicellular organisms, utilizes phagocytic cells, soluble antimicrobial peptides, and the complement system for an immediate line of defence against pathogens. Using a limited number of germline-encoded pattern recognition receptors including the Toll-like, RIG-1-like, and NOD-like receptors, the innate immune system recognizes so-called pathogen-associated molecular patterns (PAMPs). PAMPs are specific for groups of related microorganisms and represent highly conserved, mostly non-protein molecules essential for the pathogens' life cycles. Hence, escape mutants strongly reduce the pathogen's fitness. An important task of the innate immune system is to distinguish between harmless antigens and potentially dangerous pathogens. Ideally, innate immune cells should activate the adaptive immune cells only in the case of invading pathogens. The evolutionarily rather new adaptive immune system, which can be found in jawed fish and higher vertebrates, needs several days to mount an efficient response upon its first encounter with a certain pathogen. As soon as antigen-specific lymphocyte clones have been expanded, they powerfully fight the pathogen. Importantly, memory lymphocytes can often protect us from reinfections. During the development of T and B lymphocytes, many millions of different receptors are generated by somatic recombination and hypermutation of gene segments making up the antigen receptors. This process carries the inherent risk of autoimmunity, causing most inflammatory rheumatic diseases. In contrast, inadequate activation of the innate immune system, especially activation of the inflammasomes, may cause autoinflammatory syndromes.

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30

Schönthal, Axel H. Checkpoint Controls and Cancer: Volume 2: Activation and Regulation Protocols (Methods in Molecular Biology). Humana Press, 2004.

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31

Schönthal, Axel H. Checkpoint Controls and Cancer : Volume 2: Activation and Regulation Protocols. Humana Press, 2010.

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32

Burton, Derek, and Margaret Burton. Metabolism, homeostasis and growth. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198785552.003.0007.

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Metabolism consists of the sum of anabolism (construction) and catabolism (destruction) with the release of energy, and achieving a fairly constant internal environment (homeostasis). The aquatic external environment favours differences from mammalian pathways of excretion and requires osmoregulatory adjustments for fresh water and seawater though some taxa, notably marine elasmobranchs, avoid osmoregulatory problems by retaining osmotically active substances such as urea, and molecules protecting tissues from urea damage. Ion regulation may occur through chloride cells of the gills. Most fish are not temperature regulators but a few are regional heterotherms, conserving heat internally. The liver has many roles in metabolism, including in some fish the synthesis of antifreeze seasonally. Maturing females synthesize yolk proteins in the liver. Energy storage may include the liver and, surprisingly, white muscle. Fish growth can be indeterminate and highly variable, with very short (annual) life cycles or extremely long cycles with late and/or intermittent reproduction.

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33

Kirchman, David L. Elements, biochemicals, and structures of microbes. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198789406.003.0002.

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Microbiologists focus on the basic biochemical make-up of microbes, such as relative amounts of protein, RNA, and DNA in cells, while ecologists and biogeochemists use elemental ratios, most notably, the ratio of carbon to nitrogen (C:N), to explore biogeochemical processes and to connect up the carbon cycle with the cycle of other elements. Microbial ecologists make use of both types of data and approaches. This chapter combines both and reviews all things, from elements to macromolecular structures, that make up bacteria and other microbes. The most commonly used elemental ratio was discovered by Alfred Redfield who concluded that microbes have a huge impact on the chemistry of the oceans because of the similarity in nitrogen-to-phosphorus ratios for organisms and nitrate-to-phosphate ratios in the deep oceans. Although statistically different, the C:N ratios in soil microbes are remarkably similar to the ratios of aquatic microbes. The chapter moves on to discussing the macromolecular composition of bacteria and other microbes. This composition gives insights into the growth state of microbes in nature. Geochemists use specific compounds, “biomarkers”, to trace sources of organic material in ecosystems. The last section of the chapter is a review of extracellular polymers, pili, and flagella, which serve a variety of functions, from propelling microbes around to keeping them stuck in one place.

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34

Turner, Neil. Mechanisms of progression of chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0136.

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Three major hypotheses attempt to explain progressive kidney disease following diverse diseases and injuries. To varying degrees they can explain the observed risk factors for progression and the ability of interventions to lower risk. The hyperfiltration hypothesis argues that progression is due to stress on residual nephrons leading to injury and damage to remaining glomeruli. The toxicity of proteinuria hypothesis proposes that serum proteins or bound substances are toxic to tubular or tubulointerstitial cells. This sets up cycles of damage which lead to tubulointerstitial scarring. The podocyte loss hypothesis contends that proteinuria is simply a biomarker for damaged or dying podocytes, and that it is further podocyte loss that leads to progressive glomerulosclerosis. Renoprotective strategies might have direct effects on podocytes. Importantly these different hypotheses suggest different therapeutic approaches to protecting the function of damaged kidneys. Differences between repair mechanisms may explain why some injuries lead to recovery and others to progressive disease, and may suggest new targets for protective therapy.

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