Relevant bibliographies by topics / Cell and Molecular Bioscience

Academic literature on the topic 'Cell and Molecular Bioscience'

Author: Grafiati
Published: 10 December 2022
Last updated: 28 January 2023

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Journal articles on the topic "Cell and Molecular Bioscience"

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McCarthy, John. "Tackling the challenges of interdisciplinary bioscience." Nature Reviews Molecular Cell Biology 5, no. 11 (November 2004): 933–37. http://dx.doi.org/10.1038/nrm1501.

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Davidson, Eric H. "Evolutionary bioscience as regulatory systems biology." Developmental Biology 357, no. 1 (September 2011): 35–40. http://dx.doi.org/10.1016/j.ydbio.2011.02.004.

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Suiter, Tobias, Michael Laffan, Pier M. Mannucci, Christine L. Kempton, Edward H. Romond, Amy D. Shapiro, Ingvild Birschmann, et al. "Recombinant Human Von Willebrand Factor (rhVWF): First-In-Human Study Evaluating Pharmacokinetics, Demonstrating Safety and Tolerability In Type 3 Von Willebrand Disease." Blood 116, no. 21 (November 19, 2010): 237. http://dx.doi.org/10.1182/blood.v116.21.237.237.

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Abstract Abstract 237 Von Willebrand Disease (VWD) is an inherited rare bleeding disorder caused by a deficiency of von Willebrand factor (VWF). VWF is the largest soluble multimeric plasma glycoprotein, which facilitates platelet aggregation and stabilizes FVIII in the circulation. Patients with type 3 disease display severe hemorrhagic symptoms, mainly in mucosal tissues, muscle and joints. Replacement of VWF stabilizes endogenous FVIII to hemostatic levels within hours. Commercially available VWF/FVIII concentrates are plasma-derived (pd) and subject to limitations such as donor dependency, risk of blood-borne pathogen transmission, lack of high molecular weight VWF multimers, and variation in multimer composition. A novel recombinant human VWF (rhVWF) has been developed using a plasma-free method, which represents the largest protein ever produced using recombinant technology. Safety, tolerability and pharmacokinetics of the rhVWF combined at a fixed ratio with rFVIII were investigated in a Phase 1 multicenter, international clinical study in 31 patients with type 3 VWD and severe type 1 VWD. Four concentrations of rhVWF (2, 7.5, 20 and 50 IU VWF:RCo/kg) were administered in a dose-escalating manner in separate cohorts. rhVWF was well tolerated, and no thrombotic events, VWF inhibitors or other serious adverse reactions were observed. Pharmacokinetics of rhVWF/rhFVIII (50 IU VWF:RCo/kg and 38.5 IU FVIII/kg) compared with pdVWF/pdFVIII (50 IU VWF:RCo/kg and 21 IU/kg FVIII/kg) were evaluated in a sub-group of 8/31 patients using a randomized, crossover design (8-day minimum washout period). Interim data in 8 subjects show a higher degree of secondary FVIII activity with rhVWF/rhFVIII compared to pdVWF/pdFVIII (see Figure 1) that is not solely due the difference in the rhVVF:FVIII infusion ratio (1.3:1 rhVWF/rhFVIII vs. approximately 2:1 pdVWF/pdFVIII). The pharmacokinetics of the rhVWF:RCo and pdVWF:RCo were comparable and were also reflected in the VWF:Ag and collagen binding activity. Evidence is also provided for the in vivo cleavage of the ultra-high molecular weight multimers of rhVWF by endogenous ADAMTS13. In summary, interim data from the ongoing Phase 1 study, demonstrate that rhVWF is safe and well tolerated, has VWF:RCo pharmacokinetics that are comparable to pdVWF and enhances stabilization of endogenous FVIII. Multiple doses of rhVWF/rhFVIII would be expected to have beneficial effects in major surgery and severe mucosal bleeding events. These data would also support the treatment concept to administer rhVWF alone once a therapeutic baseline level of endogenous FVIII is achieved (after 1–2 doses).Figure 1:Preliminary PK data from 8 subjects post-infusion of either rhVWF/rhFVIII or pdVWF/pdFVIII. Endogenous FVIII activity reached a plateau after 6 hours and remained stable for at least 30 hours. FVIII was still elevated well above baseline at 96 hoursFigure 1:. Preliminary PK data from 8 subjects post-infusion of either rhVWF/rhFVIII or pdVWF/pdFVIII. Endogenous FVIII activity reached a plateau after 6 hours and remained stable for at least 30 hours. FVIII was still elevated well above baseline at 96 hours Disclosures: Suiter: Baxter BioScience: Employment. Laffan:Baxter BioScience: Consultancy. Mannucci:Baxter BioScience: Consultancy. Kempton:Baxter BioScience: Consultancy. Romond:Baxter BioScience: Consultancy. Shapiro: Baxter BioSci- ence: Consultancy. Birschmann:Baxter BioScience: Consultancy. Gill:Baxter BioScience: Consultancy. Ragni:Baxter BioScience: Consultancy. Turecek:Baxter BioScience: Employment. Ewenstein:Baxter Bioscience: Employment. Baxter BioScience:Baxter BioScience: Employment.
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Shi, Yun-Bo. "The right journal for the right time - Cell & Bioscience." Cell & Bioscience 1, no. 1 (2011): 1. http://dx.doi.org/10.1186/2045-3701-1-1.

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Lee, Hojae, Hengshi Yu, and Joshua Welch. "A beginner's guide to single-cell transcriptomics." Biochemist 41, no. 5 (October 18, 2019): 34–38. http://dx.doi.org/10.1042/bio04105034.

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Beginner's Guides each cover a key technique and offer the scientifically literate but not necessarily expert audience a background briefing on the underlying science of a technique that is (or will be) widely used in molecular bioscience. The series covers a mixture of techniques, including some that are well established amongst a subset of our readership but not necessarily familiar to those in different specialisms, or the reverse. This is our Beginner's Guide to single-cell transcriptomics.
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Stevenson, Brian. "Collaborative practice re-energises bioscience teaching in schools." Microbiology Australia 31, no. 1 (2010): 27. http://dx.doi.org/10.1071/ma10027.

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This year marks the first decade of operations for the Gene Technology Access Centre (GTAC). The decade has seen a grassroots initiative by a small group of eminent research scientists and dedicated personnel from the University High School in Melbourne grow into a specialist education centre in cell and molecular biology that attracts over 6000 students and their teachers each year. GTAC has not only refocused student and teacher attention on the interdisciplinary nature of contemporary biology, but has also highlighted how a ?centre model for learning?, based upon collaboration and partnerships, can exist within ?the school system? and meet the needs of students and teachers from across Victoria and beyond.
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Flier, Jeffrey S. "Irreproducibility of published bioscience research: Diagnosis, pathogenesis and therapy." Molecular Metabolism 6, no. 1 (January 2017): 2–9. http://dx.doi.org/10.1016/j.molmet.2016.11.006.

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Li, Henry Qixiang, Jingping Liu, Xiaoyu An, Na Wang, Liang Huang, Ran Wu, Jie Cai, and Jean-Pierre Wery. "Creation Of Patient Derived AML Xenografts Displaying Distinct Phenotypes and Geneotypes." Blood 122, no. 21 (November 15, 2013): 5018. http://dx.doi.org/10.1182/blood.v122.21.5018.5018.

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Abstract We have recently successfully engrafted leukemic cells from bone marrow of several AML patients into immunocompromised NOD/SCID mice. One of them, AM7577, was reported in the last year ASH1. This model displayed typical aggressive AML disease of M5 subtype, which starts at bone marrow and gradually expand to peripheral (spleen, lymphonode and peripheral blood…). AM7577 also harbors interesting genotypes of mutations for IDH2-R140Q, FLT3-ITD, DNMT3A R882H and NPM1. Our second AML PDX model, AM8096, was established similarly using bone marrow from a recurrent patient with AML-M2 disease. AML8096 displayed similarly aggressive disease and the same 100% mortality, including typical symptoms (BW loss, hunched, inactivity, labored breathing, ruffled coat and eventual mortality) and with abundant leukemic cells in bone, etc. The leukemic cells can serially be passed in mice with 100% take-rate and cause consistent disease (even with < 1e4 cells). This also creates a renewable and potentially unlimited source of leukemia cells. The leukemic cells in mice are identical to those of the original patient leukemic cells (CD45+, CD33+, CD13+, CD123+, and CD19-. However, some aspects of disease presentations are vastly different from that of AM7577. First, the peripheral symptom is significant less characterized by lower leukemic counts in peripheral blood and only slightly enlarged spleen and smaller lymphonode. Second, the leukemic cell morphology is also rather different with AM8096 demonstrating less differentiated phenotype. Third, the genotype, in contrast to AM7577, is wild-types for all the above oncogenes. While we have not identified the likely leukemogenesis driver gene for this model, we are currently performing RNAseq of AM8096, along with AM7577, in order to explore the underlying molecular mechanisms that drive both diseases. In addition, we are also investigating the drug response to standard of care (SOC), as compared to those AM7577. In summary, the two AML models could serve as useful experimental models to investigate the diverse leukemogenesis, including molecular mechanisms by genetic profiling. Ultimately, they can be used to help to identify new treatment strategies for AML. 1. Liu, e.a. A unique leukemia mouse model established from AML patient with IDH2 R140Q and FLT3-ITD mutations among other common AML mutations. ASH-2012 Annual Meeting (2012). Disclosures: Li: Crown Bioscience: Employment. Liu:Crown Bioscience: Employment. An:Crown Bioscience: Employment. Wu:Crown Bioscience: Employment. Cai:Crown Bioscience: Employment. Wery:Crown Bioscience: Employment.
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Shi, Yun-Bo. "The 2012 Ming K Jeang award for excellence in cell & bioscience." Cell & Bioscience 3, no. 1 (2013): 23. http://dx.doi.org/10.1186/2045-3701-3-23.

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Lachica, Edward. "Keeping Life in Focus New Systems Prevent Z-axis Drift in Time Lapse Studies." Microscopy Today 14, no. 4 (July 2006): 42–46. http://dx.doi.org/10.1017/s1551929500050276.

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Just two decades ago, life scientists studied biological structure, developmental anatomy and intracellular processes by describing individual snapshots of kinetic events. Today, with so much bioscience research focusing on dynamic processes that occur on the molecular, cellular and whole organ level, it is important to record events as they happen, over seconds, minutes or hours, in living cells. Photographs and camera lucida drawings of fixed, stained cells have given way to live cell imaging using fluorescent probes, warming trays to promote cell viability and cinemicrography as a method of recording events.
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Dissertations / Theses on the topic "Cell and Molecular Bioscience"

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Sargeant, Timothy John. "The effect of opiates on developing cerebral cortex : a thesis submitted to the Victoria University of Wellington in fulfilment of the requirements for the degree of Doctor of Philosophy in Cell and Molecular Bioscience /." ResearchArchive@Victoria e-Thesis, 2008. http://hdl.handle.net/10063/414.

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Reader, Karen Lee. "A quantitative ultrastructural study of oocytes during the early stages of ovarian follicular development in Booroola and wild-type sheep : a thesis submitted to the Victoria University of Wellington in fulfilment of the requirements for the degree of Master of Science in Cell and Molecular Bioscience /." ResearchArchive@Victoria e-Thesis, 2007. http://hdl.handle.net/10063/270.

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Speh, Michel Jonathan. "Transcriptional changes in stem cell-derived cardiomyocytes during extended cell cultures." Thesis, Högskolan i Skövde, Institutionen för biovetenskap, 2020. http://urn.kb.se/resolve?urn=urn:nbn:se:his:diva-18781.

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The recent advancement in the field of stem cell research gave rise to high expectations from both general and scientific audiences, and indeed, stem cells and stem cell-derived cells have an enormous potential to forward the fields of medical research and regenerative medicine. Nevertheless, there are still many obstacles and limitations associated with stem cell technology. One of those limitations is that currently no method to derive fully mature cardiomyocytes from human pluripotent stem cell is available. Instead, stem cell-derived cardiomyocytes only show basic characteristics of their adult counterparts and are thus only of limited use. However, there is experimental evidence that those cells may increase in maturity during extended culture times. To improve the understanding of those processes, a characterisation of the transcriptional changes in human embryonic stem cell-derived cardiomyocytes over two weeks was made. Using standard bioinformatics methods, including differential and enrichment analysis as well as basic machine learning technologies, changes in mRNA and miRNA transcription and several functions related to those changes could be identified. The analyses indicated increasing structural organisation in the cell cultures, increasing expression of cardiac ion channels and decreasing proliferative capacity in the cardiomyocyte cultures. Furthermore, potential dysregulations of important signalling pathways were observed. The results of this project may aid in developing protocols for differentiating stem cells into cardiomyocytes with features that are more mature than those of the currently available stem cell derived-cardiomyocytes.
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Patel, Angana Heet. "Unravelingthe molecular mechanism behind metabolic reprogramming caused by alterations of the enzyme PI3-kinase." Thesis, Högskolan i Skövde, Institutionen för biovetenskap, 2019. http://urn.kb.se/resolve?urn=urn:nbn:se:his:diva-17410.

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Oncogenes and tumor suppressor genes play a key role in cancer induction and progression. They directly or indirectly regulate critical metabolic pathways, phosphatidylinositol-3 kinase pathway being frequently activated pathway in cancer. The catalytic subunit of phosphatidylinositol-4,5-bisphosphate 3-kinase (PI3K), p110α, is the most frequently mutated kinase in human cancer, E542K, E545K, and H1047R mutations being the most common. Expression of hepatic E545K and H1047R p110α mutants in vivo shows marked and rapid increase in hepatic lipid and glycogen accumulation in mice with developmental (chronic) liver-specific deletion of p110α, which was not seen in mice when wildtype p110α is overexpressed. To investigate the logical pathways that could explain the lipid accumulation in mutant expressing mice, RNA sequencing from wildtype, knockout and mutated mouse livers was performed. Read alignment and count quantification was done using the Rsubread package and the statistical analyses are performed using the DeSeq2 package. Differentially expressed genes were identified with adjusted p-value of 0.05. Gene ontology analysis was performed on the differentially expressed genes using clusterProfiler, an R package to identify several key pathways which were upregulated and downregulated among the different sample groups. Signaling pathways related to cell cycle processes were mainly upregulated in the mutated samples when compared with the wildtype as well as knockout samples while signaling pathways related to many metabolic processes seem to be downregulated in mutated samples, even though these mutants showed upregulated metabolism by accumulation of lipids and glycogen physiologically. To confirm the results of gene expression data the results have to be cross validated with the gold standard quantitative Real Time Polymerase Chain Reaction.
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Babena, Omar. "Expression of the chloride channel CLCC1 is downregulated after 24 hours in LPS-primed THP-1 monocyte-like cell line." Thesis, Högskolan i Skövde, Institutionen för biovetenskap, 2021. http://urn.kb.se/resolve?urn=urn:nbn:se:his:diva-20377.

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Inflammation is the body's response to infection or injury and is mediated by the innate immune system. The NLRP3 inflammasome is a multi-protein complex that is a major contributor to many inflammatory disorders. Emerging evidence suggests the involvement of the Endoplasmic reticulum stress with the NLRP3 inflammasome. The endoplasmic reticulum stress is a series of stress signals that can activate the unfolded protein response and usually accompanies inflammation and eventually causes cell death. Recently, a localized endoplasmic reticulum micro-protein called the chloride clic like-1 channel was found to be involved in the endoplasmic reticulum homeostasis. Recent evidence suggests the involvement of endoplasmic reticulum stress in the inflammation pathways of the NLRP3 inflammasome. The relationship between the ER and the NLRP3 inflammasome has not been clearly described. This study aimed at investigating the expression levels of the microprotein CLCC1 to shed a light on the relationship between the endoplasmic reticulum stress and the NLRP3 inflammasome. The expression levels of CLCC1 were analyzed by qPCR in cultured monocytes under different time points of Lipopolysachaaride immuno-stimulation. The stability of expression in candidate reference genes was investigated for normalization purposes. This study reported the regulation of CLCC1 as a novel finding under prolonged LPS exposure of monocytes and stable reference genes such as GUSB and ACTB were identified. The relationship between CLCC1 and NLRP3 inflammasome priming by LPS indicated that CLCC1 is regulated and may be involved in the inflammatory mechanisms of endoplasmic reticulum stress and NLRP3 inflammasome inflammatory diseases, contributing to a potential therapeutic target in the endoplasmic reticulum and inflammasome related diseases.
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Holscher, Maxwell Arthur. "Molecular analysis of NK cell - target cell interactions." Thesis, University of Newcastle Upon Tyne, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.287147.

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Zannettino, Andrew Christopher William. "Molecular definition of stromal cell-stem cell interactions /." Title page, contents and summary only, 1996. http://web4.library.adelaide.edu.au/theses/09PH/09phz32.pdf.

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Bair, Elisabeth Laurine. "Cell-cell and cell-matrix interactions involved in cancer invasion." Diss., The University of Arizona, 2004. http://hdl.handle.net/10150/280673.

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In order for a cancer to metastasize, it must first invade through the basement membrane that surrounds it, invade blood vessels and travel through the bloodstream to a new location where it extravasates the vessel and begins growing at the new site. The mechanisms by which a cancer becomes able to invade and metastasize are currently under intense study. Interactions of the cell with its environment via cell-cell contacts, extracellular matrix (ECM) interactions, and circulating proteins are thought to play a major role in signaling for these invasive processes to occur. Upregulation of proteolytic enzymes, such as the matrix metalloproteases, is suspected of being involved in the metastatic process. Cell-cell and cell-matrix contacts via integrins and cadherins are necessary for upregulation of the matrix metalloprotease matrilysin in oral squamous cell carcinoma. In an effort to identify the factors involved in upregulation of matrilysin expression detected in a co-culture of oral squamous cell carcinoma (SCC) cells and fibroblast cells, a coculture model designed to represent the actual tumor environment, we show that inhibition of beta1 integrin, E-cadherin, and N-cadherin with blocking antibodies thoroughly decreases the induction of matrilysin in the co-culture model. This demonstrates that interactions between cancer cells and normal cells surrounding them may allow for invasion and metastasis. The protein 90K may also play a role in the invasive process of prostate cancer. It functions as an immune modulator upregulating cytokines that induce MMPs and we show that it can induce matrilysin expression in prostate cancer cells. It also functions in cell aggregation, which can help cells survive during metastasis. For this reason, expression of 90K in prostate cancer, which we examined, may be indicative of aggressive disease, making 90K a potentially useful tumor marker. Cell-matrix contacts are also important for the transmembrane matrix metalloprotease MT1-MMP cleavage of laminin-10. We demonstrate that recombinant MT1-MMP is able to cleave human laminin-10 into four distinct products. This allows for prostate cancer cell migration on laminin-10 coated substrates, which can be inhibited with the addition of MT1-MMP antisense oligonucleotides. Ln-10 cleavage also occurs in vivo in human prostate tissue, indicating that this cell-matrix interaction has in vivo relevance in human prostate cancer.
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Lee, Soo Young. "Dissecting Molecular Mechanisms of Shigella flexneri Cell-to-cell Spread." Thesis, Harvard University, 2014. http://nrs.harvard.edu/urn-3:HUL.InstRepos:13065011.

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Shigella is a causative agent of bacillary dysentery in humans. The ability of Shigella to disseminate in the intestinal epithelium is crucial for disease establishment. This process of cell-to-cell spread involves actin-based motility, which allows movement of Shigella through the cytoplasm, and the ability of Shigella to form filopodia-like membrane protrusions that are engulfed by adjacent cells. Compared to the process of Shigella actin tail assembly, which requires recruitment and activation of host actin modulators such as N-WASP and Arp2/3, the mechanism of how Shigella moves from an infected cell into neighboring cells and what host factors are involved remain poorly characterized. In this dissertation, I investigate whether members of the Ena/VASP family, as key actin regulators, or Inverse-BAR (I-BAR) family proteins, as coordinators of membrane curvature and actin dynamics, are required in dissemination of S. flexneri in a cell monolayer. Ena/VASP family proteins regulate cell migration, adhesion, shape, and cell-cell interaction. The members of the family include Vasodilator-Stimulated Phosphoprotein (VASP), Ena-VASP-like (Evl), and Mammalian enabled (Mena). We have previously shown that Mena, despite its localization to the actin tail, has no role in S. flexneri actin-based motility. Here, I investigate the role of Mena, Evl, and VASP in S. flexneri dissemination. I determine that the presence of VASP or Evl restricts cell-to-cell spread of S. flexneri. I further show evidence that the conserved EVH1 domain and phosphorylation of VASP regulate the ability of Shigella to spread. I-BAR proteins, including IRSp53 and IRTKS, contain a conserved domain that directly binds to membrane lipids and induces convex membrane deformation. This unique property and the ability of these proteins to bind F-actin and actin modulators are necessary for the formation of actin pedestals by pathogenic E. coli and filopodia. Using cells with reduced levels of IRTKS or IRSp53, I examine the role of these proteins in cell-to-cell spread and show that neither IRTKS nor IRSp53 is required for S. flexneri spread. Collectively, these results advance our understanding of host proteins that participate in S. flexneri dissemination.
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Rennel, Emma. "Molecular Mechanisms in Endothelial Cell Differentiation." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2004. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-4059.

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Books on the topic "Cell and Molecular Bioscience"

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F, Lodish Harvey, and Darnell James E, eds. Molecular cell biology. 3rd ed. New York: Scientific American Books, 1995.

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F, Lodish Harvey, ed. Molecular cell biology. 6th ed. New York: W.H. Freeman, 2007.

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M, Gottesman Michael, ed. Molecular cell genetics. New York: Wiley, 1985.

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Molecular cell biology. Reading, Mass: Addison-Wesley, 1986.

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F, Lodish Harvey, and Baltimore David, eds. Molecular cell biology. New York: Scientific American Books, 1986.

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F, Lodish Harvey, ed. Molecular cell biology. 4th ed. New York: W.H. Freeman, 2000.

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F, Lodish Harvey, ed. Molecular cell biology. 4th ed. New York: W.H. Freeman, 2000.

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F, Lodish Harvey, ed. Molecular cell biology. 5th ed. New York: W.H. Freeman and Co., 2004.

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F, Lodish Harvey, and Baltimore David, eds. Molecular cell biology. 2nd ed. New York: Scientific American Books, 1990.

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K, Rehan Virender, ed. Evolutionary biology, cell-cell communication, and complex disease. Hoboken, N.J: Wiley-Blackwell, 2012.

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Book chapters on the topic "Cell and Molecular Bioscience"

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Sachs, Leo. "Cell Differentiation and Malignancy." In Bioscience at the Physical Science Frontier, 225–42. Totowa, NJ: Humana Press, 1986. http://dx.doi.org/10.1007/978-1-4612-4834-7_14.

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Priyadarshini, Anjali, and Prerna Pandey. "Cell." In Molecular Biology, 1–28. Toronto ; New Jersey : Apple Academic Press, 2018.: Apple Academic Press, 2018. http://dx.doi.org/10.1201/b22354-1.

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Krensky, Alan M., Steven J. Mentzer, Julia L. Greenstein, Mary Crimmins, Carol Clayberger, Timothy A. Springer, and Steven J. Burakoff. "Human Cytolytic T-Lymphocyte Clones and Their Function-Associated Cell Surface Molecules." In Hybridoma Technology in the Biosciences and Medicine, 559–73. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-4964-8_35.

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Balny, Claude, and Reinhard Lange. "Optical Spectroscopic Techniques in High Pressure Bioscience." In High Pressure Molecular Science, 405–22. Dordrecht: Springer Netherlands, 1999. http://dx.doi.org/10.1007/978-94-011-4669-2_21.

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Widłak, Wiesława. "Cell Division." In Molecular Biology, 109–20. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-45361-8_7.

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Modrow, Susanne, Falke Dietrich, Uwe Truyen, and Hermann Schätzl. "Cell Damage." In Molecular Virology, 49–56. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-20718-1_5.

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Granum, Per Einar, and Gordon S. A. B. Stewart. "Molecular Biology of Clostridium perfringens Enterotoxin." In Brock/Springer Series in Contemporary Bioscience, 235–47. New York, NY: Springer New York, 1993. http://dx.doi.org/10.1007/978-1-4615-7087-5_16.

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Voordouw, Gerrit. "Molecular Biology of the Sulfate-Reducing Bacteria." In Brock/Springer Series in Contemporary Bioscience, 88–130. New York, NY: Springer New York, 1993. http://dx.doi.org/10.1007/978-1-4613-9263-7_5.

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von Eichel-Streiber, Christoph. "Molecular Biology of the Clostridium difficile Toxins." In Brock/Springer Series in Contemporary Bioscience, 264–89. New York, NY: Springer New York, 1993. http://dx.doi.org/10.1007/978-1-4615-7087-5_19.

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Bagdasarian, Michael, Yong-Eok Lee, Chanyong Lee, Menghsiao Meng, and J. Gregory Zeikus. "Molecular Biology of Xylan Utilization by Thermoanaerobes." In Brock/Springer Series in Contemporary Bioscience, 443–55. New York, NY: Springer New York, 1993. http://dx.doi.org/10.1007/978-1-4615-7087-5_33.

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Conference papers on the topic "Cell and Molecular Bioscience"

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Girsang, Ermi, I. Nyoman Ehrich Lister, Chrismis Novalinda Ginting, Sri Lestari Nasution, Suhartina Suhartina, Ubaydillah Zedd Munshy, Rizal Rizal, and Wahyu Widowati. "Antioxidant and Anti-inflammatory Activity of Salacca zalacca (Gaertn.) Voss Peel Ethanolic Extract on Lead Induced Fibroblast Cells." In 6th ICAMBBE (International Conference on Advance Molecular Bioscience & Biomedical Engineering) 2019. SCITEPRESS - Science and Technology Publications, 2019. http://dx.doi.org/10.5220/0009588200680073.

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Moritani, Yuki, Satoshi Hiyama, and Tatsuya Suda. "Molecular Communication A Biochemically-Engineered Communication System." In 2007 Frontiers in the Convergence of Bioscience and Information Technologies. IEEE, 2007. http://dx.doi.org/10.1109/fbit.2007.120.

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Lee, Jun, Taedoo Hwang, Jonghyun Lee, Sungjun Park, YoungJin Choi, Karpjoo Jeong, and Jee-In Kim. "A Web-Based Interactive Monitoring System for Molecular Simulation." In 2007 Frontiers in the Convergence of Bioscience and Information Technologies. IEEE, 2007. http://dx.doi.org/10.1109/fbit.2007.117.

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Perera, D. R. C., W. W. P. Rodrigo, A. M. M. H. Athapaththu, and P. A. D. H. N. Gunathilaka. "ESTABLISHMENT OF A MOLECULAR BASED METHOD FOR THE IDENTIFICATION OF SKIPJACK TUNA (Katsuwonus pelamis) IN LARGE SCALE FISH PROCESSING INDUSTRY." In International Conference on Bioscience and Biotechnology. TIIKM, 2016. http://dx.doi.org/10.17501/biotech.2016.1105.

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Golmohammadi, Seyed Koosha, Lukasz Kurgan, Brendan Crowley, and Marek Reformat. "Classification of Cell Membrane Proteins." In 2007 Frontiers in the Convergence of Bioscience and Information Technologies. IEEE, 2007. http://dx.doi.org/10.1109/fbit.2007.21.

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Lin, Hung-Yi, and Da-Yi Yen. "Assessing Information Quality and Distinguishing Feature Subsets for Molecular Classification." In ICBBB '20: 2020 10th International Conference on Bioscience, Biochemistry and Bioinformatics. New York, NY, USA: ACM, 2020. http://dx.doi.org/10.1145/3386052.3386061.

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Gao, Tong. "Constructing Crystalline Molecular Dipolar Rotor Arrays with Ultra-Large Dipole Moments." In ICBBB '21: 2021 11th International Conference on Bioscience, Biochemistry and Bioinformatics. New York, NY, USA: ACM, 2021. http://dx.doi.org/10.1145/3448340.3448352.

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Hwa, Kuo-Yuan, Wan Man Lin, Yung-I. Hou, and Trai-Ming Yeh. "Molecular Mimicry between SARS Coronavirus Spike Protein and Human Protein." In 2007 Frontiers in the Convergence of Bioscience and Information Technologies (FBIT '07). IEEE, 2007. http://dx.doi.org/10.1109/fbit.2007.108.

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Zhu, Jianshen, Naveed A. Azam, Kazuya Haraguchi, Liang Zhao, Hiroshi Nagamochi, and Tatsuya Akutsu. "A Method for Molecular Design Based on Linear Regression and Integer Programming." In ICBBB '22: 2022 12th International Conference on Bioscience, Biochemistry and Bioinformatics. New York, NY, USA: ACM, 2022. http://dx.doi.org/10.1145/3510427.3510431.

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Haskito, A. E. P., A. Setianingrum, F. N. A. E. P. Dameanti, and M. Fatmawati. "Organoleptic Properties Evaluation of Goat Milk Yogurt with White Rice Bran Flour Fortification." In 6th ICAMBBE (International Conference on Advance Molecular Bioscience & Biomedical Engineering) 2019. SCITEPRESS - Science and Technology Publications, 2019. http://dx.doi.org/10.5220/0009586001170121.

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Reports on the topic "Cell and Molecular Bioscience"

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DeVries, George H. Molecular Mechanisms of Schwann Cell Proliferation in NF1. Fort Belvoir, VA: Defense Technical Information Center, September 2001. http://dx.doi.org/10.21236/ada407274.

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DeVries, George H. Molecular Mechanisms of Schwann Cell Proliferation in NF1. Fort Belvoir, VA: Defense Technical Information Center, September 1999. http://dx.doi.org/10.21236/ada390950.

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DeVries, George H. Molecular Mechanisms of Schwann Cell Proliferation in NF1. Fort Belvoir, VA: Defense Technical Information Center, September 2000. http://dx.doi.org/10.21236/ada392306.

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Kaini, Ramesh. Molecular Basis of Autophagic Cell Death in Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, March 2009. http://dx.doi.org/10.21236/ada540723.

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Kaini, Ramesh. Molecular Basis of Autophagic Cell Death in Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, March 2009. http://dx.doi.org/10.21236/ada506319.

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Ray, P. M. Enzymology and molecular biology of cell wall biosynthesis. Progress report. Office of Scientific and Technical Information (OSTI), March 1993. http://dx.doi.org/10.2172/10134679.

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Itoh, Masahiko. Identification of the Molecular Determinants of Breast Epithelial Cell Polarity. Fort Belvoir, VA: Defense Technical Information Center, October 2005. http://dx.doi.org/10.21236/ada455160.

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Itoh, Masahiko. Identification of the Molecular Determinants of Breast Epithelial Cell Polarity. Fort Belvoir, VA: Defense Technical Information Center, October 2004. http://dx.doi.org/10.21236/ada432143.

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Itoh, Masahiko. Identification of the Molecular Determinants of Breast Epithelial Cell Polarity. Fort Belvoir, VA: Defense Technical Information Center, October 2006. http://dx.doi.org/10.21236/ada465842.

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Allara, David L. Characterization of the Molecular Basis of Cell Recognition at Surfaces. Fort Belvoir, VA: Defense Technical Information Center, December 1998. http://dx.doi.org/10.21236/ada384249.

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