Relevant bibliographies by topics / CDCH

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'CDCH'

Author: Grafiati
Published: 14 March 2023

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Journal articles on the topic "CDCH"

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Hermann, Petra M., Robert P. J. de Lange, Anton W. Pieneman, Andries ter Maat, and Rene F. Jansen. "Role of Neuropeptides Encoded on CDCH-1 Gene in the Organization of Egg-Laying Behavior in the Pond Snail, Lymnaea stagnalis." Journal of Neurophysiology 78, no. 6 (December 1, 1997): 2859–69. http://dx.doi.org/10.1152/jn.1997.78.6.2859.

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Hermann, Petra M., Robert P. J. de Lange, Anton W. Pieneman, Andries ter Maat, and Rene F. Jansen. Role of neuropeptides encoded on CDCH-1 gene in the organization of egg-laying behavior in the pond snail, Lymnaea stagnalis. J. Neurophysiol. 78: 2859–2869, 1997. Egg laying in the pond snail Lymnaea stagnalis is triggered by a discharge of the neuroendocrine caudodorsal cells (CDCs). The CDCs expresses three different caudorsal cell hormone (CDCH) genes. This gene family expresses, in total, 11 different peptides among which is the ovulation hormone. Besides the CDCs, the CDCH gene family is expressed in other central and peripheral neurons. In this study, we investigated the roles the different CDCH peptides play in the organization of egg-laying behavior. Egg-laying behavior is a sequence of stereotyped movements in which three phases can be distinguished: resting, turning, and oviposition. We have used the excitation of right pedal N (RPeN) motor neurons as a simple analogue of shell-turning behavior, one of the elements of egg-laying behavior. RPeN motor neurons were inhibited during the resting phase of egg laying but were subsequently excited at the onset of and during the turning phase. The excitatory effect could be evoked by application of beta3-CDCP on RPeN motor neurons in the CNS as well as in isolation but not by the ovulation hormone, alpha-CDCP or Calfluxin, the other CDCH-1 peptides tested. The ovulation hormone itself caused inhibition of RPeN motor neurons. Anti-CDCH–1 positive fiber tracts were found close to the cell bodies and axons of the RPeN motor neurons. Electrical stimulation of a nerve that contains these fibers resulted in excitation of the RPeN motor neurons. The effects of injection of CDCH-1 peptides into intact animals correlated well with the effects of these peptides on RPeN motor neurons. Injection of beta3-CDCP or alpha-CDCP into intact animals resulted in immediate turning behavior in the absence of egg laying itself. The ovulation hormone and Calfluxin had no immediate effect on the behavior. Furthermore, our data indicate that the individual CDCH-1 peptides act on different targets.
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Cabeza-Cabrerizo, Mar, Janneke van Blijswijk, Stephan Wienert, Daniel Heim, Robert P. Jenkins, Probir Chakravarty, Neil Rogers, et al. "Tissue clonality of dendritic cell subsets and emergency DCpoiesis revealed by multicolor fate mapping of DC progenitors." Science Immunology 4, no. 33 (March 1, 2019): eaaw1941. http://dx.doi.org/10.1126/sciimmunol.aaw1941.

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Conventional dendritic cells (cDCs) are found in all tissues and play a key role in immune surveillance. They comprise two major subsets, cDC1 and cDC2, both derived from circulating precursors of cDCs (pre-cDCs), which exited the bone marrow. We show that, in the steady-state mouse, pre-cDCs entering tissues proliferate to give rise to differentiated cDCs, which themselves have residual proliferative capacity. We use multicolor fate mapping of cDC progenitors to show that this results in clones of sister cDCs, most of which comprise a single cDC1 or cDC2 subtype, suggestive of pre-cDC commitment. Upon infection, a surge in the influx of pre-cDCs into the affected tissue dilutes clones and increases cDC numbers. Our results indicate that tissue cDCs can be organized in a patchwork of closely positioned sister cells of the same subset whose coexistence is perturbed by local infection, when the bone marrow provides additional pre-cDCs to meet increased tissue demand.
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Srinivasan, Jayashree, Bryan Helm, Zhe Su, Song Yi, Qi Liu, Ken Lau, and Lauren Ilyse Richie Ehrlich. "Cellular and molecular mediators of thymic DC homeostasis and activation." Journal of Immunology 208, no. 1_Supplement (May 1, 2022): 47.10. http://dx.doi.org/10.4049/jimmunol.208.supp.47.10.

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Abstract Central tolerance in the thymus ensures that autoreactive thymocytes undergo negative selection or diversion to the regulatory T cell (Treg) lineage. Thymic antigen presenting cells (APCs) such as medullary thymic epithelial cells (mTECs) and dendritic cells (DCs) present myriad self-peptides to thymocytes to induce central tolerance. We and others have previously shown that thymic cDCs can be subdivided into activated CCR7+MHC-IIhi and resting CCR7− MHC-IIlo cDC1 and cDC2 subsets with distinct functional properties, suggesting the thymic DC compartment may be more heterogeneous than previously described. Here, we performed single-cell RNA sequencing (scRNAseq) of hematopoietic APCs isolated from adult mouse thymi to comprehensively identify thymic DC subsets with distinct gene expression profiles. Our data reveal 11 transcriptionally distinct cDC subsets. We identify 6 cDC1 subsets, 3 cDC2 subsets, 2 activated CCR7+ “cDC3” subsets distinguished by expression of cDC1 and cDC2 markers, and 4 plasmacytoid DC clusters. Through functional assays, we find that the XCR1+ cDC3s are the most efficient at presenting mTEC-derived peptides on MHC-I and MHC-II, while 2 distinct cDC2 subsets are the most efficient at generating Tregs in vitro. Additional scRNA-seq and flow cytometry analyses of thymic DCs from mice in which thymocyte maturation was blocked at different developmental stages reveal candidate cellular and molecular crosstalk signals that regulate homeostasis and/or activation of pDC and cDC subsets. Our findings reveal hitherto unknown functional heterogeneity within the thymic DC compartment and identify potential interactions that support DC homeostasis and activation which in turn could impact central tolerance. Supported by NIH P01 AI139449-02
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Toka, Felix N., Lidia Szulc-Dąbrowska, Michal Koper, Justyna Struzik, and Malgorzata Gierynska. "Classical splenic dendritic cell subsets, cDC1 and cDC2, from C57BL/6 mice are more potent in stimulating the Th1 immune response than those from BALB/c mice during mousepox." Journal of Immunology 204, no. 1_Supplement (May 1, 2020): 140.20. http://dx.doi.org/10.4049/jimmunol.204.supp.140.20.

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Abstract Classical (conventional) dendritic cells (cDCs) specialize in presenting antigen to naïve T cells, and are divided into cDC1 (CD8α+) and cDC2 (CD11b+) subpopulations based on their lineage, surface phenotype and characteristics. Despite differences, both cDC subsets can stimulate CD4+ T cells and promote polarization toward a T helper 1 (Th1) phenotype. Many studies have shown that C57BL/6 (H-2b) and BALB/c (H-2d) mice readily mount vigorous Th1 or Th2 responses, respectively, upon infection with different infectious agents, including ectromelia virus (ECTV), responsible for mousepox in mice. In this work, we compared the ability of cDC subsets from mouse strains with different susceptibility to mousepox (C57BL/6 – resistant and BALB/c – susceptible) to stimulate the Th1 cytokine immune response during ECTV infection. Results showed that splenic cDC1 and cDC2 from BALB/c mice highly express MHC class II, CD83 and/or CD86 than those from C57BL/6 mice at 5 days post infection with ECTV. Despite higher activation status, both subsets of cDCs from BALB/c mice produce low amounts of Th1-polarizing cytokines, including IL-12 and IFN-γ, than those from C57BL/6 mice. Moreover, splenic cDC1 and cDC2 cells, from ECTV-infected C57BL/6 mice, stimulated higher proliferation and production of IFN-γ and IL-2 by allogeneic CD4+ T cells of C3H (H-2k) mice in a mixed leukocyte reaction than BALB/c mice. Both subsets of BALB/c cDCs up-regulated genes engaged in cell maturation and activation compared to C57BL/6 cDCs. Overall, our data indicate that both, cDC1 and cDC2 promote CD4+ T cell differentiation into IFN-γ-producing Th1 subset, and so ensure development of a strong cell-mediated immune response and recovery of C57BL/6 mice from mousepox.
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Audiger, Cindy, and Sylvie Lesage. "Merocytic dendritic cell: a new subset of conventional dendritic cells." Journal of Immunology 202, no. 1_Supplement (May 1, 2019): 118.11. http://dx.doi.org/10.4049/jimmunol.202.supp.118.11.

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Abstract Conventional dendritic cells (cDC) are potent antigen-presenting cells that induce the activation of naïve T cells in response to pathogens. cDC activity is mediated primarily by two cDC subsets, namely cDC1 and cDC2, each bearing unique properties. Recently, another DC subset, termed merocytic dendritic cells (mcDC), was defined. In contrast to both cDC1 and cDC2, mcDC are able to reverse T cell anergy, even in non-inflammatory conditions, properties that could be exploited to potentiate cancer treatments. Here, we further characterize mcDC to determine their relationship to cDCs. First, we demonstrate that mcDC express key cDC traits, namely they express the cDC-restricted transcription factor, Zbtb46, and are very potent inducers of mixed lymphocyte reactions. Second, transcriptomic studies reveal that mcDC are more closely related to cDC1 than to cDC2. In contrast, similar to cDC2, mcDC are dependent on IRF4, but not IRF8 and BATF3, two major transcription factors required for cDC1 differentiation. Third, investigating mcDC population dynamics in reconstitution kinetics studies and in parabiotic mice, we demonstrate that, as for cDC1 and cDC2, mcDCs are terminally differentiated cells. Altogether, these data demonstrate that mcDC compose novel cDC subset. Defining the properties of mcDC in mice may help identify a functionally equivalent subset in humans leading to the development of novel cancer immunotherapies.
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Burke, D. J., and D. Church. "Protein synthesis requirements for nuclear division, cytokinesis, and cell separation in Saccharomyces cerevisiae." Molecular and Cellular Biology 11, no. 7 (July 1991): 3691–98. http://dx.doi.org/10.1128/mcb.11.7.3691-3698.1991.

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Protein synthesis inhibitors have often been used to identify regulatory steps in cell division. We used cell division cycle mutants of the yeast Saccharomyces cerevisiae and two chemical inhibitors of translation to investigate the requirements for protein synthesis for completing landmark events after the G1 phase of the cell cycle. We show, using cdc2, cdc6, cdc7, cdc8, cdc17 (38 degrees C), and cdc21 (also named tmp1) mutants, that cells arrested in S phase complete DNA synthesis but cannot complete nuclear division if protein synthesis is inhibited. In contrast, we show, using cdc16, cdc17 (36 degrees C), cdc20, cdc23, and nocodazole treatment, that cells that arrest in the G2 stage complete nuclear division in the absence of protein synthesis. Protein synthesis is required late in the cell cycle to complete cytokinesis and cell separation. These studies show that there are requirements for protein synthesis in the cell cycle, after G1, that are restricted to two discrete intervals.
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Burke, D. J., and D. Church. "Protein synthesis requirements for nuclear division, cytokinesis, and cell separation in Saccharomyces cerevisiae." Molecular and Cellular Biology 11, no. 7 (July 1991): 3691–98. http://dx.doi.org/10.1128/mcb.11.7.3691.

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Protein synthesis inhibitors have often been used to identify regulatory steps in cell division. We used cell division cycle mutants of the yeast Saccharomyces cerevisiae and two chemical inhibitors of translation to investigate the requirements for protein synthesis for completing landmark events after the G1 phase of the cell cycle. We show, using cdc2, cdc6, cdc7, cdc8, cdc17 (38 degrees C), and cdc21 (also named tmp1) mutants, that cells arrested in S phase complete DNA synthesis but cannot complete nuclear division if protein synthesis is inhibited. In contrast, we show, using cdc16, cdc17 (36 degrees C), cdc20, cdc23, and nocodazole treatment, that cells that arrest in the G2 stage complete nuclear division in the absence of protein synthesis. Protein synthesis is required late in the cell cycle to complete cytokinesis and cell separation. These studies show that there are requirements for protein synthesis in the cell cycle, after G1, that are restricted to two discrete intervals.
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Peterson, T. A., L. Prakash, S. Prakash, M. A. Osley, and S. I. Reed. "Regulation of CDC9, the Saccharomyces cerevisiae gene that encodes DNA ligase." Molecular and Cellular Biology 5, no. 1 (January 1985): 226–35. http://dx.doi.org/10.1128/mcb.5.1.226-235.1985.

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We have cloned CDC9, the structural gene for Saccharomyces cerevisiae DNA ligase, and investigated its transcriptional regulation both as a function of cell cycle stage and after UV irradiation. The steady-state level of DNA ligase mRNA increases at least fourfold in late G1, after the completion of start but before S phase. This high level of CDC9 mRNA then decays with an apparent half-life of ca. 20 min and remains at a low basal level throughout the rest of the cell cycle. The accumulation of CDC9 mRNA in late G1 is dependent upon the completion of start but not the CDC7 and CDC8 functions. Exposure of cells to UV light elicits an eightfold increase in DNA ligase mRNA levels.
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Peterson, T. A., L. Prakash, S. Prakash, M. A. Osley, and S. I. Reed. "Regulation of CDC9, the Saccharomyces cerevisiae gene that encodes DNA ligase." Molecular and Cellular Biology 5, no. 1 (January 1985): 226–35. http://dx.doi.org/10.1128/mcb.5.1.226.

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We have cloned CDC9, the structural gene for Saccharomyces cerevisiae DNA ligase, and investigated its transcriptional regulation both as a function of cell cycle stage and after UV irradiation. The steady-state level of DNA ligase mRNA increases at least fourfold in late G1, after the completion of start but before S phase. This high level of CDC9 mRNA then decays with an apparent half-life of ca. 20 min and remains at a low basal level throughout the rest of the cell cycle. The accumulation of CDC9 mRNA in late G1 is dependent upon the completion of start but not the CDC7 and CDC8 functions. Exposure of cells to UV light elicits an eightfold increase in DNA ligase mRNA levels.
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Valdez, MD, Riccardo, William G. Finn, MD, Patricia Uherova, MD, Bertram Schnitzer, MD, and Charles W. Ross, MD. "Nodular Lymphocyte Predominant Hodgkin Lymphoma: An Immunophenotypic Reappraisal Based on a Single-Institution Experience." American Journal of Clinical Pathology 119, no. 2 (February 1, 2003): 192–98. http://dx.doi.org/10.1309/38rk-238f-cdch-5r22.

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Dissertations / Theses on the topic "CDCH"

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Dupré-Maquaire, Janine. "Spectroscopie moléculaire à 10 mu m des molécules toupies symétriques CDH et CDCl spectroscopie STRAK de CDCl avec structure hyperfine." Grenoble 2 : ANRT, 1986. http://catalogue.bnf.fr/ark:/12148/cb37597851v.

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CHIAPPINI, MARCO. "The construction and commissioning of the ultra low mass MEG II drift chamber for the search of the mu^+ --> e^+ gamma decay at branching ratios below 10^(−13)." Doctoral thesis, Università di Siena, 2019. http://hdl.handle.net/11365/1086892.

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The thesis work focuses on the design, construction and commissioning activities of the new cylindrical drift chamber (CDCH) of the MEG II experiment at Paul Scherrer Institut (PSI, Switzerland), in search for the lepton flavour violating mu^+ --> e^+ gamma decay. The first data taken with CDCH fully operational and integrated into the MEG II experimental apparatus are also described.
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CARENZA, CLAUDIA. "DENDRITIC CELL SUBSETS IN THE PATHOGENESIS OF HIGH GRADE GLIOMAS." Doctoral thesis, Università degli Studi di Milano, 2021. http://hdl.handle.net/2434/844781.

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Abstract Background and aims. High grade gliomas (HGGs) are aggressive brain tumours characterized by a poor prognosis and the ability to promote an immunosuppressive tumour microenvironment that impairs anti-tumor immune responses. Therefore, there is increasing interest in developing new immunotherapeutic approaches, aimed at boosting anti-tumor immune responses in HGG patients. Because HGG has shown the highest susceptibility to dendritic cell (DC) vaccines amongst other human cancers, DC-based immunotherapeutic strategies may be particularly promising in these patients. DCs are antigen presenting cells that have the unique ability to initiate antitumor immune responses, making these cells crucial in cancer immunosurveillance. They are a rare population composed of different subsets that differ each other in origin, immunophenotype and function. The differential role of different DC subsets in HGG, and in particular the subsets specifically recruited into the tumour site and the impact of HGG on the activatory/tolerogenic properties of DCs have been poorly investigated, so far. For these reasons, in this study we performed a deep characterization of circulating and tumour-infiltrating DC subsets, and investigated possible correlations between DC parameters and histopathological and molecular HGG features, patient outcome and response to treatment. To this aim, we used multiparameter flow-cytometry and single-cell RNA sequencing (scRNAseq), which allow complex analyses on high-dimensional data. Materials and methods. In this cross-sectional study we enrolled HGG patients undergoing surgery at their first diagnosis, and we applied an 18-colour flow-cytometry panel that allows the identification of DC-lineage DCs (pDCs, cDC1s, cDC2s) and inflammatory DCs (slanDCs, moDCs), and the characterisation of their activatory/inhibitory state. This panel was applied to DC characterization in the peripheral blood (n=23) and the tumour lesion (n=10) of HGG patients. Twelve whole blood samples obtained from healthy donors (HDs) and 3 healthy brain tissue samples were included as controls. scRNAseq experiments were performed on 7 tumoral samples and 2 healthy brain tissues obtained from HGG patients, by using 10x Genomics technology. Ingenuity Pathway Analysis (IPA) software was used to investigate the pathways and functions differentially activated or inhibited in infiltrating DCs. We also performed a longitudinal study on a second cohort of patients, diagnosed with recurrent HGG and enrolled in different immunotherapeutic early clinical trials (ieCTs), mainly containing immune checkpoint inhibitors (n=17). In these patients, we assessed the count and phenotype of circulating DC subsets before and at different time points after immunotherapy, by using the same 18-colour flow-cytometry panel described above. Multivariate analyses were used to correlate DC parameters with the patient outcome. Results. In the cross-sectional study, we observed by flow-cytometry that the frequency of circulating pDCs, cDC1s, cDC2s and slanDCs was significantly lower in HGG patients than HDs. DC reduction was evident only in patients affected by the most severe form of HGGs (IDHwt IV grade gliomas). The analysis of tissue DCs revealed that DC subsets were absent in healthy brain parenchyma, whereas they infiltrated HGG tumour tissues. In particular all subsets of myeloid DCs (including cDC1s, cDC2s, slanDCs, and moDCs) were observed in the tumours, whereas pDCs were observed only in a few patients. Tumour-infiltrating DCs were markedly reduced in corticosteroid-receiving patients. By performing scRNAseq, we confirmed that DCs were mostly absent in healthy brain parenchyma whereas they were present in tumour samples and could be sub-divided in 2 sub-clusters. By IPA analysis, we observed a functional dichotomy between these clusters, with the largest one being characterised by an impaired/dormancy state, as assessed by the down-regulation of pathways and functions related to pro-inflammatory responses, cell motility and cell interactions, compared with the smallest cluster characterised, on the contrary, by a more active profile. In the longitudinal study performed on relapsed HGG patients enrolled in ieCTs, we observed that patients with a positive clinical response to immunotherapeutic agents, as assessed by an increased overall survival, showed an increase in the number of circulating cDCs. Conclusions. This study demonstrated that different subsets of DCs infiltrate human HGGs, but are mainly characterized by a transcriptomic profile suggestive of a functional impairment. These results provide novel insights into the comprehension of the molecular mechanisms of DC impairment in HGG microenvironment, and pave the way for the development of novel strategies aimed at restoring the ability of DCs to activate cytotoxic anti-tumour immune cells. Our observation in the longitudinal study that an increase of cDCs correlated with a better clinical response to immunotherapy seems to support the relevant role played by DCs in the control of HGG growth. On the other hand, our study also demonstrated that corticosteroid treatment, commonly used in HGG patients for the management of cerebral oedema, reduces the number of tumour-infiltrating DCs. Based on the above considerations, this finding may suggest a negative impact of corticosteroid treatment on anti-tumour immune responses, thus supporting the use of alternative approaches to control this clinical complication. Altogether, our results support and encourage the study of DCs in HGG, in order to improve our knowledge on the role played by DCs within the immunosuppressive tumour microenvironment that characterizes this human cancer. To this aim, in the near future we plan to apply new bioinformatic tools to the analysis of single-cell data collected in HGG tumour environment that may be particularly useful for investigating the intricate interactions occurring between DCs and other HGG-infiltrating immune cells or malignant glioma cells.
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Salas, Vargas Natalia E. "CDVCH Centro de difusión del vino chileno." Tesis, Universidad de Chile, 2011. http://www.repositorio.uchile.cl/handle/2250/100425.

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El estudiar el vino como tema para proyecto de titulo, hizo posible la realización de diversas visitas a varias viñas del país. Estas experiencias permitieron la observación de un fenómeno particular: La mayoría de los visitantes de las viñas eran extranjeros, aun cuando estas estuvieran ubicadas en las cercanías de Santiago. Esta inquietud fue reafirmada tiempo después a través de entrevistas realizadas a personas vinculadas al mundo del vino (sommeliers, enólogos, Viñas de Chile), concluyendo todas estas conversaciones en un mismo punto: el poco conocimiento del vino por parte de los chilenos y la disminución en el consumo de esta bebida en el mercado nacional. Al tener noción de esta realidad, el Proyecto de Titulo comenzó a configurarse con mayor claridad. Por esta razón, es que la propuesta arquitectónica de este proyecto se plantea desde la difusión del vino chileno, promoviendo conceptos tan importantes como la historia, la cultura, el territorio y el paisaje que existe entorno al vino de nuestro país. Viendo al vino no solamente como una bebida alcohólica más, sino más bien como un valor tangible que es propio de la nación.
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Harris, Ruth V. "Phosphorylation of linker histones by cdc2 kinase." Thesis, University of Cambridge, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.336626.

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Moyano, Rodríguez Yolanda 1992. "Mitosis exit regulation by Cdc5 and PP2A-Cdc55." Doctoral thesis, Universitat Pompeu Fabra, 2019. http://hdl.handle.net/10803/668051.

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In this thesis we studied the role of PP2ACdc55 in the cytokinesis regulation and the contribution of Cdc5 kinase in the mitosis exit using budding yeast as model. Previously, it was suggested a putative PP2ACdc55 role in cytokinesis based on the elongated phenotype in absence of Cdc55. However, the PP2ACdc55 function during cytokinesis and its direct targets were not identified. In this thesis, we demonstrated that PP2ACdc55 regulates the IPC’s phosphorylation and their residence time at the bud neck. In addition, we showed that PP2ACdc55 coordinates actomyosin ring (AMR) contraction with septa formation. As a second objective, we analyzed the Net1 residues to be phosphorylated by Cdc5 kinase contributing to the Cdc14 release from the nucleolus and mitotic exit progression.
En esta tesis hemos investigado el papel de la fosfatasa PP2ACdc55 en la regulación de la citocinesis y la contribución de la quinasa Cdc5 en la salida de mitosis en la levadura de gemación. Previamente, se había sugerido un posible papel de la PP2ACdc55 en citocinesis basándose en el fenotipo elongado en ausencia de Cdc55. Sin embargo, la función de la PP2ACdc55 durante la citocinesis y sus sustratos no han sido estudiados. En esta tesis, hemos demostrado que la PP2ACdc55 regula la desfosforilación de las proteínas de IPC que regulan la citocinesis; así como, su tiempo de localización en el cuello. Además, hemos observado como la PP2ACdc55 realiza un papel en la coordinación de la contracción del anillo de actomiosina y la formación del septo. En cuanto a Cdc5, analizamos los posibles residuos de Net1 fosforilados por Cdc5 que contribuyen a la liberación de Cdc14 en la salida de mitosis.
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Glasssmith, Gareth. "Characterisation of cdc2-related kinases from Trypanosoma brucei." Thesis, University of Glasgow, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.363169.

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Ko, Tun Kiat. "Characterization of Crk7-a novel Cdc2-related kinase." Thesis, University of Cambridge, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.621784.

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Leligny, Henri. "Etude des cristaux hydratés isolés dans les diagrammes CdCl-HO, CdBr-HO et CdCl-CaCl-HO structures atomiques et propriétés cristallochimiques /." Grenoble 2 : ANRT, 1987. http://catalogue.bnf.fr/ark:/12148/cb37607240v.

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Bahman, A. M. "Studies on the CDC7 gene product of Saccharomyces cerevisiae." Thesis, University of Manchester, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.233154.

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Books on the topic "CDCH"

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Luces entre sombras: La UCV, el CDCH y la investigación universitaria. Caracas: Universidad Central de Venezuela, Consejo de Desarrollo Científico y Humanístico, 2008.

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Coach Drivers Club of Great Britain. CDC yearbook. Yate: CDC, 1999.

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CDC WONDER. Atlanta, Ga: CDC, 1997.

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Eng, Nelson F. Defining human CDC7 functional domains. Sudbury, Ont: Laurentian University, 2001.

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Taylor, Mac. Refocusing CDCR after the 2011 realignment. Sacramento, CA: Legislative Analyst's Office, 2012.

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Instruments, Texas. CDC clock-distribution circuits. [S.l.]: Texas Instruments, 1994.

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NIH/ADAMHA/CDC technology transfer. [Atlanta, Ga.?]: The Centers, 1992.

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CDC and the smallpox crusade. Atlanta, Ga: U.S. Dept. of Health and Human Services, Public Health Service, Centers for Disease Control, 1987.

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Board, National Institutes of Health (U S. ). Patent Policy. NIH/ADAMHA/CDC technology transfer. [Atlanta, Ga.?]: The Centers, 1992.

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Ogden, Horace G. CDC and the smallpox crusade. [Atlanta, Ga.?]: U.S. DHHS, PHS, Centers for Disease Control, 1987.

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Book chapters on the topic "CDCH"

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Masai, Hisao. "Cdc7." In Encyclopedia of Signaling Molecules, 975–90. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_593.

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Masai, Hisao. "Cdc7." In Encyclopedia of Signaling Molecules, 1–16. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4614-6438-9_593-1.

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van Roy, Frans, Volker Nimmrich, Anton Bespalov, Achim Möller, Hiromitsu Hara, Jacob P. Turowec, Nicole A. St. Denis, et al. "Cdc7." In Encyclopedia of Signaling Molecules, 365–73. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_593.

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Cahill, Kenneth V., and Craig N. Czyz. "Conjunctivodacryocystorhinostomy (CDCR)." In Operative Dictations in Ophthalmology, 603–6. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-45495-5_142.

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Cahill, Kenneth V., and Craig N. Czyz. "Conjunctivodacryocystorhinostomy (CDCR)." In Operative Dictations in Ophthalmology, 771–74. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-53058-7_177.

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van Roy, Frans, Volker Nimmrich, Anton Bespalov, Achim Möller, Hiromitsu Hara, Jacob P. Turowec, Nicole A. St. Denis, et al. "Cdc7 (S. cerevisiae)." In Encyclopedia of Signaling Molecules, 373. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_100228.

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Acero, Alejandro. "The CDCN Algorithm." In Acoustical and Environmental Robustness in Automatic Speech Recognition, 81–99. Boston, MA: Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-3122-7_5.

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Lackner, K. J., and D. Peetz. "CDC-Referenzmethode." In Springer Reference Medizin, 548. Berlin, Heidelberg: Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-48986-4_696.

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Lackner, K. J., and D. Peetz. "CDC-Referenzmethode." In Lexikon der Medizinischen Laboratoriumsdiagnostik, 1. Berlin, Heidelberg: Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-662-49054-9_696-1.

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Steele, Guy L., Xiaowei Shen, Josep Torrellas, Mark Tuckerman, Eric J. Bohm, Laxmikant V. Kalé, Glenn Martyna, et al. "CDC 6600." In Encyclopedia of Parallel Computing, 227. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-0-387-09766-4_2107.

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Conference papers on the topic "CDCH"

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Siyal, Amaar, Shehzad Ahmed, Waleed AlAmeri, and Emad W. Al-Shalabi. "A Systematic and Comprehensive Approach for Characterizing Capillary Desaturation Curves in Low-Permeability Carbonate Rocks." In Middle East Oil, Gas and Geosciences Show. SPE, 2023. http://dx.doi.org/10.2118/213425-ms.

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Abstract It is widely recognized that the determination of true residual oil saturation to water (Sorw) is a critical factor in predicting waterflooding performance and implementing enhanced oil recovery (EOR) methods. A particular EOR method will provide a high prospective result if there is an overestimation of Sorw. The concept of capillary desaturation curve (CDC) is used to determine how much amount of oil can be recovered, when implementing a certain EOR technique. The objective of this study is to determine the true residual oil saturation to water (Sorw) for low permeability Indiana limestone outcrops using the centrifuge technique under reservoir conditions and further generate their CDCs. In this work, three carbonate Indiana limestone outcrops with low permeability range (4-8 mD) and representative fluid samples i.e., field oil, formation water, and seawater, were utilized. The CDC was then characterized for carbonate rocks by further reducing Sorw using surfactant flooding where three anionic surfactant formulations with different IFT values were selected. A systematic approach was followed starting with conventional core analysis followed by special core analysis. For the CDC generation via surfactant flooding, three surfactant formulations having different IFT values were selected through a preliminary screening. This study showed that there is no correlation between initial water saturation (Swi) and absolute permeability for the cores tested. In addition, variations in spontaneous oil recovery was noted among cores within the same range of rock permeability, which indirectly indicates the existence of heterogeneity within each rock. Furthermore, a true Sorw of 20-29% was achieved using the centrifuge method, which was confirmed during the surfactant flooding stage. Additionally, CDC studies indicated that a critical trapping number of 10-4 was achieved for the tested cores, which is higher than most of the reported values of 10−8 to 10−6 in the literature. Accordingly, the complete desaturation of mixed-to-oil wet carbonate rocks is quite challenging since it requires a further increase in trapping number, which could possibly be achieved using ultra-low IFT surfactants. This work presents a systematic and comprehensive approach for determining true Sorw and understanding microscopically trapped oil in carbonate rocks based on CDC. The produced results would be useful in EOR screening for future surfactant flooding pilots in carbonate rocks with low permeability.
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Ni, Fan, Xing Lin, and Song Jiang. "SS-CDC." In SYSTOR '19: The 12th ACM International Systems and Storage Conference. New York, NY, USA: ACM, 2019. http://dx.doi.org/10.1145/3319647.3325834.

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"CDCs: Past, Present and Future." In Proceedings of the 45th IEEE Conference on Decision and Control. IEEE, 2006. http://dx.doi.org/10.1109/cdc.2006.376876.

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"CDCs: Past, Present and Future." In 2020 59th IEEE Conference on Decision and Control (CDC). IEEE, 2020. http://dx.doi.org/10.1109/cdc42340.2020.9303823.

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"CDCs: Past, present and future." In 2010 49th IEEE Conference on Decision and Control (CDC 2010). IEEE, 2010. http://dx.doi.org/10.1109/cdc.2010.5717562.

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Elffers, Jan, Jesús Giráldez-Cru, Stephan Gocht, Jakob Nordström, and Laurent Simon. "Seeking Practical CDCL Insights from Theoretical SAT Benchmarks." In Twenty-Seventh International Joint Conference on Artificial Intelligence {IJCAI-18}. California: International Joint Conferences on Artificial Intelligence Organization, 2018. http://dx.doi.org/10.24963/ijcai.2018/181.

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Over the last decades Boolean satisfiability (SAT) solvers based on conflict-driven clause learning (CDCL) have developed to the point where they can handle formulas with millions of variables. Yet a deeper understanding of how these solvers can be so successful has remained elusive. In this work we shed light on CDCL performance by using theoretical benchmarks, which have the attractive features of being a) scalable, b) extremal with respect to different proof search parameters, and c) theoretically easy in the sense of having short proofs in the resolution proof system underlying CDCL. This allows for a systematic study of solver heuristics and how efficiently they search for proofs. We report results from extensive experiments on a wide range of benchmarks. Our findings include several examples where theory predicts and explains CDCL behaviour, but also raise a number of intriguing questions for further study.
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"Special CDC sessions." In 2008 47th IEEE Conference on Decision and Control. IEEE, 2008. http://dx.doi.org/10.1109/cdc.2008.4738575.

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Asvany, Oskar, Stephan Schlemmer, Sandra Brünken, and Pavol Jusko. "VIBRATIONAL AND ROTATIONAL SPECTROSCOPY OF CD2H+." In 71st International Symposium on Molecular Spectroscopy. Urbana, Illinois: University of Illinois at Urbana-Champaign, 2016. http://dx.doi.org/10.15278/isms.2016.wi03.

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Parkinson, Jay, Jerome E. Dennis, and Elaine Parkinson. "When do FDA/CDRH requirements apply?" In ILSC® 2017: Proceedings of the International Laser Safety Conference. Laser Institute of America, 2017. http://dx.doi.org/10.2351/1.5056885.

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"Welcome to the 45th IEEE CDC, Invitation by the CDC Chair." In Proceedings of the 45th IEEE Conference on Decision and Control. IEEE, 2006. http://dx.doi.org/10.1109/cdc.2006.376801.

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Reports on the topic "CDCH"

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Chitanvis, Maneesha Elizabeth. LANL biosurveillance tools at CDC. Office of Scientific and Technical Information (OSTI), May 2017. http://dx.doi.org/10.2172/1361464.

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Yamada, R., J. Hawtree, K. Kaczar, R. Leverence, K. McGuire, C. Newman-Holmes, E. E. Schmidt, and J. Shallenberger. CDC field mapping device - ''ROTOTRACK''. Office of Scientific and Technical Information (OSTI), October 1985. http://dx.doi.org/10.2172/6459052.

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Velazquez-Vargas, Luis G., Tom J. Flynn, Bartev B. Sakadjian, Jinhua Bao, and Tritti Siengchum. 10 MWE CDCL Large Pilot Plant – pre-FEED Study. Office of Scientific and Technical Information (OSTI), April 2020. http://dx.doi.org/10.2172/1638256.

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Galea, Sandro, Lawrence Gostin, Alan B. Cohen, and Nicole Lurie. Eight Operational Suggestions for a Renewed CDC. Milbank Memorial Fund, January 2021. http://dx.doi.org/10.1599/mqop.2021.0105.

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McCarty, Perry L., and Alfred M. Spormann. Mechanisms, Chemistry, and Kinetics of Anaerobic Biodegradation of cDCE and VC. Office of Scientific and Technical Information (OSTI), June 1999. http://dx.doi.org/10.2172/826072.

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Mcarty, Perry L., and Alfred M. Sporman. Mechanisms, Chemistry, and Kinetics of Anaerobic Biodegradation of cDCE and VC. Office of Scientific and Technical Information (OSTI), June 2000. http://dx.doi.org/10.2172/826074.

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Parsa, Z., and E. Courant. Guide to Accelerator Physics Program Synch - CDC Version. Office of Scientific and Technical Information (OSTI), January 1987. http://dx.doi.org/10.2172/1151180.

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Foster, Stephanie, Amy M. Lavery, Suzanne K. Condon, Alisha Etheredge, Kennedy Brian, Svendsen Erik, and Breysse Patrick. Guidelines for examining unusual patterns of cancer and environmental concerns. National Center for Environmental Health (U.S.), December 2022. http://dx.doi.org/10.15620/cdc:122695.

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The Centers for Disease Control and Prevention (CDC) National Center for Environmental Health (NCEH) and the Agency for Toxic Substances and Disease Registry (ATSDR) provide scientific guidance to state, tribal, local, and territorial (STLT) health departments related to environmental health concerns. The guidelines presented here update the 2013 publication, “Investigating Suspected Cancer Clusters and Responding to Community Concerns: Guidelines from the CDC and the Council of State and Territorial Epidemiologists (CSTE)”.1 In general, STLT health departments play the primary role in examining unusual patterns of cancer in communities, including those associated with local environmental concerns. These guidelines expand the approach for these investigations. Occupation-related clusters are not included in these guidelines. Publication date from document properties. CS336302-A Guidelines-for-Examining-Unusual-Patterns-of-Cancer-and-Environmental-Concerns-h.pdf
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Parsa, Zohreh. Guide to VAX, CDC, and IBM 3090 Third Edition. Office of Scientific and Technical Information (OSTI), May 1987. http://dx.doi.org/10.2172/1118914.

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Hoerger, Thomas, Joel Segel, Ping Zhang, and Stephen Sorensen. Validation of the CDC-RTI Diabetes Cost-Effectiveness Model. Research Triangle Park, NC: RTI Press, September 2009. http://dx.doi.org/10.3768/rtipress.2009.mr.0013.0909.

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