Academic literature on the topic 'Cause and treatment of chronic fatigue syndrome'

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Journal articles on the topic "Cause and treatment of chronic fatigue syndrome"

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White, Peter D., and Trudie Chalder. "Chronic fatigue syndrome: treatment without a cause." Lancet 379, no. 9824 (April 2012): 1372–73. http://dx.doi.org/10.1016/s0140-6736(12)60197-4.

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Blitshteyn, Svetlana, and Pradeep Chopra. "Chronic Fatigue Syndrome: From Chronic Fatigue to More Specific Syndromes." European Neurology 80, no. 1-2 (2018): 73–77. http://dx.doi.org/10.1159/000493531.

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In the last decade, a group of chronic disorders associated with fatigue (CDAF) emerged as the leading cause of chronic fatigue, chronic pain, and functional impairment, all of which have been often labeled in clinical practice as chronic fatigue syndrome (CFS) or fibromyalgia. While these chronic disorders arise from various pathophysiologic mechanisms, a shared autoimmune or immune-mediated etiology could shift the focus from symptomatic treatment of fatigue and pain to targeted immunomodulatory and biological therapy. A clinical paradigm shift is necessary to reevaluate CFS and fibromyalgia diagnoses and its relationship to the CDAF entities, which would ultimately lead to a change in diagnostic and therapeutic algorithm for patients with chronic fatigue and chronic pain. Rather than uniformly apply the diagnoses of CFS or fibromyalgia to any patient presenting with unexplained chronic fatigue or chronic pain, it may be more beneficial and therapeutically effective to stratify these patients into more specific diagnoses in the CDAF group.
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Eichner, Edward R. "Chronic Fatigue Syndrome: Searching for the Cause and Treatment." Physician and Sportsmedicine 17, no. 6 (June 1989): 142–52. http://dx.doi.org/10.1080/00913847.1989.11709812.

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Murray, John B. "Psychological Aspects of Chronic Fatigue Syndrome." Perceptual and Motor Skills 74, no. 3_suppl (June 1992): 1123–36. http://dx.doi.org/10.2466/pms.1992.74.3c.1123.

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Geraghty, Keith J., and Charlotte Blease. "Cognitive behavioural therapy in the treatment of chronic fatigue syndrome: A narrative review on efficacy and informed consent." Journal of Health Psychology 23, no. 1 (September 15, 2016): 127–38. http://dx.doi.org/10.1177/1359105316667798.

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Cognitive behavioural therapy is increasingly promoted as a treatment for chronic fatigue syndrome. There is limited research on informed consent using cognitive behavioural therapy in chronic fatigue syndrome. We undertook a narrative review to explore efficacy and to identify the salient information that should be disclosed to patients. We found a complex theoretical model underlying the rationale for psychotherapy in chronic fatigue syndrome. Cognitive behavioural therapy may bring about changes in self-reported fatigue for some patients in the short term, however there is a lack of evidence for long-term benefit or for improving physical function and cognitive behavioural therapy may cause distress if inappropriately prescribed. Therapist effects and placebo effects are important outcome factors.
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Gerwin, Robert D. "A Review of Myofascial Pain and Fibromyalgia – Factors that Promote Their Persistence." Acupuncture in Medicine 23, no. 3 (September 2005): 121–34. http://dx.doi.org/10.1136/aim.23.3.121.

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Chronic muscle pain (myalgia) is a common problem throughout the world. Seemingly simple, it is actually a difficult problem for the clinician interested in determining the aetiology of the pain, as well as in managing the pain. The two common muscle pain conditions are fibromyalgia and myofascial pain syndrome. Fibromyalgia is a chronic, widespread muscle tenderness syndrome, associated with central sensitisation. It is often accompanied by chronic sleep disturbance and fatigue, visceral pain syndromes like irritable bowel syndrome and interstitial cystitis. Myofascial pain syndrome is an overuse or muscle stress syndrome characterised by the presence of trigger points in muscle. The problem these syndromes pose lies not in making the diagnosis of muscle pain. Rather, it is the need to identify the underlying cause(s) of persistent or chronic muscle pain in order to develop a specific treatment plan. Chronic myalgia may not improve until the underlying precipitating or perpetuating factor(s) are themselves managed. Precipitating or perpetuating causes of chronic myalgia include structural or mechanical causes like scoliosis, localised joint hypomobility, or generalised or local joint laxity; and metabolic factors like depleted tissue iron stores, hypothyroidism or Vitamin D deficiency. Sometimes, correction of an underlying cause of myalgia is all that is needed to resolve the condition.
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HARTZ, A. J., S. BENTLER, R. NOYES, J. HOEHNS, C. LOGEMANN, S. SINIFT, Y. BUTANI, et al. "Randomized controlled trial of Siberian ginseng for chronic fatigue." Psychological Medicine 34, no. 1 (January 2004): 51–61. http://dx.doi.org/10.1017/s0033291703008791.

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Background. Chronic fatigue greatly affects quality of life and is a common reason for consulting a physician. Since conventional therapy is often of limited help, fatigued patients may use herbal treatments. This randomized controlled trial evaluated the effectiveness of Siberian ginseng.Method. Subjects were recruited from advertisements in Iowa (82%) and members of chronic fatigue syndrome support groups (18%). Potential subjects were required to have substantial fatigue [ges ]6 months with no identifiable cause. The mean change in a fatigue measure was compared for placebo and Siberian ginseng at 1 and 2 months. Comparisons were for all subjects and for subjects with characteristics previously identified in the literature as important for categorizing chronic fatigue.Results. Ninety-six subjects were randomized to treatment groups, and 76 provided information at 2 months of follow-up. Fatigue among subjects assigned to either placebo or Siberian ginseng was substantially reduced during the study, but differences between treatment groups were not statistically significant in the full sample. Fatigue severity and duration had a statistically significant interaction with response to Siberian ginseng at the P<0·05 level. Treatment was effective at 2 months for 45 subjects with less severe fatigue (P=0·04 unadjusted for multiple comparisons) and for 41 subjects with fatigue for [ges ]5 years (P=0·09 unadjusted for multiple comparisons).Conclusion. Overall efficacy was not demonstrated. However, the findings of possible efficacy for patients with moderate fatigue suggests that further research may be of value.
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Pinikahana, Jaya, Geoff Holloway, and Neville Millen. "The Limits of Medicine and the Social Consequences for Sufferers of Chronic Fatigue Syndrome." Australian Journal of Primary Health 8, no. 2 (2002): 70. http://dx.doi.org/10.1071/py02030.

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Chronic Fatigue Syndrome (CFS) appears to be made up of several clusters of illness categories acting alone or in tandem to cause the decline of health through; fatigue/exhaustion, sensitivity/allergies, pain, general muscle and joint pains, cognitive impairment and gastro-intestinal problems. This study investigated how patients interpret, evaluate and respond to the complex and varied symptoms of Chronic Fatigue Syndrome. Data were collected from persons with CFS using a survey (n=90) and an interview (n=45). The researchers investigated how chronic fatigue syndrome is diagnosed by medical practitioners, how the label of CFS is determined and the social consequences for the patient. The results confirm the limited ability of the biomedical paradigm to diagnose adequately and treat effectively 'socially constructed' and medically ambiguous illnesses like CFS. In the absence of a legitimated regime of medical treatment for CFS, a range of often expensive treatments are employed by CFS sufferers, from formal use of pharmaceutical drugs through to 'alternative' therapies, including herbal, vitamin, homeopathic, esoteric meditative techniques, spiritual healing and general counselling are taken in no particular order.
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Sîrbu, Oana, Victoriţa Șorodoc, Alexandra Stoica, Alexandr Ceasovchih, Mihai Constantin, Laura Huiban, Gabriela Dumitrescu, Luminiţa Vâţă, and Laurenţiu Șorodoc. "A Rare Cause of Chronic Hepatitis: Celiac Disease." Internal Medicine 15, no. 4 (August 1, 2018): 55–60. http://dx.doi.org/10.2478/inmed-2018-0030.

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AbstractIntroduction. Celiac disease is a chronic bowel disease with a prevalence of 1% in the general population. This condition, immune-mediated, may exhibit multiple extra-intestinal changes, including the liver.Case presentation. We present the case of a 43-year-old patient presenting in our clinic for fatigue, associated with cytolytic and cholestatic hepatic syndrome with an onset of 10 years. During this time, the patient performed multiple investigations with the exclusion of viral, autoimmune etiology, primitive biliary cirrhosis and Wilson's disease. An abdominal ultrasound recorded an elongated, with an infundibular septum gallbladder. Abdominal computer tomography did not detect any changes. The final diagnosis is chronic alithiasic cholecystitis receiving hepatoprotective treatment with symptom relief and improved hepatic disorders. Over the past 2 years, the patient was diagnosed with osteoporosis (T score = -2.7 followed by treatment with Calcium and Vitamin D and improvement in T score to -2.1), and an iron deficiency anemia corrected with oral iron treatment. Upon resuming the anamnesis, we notice the presence of an intermittent bloating associated with diarrhea. Positive anti-transglutaminase antibodies required upper endoscopy with biopsy witch confirmed celiac disease.Conclusion. Despite the rather low prevalence of celiac disease in the etiology of hepatocytolysis, it is important to investigate its presence in the context of hepatic changes with uncertain etiology. This case motivates us to be rigorous in looking for secondary causes of hepatic impairment even in patients with apparently benign changes.
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Missailidis, Daniel, Sarah J. Annesley, and Paul R. Fisher. "Pathological Mechanisms Underlying Myalgic Encephalomyelitis/Chronic Fatigue Syndrome." Diagnostics 9, no. 3 (July 20, 2019): 80. http://dx.doi.org/10.3390/diagnostics9030080.

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The underlying molecular basis of myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) is not well understood. Characterized by chronic, unexplained fatigue, a disabling payback following exertion (“post-exertional malaise”), and variably presenting multi-system symptoms, ME/CFS is a complex disease, which demands a concerted biomedical investigation from disparate fields of expertise. ME/CFS research and patient treatment have been challenged by the lack of diagnostic biomarkers and finding these is a prominent direction of current work. Despite these challenges, modern research demonstrates a tangible biomedical basis for the disorder across many body systems. This evidence is mostly comprised of disturbances to immunological and inflammatory pathways, autonomic and neurological dysfunction, abnormalities in muscle and mitochondrial function, shifts in metabolism, and gut physiology or gut microbiota disturbances. It is possible that these threads are together entangled as parts of an underlying molecular pathology reflecting a far-reaching homeostatic shift. Due to the variability of non-overlapping symptom presentation or precipitating events, such as infection or other bodily stresses, the initiation of body-wide pathological cascades with similar outcomes stemming from different causes may be implicated in the condition. Patient stratification to account for this heterogeneity is therefore one important consideration during exploration of potential diagnostic developments.
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Dissertations / Theses on the topic "Cause and treatment of chronic fatigue syndrome"

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Rusch, Mary L. "Cognitive behavioural treatment of chronic fatigue syndrome: an in-depth case study." Thesis, Rhodes University, 2001. http://hdl.handle.net/10962/d1002555.

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Chronic Fatigue Syndrome (CFS) is a chronic disabling condition characterised by persistent physical and mental fatigue, and a variety of somatic, psychological and neurological symptoms. The aetiology of CFS is unknown and the cause of the illness remains controversial. Cognitive-behavioural therapy (CBT) is a non-pharmacological treatment approach that has been shown to be effective in treating patients with CFS. It is based on a model that hypothesises that certain maladaptive thoughts and dysfunctional beliefs may contribute to self-defeating behaviour that perpetuates symptoms and disability, and thereby impedes recovery. The main aim of this study was to critically examine the cognitive-behavioural approach of CFS outlined by Sharpe, Chalder, Palmer & Wessely (1997) by conducting three in-depth case studies. An additional aim was to present a full cognitive developmental case conceptualisation of CFS based on the theories of Leahy (1996) and Young (1994). This case study evaluated the effectiveness of a CBT treatment programme in a 48-year-old Caucasian woman diagnosed with CFS for three years. The treatment consisted of sixteen sessions of CBT and was evaluated both quantitatively and qualitatively. On the basis of this study, a set of core maladaptive schema in the area of disconnection and rejection was identified. In addition, it was found that specific compensatory schemas had developed to offset the core maladaptive schemas and that this compensatory strategy played an important role in maintaining the condition. The results also showed a significant decline in fatigue coupled with a moderate decline in functional disability and depression. A one-month follow-up interview revealed evidence of a substantial consolidation of therapeutic gains in that the improvements observed at the end of the treatment were sustained. Finally, the feasibility of incorporating a cognitive-developmental and/or schema-focused model into the standard CBT treatment model is discussed and the case study method as a research toll is evaluated in light of the findings.
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Tooley, Gregory Allan, and mikewood@deakin edu au. "The Integrity of the circadian time-keeping system in chronic fatigue syndrome." Deakin University. School of Psychology, 2000. http://tux.lib.deakin.edu.au./adt-VDU/public/adt-VDU20051017.135752.

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Chronic Fatigue Syndrome (CFS) is a debilitating condition in which severe, ongoing fatigue is the most prominent of a complex of somatic, psychological and neuropsychological symptoms. The aetiology of CFS remains uncertain and, to date, efforts to distinguish a clear pathophysiological profile for the disorder have been unsuccessful. Current evidence suggests that, rather than being a discrete disease entity with a single cause, CFS is a clinical condition resulting from the interaction of a number of pathophysiological factors, including acute infections, stress and psychiatric disorder. Recently, there has been some interest in the proposition that disordered circadian time-keeping may contribute to the development and/or course of the illness. The rationale for the investigation of circadian factors in CFS is based on the fact that disorders known to be associated with circadian dysregulation, such as jet lag and shiftwork related syndromes have a high degree of symptomatological overlap with CFS. Also, the presence of circadian disturbance could account, in part, for other phenomenological aspects of CFS, including the high rates of comorbid affective disturbance, and the reports of low-level immune dyregulation among sufferers. While several recent studies have produced some evidence of chronobiological dysregulation in CFS patients, much work remains before conclusions can be drawn about the presence, nature and clinical significance of circadian disturbance in CFS. This thesis describes a series of studies that were designed to systematically investigate: 1. whether CFS is associated with a state of circadian dysregulation, and 2. whether circadian dysregulation contributes significantly to the symptomatology of CFS. The first of the 5 studies reported here compared the circadian patterns of sleep-activity of CFS sufferers with those of healthy controls. Results indicated that CFS patients' sleep-activity cycles were significantly phase delayed compared to controls, and that some aspects of their circadian profiles of sleep-activity were related to some measures of sleep-disturbance and well-being. Studies 2 and 3 investigated the relationship between rhythms of sleep-wake and core temperature in CFS patients and healthy controls. The major finding from these studies was that sleep-wake and core temperature rhythms appear to be less effectively synchronised. Further evidence was collected that suggested that there was a relationship between circadian parameters and symptom measures in the CFS group. While this indicated that circadian dysregulation is linked in some way to the symptoms of CFS, assessment of the actual clinical significance of circadian disturbances required the use of a prospective methodology. The final two studies, therefore, report on a placebo-controlled trial of clinical interventions that were designed to restore circadian integrity to CFS patients, in order to see whether this would lead to a reduction in symptom number or severity. Results indicated that, although patients experienced improvements across a range of measures of symptoms and functional capacity, these were small in magnitude, of unlikely clinical significance, and no greater, in general, to improvements reported by patients who underwent placebo treatment. These results, along with those of the earlier studies, are discussed with respect to their implications regarding the presence and significance of circadian dysregulation. It is concluded that, while they provide evidence that CFS is associated with a degree of both internal and external circadian desynchrony, these findings suggest that circadian dysregulation is likely to be only a peripheral, contributor to the processes that generate and maintain the symptom complex. These findings are discussed with respect to how they contribute to our overall understanding of this multi-dimensional condition, and the implications they have for the continuing effort to investigate the causes and treatment of CFS.
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Sharpe, Michael Christopher. "Patients with chronic disabling medically unexplained fatigue (chronic fatigue syndrome) : a descriptive case series and randomised controlled trial of psychological treatment." Thesis, University of Cambridge, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.271706.

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Zaturenskaya, Mariya. "The Effect of Chronic Fatigue Syndrome Severity Subtype on Treatment Responsiveness." University of Cincinnati / OhioLINK, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1268426679.

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Powell, Pauline. "The efficacy of an educational intervention treatment for patients with chronic fatigue syndrome." Thesis, University of Liverpool, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.343696.

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Roos, Jo. "An investigation into predictors of outcome in pragmatic rehabilitation for chronic fatigue syndrome : dynamic illness cognitions and socialization to the treatment model." Thesis, University of Manchester, 2009. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.503668.

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Hamilton, Charlotte, and Louise Hjelm. "Upplevelsen av fysisk aktivitet vid utmattningssyndrom : En litteraturstudie." Thesis, Högskolan i Borås, Akademin för vård, arbetsliv och välfärd, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:hb:diva-8412.

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Ohälsa i form av stress har tilltagit under de senaste decennierna. Detta har lett till en ökning av antalet individer som årligen drabbas av sjukdomen utmattningssyndrom. Detta syndrom har nyligen accepterats som en egen diagnos men har ännu ingen konkret behandlingsmetod. En del av behandlingen kan utgöras av fysisk aktivitet. Trots detta ger inte alltid fysisk aktivitet önskvärd effekt. Detta kan till viss del bero på svårigheten att uppnå balans för individer med denna diagnos. Syftet med litteraturstudien är att beskriva individens upplevelse av fysisk aktivitet som behandling vid utmattningssyndrom. Författarna har valt att göra en litteraturstudie byggd på tolv artiklar av både kvalitativ och kvantitativ ansats. Detta för att utöka kunskapen inom området. Två kategorier identifierades utifrån artiklarna: “Att leva med utmattningssyndrom” och “Fysisk aktivitet som behandlingsmetod vid utmattningssyndrom”. Nämnda kategorier belyser bland annat svårigheter i nivåanpassning av fysisk aktivitet.  Resultatet visar att om aktiviteten inte individanpassas kan detta resultera i försämring av sjukdomen.
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Hart, M. J. Alexandra. "Action in Chronic Fatigue Syndrome: an Enactive Psycho-phenomenological and Semiotic Analysis of Thirty New Zealand Women's Experiences of Suffering and Recovery." Thesis, University of Canterbury. Social and Political Sciences, 2010. http://hdl.handle.net/10092/5294.

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This research into Chronic Fatigue Syndrome (CFS) presents the results of 60 first-person psycho-phenomenological interviews with 30 New Zealand women. The participants were recruited from the Canterbury and Wellington regions, 10 had recovered. Taking a non-dual, non-reductive embodied approach, the phenomenological data was analysed semiotically, using a graph-theoretical cluster analysis to elucidate the large number of resulting categories, and interpreted through the enactive approach to cognitive science. The initial result of the analysis is a comprehensive exploration of the experience of CFS which develops subject-specific categories of experience and explores the relation of the illness to universal categories of experience, including self, ‘energy’, action, and being-able-to-do. Transformations of the self surrounding being-able-to-do and not-being-able-to-do were shown to elucidate the illness process. It is proposed that the concept ‘energy’ in the participants’ discourse is equivalent to the Mahayana Buddhist concept of ‘contact’. This characterises CFS as a breakdown of contact. Narrative content from the recovered interviewees reflects a reestablishment of contact. The hypothesis that CFS is a disorder of action is investigated in detail. A general model for the phenomenology and functional architecture of action is proposed. This model is a recursive loop involving felt meaning, contact, action, and perception and appears to be phenomenologically supported. It is proposed that the CFS illness process is a dynamical decompensation of the subject’s action loop caused by a breakdown in the process of contact. On this basis, a new interpretation of neurological findings in relation to CFS becomes possible. A neurological phenomenon that correlates with the illness and involves a brain region that has a similar structure to the action model’s recursive loop is identified in previous research results and compared with the action model and the results of this research. This correspondence may identify the brain regions involved in the illness process, which may provide an objective diagnostic test for the condition and approaches to treatment. The implications of this model for cognitive science and CFS should be investigated through neurophenomenological research since the model stands to shed considerable light on the nature of consciousness, contact and agency. Phenomenologically based treatments are proposed, along with suggestions for future research on CFS. The research may clarify the diagnostic criteria for CFS and guide management and treatment programmes, particularly multidimensional and interdisciplinary approaches. Category theory is proposed as a foundation for a mathematisation of phenomenology.
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Turner, Laura. "A systematic review of cognitive behavioural therapy (CBT) for the management and treatment of Chronic Fatigue Syndrome/Myalgic Encephalomyelitis (CFS/ME) and Cognitive Behavioural Therapy (CBT) for CFS/ME : an interpretative phenomenological analysis." Thesis, University of Sheffield, 2014. http://etheses.whiterose.ac.uk/6931/.

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Frazer, Melanie. "A cognitive behavioural treatment programme for chronic fatigue syndrome sufferers." Thesis, 2012. http://hdl.handle.net/10210/7142.

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D.Litt. et Phil.
The goal of this study was to determine the impact of cognitive behavioural therapy on people who meet the international diagnostic criteria for CFS, by specifically looking at symptoms and health behaviour. Whereas CFS was originally only studied from a medical perspective, recent years have seen an increased interest from psychologists. Initially the debate centered around whether CFS was a medical or psychiatric illness, but with the development of health psychology as a recognised field of its own, the focus shifted to the investigation of how medical and psychological factors work together to create the symptomatology of CFS. The approach adopted by the research team was that CFS was a biopsychosocial illness and that biological factors such as genetic predisposition, psychological factors such as cognitions and emotions and social factors such as the pressures of families and society work together to establish the health of a person. The study thus set out to gather information regarding all of these aspects in order to add to the current body of knowledge. As previous research efforts had been critisised regarding the validity of the diagnosis of the CFS patients included in their samples, an important challenge that was faced by the researcher was to establish that all the research group participants did meet with the diagnostic criteria. It also had to be ensured that their symptoms could not be accounted for by any other medical illness that was listed as an exclusion criteria for CFS. In order to achieve this patients who had not already had the necessary medical tests done, were requested to do so. The therapeutic intervention was based on cognitive behavioural principles which had been proven to be effective in the treatment of other illnesses that had a biological as well as psychological , aspect to it, for example anxiety, fibromyalgia and chronic pain. The aspects that were addressed had been found by previous researchers to be of great importance in a therapeutic intervention for CFS. These aspects included illness beliefs, information regarding CFS, locus of control and coping mechanisms, avoidance, activity levels, negative and dysfunctional thoughts, social support, lifestyle changes, stress management, sleep, exercise and personality factors. The specific techniques that were included in the intervention were self monitoring, monitoring and modifying of dysfunctional thoughts, goal setting, relaxation and systematic desensitisation. The research group consisted of 37 patients who met the internationally accepted diagnostic criteria for CFS. The control group was a contrast group of comparable size, which included only people who were free from psychological disorders and medical illnesses. The research group participated in a cognitive behavioural intervention, which was designed around the needs of each individual. They therapy was conducted at a community clinic at Rand Afrikaans University between February 1995 and October 1995. Pretests were completed upon commencement of the therapy and the posttests upon termination of the therapy. The control group completed their pre- and posttests at the average interval of the research group pre- and posttests. In order to make a multi-dimensional assessment of the impact of the cognitive behavioural intervention on CFS, various instruments were ulitised. The first instrument was a biographical questionnaire, which recorded the name, sex, age, income group, highest formal qualification, type of work, etcetera. The second questionnaire was an adapted version of the Support and Service Utilisation Schedule, which was employed to control for the other medical and therapeutic interventions that patients participated in, for example the medication taken, alternative medical practitioners visited and sources of support. The participants were asked to maintain their use of these services that they had been using for a long period and to not embark on any new therapies and treatments while they were partaking in the cognitive behavioural intervention.
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Books on the topic "Cause and treatment of chronic fatigue syndrome"

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Chronic fatigue syndrome. Shaftsbury: Element, 1997.

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M, Gellman Lauren, ed. Chronic fatigue syndrome: A treatment guide. St. Louis, Mo: Quality Medical Pub., 1998.

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M, Gellman Lauren, ed. Chronic fatigue syndrome: A treatment guide. New York: St. Martin's Griffin, 1998.

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Jacobs, Gill. The natural way chronic fatigue syndrome. Shaftesbury, Dorset: Element, 1998.

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Leonard, Jason, ed. Understanding chronic fatigue syndrome: An empirical guide to assessment and treatment. Washington, DC: American Psychological Association, 1998.

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Bharadvaj, Daivati. Natural treatments for chronic fatigue syndrome. Westport, Conn: Praeger, 2008.

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America exhausted: Breakthrough treatments of fatigue and chronic fatigue syndrome. Flint, Mich: Vitality Press, 1997.

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Skelly, Mari. Alternative treatments for fibromyalgia and chronic fatigue syndrome. 2nd ed. Alameda CA: Hunter House, 2007.

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Ali, Majid. The canary and chronic fatigue. 2nd ed. Denville, N.J: Life Span Press, 1995.

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Dr, Murphy Gabrielle, ed. Coping better with chronic fatigue syndrome/myalgic encephalomyelitis: Cognitive behaviour therapy for chronic fatigue syndrome/myalgic encephalomyelitis (CFS/ ME). London: Karnac Books, 2009.

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Book chapters on the topic "Cause and treatment of chronic fatigue syndrome"

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Yao, Fei, Yanli You, Xuan Yin, Dhea Khiati, and Ying Xia. "Acupuncture Treatment for Chronic Fatigue Syndrome." In Translational Acupuncture Research, 165–215. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-16089-0_3.

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Mechanic, David. "Chronic Fatigue Syndrome and the Treatment Process." In Ciba Foundation Symposium 173 - Chronic Fatigue Syndrome, 318–41. Chichester, UK: John Wiley & Sons, Ltd., 2007. http://dx.doi.org/10.1002/9780470514382.ch18.

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Friedberg, Fred, and Leonard A. Jason. "Explanatory models of chronic fatigue syndrome." In Understanding chronic fatigue syndrome: An empirical guide to assessment and treatment., 31–46. Washington: American Psychological Association, 1998. http://dx.doi.org/10.1037/10273-003.

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Dudley, Matthew Z., Daniel A. Salmon, Neal A. Halsey, Walter A. Orenstein, Rupali J. Limaye, Sean T. O’Leary, and Saad B. Omer. "Do Vaccines Cause Fibromyalgia or Chronic Fatigue Syndrome (CFS)?" In The Clinician’s Vaccine Safety Resource Guide, 241–44. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-94694-8_35.

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Sharpe, Michael. "Non-Pharmacological Approaches to Treatment." In Ciba Foundation Symposium 173 - Chronic Fatigue Syndrome, 298–317. Chichester, UK: John Wiley & Sons, Ltd., 2007. http://dx.doi.org/10.1002/9780470514382.ch17.

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Friedberg, Fred, and Leonard A. Jason. "Fatigue rating scales." In Understanding chronic fatigue syndrome: An empirical guide to assessment and treatment., 65–77. Washington: American Psychological Association, 1998. http://dx.doi.org/10.1037/10273-005.

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Friedberg, Fred, and Leonard A. Jason. "An eight-session coping skills treatment program for CFS groups." In Understanding chronic fatigue syndrome: An empirical guide to assessment and treatment., 187–94. Washington: American Psychological Association, 1998. http://dx.doi.org/10.1037/10273-011.

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Friedberg, Fred, and Leonard A. Jason. "History, definition, and prevalence." In Understanding chronic fatigue syndrome: An empirical guide to assessment and treatment., 3–20. Washington: American Psychological Association, 1998. http://dx.doi.org/10.1037/10273-001.

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Friedberg, Fred, and Leonard A. Jason. "Predisposing factors." In Understanding chronic fatigue syndrome: An empirical guide to assessment and treatment., 21–30. Washington: American Psychological Association, 1998. http://dx.doi.org/10.1037/10273-002.

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Friedberg, Fred, and Leonard A. Jason. "Measurement of CFS symptoms." In Understanding chronic fatigue syndrome: An empirical guide to assessment and treatment., 49–64. Washington: American Psychological Association, 1998. http://dx.doi.org/10.1037/10273-004.

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Conference papers on the topic "Cause and treatment of chronic fatigue syndrome"

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Lyan, N. V., V. N. Lyan, and S. D. Votoropin. "Reflex therapy and UHF-acupuncture in the treatment of chronic fatigue syndrome." In 2003 13th International Crimean Conference 'Microwave and Telecommunication Technology' Conference Proceedings. IEEE, 2003. http://dx.doi.org/10.1109/crmico.2003.158751.

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Zeng, Hongbing, and Chengmei Wang. "Research on Prevention and Treatment of Chronic Fatigue Syndrome through Aerobic Exercise." In 2017 5th International Conference on Machinery, Materials and Computing Technology (ICMMCT 2017). Paris, France: Atlantis Press, 2017. http://dx.doi.org/10.2991/icmmct-17.2017.137.

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Parslow, RM, E. Anderson, A. Brigden, M. Rai, N. Mills, and E. Crawley. "G162 Adolescents and their parents’ experiences of internet delivered home treatment for chronic fatigue syndrome/me (fitnet-NHS trial)." In Royal College of Paediatrics and Child Health, Abstracts of the RCPCH Conference–Online, 25 September 2020–13 November 2020. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2020. http://dx.doi.org/10.1136/archdischild-2020-rcpch.133.

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Granberry, Rachael, Nicole Ciavarella, Robert Pettys-Baker, Mary Ellen Berglund, and Brad Holschuh. "No-Power-Required, Touch-Activated Compression Garments for the Treatment of POTS." In 2018 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/dmd2018-6886.

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Postural orthostatic tachycardia syndrome (POTS) is a clinical autonomic disorder characterized by a spike in heart rate and syncope in response to orthostasis, symptoms which then dissipate upon recumbency [1]. Additional symptoms include chronic fatigue, bloating, and nausea [2]. POTS predominately affects females (5:1) between the ages of 15 and 50 years [3]. It is estimated 1 to 3 million are affected by POTS in the United States [4]. While there is no known cure, symptom management requires a multifaceted approach, including physical exercise, counter maneuvers, high salt and fluid intake, and medications, including beta blockers and fludrocortisone [3]. Lower body compression is a core component to POTS treatment, especially during prolonged periods of upright posture. Because POTS disproportionately affects young, otherwise healthy females who have high physical and professional demands, compression garments (CG) are critical to allow this population to carry out their activities of daily living [5].
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Aronis, Ze’ev, Erez Kanka, Eyass Massarwa, Rami Haj-Ali, and Shmuel Einav. "Influence of Microcalcifications on Stress Development Within a Vulnerable Plaque’s Cap." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53197.

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Vulnerable plaques are inflamed, active, and growing lesions which are prone to complications such as rupture, luminal and mural thrombosis, intraplaque hemorrhage, and rapid progression to stenosis. Despite major advances in the prevention and treatment of this disease, it remains the leading cause of morbidity and mortality worldwide, accounting for 30% of all deaths globally [1]. The importance of stress/strain distribution is now well recognized in vascular pathophysiology, specifically in the mechanisms of plaque rupture. Finite element modeling (FEM) and advanced fluid structure interaction (FSI) studies can better characterize coronary stenosis coupling constitutive equations. Mechanical factors such as stress concentrations within a plaque (material fatigue), lesion characteristic (location, size, and composition), and flow patterns are involved in rupture of plaques. Assessment of local mechanical characteristics caused by plaque structure is important for identifying vulnerable plaques and may improve final estimation of the risk for coronary syndrome.
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Toosi, Kevin K., and Michael L. Boninger. "Wrist Kinematics and Ultrasound Measures of the Median Nerve During Computer Keyboarding." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53296.

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Carpal tunnel syndrome (CTS) is a common, costly problem in the general population and particularly in manual workers [1–3], with as many as 3 million individuals experiencing its symptoms and signs, including pain, tingling, numbness, fatigue and weakness in the hands and fingers [4]. Treatment of CTS is estimated to cost over one billion dollars a year [5]. The most prevalent theory for the pathogenesis of CTS is compression of median nerve in the carpal tunnel [6]. Although this theory is widely accepted, the cause of the compression in the carpal tunnel is not fully understood. Epidemiological research has identified several occupational risk factors associated with the development of CTS in general industry including: force, repetition, awkward/static postures, localized mechanical compression, and vibration [7]. Several studies have found greater prevalence of carpal tunnel syndrome in workers with highly repetitive manual jobs [8]. Keyboarding is a highly repetitive daily task, and its association with musculoskeletal disorders of the upper extremity has been a public health concern since the 1980s [1]. However, there are controversial results regarding the association between computer keyboarding and CTS which indicate that we have an insufficient understanding of an association between keyboarding and upper limb neuropathy. Using ultrasonographic techniques, our laboratory was able to explore acute changes in the median nerve following a one-hour keyboarding task [9].
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Reports on the topic "Cause and treatment of chronic fatigue syndrome"

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Smith, M. E. Beth, Heidi D. Nelson, Elizabeth Haney, Miranda Pappas, Monica Daeges, Ngoc Wasson, and Marian McDonagh. Diagnosis and Treatment of Myalgic Encephalomyelitis/Chronic Fatigue Syndrome. Agency for Healthcare Research and Quality, December 2014. http://dx.doi.org/10.23970/ahrqepcerta219.

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Xue, Kaiyang, Yanping Wang, Xianzhu Wang, Pingnan Chen, Caihong Xiao, Jing Fu, and Jin Cui. Clinical efficacy and safety of moxibustion in the treatment of chronic fatigue syndrome: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2021. http://dx.doi.org/10.37766/inplasy2021.4.0031.

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