Academic literature on the topic 'Cartilage'

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Journal articles on the topic "Cartilage"

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Claassen, Horst, Martin Schicht, Bernd Fleiner, Ralf Hillmann, Sebastian Hoogeboom, Bernhard Tillmann, and Friedrich Paulsen. "Different Patterns of Cartilage Mineralization Analyzed by Comparison of Human, Porcine, and Bovine Laryngeal Cartilages." Journal of Histochemistry & Cytochemistry 65, no. 6 (April 7, 2017): 367–79. http://dx.doi.org/10.1369/0022155417703025.

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Laryngeal cartilages undergo a slow ossification process during aging, making them an excellent model for studying cartilage mineralization and ossification processes. Pig laryngeal cartilages are similar to their human counterparts in shape and size, also undergo mineralization, facilitating the study of cartilage mineralization. We investigated the processes of cartilage mineralization and ossification and compared these with the known processes in growth plates. Thyroid cartilages from glutaraldehyde-perfused male minipigs and from domestic pigs were used for X-ray, light microscopic, and transmission electron microscopic analyses. We applied different fixation and postfixation solutions to preserve cell shape, proteoglycans, and membranes. In contrast to the ossifying human thyroid cartilage, predominantly cartilage mineralization was observed in minipig and domestic pig thyroid cartilages. The same subset of chondrocytes responsible for growth plate mineralization is also present in thyroid cartilage mineralization. Besides mineralization mediated by matrix vesicles, a second pattern of cartilage mineralization was observed in thyroid cartilage only. Here, the formation and growth of crystals were closely related to collagen fibrils, which served as guide rails for the expansion of mineralization. It is hypothesized that the second pattern of cartilage mineralization may be similar to a maturation of mineralized cartilage after initial matrix vesicles–mediated cartilage mineralization.
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Bender-Heine, Adam, Michelle Russell, Allen Rickards, J. Holmes, Mark Armeni, H. Lambert, and Matthew Zdilla. "Optimal Costal Cartilage Graft Selection According to Cartilage Shape: Anatomical Considerations for Rhinoplasty." Facial Plastic Surgery 33, no. 06 (December 2017): 670–74. http://dx.doi.org/10.1055/s-0037-1607972.

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AbstractCostal cartilage grafting is a commonly used reconstruction procedure, particularly in rhinoplasty. Although costal cartilage is broadly used in reconstructive surgery, there are differing opinions regarding which costal cartilage levels provide the most ideal grafts. Grafts are typically designed to match the shape of the recipient site. The shapes of costal cartilage grafts have been described as “boat-shaped,” “C-shaped,” “canoe-shaped,” “U-shaped,” “crescent-shaped,” “L-shaped,” “semilunar,” “straight,” and “Y-shaped.” The shapes of costal cartilages are thought to lend themselves to the shapes of certain grafts; however, there has been little study of the shapes of costal cartilages, and most reports have been anecdotal. Therefore, this study is aimed to detail the average shapes of the most commonly grafted cartilages (i.e., the fifth to seventh cartilages). A total of 96 cadaveric costal cartilages were analyzed through geometric morphometric analysis. The fifth costal cartilage was determined to have the straightest shape and would therefore be particularly suitable for nasal dorsum onlay grafting. The lateral portions of the sixth and, particularly, the seventh costal cartilages have the most acute curvature. Therefore, they would lend themselves to the construction of an en bloc “L”-shaped or hockey stick-shaped nasal dorsum-columellar strut graft.
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Gong, Huchen, Yutao Men, Xiuping Yang, Xiaoming Li, and Chunqiu Zhang. "Experimental Study on Creep Characteristics of Microdefect Articular Cartilages in the Damaged Early Stage." Journal of Healthcare Engineering 2019 (November 13, 2019): 1–9. http://dx.doi.org/10.1155/2019/8526436.

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Traumatic joint injury is known to cause cartilage deterioration and osteoarthritis. In order to study the mechanical mechanism of damage evolution on articular cartilage, taking the fresh porcine articular cartilage as the experimental samples, the creep experiments of the intact cartilages and the cartilages with different depth defect were carried out by using the noncontact digital image correlation technology. And then, the creep constitutive equations of cartilages were established. The results showed that the creep curves of different layers changed exponentially and were not coincident for the cartilage sample. The defect affected the strain values of the creep curves. The creep behavior of cartilage was dependent on defect depth. The deeper the defect was, the larger the strain value was. The built three-parameter viscoelastic constitutive equation had a good correlation with the experimental results and could predict the creep performance of the articular cartilage. The creep values of the microdefective cartilage in the damaged early stage were different from the diseased articular cartilage. These findings pointed out that defect could accelerate the damage of cartilage. It was helpful to study the mechanical mechanism of damage evolution.
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Wardale, R. J., and V. C. Duance. "Quantification and immunolocalisation of porcine articular and growth plate cartilage collagens." Journal of Cell Science 105, no. 4 (August 1, 1993): 975–84. http://dx.doi.org/10.1242/jcs.105.4.975.

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The collagens of growth plate and articular cartilage from 5–6 month old commercial pigs were characterised. Growth plate cartilage was found to contain less total collagen than articular cartilage as a proportion of the dry weight. Collagen types I, II, VI, IX and XI are present in both growth plate and articular cartilage whereas type X is found exclusively in growth plate cartilage. Types III and V collagen could not be detected in either cartilage. Type I collagen makes up at least 10% of the collagenous component of both cartilages. There are significant differences in the ratios of the quantifiable collagen types between growth plate and articular cartilage. Collagen types I, II, and XI were less readily extracted from growth plate than from articular cartilage following pepsin treatment, although growth plate cartilage contains less of the mature collagen cross-links, hydroxylysyl-pyridinoline and lysyl-pyridinoline. Both cartilages contain significant amounts of the divalent reducible collagen cross-links, hydroxylysyl-ketonorleucine and dehydro-hydroxylysinonorleucine. Immunofluorescent localisation indicated that type I collagen is located predominantly at the surface of articular cartilage but is distributed throughout the matrix in growth plate. Types II and XI are located in the matrix of both cartilages whereas type IX is predominantly pericellular in the calcifying region of articular cartilage and the hypertrophic region of the growth plate. Collagen type VI is located primarily as a diffuse area at the articular surface.
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Korvick, Donna, and Kyriacos Athanasiou. "Variations in the mechanical properties of cartilage from the canine scapulohumeral joint." American Journal of Veterinary Research 58, no. 9 (September 1, 1997): 949–53. http://dx.doi.org/10.2460/ajvr.1997.58.09.949.

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Abstract Objective To measure the intrinsic material properties of scapulohumeral joint cartilage in adult dogs and determine whether regional differences exist within or between the humeral and glenoid cartilages. Samples Paired shoulder joints from 7 clinically normal adult dogs. Procedure An automated indentation apparatus was used to obtain the intrinsic mechanical properties of the cartilage at 7 sites on each joint surface. Results Topographic variations in mechanical properties of the glenoid and humeral cartilages were observed. The largest aggregate modulus (HA) for the humerus was seen at the caudocentral site (0.92 MPa) and for the scapula was seen at the centrocenter site (0.84 MPa). The mean shear modulus (μ) of humeral cartilage (0.23 MPa) was significantly greater than that of the glenoid cartilage (0.19 MPa). The mean Poisson's ratio (ν) of humeral cartilage (0.24) was significantly smaller than that for the glenoid cartilage (0.29). Mean humeral cartilage aggregate modulus (0.71 MPa) was larger than the value for glenoid cartilage (0.67 MPa), but these differences were not significant. There were no significant differences in the compressive stiffness of the opposing cartilage in the canine scapulohumeral joint. Conclusions Differences in mechanical properties between opposing humeral and glenoid cartilages are not a cause of cartilage injury in the scapulohumeral joint of adult dogs. The mechanical properties of cartilage from young dogs with open physes and incomplete subchondral bone plates may be different from those of adult dogs. (Am J Vet Res 1997;58:949–953)
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Wardale, R. J., and V. C. Duance. "Characterisation of articular and growth plate cartilage collagens in porcine osteochondrosis." Journal of Cell Science 107, no. 1 (January 1, 1994): 47–59. http://dx.doi.org/10.1242/jcs.107.1.47.

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The articular and growth plate cartilages of osteochondrotic pigs were examined and compared with those from clinically normal animals. Both types of osteochondrotic cartilage showed considerable localised thickening apparently due to a lack of ossification. Histological examination of cartilage lesions demonstrated a breakdown in the normal pattern of chondrocyte maturation. Articular cartilage lesions lacked mature clones of chondrocytes in the calcifying region. Growth plate cartilage showed an accumulation of disorganised hypertrophic chondrocytes rather than the well-defined columns seen in normal tissue. The overall percentages of collagen in osteochondrotic lesions from both articular and growth plate cartilage were significantly reduced compared with levels in unaffected cartilage. There were substantial increases in the proportion of type I collagen in lesions from both osteochondrotic articular and growth plate cartilages and a reduction in the proportion of type II collagen. Type X collagen was detected in osteochondrotic but not normal articular cartilage. The proportion of type X collagen was unchanged in osteochondrotic growth plate cartilage. The levels of the collagen cross-links, hydroxylysylpyridinoline, hydroxylysyl-ketonorleucine and dehydrohydroxylysinonorleucine were radically reduced in samples from osteochondrotic growth-plate cartilage lesions when compared with normal tissue. Less dramatic changes were observed in articular cartilage although there was a significant decrease in the level of hydroxylysylketonorleucine in osteochondrotic lesions. Immunofluorescence examination of osteochondrotic lesions showed a considerable disruption of the organisation of the collagenous components within both articular and growth-plate cartilages. Normal patterns of staining of types I and VI collagen seen at the articular surface in unaffected tissue were replaced by a disorganised, uneven stain in osteochondrotic articular cartilage lesions. Incomplete removal of cartilage at the ossification front of osteochondrotic growth plate was demonstrated by immunofluorescence staining of type IX collagen. Type X collagen was produced in the matrix of the calcifying region of osteochondrotic articular cartilage by small groups of hypertrophic chondrocytes, but was not detected in normal articular cartilage. The distribution of type X collagen was unchanged in osteochondrotic growth plate cartilage.
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Holmbeck, Kenn, Paolo Bianco, Kali Chrysovergis, Susan Yamada, and Henning Birkedal-Hansen. "MT1-MMP–dependent, apoptotic remodeling of unmineralized cartilage." Journal of Cell Biology 163, no. 3 (November 10, 2003): 661–71. http://dx.doi.org/10.1083/jcb.200307061.

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Skeletal tissues develop either by intramembranous ossification, where bone is formed within a soft connective tissue, or by endochondral ossification. The latter proceeds via cartilage anlagen, which through hypertrophy, mineralization, and partial resorption ultimately provides scaffolding for bone formation. Here, we describe a novel and essential mechanism governing remodeling of unmineralized cartilage anlagen into membranous bone, as well as tendons and ligaments. Membrane-type 1 matrix metalloproteinase (MT1-MMP)–dependent dissolution of unmineralized cartilages, coupled with apoptosis of nonhypertrophic chondrocytes, mediates remodeling of these cartilages into other tissues. The MT1-MMP deficiency disrupts this process and uncouples apoptotic demise of chondrocytes and cartilage degradation, resulting in the persistence of “ghost” cartilages with adverse effects on skeletal integrity. Some cells entrapped in these ghost cartilages escape apoptosis, maintain DNA synthesis, and assume phenotypes normally found in the tissues replacing unmineralized cartilages. The coordinated apoptosis and matrix metalloproteinase-directed cartilage dissolution is akin to metamorphosis and may thus represent its evolutionary legacy in mammals.
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Li, Xue, Jin Duo Ye, Chun Qui Zhang, Qian Qian Tian, Xian Kang Wang, and Li Min Dong. "Numerical Simulation about Stretching Process in Different Layers of Cartilage." Applied Mechanics and Materials 441 (December 2013): 480–83. http://dx.doi.org/10.4028/www.scientific.net/amm.441.480.

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Cartilage with complex structure is a porous viscoelastic material. The direction of arrangement of collagen fibers in different layer regions directly affects the mechanical properties of the cartilage layer region. It is very important to use the method of numerical simulation for studying cartilage damage and repair through experimental measurements of cartilage mechanical parameters of the different layers. Because of the relatively small size of the cartilage, it is very difficult to measure mechanical parameters of cartilages by tensile test. The paper for main problems in the tensile test of cartilages, first by porcine articular cartilage compression testing, measuring the displacement of cartilage areas of different layers, according to the characteristics of the displacement determines the size of areas of different layers of cartilage, and then designed the cartilage and substrate stretching models. Model includes two forms of direct bonding and embedding bonding to simulate stretching process of different layers of the cartilage area in numerical way, displacement fields and stress-strain fields of stretching cartilage in different layer regions are derived. The numerical results show that using the way of embedded bonding can make stress of articular well-distributed without stress concentration, so it is a good way of bonding methods. Paper of the research work laid the foundation for measuring mechanical parameters of cartilage by stretch experiment.
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Wulkan, Marcelo, Alvaro Julio de Andrade Sá, and Nivaldo Alonso. "Modified technique to increase nostril cross-sectional area after using rib and septal cartilage graft over alar nasal cartilages." Acta Cirurgica Brasileira 27, no. 10 (October 2012): 713–19. http://dx.doi.org/10.1590/s0102-86502012001000008.

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PURPOSE: Describe a modified technique to increase nostril cross-sectional area using rib and septal cartilage graft over alar nasal cartilages. METHODS: A modified surgical technique was used to obtain, carve and insert cartilage grafts over alar nasal cartilages. This study used standardized pictures and measured 90 cadaveric nostril cross-sectional area using Autocad®; 30 were taken before any procedure and 60 were taken after grafts over lateral crura (30 using costal cartilage and 30 using septal cartilage). Statistical analysis were assessed using a model for repeated measures and ANOVA (Analysis of Variance) for the variable "area". RESULTS: There's statistical evidence that rib cartilage graft is more effective than septal cartilage graft. The mean area after the insertion of septal cartilage graft is smaller than the mean area under rib graft treatment (no confidence interval for mean difference contains the zero value and all P-values are below the significance level of 5%). CONCLUSIONS: The technique presented is applicable to increase nostril cross section area in cadavers. This modified technique revealed to enhance more nostril cross section area with costal cartilage graft over lateral crura rather than by septal graft.
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Souza, Thiago Sasso Carmona de, João Victor Buttini, Mariana Sasso Carmona de Souza, Pedro Aguiar Soares, Maria Fernanda Pioli Torres, and Caio Marcio Correia Soares. "MORSELIZED CARTILAGE GRAFT: AN ANALYSIS OF THE AREA AND PERIMETER MEASUREMENTS OF THIS VERSATILE OPTION FOR MODERN RHINOPLASTY." Journal of Contemporary Diseases and Advanced Medicine 1, no. 2 (August 1, 2022): 10–24. http://dx.doi.org/10.14436/jcdam.1.2.010-024.oar.

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Introduction: One of the biggest issues in the field of rhinoplasty is the use of grafts, one of which is the morselized cartilage graft. Few studies to date have sought to demonstrate the changes in size suffered by cartilage after the morselization process. Objective: To compare changes in area and perimeter of septal and auricular cartilages after being submitted to two degrees of morselization. Material and Methods: This was a cross-sectional, comparative, and analytical study. Septum and ear cartilages were separated into two groups: Septal Cartilage Group (SCG) and Auricular Cartilage Group (ACG), and evaluated in their intact, and morselized grades 1 and 2 forms. Area and perimeter measurements were taken and compared. Results: A total of 29 cartilages were analyzed, being 20 septal and 9 auricular. There was a significant increase in the measurements in the SCG according to the degree of morselization. Similar results were also obtained in the ACG, but only with a tendency towards significance. When comparing the two groups, no significant differences were identified. Discussion: Morselized cartilages are important options in rhinoplasty. The increase in the degree of morselization leads to larger grafts, but it is also linked to a decrease in cartilage viability. Despite being structurally distinct, auricular and septal cartilages showed a similar behavior of their measurements after morselization. Such information can help surgeons in choosing their grafts, providing more predictable results. Conclusion: Morselization leads to an increase in the area and perimeter of the cartilage, being greater according to the degree of the process, without significant differences between grafts of septal and auricular origin.
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Dissertations / Theses on the topic "Cartilage"

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Prado, Fabio Ornellas. "Avaliação clinicopatologica de condrossarcomas de cabeça e pescoço." [s.n.], 2006. http://repositorio.unicamp.br/jspui/handle/REPOSIP/287860.

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Orientador: Marcio Ajudarte Lopes
Tese (doutorado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba
Made available in DSpace on 2018-08-06T02:39:06Z (GMT). No. of bitstreams: 1 Prado_FabioOrnellas_D.pdf: 1979883 bytes, checksum: 0895b272310133fed868ff45f0215c36 (MD5) Previous issue date: 2006
Resumo: Os condrossarcomas são tumores malignos de etiologia desconhecida, em que as células tumorais formam tecido cartilaginoso. Embora a ocorrência seja rara, principalmente na região de cabeça e pescoço, é o segundo tumor ósseo primário maligno mais freqüente. O objetivo deste trabalho foi correlacionar os dados clinicopatológicos ao prognóstico dos pacientes portadores de condrossarcomas de cabeça e pescoço tratados no Departamento de Cabeça e Pescoço e Otorrinolaringologia do Hospital do Câncer A.C. Camargo. Foram selecionados 16 casos tratados no Hospital do Câncer A.C. Camargo entre 1953 e 2002. A idade média de acometimento no momento do diagnóstico foi de 36,5 anos, variando de 11 a 70 anos. Observou-se ligeira predileção pelo gênero masculino (56,2%). De acordo com a localização, 7 casos (43,8%) acometeram a maxila; 5 casos ocorreram em outros sítios (região etmoidal, fossa nasal [2 casos], fossa infra-temporal, região parietooccipital) e 4 pacientes (25,0%) desenvolveram condrossarcomas em mandíbula. A maioria dos casos foi tratada somente com cirurgia (6 casos, 40% do total). A sobrevida global, observada foi de 66,7% em 3 anos e 56,4% em 5 anos
Abstract: Chondrosarcomas are malignant tumors of unknown etiology, in which tumoral cells form cartilagenous tissue. Although rare in head and neck region, chondrosarcomas are the second primary osseous tumor in frequency. The aim of the present study was correlate clinicopathological data to prognostic of patients with head and neck chondrosarcomas treated in the Head and Neck and Otolaringology Department of the A.C. Camargo Cancer Hospital. There were 16 cases treated in the institution from 1953 to 2002. The mean age at diagnosis was 36.5 years, ranging from 11 to 70 years. A slight male preference was observed (56,2%). According to the location, 7 cases (43,8%) accomited maxilla; 5 cases occured at other sites (ethmoidal region, nasal fossa [2 cases], infratemporal fossa, parieto-occipital region) and 4 patients (25,0%) had mandibular lesions. Most of cases were treated with surgery alone (6 cases, 40%). The observed overall survival was 66,7% for 3-year and 56,4% for 5-year
Doutorado
Patologia
Doutor em Estomatopatologia
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Dickinson, Sally Clare. "Cartilage oligomeric matrix protein and cartilage degradation." Thesis, University of Sheffield, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.323419.

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Girdler, N. M. "The role of mandibular condylar cartilage in articular cartilage repair." Thesis, King's College London (University of London), 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.309110.

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Cook, James L. "Three-dimensional chondrocyte culture : in vitro and in vivo applications /." free to MU campus, to others for purchase, 1998. http://wwwlib.umi.com/cr/mo/fullcit?p9924877.

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Hoch, Johanna M. "SERUM CARTILAGE OLIGOMERIC MATRIX PROTEIN: A BIOMARKER FOR ACUTE ARTICULAR CARTILAGE DAMAGE." UKnowledge, 2012. http://uknowledge.uky.edu/rehabsci_etds/3.

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Bone bruise lesions (BBL) are documented on MRIs diagnosing acute knee ligament injury (AKLI). Recent evidence has indicated that a majority of patients that sustain an AKLI, especially anterior cruciate ligament (ACL) knee injury, will develop post-traumatic osteoarthritis (PTOA) 10-20 years following injury. It has been proposed that the initial damage sustained to the articular cartilage overlying BBL causes a cascade of events that may result in PTOA. Researchers have proposed a modification to treatment protocols for more severe BBL, or have stressed the need for the development of protective therapies to protect the articular cartilage. However, there are limited tools available to evaluate the clinical outcome of articular cartilage overlying BBL. Furthermore, damage to the cartilage overlying BBL may be different according to differing BBL severities. Therefore, the use of a cartilage degradation biomarker, serum cartilage oligomeric matrix protein (sCOMP) and the use of a BBL severity classification system may be useful to determine if differences exist between patients with and without BBL, and with differing BBL severities. The purpose of this dissertation was to investigate the utility of sCOMP as a biomarker for acute articular cartilage damage. The purposes of these studies were to determine the inter and intraday reliability of this marker, to document sCOMP longitudinally in collegiate athletes and following AKLI, and to determine if differences in sCOMP and self-reported pain and function exist for patients with and without BBL, and differing BBL following AKLI. The results of these studies indicated sCOMP measures had strong inter and intraday reliability. Additionally, exercise does seem to influence sCOMP levels; however, these elevations may not be clinically meaningful. Furthermore, sCOMP levels were not different between patients with BBL and without, and between differing BBL severities. The results of these studies support the use of a BBL severity classification for future research studies in order to further elucidate the outcomes of these lesions.
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Hamm, Christopher Allan Soares Marcelo B. "Functional genomic analyses of the impact of global hypomethylation and of tumor microenvironment in a rat model of human chondrosarcoma." [Iowa City, Iowa] : University of Iowa, 2009. http://ir.uiowa.edu/etd/372.

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Wong, Brian Jet-Fei. "Laser mediated cartilage reshaping." [S.l. : Amsterdam : s.n.] ; Universiteit van Amsterdam [Host], 2001. http://dare.uva.nl/document/60182.

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Getgood, Alan Martin John. "Articular cartilage tissue engineering." Thesis, University of Cambridge, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.608764.

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Makower, Anne-Marie. "Regulation of chondrocyte growth i̲n̲ v̲i̲t̲r̲o̲." Stockholm : Kongl. Carolinska Medico Chirurgiska Institutet, 1989. http://books.google.com/books?id=j0pqAAAAMAAJ.

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Tsang, Kwok-yeung. "Molecular pathogenesis of abnormal chondrocyte differentiation in a transgenic mouse model /." View the Table of Contents & Abstract, 2006. http://sunzi.lib.hku.hk/hkuto/record/B35132796.

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Books on the topic "Cartilage"

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Grässel, Susanne, and Attila Aszódi, eds. Cartilage. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-29568-8.

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Grässel, Susanne, and Attila Aszódi, eds. Cartilage. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-53316-2.

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Grässel, Susanne, and Attila Aszódi, eds. Cartilage. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-45803-8.

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Lin, Yunfeng, ed. Cartilage Regeneration. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-51617-2.

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Farr, Jack, and Andreas H. Gomoll, eds. Cartilage Restoration. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-77152-6.

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Farr, Jack, and Andreas H. Gomoll, eds. Cartilage Restoration. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-0427-9.

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Link, Thomas M., ed. Cartilage Imaging. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-8438-8.

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Sabatini, Massimo, Philippe Pastoureau, and Fr�d�ric De Ceuninck. Cartilage and Osteoarthritis. New Jersey: Humana Press, 2004. http://dx.doi.org/10.1385/1592598102.

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De Ceuninck, Fr�d�ric, Massimo Sabatini, and Philippe Pastoureau. Cartilage and Osteoarthritis. New Jersey: Humana Press, 2004. http://dx.doi.org/10.1385/1592598218.

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Smith, David W., Bruce S. Gardiner, Lihai Zhang, and Alan J. Grodzinsky. Articular Cartilage Dynamics. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-1474-2.

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Book chapters on the topic "Cartilage"

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Jiang, Yangzi, Hang Lin, and Rocky S. Tuan. "Overview: State of the Art and Future Prospectives for Cartilage Repair." In Cartilage, 1–34. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-53316-2_1.

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Jahr, Holger. "Tissue Engineering Strategies for Cartilage Repair." In Cartilage, 169–87. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-53316-2_10.

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Goebel, Lars, Liang Gao, and Henning Madry. "Animal Models in Cartilage Repair." In Cartilage, 189–206. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-53316-2_11.

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Narcisi, Roberto, Mairéad A. Cleary, Kavitha Sivasubramaniyan, Pieter A. J. Brama, and Gerjo J. V. M. van Osch. "MSC Populations for Cartilage Regeneration." In Cartilage, 35–57. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-53316-2_2.

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Oellerich, Diana, and Nicolai Miosge. "Chondrogenic Progenitor Cells and Cartilage Repair." In Cartilage, 59–72. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-53316-2_3.

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Diederichs, Solvig, and Wiltrud Richter. "Induced Pluripotent Stem Cells and Cartilage Regeneration." In Cartilage, 73–93. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-53316-2_4.

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Zellner, Johannes, and Peter Angele. "Cell-Based Cartilage Regeneration." In Cartilage, 95–107. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-53316-2_5.

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Meyer, Maximilian A., Atsushi Urita, Brian J. Cole, and Susanna Chubinskaya. "Growth Factors in Cartilage Repair." In Cartilage, 109–25. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-53316-2_6.

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Kon, Elizaveta, Giuseppe Filardo, Berardo Di Matteo, and Maurilio Marcacci. "The Role of Platelet-Rich Plasma in Cartilage Repair." In Cartilage, 127–38. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-53316-2_7.

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Schilling, Arndt F. "Subchondral Bone in Articular Cartilage Regeneration." In Cartilage, 139–50. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-53316-2_8.

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Conference papers on the topic "Cartilage"

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Jindal, Alekh, Jorge Quiané-Ruiz, and Samuel Madden. "CARTILAGE." In the 2013 international conference. New York, New York, USA: ACM Press, 2013. http://dx.doi.org/10.1145/2463676.2465258.

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Manda, Krishnagoud, and Anders Eriksson. "Simulating Metal Implants in Full Thickness Cartilage Defects." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53235.

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Damage or degeneration in the articular cartilage is a major problem that affects millions of people in the world. The biomechanical forces at a site of damage in the cartilage may make the tissue more susceptible to continued long-term degeneration. Various biological treatments are currently available, but all have drawbacks. Alternatively, a contoured articular resurfacing implant is developed to offer a treatment to such full thickness chondral defects [1,3,4]. The main goal of using metal implants, to fill the degenerated portion of the cartilage, is to seal the surrounding cartilage so that further damage can be prevented, and to re-establish the integrity of the joint articulating surface. Many researchers have studied the safety, feasibility and reliability of the metal implants in animal models from a biological point of view [3,4]. They showed promising results. Till date, the mechanical behavior of cartilages surrounding the implant has not been studied, even in animal models. It is essential to understand the time dependent behavior of the cartilages due to biphasic nature of cartilage. Any protrusion of metal implant into the joint cavity damages the opposing soft tissue [1]. In order to avoid this, the positioning of implant together with the behavior of the cartilages immediately surrounding the implant have to be studied.
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Singh, A., A. Vaziri, and H. Nayeb-Hashemi. "A Preliminary Theoretical Investigation for Developing an Artificial Meniscus." In ASME 2004 International Mechanical Engineering Congress and Exposition. ASMEDC, 2004. http://dx.doi.org/10.1115/imece2004-62139.

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Analysis of the stress distribution within cartilages of the human knee joint in response to external loads can help to understand the causes of pathological cartilage degeneration and lead to prevention of injury. Wilson et al [1] demonstrated that by modeling the cartilage material as isotropic, type-2 damage can be explained and with the assumption of transverse isotropic properties, type-1 damage could be explained. In this study, we explore the effect of meniscectomy on the shear stress distribution through the knee joint cartilages by modeling the cartilage as three layers and considering the biphasic properties of the top, middle and deep zones. The results of the analysis indicate that complete meniscectomy of the knee joint induces the maximum shear stress at the cartilages surface to increase substantially. This suggests the need for developing an artificial meniscus that could replace the damaged one.
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Sitterle, Valerie B., and David W. Roberts. "Photoactivated methods for enabling cartilage-to-cartilage tissue fixation." In Biomedical Optics 2003, edited by Lawrence S. Bass, Nikiforos Kollias, Reza S. Malek, Abraham Katzir, Udayan K. Shah, Brian J. F. Wong, Eugene A. Trowers, et al. SPIE, 2003. http://dx.doi.org/10.1117/12.476397.

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Murakami, Teruo, Nobuo Sakai, Yoshinori Sawae, Itaru Ishikawa, Natsuko Hosoda, Emiko Suzuki, and Jun Honda. "Biomechanical Aspects of Natural Articular Cartilage and Regenerated Cartilage." In In Commemoration of the 1st Asian Biomaterials Congress. WORLD SCIENTIFIC, 2008. http://dx.doi.org/10.1142/9789812835758_0028.

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McGann, Megan E., and Diane R. Wagner. "Fabrication of Cartilage-Bone Specimens for Cartilage Wear Testing." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206724.

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Experimental techniques to repair focal defects in articular cartilage involve replacing a small area of damaged cartilage with an artificial implant. An important consideration with these devices is the potential for cartilage to wear against the implant surface. To evaluate these implants and to screen for optimal materials and finishes, a method to quickly and accurately predict in vivo cartilage wear is required. Although pin-on-disc wear testers are frequently used to evaluate the wear of engineering materials, and multi-station test machines are available commercially for rapid testing, they have not often been used to test cartilage wear.
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Blum, Michelle M., and Timothy C. Ovaert. "Synthesis and Characterization of Boundary Lubricant-Functionalized PVA Gels for Biotribological Applications." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19281.

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Hyaline cartilage is a material which exhibits ideal tribological properties by maintaining naturally low friction, leading to high wear resistance in articulating joints. When damage to hyaline cartilage occurs, due to diseases such as osteoarthritis or traumatic tissue injuries, tissue regeneration is limited due to cartilage’s avascular and aneural nature. The resulting bone-on-bone contact causes serious pain and limited mobility. Current treatment options are limited to total or partial joint replacements, which are not ideal procedures due to long term failure of components and osteolysis. A vastly improved material is desirable, which better mimics the structure and excellent tribological behavior of natural cartilage.
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Sobol, Emil N., Victor N. Bagratashvili, Alexander I. Omelchenko, Alexander P. Sviridov, Emmanuel S. Helidonis, George Kavvalos, P. N. Christodoulou, et al. "Laser shaping of cartilage." In OE/LASE '94, edited by R. Rox Anderson. SPIE, 1994. http://dx.doi.org/10.1117/12.184919.

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Goyal, Neeru, and Madhur Gupta. "A Study of Osteoarthritic Human Femoral Articular Cartilage Osteoarthritic Femoral Articular Cartilage." In Annual International Conference on Microscopic and Macroscopic Anatomy. Global Science & Technology Forum (GSTF), 2014. http://dx.doi.org/10.5176/2382-6096_cmma14.10.

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Tamez-Pena, Jose G., Monica Barbu-McInnis, and Saara Totterman. "Knee cartilage extraction and bone-cartilage interface analysis from 3D MRI data sets." In Medical Imaging 2004, edited by J. Michael Fitzpatrick and Milan Sonka. SPIE, 2004. http://dx.doi.org/10.1117/12.535902.

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Reports on the topic "Cartilage"

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Spector, Myron, and Hu-Ping Hsu. Shock Wave-Stimulated Periosteum for Cartilage Repair. Fort Belvoir, VA: Defense Technical Information Center, April 2012. http://dx.doi.org/10.21236/ada574132.

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Spector, Myron, and Hu-Ping Hsu. Shock Wave-Stimulated Periosteum for Cartilage Repair. Fort Belvoir, VA: Defense Technical Information Center, December 2012. http://dx.doi.org/10.21236/ada591954.

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Spector, Myron, and Hu-Ping Hsu. Shock Wave-Stimulated Periosteum for Cartilage Repair. Fort Belvoir, VA: Defense Technical Information Center, December 2013. http://dx.doi.org/10.21236/ada600597.

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Huard, Johnny. Articular Cartilage Repair Through Muscle Cell-Based Tissue Engineering. Fort Belvoir, VA: Defense Technical Information Center, March 2011. http://dx.doi.org/10.21236/ada552048.

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Valente, Pedro, Luis Rama, Hugo Sarmento, and Ana Teixeira. Cartilage Oligomeric Matrix Protein (COMP), a potential cartilage destruction biomarker in active and healthy individuals or athletes from different sports. A systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, February 2021. http://dx.doi.org/10.37766/inplasy2021.2.0032.

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Nielson, Olivia, Dave Estrada, Mone't Alberts, Josh Eixenberger, and Raquel Brown. Optimizing ATDC5 Seeding of Graphene Foam for Cartilage Tissue Engineering. Peeref, July 2022. http://dx.doi.org/10.54985/peeref.2207p1842808.

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Lee, Francis Y. Ready to Use Tissue Construct for Military Bone & Cartilage Trauma. Fort Belvoir, VA: Defense Technical Information Center, October 2014. http://dx.doi.org/10.21236/ada613552.

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Pham, Van Phuc. In vivo functions of cartilage tissues formed from spheroid-scaffold complexes. Biomedpress, 2019. http://dx.doi.org/10.15419/arr.2019.1.

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Lee, Francis Y. Ready to Use Tissue Construct for Military Bone & Cartilage Trauma. Fort Belvoir, VA: Defense Technical Information Center, December 2015. http://dx.doi.org/10.21236/ada632352.

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Martin, James A. Promoting Cartilage Stem Cell Activity to Improve Recovery from Joint Fracture. Fort Belvoir, VA: Defense Technical Information Center, March 2012. http://dx.doi.org/10.21236/ada571622.

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