Relevant bibliographies by topics / Cardiovascular system function

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Cardiovascular system function'

Author: Grafiati
Published: 4 June 2021
Last updated: 15 February 2022

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Journal articles on the topic "Cardiovascular system function"

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Wynands, J. Earl. "Haemodynamic monitoring: Cardiovascular system function." Canadian Anaesthetists’ Society Journal 32, no. 3 (May 1985): 288–93. http://dx.doi.org/10.1007/bf03015145.

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Troshina, E. A., M. Y. Yukina, N. A. Ogneva, and N. V. Mazurina. "Thyroid function disorders and cardiovascular system." Clinical and experimental thyroidology 6, no. 1 (March 15, 2010): 12. http://dx.doi.org/10.14341/ket20106112-19.

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Becker, B. F., S. Zahler, C. Seligmann, C. Kupatt, and G. Richard. "Adenosine in the cardiovascular system: Formation and function." Clinical Biochemistry 30, no. 3 (April 1997): 246. http://dx.doi.org/10.1016/s0009-9120(97)87647-3.

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Mehrpouri, Mahdieh, Rafie Hamidpour, and Mohsen Hamidpour. "Cinnamon inhibits platelet function and improves cardiovascular system." Journal of Medicinal Plants 1, no. 73 (March 1, 2020): 1–11. http://dx.doi.org/10.29252/jmp.1.73.1.

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Sedmera, David. "Function and form in the developing cardiovascular system." Cardiovascular Research 91, no. 2 (March 2, 2011): 252–59. http://dx.doi.org/10.1093/cvr/cvr062.

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Watso, Joseph C., and William B. Farquhar. "Hydration Status and Cardiovascular Function." Nutrients 11, no. 8 (August 11, 2019): 1866. http://dx.doi.org/10.3390/nu11081866.

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Hypohydration, defined as a state of low body water, increases thirst sensations, arginine vasopressin release, and elicits renin–angiotensin–aldosterone system activation to replenish intra- and extra-cellular fluid stores. Hypohydration impairs mental and physical performance, but new evidence suggests hypohydration may also have deleterious effects on cardiovascular health. This is alarming because cardiovascular disease is the leading cause of death in the United States. Observational studies have linked habitual low water intake with increased future risk for adverse cardiovascular events. While it is currently unclear how chronic reductions in water intake may predispose individuals to greater future risk for adverse cardiovascular events, there is evidence that acute hypohydration impairs vascular function and blood pressure (BP) regulation. Specifically, acute hypohydration may reduce endothelial function, increase sympathetic nervous system activity, and worsen orthostatic tolerance. Therefore, the purpose of this review is to present the currently available evidence linking acute hypohydration with altered vascular function and BP regulation.
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McMahon, B. R. "Control of cardiovascular function and its evolution in Crustacea." Journal of Experimental Biology 204, no. 5 (March 1, 2001): 923–32. http://dx.doi.org/10.1242/jeb.204.5.923.

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Work in the last decade has shown that crustacean open circulatory systems are highly efficient and controlled in a complex manner. Control occurs at several levels. Myocardial contraction is initiated in the cardiac ganglion but constantly modulated by the central nervous system, both directly via the cardioregulatory nerves and indirectly via the neurohormonal system. Heart rate and stroke volume can be controlled independently and measurements of both are needed to assess cardiac output accurately. Haemolymph outflow from many arthropod hearts is via a complex multiarterial distribution system, and the regional distribution of cardiac output is tightly controlled via cardioarterial valves at the base of each artery. These valves contain innervated muscle, and differential contraction serves to regulate the efflux of oxygenated haemolymph into a particular system. The major influence on both the evolution and control of arthropod open blood vascular systems is efficiency of oxygen uptake and delivery. This influence is illustrated by reference to a variety of crustacean and other arthropod types.
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Brown, James M. "Ion Channels in the Cardiovascular System: Function and Dysfunction." Annals of Vascular Surgery 9, no. 2 (March 1995): 225. http://dx.doi.org/10.1016/s0890-5096(06)60609-2.

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HASSER, EILEEN M., and JULIA A. MOFFITT. "Regulation of Sympathetic Nervous System Function after Cardiovascular Deconditioning." Annals of the New York Academy of Sciences 940, no. 1 (January 25, 2006): 454–68. http://dx.doi.org/10.1111/j.1749-6632.2001.tb03698.x.

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Nichols, Colin G. "Ion Channels in the cardiovascular system: Function and dysfunction." Trends in Pharmacological Sciences 15, no. 12 (December 1994): 470–71. http://dx.doi.org/10.1016/0165-6147(94)90063-9.

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Dissertations / Theses on the topic "Cardiovascular system function"

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Maa, Ming-Hokng 1977. "Alterations in cardiovascular regulation and function assessed using cardiovascular system identification." Thesis, Massachusetts Institute of Technology, 2000. http://hdl.handle.net/1721.1/86525.

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Thesis (S.B. and M.Eng.)--Massachusetts Institute of Technology, Dept. of Electrical Engineering and Computer Science, 2000.
Includes bibliographical references (p. 65-67).
by Ming-Hokng Maa.
S.B.and M.Eng.
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Maksuti, Elira. "Imaging and modeling the cardiovascular system." Doctoral thesis, KTH, Medicinsk bildteknik, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-196538.

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Understanding cardiac pumping function is crucial to guiding diagnosis, predicting outcomes of interventions, and designing medical devices that interact with the cardiovascular system.  Computer simulations of hemodynamics can show how the complex cardiovascular system is influenced by changes in single or multiple parameters and can be used to test clinical hypotheses. In addition, methods for the quantification of important markers such as elevated arterial stiffness would help reduce the morbidity and mortality related to cardiovascular disease. The general aim of this thesis work was to improve understanding of cardiovascular physiology and develop new methods for assisting clinicians during diagnosis and follow-up of treatment in cardiovascular disease. Both computer simulations and medical imaging were used to reach this goal. In the first study, a cardiac model based on piston-like motions of the atrioventricular plane was developed. In the second study, the presence of the anatomical basis needed to generate hydraulic forces during diastole was assessed in heathy volunteers. In the third study, a previously validated lumped-parameter model was used to quantify the contribution of arterial and cardiac changes to blood pressure during aging. In the fourth study, in-house software that measures arterial stiffness by ultrasound shear wave elastography (SWE) was developed and validated against mechanical testing. The studies showed that longitudinal movements of the atrioventricular plane can well explain cardiac pumping and that the macroscopic geometry of the heart enables the generation of hydraulic forces that aid ventricular filling. Additionally, simulations showed that structural changes in both the heart and the arterial system contribute to the progression of blood pressure with age. Finally, the SWE technique was validated to accurately measure stiffness in arterial phantoms.

QC 20161115

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Schenk, Johannes. "Examination of cardiovascular function in conscious hypertensive diabetic rats." Thesis, University of British Columbia, 1991. http://hdl.handle.net/2429/30318.

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This investigation was concerned with measuring aspects of cardiac function in conscious control, diabetic, hypertensive control, and hypertensive diabetic rats. Preliminary studies were conducted to determine catheter suitability and acute responses to atropine and angiotensin II in conscious animals. The catheter-manometer was tested using a square wave impact and was shown to accurately reproduce a left ventricular pressure pulse. Intravenous atropine caused both heart rate and left ventricular +dP/dt to rise. Intravenously administered angiotensin II caused systolic blood pressure to increase dramatically. In this case heart rate fell and +dP/dt was elevated. Hypertension was induced with deoxycorticosterone acetate (DOCA) and saline drinking water. Rats were first made diabetic with streptozotocin (60 mg/kg; i.v.). One week following this, subcutaneous DOCA (25 mg/kg) was administered twice weekly and all animals received saline drinking water. Following 2 and 5 weeks of DOCA treatment rats were catheterized and resting cardiovascular function was measured. DOCA treatment caused increased systolic and diastolic blood pressures to occur in control and diabetic rats at 2 and 5 weeks. Bradycardia was also observed in DOCA-diabetic and DOCA-control rats at 2 and 5 weeks of treatment. Two and 5 week hypertensive diabetic and control rats exhibited elevated -dP/dt and +dP/dt. The rate of contraction was shown to be proportional to the magnitude of systolic blood pressure in all treatment groups. It is concluded that diabetic rats and control rats did not differ in their response to hypertension after 5 weeks of DOCA treatment.
Medicine, Faculty of
Anesthesiology, Pharmacology and Therapeutics, Department of
Graduate
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Heys, Michelle. "Life course determinants of cognitive function and cardiovascularrisk." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B46448160.

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Kemp, Paul R. "Studies on cellular energetics of the heart." Thesis, University of Oxford, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.257820.

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Onyemaechi, Clinton. "Autonomic Nerve Activity and Cardiovascular Function in the Chicken Embryo (Gallus gallus)." Thesis, University of North Texas, 2018. https://digital.library.unt.edu/ark:/67531/metadc1404516/.

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The goal of this study was to build on the historic use of the avian model of development and also to further the knowledge of autonomic nervous system (ANS) regulation of cardiovascular function in vertebrates. Vasoactive drugs sodium nitroprusside, a vasodilator and phenylephrine, a vasoconstrictor were used to study the correlation of cardiovascular function relationship with nerve activity, both sympathetic and parasympathetic (vagal). Additionally, ANG II was used to assess its effects on vagal inhibition. The present study shows that pharmacologically-induced hypertension is associated with a fall in mSNA, indicating that the capacity for sympathetic autonomic cardiovascular regulation is established by late incubation however, late-stage embryonic chickens did not show a significant increase in mSNA during hypotension. The hypotensive response of the embryo was not accompanied by the expected inhibition of vagal discharge; however a slight but insignificant reduction in vagal discharge was noted. When vagal efferent output was isolated, a significant drop in vagal efferent activity was noted in response to hypotension. The present study showed late-stage embryonic chickens lack a vagal response to hypertension in both efferent and sensory limbs. In this study, vagal discharge was reduced from baseline levels in response to Ang II. Collectively, the present study indicates that the lack of a decreased heart rate, in response to increases in Pm caused by Ang II, is due to a central inhibitory action of Ang II on the vagus. Data from the present study suggests that although autonomic interaction with the cardiovascular system in present in late-stage chicken embryos, it is still underdeveloped and possesses a limited capacity.
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Starowicz, Sharon Ann. "A non-dimensional analysis of cardiovascular function and thermoregulation." Thesis, Virginia Polytechnic Institute and State University, 1986. http://hdl.handle.net/10919/101150.

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The cardiovascular system plays a vital role in protecting the body from temperature extremes due to its unique ability to store, transport, and dissipate heat. A comprehensive study of the thermoregulatory aspects of the system is severely limited by its complexity and the interdependency of its many component variables. Before a formal study can be initiated, certain fundamental properties of the cardiovascular system must be established and the physical processes associated with heat and mass transport must first be understood. To this end, over six hundred variables relating to the system's heat transport characteristics were identified. The variables were grouped to form dimensionless quantities using the Buckingham Pi Theorem. Each dimensionless quantity, or parameter, is composed of definable physical quantities that reflect the interaction between various components of the system. From the analysis, a series of reference scales was identified and, in turn, used to facilitate the physical interpretation of each resulting parameter. As a result of this analysis, a working set of physical and experimental quantities was derived to identify significant heat and mass transport processes involved in cardiovascular thermoregulation and to establish the relative rate at which these processes occur.
M.S.
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Chan, Hiu-ting, and 陳曉庭. "The effect of diet intake on vascular function and therapeutic effect of cardiovascular medicine in patients with cardiovascular disease." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hub.hku.hk/bib/B50434342.

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Cardiovascular diseases (CVDs) remain to be the leading causes of morbidity and mortality in Hong Kong and worldwide. Among different modifiable risk factors, dietary pattern is on the major determinant for CVD and overall mortality. Other than pharmacological therapies for cardiovascular risk factors, such as hypertension, hyperlipidemia and diabetes, maintaining a healthy diet is a more sustainable method in general population to prevent CVDs. Current lifestyle intervention in the West countries focus on high intake of fruit and vegetables with more than 400g per day and limited saturated fats with less than 10% of energy, there is very limited data on impact of dietary pattern on CVDs in Chinese. Prior studies among Chinese in Hong Kong have shown that only half of the local population fell within these recommended ranges for fat, saturated fatty acid and cholesterol intakes. Several different dietary patterns have been recommended for CVDs prevention based on: i) food groups, such as Mediterranean diet, the Dietary Approaches to Stop Hypertension (DASH) diet; ii) macronutrients: the low-carbohydrate diet, low glycemic index diet, very-low- fat diet and iii) nutrition or vitamin supplement. However, the effect of different dietary patterns based on modulations of food group, macronutrients and particular micronutrients on vascular structure and function in Chinese subjects is unclear. In the first part of this thesis, the relationships between different dietary pattern and surrogate markers of subclinical atherosclerosis and vascular function in different high risk populations for CVDs were investigated. In Chapter 3, we compared the assessment of dietary pattern in Chinese using different tool, including Food Frequency Questionnaire (FFQ); Dietary Record; and Dietitian assessment. In this study, we demonstrated that suitable dietary assessments tools should be chosen for the assessment of different dietary pattern, according to characteristics of assessments. In Chapter 4, the relationship between the fruit intake and subclinical atherosclerosis as measured by carotid intimal thickness (IMT) was investigated in patient with type II diabetes mellitus (DM). Our results showed that high fruit intake was associated with lower burden of carotid atherosclerosis, independent of level of vitamin intake in patients with type II DM. In Chapter 5, we compared the impact of high carbohydrate diet on arterial stiffness between control subjects without CVDs and patients with high risk for CVDs. Our findings showed that high carbohydrate diet mainly affected patients with established CVDs, and their increased arterial stiffness was associated with an elevation of blood pressure. In Chapter 6, we determined the effect of dietary vitamin intake on oxidative stress in patients with high risk of CVDs. In those high risk patients for CVDs, we demonstrated that increased dietary intake of vitamin A, beta-carotene and alpha tocopherol were associated with decreased oxidative stress, but these relationships were not observed in those control subjects without CVDs. It is likely attributed to the higher systemic oxidative stress levels in patients with high risk of CVDs. On the other hand, food intake may also affect the clinical efficacy of cardiovascular therapies. In particularly, it has been well established that herbal intake which is commonly used by Chinese can affect the anticoagulant effect of warfarin on patients with non-valvular atrial fibrillation (AF). Thus, in this second part of the thesis, we investigated the effect of concomitant herbal intake on anticoagulation control in patients with non-valvular AF treated with warfarin. Our results showed that patients with AF treated with warfarin had limited knowledge on potential interaction between herbal substances in foods and warfarin, in which increased herbal substances intake significantly reduced the percentage time of anticoagulant effect within the therapeutic range. Moreover, a single section of education on knowledge of herbal ingredients did not improve their percentage time of therapeutic range for these patients. In conclusion, these findings suggest that dietary pattern in Chinese might have significant impact of vascular function in patients with type II DM and high risk for CVDs. Moreover, the herbal substances in the diet among Chinese could have significant impact of the therapeutic effects in some of the cardiovascular medications, such as warfarin. Future clinical studies will be needed to confirm these potential beneficial effects of particular diet intake on vascular function in patients with high risks of CVDs as well as potential interaction between herbal substances in Chinese diet and cardiovascular medications.
published_or_final_version
Medicine
Doctoral
Doctor of Philosophy
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Koon, Hon-wai Michael, and 管漢偉. "Role of endothelin-1 and nitric oxide on the cardiovascular function." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2002. http://hub.hku.hk/bib/B31245444.

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Norton, Cynthia Ann. "The Effect of Whole Wild Blueberries on Endothelial Function of the Sprague-Dawley Rat as Related to Cardiovascular Disease." Fogler Library, University of Maine, 2003. http://www.library.umaine.edu/theses/pdf/NortonCA2003.pdf.

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Books on the topic "Cardiovascular system function"

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Genes and cardiovascular function. New York: Springer, 2011.

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M, Lefer Allan, and Gee Marlys H, eds. Leukotrienes in cardiovascular and pulmonary function. New York: Liss, 1985.

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Morphology and function in MRI, cardiovascular and renal systems. Berlin: Springer-Verlag, 1989.

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High-density lipoproteins: Structure, metabolism, function, and therapeutics. Hoboken, N.J: John Wiley & Sons, Inc., 2012.

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Kontush, Anatol, and M. John Chapman. High-density lipoproteins: Structure, metabolism, function, and therapeutics. Hoboken, N.J: John Wiley & Sons, Inc., 2012.

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Your career in cardiovascular technology. Philadelphia: Saunders, 1996.

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Endothelial function and dysfunction: Improving cardiovascular patient care and outcomes in the twenty-first century. 2nd ed. Arvada, Co: 21st Century Press Books for Doctors, 2004.

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Cardiovascular dynamics: A psychophysiological study : behavioral control, type A, task performance, test anxiety, and cardiovascular responses. Berwyn [Pa.]: Swets North America, 1986.

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Ragosta, Michael. Textbook of clinical hemodynamics. Philadelphia, PA: Saunders/Elsevier, 2008.

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Cardiorespiratory assessment of the adult patient: A clinician's guide. Edinburgh: Elsevier, 2012.

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Book chapters on the topic "Cardiovascular system function"

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von Schulthess, Gustav Konrad. "Functional Aspects of the Cardiovascular System." In Morphology and Function in MRI, 33–41. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-73516-5_3.

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White, Perrin C. "Genetic Disorders of the Renin-Angiotensin-Aldsterone System Causing Hypertension." In Endocrinology of Cardiovascular Function, 281–309. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-5569-8_15.

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Hein, Lutz. "The Neuroendocrine Adrenergic System and Cardiovascular Function." In The Cardiovascular Adrenergic System, 117–32. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-13680-6_7.

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Gourgon, René, and Alain Cohen-Solal. "Arterial system, left ventricular structure and function." In Developments in Cardiovascular Medicine, 155–79. Dordrecht: Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-011-0900-0_11.

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Rushmer, Robert F. "Structure and Function of the Cardiovascular System." In Handbook of Research Methods in Cardiovascular Behavioral Medicine, 5–22. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4899-0906-0_1.

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Miyata, A. "Function of PACAP in the Cardiovascular System." In Pituitary Adenylate Cyclase-Activating Polypeptide, 277–88. Boston, MA: Springer US, 2003. http://dx.doi.org/10.1007/978-1-4615-0243-2_12.

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Berra, Cesare C. F., and Mariluce Barrasso. "Thyroid Function and Effects on Cardiovascular System." In Thyroid, Obesity and Metabolism, 221–39. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-80267-7_13.

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Berry, Colin L., and Jorge A. Sosa-Melgarejo. "Structure and function of the arterial system in hypertension." In Developments in Cardiovascular Medicine, 55–72. Dordrecht: Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-011-0900-0_5.

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Redden, John M., Kimberly L. Dodge-Kafka, and Michael S. Kapiloff. "Function to Failure: Compartmentalization of Cardiomyocyte Signaling by A-Kinase-Anchoring Proteins." In Microdomains in the Cardiovascular System, 37–57. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-54579-0_3.

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Gootman, P. M., N. Gootmant, P. D. M. V. Turlapaty, A. C. Yao, B. J. Buckleyt, and B. M. Altura. "Autonomic Regulation of Cardiovascular Function in Neonates." In Ciba Foundation Symposium 83 - Development of the Autonomic Nervous System, 70–93. Chichester, UK: John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470720653.ch4.

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Conference papers on the topic "Cardiovascular system function"

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Stevenson, D. J., P. Kolh, S. Heldmann, B. Lambermont, G. M. Shaw, A. Ghuysen, C. E. Hann, T. Desaive, G. J. Chase, and J. Revie. "Estimating the driver function of a cardiovascular system model." In UKACC International Conference on CONTROL 2010. Institution of Engineering and Technology, 2010. http://dx.doi.org/10.1049/ic.2010.0419.

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Mahmoud, Ahmed M., Daniel H. Cortes, S. Jamal Mustafa, and Osama M. Mukdadi. "High Frequency Precise Ultrasound Imaging System to Assess Mouse Hearts and Blood Vessels." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192836.

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Genetically modified mice provide a powerful tool for understanding the molecular mechanisms and pathogenesis of human cardiovascular diseases like human atherosclerosis [1]. Numerous mouse strains are available today with phenotypes relevant to human cardiovascular diseases [1,2]. These mouse strains have prompted the development of techniques for assessing the cardiovascular function and morphology of living mice. Recently, several imaging techniques have been emerged as promising non-invasive imaging modalities, such as electron-beam computed tomography, magnetic resonance imaging, positron emission tomography, optical coherent tomography, and ultrasound biomicroscopy (UBM) [3,4]. Although these systems are capable of detecting anatomic and functional information, they may not be suitable to image mouse heart vasculatures. The small size and rapid movement of mouse hearts require systems acquiring images using temporal resolution of less than 10 ms with spatial resolution of 100 μm or less [4]. However, in mice, which have extremely small coronary arteries and high heart rates, the coronary circulation constitutes a great challenge for these available imaging techniques.
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Dan, A. M., and T. L. Dragomir. "A model of the control function in the case of constant effort of the cardiovascular system." In 2012 7th IEEE International Symposium on Applied Computational Intelligence and Informatics (SACI). IEEE, 2012. http://dx.doi.org/10.1109/saci.2012.6249996.

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Reimann, G., R. Gerlini, N. Spielmann, E. Heyne, M. Szibor, V. Gailus-Durner, T. Komlodi, et al. "Defect in Complex III of the Mitochondrial Electron Transfer System Affects Cardiac Insulin Sensitivity but Not Contractile Function." In 50th Annual Meeting of the German Society for Thoracic and Cardiovascular Surgery (DGTHG). Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1725679.

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McCombie, Devin, H. Harry Asada, and Andrew Reisner. "Identification of Cardiovascular Dynamics From Peripheral Circulatory Waveforms Using Laguerre Model Blind System ID." In ASME 2004 International Mechanical Engineering Congress and Exposition. ASMEDC, 2004. http://dx.doi.org/10.1115/imece2004-60509.

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A new tool has been developed for characterizing both the global and local dynamic behavior exhibited by peripheral circulatory waveforms. The signals representing these waveforms can be derived from a variety of different cardiovascular sensor modalities measuring changes in pressure, flow, or volume. The correlation that exists between two peripheral circulatory waveform measurements taken at different locations allows this new signal-processing algorithm to identify two compact, low order dynamic models in real-time. These models identify the distinct dynamic behavior of the circulatory paths traveled by the pulsatile waveforms using output measurements alone. This new algorithm is based on a multi-channel blind system identification technique that has been reformulated to use a Laguerre basis function series expansion. Additionally, a new deconvolution algorithm has been developed to allow estimation of the unknown common input using the identified Laguerre models, which are generally inversely unstable. This new approach has been shown to provide accurate identification of vascular hemodynamics when applied to experimental data derived from a swine.
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Alomari, Mahmoud A., Dana M. Shqair, Khaldoon Alawneh, Omar F. Khabour, Mahmoud E. Nazzal, and Monsef A. Sadaqah. "Prayer-related Physical Activities for Cardiovascular Health." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0263.

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Introduction: Muslims are obligated to pray 5 times daily. Each prayer is a number of rakaa completed with a set of physical movements, preferably in the neighborhood masjid. The vascular effects of religious-related physical activities (RRPA) are not known, despite the well-known cardiovascular benefits of regular physical activity. Therefore, the current study examines the relationships of RRPA with vascular measures. Methodology: Arterial and venous indices at rest and after 5 of arterial occlusion were examined in 192 healthy participants (age: 19-85 years) using strain gauge plethysmography. The participants’ RRPA were collected in a 1-to-1 interview, including prayer (PN) and rakaa number (RN) performed, distance (DW) and time (TW) required to walk to the masjid, and the daily (DA) and weekly (WA) attendance to the masjid. Results: Resting blood flow correlated significantly with DW, TW, DA, and WA (r=0.2-0.4; p<0.01). Resting vascular resistance correlated with DW, TW, DA, and WA (r=-0.14-0.2; p<0.05). Resting venous capacitance correlated with DW, TW, DA, and WA (r=0.14-0.17; p<0.05). Resting outflow correlated with DW, TW, DA, and WA (r=0.15-0.2; p<0.05). Occlusion blood flow correlated with DW (r=0.17; p=0.02). Occlusion outflow correlated with DW and DA (r=0.16-0.17; p<0.05). Additionally, arterial and venous indices were greater (p<0.05) in the participants regularly prayed in the masjid. Discussion: The results confirm the importance of physical activities for the circulatory system. Uniquely, the data shows that RRPA might contribute to the maintenance of vascular function.
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Wagenseil, Jessica E., Chris H. Ciliberto, Russel H. Knutsen, Marilyn A. Levy, Attila Kovacs, and Robert P. Mecham. "Developmental Cardiovascular Remodeling Associated With Reduced Elastin Levels in Mice Occurs After Embryonic Day 18." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-204476.

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Elastin provides reversible extensibility and stores energy during the cardiac cycle in large blood vessels. It is only present in vertebrate animals with a closed circulatory system and high, pulsatile blood pressures. Elastin is crucial for proper cardiac function in vertebrates, as mice lacking elastin (eln-/-) die soon after birth with cardiovascular abnormalities including long, tortuous, stenotic, stiff arteries and high left ventricular (LV) pressure with low cardiac output [1, 2].
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Morrison, Tina M., Gilwoo Choi, Polina A. Segalova, Christopher K. Zarins, and Charles A. Taylor. "Age-Related Changes in the Biomechanical Cyclic Strain of the Human Thoracic Aorta." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192028.

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Large vessels in the vascular system undergo progressive degeneration with aging. The widely accepted theory is the aorta enlarges and stiffens with age [1] and vascular compliance decreases [2], which increases systolic pressure, decreases diastolic pressure, and greatly affects cardiovascular health and function. Quantifying the dynamics of an aging human aorta yields insight into changes in the vascular compliance and is vital in understanding pathogenesis and progression of disease, such as atherosclerosis and aneurysm formation [3], along with medical device implantation and design.
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9

de Jonge, Nicky, Frank P. T. Baaijens, and Carlijn C. V. Bouten. "New Tools for Understanding Extracellular Matrix Remodeling at the Micro-Level in Cardiovascular Tissue Engineering." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80098.

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Cardiovascular tissues have a prominent load-bearing function. Collagen fibers in the extracellular matrix provide strength to these tissues. In particular the content and organization of these fibers contribute to overall strength [1]. In case of changes in mechanical demand, collagen content and organization can be adapted, a process referred to as remodeling. Collagen is therefore a key factor when creating load-bearing tissues via tissue engineering (TE). In order to optimize TE constructs we want to control the collagen content and organization, by either mechanical conditioning of the construct [2] or modification of scaffold properties (degradation, structure) [3, 4]. In mechanically-induced remodeling via conditioning strategies in bioreactors, collagen and cells are generally seen as the key players [5]. Collagen is able to bear the load applied on a tissue; cells sense and react to this load [6, 7]. In cardiovascular TE myofibroblasts are the cells of main interest. Myofibroblasts are mechano-sensitive and mechanically inducible [1], and contribute to alterations in the overall collagen architecture. To be able to optimize and control the collagen architecture and remodeling, via mechanical conditioning or scaffolds, an understanding of underlying mechanisms is needed specifically. Our aim is to provide new tools to study remodeling phenomena. Therefore we developed a 3D in vitro model system to study collagen remodeling at the micro level in real-time. Here we apply the model to investigate remodeling in tissues engineered without a carrier material, or scaffold.
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Singal, Ashish, Clarence Ojo, and Rumi Faizer. "Characterization of Pulsatility and Temperature Profile During Reactive Hyperemic Response." In 2018 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/dmd2018-6805.

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Patients with peripheral arterial disease (PAD) have compromised blood flow to their extremities as a result of arterial narrowing. PAD is often associated with impairment in endothelial function which is exaggerated by injury from processes related to cardiovascular risk factors such as ageing, hypertension, hyperlipidemia, diabetes, smoking, and obesity [1]. Furthermore, patients with diabetes often have calcified arteries making standard non-invasive testing non diagnostic [2]. With increase in diabetes prevalence and concomitant PAD, a new non-invasive assessment method of arterial function that has the potential to reflect both arterial tone and response to ischemia reperfusion may be valuable. We have developed a peripheral arterial tonometry (PAT) system (previously described, [3]) that is capable of measuring pulsatility in peripheral digits. We complemented our system with simultaneous peripheral temperature measurements that could not only add value in understanding PAD, but also aid in clinical diagnoses. In this investigation, we characterized our system on healthy individuals before using it on patients suffering from arterial disease in future investigations.
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Reports on the topic "Cardiovascular system function"

1

Functional interaction between cardiovascular and respiratory systems during testing loads. Yuriy S. Vanyushin, Nikolay A. Fedorov, Natalya O. Kuznetsova, March 2020. http://dx.doi.org/10.14526/2070-4798-2020-15-1-169-175.

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