Journal articles: 'Cardiovascular regulation'

1

Speer, Mei Y., and Cecilia M. Giachelli. "Regulation of cardiovascular calcification." Cardiovascular Pathology 13, no. 2 (March 2004): 63–70. http://dx.doi.org/10.1016/s1054-8807(03)00130-3.

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Versteeg, Dirk H. G., Patricia Van Bergen, Roger A. H. Adan, and Dick J. De Wildt. "Melanocortins and cardiovascular regulation." European Journal of Pharmacology 360, no. 1 (October 1998): 1–14. http://dx.doi.org/10.1016/s0014-2999(98)00615-3.

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WEBB, D. "Endothelin and cardiovascular regulation." American Journal of Hypertension 8, no. 4 (April 1995): 18A. http://dx.doi.org/10.1016/0895-7061(95)97440-3.

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Williamson, Jon W., and William P. Morgan. "Cardiovascular regulation: Spect neuroimaging." International Journal of Sport and Exercise Psychology 3, no. 3 (January 2005): 352–62. http://dx.doi.org/10.1080/1612197x.2005.9671777.

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Trinder, John, Joanna Waloszek, Michael J. Woods, and Amy S. Jordan. "Sleep and cardiovascular regulation." Pflügers Archiv - European Journal of Physiology 463, no. 1 (October 26, 2011): 161–68. http://dx.doi.org/10.1007/s00424-011-1041-3.

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Russell, Fraser. "Urotensin II in cardiovascular regulation." Vascular Health and Risk Management Volume 4 (August 2008): 775–85. http://dx.doi.org/10.2147/vhrm.s1983.

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Sik Park, Kwon, Jang Kyu Choi, and Yang Saeng Park. "Cardiovascular Regulation during Water Immersion." APPLIED HUMAN SCIENCE Journal of Physiological Anthropology 18, no. 6 (1999): 233–41. http://dx.doi.org/10.2114/jpa.18.233.

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da Costa Martins, P. A., S. Leptidis, K. Salic, and L. J. De Windt. "MicroRNA Regulation in Cardiovascular Disease." Current Drug Targets 11, no. 8 (August 1, 2010): 900–906. http://dx.doi.org/10.2174/138945010791591322.

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Laederach-Hofmann, K., L. Mussgay, and H. Ruddel. "Autonomic cardiovascular regulation in obesity." Journal of Endocrinology 164, no. 1 (January 1, 2000): 59–66. http://dx.doi.org/10.1677/joe.0.1640059.

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Obese persons suffer from an increased mortality risk supposedly due to cardiovascular disorders related to either continuously lowered parasympathetic or altered sympathetic activation. Our cross-sectional correlation study establishes the relationship between obesity and autonomic regulation as well as salivary cortisol levels. Three patient cohorts were sampled, covering ranges of body mass index (BMI) of 27-32 (n=17), 33-39 (n=13) and above 40 kg/m(2)(n=12), and stratified for age, sex and menopausal status. Autonomic cardiovascular regulation was assessed by use of heart rate variability and continuous blood pressure recordings. Spectral analytical calculation (discrete Fourier transformation) yields indices of sympathetic and parasympathetic activation and baroreflex sensitivity. Morning salivary cortisol was concurrently collected. Contrary to expectation, BMI and waist/hip ratio (WHR) were inversely correlated with sympathetic activity. This was true for resting conditions (r=-0.48, P<0.001; r=-0.33, P<0.05 for BMI and WHR respectively) and for mental challenge (r=-0.42, P<0.01 for BMI). Resting baroreflex sensitivity was strongly related to the degree of obesity at rest (BMI: r=-0.35, P<0.05) and for mental challenge (r=-0.53, P<0.001). Salivary cortisol correlated significantly with waist circumference (r=-0.34, P=0.05). With increasing weight, no overstimulation was found but a depression in sympathetic and parasympathetic activity together with a significant reduction in baroreflex functioning and in salivary cortisol levels.

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Mingarelli, Maurizio. "The cardiovascular system renal regulation." Nephrology @ Point of Care 2, no. 1 (January 2016): pocj.5000201. http://dx.doi.org/10.5301/pocj.5000201.

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The study of kidney physiology and cardiovascular system physiology has long unveiled several points of contract from which the existence of integrated mechanisms between the two systems has readily been inferred. In conclusion, the need is felt to conduct new studies to explore how the physiologic response to neuro-vegetative stimuli correlates to the renal function level indicated by the glomerular filtration rate (GFR) in a view to demonstrating that a decreased GFR results in cardiovascular alterations whose size is directly proportional to the same GFR reduction.

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Mancia, G., G. Grassi, A. Ferrari, and A. Zanchetti. "Reflex Cardiovascular Regulation in Humans." Journal of Cardiovascular Pharmacology 7 (1985): S152—S159. http://dx.doi.org/10.1097/00005344-198500073-00018.

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12

McCloskey, D. I., and Erica K. Potter. "NEUROPEPTIDE Y AND CARDIOVASCULAR REGULATION." Clinical and Experimental Pharmacology and Physiology 18, no. 1 (January 1991): 47–49. http://dx.doi.org/10.1111/j.1440-1681.1991.tb01376.x.

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Bojic, Tijana, Djordje Radak, Biljana Putnikovic, Dragan Alavantic, and Esma Isenovic. "Methodology of monitoring cardiovascular regulation." Vojnosanitetski pregled 69, no. 12 (2012): 1084–90. http://dx.doi.org/10.2298/vsp110707019b.

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Cordes, Kimberly R., and Deepak Srivastava. "MicroRNA Regulation of Cardiovascular Development." Circulation Research 104, no. 6 (March 27, 2009): 724–32. http://dx.doi.org/10.1161/circresaha.108.192872.

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15

Ohtani, K., and S. Dimmeler. "Epigenetic regulation of cardiovascular differentiation." Cardiovascular Research 90, no. 3 (March 2, 2011): 404–12. http://dx.doi.org/10.1093/cvr/cvr019.

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16

Sessa, William C. "MicroRNA Regulation of Cardiovascular Functions." Arteriosclerosis, Thrombosis, and Vascular Biology 31, no. 11 (November 2011): 2369. http://dx.doi.org/10.1161/atvbaha.111.238311.

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17

den Ruijter, Hester M., Gerard Pasterkamp, and Saskia C. A. de Jager. "Adiponectin Regulation in Cardiovascular Disease." Arteriosclerosis, Thrombosis, and Vascular Biology 34, no. 10 (October 2014): 2180–81. http://dx.doi.org/10.1161/atvbaha.114.304380.

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18

Fadel, Paul J. "Nitric Oxide and Cardiovascular Regulation." Hypertension 69, no. 5 (May 2017): 778–79. http://dx.doi.org/10.1161/hypertensionaha.117.08999.

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19

Ansakorpi, Hanna, Juha T. Korpelainen, Päivikki Tanskanen, Heikki V. Huikuri, Antero Koivula, Uolevi Tolonen, Juhani Pyhtinen, Vilho V. Myllylä, and Jouko I. T. Isojärvi. "Cardiovascular Regulation and Hippocampal Sclerosis." Epilepsia 45, no. 8 (July 21, 2004): 933–39. http://dx.doi.org/10.1111/j.0013-9580.2004.65003.x.

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20

RAMAGE, A., and C. VILLALON. "5-Hydroxytryptamine and cardiovascular regulation." Trends in Pharmacological Sciences 29, no. 9 (September 2008): 472–81. http://dx.doi.org/10.1016/j.tips.2008.06.009.

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21

Taylor, Meghan M., and Willis K. Samson. "Adrenomedullin and central cardiovascular regulation." Peptides 22, no. 11 (November 2001): 1803–7. http://dx.doi.org/10.1016/s0196-9781(01)00522-8.

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22

Andresen, Michael C., and James H. Peters. "TRPV1, Hypertension, and Cardiovascular Regulation." Cell Metabolism 12, no. 5 (November 2010): 421. http://dx.doi.org/10.1016/j.cmet.2010.10.004.

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23

Diamond, BJ, H. Kim, J. DeLuca, and DL Cordero. "Cardiovascular regulation in multiple sclerosis." Multiple Sclerosis Journal 1, no. 3 (November 1995): 156–62. http://dx.doi.org/10.1177/135245859500100304.

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Traditional assessments of autonomic nervous system function have depended on invasive and complex procedures. Vagal power, which is the respiratory component of heart rate variability (HRV) is an alternative and non-invasive measure for indexing autonomic nervous control of the heart In the current study, 18 multiple sclerosis (MS) and 20 healthy subjects matched with respect to age, education and intelligence served as subjects. The MS group showed significantly lower vagal power during natural and paced breathing than healthy subjects. Importantly, heart rate did not differ between the two groups. If MS patients exhibit abnormalities in mechanisms mediating cardiac parasympathetic control, the impact on quality of life and vulnerability to adverse cardiac events need to be further evaluated. The results of this study may have implications with respect to the feasibility of using HRV as both a diagnostic and prognostic tool for evaluating parasympathetic nervous system dysfunction and in providing valuable information for developing more effective treatment and rehabilitation strategies.

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24

Pedrazzini, Thierry, Hans R. Brunner, and Bernard Waeber. "Neuropeptide Y and cardiovascular regulation." Current Opinion in Nephrology and Hypertension 2, no. 1 (January 1993): 106–13. http://dx.doi.org/10.1097/00041552-199301000-00016.

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25

Marshall, J. M. "Peripheral chemoreceptors and cardiovascular regulation." Physiological Reviews 74, no. 3 (July 1, 1994): 543–94. http://dx.doi.org/10.1152/physrev.1994.74.3.543.

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26

Török, Tamas, and László Rudas. "Aging and cardiovascular autonomic regulation." Canadian Journal of Anaesthesia 44, no. 6 (June 1997): 677–78. http://dx.doi.org/10.1007/bf03015457.

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27

Mikulášková, B., L. Maletínská, J. Zicha, and J. Kuneš. "The role of food intake regulating peptides in cardiovascular regulation." Molecular and Cellular Endocrinology 436 (November 2016): 78–92. http://dx.doi.org/10.1016/j.mce.2016.07.021.

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28

Shirasaka, Tetsuro, Mayumi Takasaki, and Hiroshi Kannan. "Cardiovascular effects of leptin and orexins." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 284, no. 3 (March 1, 2003): R639—R651. http://dx.doi.org/10.1152/ajpregu.00359.2002.

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Leptin, the product of the obgene, is a satiety factor secreted mainly in adipose tissue and is part of a signaling mechanism regulating the content of body fat. It acts on leptin receptors, most of which are located in the hypothalamus, a region of the brain known to control body homeostasis. The fastest and strongest hypothalamic response to leptin in ob/ob mice occurs in the paraventricular nucleus, which is involved in neuroendocrine and autonomic functions. On the other hand, orexins (orexin-A and -B) or hypocretins (hypocretin-1 and -2) were recently discovered in the hypothalamus, in which a number of neuropeptides are known to stimulate or suppress food intake. These substances are considered important for the regulation of appetite and energy homeostasis. Orexins were initially thought to function in the hypothalamic regulation of feeding behavior, but orexin-containing fibers and their receptors are also distributed in parts of the brain closely associated with the regulation of cardiovascular and autonomic functions. Functional studies have shown that these peptides are involved in cardiovascular and sympathetic regulation. The objective of this article is to summarize evidence on the effects of leptin and orexins on cardiovascular function in vivo and in vitro and to discuss the pathophysiological relevance of these peptides and possible interactions.

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29

Li, Xiang-Nong. "Hepato-cardiovascular response and its regulation." World Journal of Gastroenterology 11, no. 5 (2005): 676. http://dx.doi.org/10.3748/wjg.v11.i5.676.

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30

Bauersachs, Johann, and Thomas Thum. "Biogenesis and Regulation of Cardiovascular MicroRNAs." Circulation Research 109, no. 3 (July 22, 2011): 334–47. http://dx.doi.org/10.1161/circresaha.110.228676.

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31

Hirata, Yukio. "Recent Advance in Cardiovascular Regulation Research." Japanese Circulation Journal 57, supplementIV (1993): 1131–35. http://dx.doi.org/10.1253/jcj.57.supplementiv_1131.

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32

Head, Geoffrey A. "Baroreflexes and Cardiovascular Regulation in Hypertension." Journal of Cardiovascular Pharmacology 26, no. 2 (1995): S7–16. http://dx.doi.org/10.1097/00005344-199512020-00002.

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33

Hirooka, Yoshitaka, Yoji Sagara, Takuya Kishi, and Kenji Sunagawa. "Oxidative Stress and Central Cardiovascular Regulation." Circulation Journal 74, no. 5 (2010): 827–35. http://dx.doi.org/10.1253/circj.cj-10-0153.

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34

FUXE, K., J. A. AGUIRRE, L. F. AGNATI, G. EULER, P. HEDLUND, R. COVEÑAS, M. ZOLI, B. BJELKE, and P. ENEROTH. "Neuropeptide Y and Central Cardiovascular Regulation." Annals of the New York Academy of Sciences 611, no. 1 Central and P (November 1990): 111–32. http://dx.doi.org/10.1111/j.1749-6632.1990.tb48926.x.

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35

Porta, Alberto, Marco Di Rienzo, Niels Wessel, and Juergen Kurths. "Addressing the complexity of cardiovascular regulation." Philosophical Transactions of the Royal Society A: Mathematical, Physical and Engineering Sciences 367, no. 1892 (February 27, 2009): 1215–18. http://dx.doi.org/10.1098/rsta.2008.0292.

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36

Bruneau, Benoit G. "Epigenetic Regulation of the Cardiovascular System." Circulation Research 107, no. 3 (August 6, 2010): 324–26. http://dx.doi.org/10.1161/res.0b013e3181f17dfe.

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37

Guo, Deng-Fu, John J. Reho, Donald A. Morgan, and Kamal Rahmouni. "Cardiovascular Regulation by the Neuronal BBSome." Hypertension 75, no. 4 (April 2020): 1082–90. http://dx.doi.org/10.1161/hypertensionaha.119.14373.

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38

Rocchini, A. P. "Cardiovascular regulation in obesity-induced hypertension." Hypertension 19, no. 1_Suppl (January 1, 1992): I56. http://dx.doi.org/10.1161/01.hyp.19.1_suppl.i56.

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39

Duschek, Stefan, Jana Wörsching, and Gustavo A. Reyes del Paso. "Autonomic cardiovascular regulation and cortical tone." Clinical Physiology and Functional Imaging 35, no. 5 (July 31, 2014): 383–92. http://dx.doi.org/10.1111/cpf.12174.

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40

Suzuki, Jun-ichi, Mitsuaki Isobe, Ryuichi Morisihita, and Ryozo Nagai. "Genetic Regulation for Inflammatory Cardiovascular Disease." Journal of Molecular and Cellular Cardiology 45, no. 4 (October 2008): S6—S7. http://dx.doi.org/10.1016/j.yjmcc.2008.09.610.

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41

BENNETT, T., and S. M. GARDINER. "Involvement of vasopressin in cardiovascular regulation." Cardiovascular Research 19, no. 2 (February 1, 1985): 57–68. http://dx.doi.org/10.1093/cvr/19.2.57.

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42

Fritsche, R., and A. Jacobsson. "Ontogeny of cardiovascular regulation in amphibians." Comparative Biochemistry and Physiology Part A: Molecular & Integrative Physiology 124 (August 1999): S31. http://dx.doi.org/10.1016/s1095-6433(99)90120-4.

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43

Head, Geoffrey A. "Baroreflexes and Cardiovascular Regulation in Hypertension." Journal of Cardiovascular Pharmacology 26 (1995): S7–16. http://dx.doi.org/10.1097/00005344-199506262-00002.

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44

Laederach-Hofmann, Kurt, Lutz Mussgay, and Heinz Rüddel. "293 Autonomic cardiovascular regulation in obesity." International Journal of Psychophysiology 30, no. 1-2 (September 1998): 114–15. http://dx.doi.org/10.1016/s0167-8760(98)90292-6.

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45

Weaver, L. C., and H. V. Sparks. "Neural Regulation of the Cardiovascular System." Annual Review of Physiology 50, no. 1 (October 1988): 509–10. http://dx.doi.org/10.1146/annurev.ph.50.030188.002453.

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46

Porter, J. P., and M. J. Brody. "Spinal vasopressin mechanisms of cardiovascular regulation." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 251, no. 3 (September 1, 1986): R510—R517. http://dx.doi.org/10.1152/ajpregu.1986.251.3.r510.

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Extrahypothalamic vasopressin-containing neurons have been implicated in the central neural control of the cardiovascular system. In the present study we investigated the possibility that vasopressinergic neurons arising from the paraventricular nucleus (PVN) and terminating in the spinal cord are involved in the regulation of vasomotor functions. Vasopressin (1-17 pmol) was injected into the spinal subarachnoid space of conscious rats instrumented with Doppler flow probes and indwelling intrathecal catheters. The peptide produced a dose-related increase in arterial pressure accompanied by vasoconstriction in the mesenteric, renal, and hindquarter vascular beds. Pretreatment, intrathecally, with 0.5 nmol of the vasopressin antagonist d(CH2)5Me(Tyr)AVP completely prevented the increase in arterial pressure expected after subsequent intrathecal injection of vasopressin. However, the changes in arterial pressure and vascular resistances produced by stimulation of the PVN were not affected by the intrathecal antagonist. Stimulation of the PVN in Brattleboro rats, which lack hypothalamic and spinal vasopressin, produced hemodynamic responses similar to those produced in Long-Evans control rats. Taken together, these data suggest that spinal vasopressin can act within the spinal cord to alter vasomotor functions; however, the hemodynamic effects evoked by stimulation of the PVN do not appear to depend on spinal vasopressinergic mechanisms.

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47

Shoemaker, J. Kevin. "Peripheral Chemoreceptor Contributions to Cardiovascular Regulation." Heart Drug 4, no. 4 (2004): 190–200. http://dx.doi.org/10.1159/000082190.

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48

Hall, J. E. "Integration and regulation of cardiovascular function." Advances in Physiology Education 277, no. 6 (December 1999): S174. http://dx.doi.org/10.1152/advances.1999.277.6.s174.

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New methods in molecular biology and genetics have made possible many of the dramatic advances in physiological research that have occurred in recent years. For those of us who spend most of our time in the research laboratory, it si sometimes difficult to avoid a research-oriented, reductionist mind-set when discussing physiology with students. This article illustrates, with a few examples, the importance of conveying a "big picture" conceptual framework before discussing the details of cardiovascular physiology. Also, I have chosen examples from cardiac output and blood pressure regulation that show the importance of discussing cardiovascular physiology in terms of feedback control systems and integrating information from other areas, such as renal and endocrine physiology. Finally, I have highlighted the importance of two principles that I believe are often underemphasized in teaching physiology: mass balance and time dependence of physiological control systems.

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49

Share, L. "Role of vasopressin in cardiovascular regulation." Physiological Reviews 68, no. 4 (October 1988): 1248–84. http://dx.doi.org/10.1152/physrev.1988.68.4.1248.

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50

Minson, Jane, John Chalmers, Guy Drolet, Vimal Kapoor, Ida Llewellyn-Smith, Eric Mills, Margaret Morris, and Paul Pilowsky. "Central serotonergic mechanisms in cardiovascular regulation." Cardiovascular Drugs and Therapy 4, no. 1 (January 1990): 27–32. http://dx.doi.org/10.1007/bf00053423.

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Journal articles on the topic 'Cardiovascular regulation'

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