Journal articles on the topic 'Cardiovascular Disease Mortality'

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1

Ross, Stephanie Maxine. "Cardiovascular Disease Mortality." Holistic Nursing Practice 29, no. 1 (2015): 53–57. http://dx.doi.org/10.1097/hnp.0000000000000066.

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Liu, Jiangmei, Jinlei Qi, Peng Yin, Yunning Liu, Jinling You, Lin Lin, Maigeng Zhou, and Lijun Wang. "Cardiovascular Disease Mortality — China, 2019." China CDC Weekly 3, no. 15 (2021): 323–26. http://dx.doi.org/10.46234/ccdcw2021.087.

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3

Humblet, Olivier, Linda Birnbaum, Eric Rimm, Murray A. Mittleman, and Russ Hauser. "Dioxins and Cardiovascular Disease Mortality." Environmental Health Perspectives 116, no. 11 (November 2008): 1443–48. http://dx.doi.org/10.1289/ehp.11579.

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4

The Lancet. "Salt and cardiovascular disease mortality." Lancet 377, no. 9778 (May 2011): 1626. http://dx.doi.org/10.1016/s0140-6736(11)60657-0.

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Hagger-Johnson, Gareth, Beverly Roberts, David Boniface, Séverine Sabia, G. David Batty, Alexis Elbaz, Archana Singh-Manoux, and Ian J. Deary. "Neuroticism and Cardiovascular Disease Mortality." Psychosomatic Medicine 74, no. 6 (2012): 596–603. http://dx.doi.org/10.1097/psy.0b013e31825c85ca.

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6

Radojčić, Suzana. "Mortality from cardiovascular disease in Serbia." Zdravstvena zastita 39, no. 6 (2010): 51–56. http://dx.doi.org/10.5937/zz1004051r.

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7

Oman, D., and C. E. Thoresen. "Sex differences in cardiovascular disease mortality." American Journal of Public Health 89, no. 9 (September 1999): 1441. http://dx.doi.org/10.2105/ajph.89.9.1441.

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8

D??Addato, S., A. Dormi, C. Borghi, A. Miconi, and A. Gaddi. "Physical Activity and Cardiovascular Disease Mortality." High Blood Pressure & Cardiovascular Prevention 12, no. 3 (2005): 159. http://dx.doi.org/10.2165/00151642-200512030-00041.

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9

Slattery, M. L., and D. R. Jacobs. "Physical Fitness and Cardiovascular Disease Mortality." Journal of Cardiopulmonary Rehabilitation 8, no. 6 (June 1988): 241. http://dx.doi.org/10.1097/00008483-198806000-00013.

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10

Pilz, Stefan, Andreas Tomaschitz, Winfried März, Christiane Drechsler, Eberhard Ritz, Armin Zittermann, Etienne Cavalier, et al. "Vitamin D, cardiovascular disease and mortality." Clinical Endocrinology 75, no. 5 (October 4, 2011): 575–84. http://dx.doi.org/10.1111/j.1365-2265.2011.04147.x.

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11

Alfthan, Georg, Antti Aro, and K. Fred Gey. "Plasma homocysteine and cardiovascular disease mortality." Lancet 349, no. 9049 (February 1997): 397. http://dx.doi.org/10.1016/s0140-6736(97)80014-1.

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12

Friedman, E. "Socioeconomic inequalities in cardiovascular disease mortality." European Heart Journal 22, no. 8 (April 15, 2001): 715. http://dx.doi.org/10.1053/euhj.2000.2455.

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13

SLATTERY, MARTHA L., and DAVID R. JACOBS. "PHYSICAL FITNESS AND CARDIOVASCULAR DISEASE MORTALITY." American Journal of Epidemiology 127, no. 3 (March 1988): 571–80. http://dx.doi.org/10.1093/oxfordjournals.aje.a114832.

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14

Merom, Dafna, Ding Ding, and Emmanuel Stamatakis. "Dancing Participation and Cardiovascular Disease Mortality." American Journal of Preventive Medicine 50, no. 6 (June 2016): 756–60. http://dx.doi.org/10.1016/j.amepre.2016.01.004.

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15

Jose, Powell O., Ariel T. H. Frank, Kristopher I. Kapphahn, Benjamin A. Goldstein, Karen Eggleston, Katherine G. Hastings, Mark R. Cullen, and Latha P. Palaniappan. "Cardiovascular Disease Mortality in Asian Americans." Journal of the American College of Cardiology 64, no. 23 (December 2014): 2486–94. http://dx.doi.org/10.1016/j.jacc.2014.08.048.

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16

Yang, Jin-gang, Jue Li, Changlin Lu, Buaijiaer Hasimu, Yuejin Yang, and Dayi Hu. "Chronic Kidney Disease, All-Cause Mortality and Cardiovascular Mortality Among Chinese Patients with Established Cardiovascular Disease." Journal of Atherosclerosis and Thrombosis 17, no. 4 (2010): 395–401. http://dx.doi.org/10.5551/jat.3061.

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17

Dassanayake, Jayantha, Lyle Gurrin, Warren R. Payne, Vijaya Sundararajan, and Shyamali C. Dharmage. "Cardiovascular Disease Risk in Immigrants." Asia Pacific Journal of Public Health 23, no. 6 (December 15, 2010): 882–95. http://dx.doi.org/10.1177/1010539509360572.

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Objectives: This study systematically reviewed the peer-reviewed literature to establish morbidity and mortality from myocardial infarction (MI) and stroke among immigrant populations. Methods: The review considered only studies published between 1986 and 2008 that provided data on MI or stroke morbidity/mortality among first-generation immigrants. A prespecified search strategy identified 58 studies for possible inclusion. Of these, 12 met the inclusion criteria. Results: Immigrant MI mortality and morbidity varied by host country with no consistent pattern from one country or region. However, there was an overall trend for increasing risk of MI among immigrants worldwide. Chinese and African immigrants had consistently higher stroke mortality. Conclusion: MI and stroke incidence and prevalence among first-generation immigrants are related to both genetic and environmental factors, but the relative contribution of each is unclear. Prospective studies are needed to identify genetic and behavioral characteristics associated with stroke among Chinese immigrant populations.
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18

Soman, C. R., V. R. Kutty, S. Safraj, K. Vijayakumar, K. Rajamohanan, and K. Ajayan. "All-Cause Mortality and Cardiovascular Mortality in Kerala State of India." Asia Pacific Journal of Public Health 23, no. 6 (May 10, 2010): 896–903. http://dx.doi.org/10.1177/1010539510365100.

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Background and aim: In India, Kerala has the best health indicators, having experienced dramatic shifts in mortality and fertility. However, the mortality pattern in Kerala has not been studied. In this article, the authors analyze and report the pattern of mortality in the PROLIFE cohort (n = 161 942). Methods: Data on death were collected through regular home visits using a pretested, structured questionnaire. The data were validated by a physician. The deaths were grouped under disease categories using ICD 10. Results: Out of 4271 deaths recorded during 5 years, diseases of the circulatory system contributed 40%. Coronary heart disease was the leading cause of death in men (31.1%) and women (17.6%). Age-standardized cardiovascular disease (CVD) death rates were 490 for men and 231 for women per 100 000 person years. Conclusion: The burden of CVD deaths in this community now exceeds that of industrialized countries.
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19

Goff, David Calvin, Sadiya Sana Khan, Donald Lloyd-Jones, Donna K. Arnett, Mercedes R. Carnethon, Darwin R. Labarthe, Matthew Shane Loop, et al. "Bending the Curve in Cardiovascular Disease Mortality." Circulation 143, no. 8 (February 23, 2021): 837–51. http://dx.doi.org/10.1161/circulationaha.120.046501.

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More than 40 years after the 1978 Bethesda Conference on the Declining Mortality from Coronary Heart Disease provided the scientific community with a blueprint for systematic analysis to understand declining rates of coronary heart disease, there are indications the decline has ended or even reversed despite advances in our knowledge about the condition and treatment. Recent data show a more complex situation, with mortality rates for overall cardiovascular disease, including coronary heart disease and stroke, decelerating, whereas those for heart failure are increasing. To mark the 40th anniversary of the Bethesda Conference, the National Heart, Lung, and Blood Institute and the American Heart Association cosponsored the “Bending the Curve in Cardiovascular Disease Mortality: Bethesda + 40” symposium. The objective was to examine the immediate and long-term outcomes of the 1978 conference and understand the current environment. Symposium themes included trends and future projections in cardiovascular disease (in the United States and internationally), the evolving obesity and diabetes epidemics, and harnessing emerging and innovative opportunities to preserve and promote cardiovascular health and prevent cardiovascular disease. In addition, participant-led discussion explored the challenges and barriers in promoting cardiovascular health across the lifespan and established a potential framework for observational research and interventions that would begin in early childhood (or ideally in utero). This report summarizes the relevant research, policy, and practice opportunities discussed at the symposium.
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20

Mitter, Sumeet S., Rajesh Vedanthan, Farhad Islami, Akram Pourshams, Hooman Khademi, Farin Kamangar, Christian C. Abnet, et al. "Household Fuel Use and Cardiovascular Disease Mortality." Circulation 133, no. 24 (June 14, 2016): 2360–69. http://dx.doi.org/10.1161/circulationaha.115.020288.

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21

Han, Sae Hwang, Jane L. Tavares, Molly Evans, Jane Saczynski, and Jeffrey A. Burr. "Social Activities, Incident Cardiovascular Disease, and Mortality." Journal of Aging and Health 29, no. 2 (July 8, 2016): 268–88. http://dx.doi.org/10.1177/0898264316635565.

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Objective: This study examined the relationships between social activities, incident cardiovascular disease (CVD), and non-CVD mortality among older adults in the United States. Method: Data from the Health and Retirement Study (2006-2010) were employed. Two measures of social engagement, volunteering and informal helping, along with two measures of social participation, attendance at religious services and social group meetings, were included. Mediation models for health behaviors were estimated. Results: Multinomial logistic regression models demonstrated that volunteering provided the most consistent results in terms of a lower risk of incident CVD and mortality. Furthermore, volunteering at higher time commitments is related to lower CVD incidence and death; informally helping others at a modest time commitment is related to lower risk of death only. Health behaviors mediated the relationships. Social participation was not related to either CVD or mortality. Discussion: Social activity is a modifiable behavior that may be considered a potential health intervention.
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22

Otto, Catherine M. "Heartbeat: reducing inequities in cardiovascular disease mortality." Heart 106, no. 1 (December 16, 2019): 1–2. http://dx.doi.org/10.1136/heartjnl-2019-316324.

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23

Bradshaw, Patrick T., June Stevens, Nikhil Khankari, Susan L. Teitelbaum, Alfred I. Neugut, and Marilie D. Gammon. "Cardiovascular Disease Mortality Among Breast Cancer Survivors." Epidemiology 27, no. 1 (January 2016): 6–13. http://dx.doi.org/10.1097/ede.0000000000000394.

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24

Balkau, Beverley, Marja Pyörälä, Martin Shipley, Anne Forhan, John Jarrett, Eveline Eschwège, and Kalevi Pyörälä. "Non-cardiovascular disease mortality and diabetes mellitus." Lancet 350, no. 9092 (December 1997): 1680. http://dx.doi.org/10.1016/s0140-6736(05)64279-1.

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25

Devuyst, Olivier, and Murielle Bochud. "Uromodulin, kidney function, cardiovascular disease, and mortality." Kidney International 88, no. 5 (November 2015): 944–46. http://dx.doi.org/10.1038/ki.2015.267.

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26

Collins, Allan J. "Cardiovascular Mortality in End-Stage Renal Disease." American Journal of the Medical Sciences 325, no. 4 (April 2003): 163–67. http://dx.doi.org/10.1097/00000441-200304000-00002.

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27

L.Hancockdg, Steven, Richard T. Hoppe, Sandra J. Horning, and Saul A. Rosenberg. "Cardiovascular mortality following treatment of Hodgkin's disease." International Journal of Radiation Oncology*Biology*Physics 17 (January 1989): 141. http://dx.doi.org/10.1016/0360-3016(89)90681-0.

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28

Aleksandrova, K., T. Pischon, and C. Weikert. "Urinary Sodium Excretion and Cardiovascular Disease Mortality." JAMA: The Journal of the American Medical Association 306, no. 10 (September 13, 2011): 1083. http://dx.doi.org/10.1001/jama.2011.1291.

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29

Rebholz, C. M., and J. He. "Urinary Sodium Excretion and Cardiovascular Disease Mortality." JAMA: The Journal of the American Medical Association 306, no. 10 (September 13, 2011): 1083–84. http://dx.doi.org/10.1001/jama.2011.1292.

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30

Bochud, Murielle, Idris Guessous, and Pascal Bovet. "Urinary Sodium Excretion and Cardiovascular Disease Mortality." JAMA 306, no. 10 (September 14, 2011): 1083. http://dx.doi.org/10.1001/jama.2011.1293.

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31

Labarthe, Darwin R., and Peter A. Briss. "Urinary Sodium Excretion and Cardiovascular Disease Mortality." JAMA 306, no. 10 (September 14, 2011): 1083. http://dx.doi.org/10.1001/jama.2011.1294.

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32

Cook, Nancy R. "Urinary Sodium Excretion and Cardiovascular Disease Mortality." JAMA 306, no. 10 (September 14, 2011): 1083. http://dx.doi.org/10.1001/jama.2011.1295.

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33

Oliveira de Abreu-Silva, Erlon, and Aline Marcadenti. "Urinary Sodium Excretion and Cardiovascular Disease Mortality." JAMA 306, no. 10 (September 14, 2011): 1083. http://dx.doi.org/10.1001/jama.2011.1296.

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34

Nguyen, Jennifer L., Wan Yang, Kazuhiko Ito, Thomas D. Matte, Jeffrey Shaman, and Patrick L. Kinney. "Seasonal Influenza Infections and Cardiovascular Disease Mortality." JAMA Cardiology 1, no. 3 (June 1, 2016): 274. http://dx.doi.org/10.1001/jamacardio.2016.0433.

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35

Shlipak, Michael G., Linda F. Fried, Mary Cushman, Teri A. Manolio, Do Peterson, Catherine Stehman-Breen, Anthony Bleyer, Anne Newman, David Siscovick, and Bruce Psaty. "Cardiovascular Mortality Risk in Chronic Kidney Disease." JAMA 293, no. 14 (April 13, 2005): 1737. http://dx.doi.org/10.1001/jama.293.14.1737.

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36

Domanski, Michael, Gary Mitchell, Marc Pfeffer, James D. Neaton, James Norman, Kenneth Svendsen, Richard Grimm, Jerome Cohen, Jeremiah Stamler, and for the MRFIT Research Group. "Pulse Pressure and Cardiovascular Disease–Related Mortality." JAMA 287, no. 20 (May 22, 2002): 2677. http://dx.doi.org/10.1001/jama.287.20.2677.

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37

KRAUS, WILLIAM E., KENNETH E. POWELL, WILLIAM L. HASKELL, KATHLEEN F. JANZ, WAYNE W. CAMPBELL, JOHN M. JAKICIC, RICHARD P. TROIANO, KYLE SPROW, ANDREA TORRES, and KATRINA L. PIERCY. "Physical Activity, All-Cause and Cardiovascular Mortality, and Cardiovascular Disease." Medicine & Science in Sports & Exercise 51, no. 6 (June 2019): 1270–81. http://dx.doi.org/10.1249/mss.0000000000001939.

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38

Honarbakhsh, S., and M. Schachter. "Vitamins and cardiovascular disease." British Journal of Nutrition 101, no. 8 (October 1, 2008): 1113–31. http://dx.doi.org/10.1017/s000711450809123x.

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CVD is a major cause of mortality and morbidity in the Western world. In recent years its importance has expanded internationally and it is believed that by 2020 it will be the biggest cause of mortality in the world, emphasising the importance to prevent or minimise this increase. A beneficial role for vitamins in CVD has long been explored but the data are still inconsistent. While being supported by observational studies, randomised controlled trials have not yet supported a role for vitamins in primary or secondary prevention of CVD and have in some cases even indicated increased mortality in those with pre-existing late-stage atherosclerosis. The superiority of combination therapy over single supplementation has been suggested but this has not been confirmed in trials. Studies have indicated that β-carotene mediates pro-oxidant effects and it has been suggested that its negative effects may diminish the beneficial effects mediated by the other vitamins in the supplementation cocktail. The trials that used a combination of vitamins that include β-carotene have been disappointing. However, vitamin E and vitamin C have in combination shown long-term anti-atherogenic effects but their combined effect on clinical endpoints has been inconsistent. Studies also suggest that vitamins would be beneficial to individuals who are antioxidant-deficient or exposed to increased levels of oxidative stress, for example, smokers, diabetics and elderly patients, emphasising the importance of subgroup targeting. Through defining the right population group and the optimal vitamin combination we could potentially find a future role for vitamins in CVD.
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39

MERCER, A. J. "Long-term trends in cardiovascular disease mortality and association with respiratory disease." Epidemiology and Infection 144, no. 4 (August 5, 2015): 777–86. http://dx.doi.org/10.1017/s0950268815001818.

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SUMMARYThe recent decline in cardiovascular disease mortality in Western countries has been linked with changes in life style and treatment. This study considers periods of decline before effective medical interventions or knowledge about risk factors. Trends in annual age-standardized death rates from cerebrovascular disease, heart disease and circulatory disease, and all cardiovascular disease are reviewed for three phases, 1881–1916, 1920–1939, and 1940–2000. There was a consistent decline in the cerebrovascular disease death rate between 1891 and 2000, apart from brief increases after the two world wars. The heart disease and circulatory disease death rate was declining between 1891 and 1910 before cigarette smoking became prevalent. The early peak in cardiovascular mortality in 1891 coincided with an influenza pandemic and a peak in the death rate from bronchitis, pneumonia and influenza. There is also correspondence between short-term fluctuations in the death rates from these respiratory diseases and cardiovascular disease. This evidence of ecological association is consistent with the findings of many studies that seasonal influenza can trigger acute myocardial infarction and episodes of respiratory infection are followed by increased risk of cardiovascular events. Vaccination studies could provide more definitive evidence of the role in cardiovascular disease and mortality of influenza, other viruses, and common bacterial agents of respiratory infection.
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40

Sin, Don D., and S. F. Paul Man. "Chronic obstructive pulmonary disease: a novel risk factor for cardiovascular disease." Canadian Journal of Physiology and Pharmacology 83, no. 1 (January 1, 2005): 8–13. http://dx.doi.org/10.1139/y04-116.

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Chronic obstructive pulmonary disease (COPD) is one of the leading causes of morbidity and mortality in Canada and elsewhere. It affects 5% of all adult Canadians and is the fourth leading cause of death. Interestingly, the leading causes of hospitalizations and mortality among COPD patients are cardiovascular events. In the Lung Health Study, over 5 800 patients with mild to moderate COPD were studied. Forty-two to 48% of all hospitalizations that occurred over the study's 5-year follow-up period were related to cardiovascular complications. Various population-based studies suggest that independent of smoking, age, and gender, COPD increases the risk of cardiovascular morbidity and mortality twofold. Alarmingly, some bronchodilators, which are commonly used to treat symptoms in COPD, may increase the risk of cardiovascular morbidity and even mortality among COPD patients. In this paper, we discuss the epidemiologic evidence linking COPD and cardiovascular events as well as the potential mechanism(s) which may be responsible for this association.Key words: COPD, FEV1, cardiovascular events, C-reactive protein.
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41

Lunat, Raees. "Primary prevention of cardiovascular disease." InnovAiT: Education and inspiration for general practice 12, no. 3 (January 21, 2019): 117–22. http://dx.doi.org/10.1177/1755738018818602.

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Cardiovascular disease is one of the major causes of mortality and morbidity in the UK. Prevention of cardiovascular disease has been a prominent feature on the healthcare agenda in recent decades. Investment in both prevention strategies and primary care initiatives has aimed to lower the rates of morbidity and mortality. Cardiovascular disease incorporates stroke, coronary heart disease, and peripheral arterial disease. It is linked to other co-morbid conditions including dementia, heart failure, chronic kidney disease, and diabetes mellitus. These conditions share a pathology causing atheroma and thrombosis in arteries and reducing blood flow. This article aims to help improve understanding of cardiovascular disease and promote measures to reduce prevalence.
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42

Atem, Ngum Kikah, Sitara M. Weerakoon, Titilope Olanipekun, Temidayo Abe, and Folefac Atem. "2944 The Effects of Liver Disease on Cardiovascular Mortality Among Patients With Cardiovascular Disease." American Journal of Gastroenterology 114, no. 1 (October 2019): S1602. http://dx.doi.org/10.14309/01.ajg.0000601308.18790.95.

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43

Artero, Enrique G., Duck-chul Lee, Vanesa España-Romero, Jonathan A. Mitchell, Xuemei Sui, and Steven N. Blair. "Ideal Cardiovascular Health Assessments and All-cause and Cardiovascular Disease Mortality." Medicine & Science in Sports & Exercise 43, Suppl 1 (May 2011): 27. http://dx.doi.org/10.1249/01.mss.0000402752.80934.3d.

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44

Rosenberg, Harry M. "MISCLASSIFICATION OF RACE IN CARDIOVASCULAR DISEASE MORTALITY DATA." American Journal of Public Health 95, no. 10 (October 2005): 1673–74. http://dx.doi.org/10.2105/ajph.2005.072215.

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45

&NA;. "Methotrexate increases mortality in patients with cardiovascular disease." Reactions Weekly &NA;, no. 801 (May 2000): 5. http://dx.doi.org/10.2165/00128415-200008010-00010.

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46

Liu, Xiao, Nien Xiang Tou, Qi Gao, Xinyi Gwee, Shiou Liang Wee, and Tze Pin Ng. "Frailty and risk of cardiovascular disease and mortality." PLOS ONE 17, no. 9 (September 19, 2022): e0272527. http://dx.doi.org/10.1371/journal.pone.0272527.

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Background Prospective cohort studies suggest that frailty is associated with an increased risk of incident cardiovascular disease (CVD) morbidity and mortality, but their mechanistic and developmental relations are not fully understood. We investigated whether frailty predicted an increased risk of incident nonfatal and fatal CVD among community-dwelling older adults. Methods A population cohort of 5015 participants aged 55 years and above free of CVD at baseline was followed for up to 10 years. Pre-frailty and frailty were defined as the presence of 1–2 and 3–5 modified Fried criteria (unintentional weight loss, weakness, slow gait speed, exhaustion, and low physical activity), incident CVD events as newly diagnosed registered cases of myocardial infarction (MI), stroke, and CVD-related mortality (ICD 9: 390 to 459 or ICD-10: I00 to I99). Covariate measures included traditional cardio-metabolic and vascular risk factors, medication therapies, Geriatric Depression Scale (GDS), Mini-Mental State Exam (MMSE), and blood biomarkers (haemoglobin, albumin, white blood cell counts and creatinine). Results Pre-frailty and frailty were significantly associated with elevated HR = 1.26 (95%CI: 1.02–1.56) and HR = 1.54 (95%CI:1.00–2.35) of overall CVD, adjusted for cardio-metabolic and vascular risk factors and medication therapies, but not after adjustment for GDS depression and MMSE cognitive impairment. The HR of association between frailty status and both CVD mortality and overall mortality, however, remained significantly elevated after full adjustment for depression, cognitive and blood biomarkers. Conclusion Frailty was associated with increased risk of CVD morbidity and especially mortality, mediated in parts by traditional cardio-metabolic and vascular risk factors, and co-morbid depression and associated cognitive impairment and chronic inflammation. Given that pre-frailty and frailty are reversible by multi-domain lifestyle and health interventions, there is potential benefits in reducing cardiovascular diseases burden and mortality from interventions targeting pre-frailty and early frailty population.
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47

Cicero, Arrigo F. G., Sergio D’Addato, Francesca Santi, Alienor Ferroni, and Claudio Borghi. "Leisure-time physical activity and cardiovascular disease mortality." Journal of Cardiovascular Medicine 13, no. 9 (September 2012): 559–64. http://dx.doi.org/10.2459/jcm.0b013e3283516798.

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48

Eberhardt, Robert, and Jay Coffman. "Cardiovascular Morbidity and Mortality in Peripheral Arterial Disease." Current Drug Target -Cardiovascular & Hematological Disorders 4, no. 3 (September 1, 2004): 209–17. http://dx.doi.org/10.2174/1568006043336230.

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49

Boumendil, EF. "Mortality from non-cardiovascular disease and diabetes mellitus." Lancet 351, no. 9102 (February 1998): 598–99. http://dx.doi.org/10.1016/s0140-6736(05)78588-3.

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50

GrØnbÆk, M. "Type of Alcohol and Mortality from Cardiovascular Disease." Food and Chemical Toxicology 37, no. 9-10 (September 1999): 921–24. http://dx.doi.org/10.1016/s0278-6915(99)00077-0.

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