Log in

Relevant bibliographies by topics / Cardiomyocytes / Books

Books on the topic 'Cardiomyocytes'

To see the other types of publications on this topic, follow the link: Cardiomyocytes.

Author: Grafiati

Published: 4 June 2021

Last updated: 7 September 2024

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 50 books for your research on the topic 'Cardiomyocytes.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse books on a wide variety of disciplines and organise your bibliography correctly.

1

Skuse, Gary R., and Maureen C. Ferran, eds. Cardiomyocytes. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2572-8.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Michael, Piper Hans, and Isenberg Gerrit, eds. Isolated adult cardiomyocytes. Boca Raton, Fla: CRC Press, 1989.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

3

Skuse, Gary R., and Maureen C. Ferran. Cardiomyocytes: Methods and Protocols. New York: Humana Press, 2015.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

4

Yoshida, Yoshinori, ed. Pluripotent Stem-Cell Derived Cardiomyocytes. New York, NY: Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1484-6.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Kartha, Chandrasekharan C. Cardiomyocytes in Health and Disease. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-85536-9.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Schlüter, Klaus-Dieter, ed. Cardiomyocytes – Active Players in Cardiac Disease. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-31251-4.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Yee-Ki, Lee, and Siu Chung-Wah. Calcium Handling in hiPSC-Derived Cardiomyocytes. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-4093-2.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Luiz, Belardinelli, ed. Effects of extracellular adenosine and ATP on cardiomyocytes. Austin: Landes, 1999.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

9

Sowerby, Andrew John. Anoxia, plasma membrane structure and calcium homeostasis: Photobleaching and microflurescence investigations inisolated rat cardiomyocytes. Uxbridge: Brunel University, 1991.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

10

Lai, Laura R. B. Protein oxidation occurs in cardiomyocytes exposed to an in vitro model of hypoxia/reperfusion injury. Ottawa: National Library of Canada, 1996.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

11

Li, Fen. A novel primary cell clulture system for adult cardiomyocytes involving a 3-D extracellular matrix. Ottawa: National Library of Canada, 1996.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

12

Liu, Bohao. Leveraging the potential of human iPSC-derived cardiomyocytes: From modeling congenital heart disease to treating myocardial infarction. [New York, N.Y.?]: [publisher not identified], 2021.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

13

Kohler-Schütz, Michaela. Die Wirkung von Insulin und (-)-Phenylephrin auf pHi, (Na+)i, (Ca2+)i, (K+)i und Plasmamembranpotential von Cardiomyocyten adulter Ratten. [s.l.]: [s.n.], 1997.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

14

Piper, Hans Michael, and Gerrit Isenberg. Isolated Adult Cardiomyocytes. Edited by Hans Michael Piper and Gerrit Isenberg. CRC Press, 2019. http://dx.doi.org/10.1201/9780429290541.

Full text

APA, Harvard, Vancouver, ISO, and other styles

15

Piper, Hans Michael, and Gerrit Isenberg. Isolated Adult Cardiomyocytes. Taylor & Francis Group, 2021.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

16

Piper, Hans Michael, and Gerrit Isenberg. Isolated Adult Cardiomyocytes. Taylor & Francis Group, 2019.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

17

Skuse, Gary R., and Maureen C. Ferran. Cardiomyocytes: Methods and Protocols. Springer New York, 2016.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

18

Ikonomidis, John Sotirios. Preconditioning human ventricular cardiomyocytes. 1995.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

19

Yoshida, Yoshinori. Pluripotent Stem-Cell Derived Cardiomyocytes. Springer, 2022.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

20

Kartha, Chandrasekharan C. Cardiomyocytes in Health and Disease. Springer International Publishing AG, 2022.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

21

Rothen-Rutishauser, Barbara Maria. Dynamics of myofibrillogenesis in cardiomyocytes. 1996.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

22

Cardiomyocytes in Health and Disease. Springer International Publishing AG, 2022.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

23

Salvatori, Daniela, Harsha D. Devalla, and Robert Passier. Cells to repair the infarcted myocardium. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, José Luis de la Pompa, David Sedmera, Cristina Basso, and Deborah Henderson. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0030.

Full text

Abstract:

The adult mammalian heart has poor regenerative capacity. Loss of functional cardiomyocytes following myocardial infarction leads to the replacement of functional muscle by scar tissue. This has a detrimental effect on cardiac function and may lead to heart failure. Potential regeneration of severe cardiac damage would require replacement of dead and damaged cardiomyocytes by transplantation, recruitment of endogenous progenitor cells, or induction of cardiomyocyte proliferation. For more than a decade, clinical trials to ameliorate the injured heart have been under way. However, after evaluation of the outcome of these trials it is evident that the beneficial effects of these cell-based transplantations are only marginal, and beneficial effects, if any, are not caused by regeneration of cardiomyocytes. In recent years, alternative approaches and various cell sources have been studied and suggested for cardiac repair. Recent advances in these cell-based therapies or strategies to activate endogenous cardiac repair are discussed.

APA, Harvard, Vancouver, ISO, and other styles

24

Schlüter, Klaus-Dieter. Cardiomyocytes – Active Players in Cardiac Disease. Springer, 2016.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

25

Piper, Hans Michael, and Gerrit Isenberg. Isolated Adult Cardiomyocytes: Structure and Metabolism. Taylor & Francis Group, 2019.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

26

Piper, Hans Michael, and Gerrit Isenberg. Isolated Adult Cardiomyocytes: Structure and Metabolism. Taylor & Francis Group, 2019.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

27

Yee-Ki, Lee, and Siu Chung-Wah. Calcium Handling in HiPSC-Derived Cardiomyocytes. Springer, 2012.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

28

Piper, Hans Michael, and Gerrit Isenberg. Isolated Adult Cardiomyocytes: Structure and Metabolism. Taylor & Francis Group, 2019.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

29

Piper, Hans Michael, and Gerrit Isenberg. Isolated Adult Cardiomyocytes: Structure and Metabolism. Taylor & Francis Group, 2019.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

30

Schlüter, Klaus-Dieter. Cardiomyocytes – Active Players in Cardiac Disease. Springer, 2018.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

31

Piper, Hans Michael. Isolated Adult Cardiomyocytes: Structure and Metabolism. CRC Press, 1989.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

32

Yee-Ki, Lee, and Siu Chung-Wah. Calcium Handling in hiPSC-Derived Cardiomyocytes. Springer, 2012.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

33

Schlüter, Klaus-Dieter. Cardiomyocytes - Active Players in Cardiac Disease. Springer London, Limited, 2016.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

34

Piper, Hans Michael. Isolated Adult Cardiomyocytes: Electrophysiology and Contractile Function. CRC Press, 1989.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

35

Effects of Extracellular Adenosine and Atp on Cardiomyocytes. International Thomson Publishing Services, 1998.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

36

Calcium Handling in HipscDerived Cardiomyocytes Springerbriefs in Stem Cells. Springer, 2012.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

37

Mumford, David Arthur. Modelling cardiac function using strands of cultured neonatal rat cardiomyocytes. 1985.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

38

Bauwens, Celine L. A scalable bioprocess for generating embyronic stem cell derived cardiomyocytes. 2004.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

39

Musso, Gabriel A. Effects of PP2a inhibition during ischemia/reperfusion on apoptosis in cardiomyocytes. 2005.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

40

Musso, Gabriel A. Effects of PP2a inhibition during ischemia/reperfusion on apoptosis in cardiomyocytes. 2005.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

41

David, Peter Guido. On the role of the tumor suppressor protein p53 in cultured rat cardiomyocytes. 1998.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

42

Elbel, Jörg Andreas. Sol-gel derived titania as culture substrate for long term culturing of differentiated cardiomyocytes. 2001.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

43

Polontchouk, Lioudmila Olegovna. Cell-cell and cell-substratum interactions in the long-term culture of adult cardiomyocytes. 1999.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

44

Harder, Beatrice Andrea. Remodeling of cytoskeletal and contractile structures by growth factors and thyroid hormone in clutured cardiomyocytes. 1997.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

45

Miquerol, Lucile. Origin and development of the cardiac conduction system. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, José Luis de la Pompa, David Sedmera, Cristina Basso, and Deborah Henderson. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0015.

Full text

Abstract:

The cardiac conduction system represents the ‘wiring’ of the heart and orchestrates the propagation of the electrical activity to synchronize heartbeats. It is built from specialized cardiomyocytes expressing a subset of ion channels and gap junctions indispensable for their electrophysiological properties. Although representing only a very small volume of the heart, the conduction system plays a crucial role in the appearance of cardiac arrhythmias. The cells forming the conduction system are derived from the same cardiac progenitors as the working cardiomyocytes, and the choice between these two fates is acquired during embryonic development. The components of the conduction system are progressively established during cardiac morphogenesis and converge to form an integrated electrical system in the definitive heart. This chapter will discuss recent advances using mouse genetic approaches which have improved understanding of the cellular origin and the transcriptional regulatory networks involved in the development of the conduction system.

APA, Harvard, Vancouver, ISO, and other styles

46

Cohen, Gideon. Studies on the role of K+-ATP channels in the preconditioning of cultured human ventricular cardiomyocytes. 2001.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

47

Yilmaz, Ali, and Anca Florian. Myocarditis: imaging techniques. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0367.

Full text

Abstract:

The clinical presentation of myocarditis is multifaceted and electrocardiogram (ECG) changes as well as biomarkers tend to be non-specific. Therefore, the diagnosis of myocarditis can be challenging and should be based on an integrated approach including patient history, physical examination, non-invasive tests such as ECG and serum biomarkers, and non-invasive cardiac imaging. As myocarditis may lead to global ventricular dysfunction, regional wall motion abnormalities, and/or diastolic dysfunction, echocardiography should be routinely performed. However, hallmarks of acute myocarditis comprise structural changes such as cardiomyocyte swelling, an increase in extracellular space and water content, accumulation of inflammatory cells, potential necrosis or apoptosis of cardiomyocytes, and myocardial remodelling with fibrotic tissue replacement that can be depicted by cardiovascular magnetic resonance. Nuclear techniques are still not routinely recommended for the work-up of myocarditis—with the possible exception of suspected sarcoidosis—due to limited data, limited diagnostic specificity, limited availability, and risk from radiation exposure. This chapter focuses on those non-invasive cardiac imaging techniques that are used in daily clinical practice for work-up of suspected myocarditis. However, as research continues and novel imaging techniques become available, it is hoped that even more accurate and timely diagnosis of myocarditis will be possible in the near future.

APA, Harvard, Vancouver, ISO, and other styles

48

Elliott, Perry, Kristina H. Haugaa, Pio Caso, and Maja Cikes. Restrictive cardiomyopathy and arrhythmogenic right ventricular cardiomyopathy. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0044.

Full text

Abstract:

Restrictive cardiomyopathy is a heart muscle disorder characterized by increased myocardial stiffness that results in an abnormally steep rise in intraventricular pressure with small increases in volume in the presence of normal or decreased diastolic left ventricular volumes and normal ventricular wall thickness. The disease may be caused by mutations in a number of genes or myocardial infiltration. Arrhythmogenic right ventricular cardiomyopathy is an inherited cardiac muscle disease associated with sudden cardiac death, ventricular arrhythmias, and cardiac failure. It is most frequently caused by mutations in desmosomal protein genes that lead to fibrofatty replacement of cardiomyocytes, right ventricular dilatation, and aneurysm formation.

APA, Harvard, Vancouver, ISO, and other styles

49

Menasché, Philippe. Stem Cell Therapy Post-AMI. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199544769.003.0010.

Full text

Abstract:

• Experimental studies suggest that bone marrow-derived stem cells can improve function of infarcted myocardium• This benefit seems to involve paracrine signalling and limitation of left ventricular remodelling rather than true regeneration of cardiomyocytes from donor cells• These experimental findings have been translated in the clinical setting into significant, although moderate, improvements in cardiac function and LV remodelling but the extent to which these benefits impact on event-free long term survival remains to be determined• Optimisation of this therapeutic strategy will require a more comprehensive characterisation of cell functionality and an improvement in the methods used in cell transfer, engraftment, survival and integration.

APA, Harvard, Vancouver, ISO, and other styles

50

Meilhac, Sigolène M. Cardiac growth I: Cardiomyocyte proliferation. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, José Luis de la Pompa, David Sedmera, Cristina Basso, and Deborah Henderson. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0009.

Full text

Abstract:

Efficient contraction of the heart depends on the size and oriented architecture of the myocardium. This is severely compromised by myocardial infarction or in cardiomyopathies. Deciphering the mechanisms underlying heart growth has attracted much attention over the past decade, after the demonstration that the mammalian heart has some potential to regenerate, thus raising hopes that heart repair may become a reality. The mechanisms of cardiac growth during development have been well studied in the mouse model, taking advantage of sophisticated genetic engineering and new tools for tracking cell lineages and behaviour. We discuss the current view of the intrinsic regulation of cardiomyocyte behaviour, as well as how it is modulated by interplay with other cardiac cell types or with the environment. Such fundamental knowledge is important for understanding the origin of congenital heart defects and for the development of novel strategies of heart repair.

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!