Log in

Relevant bibliographies by topics / Cardiac myocytes / Books

To see the other types of publications on this topic, follow the link: Cardiac myocytes.

Books on the topic 'Cardiac myocytes'

Author: Grafiati

Published: 4 June 2021

Last updated: 7 September 2023

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 29 books for your research on the topic 'Cardiac myocytes.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse books on a wide variety of disciplines and organise your bibliography correctly.

1

sirna, Josephine Barbara. Iron regulation in neonatal rat cardiac myocytes. Ottawa: National Library of Canada, 1998.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

2

Wu, Bingruo. Expression of inhibin subunits in rat heart and cardiac myocytes. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1993.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

3

Rizek, Randy. Ionic selectivity and regulation of maitotoxin-activated nonselective cation channels in rat cardiac myocytes. Ottawa: National Library of Canada, 2003.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

4

A, Clark William, Decker Robert S, Borg Thomas K. 1943-, and National Heart, Lung, and Blood Institute., eds. Biology of isolated adult cardiac myocytes: Proceedings of the National Heart, Lung, and Blood Institute-sponsored workshop "Biology of isolated adult cardiac myocytes," held September 22-25, 1987, at Asilomar Conference Center, Pacific Grove, California, USA. New York: Elsevier, 1988.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

5

Hariharan, Venkatesh. The Effects of Arrhythmogenic Right Ventricular Cardiomyopathy-Causing Proteins on the Mechanical and Signaling Properties of Cardiac Myocytes. [New York, N.Y.?]: [publisher not identified], 2014.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

6

Larry and Horti Fairberg Cardiac Workshop (6th 2009 Haifa, Israel). Analysis of cardiac development: From embryo to old age. Edited by Beyar Rafael, Landesberg Amir, and New York Academy of Sciences. Boston: Blackwell Pub. on behalf of the New York Academy of Sciences, 2010.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

7

S, Sideman, Beyar Rafael, Landesberg Amir, and New York Academy of Sciences, eds. Interactive and integrative cardiology. Boston, Mass: Blackwell Pub. on behalf of the New York Academy of Sciences, 2006.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

8

1944-1988, Robinson T. F., and Kinne Rolf K. H, eds. Cardiac myocyte-connective tissue interactions in health and disease. Basel: Karger, 1990.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

9

pepper, C. B. Modulation of cardiac myocytr contraction by the vascular endothelium. Birmingham: University of Birmingham, 1996.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

10

Jenkins, Kim. The role of phosphoinositide hydrolysis and protein kinase C activation in cardiac myocyte hypertrophy. Birmingham: University of Birmingham, 1994.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

11

Kung, Shu hung. The role that bacterial DNA and RNA play in PKR and JNK signalling in cardiac myocyte and 2FTGH cells. Sudbury, Ont: Laurentian University, 2005.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

12

Isolated Adult Cardiac Myocytes: Structure, Function and Metabolism. Springer, 1985.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

13

Hong, Charles C., Ada S. Ao, and Jijun Hao. Chemical Biology in Regenerative Medicine: Bridging Stem Cells and Future Therapies. Wiley & Sons, Incorporated, John, 2014.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

14

An Essential Guide to Cardiac Cell Therapy. Informa Healthcare, 2006.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

15

Taillefer, Raymond, and Frans J. Th Wackers. Kinetics of Conventional and New Cardiac Radiotracers. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199392094.003.0004.

Full text

Abstract:

The kinetics of radiotracers, that is the mode of uptake, retention and release from the myocardium, are relevant for designing and implementing optimized nuclear cardiac imaging protocols. This chapter addresses the kinetics of commonly used radiotracers for imaging myocardial perfusion, sympathetic neuronal function and cardiac metabolism, both with SPECT and PET cardiac imaging. The optimal timing of imaging after injection either at stress or at rest is determined by rate of uptake in the heart and adjacent organs, as well as the residence time of radiotracers within the myocytes. The efficiency of myocardial extraction over a wide range myocardial blood flows is relevant for reliable detection of obstructive coronary artery disease and absolute quantification of regional myocardial blood flow. For each cardiac imaging agent the cellular mechanism of uptake and its release or retention are discussed with an emphasis on the clinical impact of these parameters.

APA, Harvard, Vancouver, ISO, and other styles

16

Pathogenesis of Heart Failure, An Issue of Heart Failure Clinics (The Clinics: Internal Medicine). Saunders, 2005.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

17

Hong, Charles C., Ada S. Ao, and Jijun Hao. Chemical Biology in Regenerative Medicine: Bridging Stem Cells and Future Therapies. Wiley, 2014.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

18

Hong, Charles C., Ada S. Ao, and Jijun Hao. Chemical Biology in Regenerative Medicine: Bridging Stem Cells and Future Therapies. Wiley & Sons, Limited, John, 2014.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

19

Hong, Charles C., Ada S. Ao, and Jijun Hao. Chemical Biology in Regenerative Medicine: Bridging Stem Cells and Future Therapies. Wiley & Sons, Incorporated, John, 2014.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

20

Hong, Charles C., Ada S. Ao, and Jijun Hao. Chemical Biology in Regenerative Medicine: Bridging Stem Cells and Future Therapies. Wiley & Sons, Incorporated, John, 2014.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

21

Mungrue, Imran N. Conditional over-expression of inducible nitric oxide synthase (iNOS) in cardiac and arterial myocytes of transgenic mice. 2003.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

22

Walsh, Richard A. Molecular Mechanisms of Cardiac Hypertrophy and Failure. Informa Healthcare, 2004.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

23

(Editor), Samuel Sideman, Rafael Beyar (Editor), and Amir Landesberg (Editor), eds. The Communicative Cardiac Cell (Annals of the New York Academy of Sciences). New York Academy of Sciences, 2005.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

24

Larry and Horti Fairberg Cardiac Workshop. Cardiac Engineering: From Genes and Cells to Structure and Function (Annals of the New York Academy of Sciences). New York Academy of Sciences, 2004.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

25

Cardiac Engineering: From Genes And Cells To Structure And Function (Annals of the New York Academy of Sciences, V. 1015). New York Academy of Sciences, 2004.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

26

Calcium Handling in HipscDerived Cardiomyocytes Springerbriefs in Stem Cells. Springer, 2012.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

27

Sánchez-Quintana, Damián, and José Angel Cabrera. Normal atrial and ventricular myocardial structures. Edited by Yen Ho. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0014.

Full text

Abstract:

The heart functions by means of a three-dimensional arrangement of myofibres supported by an extracellular matrix which plays an important role in maintaining the size and shape of the heart. In both atria, the structure of the walls and the atrial septum confers a three-dimensional arrangement of muscle bundles and myoarchitecture that allows preferential electrical intra- and interatrial conduction which is important for a better understanding of atrial activation and arrhythmias. The myoarchitecture within the ventricular walls has a three-dimensional arrangement of myofibres, within a supporting matrix of fibrous tissue, which changes orientation from being oblique in the subepicardium to circumferential in the middle and to longitudinal in the subendocardium, allowing the chambers to change in shape and size through the cardiac cycle. Within each ventricle, the circumferential portion is the thickest transmurally, with the longitudinal portion the thinnest. The three-dimensional arrangement of the ventricular mesh serves to realign the myocytes during ventricular contraction, accounting for the extent of systolic mural thickening. Abnormal myoarchitecture in combination with alterations in the connective tissue matrix provide the structural basis for abnormalities in myocardial function.

APA, Harvard, Vancouver, ISO, and other styles

28

Thygesen, Kristian, Joseph S. Alpert, Allan S. Jaffe, and Harvey D. White. The universal definition of myocardial infarction. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0041.

Full text

Abstract:

Myocardial infarction is defined pathologically as myocyte necrosis due to prolonged ischaemia. These conditions are met when there is a detection of a rise and/or fall of cardiac biomarkers, preferably troponins, with at least one value above the 99th percentile of the upper reference limit, together with evidence of myocardial ischaemia, as recognized by at least one of the following: symptoms of ischaemia, electrocardiographic changes of new ischaemia, the development of pathological Q waves, imaging evidence of a new loss of viable myocardium or new regional wall motion abnormality, or the identification of an intracoronary thrombus by angiography or autopsy.

APA, Harvard, Vancouver, ISO, and other styles

29

Alexander, Kevin. Myocarditis and Pericarditis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0019.

Full text

Abstract:

Infectious myocarditis is a primary, inflammatory cardiomyopathy that can lead to cardiomyocyte toxicity via direct myocyte invasion, toxin production, and/or stimulation of a chronic inflammatory response through antigenic mimicry. Its incidence is difficult to determine due to significant disease heterogeneity and the lack of a noninvasive gold standard for diagnosis. Often, the causative pathogen is not identified; in cases where it is, appropriate anti-infective agents may be used. Treatment is primarily supportive. Acute infectious pericarditis involves inflammation of the parietal and visceral layers of the pericardial sac that surround the heart. Because infectious pericarditis usually has a viral etiology, antibiotics are only started if blood or pericardial effusion cultures demonstrate a bacterial or fungal cause. Purulent pericarditis and cardiac tamponade should be treated with drainage via either pericardiocentesis or a pericardiotomy. Pericardial resection is the only treatment for constrictive pericarditis.

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!