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Journal articles on the topic 'CARDIAC CONDITIONS'

Author: Grafiati
Published: 11 September 2023

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1

Dutton, Emily. "Tackling cardiac conditions." BSAVA Companion 2017, no. 5 (May 1, 2017): 4–7. http://dx.doi.org/10.22233/20412495.0517.4.

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2

Hia, Cindy Ping Ping, and William Chin Ling Yip. "Childhood Cardiac Conditions." Singapore Family Physician 46, no. 5 (July 1, 2020): 37–42. http://dx.doi.org/10.33591/sfp.46.5.u6.

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3

Hudzik, B., and L. Polonski. "Neglected conditions: Cardiac tumours." Canadian Medical Association Journal 186, no. 6 (March 31, 2014): 452–53. http://dx.doi.org/10.1503/cmaj.114-0024.

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4

Peck, Michel D., and Amy L. Ai. "Chapter 2; Cardiac Conditions." Journal of Gerontological Social Work 50, sup1 (May 13, 2008): 11–44. http://dx.doi.org/10.1080/01634370802137777.

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5

Li, Yuehua, Chenghui Zhou, Xianliang Zhou, Lei Song, and Rutai Hui. "PAPP-A in cardiac and non-cardiac conditions." Clinica Chimica Acta 417 (February 2013): 67–72. http://dx.doi.org/10.1016/j.cca.2012.12.006.

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6

Mitkowski, Przemysław. "Electrotherapy in acute cardiac conditions." In a good rythm 1, no. 50 (April 15, 2019): 4–7. http://dx.doi.org/10.5604/01.3001.0013.1682.

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Cardiac arrhythmia is one of the main reasons of admission to emergency units. Arrhythmia can be alone symptom, effect of compromise of stable disease or be caused by acute cardiac event. Temporary pacing according to current guidelines should be limited to advanced atrio-venricular block, life-threatening bradycardia related to cardiac procedures (ie. PCI). This method should not be used routinely but only when chronotro­pic drugs are ineffective. Likelihood of restoration of proper atrio-ventricular conduction, when block persists over 2 days is exiguous so decision of permanent system implantation should be taken earlier. Temporary pacing with stiff lead is related to increased risk of perforation and correlates with an increased risk of permanent device infection. Overdrive pacing should be considered in case of refractory to treatment recurrent ventricular arrhythmias. Immediate implantation of cardioverter-defibrillator should be performed in secondary prevention, when arrhythmia seems not to be related to potentially reversible cause. Frequently recurrent or persistent arrhythmias should be treated pharmacologically or with ablation before system implantation. In randomized trials it was not proved that implantation of ICD based on early risk stratification after myocardial infarction give any benefits. Experts point out the group of patients who could benefit from early (within 40 days after MI) implantation of an ICD. Cardiac resynchronization system implantation may be considered as a thera­py option to decrease congestion. Such treatment may allow the withdrawal of inotropic agents. Treatment of acute cardiac conditions with electrotherapy methods could be an effective therapy worth in selected group of patients.
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Statuta, Siobhan M., Erin S. Barnes, and John M. MacKnight. "Non-Cardiac Conditions that Mimic Cardiac Symptoms in Athletes." Clinics in Sports Medicine 41, no. 3 (July 2022): 389–404. http://dx.doi.org/10.1016/j.csm.2022.02.003.

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8

Sarmast, Syed Abdul, and Jeffrey M. Schussler. "Monozygotic Twins with Identical Cardiac Conditions." Baylor University Medical Center Proceedings 24, no. 2 (April 2011): 104–6. http://dx.doi.org/10.1080/08998280.2011.11928694.

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9

Roos-Hesselink, J. W., J. J. Duvekot, and S. A. Thorne. "Pregnancy in high risk cardiac conditions." Heart 95, no. 8 (August 26, 2008): 680–86. http://dx.doi.org/10.1136/hrt.2008.148932.

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10

Jain, Deepali, Joseph J. Maleszewski, and Marc K. Halushka. "Benign cardiac tumors and tumorlike conditions." Annals of Diagnostic Pathology 14, no. 3 (June 2010): 215–30. http://dx.doi.org/10.1016/j.anndiagpath.2009.12.010.

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11

Gloag, D. "Rehabilitation of patients with cardiac conditions." BMJ 290, no. 6468 (February 23, 1985): 617–20. http://dx.doi.org/10.1136/bmj.290.6468.617.

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12

Bethell, H. "Rehabilitation of patients with cardiac conditions." BMJ 290, no. 6480 (May 18, 1985): 1513–14. http://dx.doi.org/10.1136/bmj.290.6480.1513-c.

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13

Chope, Katherine B. "Cardiac/Cardiovascular Conditions Affecting Sport Horses." Veterinary Clinics of North America: Equine Practice 34, no. 2 (August 2018): 409–25. http://dx.doi.org/10.1016/j.cveq.2018.04.001.

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14

Moulson, Nathaniel, Saul Isserow, and James McKinney. "Lifestyle Considerations in Genetic Cardiac Conditions Associated With Sudden Cardiac Death." Canadian Journal of Cardiology 38, no. 4 (April 2022): 544–48. http://dx.doi.org/10.1016/j.cjca.2021.12.014.

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15

Francis, Gary S., and W. H. Wilson Tang. "Cardiac troponins in renal insufficiency and other non-ischemic cardiac conditions." Progress in Cardiovascular Diseases 47, no. 3 (November 2004): 196–206. http://dx.doi.org/10.1016/j.pcad.2004.07.005.

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16

Gualandro, Danielle M., Christian Puelacher, and Christian Mueller. "High-sensitivity cardiac troponin in acute conditions." Current Opinion in Critical Care 20, no. 5 (October 2014): 472–77. http://dx.doi.org/10.1097/mcc.0000000000000132.

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17

Bellchambers, Julie, Emma Neves, and Alison Pottle. "Inherited cardiac conditions: examining two patient cases." British Journal of Cardiac Nursing 12, no. 8 (August 2, 2017): 387–96. http://dx.doi.org/10.12968/bjca.2017.12.8.387.

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18

Agrafioti, Foteini, and Dimitrios Hatzinakos. "ECG biometric analysis in cardiac irregularity conditions." Signal, Image and Video Processing 3, no. 4 (September 2, 2008): 329–43. http://dx.doi.org/10.1007/s11760-008-0073-4.

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19

Thow, Morag K., Gillian Armstrong, and Danny Rafferty. "Non-cardiac Conditions and Physiotherapy in Phase III Cardiac Rehabilitation Exercise Programmes." Physiotherapy 89, no. 4 (April 2003): 233–37. http://dx.doi.org/10.1016/s0031-9406(05)60154-x.

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20

Ingles, Jodie, and James S. Ware. "What Is the Risk of Sudden Cardiac Arrest in Inherited Cardiac Conditions?" Journal of the American College of Cardiology 75, no. 21 (June 2020): 2708–10. http://dx.doi.org/10.1016/j.jacc.2020.04.010.

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21

S, Karthik, M. B Sridevi, and Evangeline Cynthia D. "Dental management of children with congenital cardiac conditions." International Dental Journal of Student Research 8, no. 2 (August 15, 2020): 94–95. http://dx.doi.org/10.18231/j.idjsr.2020.019.

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22

Villacorta-Lyew, Rachel, Brooks Laselle, Joseph P. Mazzoncini, Emily Merchant, and Peter J. Buckley. "Cardiac Pathological Conditions in Young Soldiers: Case Series." Military Medicine 173, no. 11 (November 2008): 1122–31. http://dx.doi.org/10.7205/milmed.173.11.1122.

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23

Owlia, Mohammad Bagher, Seyed Mohammad Yousof Mostafavi Pour Manshadi, and Nafiseh Naderi. "Cardiac Manifestations of Rheumatological Conditions: A Narrative Review." ISRN Rheumatology 2012 (October 17, 2012): 1–10. http://dx.doi.org/10.5402/2012/463620.

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Cardiovascular diseases are common in systemic rheumatologic diseases. They can be presented at the time of diagnosis or after diagnosis. The cardiac involvements can be the first presentation of rheumatologic conditions. It means that a patient with rheumatologic disease may go to a cardiologist when attacked by this disease at first. These manifestations are very different and involve different structures of the heart, and they can cause mortality and morbidity of patients with rheumatologic diseases. Cardiac involvements in these patients vary from subclinical to severe manifestations. They may need aggressive immunosuppressive therapy. The diagnosis of these conditions is very important for choosing the best treatment. Premature atherosclerosis and ischemic heart disease are increased in rheumatoid arthritis and systemic lupus erythematosus, and may be causes of mortality among them. The aggressive control of systemic inflammation in these diseases can reduce the risk of cardiovascular disease especially ischemic heart disease. Although aggressive treatment of primary rheumatologic diseases can decrease mortality rate and improve them, at this time, there are no specific guidelines and recommendations, to include aggressive control and prevention of traditional risk factors, for them.
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24

Abramov, G. V., D. G. Abramov, and I. P. Polovinkin. "Oscillogram processing algorithm when monitoring cardiac patients’ conditions." Journal of Physics: Conference Series 1902, no. 1 (May 1, 2021): 012094. http://dx.doi.org/10.1088/1742-6596/1902/1/012094.

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25

Chenzbraun, Adrian. "Non-ischaemic cardiac conditions: role of stress echocardiography." Echo Research and Practice 1, no. 1 (August 2014): R1—R7. http://dx.doi.org/10.1530/erp-14-0030.

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Stress echocardiography (SE) has a unique ability for simultaneous assessment of both functional class and exercise-related haemodynamic changes and as such is increasingly recognised for the evaluation of non-coronary artery disease pathologies. Some indications such as valvular heart disease or hypertrophic cardiomyopathy have been well established already, while others such as diastolic exercise testing are emerging of late. This paper addresses the main and best established indications for SE in non-ischaemic conditions, providing a practical perspective correlated with updated guidelines.
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26

Whight, Suzanne. "Nursing common cardiac conditions in cats and dogs." Veterinary Nursing Journal 24, no. 12 (December 2009): 17–19. http://dx.doi.org/10.1080/17415349.2009.11013147.

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27

Tavora, Fabio, and Allen Burke. "Pathology of cardiac tumours and tumour-like conditions." Diagnostic Histopathology 16, no. 1 (January 2010): 1–9. http://dx.doi.org/10.1016/j.mpdhp.2009.10.002.

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28

Chan, Titus, Jane Di Gennaro, Stephanie Burns Wechsler, and Susan L. Bratton. "Complex Chronic Conditions Among Children Undergoing Cardiac Surgery." Pediatric Cardiology 37, no. 6 (March 31, 2016): 1046–56. http://dx.doi.org/10.1007/s00246-016-1387-6.

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29

Lee, Jan Hau, and Janil Puthucheary. "Transport of Critically III Neonates with Cardiac Conditions." Air Medical Journal 29, no. 6 (November 2010): 320–22. http://dx.doi.org/10.1016/j.amj.2010.05.001.

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30

SMITS, J. "Pharmacological modulation of cardiac remodeling in pathophysiological conditions." Journal of Molecular and Cellular Cardiology 23 (July 1991): S44. http://dx.doi.org/10.1016/0022-2828(91)90649-7.

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31

Cheek, Dennis J., Johnathon S. Hawkins, and Melissa McIntire Sherrod. "Unlocking the secrets of 2 common cardiac conditions." Nursing 39, no. 1 (January 2009): 52–56. http://dx.doi.org/10.1097/01.nurse.0000343458.88870.a9.

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32

Baruteau, Alban-Elouen, David J. Tester, Jamie D. Kapplinger, Michael J. Ackerman, and Elijah R. Behr. "Sudden infant death syndrome and inherited cardiac conditions." Nature Reviews Cardiology 14, no. 12 (September 7, 2017): 715–26. http://dx.doi.org/10.1038/nrcardio.2017.129.

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33

Bennett, Richard, Timothy Campbell, and Saurabh Kumar. "Catheter and Device Management of Inherited Cardiac Conditions." Heart, Lung and Circulation 29, no. 4 (April 2020): 594–606. http://dx.doi.org/10.1016/j.hlc.2019.12.009.

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34

Djukanovic, Nina, Vladimir Jakovljevic, and Vujadin Mujovic. "Evaluation of myocardial relaxation in conditions of cardiac remodeling." Medical review 62, no. 11-12 (2009): 555–68. http://dx.doi.org/10.2298/mpns0912555d.

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The term cardiodynamics refers to dynamic events associated with cardiac contraction and relaxation. The occurring wave of excitement spreads very quickly along the entire atrial musculature and after a brief AV retention it affects all muscle cells of the ventricles. Excitation, that is, the increase in action potentials, precedes the contraction of the myocardium, which follows the 'all or none' rule. Each contraction results in relaxation of the myocardium, so that the contraction and relaxation cycles continually follow each other in succession. The entire cardiodynamics, hemodinamics, i.e. signaling mechanisms of the heart are altered in the remodeling (alternation) condition of the left ventricular myocardium, i.e. the musculature and the whole arterial wall. Remodeling of the cardiac wall and layers of the arterial wall is a negative factor, because it leads to disturbances of the cardiac contraction and relaxation cycles and incites progression of the arterial hypertension, emergence of atherosclerosis and arterial stenosis. Today, the genetic base of the cardiac remodeling is the object of intensive studies. Cardiomyopathies are primary disorders of the myocardium associated with abnormalities of the cardiac wall thickness, the size of chambers, contractions, relaxations, signal conduct and rhythm. They are the major cause of morbidity and mortality for all age groups. Mechanisms of these events on the molecular level will be discussed in the following study.
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35

Jianhui, Li, Nathalie Rosenblatt-Velin, Noureddine Loukili, Pal Pacher, François Feihl, Bernard Waeber, and Lucas Liaudet. "Endotoxin impairs cardiac hemodynamics by affecting loading conditions but not by reducing cardiac inotropism." American Journal of Physiology-Heart and Circulatory Physiology 299, no. 2 (August 2010): H492—H501. http://dx.doi.org/10.1152/ajpheart.01135.2009.

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Acute myocardial dysfunction is a typical manifestation of septic shock. Experimentally, the administration of endotoxin [lipopolysacharride (LPS)] to laboratory animals is frequently used to study such dysfunction. However, a majority of studies used load-dependent indexes of cardiac function [including ejection fraction (EF) and maximal systolic pressure increment (dP/d tmax)], which do not directly explore cardiac inotropism. Therefore, we evaluated the direct effects of LPS on myocardial contractility, using left ventricular (LV) pressure-volume catheters in mice. Male BALB/c mice received an intraperitoneal injection of E. coli LPS (1, 5, 10, or 20 mg/kg). After 2, 6, or 20 h, cardiac function was analyzed in anesthetized, mechanically ventilated mice. All doses of LPS induced a significant drop in LV stroke volume and a trend toward reduced cardiac output after 6 h. Concomitantly, there was a significant decrease of LV preload (LV end-diastolic volume), with no apparent change in LV afterload (evaluated by effective arterial elastance and systemic vascular resistance). Load-dependent indexes of LV function were markedly reduced at 6 h, including EF, stroke work, and dP/d tmax. In contrast, there was no reduction of load-independent indexes of LV contractility, including end-systolic elastance (ejection phase measure of contractility) and the ratio dP/d tmax/end-diastolic volume (isovolumic phase measure of contractility), the latter showing instead a significant increase after 6 h. All changes were transient, returning to baseline values after 20 h. Therefore, the alterations of cardiac function induced by LPS are entirely due to altered loading conditions, but not to reduced contractility, which may instead be slightly increased.
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36

Rucinski, Cynthia, Annika Winbo, Luciana Marcondes, Nikki Earle, Martin Stiles, Rachael Stiles, Darren Hooks, et al. "A Population-Based Registry of Patients With Inherited Cardiac Conditions and Resuscitated Cardiac Arrest." Journal of the American College of Cardiology 75, no. 21 (June 2020): 2698–707. http://dx.doi.org/10.1016/j.jacc.2020.04.004.

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37

Alor, Sahoo, Strong Laura, Jensen Alexander, Flaherty Daniel, Anand Mukul, Alromheen Hassan, and Singh, Sarabjeet. "Elevated Lipoprotein (a) in Cardiac Outcomes: A Review." Clinical Cardiology and Cardiovascular Interventions 3, no. 8 (October 7, 2020): 01–06. http://dx.doi.org/10.31579/2641-0419/083.

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Context Elevated lipoprotein (a) [Lp(a)] levels is an often-ignored risk factor for major adverse cardiac events (MACEs) in humans. Even after accounting for established risk factors (discussed in the text), some residual risk can still be independently attributed to elevated Lp(a) levels. Current guidelines dictating normal and elevated Lp(a) levels and subsequent treatment have proven haphazard due to unstandardized studies. Many studies offer cutoff values in units mg/dL, which do not account for the heterogeneity of Lp(a). Interpretation of elevated Lp(a) necessitates consideration of ethnicity necessary for proper predictions. Numerous studies detail the effects of elevated Lp(a) in relation to myocardial infarction, aortic valve stenosis, and atherosclerosis, among other conditions. This article aims to clarify the numerous cutoffs and guidelines presented. Methods Searches were primarily conducted through Google, PubMed.gov, and cochrane.org. Results Elevated Lp(a) seems to correlate with the incidence of MACEs and should be considered when assessing risk. Specific cutoff values remain quite unclear. Conclusions We urge for further detailed investigation on the effects of elevated Lp(a) on cardiac outcomes with the use of isoform independent assays. Particular attention should be given to ethnicity when assigning risk cutoffs for cardiac conditions. Proprotein convertase subtilisin/kexin type 9 (PCSK-9) inhibitors can lower Lp(a) levels significantly and should be investigated.
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38

Farshadmand, Jonathan, Ofek Hai, Roman Zeltser, and Amgad N. Makaryus. "Utility of Cardiac Power Hemodynamic Measurements in the Evaluation and Risk Stratification of Cardiovascular Conditions." Healthcare 10, no. 12 (November 30, 2022): 2417. http://dx.doi.org/10.3390/healthcare10122417.

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Despite numerous advancements in prevention, diagnosis and treatment, cardiovascular disease has remained the leading cause of mortality globally for the past 20 years. Part of the explanation for this trend is persistent difficulty in determining the severity of cardiac conditions in order to allow for the deployment of prompt therapies. This review seeks to determine the prognostic importance of cardiac power (CP) measurements, including cardiac power output (CPO) and cardiac power index (CPI), in various cardiac pathologies. CP was evaluated across respective disease-state categories which include cardiogenic shock (CS), septic shock, transcatheter aortic valve replacement (TAVR), heart failure (HF), post-myocardial infarction (MI), critical cardiac illness (CCI) and an “other” category. Literature review was undertaken of articles discussing CP in various conditions and this review found utility and prognostic significance in the evaluation of TAVR patients with a significant correlation between one-year mortality and CPI; in HF patients showing CPI and CPO as valuable tools to assess cardiac function in the acute setting; and, additionally, CPO was found to be an essential tool in patients with CCI, as the literature showed that CPO was statistically correlated with mortality. Cardiac power and the derived measures obtained from this relatively easily obtained variable can allow for essential estimations of prognostic outcomes in cardiac patients.
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39

Goldenberg, Paula C., Betsy J. Adler, Ashley Parrott, Julia Anixt, Karen Mason, Jannel Phillips, David S. Cooper, Stephanie M. Ware, and Bradley S. Marino. "High burden of genetic conditions diagnosed in a cardiac neurodevelopmental clinic." Cardiology in the Young 27, no. 3 (September 19, 2016): 459–66. http://dx.doi.org/10.1017/s104795111600072x.

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AbstractBackgroundThere is a known high prevalence of genetic and clinical syndrome diagnoses in the paediatric cardiac population. These disorders often have multisystem effects, which may have an important impact on neurodevelopmental outcomes. Taken together, these facts suggest that patients and families may benefit from consultation by genetic specialists in a cardiac neurodevelopmental clinic.ObjectiveThis study assessed the burden of genetic disorders and utility of genetics evaluation in a cardiac neurodevelopmental clinic.MethodsA retrospective chart review was conducted of patients evaluated in a cardiac neurodevelopmental clinic from 6 December, 2011 to 16 April, 2013. All patients were seen by a cardiovascular geneticist with genetic counselling support.ResultsA total of 214 patients were included in this study; 64 of these patients had a pre-existing genetic or syndromic diagnosis. Following genetics evaluation, an additional 19 were given a new clinical or laboratory-confirmed genetic diagnosis including environmental such as teratogenic exposures, malformation associations, chromosomal disorders, and single-gene disorders. Genetic testing was recommended for 112 patients; radiological imaging to screen for congenital anomalies for 17 patients; subspecialist medical referrals for 73 patients; and non-genetic clinical laboratory testing for 14 patients. Syndrome-specific guidelines were available and followed for 25 patients with known diagnosis. American Academy of Pediatrics Red Book asplenia guideline recommendations were given for five heterotaxy patients, and family-based cardiac screening was recommended for 23 families affected by left ventricular outflow tract obstruction.ConclusionGenetics involvement in a cardiac neurodevelopmental clinic is helpful in identifying new unifying diagnoses and providing syndrome-specific care, which may impact the patient’s overall health status and neurodevelopmental outcome.
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40

Öztürk, Selçuk, and Y. Murat Elçin. "Cardiac Stem Cell Characteristics in Physiological and Pathological Conditions." Current Pharmaceutical Design 24, no. 26 (November 14, 2018): 3101–12. http://dx.doi.org/10.2174/1381612824666180903123817.

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For nearly a century, the adult heart was considered as a post-mitotic organ. The discovery of a resident cardiac stem cell (CSC) population in the heart has dramatically undermined this notion with the support of encouraging preclinical and clinical studies aiming to regenerate the damaged heart after a myocardial infarction (MI). There are two ways to obtain CSCs for transplantation: Allogeneic and autologous sources. Autologous cells may be obtained from the patients’ own tissue. Obtaining cells from diseased patients may contain a risk for altered stem cell characteristics. In addition to MI, these patients may also suffer from pathological conditions such as hypertension, diabetes mellitus, heart failure, congenital heart disease or cancer, which are known to alter CSC characteristics. It is also known that physiological conditions such as aging and death affect CSC functions in the heart. Our knowledge about the CSC characteristics in various physiological and pathological conditions may shed light on our opinion about the regenerative capacity and biological activity of these cells in these situations. Defining these properties may guide the researchers and clinicians in choosing and obtaining the most qualified CSC populations for cardiac regenerative medicine therapies. The purpose of this review is to describe the alterations in CSC characteristics in various physiological and pathological conditions.
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41

ISHIDE, Nobumasa. "Intracellular Calcium Modulators for Cardiac Muscle in Pathological Conditions." Japanese Heart Journal 37, no. 1 (1996): 1–17. http://dx.doi.org/10.1536/ihj.37.1.

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42

Carey, Mary G., Salah S. Al-Zaiti, Teri M. Kozik, and Michele M. Pelter. "Preoperative Screening 12-Lead Electrocardiogram Reveals Correctable Cardiac Conditions." American Journal of Critical Care 29, no. 6 (November 1, 2020): 493–94. http://dx.doi.org/10.4037/ajcc2020827.

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43

Lawless, Christine E. "Return-to-Play Decisions in Athletes with Cardiac Conditions." Physician and Sportsmedicine 37, no. 1 (April 2009): 80–91. http://dx.doi.org/10.3810/psm.2009.04.1686.

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44

Bennett, Christina. "Thyroid Cancer Survivors at Risk for Cardiac Conditions, Osteoporosis." Oncology Times 39, no. 5 (March 2017): 33. http://dx.doi.org/10.1097/01.cot.0000514196.19834.7f.

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45

Fox, Kim. "Future perspectives of If inhibition in various cardiac conditions." European Heart Journal Supplements 7, suppl_H (September 1, 2005): H33—H36. http://dx.doi.org/10.1093/eurheartj/sui051.

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46

Zhang, Jun, David A. Liem, Michael Mueller, Yueju Wang, Chenggong Zong, Ning Deng, Thomas M. Vondriska, et al. "Altered Proteome Biology of Cardiac Mitochondria Under Stress Conditions." Journal of Proteome Research 7, no. 6 (June 2008): 2204–14. http://dx.doi.org/10.1021/pr070371f.

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47

Maglaveras, N., A. V. Sahakian, and G. A. Myers. "Boundary conditions in simulations of cardiac propagating action potentials." IEEE Transactions on Biomedical Engineering 35, no. 9 (1988): 755–58. http://dx.doi.org/10.1109/10.7277.

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48

Cesarovic, Nikola, Miriam Lipiski, Volkmar Falk, and Maximilian Y. Emmert. "Cardiac electrophysiology: purpose tailored animal models for complex conditions." European Heart Journal 41, no. 21 (May 15, 2020): 2037. http://dx.doi.org/10.1093/eurheartj/ehaa328.

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Kimura, Takehiro, Seiji Takatsuki, Shunichiro Miyoshi, Mei Takahashi, Emiyu Ogawa, Yoshinori Katsumata, Takahiko Nishiyama, et al. "Optimal conditions for cardiac catheter ablation using photodynamic therapy." Europace 17, no. 8 (January 6, 2015): 1309–15. http://dx.doi.org/10.1093/europace/euu335.

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Ribeiro, Helder, Renato Margato, Sofia Carvalho, Catarina Ferreira, Ana Rebelo, Alberto Ferreira, Paulo Subtil, Pedro Mateus, and J. Ilídio Moreira. "P0834 INFECTIVE ENDOCARDITIS IN PATIENTS WITHOUT PREDISPOSING CARDIAC CONDITIONS." European Journal of Internal Medicine 20 (May 2009): S271. http://dx.doi.org/10.1016/s0953-6205(09)60853-0.

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