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Books on the topic 'Capsaicin'

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Published: 4 June 2021

Last updated: 11 March 2023

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1

Abdel-Salam, Omar M. E., ed. Capsaicin as a Therapeutic Molecule. Basel: Springer Basel, 2014. http://dx.doi.org/10.1007/978-3-0348-0828-6.

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2

N, Wood John, ed. Capsaicin in the study of pain. London: Academic Press, 1993.

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3

Srivastava, Sanjay K., ed. Role of Capsaicin in Oxidative Stress and Cancer. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-6317-3.

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4

T, Stock Melissa, and Hunter Kellye, eds. The healing powers of peppers: Chile pepper recipes and folk remedies for better health and living. New York: Three Rivers Press, 1998.

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5

Manteghian, M. Extraction of chemicals from plants: capsaicin ( chilli flavour ) from capsicum. Manchester: UMIST, 1985.

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6

Law, D. Studies related to the production of capsaicin by capsicum frutescens cells. Manchester: UMIST, 1993.

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7

Salazar, Mario Alfonso, and Jose Miguel Ortega. Peppers: Nutrition, consumption, and health. Hauppauge, N.Y: Nova Science Publishers, 2011.

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8

Christensen, Richard G. Preliminary investigation of Oleoresin Capsicum. Washington, D.C: U.S. Dept. of Justice, Office of Justice Programs, National Institute of Justice, 1995.

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9

DeWitt, Dave. El poder curativo de los pimientos: Con recetas de chiles y remedios caseros para mejorar la vida y la salud. Nueva York: Random House Español, 2001.

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10

Kwan, Chun Leung. Neuroplasticity of rat trigeminal subnucleus oralis neurons after tooth pulp deafferentation and after depletion of somatosensory c-fiber affrents with neonatal capsaicin treatment. Ottawa: National Library of Canada, 1994.

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11

Kwan, Chun Leung. Neuroplasticity of rat trigeminal subnucleus oralis neurons after tooth pulp deafferentation and after depletion of somatosensory C-fiber : afferent with neonatal capsaicin treatment. [Toronto: Faculty of Dentistry, University of Toronto], 1994.

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12

Golubkina, Nadezhda, Elena Kekina, Anna Molchanova, and Sergey Nadezhkin. Antioxidants of plants and methods of their definition. ru: INFRA-M Academic Publishing LLC., 2020. http://dx.doi.org/10.12737/1045420.

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The monograph presents the most simple and widely used methods for determining the most important of plant antioxidants: vitamin C, polyphenols, carotenoids, capsaicin, and belinovich photosynthetic pigments, flavonoids, anthocyanins, alkaloids, tannins, and minerals antioxidant: selenium and iodine. Special attention is paid to methods of extraction of antioxidants, providing maximum extraction of antioxidants from plant material, and the correct selection of the most appropriate method of analysis of one or another component. Provides detailed information developed by the authors method of using thin layer chromatography to assess the carotenoid composition of tomatoes and peppers. The data presented here include results of research conducted on the basis of FICO, as well as the latest developments of foreign scientists devoted to natural antioxidants and methods of their determination. Presented in this monograph methodology was successfully tested in the laboratory and analytical Department of PNCO in 2012-2018. For students and teachers and all interested in horticulture and agriculture.

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13

Nagy, Istvan. The capsaicin receptor. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0027.

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The landmark paper discussed in this chapter is ‘The capsaicin receptor: A heat activated ion channel in the pain pathway’, published by Caterina et al. in 1997. The identification of the molecular basis for the sensitivity of a major proportion of nociceptive primary sensory neurons for capsaicin, the pungent agent in chilli pepper, was undoubtedly one of the most significant pain-related discoveries in the twentieth century, for at least three reasons. First, the mechanism for capsaicin-induced responses could unequivocally be explained. Second, the discovery heralded the starting point for the development of a highly promising, mechanism-based means of analgesia. Third, the discovery also sparked studies which resulted in the discovery of the major cation channel family, the transient receptor potential (TRP) ion channel family, several members of which have also become putative targets for the development of analgesics.

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14

Muhammed, Majeed, ed. Capsaicin: The anti-arthritic phytochemical. Piscataway, NJ: Nutriscience Publishers, 1997.

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15

Abdel-Salam, Omar M. E. Capsaicin As a Therapeutic Molecule. Springer London, Limited, 2014.

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16

Capsaicin As a Therapeutic Molecule. Springer, 2014.

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17

Capsaicin: Food Sources, Medical Uses and Health Implications. Nova Science Pub Inc, 2014.

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18

Mozsik, Gyula, ed. Capsaicin and its Human Therapeutic Development. InTech, 2018. http://dx.doi.org/10.5772/intechopen.73124.

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19

Mózsik, Gyula. Capsaicin and Its Human Therapeutic Development. IntechOpen, 2018.

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20

Blokdijk, G. J. Capsaicin; A Clear and Concise Reference. CreateSpace Independent Publishing Platform, 2018.

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21

Srivastava, Sanjay K. Role of Capsaicin in Oxidative Stress and Cancer. Springer London, Limited, 2013.

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22

Srivastava, Sanjay K. Role of Capsaicin in Oxidative Stress and Cancer. Springer, 2013.

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23

Srivastava, Sanjay K. Role of Capsaicin in Oxidative Stress and Cancer. Springer, 2015.

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24

Li, Albert Steven. Structural studies of TRPV1 activation by capsaicin. 2009.

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25

Wood, John N. Capsaicin in the Study of Pain (Neuroscience Perspectives). Academic Press, 1993.

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26

Tang, Michelle Lai Yee. Effect of local anaesthesia on capsaicin-induced neurogenic inflammation. 2003, 2003.

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27

Daniell, Grace. The functional role of capsaicin-sensitive baro- and chemo-afferents. 1990.

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28

Tamura, Connie S. Capsaicin-sensitive enteric neurons: Anatomy, function and role in the short-term control of food intake. 1995.

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29

Mozsik, Gyula, Andras Debreceni, and Kazuichi Okazaki. The Capsaicin- And Helicobacter Strains-Induced Cellular Mechanisms of the Gastric Mucosa in Animals and Humans. Akademiai Kiado, 2001.

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30

Mozsik, Gyula, Omar M. E. Abdel-Salam, and Koji Takeuchi, eds. Capsaicin - Sensitive Neural Afferentation and the Gastrointestinal Tract: from Bench to Bedside. InTech, 2014. http://dx.doi.org/10.5772/57289.

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31

O'Connell, Finbarr. Enhanced sensitivity of capsaicin-sensitive afferent airway nerves inthe pathogenesis of cough. 1994.

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32

Salam, Omar Abdel. Capsaicin - Sensitive Neural Afferentation and the Gastrointestinal Tract: From Bench to Bedside. Intechopen, 2014.

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33

Nagy, Istvan. VR1 in inflammatory thermal hyperalgesia. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0028.

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The landmark paper discussed in this chapter, published by Davis et al. in 2000, describes the role of the capsaicin receptor, which is called transient receptor potential cation channel subfamily vanilloid member 1 (TRPV1), in inflammatory thermal hyperalgesia. Capsaicin, the pungent agent found in hot peppers, has been linked to pain for centuries because it induces a burning pain sensation which, after prolonged application of the agent, turns into analgesia. Because of this, capsaicin has been used to relieve pain, most likely since prehistoric times. The elucidation of the role of TRPV1 in nociceptive processing was heralded as the starting point for the development of agents which would revolutionize pain management. Unfortunately, that promise is yet to be realized and apparently we need a more detailed understanding of the role of TRPV1 in physiological and pathological processes in order to fulfil the analgesic potential of drugs acting on this receptor.

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34

Schutze, Christopher. Capsaicin: Der Chili-Inhaltsstoff -Wirkungen Auf Den Menschlichen Körper-Auf Basis Wissenschaftlicher Erkenntnisse. Independently Published, 2019.

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35

South, Elizabeth H. Depletion of substance P immunoreactivity and alterations in cholecystokinin-induced reduction of food intake following capsaicin treatment. 1986.

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36

Abhishek, Abhishek, Adrian Jones, and Michael Doherty. Topical pharmacological treatments. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0028.

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Topical pharmacological agents such as non-steroidal anti-inflammatory drugs (NSAIDs) and capsaicin are widely recommended as first-line analgesics in the treatment of osteoarthritis (OA) of the knee, hand, and potentially other peripheral joints in view of their safety and efficacy. Although initial studies were short in duration (2–4 weeks), recent randomized controlled trials have confirmed the efficacy of topical NSAIDs over longer (12-week) study periods. Systematic reviews demonstrate that their efficacy can be equivalent to oral NSAIDs for OA pain, but they have a significantly better systemic toxicity profile than the corresponding oral formulations. Topical capsaicin is less well studied than topical NSAIDs but has been demonstrated to be effective in several placebo-controlled clinical trials. Local warming and an uncomfortable burning sensation is a common problem with initial applications, but this subsides with continued treatment and can be minimized by using a low-strength preparation (e.g. 0.025%) initially. Several other topical treatments such as drug-free transfersome gel and local lignocaine patches have been shown to be effective in controlling pain due to OA. However, they have been studied in relatively few studies and currently are not recommended for general use.

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37

(Editor), Annika B. Malmberg, and Keith R. Bley (Editor), eds. Turning up the Heat on Pain: TRPV1 Receptors in Pain and Inflammation (Progress in Inflammation Research). Birkhäuser Basel, 2005.

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38

Hunter, Kellye, Dave Dewitt, and Melissa T. Stock. El Poder Curativo De Los Pimientos/The Healing Powers of Peppers. Random House Espanol, 2001.

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39

Otis, James A. D. Non-Opioid Pharmacotherapies for Chronic Pain (DRAFT). Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190265366.003.0015.

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The objective of chapter 15 is to describe analgesic approaches to chronic pain, excluding opioids. As such, it emphasizes, first, the available pharmacotherapies; and then procedures. The pharmacotherapies divide into analgesics, such as non-steroidal anti-inflammatory drugs (NSAIDs); adjuvant analgesics, such as tricyclic antidepressants and anticonvulsants; oral anesthetic agents (cardiotropics); adrenergic agonists; topical agents such as capsaicin and local anesthetic solutions and ointments; and muscle relaxants such as cyclobenzaprine, tizanidine, and baclofen. Interventions include many best administered by anesthesiologists such as infusions of anesthetic agents; trigger point injections; local and regional blockade, spinal injections including corticosteroids; and electrical spinal cord stimulation. A text box is provided with additional resources.

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40

Stammen, Katherine, Harish Siddaiah, Cody Brechtel, Elyse M. Cornett, Charles J. Fox, and Alan D. Kaye. Pain Management for General Surgery. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190457006.003.0006.

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Pain is multidimensional and subjective, which makes it difficult to treat. Newer treatment modalities have been under development with a better understanding of pain pathways in recent years. These treatments take advantage of the multifactorial components of pain, including agents such as ketamine, capsaicin, gabapentin, pregabalin, long-acting opioids, peripheral nerve blockade, and patient-controlled analgesia. Numerous studies have revealed not only efficacy but additive and/or synergistic effects when multiple agents are utilized for pain management. Overall, adequate perioperative pain control is important both in an acute setting and in preventing the development of a chronic pain condition, which causes significant short- and long-term negative consequences. Best practice strategies are being utilized based on clinical studies to reduce pain and improve patient needs after surgery.

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41

Finnerup, Nanna Brix, and Troels Staehelin Jensen. Management issues in neuropathic pain. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656097.003.0133.

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Neuropathic pain is a common complication to cancer, cancer treatment, HIV, and other conditions that may affect the somatosensory nervous system. Neuropathic pain may be present in up to 40% of cancer patients and may persist independently of the cancer and affect the quality of life in disease-free cancer survivors. Particular surgical treatment and chemotherapy may cause chronic persistent neuropathic pain in cancer survivors. The diagnosis of neuropathic pain can be challenging and requires documentation of a nervous system lesion and pain in areas of sensory changes. The pharmacological treatment may include tricyclic antidepressants, selective serotonin noradrenaline reuptake inhibitors (duloxetine or venlafaxine), calcium channel α2↓ agonists (gabapentin or pregabalin), and opioids. Topical lidocaine and capsaicin, NMDA antagonists, carbamazepine, oxcarbazepine, and cannabinoids may be indicated. Due to limited efficacy or intolerable side effects at maximal doses, combination therapy is often required and careful monitoring of effect and adverse reactions is important.

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42

1966-, Malmberg Annika B., and Bley Keith R, eds. Turning up the heat on pain: TRPVI receptors in pain and inflammation. Boston, MA: Birkhauser Verlag, 2005.

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43

Wenham, Claire Y. J., and Philip G. Conaghan. Osteoarthritis—management. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0140.

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Osteoarthritis (OA) is a common condition which often causes pain and functional limitation, significantly impacting on a person's quality of life. A comprehensive assessment of the impact of OA should be performed before selecting therapies and treatment goals. Current recommended therapies include a combination of pharmacological and non-pharmacological therapies, which should be considered for all people with OA, regardless of anatomical site of involvement. Non-pharmacological treatments include education, muscle strengthening and aerobic exercises, weight loss if appropriate, splints and devices, and aids. Pharmacological therapies include paracetamol, oral and topical non-steroidal anti-inflammatory drugs, topical capsaicin, intra-articular corticosteroid injections, and opioids. Many existing therapies have only a small analgesic effect size and, in the case of drug therapies, may be associated with important side effects, so an individual's symptoms and comorbidities must be taken into account when selecting therapies. For those who do not respond to these treatments, surgery such as a total joint arthroplasty may be required. There is a strong need for new analgesic treatments for OA. As it is becoming increasingly clear that the sources of pain in OA are complex and multifactorial, future treatments for OA will need to target both peripheral and central pain mechanisms.

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