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Relevant bibliographies by topics / Cancer transcription / Books

Books on the topic 'Cancer transcription'

To see the other types of publications on this topic, follow the link: Cancer transcription.

Author: Grafiati

Published: 11 March 2023

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1

Kumar, Rakesh, ed. Nuclear Signaling Pathways and Targeting Transcription in Cancer. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-8039-6.

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2

Rakesh, Kumar. Nuclear signaling pathways and targeting transcription in cancer. New York: Humana Press, 2014.

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3

B, La Thangue Nicholas, and Bandara Lasantha R, eds. Targets for cancer chemotherapy: Transcription factors and other nuclear proteins. Totowa, N.J: Humana Press, 2002.

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4

B, La Thangue Nicholas, and Bandara Lasantha R, eds. Targets for cancer chemotherapy: Transcription factors and other nuclear proteins. Totowa, N.J: Humana Press, 2002.

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5

1939-, Wilson Samuel H., and Hoagland Mahlon B, eds. Cancer biology and biosynthesis. Boca Raton, Fla: CRC Press, 1991.

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6

Sanno, Naoko. Cytochemical and molecular biological aspects of the pituitary and pituitary adenomas: Cell differentiation and transcription factors. Jena, Germany: Urban & Fischer, 2001.

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7

Pappas, Kyrie Jean. Transcriptional control of tumor suppressor genes in cancer. [New York, N.Y.?]: [publisher not identified], 2017.

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8

Eric, Verdin, ed. Histone deacetylases: Transcriptional regulation and other cellular functions. Totowa, N.J: Humana Press, 2006.

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9

Kumar, Rakesh. Nuclear Signaling Pathways and Targeting Transcription in Cancer. Humana, 2015.

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10

Thurston, David E., and Khondaker Miraz Rahman. Small-Molecule Transcription Factor Inhibitors in Oncology. Royal Society of Chemistry, The, 2018.

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11

Rahman, Khondaker Miraz, and David Thurston. Small-Molecule Transcription Factor Inhibitors in Oncology. Royal Society of Chemistry, The, 2018.

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12

Rahman, Khondaker Miraz, and David Thurston. Small-Molecule Transcription Factor Inhibitors in Oncology. Royal Society of Chemistry, The, 2018.

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13

Nicholas B. La Thangue (Editor) and Lasantha R. Bandara (Editor), eds. Targets for Cancer Chemotherapy (Cancer Drug Discovery and Development). Humana Press, 2002.

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14

Thangue, Nicholas B. La, and Lasantha R. Bandara. Targets for Cancer Chemotherapy: Transcription Factors and Other Nuclear Proteins. Humana Press, 2010.

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15

Thangue, Nicholas B. La, and Lasantha R. Bandara. Targets for Cancer Chemotherapy: Transcription Factors and Other Nuclear Proteins. Humana Press, 2012.

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16

Thangue, Nicholas B. La, and Lasantha R. Bandara. Targets for Cancer Chemotherapy: Transcription Factors and Other Nuclear Proteins. Humana Press, 2002.

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17

Hodgkiss, Andrew. Introduction to cancer biology. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198759911.003.0001.

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A brief introduction to cancer biology, aimed at psychiatrists, is offered. Selective DNA transcription, the cell cycle, receptor tyrosine kinases, and cell signalling pathways are introduced, using the EGFR/RAS/MAPK pathway as an exemplar. The molecular pathology of oncogenesis is summarized, including discussion of oncogenes, tumour suppressor genes, and examples of driver mutations. The exploitation of such mutations in stratified medicine, using molecularly targeted agents, is mentioned. Finally, Hanahan and Weinberg’s six hallmarks of cancer are listed, adding angiogenesis and metastasis to the picture of oncogenesis.

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18

McFarland, Daniel C., and Jimmie Holland. Depression and Cancer. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190603342.003.0006.

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The relationship between depression and cancer has long captured the imagination of clinicians and the lay population. Therefore, the science behind this putative relationship is paramount to determine reality from myth. This chapter begins with a historical and relevant clinical overview from within the context of psycho-oncology and psychoneuroimmunology. An exploration of the association between cancer and depression follows by reviewing cancer initiation and progression data in the context of depression. Biological correlates of the stress response in depression, inflammation, and its effects on cancer are presented. Social attributes to these biological phenomena are also evaluated through the putative mechanisms of epigenetics and the stress response. The strongest data for the relationship between depression and cancer fall into four distinct areas: (1) the cytokine hypothesis of depression; (2) dysregulation of the HPA, glucocorticoids, and diurnal circadian rhythms; (3) enhanced sympathetic nervous system activity; and (4) alterations in DNA protein transcription/epigenetics.

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19

Alves, Ines Teles, Jan Trapman, and Guido Jenster. Molecular biology of prostate cancer. Edited by James W. F. Catto. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0059.

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Prostate cancer is a heterogeneous disease that arises through the acquisition of key malignant hallmarks. At the molecular level, prostate tumours are dependent upon the androgen receptor pathway, which affects cell function, growth, and behaviour through downstream androgen-regulated genes. Prostate cancer requires this activity and manipulates the AR pathway to maintain signalling. For example, mutation of the AR (to bind ligands other than androgens) or amplification/duplication of the AR allows signalling to continue in the absence of testosterone. Around 50% of prostate cancers have a gene fusion between the androgen-regulated component of the TMPRSS2 gene and a transcription factor (e.g. ETS family members ERG and ETV1). This results in aberrant androgen stimulated cell growth. Current research is using molecular knowledge to identify biomarkers, such as PCA3, and new therapies, such as enzalutamide or abiraterone acetate.

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20

Bins, Adriaan D. Induction and Analysis of Antigen-specific T cell Responses in Melanoma Patients and Animal Models. Amsterdam University Press, 2007.

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21

Connor, Thomas, and Patrick H. Maxwell. Hypoxia-inducible factor and renal disorders. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0331.

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Hypoxia-inducible factors (HIFs) are transcription factors that control the cellular response to changes in oxygen levels. This response is common to all cells in the body and is highly conserved in evolution. The kidney exhibits steep gradients in oxygenation which are important in the homeostatic response to anaemia. The cellular response to low levels of oxygen (hypoxia) also plays a role in such diverse processes as acute kidney injury, the progression of chronic kidney disease, and kidney cancer. There is now considerable interest in using drugs to manipulate the HIF response to treat these varied conditions.

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22

Cold Spring Harbor Symposia on Quantitative Biology: Molecular Genetics of Cancer (Cold Spring Harbor Symposia on Quantitative Biology). Cold Spring Harbor Laboratory Press, 1995.

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23

Zhou, Wen, and Carol Prives, eds. Transcriptional Regulation in Cancers and Metabolic Diseases. Frontiers Media SA, 2015. http://dx.doi.org/10.3389/978-2-88919-712-5.

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24

Ye, Qinong, Changliang Shan, Binghui Li, Pei Wang, and Bin Yuan, eds. Post-Transcriptional and Post-Translational Regulation of Cancer Metabolism. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88974-004-8.

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25

Abbas, Ata, and Rais Ahmad Ansari, eds. Epigenetic and Transcriptional Dysregulations in Cancer and Therapeutic Opportunities. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88976-657-4.

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26

Verdin, Eric. Histone Deacetylases: Transcriptional Regulation and Other Cellular Functions. Humana Press, 2010.

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27

Verdin, Eric. Histone Deacetylases: Transcriptional Regulation and Other Cellular Functions. Humana Press, 2007.

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28

(Editor), Bruce Stillman, and David Stewart (Editor), eds. Regulatory RNAs. Cold Spring Harbor Laboratory Press, 2007.

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29

Verdin, Eric. Histone Deacetylases: Transcriptional Regulation and Other Cellular Functions (Cancer Drug Discovery and Development). Humana Press, 2006.

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30

Chaudhry, Raazia. Transcriptional profiling of xenografts as a model system for classification of lung cancer. 2004.

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31

Histone deacetylases: Transcriptional regulation and other cellular functions. Totowa, NJ: Humana Press, 2006.

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32

Laboratory, Cold Spring Harbor. Cold Spring Harbor Symposia on Quantitative Biology: The Cell Cycle (Cold Spring Harbor Symposia on Quantitative Biology) (Cold Spring Harbor Symposia on Quantitative Biology). Cold Spring Harbor Laboratory Press, 1992.

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33

Laboratory, Cold Spring Harbor. Cold Spring Harbor Symposia on Quantitative Biology. Cold Spring Harbor Laboratory Press, 1998.

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34

Cold Spring Harbor Symposia on Quantitative Biology: Function & Dysfunction in the Nervous System (Cold Spring Harbor Symposia on Quantitative Biology) ... Harbor Symposia on Quantitative Biology). Cold Spring Harbor Laboratory Press, 1997.

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35

Cold Spring Harbor Symposia on Quantitative Biology: DNA and Chromosomes (Cold Spring Harbor Symposia on Quantitative Biology). Cold Spring Harbor Laboratory Press, 1994.

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36

Laboratory, Cold Spring Harbor. Cold Spring Harbor Symposia on Quantitative Biology: Immunological Recognition (Cold Spring Harbor Symposia on Quantitative Biology). Cold Spring Harbor Laboratory Press, 1990.

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37

New York Academy of Sciences Staff (Contributor) and Marc Diederich (Editor), eds. Signal Transduction Path Part C: Stress Signaling and Transcriptional Control (Annals of the New York Academy of Sciences). Blackwell Publishing Limited, 2007.

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