Academic literature on the topic 'Cancer – Mortality – New South Wales'

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Journal articles on the topic "Cancer – Mortality – New South Wales"

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McCredie, Margaret, Marylon S. Coates, Peter Day, and Jane C. Bell. "Changes in cancer incidence and mortality in New South Wales." Medical Journal of Australia 163, no. 10 (November 1995): 520–23. http://dx.doi.org/10.5694/j.1326-5377.1995.tb124717.x.

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Taylor, Richard, Stephen Morrell, Jane Estoesta, and Ann Brassil. "Mammography Screening and Breast Cancer Mortality in New South Wales, Australia." Cancer Causes & Control 15, no. 6 (August 2004): 543–50. http://dx.doi.org/10.1023/b:caco.0000036153.95908.f2.

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Burnley, I. H. "Mortality from respiratory system cancer in New South Wales and Sydney." Australian Journal of Public Health 16, no. 3 (February 12, 2010): 251–61. http://dx.doi.org/10.1111/j.1753-6405.1992.tb00063.x.

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Supramaniam, Rajah, Hari Grindley, and Lisa Jackson Pulver. "Cancer mortality in Aboriginal people in New South Wales, Australia, 1994-2002." Australian and New Zealand Journal of Public Health 30, no. 5 (October 2006): 453–56. http://dx.doi.org/10.1111/j.1467-842x.2006.tb00463.x.

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Burnley, I. H. "Stomach cancer mortality in New South Wales and Sydney, 1980 to 1985." Australian Journal of Public Health 15, no. 2 (February 12, 2010): 88–100. http://dx.doi.org/10.1111/j.1753-6405.1991.tb00317.x.

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Bell, Jane C., Margaret McCredie, Marylon S. Coates, and Bruce K. Armstrong. "Trends in colorectal cancer incidence and mortality in New South Wales, 1973–1992." Medical Journal of Australia 166, no. 4 (February 1997): 178–81. http://dx.doi.org/10.5694/j.1326-5377.1997.tb140070.x.

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Burnley, I. H. "Disadvantage and male cancer incidence and mortality in New South Wales 1985–1993." Social Science & Medicine 45, no. 3 (August 1997): 465–76. http://dx.doi.org/10.1016/s0277-9536(96)00366-8.

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Smith, David, Richard Taylor, and Marylon Coates. "Socioeconomic differentials in cancer incidence and mortality in urban New South Wales, 1987-1991." Australian and New Zealand Journal of Public Health 20, no. 2 (April 1996): 129–37. http://dx.doi.org/10.1111/j.1753-6405.1996.tb01806.x.

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Smith, Ross C., Nicola Creighton, Reginald V. Lord, Neil D. Merrett, Gregory W. Keogh, Winston S. Liauw, and David C. Currow. "Survival, mortality and morbidity outcomes after oesophagogastric cancer surgery in New South Wales, 2001–2008." Medical Journal of Australia 200, no. 7 (April 2014): 408–13. http://dx.doi.org/10.5694/mja13.11182.

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Lee, Margaret, and Peter Gibbs. "Survival, mortality and morbidity outcomes after oesophagogastric cancer surgery in New South Wales, 2001–2008." Medical Journal of Australia 201, no. 8 (October 2014): 447. http://dx.doi.org/10.5694/mja14.00870.

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Dissertations / Theses on the topic "Cancer – Mortality – New South Wales"

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Amin, Janaki Public Health &amp Community Medicine Faculty of Medicine UNSW. "Hepatitis B and C associated cancer and mortality: New South Wales, 1990-2002." Awarded by:University of New South Wales. School of Public Health and Community Medicine, 2006. http://handle.unsw.edu.au/1959.4/27338.

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This thesis examines cancer and mortality rates among people diagnosed with hepatitis B (HBV) and C (HCV) infection in New South Wales (NSW) from 1990 through 2002, by linking hepatitis notifications with the NSW Central Cancer Registry (CCR) and National Death Index. Of the 39101 HBV, 75834 HCV and 2604 HBV/HCV co-infection notifications included 1052, 1761 and 85 were linked to cancer notifications and 1233, 4008 and 186 were linked to death notifications respectively. Of 2072 hepatocellular carcinoma (HCC) notifications to the CCR 323, 267 and 85 were linked to HBV, HCV and HBV/HCV co-infection notifications. Incidence of HCC was 6.5, 4.0 and 5.9 per 1000 person years for HBV, HCV and HBV/HCV co-infected groups. Risk of HCC in those diagnosed with hepatitis was 20 to 30 times greater than the standard population. There was a marginally statistically significant increased risk of immunoproliferative malignancies associated with HCV infection (SIR=5.6 95% CI 1.8 ???17.5). Risk of death for those with hepatitis was significantly greater, 1.5 to 5 fold, than the general population with the greatest risk among those with HBV/HCV co-infection. The primary cause of HBV deaths was liver related, particularly HCC, whereas in the HCV groups drug related deaths were most frequent. Among people with HCV, risk of dying from drug related causes was significantly greater than from liver related causes (p=0.012), with the greatest increased risk in females age 15- 24 years (SMR 56.9, 95%CI 39.2???79.9). Median age at diagnosis of HCC varied markedly by country of birth and hepatitis group: HBV 66, 63 and 57years ; HCV 51, 68 and 71 years; unlinked 69, 70 and 64 years for Australian, European, and Asian-born groups, respectively (P<0.0001 for all groups). While the risk of cancer, particularly HCC, is elevated among people with HBV and HCV infection, the absolute risk remains low. Young people with HCV face a higher mortality risk from continued drug use than from liver damage related to their HCV infection. The influence of IDU in the epidemiology of HCC in New South Wales was possibly reflected in the varying distributions of age and country of birth.
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Kariminia, Azar Public Health &amp Community Medicine Faculty of Medicine UNSW. "Death among a cohort of prisoners in New South Wales Australia ??? a data linkage study." Awarded by:University of New South Wales. School of Public Health and Community Medicine, 2007. http://handle.unsw.edu.au/1959.4/32476.

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This thesis examines mortality rates among adults who experienced full-time imprisonment in New South Wales between January 1988 and December 2002, by record linkage to the Australian National Death Index. The cohort included 76383 men and 8820 women. Over a mean follow-up of 7.7 years, 5137 deaths (4724 men, 423 women) were identified. Three hundred and three deaths (295 men, eight women) occurred in custody. The median age at death was 36.6 years for men and 32.7 years for women. The prominent causes of death were drug overdose, suicide, accidental and cardiovascular disease. The crude mortality rate was 797 per 100000 person-years for men and 685 per 100000 person-years for women. Risk of mortality was 3.7 times greater in male and 7.8 times greater in female prisoners than the standard population. The excess mortality was substantially raised following release from prison in both men (standardised mortality ratio 4.0 vs 1.7) and women (standardised mortality ratio 8.2 vs 2.1). The period of highest risk of death was the first two weeks after release. Drug overdose was the main cause of death, responsible for 68% of the deaths in the first two weeks for men and for 90% of the deaths in this period for women. In men, there was also a clustering of suicide directly after release. Prisoners admitted to prison psychiatric hospital, repeat offenders and those in the early stage of followup were at increased risk of mortality. Violent offenders were overrepresented in suicide figures and property offenders in death from overdose. Minority groups, in particular men, had a lower risk of death than white people. The above findings reinforce how disadvantaged prisoners are, measured by mortality as the most fundamental scale of human wellbeing. Prison represents a potential opportunity for treatment and public health intervention to address some of the health problems underlying the high mortality found in this study. The key challenge is, however, to provide a continuum of care between the prison and community.
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Vinod, Shalini Kavita Public Health &amp Community Medicine Faculty of Medicine UNSW. "A lung cancer patterns of care study in the South Western Sydney Area Health Service." Awarded by:University of New South Wales. School of Public Health and Community Medicine, 2004. http://handle.unsw.edu.au/1959.4/22463.

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Background: The South Western Sydney Area Health Service (SWSAHS) contains many areas of socio-economic disadvantage and ethnic diversity. It has a high incidence of lung cancer, which is the leading cause of cancer deaths. The aims of this study were to document lung cancer patterns of care (POC) for SWSAHS residents, compare POC before and after the opening of an oncology centre in SWSAHS and compare POC with other areas in NSW. Methods: The study population consisted of SWSAHS residents diagnosed with lung cancer in 1993 and 1996. A clinical audit of medical records was performed to extract details on patient demographics, management of lung cancer and outcomes. Collaborating investigators performed identical studies in the Northern Sydney Area Health Service (NSAHS) and the Hunter Area Health Service (HAHS) for lung cancers diagnosed in 1996. Results: The SWSAHS study population comprised 527 patients. Nine percent did not have a pathological diagnosis. Twelve percent did not see a lung cancer specialist. Twenty-eight percent did not receive any treatment throughout the course of their illness. The median survival was 6.7 months and five-year actuarial survival was 8% (95% CI 6%-10%). Increasing age and poorer performance status were associated with a lower likelihood of obtaining a pathological diagnosis, specialist referral and treatment. Socio-economic factors did not influence POC. The establishment of an oncology center resulted in more referrals to medical oncologists and palliative care services. Other aspects of POC and survival were similar. Variability in POC was noted between SWSAHS, NSAHS and HAHS. HAHS residents were almost twice as likely not to have pathological confirmation of diagnosis or treatment. Despite this survival was not significantly different. Conclusions: This study has identified deficiencies in the management of lung cancer. To improve outcomes, referral to specialists and utilisation of treatment, particularly radiotherapy and chemotherapy, needs to be increased. Ageist and nihilistic attitudes need to be overcome. Prospective data collection is necessary to ensure quality of patient care. The formation of national guidelines for the management of lung cancer will play an important role in achieving better outcomes.
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Books on the topic "Cancer – Mortality – New South Wales"

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Nsw Commission for Children & Young Peop. Sudden Unexpected Deaths in Infancy: The New South Wales Experience. Nsw Commission for Children & Young People, 2005.

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Alan, Gray, and National Centre for Epidemiology and Population Health (Australia), eds. A Matter of life and death: Contemporary aboriginal mortality : proceedings of a workshop of the National Centre for Epidemiology and Population Health held at Kioloa, New South Wales, 10-12 July 1989. Canberra: Aboriginal Studies Press, 1990.

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Book chapters on the topic "Cancer – Mortality – New South Wales"

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Lassau, Scott A., Brendan Ryan, Robert Close, Chris Moon, Pascal Geraghty, Ann Coyle, and John Pile. "Home ranges and mortality of a roadside Koala Phascolarctos cinereus population at Bonville, New South Wales." In Too close for comfort, 127–36. P.O. Box 20, Mosman NSW 2088, Australia: Royal Zoological Society of New South Wales, 2008. http://dx.doi.org/10.7882/fs.2008.018.

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Henry, David A., Pamela R. Hall, Anne Johnston, and Annette Dobson. "NSAIDs as a Cause of Morbidity and Mortality from Peptic Ulcer Complications in New South Wales." In Pharmacoepidemiology, 255–60. Routledge, 2018. http://dx.doi.org/10.1201/9780203743669-24.

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Robinson, Max, Keith Hunter, Michael Pemberton, and Philip Sloan. "Oral cancer." In Soames' & Southam's Oral Pathology. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780199697786.003.0008.

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The term ‘oral cancer’ encompasses all malignant neoplasms affecting the oral cavity. The majority, greater than 90%, are squamous cell car¬cinomas. The remainder are uncommon and comprise minor salivary gland adenocarcinomas, malignant melanoma, sarcomas, haemato-logical malignancies, and metastases to the oral cavity from cancers at other sites. Oral squamous cell carcinoma is a malignant epithelial neoplasm that arises from the lining mucosa of the oral cavity. The tumour shows vary¬ing degrees of squamous differentiation and is characterized by invasion of local structures and metastasis to regional lymph nodes, followed by metastasis to other organ systems (e.g. lungs and bones) later in the course of the disease. Epidemiological data pertaining to oral cancer can be difficult to evalu¬ate because of variations in the methods of data collection (Box 3.1). Notwithstanding these confounding variables, a database produced by the International Agency for Research on Cancer (GLOBOCAN), esti-mated there were over 400,000 new cases of lip, oral, and pharyngeal cancer worldwide in 2012, placing the disease in ninth position with breast, prostate, lung, colorectal, cervical, stomach, liver, and uterine cancer being more common. These data suggest that oral cancer is uncommon, but there are enormous variations worldwide. Whereas oral cancer is relatively uncommon in the UK, accounting for 2% of all cancers, in India and parts of South-East Asia it is the most common malignant neoplasm and accounts for around a third of all cancers. Furthermore, the incidence rates for large countries, such as India and the USA, conceal regional and ethnic variations. For example, incidence rates tend to be higher in urban as opposed to rural communities, and in the USA are higher for blacks than whites. In the United Kingdom, inci¬dence rates are slightly higher in Scotland than in England and Wales. In the United Kingdom the incidence of oral cancer is 9 per 100,000 of the population, which represents around 6,800 new cases per annum. The disease is more common in men than in women; the male:female ratio is currently 2:1. Oral cancer incidence increases with age, and the majority of cases (greater than two-thirds) are diagnosed after the age of 50 years old; less than 5% occur in individuals below the age of 40 years old.
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Conference papers on the topic "Cancer – Mortality – New South Wales"

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Nair-Shalliker, Visalini, David P. Smith, Sam Egger, Ann Marie Hughes, Mark Clements, Anne Kricker, and Bruce K. Armstrong. "Abstract 5482: Sun exposure and prostate cancer risk in New South Wales, Australia: A case control study." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-5482.

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Jacklyn, Gemma, Kevin McGeechan, Les Irwig, Nehmat Houssami, Stephen Morrell, Katy Bell, and Alexandra Barratt. "48 Trends in stage-specific breast cancer incidence in new south wales, australia: insights from 25 years of screening mammography." In Preventing Overdiagnosis, Abstracts, August 2018, Copenhagen. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/bmjebm-2018-111070.48.

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Salagame, Usha G., Emily Banks, Dianne O’Connell, Sam Egger, and Karen Canfell. "Abstract 2285: Menopausal hormone therapy (MHT) use and breast cancer risk by receptor subtypes: results from the New South Wales Cancer Lifestyle and EvAluation of Risk (CLEAR) study." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-2285.

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Batra, Ankit. "Clinical comparison of toxicity pattern of two linear quadratic model-baesd fractionation schemes of high-dose-rate intracavitary brachytherapy for cervical cancer." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685255.

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Introduction: Carcinoma cervix is the fourth (GLOBACON 2012) most common cancer among women worldwide, and the main cancer affecting women in Sub-Saharan Africa, Central America and south-central Asia. In India, approx. 1,23,000 (GLOBACON 2012) new cases of carcinoma cervix are diagnosed each year. Brachytherapy is an integral part of treatment of cancer cervix. In the context of a developing country like us where maximum utilization of the resource is of prime importance to provide treatment to the large patient cohort, shortening the treatment duration and number of fractions always increases efficiency. In order to maximize the logistic benefits of HDR-BT while improving patient compliance and resource sparing, various fractionation regimens are used. Fractionation and dose adjustments of the total dose are radiobiologically important factors in lowering the incidence of complications without compromising the treatment results. Aim: To compare patient outcomes and complications using two linear-quadratic model-based fractionation schemes of high-dose-rate intracavitary brachytherapy (HDR-IC) used to treat cervical cancer. Materials and Methods: A prospective randomized study on 318 patients, with histologically proven advanced carcinoma cervix (stages IIB-IIIB) was enrolled in the study. All patients received External Beam Radio Therapy (EBRT) 50 Gy in 25 fractions with concurrent chemotherapy (cisplatin 35 mg/m2) followed by IntraCavitary brachytherapy using high dose rate equipment. Patients were randomised after completion of EBRT into two arms: (1) Arm 1: HDR ICRT 6.5 Gy per fraction for 3 fractions, a week apart. (2) Arm 2: HDR ICRT, 9 Gy per fraction for 2 fractions, 1 week apart. On completion of treatment, patients were assessed monthly for 3 months followed by 3 monthly thereafter. Treatment response was assessed according to WHO criteria after one month of completion of radiotherapy. The RTOG criteria were used for radiation induced toxicities. We analyzed late toxicities in terms of Rectal, Bladder, Small Bowel toxicity and Vaginal Stenosis. Results: Acute reactions in both the groups were comparable. None of the patient developed Grade 4 toxicity in our study and no toxicity related mortality was encountered. A slightly high frequency of late toxicity was observed in 9Gy Arm patients but was not statistically significant. Conclusion: In our setup, HDR brachytherapy at 9 Gy per fraction in two fractions is safe, effective and resource saving method with good local control, survival, and manageable normal tissue toxicity.
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