Relevant bibliographies by topics / Cancer identity

Academic literature on the topic 'Cancer identity'

Author: Grafiati
Published: 10 December 2022
Last updated: 28 January 2023

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Journal articles on the topic "Cancer identity"

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Mathieson, Cynthia M., and Henderikus J. Stam. "Renegotiating identity: cancer narratives." Sociology of Health and Illness 17, no. 3 (June 1995): 283–306. http://dx.doi.org/10.1111/1467-9566.ep10933316.

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Male, Dana A., Karen D. Fergus, and Kimberley Cullen. "Sexual identity after breast cancer." Current Opinion in Supportive and Palliative Care 10, no. 1 (March 2016): 66–74. http://dx.doi.org/10.1097/spc.0000000000000184.

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Kiberstis, Paula A. "Evading cancer drugs by identity fraud." Science 355, no. 6320 (January 5, 2017): 35.16–37. http://dx.doi.org/10.1126/science.355.6320.35-p.

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Wou, C., N. Chaston, and S. Doughan. "Mistaken identity: endometrial or rectal cancer?" Case Reports 2014, may20 1 (May 20, 2014): bcr2013202874. http://dx.doi.org/10.1136/bcr-2013-202874.

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Roy, Nilotpal, and Matthias Hebrok. "Regulation of Cellular Identity in Cancer." Developmental Cell 35, no. 6 (December 2015): 674–84. http://dx.doi.org/10.1016/j.devcel.2015.12.001.

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Ruffenach, G., S. Bonnet, S. Rousseaux, S. Khochbin, S. Provencher, and F. Perros. "Identity crisis in pulmonary arterial hypertension." Pulmonary Circulation 8, no. 1 (December 5, 2017): 204589321774605. http://dx.doi.org/10.1177/2045893217746054.

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Pulmonary arterial hypertension (PAH) shares many hallmarks with cancer. Cancer cells acquire their hallmarks by a pathological Darwinian evolution process built on the so-called cancer cell “identity crisis.” Here we demonstrate that PAH shares the most striking features of the cancer identity crisis: the ectopic expression of normally silent tissue-specific genes.
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Sehjae Chun. "Identity Excised: Mastectomy and Women’s Identity in Audre Lorde’s The Cancer Journals." English & American Cultural Studies 9, no. 2 (August 2009): 185–206. http://dx.doi.org/10.15839/eacs.9.2.200908.185.

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Cheung, Sze Yan, and Paul Delfabbro. "Are you a cancer survivor? A review on cancer identity." Journal of Cancer Survivorship 10, no. 4 (February 11, 2016): 759–71. http://dx.doi.org/10.1007/s11764-016-0521-z.

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Huang, Qin, Matthew Read, Jason S. Gold, and Xiao Ping Zou. "Unraveling the identity of gastric cardiac cancer." Journal of Digestive Diseases 21, no. 12 (October 28, 2020): 674–86. http://dx.doi.org/10.1111/1751-2980.12945.

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Kiberstis, P. A. "Clues to cancer from an identity change." Science 352, no. 6293 (June 23, 2016): 1530–32. http://dx.doi.org/10.1126/science.352.6293.1530-q.

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Dissertations / Theses on the topic "Cancer identity"

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Masson, Sarah Jane. "Identity, meaning making and cancer survivorship." Thesis, University of Exeter, 2014. http://hdl.handle.net/10871/15372.

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Purpose: Many lives are affected by cancer. The number of people in England who have had a diagnosis of cancer exceeds one million. Previous research shows that one third of patients have unmet needs post-discharge from cancer treatment, including psychological issues such as negative impacts on self-identity and a lack of meaning in life. Studies have identified identity as an important factor in meaning making, but evidence regarding cancer’s impact on identity is limited to specific cancer sites and specific identity roles. Little is known about cancer’s general impact on global identity or how threats to identity relate to meaning making. The aim of this study was to understand patients’ experiences of cancer’s impact on their identity and what sense they made of these experiences. Methods: Twelve participants in the post-treatment phase of cancer shared their experiences in individual semi-structured interviews. Key themes regarding identity and meaning making in the post-treatment phase were identified using interpretative phenomenological analysis (IPA). Results and Conclusions: Four key themes in the participants’ experiences were identified. These were 1) disrupted identity roles, 2) highlights what is important, 3) focused on priorities, and 4) reducing awareness of loss and uncertainty. Relevant literature and implications for future research and clinical practice are discussed.
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Ng, Tsz-yin Carina. "Illness, ideology, and identity the "pregnancy" of cancer /." Click to view the E-thesis via HKUTO, 2007. http://sunzi.lib.hku.hk/hkuto/record/B38671074.

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Ng, Tsz-yin Carina, and 伍紫燕. "Illness, ideology, and identity: the "pregnancy" of cancer." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B38671074.

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Palmer-Wackerly, Angela Lynn. "Illness Identity, Social Support, and Cancer Treatment Decision-Making." The Ohio State University, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=osu1434452561.

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van, Willigen Marieke Minke. "Collective identity and activist strategies in the breast cancer movement." The Ohio State University, 1993. http://rave.ohiolink.edu/etdc/view?acc_num=osu1382032315.

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Duvall, Kathryn L., Kelly A. Dorgan, and Sadie P. Hutson. "Personal Identity Changes of Female Cancer Survivors in Southern Appalachia." Digital Commons @ East Tennessee State University, 2012. https://dc.etsu.edu/etsu-works/1228.

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Navigating personal identity changes through the cancer journey can be challenging, especially for women in a culture that places emphasis on traditional gender roles and values close-knit families. Drawing on a story circule approach, this study examined the intersecting identities of female cancer survivors in southern Appalachia. Stories of 29 female Appalachian cancer survivors from Northeast Tennessee and Southwest Virginia were collected via a mixed methods approach in either a day-long story circule (N-26) or an in-depth interview (N=3). Transcripts from both phases were audio-recorded and transcribed verbatim; NVivo 8.0 facilitated qualitative content analysis of the data. Inductive analysis revealed that women in this study appeared in struggle with (1) maintaining place in the family, (2) mothering, and (3) navigating physical changes. Ideas of family versus self appeared to overlap and intertwine with how women in Appalachia navigate personal identify changes through the cancer journey.
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Mourão, Larissa. "Unravelling the identity and fate of Notch1-expressing cells within intestinal tumours." Thesis, Paris 6, 2017. http://www.theses.fr/2017PA066586/document.

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Stem cells and cancer are inextricably linked and many tumours, including colorectal cancers, contain a small population of self-renewing cells, referred to as cancer stem cells (CSCs), able to give rise to proliferating but progressively differentiating cells that contribute to the cellular heterogeneity typical of solid tumours. Thus, the identification of CSCs and the factors that regulate their behaviour should have a profound impact on cancer treatment. Notch signalling controls the maintenance and differentiation of stem cells in several tissues, including the intestine, where it is essential for stem cells maintenance. Based on these premises, my work was aimed at identifying and characterising the cells that express the Notch1 receptor in intestinal tumours in vivo, with the objective of getting insights into the cellular hierarchy of colon cancer cells. We found that the Notch1 receptor is expressed in rare undifferentiated tumour cells that present self-renewal and multipotency in vivo, as they indefinitely give rise to marked differentiated tumour cells and fuel tumour growth. Our analysis on the transcriptomic profile of these cells confirmed our in vivo observations that Notch1+ tumour cells represent a specific population of highly proliferative tumour cells, expressing several, but not all, known markers of normal intestinal stem cells (ISCs). Indeed, their transcriptional signature highly correlates with normal ISCs. Given that the tumour cells we characterised appear not to carry Apc mutations, we hypothesise that during the earlies steps of tumourigenesis, normal Notch1+ ISCs are engulfed within the nascent tumour (in aberrant hyperproliferative crypts) and are able to grow and expand within this new ecosystem, as they are supported by extrinsic secreted growth factors from the neighbouring mutant cells. The concept that normal ISCs might contribute to tumour expansion highlights the complications that patients can encounter during treatment, since these cells share many features with their wild-type counterparts, making therapy deleterious to normal ISCs
Les cellules souches et le cancer sont inextricablement liés et de nombreuses tumeurs, y compris les cancers colorectaux, contiennent une petite population de cellules auto-renouvelables, appelées cellules souches cancéreuses (CSCs), capables de donner naissance à des cellules proliférantes mais progressivement différenciatrices qui contribuent à l'hétérogénéité cellulaire typique des tumeurs solides. Ainsi, l'identification des CSC et des facteurs qui régissent leur comportement devrait avoir un impact profond sur le traitement du cancer. Notch signale le contrôle le maintien et la différenciation des cellules souches dans plusieurs tissus, y compris l'intestin, où elles sont essentielles au maintien des cellules souches. Sur la base de ces prémisses, mes travaux visaient à identifier et caractériser les cellules qui expriment le récepteur Notch1 dans les tumeurs intestinales in vivo, dans le but de mieux comprendre la hiérarchie cellulaire des cellules cancéreuses du colon. Nous avons constaté que le récepteur Notch1 s'exprime dans de rares cellules tumorales indifférenciées qui se renouvellent et se multiplient in vivo, car elles donnent lieu indéfiniment à une différenciation marquée des cellules tumorales et à une croissance tumorale. Notre analyse du profil transcriptomique de ces cellules a confirmé nos observations in vivo selon lesquelles les cellules tumorales Notch1+ représentent une population spécifique de cellules tumorales hautement prolifératives, exprimant plusieurs marqueurs connus, mais pas tous, des cellules souches intestinales normales (CSI). En effet, leur signature transcriptionnelle est fortement corrélée avec les CSI normaux. Étant donné que les cellules tumorales que nous avons caractérisées ne semblent pas être porteuses de mutations Apc, nous supposons que durant les premières étapes de la tumorigénèse, les CSI normales Notch1+ sont englouties dans la tumeur naissante (dans des cryptes hyperprolifératives aberrantes) et sont capables de croître et de s'étendre dans ce nouvel écosystème, car elles sont soutenues par les facteurs de croissance extrinsèques des cellules mutantes voisines. Le concept selon lequel les CSI normaux pourraient contribuer à l'expansion tumorale met en évidence les complications que les patients peuvent rencontrer pendant le traitement, puisque ces cellules partagent de nombreuses caractéristiques avec leurs homologues de type sauvage, ce qui rend le traitement délétère pour les CSI normaux
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Soltermann, Tanya C. "Emergence of a Cancer Identity in Emerging Adulthood: Weblogs as Illness Narratives." Thèse, Université d'Ottawa / University of Ottawa, 2014. http://hdl.handle.net/10393/30653.

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The focus of this research is on the specific relational and particular circumstances that result in an emerging cancer identity expressed through the daily lived- experiences of emerging adults via personal weblogs. Identity, a complex term in its own right, is discussed here under the rubric of social identity as processual, therefore it is expected that an emerging cancer identity will develop as the participants begin to narrativize their daily experiences with cancer on their weblogs. By critically engaging with notions of emerging adulthood theories with theories on the sociology of death and dying and illness narratives, this research seeks to understand the specific psychosocial changes that occur as the participants engage with their illness on their weblogs, which arguably contributes to an emerging cancer identity.
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Duvall, Kathryn L. "Surviving Cancer in Appalachia: A Qualitative Study of Family Cancer Communication and Changing Personal Identities Through the Cancer Journey." Digital Commons @ East Tennessee State University, 2010. https://dc.etsu.edu/etd/1677.

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The Appalachian region is known for its beautiful mountains, close-knit communities, and health care disparities including higher rates of cancer and premature mortality. Being diagnosed with cancer in the region may present a unique experience for survivors in regards to family cancer communication and changing personal identities. In a multiphasic study, the stories of 29 female Appalachian cancer survivors were collected through either a day-long modified story circle event (n=26) or an in-depth interview (n=3). Qualitative content analysis was used to identify emergent themes in the data. The analysis revealed 5 types of family cancer communication and five barriers to family cancer communication. The analysis additionally revealed the identity struggle women experience between maintaining traditional Appalachian gender roles and surviving cancer. These findings suggest that female Appalachian cancer survivors appear to have additional challenges that may make the cancer experience in Appalachia unique.
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Reiter, Paul Lawrence. "Appalachian Self-Identity, Cervical Cancer Screening, and Risky Sexual Behavior Among Women in Ohio Appalachia." The Ohio State University, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=osu1210958568.

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Books on the topic "Cancer identity"

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1948-, Koppelman Kent L., ed. Robbery and redemption: Cancer as identity theft. Amityville, N.Y: Baywood Pub. Co., 2011.

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Risky genes: Genetics, breast cancer, and Jewish identity. Abingdon, Oxon: Routledge, 2013.

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Beck, Guy. Hello, my name Is not cancer: One man's uncensored look at life, death, and identity. Minneapolis: Huff Publishing Associates/Quill House Publishers, 2012.

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Nakahara, Seiichirō. Kanon =: Canon. Tōkyō: Kawade shobō shinsha, 2014.

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Lambert, Sonia. Three mothers. New York: Berkley Books, 2008.

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A stone boat. New York: Plume, 1996.

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Solomon, Andrew. A stone boat. London: Faber and Faber, 1995.

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Three mothers. London: Piatkus, 2007.

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Srivastava, Sudhir. The early detection research network: Translational research to identify early cancer and cancer risk. Washington, D.C.]: U.S. Dept. of Health and Human Services, National Institutes of Health, National Cancer Institute, 2005.

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Rebecca, Sutphen, and Steligo Kathy, eds. Confronting hereditary breast and ovarian cancer: Identify your risk, understand your options, change your destiny. Baltimore: Johns Hopkins University Press, 2012.

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Book chapters on the topic "Cancer identity"

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Ouzounova, Maria, Hector Hernandez-Vargas, and Zdenko Herceg. "Epigenetic Identity in Cancer Stem Cells." In Stem Cells & Regenerative Medicine, 127–39. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-860-7_8.

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Gwyn, Richard. "Processes of Refiguration: Shifting Identities in Cancer Narratives." In Discourse, the Body, and Identity, 209–24. London: Palgrave Macmillan UK, 2003. http://dx.doi.org/10.1057/9781403918543_10.

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Imura, Tetsuya. "Adult Neural Stem Cells; Identity and Regulation." In Stem Cells and Cancer Stem Cells, Volume 4, 77–84. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-2828-8_8.

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Müller, Alex. "Sexual Orientation, Gender Identity and Cancer in South Africa." In LGBT Populations and Cancer in the Global Context, 35–47. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-06585-9_4.

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Agénor, Madina. "What Are the Numbers? The Epidemiology of Cancer by Sexual Orientation and Gender Identity." In Cancer and the LGBT Community, 117–40. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-15057-4_8.

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Tullio-Pow, S., K. Schaefer, J. Nyhof-Young, and M. Strickfaden. "Sleepwear for Breast Cancer Survivors: Enacting Inclusion Through Feminine Identity and Attachments." In Designing for Inclusion, 23–34. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-43865-4_3.

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Dumitru, Raluca, and Guang Hu. "Maintenance of Human Embryonic Stem Cell Identity and Inhibition of Extraembryonic Differentiation: Role of CNOT1, CNOT2 and CNOT3." In Stem Cells and Cancer Stem Cells, Volume 11, 3–14. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-7329-5_1.

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Sahin, Ugur, Geng Li, Özlem Türeci, and Michael Pfreundschuh. "Recognition of human tumors: SEREX expression cloning to identify tumour antigens." In Cancer Immunology, 45–57. Dordrecht: Springer Netherlands, 2001. http://dx.doi.org/10.1007/978-94-017-0963-7_3.

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Fleck, Leonard M. "Just Caring: Precision Health vs. Ethical Ambiguity: Can we Afford the Ethical and Economic Costs?" In Human Perspectives in Health Sciences and Technology, 205–33. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-92612-0_13.

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AbstractMany see “precision medicine” and “precision health” complementing one another. Precision medicine is critical when we have a life-threatening cancer that could be managed with a targeted cancer therapy. Still, we would rationally prefer whatever medicine might offer that would either prevent the emergence of that cancer or treat it in its earliest stages, the goal of precision health. Dr. Raza, an oncologist, argues in a book titled “The First Cell” that we should abandon paying for targeted therapies for metastatic cancer, and use that money to detect cancer up front using “liquid biopsies” to identify cancer cells in the blood at $500 each, or $100 billion per year. However, I argue that this would be neither a wise nor just use of limited health care resources. Granted, targeted therapies for most patients yield costly marginal gains in life expectancy. Still, we would be sacrificing identified lives for the statistical lives we hoped to save with liquid biopsy tests. We could do Whole Genome Sequencing of the entire population at $3000 per person, looking for the 10% of the population with a heritable cancer, again neither a wise nor just use of limited health care resources. We could fund research to identify biomarkers that would identify cancer patients who would be strong responders to targeted therapies and deny these therapies at social expense to moderate responders. The money saved could be used for targeted precision health efforts. What is really fair? Answering this question requires developing processes of fair and inclusive rational democratic deliberation. What choices are our healthy selves willing to pay for and live with if we develop cancer?
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Lambregts, Doenja M. J., and Regina G. H. Beets-Tan. "How Can We Identify Local Relapse?" In Multidisciplinary Management of Rectal Cancer, 113–19. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-43217-5_15.

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Conference papers on the topic "Cancer identity"

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Delattre, Olivier, G. A. Franzetti, K. Laud-Duval, A. Brisac, Valentina Boeva, Caroline Louis-Brennetot, Agathe Peltier, et al. "Abstract IA02: Identity shifts in pediatric cancers." In Abstracts: AACR Special Conference: Pediatric Cancer Research: From Basic Science to the Clinic; December 3-6, 2017; Atlanta, Georgia. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.pedca17-ia02.

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Thong, M., EM Wolschon, L. Koch-Gallenkamp, A. Waldmann, H. Brenner, and V. Arndt. "“Still cancer patient” self-identity is associated with healthcare use among cancer survivors: a population-based study." In Gemeinsam forschen – gemeinsam handeln. Georg Thieme Verlag KG, 2017. http://dx.doi.org/10.1055/s-0037-1605842.

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Nogueira, Ingrid, Amanda Araújo, Maria Tereza Morano, Antonio George Cavalcante, Pedro Felipe de Bruin, Johana Susan Paddison, Clarissa Magalhaes, et al. "Assessment of fatigue using the identity-consequence fatigue scale in patients with lung cancer." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa2498.

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Horst, KC, KE Fero, K. Haimovitz, and CS Dweck. "Abstract P6-08-10: Cancer as self: a novel assessment of patient identity as it relates to a cancer diagnosis." In Abstracts: Thirty-Fifth Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 4‐8, 2012; San Antonio, TX. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/0008-5472.sabcs12-p6-08-10.

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Bond, David J., and John D. Lewis. "Abstract 5070: CD44 alternative splice variants are associated with prostate cancer cell identity and migration." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-5070.

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Beck, Tim N., Vladislav Korobeynikov, Alexander Kudinov, Rachel Georgeopoulos, Emmanuelle Nicolas, Margret B. Einarson, Yan Zhou, et al. "Abstract B25: Anti-Mullerian hormone (AMH) supports epithelial identity and survival signaling in lung cancer." In Abstracts: Fourth AACR International Conference on Frontiers in Basic Cancer Research; October 23-26, 2015; Philadelphia, PA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.fbcr15-b25.

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Furth, Noa, Ioannis Pateras, Ron Rotkopf, Ina Schmitt, Deborah Bakaev, Anat Gershoni, Randy Johnson, Vassilis Gorgoulis, Moshe Oren, and Yael Aylon. "Abstract IA20: LATS1 and LATS2 suppress breast cancer progression by maintaining cell identity and metabolic state." In Abstracts: AACR Special Conference on the Hippo Pathway: Signaling, Cancer, and Beyond; May 8-11, 2019; San Diego, CA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1557-3125.hippo19-ia20.

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Peltier, Agathe, Caroline Louis-Brennetot, Cécile Pierre-Eugène, Cécile Thirant, Simon Durand, Divya Sahu, Sandrine Grossetête-Lalami, et al. "Abstract B78: Role of noradrenergic core regulatory circuitry transcription factors in neuroblastoma cell identity." In Abstracts: AACR Special Conference on the Advances in Pediatric Cancer Research; September 17-20, 2019; Montreal, QC, Canada. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.pedca19-b78.

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Le Magnen, Clémentine, Aditya Dutta, and Cory Abate-Shen. "Abstract 88: The Nkx3.1 homeobox gene maintains prostatic identity while its loss leads to prostate cancer initiation." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-88.

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Teteh, Dede K., Marissa Ericson, Eudora Mitchell, Phyllis Clark, Rick Kittles, and Susanne Montgomery. "Abstract A044: Black identity, hair product use, and breast cancer: Exploring breast health issues in Black women." In Abstracts: Eleventh AACR Conference on The Science of Cancer Health Disparities in Racial/Ethnic Minorities and the Medically Underserved; November 2-5, 2018; New Orleans, LA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7755.disp18-a044.

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Reports on the topic "Cancer identity"

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Freire, Mariana, Diana Martins, Maria Filomena Botelho, and Fernando Mendes. Biomarkers of resistance mechanisms in innovative lung cancer treatments - A systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2022. http://dx.doi.org/10.37766/inplasy2022.9.0011.

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Review question / Objective: This systematic review aims to provide an overview of the immunotherapy resistance mechanisms and identify potential biomarkers associated with immunotherapy response in NSCLC, as well as examine new treatment options to overcome this hurdle. Condition being studied: Lung Cancer (LC) remains one of the leading cancers worldwide. In 2020, were globally estimated 2 206 771 new cases and 1 796 144 deaths, representing the uttermost frequent cause of cancer death. LC is classified histologically into small cell lung cancer (SCLC) or non-small cell lung cancer (NSCLC), being the last one the most common, representing 80 to 85% of all LC. The three predominantly subtypes of NSCLC are lung adenocarcinoma (LUAD), lung squamous cell carcinoma (LUSC) and large cell carcinoma (LCLC). NSCLC is usually diagnosed in advanced-staged disease due to ambiguous and delayed severe symptoms.
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Esteva, Francisco J. Proteomic Analysis to Identify Novel Circulating Breast Cancer Markers. Fort Belvoir, VA: Defense Technical Information Center, June 2004. http://dx.doi.org/10.21236/ada426268.

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Esteva, Francisco J. Proteomic Analysis to Identify Novel Circulating Breast Cancer Markers. Fort Belvoir, VA: Defense Technical Information Center, June 2006. http://dx.doi.org/10.21236/ada467768.

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Wang, Yan, Wenpeng Song, Sicheng Zhou, Jie Tian, Yingxian Dong, Jue Li, Junke Chang, et al. Increased risk for subsequent primary lung cancer among female hormone-related cancer patients: a meta-analysis based on over four million cases. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, July 2022. http://dx.doi.org/10.37766/inplasy2022.7.0044.

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Review question / Objective: To identify the risk of lung cancer in FHRC patients compared to the general population. Condition being studied: The incidence rate of lung cancer in women is obviously increasing over the past decade and previous evidence have indicated the significant relationship between disturbances in hormone levels and the risk of lung cancer. Therefore, we hypothesized female hormone-related cancer (FHRC), including the breast, endometrial, cervix, and ovary cancer, patients may experience a higher risk of developing subsequent lung cancer.
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Patankar, Manish S. Mining the Immune Cell Proteome to Identify Ovarian Cancer-Specific Biomarkers. Fort Belvoir, VA: Defense Technical Information Center, March 2012. http://dx.doi.org/10.21236/ada564144.

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6

Patankar, Manish S. Mining the Immune Cell Proteome to Identify Ovarian Cancer-Specific Biomarkers. Fort Belvoir, VA: Defense Technical Information Center, November 2013. http://dx.doi.org/10.21236/ada594703.

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7

Li, Zhenqi, Guangfu Zhang, Jia Liu, and Xiaolin Li. Risk factors for gallbladder Cancer:A meta-analysis based on nearly a decade of research. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2022. http://dx.doi.org/10.37766/inplasy2022.4.0065.

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Abstract:
Review question / Objective: Gallbladder cancer is a rare tumor that is mostly advanced once detected. The efficacy of surgical treatment is still controversial. Therefore, primary prevention of gallbladder cancer is important. There are many studies on risk factors for gallbladder cancer, but at present it is difficult to identify independent risk factors for gallbladder cancer, except for a history of symptomatic chronic cholecystitis and malignant transformation of a single polyp. Laparoscopic cholecystectomy is popular worldwide and can be a preventive procedure for gallbladder cancer in addition to resolving benign lesions. This study makes a meta-analysis of the latest research results exploring the risk factors of gallbladder cancer in the last decade , expecting to provide evidence-based medical support for the prevention of gallbladder cancer at the clinical level, and to provide some ideas to guide the surgical indications for LC and future research related to gallbladder cancer. Subject of study: Gallbladder cancer. Study content: Risk factors. Type of study: case-control or cohort study. Extract the value: OR, HR, RR.
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8

Gelman, Irwin H. High Throughput Screen to Identify Novel Drugs that Inhibit Prostate Cancer Metastasis. Fort Belvoir, VA: Defense Technical Information Center, October 2005. http://dx.doi.org/10.21236/ada454304.

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9

Gelman, Irwin H. High Throughput Screen to Identify Novel Drugs That Inhibit Prostate Cancer Metastasis. Fort Belvoir, VA: Defense Technical Information Center, October 2006. http://dx.doi.org/10.21236/ada462781.

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10

Fuqua, Suzanne A. Fellowship to Identify New Mechanisms of Tamoxifen Resistance in Breast Cancer Patients. Fort Belvoir, VA: Defense Technical Information Center, May 1999. http://dx.doi.org/10.21236/ada383255.

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